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Relevant bibliographies by topics / Alcoholic Liver cirrhosis

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Academic literature on the topic 'Alcoholic Liver cirrhosis'

Author: Grafiati

Published: 4 June 2021

Last updated: 26 July 2025

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Journal articles on the topic "Alcoholic Liver cirrhosis"

1

Bell, Helge, Nils Raknerud, Jan A. Falch, and Egil Haug. "Inappropriately low levels of gonadotrophins in amenorrhoeic women with alcoholic and non-alcoholic cirrhosis." European Journal of Endocrinology 132, no. 4 (1995): 444–49. http://dx.doi.org/10.1530/eje.0.1320444.

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Bell H, Raknerud N, Falch JA, Haug E. Inappropriately low levels of gonadotrophins in amenorrhoeic women with alcoholic and non-alcoholic cirrhosis. Eur J Endocrinol 1995;132:444–9. ISSN 0804–4643 We investigated a group of 111 amenorrhoeic females with associated liver disease. These comprised alcoholic cirrhotics (N = 38), non-alcoholic cirrhotics (N = 12), non-cirrhotic alcoholics (N = 21) and those suffering from other chronic liver diseases (N = 40) admitted to our medical department from 1986 to 1991. The serum levels of luteinizing hormone (LH) and follicle-stimulating hormone (FSH), oe

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Johansson, U., F. Johnsson, B. Joelsson, M. Berglund, and B. Åkesson. "Selenium status in patients with liver cirrhosis and alcoholism." British Journal of Nutrition 55, no. 2 (1986): 227–33. http://dx.doi.org/10.1079/bjn19860029.

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1. Selenium status and blood levels of other nutrients related to lipid peroxidation were studied in patients with advanced alcoholic liver cirrhosis, in male alcoholics who had been abstinent for at least 1 month and in healthy control subjects.2. Plasma Se was decreased in alcoholic cirrhosis but not among alcoholics in abstinence. Platelet glutathione peroxidase (EC 1. 11. 1.9; GSH-Px) activity was decreased in cirrhotic patients, whereas GSH-Px in blood and plasma was the same as in controls for both groups of patients.3. Plasma retinol and plasma α-tocopherol were decreased in alcoholic c

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Prystupa, Andrzej, Paweł Kiciński, Jarosław Sak, Anna Boguszewska-Czubara, Anna Toruń-Jurkowska та Wojciech Załuska. "Proinflammatory Cytokines (IL-1α, IL-6) and Hepatocyte Growth Factor in Patients with Alcoholic Liver Cirrhosis". Gastroenterology Research and Practice 2015 (2015): 1–7. http://dx.doi.org/10.1155/2015/532615.

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Background. The aim of the study was to assess the activity of interleukin-1α, interleukin-6, and hepatocyte growth factor protein (HGF) in serum of patients with alcoholic liver cirrhosis.Materials and Methods. Sixty patients with alcoholic liver cirrhosis treated in various hospitals were randomly enrolled. The stage of cirrhosis was assessed according to the Child-Turcotte-Pugh scoring system. The control group consisted of ten healthy persons without liver disease, who did not drink alcohol. Additionally, the group of alcoholics without liver cirrhosis was included in the study. The activi

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Chang, Binxia, Baosen Li, Ying Sun, et al. "Changes in Etiologies of Hospitalized Patients with Liver Cirrhosis in Beijing 302 Hospital from 2002 to 2013." Mediators of Inflammation 2017 (2017): 1–5. http://dx.doi.org/10.1155/2017/5605981.

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Background. Over the last 20 years, the prevalence of hepatitis B virus (HBV) infection in China has decreased gradually due to the application of a national HBV vaccination program. In contrast, the prevalence of alcoholic liver disease (ALD), nonalcoholic fatty liver disease, autoimmune liver disease, and drug-induced liver injury has markedly increased. Methods. We conducted a retrospective review of 82,562 hospitalized patients diagnosed with liver cirrhosis in Beijing 302 Hospital from 2002 to 2013. Results. The top four etiologies of cirrhosis were HBV, HCV, ALD, and autoimmune liver dis

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Zhang, Zhenzhen, Guomin Xie, Li Liang, et al. "RIPK3-Mediated Necroptosis and Neutrophil Infiltration Are Associated with Poor Prognosis in Patients with Alcoholic Cirrhosis." Journal of Immunology Research 2018 (November 25, 2018): 1–7. http://dx.doi.org/10.1155/2018/1509851.

