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Littérature scientifique sur le sujet « Tobacco Smoke Pollution »

Auteur : Grafiati
Publié le 4 juin 2021
Mis à jour le 30 juillet 2024

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Articles de revues sur le sujet "Tobacco Smoke Pollution"

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Petersen, Anne Berit, Natassia Muffley, Khamphithoun Somsamouth et Pramil N. Singh. « Smoked Tobacco, Air Pollution, and Tuberculosis in Lao PDR : Findings from a National Sample ». International Journal of Environmental Research and Public Health 16, no 17 (23 août 2019) : 3059. http://dx.doi.org/10.3390/ijerph16173059.

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In 2017, more than half of the global burden of incident tuberculosis (TB) came from the Western Pacific region. In Lao People’s Democratic Republic (PDR), the high rates of tobacco use and use of polluting biomass fuels for cooking (e.g., wood, charcoal, crop waste, dung) represent significant risk factors for TB. The purpose of this study was to determine the association between self-reported (1) smoking and TB; and (2) exposure to air pollution (from both cooking fires and environmental tobacco smoke) and TB among adults in Lao PDR. We analyzed data from the 2012 National Adult Tobacco Survey (NATSL) of Lao PDR—a multi-stage stratified cluster sample of 9706 subjects from 2822 households located in all 17 provinces. Utilizing a nationally representative sample and inferential, multivariable methods, we observed a significant increase in odds of self-reported TB among those who smoked tobacco (OR = 1.73, 95% CI = (1.00 to 2.98)). Larger multivariable models identified independent contributions from exposure to tobacco pipes (OR = 21.51, 95% CI = (6.34 to 72.89)) and communal outdoor fires (OR = 2.27, 95% CI = (1.15 to 4.49)). An index measuring combined exposure to smoked tobacco, environmental tobacco smoke in enclosed workspace, indoor cooking fire, trash fires, and other outdoor communal fires also showed a positive association (OR per added exposure = 1.47, 95% CI = (1.14 to 1.89)). The findings of this study underscore the need for multi-sectoral collaboration between tobacco control, environmental health, TB prevention and treatment programs, national authorities, policy makers, civil groups, and the private sector to address the convergence of potential risk factors impacting respiratory health in Lao PDR.
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Crawford, W. Allan. « Indoor Air Pollution and Environmental Tobacco Smoke ». Journal of the Royal Society of Health 109, no 1 (février 1989) : 30–33. http://dx.doi.org/10.1177/146642408910900114.

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Puspitasari, Mardiana Dwi, et Riza Fatma Arifa. « Progress in Reducing Indoor Tobacco Smoke Pollution Toward the Establishment of Kabupaten/Kota Layak Anak : Evidence From the 2012 and 2017 Indonesia Demographic and Health Surveys ». Jurnal Bina Praja 15, no 1 (avril 2023) : 193–206. http://dx.doi.org/10.21787/jbp.15.2023.193-206.

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In 2017, only about 26.68 percent of Indonesian households were smoke-free. Previous research established a relationship between the health of children under five and indoor tobacco smoke pollution. According to Presidential Regulation 25 of 2021, some indicators that should be met for establishing a child-friendly city/ regency (KLA) are the child's rights to health and family environment. Data from the Indonesia Demographic and Health Surveys (IDHS) 2012 and 2017 assessed progress toward smoke-free homes by evaluating the interaction between child age and indoor tobacco smoking at the urban-rural area and regional levels. T-test analysis was used to determine the statistical significance. Findings revealed a higher prevalence of indoor tobacco smoking in rural areas. Over time, the prevalence of indoor tobacco smoking in Indonesian rural areas decreased by 0.84 percent. There was no significant reduction in Kalimantan, Nusa Tenggara, or Maluku-Papua. Furthermore, Sulawesi experienced a significant increase of approximately 4.54 percent. From 2012 to 2017, the prevalence of indoor tobacco smoking was higher in households with children under five, home crowding, poor households, and households with a low education household head. Therefore, interventions should focus on rural areas. City/regency governments in Sulawesi, Kalimantan, Nusa Tenggara, and Maluku-Papua should be more concerned about indoor tobacco smoke pollution. Raising family awareness about the dangers of indoor tobacco smoke pollution could be targeted at households with children under the age of five, overcrowded households, low-income households, and households with a low-education household head.
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Fetterman, Jessica L., Melissa J. Sammy et Scott W. Ballinger. « Mitochondrial toxicity of tobacco smoke and air pollution ». Toxicology 391 (novembre 2017) : 18–33. http://dx.doi.org/10.1016/j.tox.2017.08.002.

