Relevant bibliographies by topics / Activated chain end

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Activated chain end'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Activated chain end"

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Zhang, Lei, Fangyuan Zhou, Zhenjiang Li, et al. "Tunable hydantoin and base binary organocatalysts in ring-opening polymerizations." Polymer Chemistry 11, no. 35 (2020): 5669–80. http://dx.doi.org/10.1039/d0py00812e.

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Kalafatis, M., MD Rand, RJ Jenny, YH Ehrlich, and KG Mann. "Phosphorylation of factor Va and factor VIIIa by activated platelets." Blood 81, no. 3 (1993): 704–19. http://dx.doi.org/10.1182/blood.v81.3.704.704.

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Abstract Platelet activation leads to the incorporation of 32[PO4(2-)] into bovine coagulation factor Va and recombinant human factor VIII. In the presence of the soluble fraction from thrombin-activated platelets and (gamma-32P) adenosine triphosphate, radioactivity is incorporated exclusively into the M(r) = 94,000 heavy chain (H94) of factor Va and into the M(r) = 210,000 to 90,000 heavy chains as well into the M(r) = 80,000 light chain of factor VIII. Proteolysis of the purified phosphorylated M(r) = 94,000 factor Va heavy chain by activated protein C (APC) gave products of M(r) = 70,000,
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Kalafatis, M., MD Rand, RJ Jenny, YH Ehrlich, and KG Mann. "Phosphorylation of factor Va and factor VIIIa by activated platelets." Blood 81, no. 3 (1993): 704–19. http://dx.doi.org/10.1182/blood.v81.3.704.bloodjournal813704.

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Platelet activation leads to the incorporation of 32[PO4(2-)] into bovine coagulation factor Va and recombinant human factor VIII. In the presence of the soluble fraction from thrombin-activated platelets and (gamma-32P) adenosine triphosphate, radioactivity is incorporated exclusively into the M(r) = 94,000 heavy chain (H94) of factor Va and into the M(r) = 210,000 to 90,000 heavy chains as well into the M(r) = 80,000 light chain of factor VIII. Proteolysis of the purified phosphorylated M(r) = 94,000 factor Va heavy chain by activated protein C (APC) gave products of M(r) = 70,000, 24,000, a
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Tokar, R., P. Kubisa, S. Penczek, and A. Dworak. "Cationic polymerization of glycidol: coexistence of the activated monomer and active chain end mechanism." Macromolecules 27, no. 2 (1994): 320–22. http://dx.doi.org/10.1021/ma00080a002.

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Rasines Mazo, Alicia, Thi Nga Tran, Wenhao Zhang, et al. "Blue LED light-activated RAFT polymerization of PEG acrylate with high chain-end fidelity for efficient PEGylation." Polymer Chemistry 11, no. 32 (2020): 5238–48. http://dx.doi.org/10.1039/d0py00838a.

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Lee, Wei-Lih, Michelle A. Kaiser, and John A. Cooper. "The offloading model for dynein function." Journal of Cell Biology 168, no. 2 (2005): 201–7. http://dx.doi.org/10.1083/jcb.200407036.

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During mitosis in budding yeast, dynein moves the mitotic spindle into the mother-bud neck. We have proposed an offloading model to explain how dynein works. Dynein is targeted to the dynamic plus end of a cytoplasmic microtubule, offloads to the cortex, becomes anchored and activated, and then pulls on the microtubule. Here, we perform functional studies of dynein intermediate chain (IC) and light intermediate chain (LIC). IC/Pac11 and LIC/Dyn3 are both essential for dynein function, similar to the heavy chain (HC/Dyn1). IC and LIC are targeted to the distal plus ends of dynamic cytoplasmic m
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Bednarek, Melania, Przemysław Kubisa, and Stanisław Penczek. "Coexistence of Activated Monomer and Active Chain End Mechanisms in Cationic Copolymerization of Tetrahydrofuran with Ethylene Oxide." Macromolecules 32, no. 16 (1999): 5257–63. http://dx.doi.org/10.1021/ma9900939.