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Alcoholic cirrhosis is an end-stage liver disease with impaired survival and often requires liver transplantation. Recent data suggests that receptor-interacting protein kinase-3- (RIPK3-) mediated necroptosis plays an important role in alcoholic cirrhosis. Additionally, neutrophil infiltration is the most characteristic pathologic hallmark of alcoholic hepatitis. Whether RIPK3 level is correlated with neutrophil infiltration or poor prognosis in alcoholic cirrhotic patients is still unknown. We aimed to determine the correlation of RIPK3 and neutrophil infiltration with the prognosis in the e

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Silveira, Luciano Alves Matias da, Bianca Gonçalves Silva Torquato, Mariana Silva Oliveira, et al. "Implications of alcoholic cirrhosis in atherosclerosis of autopsied patients." Revista da Associação Médica Brasileira 63, no. 4 (2017): 336–40. http://dx.doi.org/10.1590/1806-9282.63.04.336.

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Summary Introduction: Alcoholism is a major public health problem, which has a high social cost and affects many aspects of human activity. Liver disease is one of the first consequences of alcohol abuse, and steatosis, liver cirrhosis and hepatitis may occur. Other organs are also affected with pathological changes, such as pancreatitis, cardiomyopathies, dyslipidemias and atherosclerosis. Objective: To identify the occurrence and degree of atherosclerosis in alcohol-dependent individuals with liver cirrhosis, observing macroscopic and microscopic changes in lipid and collagen deposits and in

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Gaurav R. Waghmare, Vinayak A. Katekar, and Swati P. Deshmukh. "A review: Alcoholic liver disease pathophysiology, current molecular and clinical perspectives." GSC Biological and Pharmaceutical Sciences 25, no. 2 (2023): 014–24. http://dx.doi.org/10.30574/gscbps.2023.25.2.0424.

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Alcohol-related liver damage (ALD) is a term used for a variety of liver conditions comprising cirrhosis, fibrosis, and steatohepatitis with chronic inflammation that is predominantly brought on by heavy alcohol consumption. ALD is currently regarded as one of the most common causes of liver disease-related mortality on a global scale. The majority of cirrhosis in the liver cases seen in district general hospitals in the UK are caused by alcohol, which is a key factor in the Western world. Alcoholic cirrhosis, acute alcoholic hepatitis, and alcoholic fatty liver (steatosis) are the three most

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Gaurav, R. Waghmare, A. Katekar Vinayak, and P. Deshmukh Swati. "A review: Alcoholic liver disease pathophysiology, current molecular and clinical perspectives." GSC Biological and Pharmaceutical Sciences 25, no. 2 (2023): 014–24. https://doi.org/10.5281/zenodo.10579109.

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Alcohol-related liver damage (ALD) is a term used for a variety of liver conditions comprising cirrhosis, fibrosis, and steatohepatitis with chronic inflammation that is predominantly brought on by heavy alcohol consumption. ALD is currently regarded as one of the most common causes of liver disease-related mortality on a global scale. The majority of cirrhosis in the liver cases seen in district general hospitals in the UK are caused by alcohol, which is a key factor in the Western world. Alcoholic cirrhosis, acute alcoholic hepatitis, and alcoholic fatty liver (steatosis) are the three most

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Girleanu, Irina, Anca Trifan, Laura Huiban, et al. "The Risk of Clostridioides difficile Infection in Cirrhotic Patients Receiving Norfloxacin for Secondary Prophylaxis of Spontaneous Bacterial Peritonitis—A Real Life Cohort." Medicina 57, no. 9 (2021): 964. http://dx.doi.org/10.3390/medicina57090964.

