Academic literature on the topic 'Diffuse parenchymal lung diseases'
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Journal articles on the topic "Diffuse parenchymal lung diseases"
Tomassetti, Sara, Claudia Ravaglia, and Venerino Poletti. "Diffuse parenchymal lung disease." European Respiratory Review 26, no. 144 (April 26, 2017): 170004. http://dx.doi.org/10.1183/16000617.0004-2017.
Full textPoletti, Venerino, Claudia Ravaglia, and Sara Tomassetti. "Transbronchial cryobiopsy in diffuse parenchymal lung diseases." Current Opinion in Pulmonary Medicine 22, no. 3 (May 2016): 289–96. http://dx.doi.org/10.1097/mcp.0000000000000272.
Full textMartinez, Fernando J., and Michael P. Keane. "Update in Diffuse Parenchymal Lung Diseases 2005." American Journal of Respiratory and Critical Care Medicine 173, no. 10 (May 15, 2006): 1066–71. http://dx.doi.org/10.1164/rccm.2601011.
Full textRaghunath, Sushravya, Srinivasan Rajagopalan, Ronald A. Karwoski, Fabien Maldonado, Tobias Peikert, Teng Moua, Jay H. Ryu, Brian J. Bartholmai, and Richard A. Robb. "Quantitative Stratification of Diffuse Parenchymal Lung Diseases." PLoS ONE 9, no. 3 (March 27, 2014): e93229. http://dx.doi.org/10.1371/journal.pone.0093229.
Full textWalsh, Sinead M., and Anthony W. O’Regan. "Diffuse Parenchymal Lung Diseases in the Elderly." Current Geriatrics Reports 7, no. 3 (July 13, 2018): 174–80. http://dx.doi.org/10.1007/s13670-018-0249-x.
Full textShlobin, Oksana A., A. Whitney Brown, and Steven D. Nathan. "Pulmonary Hypertension in Diffuse Parenchymal Lung Diseases." Chest 151, no. 1 (January 2017): 204–14. http://dx.doi.org/10.1016/j.chest.2016.08.002.
Full textFarag, TaghreedS, ZeinabR Adawy, LobnaK Sakr, and HanaaS Abdellateef. "Transthoracic ultrasonographic features of diffuse parenchymal lung diseases." Egyptian Journal of Bronchology 11, no. 3 (2017): 179. http://dx.doi.org/10.4103/ejb.ejb_3_17.
Full textTsangaris, Iraklis, Georgios Tsaknis, Anastasia Anthi, and Stylianos E. Orfanos. "Pulmonary Hypertension in Parenchymal Lung Disease." Pulmonary Medicine 2012 (2012): 1–14. http://dx.doi.org/10.1155/2012/684781.
Full textBraun, Sarah, Marion Ferner, Kai Kronfeld, and Matthias Griese. "Hydroxychloroquine in children with interstitial (diffuse parenchymal) lung diseases." Pediatric Pulmonology 50, no. 4 (December 9, 2014): 410–19. http://dx.doi.org/10.1002/ppul.23133.
Full textPulagam, Ammi Reddy, Giri Babu Kande, Venkata Krishna Rao Ede, and Ramesh Babu Inampudi. "Automated Lung Segmentation from HRCT Scans with Diffuse Parenchymal Lung Diseases." Journal of Digital Imaging 29, no. 4 (March 9, 2016): 507–19. http://dx.doi.org/10.1007/s10278-016-9875-z.
Full textDissertations / Theses on the topic "Diffuse parenchymal lung diseases"
Sharp, Charles Michael Francis. "Clinical outcomes in diffuse parenchymal lung disease." Thesis, University of Bristol, 2017. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.723507.
Full textMerola, Pietro Krauspenhar. "Avaliação funcional da musculatura respiratória e periférica e sua relação com a capacidade de exercício e dispneia em pacientes com pneumonias intersticiais fibrosantes." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2016. http://hdl.handle.net/10183/143039.
Full textBackground: Pectoralis muscle area (PMA) is an easily derived computed tomography-based assessment that can provide insight into clinical features of other skeletal muscles. Respiratory and locomotor muscle dysfunction has been increasingly recognized in patients with interstitial lung disease (ILD). Its contribution to exercise performance has been controversial. We aimed investigate if PMA is related with respiratory and locomotor skeletal muscle strength in ILD patients, and if skeletal muscle function is compromised and independently related with exercise capacity and dyspnea in these patients. Methods: Cross-sectional study where subjects performed incremental cycling cardiopulmonary exercise testing with maximal inspiratory pressure (MIP), maximal expiratory pressure (MEP), and quadriceps maximal voluntary contraction (MVC) before and after exercise.
