Relevant bibliographies by topics / Early onset age

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Early onset age'

Author: Grafiati
Published: 11 September 2021
Last updated: 7 February 2022

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Journal articles on the topic "Early onset age"

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Mezzich, Ada, Ralph Tarter, Levent Kirisci, Duncan Clark, Oscar Buckstein, and Cynthia Martin. "Subtypes of Early Age Onset Alcoholism." Alcoholism: Clinical and Experimental Research 17, no. 4 (August 1993): 767–70. http://dx.doi.org/10.1111/j.1530-0277.1993.tb00838.x.

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Ferreiro, Ana. "Early-onset myopathies: Entering a new age." Seminars in Cell & Developmental Biology 64 (April 2017): 158–59. http://dx.doi.org/10.1016/j.semcdb.2017.03.002.

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Verdino, V., A. Goracci, S. Bolognesi, A. Di Muro, F. Pieraccini, and A. Fagiolini. "Early age at onset of psychotic symptoms." European Neuropsychopharmacology 29 (2019): S403. http://dx.doi.org/10.1016/j.euroneuro.2018.11.610.

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Jefferies, Kate, and Niruj Agrawal. "Early-onset dementia." Advances in Psychiatric Treatment 15, no. 5 (September 2009): 380–88. http://dx.doi.org/10.1192/apt.bp.107.004572.

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SummaryDementia is is stereotypically associated with older people. However, in a significant minority it can affect people in their 40s and 50s, or even younger. Currently there is a lack of awareness, even among healthcare professionals, and there is a dearth of appropriate services for such patients. Despite the attention given to this condition by National Institute for Health and Clinical Excellence guidelines, provision of specialist early-onset dementia services in the UK remains patchy. Carers and patients often find themselves being passed ‘from pillar to post’ between psychiatry and neurology, and also between adult, old age and liaison psychiatry. The responsibility for identifying available and appropriate help is often left with carers. This leads to unnecessary delays, causes undue distress to patients and places an added burden on carers.
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MANNO, REBECCA L., FREDRICK M. WIGLEY, ALLAN C. GELBER, and LAURA K. HUMMERS. "Late-age Onset Systemic Sclerosis." Journal of Rheumatology 38, no. 7 (June 17, 2011): 1317–25. http://dx.doi.org/10.3899/jrheum.100956.

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Objective.Although patients who develop systemic sclerosis (SSc) later in life (≥ 65 yrs) may express the entire clinical spectrum of disease, we hypothesize that patients with late-age onset SSc incur a different risk for specific organ manifestations of disease compared to those with early-age onset SSc.Methods.In total, 2300 patients with SSc were evaluated between 1990 and 2009 and reviewed from a university-based scleroderma center cohort. Demographic profile, SSc subtype, autoantibody status, Medsger severity scores, pulmonary function tests, echocardiography, and right heart catheterization measures were compared between late-age onset vs younger-age onset patients with SSc.Results.Overall, 2084 patients (91%) developed SSc prior to age 65, while 216 (9%) were ≥ 65 years. Late-age onset patients had a significantly higher proportion of anticentromere antibodies (42% vs 27%; p = 0.001) compared to early-age onset patients. Risk of pulmonary hypertension (OR 1.76, 95% CI 1.00, 3.12), muscle weakness (OR 1.85, 95% CI 1.30, 1.64), renal impairment (OR 2.83, 95% CI 1.98, 4.04), and cardiac disease (OR 2.69, 95% CI 1.92, 3.78) was greater among those with late-age onset SSc; although risk of digital ischemia (OR 0.64, 95% CI 0.47, 0.86) was reduced. The cumulative incidence of pulmonary hypertension at 5 years was greater among those with late-age onset SSc (9%) compared to those with early-age onset SSc (2.7%; log-rank, p < 0.001).Conclusion.These findings suggest that older patients with SSc are at greater risk for pulmonary hypertension, renal impairment, cardiac disease, and muscle weakness. Awareness of the distinct risk for specific organ manifestations in SSc, in particular pulmonary hypertension, should guide the care of patients with SSc whose disease begins after age 65.
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Hughes, Anne E., Weihua Meng, Stephen Bridgett, and Declan T. Bradley. "RareCFHmutations and early-onset age-related macular degeneration." Acta Ophthalmologica 94, no. 3 (August 13, 2015): e247-e248. http://dx.doi.org/10.1111/aos.12822.

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Fragoso, Yara D., and Paulo Christian Machado. "Hemicrania Continua With Onset at an Early Age." Headache: The Journal of Head and Face Pain 38, no. 10 (November 1998): 792–93. http://dx.doi.org/10.1046/j.1526-4610.1998.3810792.x.

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Kumar, Tinni, Suzan G. Mandla, and Wenda L. Greer. "Familial myelodysplastic syndrome with early age of onset." American Journal of Hematology 64, no. 1 (May 2000): 53–58. http://dx.doi.org/10.1002/(sici)1096-8652(200005)64:1<53::aid-ajh9>3.0.co;2-9.

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Sharman, Steve, Raegan Murphy, John Turner, and Amanda Roberts. "Psychosocial correlates in treatment seeking gamblers: Differences in early age onset gamblers vs later age onset gamblers." Addictive Behaviors 97 (October 2019): 20–26. http://dx.doi.org/10.1016/j.addbeh.2019.05.013.

