Relevant bibliographies by topics / ABCA1/ABCG1

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'ABCA1/ABCG1'

Author: Grafiati
Published: 6 July 2024
Last updated: 31 July 2025

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Journal articles on the topic "ABCA1/ABCG1"

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Esobi, Ikechukwu, Oladosu Olanrewaju, Jing Echesabal-Chen, and Alexis Stamatikos. "Utilizing the LoxP-Stop-LoxP System to Control Transgenic ABC-Transporter Expression In Vitro." Biomolecules 12, no. 5 (2022): 679. http://dx.doi.org/10.3390/biom12050679.

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ABCA1 and ABCG1 are two ABC-transporters well-recognized to promote the efflux of cholesterol to apoAI and HDL, respectively. As these two ABC-transporters are critical to cholesterol metabolism, several studies have assessed the impact of ABCA1 and ABCG1 expression on cellular cholesterol homeostasis through ABC-transporter ablation or overexpressing ABCA1/ABCG1. However, for the latter, there are currently no well-established in vitro models to effectively induce long-term ABC-transporter expression in a variety of cultured cells. Therefore, we performed proof-of-principle in vitro studies t
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Tavoosi, Zahra, Hemen Moradi-Sardareh, Massoud Saidijam, et al. "Cholesterol Transporters ABCA1 and ABCG1 Gene Expression in Peripheral Blood Mononuclear Cells in Patients with Metabolic Syndrome." Cholesterol 2015 (December 15, 2015): 1–6. http://dx.doi.org/10.1155/2015/682904.

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ABCA1 and ABCG1 genes encode the cholesterol transporter proteins that play a key role in cholesterol and phospholipids homeostasis. This study was aimed at evaluating and comparing ABCA1 and ABCG1 genes expression in metabolic syndrome patients and healthy individuals. This case-control study was performed on 36 patients with metabolic syndrome and the same number of healthy individuals in Hamadan (west of Iran) during 2013-2014. Total RNA was extracted from mononuclear cells and purified using RNeasy Mini Kit column. The expression of ABCA1 and ABCG1 genes was performed by qRT-PCR. Lipid pro
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Miroshnikova, V. V., A. A. Panteleeva, E. A. Bazhenova, et al. "Regulation of ABCA1 and ABCG1 gene expression in the intraabdominal adipose tissue." Biomeditsinskaya Khimiya 62, no. 3 (2016): 283–89. http://dx.doi.org/10.18097/pbmc20166203283.

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Tissue specific expression of genes encoding cholesterol transporters ABCA1 and ABCG1 as well as genes encoding the most important transcriptional regulators of adipogenesis – LXRa, LXRb, PPARg and RORa has been investigated in intraabdominal adipose tissue (IAT) samples.A direct correlation between the content of ABCA1 and ABCG1 proteins with RORa protein level (r=0.480, p<0.05; r=0.435, p<0.05, respectively) suggests the role of the transcription factor RORa in the regulation of IAT ABCA1 and ABCG1 protein levels. ABCA1 and ABCG1 gene expression positively correlated with obesity indic
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Porsch-Özcürümez, Mustafa, Thomas Langmann, Susanne Heimerl, et al. "The Zinc Finger Protein 202 (ZNF202) Is a Transcriptional Repressor of ATP Binding Cassette Transporter A1 (ABCA1) and ABCG1 Gene Expression and a Modulator of Cellular Lipid Efflux." Journal of Biological Chemistry 276, no. 15 (2001): 12427–33. http://dx.doi.org/10.1074/jbc.m100218200.

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The zinc finger gene 202 (ZNF202) located within a hypoalphalipoproteinemia susceptibility locus on chromosome 11q23 is a transcriptional repressor of various genes involved in lipid metabolism. To provide further evidence for a functional linkage between ZNF202 and hypoalphalipoproteinemia, we investigated the effect of ZNF202 expression on ATP binding cassette transporter A1 (ABCA1) and ABCG1. ABCA1 is a key regulator of the plasma high density lipoprotein pool size, whereas ABCG1 is another mediator of cellular cholesterol and phospholipid efflux in human macrophage. We demonstrate here tha
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Cheng-Mao, Xie, Long Yan, Lin Li, Jin Hua, Wang Xiao-Ju, and Zhang Jie-Wen. "Placental ABCA1 Expression Is Increased in Spontaneous Preterm Deliveries Compared with Iatrogenic Preterm Deliveries and Term Deliveries." BioMed Research International 2017 (2017): 1–9. http://dx.doi.org/10.1155/2017/8248094.