Jacob, Joseph. "Computer based analysis of diffuse fibrosing lung diseases." Thesis, Imperial College London, 2015. http://hdl.handle.net/10044/1/52803.
Full textAbdallah, Atiyeh Mahmoud. "The genetics of co-stimulation in diffuse (pro-fibrotic) lung diseases." Thesis, Imperial College London, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.439005.
Full textCamargo, Leandro do Nascimento. "O efeito do anti-IL-17 na inflama~ção, remodelamento e estresse oxidativo em modelo experimental de inflamação pulmonar alérgica crônica exacerbado por LPS." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/5/5165/tde-15082018-082634/.
Full textINTRODUCTION: Inflammation plays a central role in the development of asthma, which is considered an allergic disease with a classic Th2 inflammatory profile. However, IL-17 profile cytokines have been studied to better understand their involvement in the pathophysiology of this disease. Severe asthmatic patients have frequent exacerbations, the infectious cause being one of the main triggers and that can perpetuate the inflammatory responses. The Th17 response may be clearly associated with these events. OBJECTIVE: This study evaluates the effects of anti-IL-17 monoclonal antibody therapy on alveolar septa in an experimental model of chronic allergic lung inflammation of asthma exacerbated by LPS. METHODS: Sixty male BALB / c mice were used, being sensitized with intraperitoneal ovalbumin and repeatedly exposed to inhalation with ovalbumin, followed by treatment with or without anti-IL-17. Twenty-four hours before the end of the 29-day experimental protocol, two groups received intratracheal LPS (0.1 mg / ml, OVA-LPS and anti-IL-17 OVA-LPS groups). Afterwards, we evaluated bronchoalveolar lavage fluid (BALF), by morphometry we quantified the recruitment of antigen-presenting cells (FOXP3 and dendritic cells), eosinophils, the cellular expression of proinflammatory cytokines (TNF-alpha, IL-2, IL IL-5, IL-6, IL-13 and IL-17), anti-inflammatory cytokines (IL-10), chemokine (TARC), and quantification of IL6 by RT-PCR. We also evaluated elements of the extracellular matrix (collagen fibers type I and III, MMP-9, MMP-12, TIMP-1, TGF-beta, actin, decorin, lumicam, biglican and fibronectin), alveolar septum edema, oxidative stress through the marker iNOS and 8-iso-PGF2? and the signaling pathways NF-kB and Rho kinase. In addition, we evaluated of the mean alveolar diameter. RESULTS: The animals of the OVA-LPS group presented a potentiation of the all responses compared to OVA (p < 0.05), except for: expression of IL-17, TNFalpha, NF-kB, TIMP-1 and in the volume fraction of type I collagen fibers, decorin, lumicam and actin. Treatment with anti-IL-17 (OVA-anti-IL17 group) attenuated all parameters compared to OVA group (p < 0.05). The animals of the chronic allergic inflammation model exacerbated by LPS and treated with anti-IL-17 (OVA-LPS anti-IL-17 group) had a decreased of the following parameters compared to OVA-LPS: total bronchoalveolar lavage cells number, FOXP3 and dendritic positive cells, CD4 + and CD8 + numbers, of cells positive for IL-10 antiinflammatory, and pro-inflammatory cytokines (TNF-alfa, IL-2, IL-4, IL-5, IL-6, IL- 13 and IL-17); chemokine (TARC), genic expression IL6; edema in the alveolar septum; extracellular matrix elements (volume fraction of collagen fibers type I and III, actin, decorin, biglican, lumicam, fibronectin, and expression of TGF-beta, MMP-9, MMP-12 and TIMP-1 positive cells) of the oxidative stress pathways response (iNOS positive cells and isoprostane volume fraction 8-isoPGF-2alfa) NFkB and Rho kinase 1 and 2 positive cells (p < 0.05). There were no differences in mean alveolar diameter among all experimental groups. CONCLUSION: These data suggest that inhibition of IL-17 may be a promising therapeutic pathway for the treatment of chronic allergic inflammation, even during an exacerbation, and may contribute to the control of Th1 / Th2 / Th17 inflammation, chemokine expression, extracellular matrix remodeling and oxidative stress. Signaling pathways of NF-kB and Rho-kinase are involved in the control of these responses in this model of chronic allergic inflammation exacerbated by LPS
Tarando, Sebastian Roberto. "Quantitative follow-up of pulmonary diseases using deep learning models." Thesis, Evry, Institut national des télécommunications, 2018. http://www.theses.fr/2018TELE0008/document.
