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1

Dickerson, James L. Cirrhosis: An essential guide for the newly diagnosed. Marlow, 2006.

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2

Miron, Jeffrey A. The effect of alcohol prohibition on alcohol consumption. National Bureau of Economic Research, 1999.

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3

Dills, Angela K. Alcohol prohibition and cirrhosis. National Bureau of Economic Research, 2003.

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4

Fe, Caces M., Stinson Frederick S, Elliott Steven D, and National Institute on Alcohol Abuse and Alcoholism (U.S.). Alcohol Epidemiologic Data System, eds. County alcohol problem indicators, 1979-1985. 3rd ed. U.S. Dept. of Health and Human Services, Public Health Service, Alcohol, Drug Abuse, and Mental Health Administration, National Institute on Alcohol Abuse and Alcoholism, 1991.

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5

Yokoe, Ryosuke. Alcohol and Liver Cirrhosis in Twentieth-Century Britain. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-27107-6.

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6

S, Stinson Frederick, Proudfit Ann H, and National Institute on Alcohol Abuse and Alcoholism (U.S.). Alcohol Epidemiologic Data System., eds. U.S. alcohol epidemiologic data reference manual. 4th ed. the Institute, 1994.

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7

Shannon, Joyce Brennfleck. Alcoholism sourcebook: Basic consumer health information about alcohol use, abuse, and addiction, including facts about the physical consequences of alcohol abuse, such as brain changes and problems with cognitive functioning, cirrhosis and other liver diseases, cardiovascular disease, pancreatitis, and alcoholic neuropathy, and the effects of alcohol on reproductive health and fetal development, mental health problems associated with alcohol abuse, and alcohol's impact on families, workplaces, and the community ; along with information about underage drinking, alcohol treatment and recovery, a glossary of related terms, and directories of resources for more information. 3rd ed. Omnigraphics, 2010.

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8

Keshav, Satish, and Alexandra Kent. Alcoholic liver disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0211.

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Alcoholic liver disease develops in excessive drinkers and can manifest in three forms: alcoholic fatty liver (steatosis; >80%), alcoholic hepatitis (10%–35%), and cirrhosis (10%). The more alcohol consumed, the greater the risk of alcoholic liver disease, although other factors may also be involved. Alcohol can cause significant damage without producing any symptoms, and many patients will only have liver dysfunction detected on routine blood tests. Many patients report non-specific symptoms, such as anorexia, morning nausea, diarrhoea, and vague right upper quadrant abdominal pain. The un
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9

Temas sobre patología hepática. [s.n], 1986.

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10

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Fatty liver disease in children. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0055.

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Demographics 406Pathophysiology 406Differential diagnoses 407Presenting features 407Investigation 408Management 409Fatty liver disease is now increasingly recognized in children, particularly in the setting of obesity.The term non-alcoholic steatohepatitis (NASH) was first coined in 1980 by Ludwig to describe a pattern of liver injury in adults in which the liver histology was consistent with alcoholic hepatitis, but in whom significant alcohol consumption was denied. NASH can be considered as part of a broader spectrum of non-alcoholic fatty liver disease that extends from simple steatosis th
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11

Fatty Liver Disease: Education for Patients and the Public. Exon Publications, 2025. https://doi.org/10.36255/fatty-liver-disease.

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Fatty Liver Disease is a condition in which excess fat accumulates in the liver, leading to potential liver damage and health complications. This article provides a comprehensive guide to fatty liver disease, including its causes, symptoms, diagnosis, treatment, and prevention. It explains how the liver functions and why fat buildup can interfere with its ability to process nutrients, break down toxins, and regulate metabolism. The article discusses the two main types of fatty liver disease: non-alcoholic fatty liver disease (NAFLD), which is associated with obesity and metabolic conditions, a
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12

Carton, James. Hepatobiliary pathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0008.