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Oliveira, Maria João, Inês Franco, Ana Gonçalves et Ivone Pascoal. « O Tabagismo em Terceira Mão : Um Conceito a (Re)Lembrar ! » Acta Médica Portuguesa 28, no 5 (1 septembre 2015) : 676. http://dx.doi.org/10.20344/amp.6858.

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Yaksic, Mateo Sainz, Mauro Tojo, Alberto Cukier et Rafael Stelmach. « Profile of a Brazilian population with severe chronic obstructive pulmonary disease ». Jornal de Pneumologia 29, no 2 (avril 2003) : 64–68. http://dx.doi.org/10.1590/s0102-35862003000200004.

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Chronic obstructive pulmonary disease (COPD) is a public health problem. Tobacco smoking is the major cause, but not the only one. Air pollution, exposure to chemicals, environmental smoke exposure, and passive smoking are among other contributing causes; being viral and bacterial infections also risk factors. Gender and weight are associated to the severity of the disease. Co-morbidity is frequent. OBJECTIVE: To characterize a population of COPD outpatients followed at an outsourced medical service. METHODS: Questionnaires were applied to patients with COPD. The data included gender, age, weight, body mass index (BMI), oxygen delivery users, and FEV1, exposure to tobacco smoke, exposure to wood smoke, history of tuberculosis and co-morbid diseases. RESULTS: Of the 70 patients enrolled in the study, 70% (49) were men with an average age of 64 ± 10 years, average weight of 63 ± 16 kg and average BMI of 22 ± 5 kg/m². Mean FEV1 was 35 ± 14% and 45.7% were oxygen dependent. Nine (12.8%) patients never smoked, while 78.8% had quit tobacco smoking, (38 ± 11 pack/years was the average). Nine (12.8%) smoked corn husk cigarettes. Eighteen (25.7%) were exposed to wood smoke. Eleven (15.7%) patients had tuberculosis, 5.7% complained of asthma symptoms, 2.8% had bronchiectasis, 11.4% diabetes mellitus, 51.4% hypertension, and 20% Cor pulmonale. CONCLUSION: Other possible COPD etiologies must be investigated. Determinants of the pulmonary injury could be environmental smoke exposure associated to former infections. Men with low BMI are typically representative of this severe patient population. Hypertension and Cor Pulmonale are frequent co-morbidity factors.
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GARDEZI, SHAH NAWAZ HASSAN. « PASSIVE SMOKING ». Professional Medical Journal 12, no 04 (31 décembre 2005) : 354–56. http://dx.doi.org/10.29309/tpmj/2005.12.04.5080.

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Definition: Breathing other people’s smoke is calledpassive, involuntary or second hand smoking.Types:- Side stream – from burning tip of the cigaretteMaintenance – inhaled and exhaled by smoker.Environmental tobacco smoke (ETS) is a major sourceof indoor air pollution.
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Hulka, Barbara S. « The health consequences of environmental tobacco smoke ». Environmental Technology Letters 9, no 6 (juin 1988) : 531–38. http://dx.doi.org/10.1080/09593338809384601.

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Aviado, Domingo M. « Suspected pulmonary carcinogens in environmental tobacco smoke ». Environmental Technology Letters 9, no 6 (juin 1988) : 539–44. http://dx.doi.org/10.1080/09593338809384602.

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Cain, William S., Tarik Tosun, Lai-Chu See et Brian Leaderer. « Environmental tobacco smoke : Sensory reactions of occupants ». Atmospheric Environment (1967) 21, no 2 (janvier 1987) : 347–53. http://dx.doi.org/10.1016/0004-6981(87)90011-4.

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Thèses sur le sujet "Tobacco Smoke Pollution"

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Nanwani, Shalini Suresh. « Environmental tobacco smoke and wellbeing ». Thesis, Click to view the E-thesis via HKUTO, 2003. http://sunzi.lib.hku.hk/hkuto/record/B31971209.

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Chan, Tsz Tung. « Removal and leakage of environmental tobacco smoke from model smoking room / ». View abstract or full-text, 2009. http://library.ust.hk/cgi/db/thesis.pl?MECH%202009%20CHAN.