Full textInfiltrative lung diseases (ILDs) enclose a large group of irreversible lung disorders which require regular follow-up with computed tomography (CT) imaging. A quantitative assessment is mandatory to establish the (regional) disease progression and/or the therapeutic impact. This implies the development of automated computer-aided diagnosis (CAD) tools for pathological lung tissue segmentation, problem addressed as pixel-based texture classification. Traditionally, such classification relies on a two-dimensional analysis of axial CT images by means of handcrafted features. Recently, the use of deep learning techniques, especially Convolutional Neural Networks (CNNs) for visual tasks, has shown great improvements with respect to handcrafted heuristics-based methods. However, it has been demonstrated the limitations of "classic" CNN architectures when applied to texture-based datasets, due to their inherently higher dimension compared to handwritten digits or other object recognition datasets, implying the need of redesigning the network or enriching the system to learn meaningful textural features from input data. This work addresses an automated quantitative assessment of different disorders based on lung texture classification. The proposed approach exploits a cascade of CNNs (specially redesigned for texture categorization) for a hierarchical classification and a specific preprocessing of input data based on locally connected filtering (applied to the lung images to attenuate the vessel densities while preserving high opacities related to pathologies). The classification targeting the whole lung parenchyma achieves an average of 84% accuracy (75.8% for normal, 90% for emphysema and fibrosis, 81.5% for ground glass)
"Spectrum of diffuse parenchymal lung disease with special reference to idiopathic pulmonary fibrosis: experience at Charlotte Maxeke Johannesburg Academic Hospital." Thesis, 2017. https://hdl.handle.net/10539/25322.
Full textBackground Diffuse parenchymal lung diseases (DPLD) encompass a group of diseases with a wide range of causes with varied presentations and prognosis. The known causes include occupational or environmental exposure, drug induced lung diseases, hypersensitivity pneumonitis and connective tissue disease (CTD). Among the DPLD with unknown cause, idiopathic pulmonary fibrosis (IPF) is the commonest and has the worst outcome. The earlier publications from this country described cryptogenic fibrosing alveolitis. With subsequent characterisation of idiopathic interstitial pneumonia (IIP), and an increase in the burden of IPF reported worldwide, we evaluated the clinical features of patients with IPF in the South African context. Objectives To evaluate the clinical spectrum of DPLD encountered in Johannesburg, South Africa, and to describe the clinical profile of patients with IPF. Methods A retrospective record review of patient files was conducted who attended the Charlotte Maxeke Johannesburg Academic Hospital (CMJAH) Respiratory Clinic in the past 5 years from January 2011 to December 2015. Patients with DPLD were identified and the diagnoses were noted. The records of patients with IPF were further analysed. v Results We identified 132 patients with DPLD. Sarcoidosis (37.8%), IPF (21.2%), connective tissue associated diffuse parenchymal lung disease (CTD-DPLD) (14.3%), and hypersensitivity pneumonitis (HP) (9.8%) were the four most common subtypes. IPF was seen in all racial groups. Of the 28 patients with IPF in our cohort, there was a slight female predominance (1.3:1). The mean age of the patients in our study was 63.8 years and the majority were Whites. Cough (96.4%), dyspnoea (92.8%) and bilateral crackles (96.4%) were the commonest clinical features. The majority of patients (78.5%) were diagnosed by high resolution computerised tomography (HRCT) scan. Conclusion IPF is the second most common DPLD disease encountered after sarcoidosis at the CMJAH. IPF is seen in all racial groups in Johannesburg, South Africa, and the characteristics of patients with IPF are similar to those seen in other parts of the world.
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Κορφιάτης, Παναγιώτης. "Image analysis methods for diagnosis of diffuse lung disease in multi-detector computed tomography." Thesis, 2010. http://nemertes.lis.upatras.gr/jspui/handle/10889/4778.