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Levy-Lahad, Ephrat, Amnon Lahad, Ellen M. Wijsman, Thomas D. Bird, and Gerard D. Schellenberg. "Apolipoprotein E genotypes and age of onset in early-onset familial Alzheimer's disease." Annals of Neurology 38, no. 4 (October 1995): 678–80. http://dx.doi.org/10.1002/ana.410380420.

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Dissertations / Theses on the topic "Early onset age"

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Owens, Ann M. "Culture, age, gender factors in the early onset of eating disorders /." Instructions for remote access. Click here to access this electronic resource. Access available to Kutztown University faculty, staff, and students only, 1994. http://www.kutztown.edu/library/services/remote_access.asp.

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Gulledge, Laura M. "GENDER DIFFERENCES IN AGE OF ONSET FOR DELINQUENCY:RISK FACTORS AND CONSEQUENCES." Scholar Commons, 2006. http://scholarcommons.usf.edu/etd/3828.

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The age of onset of delinquency has long been viewed as a primary indicator for further delinquency and criminality. However, studies on the risk factors for onset, and future delinquency have focused predominantly on males. The purpose of this study was to explore gender differences and similarities in risk factors for onset and frequency of arrest. The data used in these analyses were from a longitudinal study, Pathways to Adulthood: A Three Generational Urban Study, 1960-1994. Sixty-six percent (N=1,758) of the eligible children completed the final survey. Of these children, only 515 were used in this particular study because they had documented ages of first arrest. It is hypothesized that 1) female "early" onset occurs at a later age from that of male "early" onset, 2) risk factors predictive of early onset will differ across gender, and 3) "early" onset in females will be predictive of frequency of subsequent arrests.With these data, the author uses OLS regression, logistic regression, and negative binomial regression to evaluate these hypotheses regarding age of onset, risk factors for onset, and frequency of arrest. Insufficient evidence was found to support the hypotheses of the current study. A discussion of the findings, as well as implications and calls for future research are discussed
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Malek, Naveed. "Variation in Parkinson's disease : age, gender, genotype and phenotype correlations in early onset disease." Thesis, University of Glasgow, 2014. http://theses.gla.ac.uk/5602/.

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There is a wide variation in the phenotypic expression, progression rates, therapy response and complications in Parkinson’s disease (PD). The primary research objective in this thesis was to analyse the variation in the 4 domains of phenotypic expression i.e. motor, non-motor, cognitive, and quality of life in a subset of early onset Parkinson’s disease (EOPD) patients from the PRoBaND study, in the United Kingdom. The secondary objective was to explore the factors responsible for this variation or heterogeneity in the clinical characteristics. Linking genotypes with phenotypes, besides evaluating environmental risk factors and iatrogenic influences, represents one mechanism of understanding this variation in the phenotypic expression of PD. We found subtle but significant variation across all domains of symptoms examined in this thesis by classifying patients into groups according to motor subtype, gender, age at diagnosis and heritability of the parkinsonian trait, despite statistically insignificant differences in risk factors such as head trauma, exposure to pesticides (including herbicides, insecticides, fungicides and fumigants), heavy metals, caffeine and a past history of oophorectomy (in females) with the exception of smoking (p=0.046) and exposure to solvents, which were more common in males compared to females (p<0.001). There were differences in the prevalence of motor symptoms such as balance problems being more prevalent in the postural instability gait difficulty (PIGD) subtype compared to the tremor dominant PD (TDPD) and ‘Mixed’ motor subtypes both subjectively (p<0.001) and objectively (p<0.001). Other axial problems such as speech difficulties and freezing were also more prevalent in those with the PIGD phenotype compared to the other motor subtypes both subjectively (p=0.004, p<0.001) and objectively (p=0.002, p<0.001). There was also variation in the prevalence of motor complications such as dyskinesia (p<0.001) and dystonia (p=0.020), being more prevalent in the PIGD subtype compared to other motor subtypes. 8 The prevalence of certain non-motor symptoms such as pain (p=0.022) and features of gastrointestinal dysfunction e.g. prandial bloating (p=0.024) and constipation (p=0.022) were more commonly reported by females compared to males. There were also differences in the prevalence of cognitive impairment (p=0.049) and neurobehavioural characteristics such as anxiety (p=0.002) and depression (p=0.006), after the diagnosis of PD, being more prevalent in PIGD compared to other motor subtypes. Finally, these differences contributed to the variation in the independence of activities of daily living scores which were lower in those with the PIGD phenotype compared to other motor subtypes (p<0.001). There were some differences in exposure to environmental risk factors for PD but not sufficient to explain all the variation. Iatrogenic influences from drugs contributed in part to the phenotypic variation. 10% of the cases in the EOPD cohort tested positive for mutations in one of three genes screened i.e. LRRK2, GBA and Parkin; their DNA remains banked and there is scope to test these cases for mutations in other genes, relevant to PD, in the future. There were too small numbers of cases in each subgroup to draw definite conclusions about the exact influence of genes on the overall phenotypic variation but differences between Parkin mutation carriers and gene test negative ‘controls’ such as early age of onset and long disease duration were obvious. PRoBaND is linked to other similar research studies in the UK, with the stated aim of sharing datasets, in the hope that larger numbers of patients and their DNA samples will increase the power, in statistical terms, to test hypotheses about the role of genetic markers in influencing the course and expression of symptoms. Our current understanding of PD as a complex trait suggests both genetic and environmental influences (including iatrogenic factors if patients are treated) play a role in the phenotypic expression of this condition. A lot more remains to be explored to improve our understanding of the finer details and molecular mechanisms underlying the variation in this disease.
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Becker, Miriam Mimi. "A Retrospective Study Between The Relationships Of Gender, Age Of Onset, And Frequency Of Problematic Behaviors In Early Onset Bipolar Disorder." Cleveland State University / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=csu1213290555.