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Objective. Abnormal expression of ABCA1 and ABCG1 in the placenta can elicit lipid metabolism disorder and adverse pregnancy outcomes. However, whether it is associated with preterm delivery remains unclear. Our present study aimed to evaluate the relationship between abnormal expression of ABCA1 or ABCG1 and preterm delivery. Methods. Maternal blood and placental tissues from women with spontaneous deliveries (SPD), iatrogenic deliveries (IPD), and term deliveries (TD) were collected. The lipid content and expression of ABCA1 and ABCG1 were subsequently measured. Results. Compared with IPD an
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Oladosu, Olanrewaju, Ikechukwu C. Esobi, Rhonda R. Powell, Terri Bruce, and Alexis Stamatikos. "Dissecting the Impact of Vascular Smooth Muscle Cell ABCA1 versus ABCG1 Expression on Cholesterol Efflux and Macrophage-like Cell Transdifferentiation: The Role of SR-BI." Journal of Cardiovascular Development and Disease 10, no. 10 (2023): 416. http://dx.doi.org/10.3390/jcdd10100416.

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Cholesterol-laden macrophages are recognized as a major contributor to atherosclerosis. However, recent evidence indicates that vascular smooth muscle cells (VSMC) that accumulate cholesterol and transdifferentiate into a macrophage-like cell (MLC) phenotype also play a role in atherosclerosis. Therefore, removing cholesterol from MLC may be a potential atheroprotective strategy. The two transporters which remove cholesterol from cells are ABCA1 and ABCG1, as they efflux cholesterol to apoAI and HDL, respectively. In this study, the well-characterized immortalized VSMC line MOVAS cells were ed
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Mani, Orlando, Meike Körner, Martin T. Sorensen, et al. "Expression, localization, and functional model of cholesterol transporters in lactating and nonlactating mammary tissues of murine, bovine, and human origin." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 299, no. 2 (2010): R642—R654. http://dx.doi.org/10.1152/ajpregu.00723.2009.

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Members of the ATP-binding cassette (ABC) transporters play a pivotal role in cellular lipid efflux. To identify candidate cholesterol transporters implicated in lipid homeostasis and mammary gland (MG) physiology, we compared expression and localization of ABCA1, ABCG1, and ABCA7 and their regulatory genes in mammary tissues of different species during the pregnancy-lactation cycle. Murine and bovine mammary glands (MGs) were investigated during different functional stages. The abundance of mRNAs was determined by quantitative RT-PCR. Furthermore, transporter proteins were localized in murine
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Lin, Hung-Chih, Chong-Kuei Lii, Hui-Chun Chen, Ai-Hsuan Lin, Ya-Chen Yang, and Haw-Wen Chen. "Andrographolide Inhibits Oxidized LDL-Induced Cholesterol Accumulation and Foam Cell Formation in Macrophages." American Journal of Chinese Medicine 46, no. 01 (2018): 87–106. http://dx.doi.org/10.1142/s0192415x18500052.

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oxLDL is involved in the pathogenesis of atherosclerotic lesions through cholesterol accumulation in macrophage foam cells. Andrographolide, the bioactive component of Andrographis paniculata, possesses several biological activities such as anti-inflammatory, anti-oxidant, and anticancer functions. Scavenger receptors (SRs), including class A SR (SR-A) and CD36, are responsible for the internalization of oxLDL. In contrast, receptors for reverse cholesterol transport, including ABCA1 and ABCG1, mediate the efflux of cholesterol from macrophage foam cells. Transcription factor liver X receptor
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Мирошникова, В. В., А. А. Пантелеева, И. А. Побожева та ін. "Аdipose tissue expression of ABCA1 and ABCG1 transporters genes in obesity, metabolic syndrome and ischemic heart disease". Nauchno-prakticheskii zhurnal «Medicinskaia genetika», № 5(214) (29 травня 2020): 56–57. http://dx.doi.org/10.25557/2073-7998.2020.05.56-57.