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唐海寧 et Hoi-ning Mandy Tong. « Evidence-based practice guidelines : nurses' interventions for care-takers of paediatric in-patients to reducechildren's environmental tobacco smoke exposure ». Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43251547.

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Tong, Hoi-ning Mandy. « Evidence-based practice guidelines nurses' interventions for care-takers of paediatric in-patients to reduce children's environmental tobacco smoke exposure / ». Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B43251547.

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Campbell, Robert Charles James. « Speciation of metals and metalloids in tobacco and tobacco smoke : implications for health and regulation ». Thesis, University of St Andrews, 2014. http://hdl.handle.net/10023/5728.

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Some metals and metalloids make significant contributions to the harmful effects of tobacco consumption although understanding the mechanisms involved in toxicity is hampered by the lack of information on their chemical and valence species, both in tobacco and in smoke. This research addresses the speciation of the metals and metalloids most frequently implicated, particularly those elements that exist in nature in multiple valence states, namely arsenic (As) and chromium (Cr), there being considerable differences in toxicity with oxidation state. A strategy was devised to overcome some of the problems that have thwarted earlier attempts at speciation. Firstly tobacco plants were cultivated under controlled conditions in compost burdened with high levels of metals and metalloids resulting in leaf with up to 250 µg g⁻¹ As, although Cr uptake was less successful. Secondly valence speciation even at the exceptionally low concentrations of As and Cr in smoke from unburdened tobacco was achieved with XANES analysis using the exceptionally bright Diamond synchrotron source. This revealed that combustion of tobacco has a marked effect on valence speciation with As(III), the reduced form of As, dominating (and persisting) in condensate of tobacco smoke while ash is dominated by the oxidised form, As(V). Chromium also appears to be present in smoke mainly as reduced Cr(III) species. HPLC-ICPMS analysis of arsenic indicates the dominance of inorganic over organic species (~4:1). Other metals were investigated in less detail. These findings establish that arsenic is present in smoke in its most toxic form and represents a significant risk to health. Conversely smokers appear to be exposed to the less harmful species of chromium. These results support a recent WHO report that includes As but not Cr in a list of four metals and metalloids recommended for regulation in crops and commercial products in the interests of public health.
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Talabi, Taiwo. « Workers who continue to be occupationally exposed to second-hand tobacco smoke (SHS) in the UK ». Available from the University of Aberdeen Library and Historic Collections Digital Resources. Restricted no access until Sept. 20, 2009, 2008. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=25325.

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Almeida, Nisha. « Measures of maternal tobacco smoke exposure and foetal growth ». Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=112375.

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Objective. Most biomarker studies of maternal smoking have been based on a single blood or urinary cotinine value, which is inadequate in capturing maternal tobacco exposure over the entire pregnancy. This thesis used maternal hair biomarkers to investigate the association between maternal active and passive smoking, and birthweight for gestational age (BW for GA).
Methods. Subjects were 444 term controls drawn from 5,337 participants of a multi-centre nested case-control study of preterm birth in Montreal. Maternal hair, collected after delivery, was measured for average nicotine and cotinine concentration across the pregnancy, assuming hair growth of 1 cm/month. The BW for GA z-score used Canadian population-based standards. Multiple linear regression was used to assess effects on the z-score, after controlling for potential confounders.
Results. In regression models for maternal active smoking analysis, the addition of hair nicotine to models containing either self-report or hair cotinine or both self-report and cotinine explained significantly more variance in the BW for GA z-score (p=0.009, p=0.017, and p=0.033, respectively). In maternal passive smoking analysis, no significant effect of ETS on BW for GA was found using hair biomarkers.
Conclusion. These results indicate that hair biomarkers are sensitive tools capable of predicting reductions in birthweight for maternal active smoking. The stronger results obtained for nicotine are reflective of the fact that hair nicotine is a better measure of maternal smoking, but it could also suggest that nicotine plays an aetiologic role in affecting foetal growth.
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Peter, Justin R. « Numerical investigation of aerosol dynamics : evaluation of the Hamaker constant for environmental tobacco smoke ». Thesis, Queensland University of Technology, 1996. https://eprints.qut.edu.au/36917/1/36917_Peter_1996.pdf.