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Morgan, Erin A. "The impact of school and parent attachment on rural adolescents' age at first intercourse: A comparison of contexts." Thesis, Virginia Tech, 2002. http://hdl.handle.net/10919/33565.

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This cross-sectional survey study investigates the relationship between school attachment (SA) and adolescentsâ age at first intercourse (DV), as well as the influence of SA on DV in comparison to the influences of parent attachment (PA), other parent and school factors, and individual factors. Early first intercourse is defined as prior to age 15. Participants are 1,757 mostly African-American and White 7th through 12th grade adolescent boys and girls in five rural counties of a Mid-Atlantic state. Bivariate correlations comparing SA and PA revealed significant and positive correlations between SA and DV (p<.001), as well as PA and DV (p<.001). Linear regressions including only SA and PA showed SA was most predictive of DV for adolescents reporting the lowest (p<.05) and highest (p<.001) levels of PA. For those reporting moderate attachment to parents, SA was not predictive of DV. Several ethnic and gender differences are discussed. Finally, when the influence of individual, parent, and school contexts was compared using entry in a regression by blocks, SA was no longer a significant predictor of DV, and school variables did not account for a significant portion in the variance of age at first intercourse. Parent attachment was a significant and negative predictor, indicating that other parent, individual, and community variables are more influential. Implications are discussed.
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Ahuja, Manik, Manul Awasthi, Kathie Records, and Rabindra Raj Lamichhane. "Early Age of Alcohol Initiation and its Association with Suicidal Behaviors." Digital Commons @ East Tennessee State University, 2021. https://dc.etsu.edu/etsu-works/8842.

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Objective: The relationship between alcohol use and suicidal behaviors is well-accepted, but less is known about the contribution of its early initiation. This study was designed to test the association of early alcohol initiation versus later initiation with suicidal ideation and attempt in an ethnically diverse sample. Methods: The Collaborative Psychiatric Epidemiology Surveys (CPES), 2001-2003 (n = 20,013), database was used. A total of 13,867 participants were selected included 56.9% females and 43.1% males. Race and ethnicity were reported as 28.8% non-Hispanic White, 39.1% Black, 20.3% Latino, and 11.9% Asian. Logistic regression analyses tested the associations between early (< =14 years) and later (> =15) age alcohol initiation with suicide ideation and attempts. Alcohol initiation was indexed by self-report of the first time that any alcohol product was consumed. Potential confounders were controlled. Results: Early alcohol initiation was associated with higher odds (AOR = 3.64, 95% CI [2.51, 5.28]) of suicide ideation as compared with adults who had initiated > = age 15 (AOR = 2.11, 95% CI [1.46, 3.04]). Early age initiation was also associated with higher odds (AOR = 3.81, 95% CI [2.02, 7.18]) of lifetime suicide attempt versus later age initiators (AOR = 2.03, 95% CI [1.08, 3.79]). Significant differences were found between early and later age of initiation. Conclusion: Early age of alcohol initiation has profoundly increased odds of suicide ideation or attempt. It is critical that effective prevention programs for children and their caregivers be implemented to prevent or delay alcohol initiation and lessen the risk for future suicidal behaviors.
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Osazee, Osarueme J., Jodi L. Southerland, Shimin Zheng, Megan Quinn, Yan Cao, Deborah L. Slawson, and Lori Paisley. "Early Age of Onset of Tobacco, Alcohol, and Marijuana Use Among Middle School Youth in Tennessee: Does Place Matter?" Digital Commons @ East Tennessee State University, 2015. https://dc.etsu.edu/etsu-works/101.

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Introduction. Among U.S. adolescents (12-17 years), tobacco, alcohol and marijuana are commonly used substances. Recent data suggests that although trends in tobacco use have declined, marijuana and alcohol use rates are steadily increasing, especially among younger adolescents. The main purpose of this study was to characterize differences in tobacco, alcohol and marijuana onset among Appalachian and nonAppalachian middle school students in Tennessee. At present, there is limited research on tobacco and drug use among younger adolescents in Tennessee and Appalachia as a whole. Therefore, these findings have important implications for the assessment and prevention of risk behaviors among adolescents regionally and may help to establish priorities for policy and preventive measures. Methods. This is a secondary data analysis of the Youth Risk Behavior Survey (YRBS), consisting of data collected in 2010 from a representative sample (n=65,182) of middle schoolers in Tennessee. All analyses were performed on weighted data so that results represent all middle school students in Tennessee. Primary outcome variables included early age of onset (age) of alcohol, tobacco, and marijuana use. The primary exposure variable was region (Appalachia versus non-Appalachia). Multiple logistic regression was used to determine the association between early age of onset of cigarette, alcohol, and marijuana use, and region controlling for personal characteristics, other substance use, suicidal behaviors, body mass index, weight misperception and extreme weight control behaviors. Odds ratios (ORs) and corresponding 95% confidence intervals (CIs) were reported. Results. Early initiators of cigarette, alcohol or marijuana had a two to four fold increase risk for experimentation with other substances compared to late age of onset, with cigarette use posing the highest risk [OR: 4.73, C.I. (4.47, 5.01)]. Middle schoolers in Appalachia were at greater risk for early age of onset of cigarette [OR: 1.502, C.I. (1.421, 1.587)] and marijuana use [OR: 1.268, C.I. (1.169, 1,375)] compared to non-Appalachia middle schoolers, and 80.9% less likely to use alcohol prior to age 12 [OR: 0.809, C.I. (0.777, 0.843). Other differences were observed for risk of early onset of use and gender, race/ethnicity, age, other substance use and EWCB. Conclusion. Differences in the pattern of substance use were observed for middle schoolers in Appalachia and non-Appalachia Tennessee. Better understanding of these differences will help inform public health policy and practice targeting cigarette, alcohol and marijuana use in the region.
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Innala, Lena. "Early rheumatoid arthritis aspects of severity and co-morbidity." Doctoral thesis, Umeå universitet, Reumatologi, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-88477.