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Ожирение ассоциировано с повышенным риском развития метаболических нарушений и сердечно-сосудистых заболеваний. В нашем исследовании мы показали, что экспрессия генов транспортеров холестерина ABCA1 и ABCG1 в жировой ткани может играть роль в развитии ожирения, дислипидемии, метаболического синдрома и ишемической болезни сердца (ИБС). Obesity is linked to increased cardiometabolic risk. Our study shows that cholesterol transporters ABCA1 and ABCG1 gene expression plays a role in development of obesity, dyslipidemia, metabolic syndrome and ischemic heart disease.
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Delvecchio, Christopher J., Patricia Bilan, Parameswaran Nair, and John P. Capone. "LXR-induced reverse cholesterol transport in human airway smooth muscle is mediated exclusively by ABCA1." American Journal of Physiology-Lung Cellular and Molecular Physiology 295, no. 5 (2008): L949—L957. http://dx.doi.org/10.1152/ajplung.90394.2008.

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The association of hypercholesterolemia and obesity with airway hyperresponsiveness has drawn increasing attention to the potential role of cholesterol and lipid homeostasis in lung physiology and in chronic pulmonary diseases such as asthma. We have recently shown that activation of the nuclear hormone receptor liver X receptor (LXR) stimulates cholesterol efflux in human airway smooth muscle (hASM) cells and induces expression of the ATP-binding cassette (ABC) transporters ABCA1 and ABCG1, members of a family of proteins that mediate reverse cholesterol and phospholipid transport. We show he
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Dissertations / Theses on the topic "ABCA1/ABCG1"

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Matsuda, Akihiro. "(24S)-Hydroxycholesterol efflux from neuronal cells by ABC proteins." Kyoto University, 2014. http://hdl.handle.net/2433/185211.

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Kyoto University (京都大学)<br>0048<br>新制・課程博士<br>博士(農学)<br>甲第17986号<br>農博第2033号<br>新制||農||1019(附属図書館)<br>学位論文||H26||N4811(農学部図書室)<br>80830<br>京都大学大学院農学研究科応用生命科学専攻<br>(主査)教授 植田 和光, 教授 植田 充美, 教授 三芳 秀人<br>学位規則第4条第1項該当
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Brüggmann, Nina [Verfasser]. "Untersuchungen zum ABCA1-/ABCG1-vermittelten Cholesterin-Efflux in humanen Kontroll- und Tangierfibroblasten / Nina Brüggmann." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2021. http://d-nb.info/1228861153/34.

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Zago, Vanessa Helena de Souza 1984. "Estudo molecular dos genes ABCA1, ABCG1, ABCG5, ABCG8 e SCARB1 em amostra populacional brasileira assintomática." [s.n.], 2015. http://repositorio.unicamp.br/jspui/handle/REPOSIP/312594.

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Orientadores: Eliana Cotta de Faria, Helena Coutinho Franco de Oliveira, Daniel Zanetti Scherrer<br>Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-26T20:49:38Z (GMT). No. of bitstreams: 1 Zago_VanessaHelenadeSouza_D.pdf: 5059555 bytes, checksum: 854d9d1d1674a14d2ebcf5798acc31b3 (MD5) Previous issue date: 2015<br>Resumo: Dado o importante papel desempenhado pelos transportadores ATP binding cassete A1 (ABCA1), G1 (ABCG1), G5 (ABCG5), G8 (ABCG8) e pelo scavenger receptor class B type I (SR-BI) para a homeostase corpóre
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Monzel, Judith Verena [Verfasser]. "Regulation der Cholesteroltransporter ABCA1 und ABCG1 im Kontext der Doxorubicin-induzierten Kardiotoxizität / Judith Verena Monzel." Greifswald : Universitätsbibliothek Greifswald, 2017. http://d-nb.info/1143131673/34.

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Aleidi, Shereen Mohammad Suleimann. "Characterization of the Post-Translational Regulation of the ABCA1 and ABCG1 Lipid Transporters by E3 Ligases." Thesis, The University of Sydney, 2016. http://hdl.handle.net/2123/15477.