Full textΗ Διάμεση Νόσος (ΔΝ) του πνεύμονα αποτελεί το 15% των παθήσεων του πνεύμονα που εμφανίζονται στην κλινική πρακτική. Η ΔΝ επηρεάζει κυρίως το πνευμονικό παρέγχυμα και εμφανίζεται στις εικόνες Υπολογιστικής Τομογραφίας (ΥΤ) του πνεύμονα με την μορφή διάχυτων περιοχών χαρακτηριστικών προτύπων υφής που παρεκκλίνουν από αυτό του φυσιολογικού παρεγχύματος. Η Πολυτομική Υπολογιστική Τομογραφία (ΠΥΤ) επιτρέπει την απόκτηση τρισδιάστατων απεικονίσεων με σημαντική μείωση του χρόνου λήψης και αποτελεί την απεικονιστική τεχνική επιλογής για την ποσοτικοποίηση και τη διάγνωση της ΔΝ. Η διάγνωση της ΔΝ χαρακτηρίζεται από μειωμένη διαγνωστική ακρίβεια χαρακτηρισμού και ακρίβεια ποσοτικοποίησης έκτασης ακόμα και για τον έμπειρο ακτινολόγο, αλλά και από χαμηλή επαναληψιμότητα. Η δυσκολία διάγνωσης οφείλεται στη μειωμένη ικανότητα του ανθρώπινου παράγοντα ως προς το καθορισμό έκτασης των προτύπων υφής λόγω ομοιότητας ακτινολογικής εμφάνισης τους σε συνδυασμό με το φόρτο εργασίας του ακτινολόγου και τον αυξημένο όγκο δεδομένων της ΠΥΤ. Αυτοματοποιημένα συστήματα ανάλυσης εικόνας μπορούν να αντιμετωπίσουν τα παραπάνω προβλήματα παρέχοντας σημαντική υποβοήθηση στο έργο της διάγνωσης και παρακολούθησης της νόσου. Η ανάπτυξη αυτοματοποιημένων συστημάτων ανάλυσης εικόνας για υποβοήθηση διάγνωσης στην ΥΤ του πνεύμονα έχει αποτελέσει θέμα εκτεταμένης έρευνας την τελευταία δεκαετία με ένα μικρό τμήμα της να επικεντρώνεται στο χαρακτηρισμό και ποσοτικοποίηση της έκτασης της ΔΝ. Σημαντικά στάδια προεπεξεργασίας των συστημάτων αυτών αποτελούν οι τμηματοποίησεις των Πνευμονικών Πεδίων (ΠΠ) και του αγγειακού δένδρου για τον καθορισμό του προς ανάλυση όγκου του πνευμονικού παρεγχύματος. Τα έως σήμερα προταθέντα συστήματα αυτόματης ανίχνευσης και ποσοτικοποίησης της έκτασης της ΔΝ αξιοποιούν κυρίως μεθόδους ανάλυσης δισδιάστατης (2Δ) υφής εικόνας, ενώ μόνο δύο μελέτες έως σήμερα έχουν αξιοποιήσει ανάλυση 3Δ υφής. Συγκεκριμένα, μέθοδοι ανάλυσης υφής εικόνας που έχουν αξιοποιηθεί είναι: στατιστική 1ης τάξης (ιστόγραμμα), μήτρες συνεμφάνισης αποχρώσεων του γκρι (Grey level Co-occurrence Matrices), μήτρες μήκους διαδρομής απόχρωσης του γκρι (Gray Level Run Length Matrices), υπογραφές ιστογράμματος και Fractals. Ο χαρακτηρισμός και η ποσοτικοποίηση περιοχών του πνευμονικού παρεγχύματος που αντιστοιχούν σε φυσιολογικό παρέγχυμα και υποκατηγορίες παθολογίας υλοποιείται με μεθόδους επιβλεπόμενης ταξινόμησης προτύπων όπως: τεχνητά νευρωνικά δίκτυα (Artificial Neural Networks, ΑΝΝ), Bayesian ταξινομητής, ανάλυση γραμμικού διαχωρισμού ( Linear Discriminant Analysis, LDΑ) και ταξινομητής πλησιέστερου γείτονα (k-Nearest Neighboor, k-NN). Στα έως σήμερα προταθέντα συστήματα, η τμηματοποίηση των ΠΠ υλοποιείται με συμβατικές μεθόδους τμηματοποίησης με βάση τις αποχρώσεις του γκρί (τιμές έντασης) εικονοστοιχείων. Ανοικτό ζήτημα παραμένει και η αξιολόγηση της επίδρασης των σταδίων προ-επεξεργασίας (τμηματοποίηση ΠΠ και αγγειακού δένδρου) στην ακρίβεια συστημάτων χαρακτηρισμού και ποσοτικοποίησης της έκτασης της ΔΝ. Τέλος, η αξιολόγηση της αλληλεπίδρασης αυτόματων συστημάτων ποσοτικοποίησης και ακτινολόγου στη λήψη αποφάσεων χαρακτηρισμού και ποσοτικοποίησης της έκτασης που αφορούν την ΔΝ δεν έχει διερευνηθεί. Η παρούσα διδακτορική διατριβή επικεντρώνεται στην ανάπτυξη ολοκληρωμένου