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Human exposure to atmospheric pollutants (aerosols) depends on the nature and concentration of the pollutants, and also on the size distribution of the particulate phase of the aerosol. One such commonly encountered aerosol of significant physiological importance is Environmental Tobacco Smoke (ETS). Knowledge of the time evolution of combustion aerosols is necessary to understand the interactions which shape the aerosol size distribution, and thus lead to conclusions as to the health risk associated with exposure to ETS. A systematic numerical investigation of the dynamics of aerosol coagulation with the inclusion of van der Waal's forces has been undertaken. Numerical results are compared with those obtained experimentally for the size distribution ofETS in the particle range, 10 nm < particle diameter < 1000 nm. These particles diameters lie in the Cunningham Slip-Flow region, between the free-molecule and continuum regions. The newly developed numerical method allows direct simulation to obtain the size distributions of aging aerosols of arbitrary diameter. Considering that other processes such as condensation and wall deposition are not considered, results obtained theoretically are in excellent agreement with those obtained experimentally. The comparison of experimental and numerical results enables conclusions to be made as the value of the Hamaker constant for ETS. Although the Hamaker constant is only defined for a pure substance, the method allows an average value over all the constituents of ETS to be evaluated. This value can then be used as input for any further modelling ofETS.
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Schulz-Katterbach, Michèle Sabrina. « Cannabis and caries - does regular cannabis use increase the risk of caries in cigarette smokers ? / ». [S.l.] : [s.n.], 2009. http://opac.nebis.ch/cgi-bin/showAbstract.pl?sys=000297946.

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Gudi, Girish Srinivas. « Evaluation of non-invasive biomarkers for carcinogenic exposure to cigarette smoke ». Morgantown, W. Va. : [West Virginia University Libraries], 1999. http://etd.wvu.edu/templates/showETD.cfm?recnum=661.

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Thesis (M.S.)--West Virginia University, 1999.
Title from document title page. Document formatted into pages; contains xi, 107 p. : ill. (some col.) Includes abstract. Includes bibliographical references (p. 98-107).
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Livres sur le sujet "Tobacco Smoke Pollution"

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Massachusetts. Department of Public Health. Secondhand smoke (Environmental tobacco smoke). Boston, MA : Massachusetts Dept. of Public Health, 1988.

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Massachusetts. Department of Public Health. Secondhand smoke (Environmental tobacco smoke). Boston, MA : Massachusetts Dept. of Public Health, 1987.

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R, Watson Ronald, et Witten Mark L. 1953-, dir. Environmental tobacco smoke. Boca Raton, Fla : CRC Press, 2001.

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Farrelly, Matthew C. Youth exposure to environmental tobacco smoke. Washington, DC : American Legacy Foundation, 2001.

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D, Tollison Robert, dir. Clearing the air : Perspectives on environmental tobacco smoke. Lexington, Mass : Lexington Books, 1988.

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California Environmental Protection Agency. Office of Environmental Health Hazard Assessment. et National Cancer Institute (U.S.), dir. Health effects of exposure to environmental tobacco smoke : The report of the California Environmental Protection Agency. [Bethesda, MD] : U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, National Cancer Institute, 1999.

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California Environmental Protection Agency. Office of Environmental Health Hazard Assessment., dir. Health effects of exposure to environmental tobacco smoke : Final report. [Sacramento, Calif.?] : The Office, 1997.

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New Hampshire. Office of Community and Public Health. Secondhand smoke in New Hampshire. Concord, N.H.] : The Dept., 2003.

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IARC Working Group on the Evaluation of Carcinogenic Risks to Humans. Tobacco smoke and involuntary smoking. [Lyon, France] : IARC Press, 2004.

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United States. Environmental Protection Agency., dir. Secondhand smoke : What you can do about secondhand smoke as parents, decisionmakers, and building occupants. [Washington, DC] : EPA, 1996.

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Chapitres de livres sur le sujet "Tobacco Smoke Pollution"

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Wong, C. M., Z. G. Hu, T. H. Lam, A. J. Hedley et J. Peters. « Environmental tobacco smoke, air pollution and respiratory symptoms in non-smoking housewives in Hong Kong ». Dans Tobacco : The Growing Epidemic, 150–53. London : Springer London, 2000. http://dx.doi.org/10.1007/978-1-4471-0769-9_58.

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Hugod, C. « Indoor Air Pollution from Tobacco Smoke as Seen by Scientists in Governmental Administration ». Dans Indoor Air Quality, 390–92. Berlin, Heidelberg : Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-83904-7_46.