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This thesis present a series of experimental approaches that aim to characterize the roles of three E3 ubiquitin ligase enzymes in the post-translational regulation of two ABC lipid transporters; ABCA1 and ABCG1. These E3 ligases, namely HECTD1 ( Homologous to E6Ap C-terminus Domain 1), HUWE1 (HECT, UBA, and WWE domain containing 1), and NEDD4-1 (Neural precursor cell-expressed developmentally down regulated gene 4), were identified during a previous work in our laboratory using peptide mass spectrometry analysis as associate proteins with ABCG1. The experiments presented in the following chap
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Guilbaud, Emma. "Nouvelles altérations métaboliques des cancers bronchiques non à petites cellules : rôle de l’efflux du cholestérol et de la mitophagie." Electronic Thesis or Diss., Université Côte d'Azur, 2021. http://theses.univ-cotedazur.fr/2021COAZ6021.

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Le cancer bronchique non à petites cellules (CBNPC), représentant jusqu'à 85% des cas de cancer de poumon, est la principale cause de décès par cancer dans le monde. La majorité des patients sont diagnostiqués à un stade avancé de cancer et malgré les progrès thérapeutiques, le taux moyen de survie à 5 ans est d’environ 20%. De ce fait, une meilleure compréhension de la biologie des cellules cancéreuses et l'exploration des vulnérabilités des CBNPC devraient ouvrir de nouvelles voies pour le développement de stratégies thérapeutiques. Dans ce contexte, je me suis intéressée aux altérations mét
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Teixeira, Mayza Dalcin. "Estudo da associação de polimorfismos dos genes FTO, ABCA1, ABCA7 e ABCG1 com marcadores de obesidade e perfil lipídico em mulheres obesas." reponame:Repositório Institucional da UFPR, 2017. http://hdl.handle.net/1884/47563.

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Orientadora : Profª. Drª. Lupe Furtado-Alle<br>Coorientadora : Drª Luciane Viater Tureck<br>Dissertação (mestrado) - Universidade Federal do Paraná, Setor de Ciências Biológicas, Programa de Pós-Graduação em Genética. Defesa: Curitiba, 31/03/2017<br>Inclui referências : f. 85-88<br>Resumo: A maioria dos casos de obesidade e de dislipidemias possui origem complexa, pois é resultante da interação entre fatores genéticos e ambientais. Diversos genes têm sido relacionados com a susceptibilidade a estas doenças, incluindo variantes alélicas dos genes FTO, ABCA1, ABCA7 e ABCG1. Desse modo, o objetiv
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Nascimento, Gabrielle Araújo do. "Avaliação do efeito de polimorfismos nos genes FTO, ABCA1, ABCA7 e ABCG1 sobre indicadores de obesidade e dislipidemias em crianças e adolescentes submetidos a treinamentos físico." reponame:Repositório Institucional da UFPR, 2017. http://hdl.handle.net/1884/47393.

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Orientadora : Profª Drª Luciane Viater Tureck<br>Coorientadora : Profª Drª Lupe Furtado Alle<br>Dissertação (mestrado) - Universidade Federal do Paraná, Setor de Ciências Biológicas, Programa de Pós-Graduação em Genética. Defesa: Curitiba, 27/03/2017<br>Inclui referências<br>Resumo: A obesidade e as dislipidemias geralmente estão associadas, e na maior parte dos casos possuem origem complexa, sendo decorrentes da interação entre os fatores ambientais e fatores genéticos. Dentre os fatores genéticos já conhecidos encontram-se genes relacionados ao metabolismo, como o gene FTO (Fat Mass and Obes
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Book chapters on the topic "ABCA1/ABCG1"

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Yu, Xiao-Hua, and Chao-Ke Tang. "ABCA1, ABCG1, and Cholesterol Homeostasis." In HDL Metabolism and Diseases. Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-1592-5_7.

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Terasaka, Naoki. "Sterol Efflux by ABCA1 and ABCG1." In The HDL Handbook. Elsevier, 2010. http://dx.doi.org/10.1016/b978-0-12-382171-3.10010-5.

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Yuan, Z., X. Tan, X. Xu, et al. "Arsenic impaired cholesterol efflux by inhibiting ABCA1 and ABCG1." In Arsenic in the Environment - Proceedings. CRC Press, 2016. http://dx.doi.org/10.1201/b20466-203.

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