Academic literature on the topic 'F.N.A.T'

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Journal articles on the topic "F.N.A.T"

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Morton, K. D. "Fine Needle Aspiration Cytology of Lesions of the Head and Neck and Factors Affecting Outcome." Scottish Medical Journal 34, no. 5 (October 1989): 523–25. http://dx.doi.org/10.1177/003693308903400505.

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Fine Needle Aspiration Cytology (F.N.A.C.) has been used as a diagnostic tool by the ENT departments in Tayside since the end of 1985. This paper discusses the results of our initial experience with this and outlines some of the diagnositc pitfalls. This is a useful and accurate procedure with many advantages over diagnostic biopsy.
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Pérez Dueñas, Virginia, Isabel Torres Sánchez, Francisco García Río, Emilio Valbuena Durán, Blanca Vicandi Plaza, and Jose María Viguer García-Moreno. "Usefulness CT-guided F.N.A.C. in the Diagnosis of Mediastinal Lesions." Archivos de Bronconeumología ((English Edition)) 46, no. 5 (May 2010): 223–29. http://dx.doi.org/10.1016/s1579-2129(10)70057-4.

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Abedalrahman, Sarab K. "Accuracy of Fine Needle Aspiration Biopsy (F.N.A.B) in Diagnosis of Breast Lump." AL-Kindy College Medical Journal 15, no. 2 (January 30, 2020): 9–12. http://dx.doi.org/10.47723/kcmj.v15i2.152.

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Background: Breast cancer is the first one among Iraqi females. Most of them present later for diagnosis. Early detection center in tertiary hospital practice uses FNAB for early diagnosis. Publications on accuracy of this detection are scarce. Objective: To test the accuracy of FNAB in breast lump diagnosis. Methods: Diagnostic test accuracy study, on 204 women with breast lump, attending the oncology department in 2017. Results: Fine-needle aspiration biopsy diagnosis of histologically malignant cases were, malignant in 89 (87.3%), suspicious of malignancy in 5 (4.9%), and benign in 4 (3.9%). Complete sensitivity was 87.3%, and specificity was 100%, with 12.7% false negative results and no false positive cases. The accuracy was 94%.
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Tremblay, Pierre-André. "Granjon, Marie-Christine. L’Amérique de la contestation : Les années soixante aux États-Unis. Paris, Presses de la F.N.S.P., 1985, 656 p." Études internationales 19, no. 3 (1988): 592. http://dx.doi.org/10.7202/702407ar.

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5

A M, Damodaran, Gururaja Prasad, Rangaswamy M, and Manjunath G V. "F.N.A . C DIAGNOSIS OF A PURE LEYDIG TUMOUR OF THE TESTIS PRESENTING WITH GYNAECOMASTIA AND INFERTILITY : A RARE CASE REPORT." Journal of Evidence Based Medicine and Healthcare 1, no. 15 (December 15, 2014): 1962–64. http://dx.doi.org/10.18410/jebmh/2014/285.

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Delbaere, Anne, Pierre J. M. Bergmann, Christine Gervy-Decoster, Michel Staroukine, and Yvon Englert. "Angiotensin II immunoreactivity is elevated in ascites during severe ovarian hyperstimulation syndrome: implications for pathophysiology and clinical management**Supported by grant 1.5.218.94F from the Fonds National de la Recherche Scientifique (F.N.R.S.), Brussels, Belgium." Fertility and Sterility 62, no. 4 (October 1994): 731–37. http://dx.doi.org/10.1016/s0015-0282(16)56997-0.

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7

Kapralova, Katarina, Monika Horvathova, Jana Kucerova, Dagmar Pospisilova, Emilie Leroy, Stefan N. Constantinescu, and Vladimir Divoky. "JAK2 E846D Germline Mutation Associated with Erythrocytosis Causes Increased and Prolonged Epo-Induced Activation of STAT5." Blood 124, no. 21 (December 6, 2014): 4008. http://dx.doi.org/10.1182/blood.v124.21.4008.4008.