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Delcroix, Damien, Blanca Martín-Vaca, Didier Bourissou, and Christophe Navarro. "Ring-Opening Polymerization of Trimethylene Carbonate Catalyzed by Methanesulfonic Acid: Activated Monomer versus Active Chain End Mechanisms." Macromolecules 43, no. 21 (2010): 8828–35. http://dx.doi.org/10.1021/ma101461y.

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McLaughlin, J. "Long-chain fatty acid sensing in the gastrointestinal tract." Biochemical Society Transactions 35, no. 5 (2007): 1199–202. http://dx.doi.org/10.1042/bst0351199.

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The gastrointestinal tract actively responds to the presence of nutrients in the lumen, and there is a high level of specificity to these responses. This specificity exists for different nutrient types and anatomical regions, with physiologically appropriate target end-organ responses. This review will journey from outlining the effects of dietary fat on digestive function and feeding behaviour to the evidence for lipid sensory cells, particularly the enteroendocrine system, in the epithelium. It will also outline potential molecular bases for these mechanisms. This will deal exclusively with
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Sangermano, Marco, Mehmet Atilla Tasdelen, and Yusuf Yagci. "Photoinitiated curing of mono- and bifunctional epoxides by combination of active chain end and activated monomer cationic polymerization methods." Journal of Polymer Science Part A: Polymer Chemistry 45, no. 21 (2007): 4914–20. http://dx.doi.org/10.1002/pola.22239.

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Dissertations / Theses on the topic "Activated chain end"

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Pasquet, Cédric. "Disproportionation and ring-opening polymerization of silmethylene-siloxane derivatives." Thesis, Lyon, INSA, 2013. http://www.theses.fr/2013ISAL0072.

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Les poly(silméthylènesiloxane)s, aussi dénommés silicones hybrides, sont constitués d’un squelette portant un groupement méthylène entre 2 unités SiOSi. La synthèse de ces polymères par polycondensation ne permet pas d’atteindre des masses molaires très élevées, contrairement aux produits issus de la polymérisation par ouverture de cycle. Ce manuscrit décrit la procédure choisie pour aboutir à une synthèse contrôlée de poly(silméthylènesiloxane)s, des précurseurs (monomères, amorceurs) aux polymères. Le monomère cyclique silméthylène a été préparé par cyclisation de le 1,3-dichlorotetraméthyls
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Book chapters on the topic "Activated chain end"

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Fajmut, Aleš. "Molecular Mechanisms and Targets of Cyclic Guanosine Monophosphate (cGMP) in Vascular Smooth Muscles." In Muscle Cell and Tissue - Novel Molecular Targets and Current Advances [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.97708.

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Molecular mechanisms and targets of cyclic guanosine monophosphate (cGMP) accounting for vascular smooth muscles (VSM) contractility are reviewed. Mathematical models of five published mechanisms are presented, and four novel mechanisms are proposed. cGMP, which is primarily produced by the nitric oxide (NO) dependent soluble guanylate cyclase (sGC), activates cGMP-dependent protein kinase (PKG). The NO/cGMP/PKG signaling pathway targets are the mechanisms that regulate cytosolic calcium ([Ca2+]i) signaling and those implicated in the Ca2+-desensitization of the contractile apparatus. In addition to previous mathematical models of cGMP-mediated molecular mechanisms targeting [Ca2+]i regulation, such as large-conductance Ca2+-activated K+ channels (BKCa), Ca2+-dependent Cl− channels (ClCa), Na+/Ca2+ exchanger (NCX), Na+/K+/Cl− cotransport (NKCC), and Na+/K+-ATPase (NKA), other four novel mechanisms are proposed here based on the existing but perhaps overlooked experimental results. These are the effects of cGMP on the sarco−/endo- plasmic reticulum Ca2+-ATPase (SERCA), the plasma membrane Ca2+-ATPase (PMCA), the inositol 1,4,5-trisphosphate (IP3) receptor channels type 1 (IP3R1), and on the myosin light chain phosphatase (MLCP), which is implicated in the Ca2+-desensitization. Different modeling approaches are presented and discussed, and novel model descriptions are proposed.
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Conference papers on the topic "Activated chain end"

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Strachan, B. Scott, and Steven Shaw. "Subharmonic Resonance Cascades in a Class of Coupled Resonators." In ASME 2011 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2011. http://dx.doi.org/10.1115/detc2011-48599.