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Background Rheumatoid arthritis (RA) is a systemic progressive destructive joint disease with an increased risk for co-morbidity and premature death if untreated. Cardiovascular disease (CVD) is the main cause of death but also other co-morbid conditions contribute to the patient’s shorter life expectancy. Inflammation is important for the development of CVD, but knowledge of its relationship with other co-morbidities is sparse. Early disease modifying anti rheumatic drugs (DMARDs) can suppress disease activity and improve the long-term outcome. The aim of this thesis was to evaluate prospectively aspects of disease activity and severity in a large cohort of patients with early RA. Predictive and prognostic markers, e.g., antibodies against citrullinated proteins/peptides (ACPAs), occurring in early disease and with implications for disease outcome and co-morbidity were evaluated. Methods Patients with early RA (i.e., symptomatic for ≤12 months) have, since December 1995, been consecutively included in a large survey of prospective and observational studies on the progression of RA and the development of co-omorbidity. Autoantibodies, inflammatory, genetic markers and radiographs have been analyzed. In paper I, 210 RA patients and 102 controls were followed regularly for two years. The predictive value of four different ACPAs in relation to disease activity and radiological progression was evaluated. In Paper II (n = 700) and in Papers III-IV (n =950), patients with early RA from the four northern-most counties of Sweden were followed regularly for 5 years. Data on risk factors and co-morbidity was collected, according to the study protocol, from clinical records and self-reported questionnaires from patients at inclusion into the study cohort and after five years. The predictive value of traditional and potential disease related risk factors for new cardiovascular events (CVE) was evaluated (II). In Paper III, the impact of age at the onset RA, stratified as being young onset RA (<58 years; YORA) and late onset RA (≥58 years; LORA) on disease activity, severity and chosen treatment, was evaluated. In Paper IV, the development of new co-morbidities after RA onset and their relation to inflammatory activity was assessed. Results The presence of anti-mutated citrullinated vimentin (MCV ) antibodies was associated with a more severe disease course, estimated by disease activity score, erythrocyte sedimentation rate (ESR) and swollen joint count after 24 months, compared with anti-CCP2, anti-CCP3, and anti CCP3.1 antibodies. In Paper II, the incidence of a new CVE during 5 years was explained by several of the traditional CV risk factors, and potentiated by a high disease activity. Treatment with DMARDs decreased the risk. In Paper III, LORA patients were associated with greater disease activity/severity at disease onset and over time compared with YORA who were more often ACPA positive. YORA patients were treated earlier with DMARDs, whilst LORA patients were more often treated with corticosteroids and less so with DMARDs early in the course of their disease. In Paper IV, 53%of patients already had one or more co-morbidities already at the onset of RA. After 5 years, 41% of the patients had developed at least one new co-morbidity. ESR at baseline and accumulated disease activity were associated with a new co-morbidity after five years. Conclusion Early RA patients sero-positive for anti- MCV antibodies appeared to have a higher disease activity over time. The occurrence of a new CVE in early RA patients was predicted by traditional risk factors for CVD which were potentiated by a high disease activity. Treatment with DMARDs decreased the risk. Patients with young onset of RA were associated with a higher frequency of ACPA. Late onset of RA was associated with higher disease activity/severity at inclusion and over time. However, LORA patients were more often treated with corticosteroids and less so with DMARDs early in the disease course. Development of a new co-morbidity during the five years following diagnosis was related to ESR.
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Behrendt, Silke, Hans-Ulrich Wittchen, Michael Höfler, Roselind Lieb, and Katja Beesdo. "Transitions from first substance use to substance use disorders in adolescence: Is early onset associated with a rapid escalation?" Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2013. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-110746.

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Background: Early substance use (SU) in adolescence is known to be associated with an elevated risk of developing substance use disorders (SUD); it remains unclear though whether early SU is associated with more rapid transitions to SUD. Objective: To examine the risk and speed of transition from first SU (alcohol, nicotine, cannabis) to SUD as a function of age of first use. Methods: N = 3021 community subjects aged 14–24 years at baseline were followed-up prospectively over 10-years. SU and SUD were assessed using the DSM-IV/M-CIDI. Results: (1) The conditional probability of substance-specific SU-SUD transition was the greatest for nicotine (36.0%) and the least for cannabis (18.3% for abuse, 6.2% for dependence) with alcohol in between (25.3% for abuse; 11.2% for dependence). (2) In addition to confirming early SU as a risk factor for SUD we find: (3) higher age of onset of any SU to be associated with faster transitions to SUD, except for cannabis dependence. (4) Transitions from first cannabis use (CU) to cannabis use disorders (CUD) occurred faster than for alcohol and nicotine. (5) Use of other substances co-occurred with risk and speed of transitions to specific SUDs. Conclusion: Type of substance and concurrent use of other drugs are of importance for the association between age of first use and the speed of transitions to substance use disorders. Given that further research will identify moderators and mediators affecting these differential associations, these findings may have important implications for designing early and targeted interventions to prevent disorder progression.
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Behrendt, Silke, Hans-Ulrich Wittchen, Michael Höfler, Roselind Lieb, and Katja Beesdo. "Transitions from first substance use to substance use disorders in adolescence: Is early onset associated with a rapid escalation?" Technische Universität Dresden, 2009. https://tud.qucosa.de/id/qucosa%3A26833.