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Abstract Until now, disease-causing JAK2 mutations have been associated with clonal myeloproliferative neoplasms (acquired mutations) or with polyclonal hereditary thrombocytosis (congenital mutations). Here we present the first example of a JAK2 mutation associated with congenital erythrocytosis. We studied a young patient with congenital erythrocytosis who inherited two heterozygous mutations, an E846D mutation in JAK2 from the mother and a Q157H in PHD2 from the father. Each mutation apparently sufficed to induce EPO-hypersensitivity in BFU-E progenitors, since BFU-Es from both parents were hypersensitive to EPO, as were those of the patient. Strikingly, only the patient, but not his parents exhibited an erythrocytosis phenotype. Expression analysis of granulocyte mRNA revealed markers of inappropriately increased signaling mediated by hypoxia sensing pathway (HIF) (Kapralova, Blood, 2014,16;123:391-4) in the propositus and his father. This result suggested that the polycythemia phenotype of the propositus could result from a combined impact of otherwise clinically silent defects co-inherited from both parents and affecting EPOR/JAK2 and HIF signaling pathways. Using a Ba/F3-EPOR cellular model we have characterized the impact of JAK2 E846D on EPO-induced JAK2-mediated signaling, cell cycle progression and cell responsiveness to limited concentrations of EPO. We demonstrated prolonged, EPO-dependent JAK2 and STAT5 activation caused by JAK2 E846D, which was mitigated by a JAK2 inhibitor, AZ-960. However, in contrast to the oncogenic V617F mutation, E846D did not trigger factor-independent constitutive signaling. Also unlike the oncogenic V617F or the JAK2 germline mutations associated with hereditary thrombocytosis, the E846D did not activate STAT1, in agreement with differential signaling of STATs in different clinical phenotypes associated with activating JAK2 mutations. The thrombocytosis phenotype (ET for acquired and hereditary thrombocytosis for germline) is promoted by STAT1 signaling, while STAT5 signaling results in erythroid hyper-proliferation. Using BrdU incorporation assay and growth factor starved Ba/F3 cells we also demonstrated that JAK2 E846D - when compared with JAK2 wild type - stimulates more rapid re-entry of the transfectants into the S phase in response to low concentration of EPO. In agreement with immunoblot analyses JAK2 E846D did not support growth factor-independent proliferation in cytokine-free media; however it provided improved survival in limiting concentrations of EPO. The proliferation advantage of JAK2 E846D transfectants was diminished by AZ-960. These results showed that E846D substitution lacks features of a constitutive gain-of-function mutation and were consistent with its germline transmission and with non-clonal hematopoiesis observed in the JAK2 E846D-positive mother. Structural modeling based on the X-ray crystal structures of inactive and active kinase domain explain the mechanism of hypersensitivity and prolonged signaling. The homologous residue of E846 in the JAK2 pseudokinase domain (JH2) is N542, a residue that is targeted by the exon 12 activating mutations in polycythemia vera, and a close residue to this homologous position in JH2 is R564 recently found to be mutated in hereditary thrombocytosis. Thus the E846D mutation belongs to a conformational hotspot in kinases, but this mutation is only inducing hypersensitivity and prolonged action and not constitutive activation. In conclusion, JAK2 E846D mutation associated with congenital erythrocytosis confers hypersensitivity to EPO, prolonged and selective but not constitutive STAT5 activation, and requires additional cooperating event, likely affecting the HIF signaling, for its full clinical expression. (KK, MH, JK contributed equally to this work.) Funding: Czech Science Foundation P301/12/1503, Czech Ministry of Health NT13587-4, Education for Competitiveness Operational Program CZ.1.07/2.3.00/20.0164 and Palacky University (IGA_LF_2014_011). JK was supported by CZ.1.07/2.3.00/30.0041, EL by a FRIA PhD fellowship. SNC was supported from the F.R.S.-F.N.R.S., Belgium, the Salus Sanguinis Foundation, the Action de Recherche Concertée projects MEXP31C1 and ARC10/15-027 of the University catholique de Louvain, Brussels, the Fondation contre le Cancer, Brussels, the PAI Programs BCHM61B5 and Belgian Medical Genetics Initiative. Disclosures Constantinescu: Shire: Consultancy, Speakers Bureau; Novartis: Consultancy, Membership on an entity's Board of Directors or advisory committees, Speakers Bureau; Personal Genetics: Membership on an entity's Board of Directors or advisory committees; Amgen: Speakers Bureau.
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8

Pant, Bhawana, Sanjay Gaur, and Prabhat Pant. "F.N.A.C. AS DIAGNOSTIC TOOL OF PAROTID TUMORS AND ASSOCIATED PITFALLS IN CORRELATION WITH HISTOPATHOLOGY." International Journal of Medical and Biomedical Studies 4, no. 1 (January 30, 2020). http://dx.doi.org/10.32553/ijmbs.v4i1.1153.