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Background and Objectives: Spontaneous bacterial peritonitis (SBP) is a life-threatening complication of liver cirrhosis. Antibiotic prophylaxis is effective but can lead to an increased incidence of Clostridioides difficile infection (CDI). The aim of this study was to evaluate the incidence of CDI and the risk factors in cirrhotic patients with a previous episode of SBP receiving norfloxacin as secondary prophylaxis. Materials and Methods: We performed a prospective, cohort study including patients with liver cirrhosis and SBP, successfully treated over a 2-year period in a tertiary universi

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Anaize Tonon do Amaral, Joice, Renata Salatini, Claudia Arab, et al. "Non-Alcoholic Cirrhosis and Heart Rate Variability: A Systematic Mini-Review." Medicina 56, no. 3 (2020): 116. http://dx.doi.org/10.3390/medicina56030116.

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Background and Objectives: Cirrhosis is a liver disease that causes about one million deaths annually worldwide. The estimated cirrhosis prevalence ranges from 4.5–9.5% in the general population. Up to 40% of cirrhotic patients are asymptomatic and may be diagnosed late. Studies have described the importance of the functions of the liver and autonomic nervous system (ANS) and their relationship. There is limited information available on non-alcoholic cirrhosis and heart rate variability (HRV), which is a measure of the ANS. This study aimed to evaluate cardiac autonomic modulation through HRV

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Dissertations / Theses on the topic "Alcoholic Liver cirrhosis"

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Stokkeland, Knut. "Studies on alcoholic liver disease /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-853-3/.

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Le, Moine Olivier. "Interleukin-10 in liver ischaemia-reperfusion injury and alcoholic cirrhosis." Doctoral thesis, Universite Libre de Bruxelles, 1996. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/212296.

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Gunnarsdóttir, Steingerður Anna /. "Liver cirrhosis : epidemiological and clinical aspects /." Göteborg : Section of Gastroenterology and Hepatology, Department of Internal Medicine, Sahlgrenska University Hospital, Göteborg University, 2008. http://hdl.handle.net/2077/10132.

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Virstiuk, N. O., I. Kobitovuch, Nataliia Oleksyivna Slyvka, and O. V. Virstiuk. "N-terminal pro-brain natriuretic peptide and prognosis of alcoholic liver cirrhosis course." Thesis, The international liver congress. - Barselona, Spain, 13-17.04.2016, 2016. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/11745.

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Satishchandran, Abhishek. "The Mechanistic Role and Therapeutic Potential of microRNA-122 in Alcoholic Liver Disease: A Dissertation." eScholarship@UMMS, 2016. https://escholarship.umassmed.edu/gsbs_diss/838.

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Chronic alcohol use results in accelerated liver injury, leading to alcoholic steatohepatitis, cirrhosis, and hepatocellular carcinoma. However, due to the complex nature of this disease process, a central, druggable mechanism has remained elusive. microRNAs are potent post-transcriptional regulators of gene expression. A single miRNA has the ability to regulate hundreds of pathways simultaneously, defining cellular fate and function. microRNA-122 (miR-122), the most abundant miRNA in hepatocytes, has a demonstrated role as an tumor suppressor, regulator of hepatocyte metabolism, and hepatic d

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Satishchandran, Abhishek. "The Mechanistic Role and Therapeutic Potential of microRNA-122 in Alcoholic Liver Disease: A Dissertation." eScholarship@UMMS, 2004. http://escholarship.umassmed.edu/gsbs_diss/838.

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Chronic alcohol use results in accelerated liver injury, leading to alcoholic steatohepatitis, cirrhosis, and hepatocellular carcinoma. However, due to the complex nature of this disease process, a central, druggable mechanism has remained elusive. microRNAs are potent post-transcriptional regulators of gene expression. A single miRNA has the ability to regulate hundreds of pathways simultaneously, defining cellular fate and function. microRNA-122 (miR-122), the most abundant miRNA in hepatocytes, has a demonstrated role as an tumor suppressor, regulator of hepatocyte metabolism, and hepatic d

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Fassnacht, Ryan. "Molecular Mechanisms Involved Involved in the Interaction Effects of HCV and Ethanol on Liver Cirrhosis." VCU Scholars Compass, 2010. http://scholarscompass.vcu.edu/etd/2246.