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We consider a chain of N nonlinear resonators with natural frequency ratios of approximately 2:1 along the chain and weak nonlinear coupling of a form that allows energy to flow between resonators. Specifically, the coupling is such that the response of one resonator parametrically excites the next resonator in the chain, and also creates a resonant backaction on the previous resonator in the chain. This class of systems, which is being proposed for micro-electro-mechanical frequency dividers, is shown to have rich dynamical behavior. Of particular interest is the case when the high frequency
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Shafer, Michael W., Heidi P. Feigenbaum, and Diego Ricardo Higueras Ruiz. "A Novel Biomimetic Torsional Actuator Design Using Twisted Polymer Actuators." In ASME 2017 Conference on Smart Materials, Adaptive Structures and Intelligent Systems. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/smasis2017-3803.

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Artificial muscle systems have the potential to impact many technologies ranging from advanced prosthesis to miniature robotics. Recently, it has been shown that twisting drawn polymer monofilaments, such as nylon fishing line or sewing thread, can result in a biomimetic thermally activated torsional actuator. The actuation phenomenon in these twisted polymer actuators (TPAs) is thought to be a result of an untwisting that occurs about the fiber’s axis due to an anisotropic thermal expansion. Before being twisted, the precursor fibers are comprised of polymer chains that are aligned axially. D
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Omar, M. N., C. D. Lee, and K. G. Mann. "INACTIVATION OF FACTOR Va BY PIASMIN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643883.

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The inactivation of Factor Va by plasmin was studied in the presence and absence of phospholipid vesicles and calcium ions. The action of plasmin resulted in a rapid loss of the ability of Factor Va to serve as a cofactor to Factor Xa , as judged by clotting assays and . direct assays of prothrombin activation using the fluorophore, dansylarginine N-(3-ethyl-1,5-pentanediyl) amide (DAPA) . The rate of Factor Va inactivation catalyzed by plasmin was markedly enhanced by the addition of phospholipid vesicles (PCPS), suggesting that the action of plasmin on Factor Va may be a membrane bound pheno
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Kooistra, T., J. A. van den Berg, H. A. M. Tüns, G. Platenburg, D. Rijken, and E. A. van den Berg. "BUTYRATE SPECIFICALLY STIMULATES TISSUE-TYPE PLASMINOGEN ACTIVATOR SYNTHESIS IN CULTURED HUMAN ENDOTHELIAL CELLS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644656.

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In a search for compounds that can enhance tissue-type plasminogen activator (t-PA) synthesis in cultured human endothelial cells we found that dibutyryl cyclic AMP (at a concentration of 2 mM) led to a several-fold increase in t-PA production by endothelial cells over a 24 h incubation period. Further researchshowed that this stimulating effectcould be explained by the slow liberation of butyrate, as the effect could be reproduced by addition of free butyrate to the medium, but notby addition of 8-bromo cyclic AMP. With butyrate, an accelerated accumulation of t-PA antigen in the conditioned
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Nguyen, G., L. Bara, M. Samama, et al. "COMPARISON OF SINGLE-CHAIN AND TWO-CHAIN RECOMBINANT TISSUE-PLAS-MINOGEN ACTIVATOR (t-PA) IN VITRO. TURNOVER OF SINGLE-CHAIN t-PA IN ANIMALS AND MAN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644409.