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Background: Early substance use (SU) in adolescence is known to be associated with an elevated risk of developing substance use disorders (SUD); it remains unclear though whether early SU is associated with more rapid transitions to SUD. Objective: To examine the risk and speed of transition from first SU (alcohol, nicotine, cannabis) to SUD as a function of age of first use. Methods: N = 3021 community subjects aged 14–24 years at baseline were followed-up prospectively over 10-years. SU and SUD were assessed using the DSM-IV/M-CIDI. Results: (1) The conditional probability of substance-specific SU-SUD transition was the greatest for nicotine (36.0%) and the least for cannabis (18.3% for abuse, 6.2% for dependence) with alcohol in between (25.3% for abuse; 11.2% for dependence). (2) In addition to confirming early SU as a risk factor for SUD we find: (3) higher age of onset of any SU to be associated with faster transitions to SUD, except for cannabis dependence. (4) Transitions from first cannabis use (CU) to cannabis use disorders (CUD) occurred faster than for alcohol and nicotine. (5) Use of other substances co-occurred with risk and speed of transitions to specific SUDs. Conclusion: Type of substance and concurrent use of other drugs are of importance for the association between age of first use and the speed of transitions to substance use disorders. Given that further research will identify moderators and mediators affecting these differential associations, these findings may have important implications for designing early and targeted interventions to prevent disorder progression.
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Books on the topic "Early onset age"

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Kuna, Martin. Počátky raného středověku v Čechách: Archeologický výzkum sídelní aglomerace kultury pražského typu v Roztokách = The onset of the early middle ages in Bohemia : archaeological research at a large settlement site of the Prague-type culture at Roztoky. Praha: Archeologický ústav AV ČR, 2005.

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Kuna, Martin. Počátky raného středověku v Čechách: Archeologický výzkum sídelní aglomerace kultury pražského typu v Roztokách = The onset of the early middle ages in Bohemia : archaeological research at a large settlement site of the Prague-type culture at Roztoky. Praha: Archeologický ústav AV ČR, 2005.

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Bjornsson, Andri S. Age at Onset and Clinical Course of Body Dysmorphic Disorder. Edited by Katharine A. Phillips. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190254131.003.0010.

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This chapter reviews studies that have been conducted to determine the age at onset and course of illness of body dysmorphic disorder (BDD). Age at onset has been examined in two large samples, and the mean age at onset was the same in both studies (16.7 years). About two thirds of people had onset of BDD before age 18. There were more similarities than differences between the early-onset group (before age 18) and the later-onset group, although the early-onset group had a higher prevalence of attempted suicide than the late-onset group in both samples. The clinical course of BDD is often chronic unless appropriate treatment is received. These findings point to the need for early education on healthy body image and treatment interventions for BDD, especially among adolescents.
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Vasudev, Akshya. Manic syndromes in old age. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0044.

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Manic syndromes in the elderly are different from those seen in the younger bipolar population. They are a heterogenous group but can probably be divided into two main groups based on age of onset of the illness: late onset bipolar disorder (LOB) and early onset bipolar disorder (EOB). This chapter elaborates on differences in these two groups based on epidemiological data findings, clinical presentation, aetiopathogenesis and management. Latest concepts with regards to the vascular mania hypothesis, neuroimaging findings, cognitive impairment in bipolar disorder are also dealt with. A critical review of pharmacological management options is also provided with reference to recently published data on mood stabilisers, antipsychotic and antidepressant usage for this age group.
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Jones, Matthew, and Jennifer Thompson. Atypical presentations of Alzheimer’s disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198779803.003.0005.

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Alzheimer’s disease usually presents in older age with progressive episodic memory loss. Atypical presentations of Alzheimer’s disease occur and involve non-amnestic and early-onset forms of the disease. Posterior cortical atrophy (PCA) and logopenic progressive aphasia (lvPPA) are two well-described syndromes that are most commonly due to atypical presentations of Alzheimer’s disease. PCA is a higher-order disturbance of vision whilst lvPPA is characterized by hesitant speech with word-finding difficulties and problems with repetition of words and phrases. Early-onset Alzheimer’s disease presents before the age of 65 and typically consists of a constellation of progressive cortical deficits including language disturbance, apraxia, visuospatial deficits, and poor working memory. Alzheimer’s disease may rarely be inherited because of an autosomal dominant mutation in one of three genes (PSEN1, PSEN2, and APP). Recognition and accurate diagnosis of these atypical forms is vital to ensure patients receive the most appropriate care and treatment.
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Williams, J. Corey, and Hanna E. Stevens. Treatment of Early-Onset Schizophrenia Spectrum Disorders (TEOSS) Study. Edited by Ish P. Bhalla, Rajesh R. Tampi, Vinod H. Srihari, and Michael E. Hochman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190625085.003.0009.