συστήματος ανάλυσης εικόνας το οποίο χαρακτηρίζει και ποσοτικοποιεί την έκταση περιοχών με ΔΝ σε απεικονίσεις ΠΥΤ θώρακος, στοχεύοντας στη βελτιστοποίηση όλων των σταδίων του, καθώς και στην αξιολόγηση της συμβολής του συστήματος στην λήψη διαγνωστικών αποφάσεων. Για το σκοπό αυτό διερευνώνται τεχνικές 3Δ ενίσχυσης εικόνας, 3Δ τμηματοποίησης εικόνας καθώς και 3Δ χαρακτηριστικά υφής εικόνας σε συνδυασμό με επιβλεπόμενα και μη επιβλεπόμενα συστήματα ταξινόμησης. Συγκεκριμένα η συμβολή της παρούσας διατριβής επικεντρώνεται στα ακόλουθα: • Ανάπτυξη μεθόδων τμηματοποίησης των ΠΠ και του αγγειακού δένδρου παρουσία παθολογίας. • Διερεύνηση της συμβολής αλγορίθμων εξαγωγής 3Δ υφής εικόνας στην ακρίβεια μεθόδων ταξινόμησης προτύπων ΔΝ. • Βελτιστοποίηση μεθόδων χαρακτηρισμού και ποσοτικοποίησης έκτασης με χρήση τεχνικών επιβλεπόμενης και μη επιβλεπόμενης ταξινόμησης. • Αξιολόγηση της επίδρασης των σταδίων προεπεξεργασίας στην ακρίβεια συστημάτων ποσοτικοποίησης. • Αξιολόγηση της συμβολής συστημάτων ποσοτικοποίησης στη διάγνωση της ΔΝ.
Books on the topic "Diffuse parenchymal lung diseases"
Poletti, Venerino, ed. Transbronchial cryobiopsy in diffuse parenchymal lung disease. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-14891-1.
Full textMD) Hales Lung Conference (1st 2009 Baltimore. Proceedings of 2009 Hales Lung Conference: Clinical and pathophysiolgic aspects of diffuse parenchymal disease, April 27, 2009, Baltimore, MD : clinical and pathophysiologic aspects of diffuse parenchymal lung disease. Ellicott City, MD: BoBitField, 2011.
Find full textCancellieri, Alessandra, Giorgia Dalpiaz, Mario Maffessanti, Alberto Pesci, Roberta Polverosi, and Maurizio Zompatori. Diffuse Lung Diseases. Edited by Mario Maffessanti and Giorgia Dalpiaz. Milano: Springer Milan, 2006. http://dx.doi.org/10.1007/88-470-0430-6.
Full textGupta, Nishant, Kathryn A. Wikenheiser-Brokamp, and Francis X. McCormack, eds. Diffuse Cystic Lung Diseases. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-63365-3.
Full textDalpiaz, Giorgia, and Alessandra Cancellieri, eds. Atlas of Diffuse Lung Diseases. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-42752-2.
Full textV, Colby Thomas, ed. Surgical pathology of diffuse infiltrative lung disease. Orlando: Grune & Stratton, 1987.
Find full textBush, Andrew, and Jane C. Davies. Paediatric respiratory disease: Parenchymal diseases : an atlas of investigation and management. Oxford, UK: Clinical Pub., 2011.
Find full textCostabel, U., R. M. du Bois, and J. J. Egan, eds. Diffuse Parenchymal Lung Disease. S. Karger AG, 2007. http://dx.doi.org/10.1159/isbn.978-3-318-01377-1.
Full textBook chapters on the topic "Diffuse parenchymal lung diseases"
Bratis, Livia, and Thomas F. Morley. "Diffuse Parenchymal Lung Diseases." In Challenging Cases in Pulmonology, 233–49. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-7098-5_14.
Full textClement, Annick, and Brigitte Fauroux. "Interstitial Lung Diseases in Children." In Diffuse Parenchymal Lung Disease, 323–31. Basel: KARGER, 2007. http://dx.doi.org/10.1159/000102701.
Full textChilosi, Marco, Bruno Murer, and Venerino Poletti. "Diffuse Parenchymal Lung Diseases – Histopathologic Patterns." In Diffuse Parenchymal Lung Disease, 44–57. Basel: KARGER, 2007. http://dx.doi.org/10.1159/000102625.
