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Books on the topic 'Receptors fibroblast growth factor'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 February 2022

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1

Burgar, Helen Rachel. Fibroblast growth factor receptor signalling. University of Birmingham, 2002.

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2

Walters, Jean Elizabeth. Immunochemical studies on fibroblast growth factor-1 and fibroblast growth factor receptor 1. Oxford Brookes University, 1998.

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3

Smith, Conrad. Developmental changes in the injury induced expression of fibroblast growth factor-2 and fibroblast growth factor receptor 1 in the rat brain. University of Birmingham, 1998.

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4

Thompson, Stuart David. Fibroblast growth factor 2 and its receptor, FGFR 1 in the normal thyroid and multinodular goitre. University of Birmingham, 2000.

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5

1945-, Lippman Marc E., Ortho Pharmaceutical Corporation, and University of California, Los Angeles., eds. Growth regulation of cancer: Proceedings of an Ortho-UCLA Symposium on Growth Regulation of Cancer, held at Park City, Utah, January 17-23, 1987. Liss, 1988.

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6

Plowright, Elizabeth Emma. Expression of fibroblast growth factor receptor 3 in multiple myeloma causes II-6-independence and enhanced survival. National Library of Canada, 1999.

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7

Clarke, Wendy Elizabeth. Intracellular compartmentalisation of fibroblast growth factor-2 and associated high and low affinity receptors after cerebral injury to the adult rat brain. University of Birmingham, 1999.

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8

Onwuazor, Obiageli Nneka. Mutation, SNP and isoform analysis of fibroblast growth factor receptor 3 (FGFR3) in 150 newly diagnosed multiple myeloma patients. National Library of Canada, 2003.

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9

Cocks, Helen Catherine. Fibroblast growth factor in the thyroid. University of Birmingham, 2003.

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10

Lyn-Cook, Richard X. Cortical development in fibroblast growth factor 2 knockout mice. s.n.], 1999.

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11

Madshus, Inger Helene, ed. Signalling from Internalized Growth Factor Receptors. Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-540-69494-6.

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12

Alexis, Michael N., and Constantin E. Sekeris, eds. Activation of Hormone and Growth Factor Receptors. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1936-5.

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13

Daley, Sandra J. Basic fibroblast growth factor mediates neointimal formation in porcine aortic explants. National Library of Canada = Bibliothèque nationale du Canada, 1997.

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14

McGuckin, C. P. The role of growth factor receptors in leukaemia. The Author], 1991.

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15

Das, Manjusri. Selected abstracts on oncogenes and epidermal growth factor receptors. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1986.

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16

Boros, Peter. Hepatocyte growth factor: The basic principles. R.G. Landes, 1999.

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17

NATO Advanced Research Workshop on Molecular Mechanisms and Consequences of Activation of Hormone and Growth Factor Receptors (1988 Nauplion, Greece). Activation of hormone and growth factor receptors: Molecular mechanisms and consequences. Kluwer Academic Publishers, 1990.

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18

Cellular receptors for human growth hormone: Quantitative aspects and clinical applications. Leuven University Press, 1988.

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19

Wang, David I. Kuo. The effects of fibroblast growth factor 2 on the early stages on in vitro endothelial wound repair. National Library of Canada, 1998.

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20

Hildahl, Jon. Endocrine regulation of flatfish metamorphosis: Growth hormone, insulin-like growth factor-I and their receptors in Atlantic halibut. Göteborg University, Department of Zoology/Zoophysiology, 2007.

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21

Hildahl, Jon. Endocrine regulation of flatfish metamorphosis: Growth hormone, insulin-like growth factor-I and their receptors in Atlantic halibut. Göteborg University, Department of Zoology/Zoophysiology, 2007.

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22

Khesaifan, Mahler M. K. Transferrin and haemopoietic growth factor receptors on the human leukaemia U937 cell line: Observation by confocal microscopy. The Author], 1994.

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23

Sherbet, G. V. Growth factors and their receptors in cell differentiation, cancer and cancer therapy. Elsevier, 2011.

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24

Leyck, Dieken Markus, Federwisch Matthias, De Meyts Pierre, and Wollmer Axel 1935-, eds. Insulin & related proteins: Structure to function and pharmacology. Kluwer Academic Publishers, 2002.

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25

Birchmeier, Carmen. Growth factors in development. Academic, 2011.

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26

Kazue, Takano, Hizuka Naomi, and Takahashi Shinʼichirō 1959-, eds. Molecular mechanisms to regulate the activities of insulin-like growth factors: Proceedings of the 4th International Symposium on Insulin-like Growth Factors, at Tokyo International Forum, Tokyo, Japan, 21-24 October 1997. Elsevier, 1998.

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27

International Symposium on Molecular and Cellular Biology of Insulin and IGFs (3rd 1990 Gainesville, Fla.). Molecular biology and physiology of insulin and insulin-like growth factors. Plenum Press, 1991.

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28

C, Baxter R., Gluckman Peter D, and Rosenfeld Ron G, eds. The insulin-like growth factors and their regulatory proteins: Proceedings of the Third International Symposium on Insulin-Like Growth Factors, Sydney, 6-10 February 1994. Excerpta Medica, 1994.

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29

Endocrine Fgfs and Klothos Advances in Experimental Medicine and Biology. Springer, 2012.

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30

Zhang, Zhe. Cell Surface Heparan Sulfate Proteoglycans As Co-Receptors for Fibroblast Growth Factor. Leuven Univ Pr, 2002.