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This chapter provides a summary of a landmark study in child and adolescent psychiatry that addresses the treatment of youth with psychotic disorders. Are second generation antipsychotics superior to first generation antipsychotics in the treatment of early-onset schizophrenia spectrum disorders? Starting with that question, it describes the basics of the study, including funding, study locations, who was studied, how many patients, study design, study intervention, follow-up, endpoints including treatment response and adverse events, results, and criticism and limitations. No differences in symptom change were found between groups, but each group had a specific set of adverse events that distinguished it. The chapter briefly reviews other relevant studies and information, discusses implications, and concludes with a relevant clinical case.
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Lleo, Alberto, and Rafael Blesa. Clinical course of Alzheimer’s disease. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199569854.003.0002.

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• Alzheimer’s disease is an age-related neurodegenerative disorder, with onset usually in late life, characterized by cognitive impairment, a variety of behavioural symptoms, and restrictions in the activities of daily living• The initial symptom is episodic memory loss, in particular in delayed recall of visual and/or verbal material. Immediate and remote memory is usually preserved in early stages...
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Xu, Huji, Feng Huang, Chan-Bum Choi, and Tae-Hwan Kim. Axial spondyloarthritis in Asia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0028.

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The prevalence of axial spondyloarthritis (axSpA) in Chinese and Korean populations is not dissimilar to that in Caucasians. However, the age of onset is younger and peripheral arthritis of the lower limb is relatively more common. Multicenter studies have revealed similarities and differences among different ethnic backgrounds. For example, the dominant HLA-B27 subtype in Chinese SpA is B2704, while it is B2705 in Koreans. Both Chinese and Korean rheumatologists have adopted ASAS/EULAR guidelines to manage axSpA. TNFi are covered by insurance in Korea but not in China. Owing to early onset and the high cost of TNFi therapy, axSpA has placed enormous burdens on both patients and society in Asia. To achieve a higher HRQoL, rheumatologists are bringing forward cost-effective and treat-to-target therapeutic strategies by integrating nonpharmacological treatment, traditional medications, and biologics in a multimodality setting. Further basic and clinical studies in Asian populations are needed to improve decision-making in clinical practice.
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Lynch, Bernadette, and Aine Burns. The patient with scleroderma. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0165.

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Scleroderma is tightness, thickening, and non-pitting induration of skin. Two forms of the skin disease are described. Limited cutaneous systemic sclerosis (lcSSc) which occurs distal to the wrists (or ankles) and/or over the face and neck, often associated with longstanding Raynaud’s phenomenon, and diffuse cutaneous systemic sclerosis (dcSSc) where truncal as well as acral skin involvement occurs as well as tendon friction rubs. In this latter condition the onset of the skin changes occurs within 1 year of onset of Raynaud’s phenomenon; however, the skin involvement may precede onset of vascular symptoms.The skin manifestations are the outward manifestation of a systemic disease, systemic sclerosis. Lung, heart, and gut involvement are frequent. Scleroderma renal crisis, usually presenting as accelerated hypertension and acute kidney injury, is one of the most severe complications of this disease. Autoantibodies against RNA polymerase are associated with scleroderma renal crisis. It occurs in 12% of dcSSc and 2% of lcSSc patients (men and women) and carries a high morbidity and mortality although careful supportive care and blood pressure management using angiotensin converting enzyme inhibitors (ACEI) or angiotensin-II receptor blockers have improved short-term outcomes. In general, beta blockers should be avoided in the early management.Approximately two-thirds of patients require dialysis, of these many recover enough function to come off dialysis. Higher blood pressure and younger age at presentation have a better prognosis. ACEIs should be continued even after dialysis is established as the latter increases the chance of late recovery. Average time to coming off dialysis is 11 months but recovery is uncommon after 24 months. After a crisis renal function continues to improve for several years.
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Staedtke, Verena, and Eric H. Kossoff. Epilepsy Syndromes in Childhood. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0074.

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Epilepsy syndromes of childhood are a heterogeneous group of disorders that occur at specific neurodevelopmental stages, with a variable prognosis ranging from benign to catastrophic. In clinical practice they are categorized based on seizure type, age of onset, clinical presentation, electroencephalographic (EEG) findings, as well as response to treatment. In addition, recent advancements in neuroimaging and genetic testing have become important diagnostic tools revealing underlying defects for some of these syndromes. This knowledge has consequences for clinical practice, as it opens new perspectives for early diagnosis, prognosis and treatment. Here, we provide an up-to-date overview of the most common pediatric epilepsy syndromes, their clinical findings, associated EEG findings, and clinical management.
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Book chapters on the topic "Early onset age"

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Win, Aung K., Garrett Friedman, and Jose G. Guillem. "Early-Age-of-Onset Colorectal Carcinoma: An Emerging Public Health Issue." In Management of Hereditary Colorectal Cancer, 1–9. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-26234-1_1.

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Patel, Swati G., Caitlin C. Murphy, Christopher H. Lieu, and Heather Hampel. "Early age onset colorectal cancer." In Advances in Cancer Research, 1–37. Elsevier, 2021. http://dx.doi.org/10.1016/bs.acr.2021.03.001.

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Early, Theresa. "Early-Onset Schizophrenia." In Evidence-Based Practice in School Mental Health, 176–93. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780190886578.003.0005.