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The leading causes of liver disease are Hepatitis C virus infection and chronic alcohol abuse. Alcohol accelerates liver disease in HCV but the mechanisms are poorly understood. The identification of molecular gene expression profiles on human liver tissue was performed using microarrays. Samples were obtained from alcoholic-cirrhotic, HCV-cirrhotic, HCV/alcohol-cirrhotic and control non-cirrhotic liver tissue. Probe set expression summaries were calculated using RMA. Probe set level linear models were fit where probe set expression was modeled by HCV status, alcohol status, and the interactio

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Slyvka, N. O., N. G. Virstiuk, I. A. Plesh, and V. A. Gaidukov. "Low cardiac output predicts development of hepatorenal syndrome and survival in patients with alcoholic liver cirrhosis." Thesis, Матерiали 97-ї пiдсумковоi' наукової конференцiї професорсько-викладацького персоналу вищого державного навчального закладу України «Буковинський державний медичний унiверситет» (Чернiвцi, 15, 17 ,22 лютого 2016 р.) - Чернiвцi: Медунiверситет, 2016, 2016. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/10380.

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Kruger, F. C. "A study of non-alcoholic fatty liver disease (NAFLD) in South African patients and analysis of candidate genes in insulin resistance and fatty acid oxidation." Thesis, Stellenbosch : Stellenbosch University, 2008. http://hdl.handle.net/10019.1/1415.

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Thesis (PhD (Medicine. Internal Medicine))--Stellenbosch University, 2008.<br>Non-alcoholic fatty liver disease (NAFLD) is the most prevalent chronic liver disease in Western countries, extending from steatosis (FLD) to steatohepatitis (NASH). Differentiation between NASH and nonprogressive NAFLD is difficult on clinical grounds therefore a need exists to identify reliable biomarkers of disease progression. The aims of the study were 1) to describe the disease profile of NAFLD/NASH in South African patients of the Western Cape, 2) to investigate the metabolic derangements associated with

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Trepo, Eric. "Role of genetic factors in the progression of fibrosis in alcoholic liver disease and chronic hepatitis C." Doctoral thesis, Universite Libre de Bruxelles, 2012. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209659.

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La maladie alcoolique du foie (MAF) et l’hépatite C chronique (HCC) sont les causes les plus fréquentes de cirrhose, carcinome hépatocellulaire (CHC) et transplantation hépatique dans les pays industrialisés. La fibrose hépatique est le stigmate lésionnel de la progression de la maladie vers la cirrhose comme dans toutes les hépatopathies chroniques. Certains facteurs de risque cliniques environnementaux ont été identifiés. Toutefois, ils n’expliquent pas l’extrême variabilité individuelle de la progression de la fibrose. L’influence ethnique à développer une maladie plus sévère ainsi que les

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Books on the topic "Alcoholic Liver cirrhosis"

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Dickerson, James L. Cirrhosis: An essential guide for the newly diagnosed. Marlow, 2006.

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Miron, Jeffrey A. The effect of alcohol prohibition on alcohol consumption. National Bureau of Economic Research, 1999.

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Dills, Angela K. Alcohol prohibition and cirrhosis. National Bureau of Economic Research, 2003.

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Fe, Caces M., Stinson Frederick S, Elliott Steven D, and National Institute on Alcohol Abuse and Alcoholism (U.S.). Alcohol Epidemiologic Data System, eds. County alcohol problem indicators, 1979-1985. 3rd ed. U.S. Dept. of Health and Human Services, Public Health Service, Alcohol, Drug Abuse, and Mental Health Administration, National Institute on Alcohol Abuse and Alcoholism, 1991.

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Yokoe, Ryosuke. Alcohol and Liver Cirrhosis in Twentieth-Century Britain. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-27107-6.

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S, Stinson Frederick, Proudfit Ann H, and National Institute on Alcohol Abuse and Alcoholism (U.S.). Alcohol Epidemiologic Data System., eds. U.S. alcohol epidemiologic data reference manual. 4th ed. the Institute, 1994.