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Two preparations of recombinant t-PA obtained from mammalian cells (Boehringer Ingelheim) containing mainly single-chain forms t-PA (sct-PA > 95%) or two-chain forms t-PA (tct-PA > 70%) were compared in vitro for their thrombolytic and fibrinogeno-lytic properties.Thrombolytic activities studied on human hanging clots were similar for both preparations, at plasma concentrations ranging from 62.5 to 1,000 IU/ml. Fibrinogenolysis appeared for both preparations at concentrations higher than 500 IU/ml, after a 90 min incubation at 37°C. For higher concentrations, fibrinogenolysis was dose-de
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Lu, X. Lucas, Bo Huo, Andrew D. Baik, and X. Edward Guo. "Intercellular Calcium Wave Propagation in Linear and Circuit-Like Bone Cell Networks." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19365.

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Intracellular calcium ([Ca2+]i) transients in response to mechanical stimulation can be propagated to neighboring cells in bone cell networks, which provides an essential mechanism for cell-cell communication in bone. Transfer of intracellular second messengers (e.g., IP3 and Ca2+) through gap junction pores and the diffusion of extracellular ATP to activate membrane receptors have long been conjectured as the two major pathways for intercellular Ca2+ wave propagation [1]. In this study, by comparing the calcium wave in open-end linear and looped circuit-like cell chains, the roles of gap junc
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Schmitz-Huebner, U., H. Ostermann, D. G. Mathey, J. Schofer, Ch Diefenbach, and R. Erbel. "INFLUENCE OF RECOMBINANT PRO-UROKINASE ON THE HEMOSTATIC SYSTEM IN PATIENTS WITH ACUTE MYOCARDIAL INFARCTION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643572.

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Recombinant unglycosylated pro-urokinase (recombinant single chain urokinase-type plasminogen activator, CG4509) was given to twelve patients (pts.) with acute myocardial infarction as a 20 mg bolus followed by a 60 mg intravenous infusion (iv.inf.) over 1 hour and to twelve pts. as a 10 mg bolus followed by a 30 mg iv.inf. over 1 hour. Reperfusion was angiographically confirmed in 9/12 pts. with the higher dose and in 6/12 pts. who obtained the lower dose. Different parameters of hemostasis were determined before administration, 30 min after the beginning of inf.,at the end of inf., 60 min th
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Stump, D. C., E. J. Topol, R. Califf, A. B. Chen, A. Hopkins, and A. D. Collen. "RESULTS OF COAGULATION - FIBRINOLYSIS ANALYSES IN 386 PATIENTS WITH ACUTE MYOCARDIAL INFARCTION TREATED WITH RECOMBINANT TISSUE-TYPE PLASMINOGEN ACTIVATOR (rt-PA) (TAMI TRIAL)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643746.

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Three hundred eighty-six patients with acute myocardial infarction received up to 150 mg rt-PA (single chain) IV either over 8 h (60mg over 1 h, 20 mg/h for 2 h,10 mg/h for 5 h)(173 pts) or over"5h(1 mg/kg over 1 h, remainder over 4 h) (213 pts),before randomization to early or late angioplasty. Blood was collected on a lyophilized mixture of citrate and the t-PA inhibitor D-Phe-Pro-Arg-CH2C1 (PPACK), to maximally prevent in vitro fibrinolytic activation and concomitant fibrinogen degradation. The plasma rt-PA level increased to 2.4±2.0 /μg/ml(mean +SD)and 1.7 ±1.3 /μg/ml after 3h and to 1.0 ±
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Welzel, D., and H. Wolf. "CLINICAL RESEARCH ON SINGLE-CHAIN UROKINASE-TYPE PLASMINOGEN ACTIVATOR (SCU-PA) IN GERMANY. RESULTS IN PATIENTS WITH ACUTE MYOCARDIAL INFARCTION (AMI)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642959.

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The German multicenter trial on scu-PA so far comprises 63 patients with AMI that received scu-PA intravenously in increasing dosages from 2mio units (15 mg) up to 9mio units (69 mg), combined in a bolus injection and a subsequent infusion over one hour. Another 18 patients received 200.000 IU urokinase combined with scu-PA 6.5mio units/h. All patients were anticoagulated prior to the lytic therapy with a bolus injection of 5000 IU heparin, and subsequently received heparin in a dosage maintaining the aPTT-prolonga-tion at 60 to 80 sec. The application of scu-PA up to 4mio units lead to a reop
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