Full textBehr, Jürgen. "Novel Aspects of Treatment for Interstitial Lung Diseases." In Diffuse Parenchymal Lung Disease, 117–26. Basel: KARGER, 2007. http://dx.doi.org/10.1159/000102632.
Full textCollard, Harold R. "Diseases: Other Entities of the Idiopathic Interstitial Pneumonias." In Diffuse Parenchymal Lung Disease, 175–84. Basel: KARGER, 2007. http://dx.doi.org/10.1159/000102689.
Full textIrani, Sarosh, and Annette Boehler. "Lung Transplantation for Diffuse Parenchymal Lung Disease." In Diffuse Parenchymal Lung Disease, 332–40. Basel: KARGER, 2007. http://dx.doi.org/10.1159/000102702.
Full textPoletti, Venerino, Pier Luigi Zinzani, Sara Tomassetti, and Marco Chilosi. "Lymphoproliferative Lung Disorders." In Diffuse Parenchymal Lung Disease, 307–22. Basel: KARGER, 2007. http://dx.doi.org/10.1159/000102700.
Full textOlson, Amy L., and Marvin I. Schwarz. "Diffuse Alveolar Hemorrhage." In Diffuse Parenchymal Lung Disease, 250–63. Basel: KARGER, 2007. http://dx.doi.org/10.1159/000102695.
Full textMetze, Dieter, Tam Nguyen, Birgit Haack, Alexander K. C. Leung, Noriko Miyake, Naomichi Matsumoto, A. J. Larner, et al. "Diffuse Parenchymal Lung Disease." In Encyclopedia of Molecular Mechanisms of Disease, 533. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_7710.
Full textGomez, Antonio D., and Talmadge E. King Jr. "Classification of Diffuse Parenchymal Lung Disease." In Diffuse Parenchymal Lung Disease, 1–10. Basel: KARGER, 2007. http://dx.doi.org/10.1159/000102577.
Full textConference papers on the topic "Diffuse parenchymal lung diseases"
Ashiq, J., FM Daroowalla, and PS Richman. "Making a Decision on Lung Biopsy in Diffuse Parenchymal Lung Diseases." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a4047.
Full textStrumiliene, Edita, and Vygantas Gruslys. "Complications - based learning curve of transbronchial lung cryobiopsy in diffuse parenchymal lung diseases." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.1191.
Full textOkumus, Gulfer, Berker Ozkan, Zuleyha Bingol, Melike Ulker, Berivan Karatekin, Esen Kiyan, Zeki Kilicaslan, Murat Kara, Dilek Yılmazbayhan, and Alper Toker. "Surgical biopsy results of our patients with diffuse parenchymal lung diseases." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2990.
Full textBehera, Digambar, Sahajal Dhooria, Ritesh Agarwal, Inderpaul Singh Sehgal, Mandeep Garg, Amanjit Bal, and Ashutosh Nath Aggarwal. "Spectrum of diffuse parenchymal lung diseases at a tertiary care center in India." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa875.
Full textCunha Machado, Daniela Patrícia, Gisela Lage, Catarina Marques, Filipa Lima, Inês Marques, Carla Nogueira, Ana Oliveira, et al. "Transbronchial cryobiopsy from two different lobes versus one lobe in diffuse parenchymal lung diseases." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.oa2164.
Full textKoslow, M., E. S. Edell, T. Moua, A. C. Roden, E. S. Yi, P. Decker, and J. H. Ryu. "Bronchoscopic Lung Biopsy for Diffuse Parenchymal Lung Disease: Comparison of Forceps with Cryobiopsy." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4317.
Full textHanta, Ismail, Efraim Güzel, Oya Baydar, Emine Kiliçbagir, and Alper Avci. "A case of Non-Hodgkin lyphoma mimicking diffuse parenchymal lung disease." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa3719.
Full textFedyna, S., W. Bulman, C. Shu, A. Saqi, J. Sonett, and P. Stanifer. "A Clinocopathological Study of Lung Cancer in Patients with Non-Idiopathic Pulmonary Fibrosis (IPF) Diffuse Parenchymal Lung Diseases." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a3367.
Full textMassoptier, Laurent, Avishkar Misra, and Arcot Sowmya. "Automatic lung segmentation in HRCT images with diffuse parenchymal lung disease using graph-cut." In 2009 24th International Conference Image and Vision Computing New Zealand (IVCNZ). IEEE, 2009. http://dx.doi.org/10.1109/ivcnz.2009.5378398.
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