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Shannon, Joyce Brennfleck. Alcoholism sourcebook: Basic consumer health information about alcohol use, abuse, and addiction, including facts about the physical consequences of alcohol abuse, such as brain changes and problems with cognitive functioning, cirrhosis and other liver diseases, cardiovascular disease, pancreatitis, and alcoholic neuropathy, and the effects of alcohol on reproductive health and fetal development, mental health problems associated with alcohol abuse, and alcohol's impact on families, workplaces, and the community ; along with information about underage drinking, alcohol treatment and recovery, a glossary of related terms, and directories of resources for more information. 3rd ed. Omnigraphics, 2010.

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Keshav, Satish, and Alexandra Kent. Alcoholic liver disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0211.

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Alcoholic liver disease develops in excessive drinkers and can manifest in three forms: alcoholic fatty liver (steatosis; >80%), alcoholic hepatitis (10%–35%), and cirrhosis (10%). The more alcohol consumed, the greater the risk of alcoholic liver disease, although other factors may also be involved. Alcohol can cause significant damage without producing any symptoms, and many patients will only have liver dysfunction detected on routine blood tests. Many patients report non-specific symptoms, such as anorexia, morning nausea, diarrhoea, and vague right upper quadrant abdominal pain. The un

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Temas sobre patología hepática. [s.n], 1986.

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Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Fatty liver disease in children. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0055.

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Demographics 406Pathophysiology 406Differential diagnoses 407Presenting features 407Investigation 408Management 409Fatty liver disease is now increasingly recognized in children, particularly in the setting of obesity.The term non-alcoholic steatohepatitis (NASH) was first coined in 1980 by Ludwig to describe a pattern of liver injury in adults in which the liver histology was consistent with alcoholic hepatitis, but in whom significant alcohol consumption was denied. NASH can be considered as part of a broader spectrum of non-alcoholic fatty liver disease that extends from simple steatosis th

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Book chapters on the topic "Alcoholic Liver cirrhosis"

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Leung, Alexander K. C., William Lane M. Robson, Carsten Büning, et al. "Liver Cirrhosis, Alcoholic." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1067.

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Theisler, Charles. "Liver Disease—Alcoholic Hepatitis/Cirrhosis." In Adjuvant Medical Care. CRC Press, 2022. http://dx.doi.org/10.1201/b22898-208.

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Siskind, Eric, and Rolf N. Barth. "Alcoholic Cirrhosis, Transplantation, and Recurrence of Disease." In Disease Recurrence After Liver Transplantation. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-2947-4_7.

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Bowden, David, and Cäcilia S. Reiner. "Diffuse Liver Disease." In IDKD Springer Series. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-27355-1_6.

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AbstractDiffuse liver disease is multifactorial and may arise from metabolic, storage, vascular or neoplastic disease or from drug-related toxicities. Metabolic disease, in particular non-alcoholic fatty liver disease and its sequela of cirrhosis, is an increasing worldwide epidemic with decompensation and the development of malignancy a major concern. This chapter aims to summarize current radiologic techniques in the early detection of chronic/diffuse liver diseases and describes the imaging features of the most common important associated lesions.

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Schwake, Jonathon W., Dawn M. Torres, and Stephen A. Harrison. "The Pointy End of the NAFLD Iceberg: Cirrhosis, Portal Hypertension, and Liver Failure." In Non-Alcoholic Fatty Liver Disease. Wiley-Blackwell, 2013. http://dx.doi.org/10.1002/9781118556153.ch16.

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Miyake, Teruki, and Yoichi Hiasa. "Liver Cirrhosis with Steatohepatitis: Nonalcoholic Steatohepatitis and Alcoholic Steatohepatitis." In The Evolving Landscape of Liver Cirrhosis Management. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-7979-6_1.

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Lefkowitch, Jay H. "Non-alcoholic fatty liver disease (NAFLD) as cause of cryptogenic cirrhosis." In Clinical Dilemmas in Non-Alcoholic Fatty Liver Disease. John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118924938.ch5.