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Early-onset schizophrenia (EOS) is a serious psychotic disorder that affects children as young as 12 years of age. Although EOS is typically diagnosed in the specialty mental health sector, increasing the mental health literacy of school personnel is a Tier 1 intervention that can help identify youth at high risk. Tier 2 intervention could include group psychoeducation for youth at high risk and their families. Mental health treatment of EOS occurs in acute, stabilization, and maintenance phases. Youth who are experiencing EOS likely will need accommodations in school. School mental health personnel should educate teachers about accommodations and help them prepare to identify and avoid distressing stimuli, allow alternative schoolwork and activities to avoid provoking delusions, and provide safety for de-escalation. Collaboration with parents and community providers also is critical for students with EOS. A case example illustrates multitiered supports for an adolescent from diagnosis to returning to core classes.
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Lee, Ellen E., Baichun Hou, Ipsit V. Vahia, and Dilip V. Jeste. "Late-onset schizophrenia." In Oxford Textbook of Old Age Psychiatry, 671–94. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198807292.003.0043.

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Late-onset schizophrenia remains an understudied subtype of schizophrenia, despite growing recognition of its impact and distinction from early-onset schizophrenia. This chapter reviews the existing literature on late-onset schizophrenia including beginning with the nomenclature and epidemiology. Then we provide a review of key risk factors and correlates—including genetic risk, sex differences, comorbid sensory loss and physical illness, cognitive and psychiatric symptoms, sociodemographic factors, adverse life events, neuropathology, and inflammation. The chapter ends with clinical issues, including symptoms, differential diagnosis, treatments, and prognosis. Recent studies have examined the role of oestrogen treatments and a new therapy for tardive dyskinesia therapy as well as inflammatory mechanisms in schizophrenia.
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Ellajosyula, Ratnavalli. "Early onset Alzheimer’s disease." In Oxford Textbook of Neurologic and Neuropsychiatric Epidemiology, edited by Carol Brayne, Valery L. Feigin, Lenore J. Launer, and Giancarlo Logroscino, 75–82. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198749493.003.0009.

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The term ‘early onset Alzheimer’s disease’ (EOAD) is used when symptoms of Alzheimer’s disease (AD) occur in patients younger than 65 years. EOAD is an uncommon condition and data on epidemiology is limited. Prevalence rates range from 15 to 200 and incidence rates 2.4–22.6 per 100,000 population. Prevalence rates increase with age similar to that for late onset AD. The prevalence of autosomal dominant EOAD is 5.2 per 100,000. Half of these patients have an underlying mutation in amyloid precursor protein, presenilin 1 or 2 genes. Apolipoprotein E genotype is a risk factor for EOAD and homozygotes have an earlier age of onset. Methodological issues and geographical location make comparisons across epidemiological studies difficult. Further cross-national and cross-cultural studies with standardized methodology are necessary to understand the role of risk and protective factors, as well as to estimate the burden of the disease.
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Mussen, Lauren, and Akshya Vasudev. "Manic syndromes in old age." In Oxford Textbook of Old Age Psychiatry, 637–54. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198807292.003.0041.

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Manic syndromes in the elderly are different from those seen in the younger bipolar population. They are a heterogeneous group but can probably be divided into two main groups based on age of onset of the illness: late-onset bipolar disorder (LOB) and early-onset bipolar disorder (EOB). This chapter elaborates on differences in these two groups based on epidemiological data findings, clinical presentation, aetiopathogenesis, and management. It also discusses the latest concepts with regards to the vascular mania hypothesis, neuroimaging findings, and cognitive impairment in bipolar disorder (BD). It provides a critical review of pharmacological management options with reference to recently published data on mood stabilizers, antipsychotics, and antidepressant usage for this age group.
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Patterson, Gerald R. "Coercion as a basis for early age of onset for arrest." In Coercion and Punishment in Long-Term Perspectives, 81–105. Cambridge University Press, 1995. http://dx.doi.org/10.1017/cbo9780511527906.005.

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Huestegge, Lynn, Ralph Radach, Hans-Juergen Kunert, and Dieter Heller. "Visual search in long-term cannabis users with early age of onset." In The Brain's eye: Neurobiological and clinical aspects of oculomotor research, 377–94. Elsevier, 2002. http://dx.doi.org/10.1016/s0079-6123(02)40064-7.

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Sisk, Cheryl L., and Russell D. Romeo. "Puberty and Adolescence in a Lifespan Context." In Coming of Age, 144–60. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780195314373.003.0009.

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The final chapter zooms out to explore some bigger picture questions about puberty and adolescence that remain unanswered; the authors provide their perspectives on these larger issues. For instance, how do we define the end of adolescence, and how do we know when adulthood has been achieved? Are cultural expectations partially responsible for the protracted nature of human adolescent brain development? How does the timing of puberty (early vs. late bloomers) influence psychosocial development and risk of psychopathology in females and males? What are the potential consequences of medically suspending puberty onset in gender dysphoric youth? Is adolescence an experience-expectant (a specific type of experience is absolutely required for normal development) or experience-dependent (specific experiences influence developmental trajectory) developmental period? Finally, the limitations of animal models for understanding human adolescent development are discussed and experimental approaches for future research are recommended.
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Camacho, M. Catalina, Lauren S. Wakschlag, and Susan B. Perlman. "Early Childhood Irritability." In Irritability in Pediatric Psychopathology, edited by Amy Krain Roy, Melissa A. Brotman, and Ellen Leibenluft, 73–93. Oxford University Press, 2019. http://dx.doi.org/10.1093/med-psych/9780190846800.003.0005.