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31

Fibroblast Growth Factor Receptor (FGFR) Signaling Pathway in Tumor. MDPI, 2020. http://dx.doi.org/10.3390/books978-3-03936-785-6.

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32

Steinfeld, R. Modulation of the Interaction Between Fibroblast Growth Factor-2 and Fibroblast Growth Factor Receptor-1 by Cell Surface Heparan Sulfate Proteoglycans. Leuven University Press, 1996.

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33

Cimpean, Anca Maria, Andreea Adriana Jitariu, and Marius Raica. Growth Factors and Their Corresponding Receptors as Targets for Ovarian Cancer Therapy. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190248208.003.0011.

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Ovarian cancer remains one of the most aggressive and difficult to manage malignancies regarding evaluation and therapeutic options. The high mortality persists despite extensive research in the field. Current conventional chemotherapy does not improve disease-free survival and does not decrease recurrences amongst patients. This calls for a stringent reconsideration of the drugs selection, focused on the most targeted strategies and personalization of the therapy. Targeted agents against growth factors and their corresponding receptors are already approved as first- or second-line neoadjuvant

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34

Gutiérrez, Orlando M. Fibroblast growth factor 23, Klotho, and phosphorus metabolism in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0119.

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Fibroblast growth factor 23 (FGF23) and Klotho have emerged as major hormonal regulators of phosphorus (P) and vitamin D metabolism. FGF23 is secreted by bone cells and acts in the kidneys to increase urinary P excretion and inhibit the synthesis of 1,25 dihydroxyvitamin D (1,25(OH)2D) and in the parathyroid glands to inhibit the synthesis and secretion of parathyroid hormone. Phosphorus excess stimulates FGF23 secretion, likely as an appropriate physiological adaptation to maintain normal P homeostasis by enhancing urinary P excretion and diminishing intestinal P absorption via lower 1,25(OH)

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35

Growth regulation of cancer: Proceedings of an Ortho-UCLA Symposium on Growth Regulation of Cancer, held at Park City, Utah, January 17-23, 1987 (UCLA symposia on molecular and cellular biology). Liss, 1988.

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36

Growth Regulation of Cancer (UCLA Symposia on Molecular and Cellular Biology). John Wiley & Sons Inc, 1990.

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37

1945-, Lippman Marc E., Dickson Robert B. 1952-, and University of California, Los Angeles., eds. Growth regulation of cancer II: Proceedings of a UCLA symposium, held at Keystone, Colorado, January 21-27, 1989. Wiley-Liss, 1990.

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38

White, Kenneth, Christian Faul, and Orlando Gutierrez. Fibroblast Growth Factor 23. Elsevier, 2021.

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39

Fibroblast Growth Factor 23. Elsevier, 2021. http://dx.doi.org/10.1016/c2018-0-03274-0.

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40

Baird, Andrew, 1954 July 27-, Klagsbrun Michael, and New York Academy of Sciences., eds. The Fibroblast growth factor family. New York Academy of Sciences, 1991.

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41

Pedro, Cuevas, ed. Fibroblast growth factor in the cardiovascular system. I. Holzapfel, 2002.

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42

Meier, Petra M., and Thomas O. Erb. Craniosynostosis and Apert Syndrome. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0021.

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Apert syndrome is a complex, progressive multisystem condition of the craniosynostosis spectrum originating from a fibroblast growth factor receptor disorder. Multidisciplinary treatment teams may include craniofacial surgery, neurosurgery, otolaryngology, ophthalmology, oro-maxillofacial surgery, and pediatric intensive care. Secondary to midface hypoplasia, children often present with a compromised airway and have a high incidence of sleep disorders. Anesthetic considerations include difficult airway assessment, the presence of obstructive sleep apnea syndrome, and increased intracranial pre

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43

Padela, Sanna. Hepatocyte growth factor and fibroblast growth factor-7 in neonatal lung development and injury. 2006.

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44

Madshus, I. H. Signalling from Internalised Growth Factor Receptors. Springer Berlin Heidelberg, 2010.

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45

H, Madshus I., ed. Signalling from internalised growth factor receptors. Springer, 2004.

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46

Ashton, Anthony. Fibroblast Growth Factor: Methods and Protocols (Methods in Molecular Medicine). Humana Press, 2001.

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47

Huang, Kui. Vascular Endothelial Growth Factor Receptors in Angiogenesis. Uppsala Universitet, 2001.

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48

Boddeke, Erik W. G. M., Bart J. L. Eggen, and Knut P. H. Biber. Cytokine, Chemokine, and Growth Factor Receptors and Signaling. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199794591.003.0022.

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This is a digitally enhanced text. Readers can also see the coverage of this topic area in the second edition of Neuroglia. The second edition of Neuroglia was first published digitally in Oxford Scholarship Online and the bibliographic details provided, if cited, will direct people to that version of the text. Readers can also see the coverage of this topic area in the ...

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49

Elder, Grahame J. Metabolic bone disease after renal transplantation. Edited by Jeremy R. Chapman. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0288.

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Patients who undergo kidney transplantation have laboratory, bone, and soft tissue abnormalities that characterize chronic kidney disease mineral and bone disorder (CKD-MBD). After successful transplantation, abnormal values of parathyroid hormone, fibroblast growth factor 23, calcium, phosphate, vitamin D sterols, and sex hormones generally improve, but abnormalities often persist. Cardiovascular risk remains high and is influenced by prevalent vascular calcification, and fracture risk increases due to a combination of abnormal bone ‘quality’, compounded by immunosuppressive drugs and reducti

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50

Boros, Peter. Hepatocyte Growth Factor: The Basic Principles. Landes Bioscience, 2002.

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