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Mioni, D., M. De Bona, U. Cillo, et al. "Liver Transplantation (OLT) for Alcoholic Cirrhosis with Respect to Recidivism." In Retransplantation. Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-0-585-38142-8_44.

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Jamal, Mohammad H., and Rawan El-Abd. "Sleeve Gastrectomy in Non-alcoholic Steatohepatitis (NASH) and Liver Cirrhosis." In Laparoscopic Sleeve Gastrectomy. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-57373-7_15.

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Vinutha, M. R., J. Chandrika, Balachandran Krishnan, and Sujatha Arun Kokatnoor. "Applying Ensemble Techniques for the Prediction of Alcoholic Liver Cirrhosis." In Data Science and Security. Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-2211-4_39.

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Conference papers on the topic "Alcoholic Liver cirrhosis"

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Kuchinskaya, E. V., and T. S. Apanasenka. "ENZYMATIC ANALYSIS OF BLOOD SERUM OF PATIENTS WITH ALCOHOLIC LIVER INTOXICATION." In SAKHAROV READINGS 2021: ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. International Sakharov Environmental Institute, 2021. http://dx.doi.org/10.46646/sakh-2021-1-278-281.

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Alcohol abuse is a serious medical and social problem. Long-term alcohol intake has a negative effect on all organs, but the liver is most susceptible to its influence, since it is in the liver that ethanol is oxidized. The term alcoholic liver disease is used to refer to the many changes in the liver associated with alcohol. It manifests itself in three main clinical and morphological forms - alcoholic steatosis, accounting for 60-90% of cases of alcoholic liver disease, alcoholic hepatitis, observed in 10-30% of cases, and alcoholic liver cirrhosis in 8-20% of cases. Alcoholic liver disease

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Kraus, Nico, FrankErhard Uschner, Magnus Moeslein, et al. "New Rat Model of Alcoholic Liver Cirrhosis Developing Acute-on-Chronic-Liver failure." In 38. Jahrestagung der Deutsche Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag, 2022. http://dx.doi.org/10.1055/s-0041-1740677.

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Prakash, K., and S. Saradha. "A Deep Learning Approach for Classification and Prediction of Cirrhosis Liver: Non Alcoholic Fatty Liver Disease (NAFLD)." In 2022 6th International Conference on Trends in Electronics and Informatics (ICOEI). IEEE, 2022. http://dx.doi.org/10.1109/icoei53556.2022.9777239.

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Israni, A., C. Shah, and U. Patel. "A Rare Case of Spontaneous Fungal Peritonitis in a Patient with Alcoholic Liver Cirrhosis." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a2934.

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Kraus, Nico, Magnus Moeslein, Robert Schierwagen, et al. "New Rat Model of Advanced Non-Alcoholic-Steatohepatitis-Cirrhosis Developing Acute-on-Chronic-Liver failure." In 38. Jahrestagung der Deutsche Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag, 2022. http://dx.doi.org/10.1055/s-0041-1740678.

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Buttler, Laura, Anja Tiede, Marie Griemsmann, et al. "Impact of thiamine (vitamin B1) supplementation on hepatic encephalopathy and mortality in patients with decompensated alcoholic liver cirrhosis." In 40. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag, 2024. http://dx.doi.org/10.1055/s-0043-1777521.

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Korninger, C., W. Klepetko, J. Miholic, Ch Schwarz, and K. Lechner. "RANDOMIZED TRIAL OF ANTITHROMBIN III VERSUS PLACEBO IN PATIENTS UNDERGOING PERITONEO-VENOUS SHUNT OPERATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644364.

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A randomized trial of Antithrombin III (AT III) versus placebo was performed in patients undergoing peritoneo-venous shunt operation because of intractable ascites. 10 patients with alcoholic liver cirrhosis (Child's stages B and C) were enrolled. Randomization was performed according to the stage of liver disease and to preoperative AT III levels. AT III concentrate (kindly provided by Kabi) was infused in 5 patients, twice daily for 4 days, at a dose of 20 U/kg BW, starting 12 hours prior to operation. Coagulation studies were performed preoperatively, and on postoperative days 1, 3 and 7.In

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Modi, P., B. Tuppekar, D. Ahire, A. Nagpal, G. Nair, and A. Uppe. "Sudden Onset Unilateral Massive Pleural Effusion with Ascites in a Case of Cirrhosis Due to Non-Alcoholic Fatty Liver Disease." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a1939.