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The preschool age (3–6 years) is a unique period for development as both emotion regulation and foundational brain development are rapidly maturing. Emotions such as frustration are common during this age; however, there is extensive variability along the spectrum from typical to atypical. This juxtaposition of normative misbehaviors with stable irritable temperament presents unique challenges to the differentiation of normative variation from the onset of clinical problems. As such, recent research has focused efforts on improving methods for differentiating normative and clinically concerning behavior. Improved neuroimaging tools, in combination with behavioral and clinical assessment, have provided an additional tool for assessing pediatric irritability. The authors believe that joint consideration of brain–behavior atypicalities will enhance early identification of clinically concerning irritability. To this end, this chapter aims to summarize the rapid development occurring during the preschool years, describe advancements in developmentally appropriate irritability assessments, and integrate these measurements within a neurodevelopmental framework.
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Conference papers on the topic "Early onset age"

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Gilio, M., G. Tramontano, U. Bottoni, A. Padula, and S. D’Angelo. "AB0938 Treatment patterns in early psoriatic arthritis according to the age of onset." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.2197.

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Middlebrooks, Candace D. "Abstract 2408: Analysis of theCDKN2Agene in FAMMM Syndrome families reveals early age of onset for additional syndromic cancers." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-2408.

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Middlebrooks, Candace D. "Abstract 2408: Analysis of theCDKN2Agene in FAMMM Syndrome families reveals early age of onset for additional syndromic cancers." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-2408.

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Singh, Neeraj K., and Ravindra Kumar Singh. "Comprehensive Epilepsy Early Warning System and Seizure Alarm." In ASME 2009 4th Frontiers in Biomedical Devices Conference. ASMEDC, 2009. http://dx.doi.org/10.1115/biomed2009-83042.

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Epilepsy is characterized by unprovoked, recurring seizures that disrupt the nervous system and can cause mental and physical dysfunction. It affects all age groups. About 14% of epilepsy patients are under 15 years old and 24% are over 64, with 62% being between those ages. There is an increased risk of death due to accidents, suicide and other medical conditions. There are also cases where the death appears to be directly related to epilepsy itself, usually referred to as Sudden Unexpected Death in Epilepsy (SUDEP). This information is not always passed from doctor to patient, for various reasons. Epilepsy share is an uncontrolled electrical discharge from nerve cells in the cerebral cortex. This is the part of the brain that integrates higher mental functions, general movement, and the functions of the internal organs in the abdominal cavity, perception, and behavioral reactions. The early warning system will be based on three factors: 1. Detection of abnormal electrical activity of brain. 2. Detection of abnormal breathing (too fast or too slow) that almost always accompanies the onset of an attack. 3. Unusual sensations or movements of parts of body such as muscles relaxation and tightening.
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Divjan, Adnan, Maria Jose Rosa, Marilyn Reyes, Lori Hoepner, Beverley J. Sheares, Hanjie Zhang, Frederica P. Perera, Rachel L. Miller, and Matthew S. Perzanowski. "Exhaled No At Age 7-11 Years Is Elevated With Early Life But Not Recent Onset Of Allergic Sensitization." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4472.

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Johnson, Katherine A., Philip B. Emmerich, Anna L. Lippert, Cheri A. Pasch, Linda Clipson, Wei Zhang, Kristina A. Matkowskyj, and Dustin A. Deming. "Abstract 2734: Impact of the mutation profile and versican status on lymphocyte infiltration in early age onset colorectal cancer." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-2734.

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Galuppi, E., I. Farina, C. De Giorgio, CA Scirè, and M. Govoni. "THU0090 Influence of age at disease onset on clinical, functional, and ultrasonographic outcomes in a monocentric early rheumatoid arthritis cohort." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.5011.

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Lippert, Anna, Katherine A. Johnson, Philip B. Emmerich, Cheri A. Pasch, Linda Clipson, Kristina A. Matkowskyi, Wei Zhang, and Dustin A. Deming. "Abstract 3166: Impact of cancer associated fibroblast phenotypes on the infiltration of t-lymphocytes in early age onset colorectal cancer." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-3166.

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Quinn, Kyle P., Jason F. Luck, Roger W. Nightingale, and Beth A. Winkelstein. "The Onset of Structural Yield During Tensile Loading Increases With Age in the Pediatric PMHS Cervical Spine." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204771.

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Motor vehicle crashes are the leading cause of injury to the pediatric spine, and the mortality rate of pediatric spinal trauma victims has been approximated at 25–32% [1]. In addition, non-fatal painful traumatic injuries resulting from motor vehicle crashes or sports-related activities contribute to an estimated prevalence of neck pain of 21–41% in children and adolescents [2]. It has been hypothesized that the anatomy of the pediatric neck, combined with a relatively large head mass, presents an increased risk for traumatic inertial loading in that population, particularly during early development. However, the relationship between mechanical metrics related to pain and age remains undefined, limiting the development of meaningful estimates of tolerance to painful injury in this population.
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Argyropoulou, Ourania, Andreas Goules, Evangelia Zampeli, Maria Mavromati, Clio Mavragani, Fotini Skopouli, Haralampos M. Moutsopoulos, and Athanasios Tzioufas. "FRI0222 ANALYSIS OF CLINICAL AND SEROLOGICAL PICTURE OF PATIENTS WITH PRIMARY SJöGREN’S SYNDROMEAND AN EARLY DISEASE ONSET AT AGE BEFORE 35 YEARS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.7105.

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