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Repace, James L. « Tobacco Smoke Pollution ». Dans Nicotine Addiction : Principles and Management, 129–42. Oxford University PressNew York, NY, 1993. http://dx.doi.org/10.1093/oso/9780195064414.003.0007.

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Abstract Inhaling the smoke from cigarettes, pipes, and cigars delivers nicotine to the brain more quickly and efficently than chewing tobacco leaf (Chapter l}. However, the practice of burning tobacco leaves indoors ex-poses other people to indoor air pollution from tobacco combustion products containing many chemicals known to be harmful to human health. Although society, in the interest of public health, has long imposed quality standards for food, water, and in-deed, for outdoor air, it has been slow to re-quire that the indoor air be of a quality that will prevent morbidity and mortality. In general, the same amount of contaminant deposited on the lung surface from inhaled air that we breathe has greater potential for harm than an equal amount ingested in food or water, due to differences in absorption efficiency between the pulmonary and gastro-intestinal membranes. For example, in a healthy adult, 5% of a gram of lead from a chip of accidentally ingested paint will be absorbed, while 95% of the same amount of lead inhaled as a fume from automobile exhaust will be absorbed.
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Benatar, David. « Smoking ». Dans Very Practical Ethics, 112–44. Oxford University PressNew York, NY, 2024. http://dx.doi.org/10.1093/9780197780831.003.0004.

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Abstract This chapter discusses the ethics of smoking (tobacco). There are ethical questions to be asked even when one smokes alone. However, the chapter focuses on smoking in the presence of non-smokers. There are two kinds of arguments against the permissibility of smoking in the presence of others—a harm-based argument and an offence-based argument. The chapter considers and rejects the objection that the risk increment to non-smokers is too negligible to impose a duty on the smoker. (This involves a comparison with car pollution.) Although offence arguments are typically weaker than harm arguments, it is argued that the particular kind of offence caused by smoke, along with some other factors, can ground a duty not to smoke in the presence of others. Attention is also given to electronic cigarettes, to obtaining consent from non-smokers to smoke in their presence, and to some additional ethical issues to which smoking gives rise.
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Dehghani, Mohammad Hadi, Parnia Bashardoust, Danial Nayeri, Mohammad Rezvani Ghalhari, Niloofar Borhani Yazdi, Farzaneh Jajarmi, Rama Rao Karri et Nabisab Mujawar Mubarak. « A comprehensive review of the potential outcomes of exposure to tobacco smoke or secondhand smoke ». Dans Health Effects of Indoor Air Pollution, 167–89. Elsevier, 2024. http://dx.doi.org/10.1016/b978-0-443-16090-5.00004-0.

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S, Megan, Mark A, Eric Dubuis et Maria G. « Tobacco Smoke Induced Cough : Mechanisms Driving Acute and Chronic Cough Pathology ». Dans Advanced Topics in Environmental Health and Air Pollution Case Studies. InTech, 2011. http://dx.doi.org/10.5772/21907.

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Münzel, Thomas, Sanjay Rajagopalan, Mette Sørenson, Dave Newby et Robert D. Brook. « Introduction ». Dans ESC CardioMed, sous la direction de Thomas Münzel, 3099–101. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0748.

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Cardiovascular disease represents the result of underlying genetic predisposition and lifetime exposure to multiple environmental factors. The past century has seen a revolution in our understanding of the importance of modifiable risk factors such as diet, exercise, and smoking. Exposure to environmental pollutants, be it in the air, water, or physical environment, is increasingly recognized as a silent, yet important determinant of cardiovascular disease. The cardiovascular system is highly vulnerable to a variety of environmental insults, including tobacco smoke, solvents, pesticides, and other inhaled or ingested pollutants, as well as extremes in noise and temperature. While our understanding of multiple environmental factors continues to evolve, it is estimated that environmental air pollution and noise pollution alone contribute a substantial burden attributable to environmental factors as we currently understand them.
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Navratilova, Zdenka, Eva Kominkova et Martin Petrek. « The Immune Response in the Pathophysiology of Pulmonary Diseases ». Dans Human Physiology - Annual Volume 2023 [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.112587.