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F.NA.C has been used for ages as a safe and economical tool for fast preoperative diagnosis of parotid tumors. It has certain pitfall which sometimes leads to misdiagnosis and consequently it may have affect on treatment of the tumors. Keeping in view of the diverse classification of parotid tumors’ information from cytology should be combined with radiology as well as clinical diagnosis. Aim: To discuss some cases where there was discrepancy between cytological diagnosis and histopathological result and also suggest measures to improve the efficacy of F.N.A.C. Material and methods: The study includes 50 cases of parotid tumours who presented to the department of ENT at Government medical college Haldwani which is a tertiary referral centre during 2009 to 2016. Only adult patients were included and inflammatory swelling were excluded from the study. All patients evaluated Contrast enhanced computerized tomography(CECT) and Magnetic resonance imaging (MRI) followed by Fine needle aspiration cytology .Preoperative diagnosis was made upon the findings of the above investigations and different types of parotid surgeries were done. . Final diagnosis was made on histopathological examination. Result :The most common tumour came out to be pleomorphic adenoma (23 cases-46%) followed by mucoepidermoid carcinoma(12cases-24%). In ten cases there was no clear cut association between cytological diagnosis and final histopathological diagnosis. Conclusion: FNAC is highly sensitive and specific technique for diagnosis of many salivary gland swellings. FNAC can be used preoperatively to avoid unnecessary surgery and biopsy. Details of clinical information and radiologic features may help the pathologist to arrive at the appropriate diagnosis and reduce false interpretation. Pitfalls may also occur with improper technique of FNAC which can be overcome by proper caution.
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Mishra, Jyotirmayee, Sonal Seth, Shobhagini Nayak, and Kailash Chandra Agrawal. "Spectrum of Salivary Gland Lesions in Western Odisha with the Diagnostic Value of F.N.A.C. Correlating with Histomorphology." Annals of International medical and Dental Research 4, no. 4 (June 24, 2018). http://dx.doi.org/10.21276/aimdr.2018.4.4.pt2.

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Lal, Dr Manohar. "Ultrasound Guided F.N.A.C. is the Excellent Methods for the Diagnosis of Pyogenic Liver Abscess (PLA) in patients Attending in Tertiary Care Hospital, at N.M.C.H. Patna." Journal of Medical Science And clinical Research 7, no. 6 (June 11, 2019). http://dx.doi.org/10.18535/jmscr/v7i6.60.

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Books on the topic "F.N.A.T"

1

Valente, José Carlos. Estado Novo e alegria no trabalho: Uma história política da FNAT (1935-1958). Lisboa: Edições Colibri, 1999.

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2

Lewin, Christophe. Le retour des prisonniers de guerre français: Naissance et développement de la F.N.P.G., 1944-1952. Paris: Publications de la Sorbonne, 1986.

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Journée, de recherches F. N. R. S. du Groupe de géographie humaine théorique et satellitaire (1987 Université de Liège). Télédétection satellitaire et espaces urbains: Actes de la Journée de recherches F.N.R.S. du Groupe de géograph[i]e humaine théorique et quantitative, 27 mai 1987, Université de Liège. Liège: Société géographique de Liège, 1987.

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International Conference of Cypriote Studies (1st 1989 Brussels, Belgium, etc.). Cypriote terracottas: Proceedings of the First International Conference of Cypriote Studies, Brussels-Liège-Amsterdam, 29 May-1 June 1989 : organized by the "Groupe de contact interuniversitaire d'études chypriotes"/"Interuniversitaire contactgroep voor Cyprische Studies", F.N.R.S./N.F.W.O. (Belgium). Brussels-Liège: A.G. Leventis Foundation, Vrije Universiteit Brussel-Université de Liège, 1991.

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Le Traitement de l'information marketing et finaniere dans la experts : Eric de Bodt Aspirant F.N.R.S. Louvain-la-Neuve : 1992, 1992.

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Cypriote terracottas: Proceedings of the First International Conference of Cypriote Studies, Brussels-Liege-Amsterdam, 29 May-1 June 1989 : Organized by ... Studies", F.N.R.S./N.F.W.O. (Belgium). A.G. Leventis Foundation, Vrije Universiteit Brussel-Universite de Liege, 1991.

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Book chapters on the topic "F.N.A.T"

1

Lewin, Christophe. "Chapitre V. La guerre froide de la F.N.P.G. effectifs, cadres et luttes internes." In Le retour des prisonniers de guerre français, 205–72. Éditions de la Sorbonne, 1986. http://dx.doi.org/10.4000/books.psorbonne.70519.

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