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Vizotto, Iara Desiree, Juliana da Cruz Nunes Silva, Maryellen Orbolato Patussi, Luiza Mahiara Calixto Zussa, and Paulo Giovani Padilha dos Santos. "DOENÇA HEPÁTICA ALCOÓLICA." In I Congresso Brasileiro de Saúde Pública On-line: Uma abordagem Multiprofissional. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/2820.

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Introdução: A doença hepática alcoólica (ALD), reporta-se aos danos que acometem o fígado e suas funções, devido ao excesso do consumo de álcool, resultando em várias patologias: esteatose, hepatite alcoólica, esteatohepatite, hepatite crônica com fibrose, cirrose e câncer de fígado. A patogênese por trás da doença hepática alcoólica (ALD) possui mecanismos complexos devido ao comprometimento imunológico, sendo considerada a segunda causa mais comum de morte humana. Consumir bebida alcoólica a longo prazo em excesso, pode causar uma inflamação por meio de acúmulo de algumas substâncias que cau

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Harberts, A., F. Piecha, M. Qi, et al. "Stage-specific transcriptional programming of the liver during progressively decompensating alcohol-associated cirrhosis." In Viszeralmedizin 2023 77. Jahrestagung der DGVS mit Sektion Endoskopie Herbsttagung der Deutschen Gesellschaft für Allgemein- und Viszeralchirurgie mit den Arbeitsgemeinschaften der DGAV und Jahrestagung der CACP. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1771832.

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Reports on the topic "Alcoholic Liver cirrhosis"

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Ismaiel, Abdulrahman, Oana Ciobanu, Mohamed Ismaiel, et al. Atherogenic Index of Plasma in Non-Alcoholic Fatty Liver Disease: Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.8.0043.

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Review question / Objective: P - Non-alcoholic fatty liver disease (NAFLD) I - Atherogenic index of plasma (AIP) C - Imaging and histopathology O - Mean difference and Area Under the Curve S - Observational studies. Condition being studied: Non-alcoholic fatty liver disease (NAFLD), is a common liver disease characterized by the presence of excessive fat build up within hepatocytes, in the absence of other conditions that result in hepatic steatosis and with little to no alcohol consumption. It refers to a broad range of conditions including steatosis, non-alcoholic steatohepatitis (NASH) and

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Jin, Dachuan, Gao Peng, Shunqin Jin, Tao Zhou, Baoqiang Guo, and Guangming Li. Comparison of therapeutic effects of anti-diabetic drugs on non-alcoholic fatty liver disease patients without diabetes: A network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.11.0014.

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Review question / Objective: To evaluate the efficacy of different anti-diabetic drugs in the treatment of non-diabetic non-alcoholic disease by network meta-analysis, and find the best intervention. Condition being studied: Non-alcoholic fatty liver disease (NAFLD) refers to the disease in which the liver fat content exceeds 5%, and excludes the secondary causes of alcohol, infection, drugs or other specific metabolic diseases. As a spectrum of disorders, it includes hepatocyte steatosis and steatohepatitis at the initial stage, liver fibrosis at the later stage, cirrhosis at the final stage,

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Liu, Xiaopei, Dan Liu, and Cong’e Tan. Gut microbiome-based machine learning for diagnostic prediction of liver fibrosis and cirrhosis: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.5.0133.

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Review question / Objective: The invasive liver biopsy is the gold standard for the diagnosis of liver cirrhosis. Other non-invasive diagnostic approaches, have been used as alternatives to liver biopsy, however, these methods cannot identify the pathological grade of the lesion. Recently, studies have shown that gut microbiome-based machine learning can be used as a non-invasive diagnostic approach for liver cirrhosis or fibrosis, while it lacks evidence-based support. Therefore, we performed this systematic review and meta-analysis to evaluate its predictive diagnostic value in liver cirrhos

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