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Chronic obstructive pulmonary disease (COPD) is the world’s third leading cause of death. The number of patients with asthma is increasing in developed countries. We review here the main features of pathophysiology in these obstructive diseases. Tobacco smoke and other air pollution stimulate chronic inflammation in COPD. Asthma is a type 1 hypersensitivity that is a response to various allergens. In both pathologies, chronic inflammatory response leads to airway remodeling, significantly impacting lung function and a patient’s daily activity. Besides imaging techniques, a critical diagnostic tool is a pulmonary function test with characteristic obstructive patterns and respiratory symptoms. Sarcoidosis is discussed as an example of a restrictive disease. Finally, we shortly highlight the direction of current research.
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Prescott, Eva, Torben Jørgensen, Maja-Lisa Løchen, Jonathan Pearson-Stuttard et Simon Capewell. « Risk factor intervention at the population level ». Dans ESC CardioMed, sous la direction de Massimo Piepoli, 873–82. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0210.

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Great health benefits can be achieved if preventive efforts are focused on whole populations rather than mainly on high-risk individuals. Tobacco smoking, unhealthy diet, physical inactivity, alcohol abuse, and air pollution are responsible for a large proportion of global deaths and loss of disability-adjusted life years and there are documented effects of interventions at the population level on all of these factors. Population-based strategies include fiscal measures (i.e. taxation and subsidies), international, national, and regional policy regulations and legislation (e.g. smoke-free policies, rules for marketing, food production), and environmental changes (e.g. access to recreational areas, convenience stores), and have the advantages of addressing cardiovascular health over the entire life course, reducing health inequalities and saving costs when compared to individual-level interventions.
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Vilcins, Dwan, et Peter D. Sly. « Asthma, Allergy, and the Environment ». Dans Textbook of Children's Environmental Health, 650–60. 2e éd. Oxford University PressNew York, 2024. http://dx.doi.org/10.1093/oso/9780197662526.003.0048.

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Abstract Asthma is disease in which the normal development of the respiratory and immune systems is altered by interactions between harmful environmental exposures and underlying genetic predispositions. The major risk factors for asthma in early life are a family history of asthma and allergies, reflecting a genetic predisposition; sensitization to perennial aeroallergens; and recurrent severe lower respiratory infections, especially those associated with fever and/or wheeze. Reduced lung function appears to be an independent risk factor for asthma. Environmental exposures—either prenatal exposures or exposures in early postnatal life—that limit lung growth or delay maturation of the immune system further increase the risk of asthma. Although environmental controls may not truly prevent asthma, strategies such as eliminating exposure of pregnant women, infants, and young children to tobacco smoke, air pollution, and ambient pollutants are likely to decrease asthma.
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Actes de conférences sur le sujet "Tobacco Smoke Pollution"

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Geens, A. J., H. Al-Madfai et D. G. Snelson. « Impacts of ventilation : studies on “environmental tobacco smoke” ». Dans AIR POLLUTION 2008. Southampton, UK : WIT Press, 2008. http://dx.doi.org/10.2495/air080491.

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Vanker, Aneesa, Whitney Barnett, Polite M. Nduru, Kirsty Brittain, Robert P. Gie et Heather J. Zar. « Indoor air pollution and tobacco smoke exposure in an African birth cohort study ». Dans Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa4510.

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Chaya, Shaakira, Rae Macginty, Zoltan Hantos, Carvern Jacobs, Aneesa Vanker, Graham Hall, Heather Zar et Diane Gray. « Indoor pollution or tobacco smoke exposure and lung function at 3 years in an African birth cohort ». Dans ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.oa1597.

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van Gemert, Frederik, Evelyn Brakema, Rianne Van Der Kleij, Sian Williams, Rupert Jones, Bruce Kirenga, Shamim Buteme et al. « Development and implementation of an awareness programme addressing household air pollution and tobacco smoke : a FRESH AIR project ». Dans ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa728.

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Vanker, Aneesa, Felix Dube, Whitney Barnett, Polite Nduru, Robert Gie, Mark Nicol et Heather Zar. « Impact of tobacco smoke exposure or indoor air pollution on nasopharyngeal bacteria in African infants in a birth cohort study ». Dans ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa1857.

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Biagini Myers, Jocelyn M., Gurjit K. Khurana Hershey et Grace K. LeMasters. « Environmental Tobacco Smoke (ETS) Exposure Histories In Children - Are We Asking The Right Questions ? The Cincinnati Childhood Allergy And Air Pollution Study (CCAAPS) ». Dans American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4032.

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