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This chapter discusses hepatobiliary pathology and covers acute viral hepatitis, chronic viral hepatitis, alcoholic liver disease, non-alcoholic fatty liver disease, autoimmune hepatitis (AIH), primary biliary cholangitis (PBC), primary sclerosing cholangitis (PSC), Wilson’s disease, hereditary haemochromatosis, cirrhosis, benign liver lesions, hepatocellular carcinoma (liver cell cancer), intrahepatic cholangiocarcinoma, cholecystitis, and extrahepatic bile duct carcinoma.
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13

Carton, James. Hepatobiliary pathology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199591633.003.0007.

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Acute viral hepatitis 116Chronic viral hepatitis 117Alcoholic liver disease 118Non-alcoholic fatty liver disease 119Autoimmune hepatitis 120Primary biliary cirrhosis 121Primary sclerosing cholangitis 122Wilson's disease 123Hereditary haemochromatosis 124Cirrhosis 125Benign liver lesions 126Hepatocellular carcinoma 127Intrahepatic cholangiocarcinoma ...
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14

Gapstur, Susan M., and Philip John Brooks. Alcohol and Cancer Risk. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0012.

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In 2010, alcoholic beverage consumption caused an estimated 3.3 million deaths worldwide, and contributed to injuries, violence, liver cirrhosis, social disruption and at least seven different types of cancer. The International Agency for Research on Cancer (IARC) classifies exposure to both ethanol in alcoholic beverages and acetaldehyde, the primary metabolite of ethanol, as carcinogenic to humans (Group 1) based on “sufficient” evidence that alcoholic beverage consumption is causally related to cancers of the oral cavity, pharynx, larynx, esophagus, liver, colorectum and female breast. The
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15

Liver Cirrhosis: Education for Patients and the Public. Exon Publications, 2025. https://doi.org/10.36255/liver-cirrhosis.

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Liver cirrhosis is a serious condition where healthy liver tissue is replaced by scar tissue, leading to a decline in liver function over time. This article provides a complete guide for patients, families, and the general public to understand what liver cirrhosis is, how it develops, and what can be done to manage it. It begins with an overview of the condition and explains the different types, including those caused by alcohol, viral infections, and metabolic diseases. The article outlines major risk factors like chronic hepatitis, alcohol abuse, obesity, and genetic disorders, helping reade
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16

Hyman, Mark. Reverse Liver Cirrhosis : Liver Cirrhosis: Getting Rid of Cirrhosis, Fatty Liver, Chronic Alcoholism, Hepatitis from Your Liver. Independently Published, 2019.

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17

Yokoe, Ryosuke. Alcohol and Liver Cirrhosis in Twentieth-Century Britain. Springer International Publishing AG, 2024.

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18

Hayes, Kevin. Alcoholism SB, 6th Ed: Basic Consumer Health Information about Alcohol Use, Abuse, and Addiction, Including Facts about the Physical Consequences of Alcohol Abuse, Such As Brain Changes and Problems with Cognitive Functioning, Cirrhosis and Other Liver Diseases, Cardiovascular Disease, Pancreatitis, and Alcoholic Neuropathy, and the Effects of Alcohol on Reproductive Health and Fetal Development, Mental Health Problems. Omnigraphics, Incorporated, 2021.

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19

Cárdenas, Andrés, and Pere Ginès. The patient with hepatorenal syndrome. Edited by Giuseppe Remuzzi. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0169_update_001.

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Hepatorenal syndrome (HRS) is a dreaded and common complication of patients with end-stage liver disease. The syndrome is characterized by functional renal failure due to renal vasoconstriction in the absence of underlying kidney pathology. The pathogenesis of HRS is the result of an extreme underfilling of the arterial circulation secondary to an arterial vasodilation located in the splanchnic circulation. This phenomenon triggers a compensatory response with activation of vasoconstrictor systems leading to intense renal vasoconstriction.Besides HRS, there are several other causes of renal fa
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20

Chronic Liver Damage (International congress series). Elsevier, 1990.

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21

Yokoe, Ryosuke. Liver Cirrhosis and the Politics of Alcohol in Twentieth-Century Britain: Drinking in the Science. Springer International Publishing AG, 2023.

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22

Kan, Xuegui. Relationship between alcohol consumption and hepatitis B virus (HBV) infection as risk factors in liver cirrhosis. 1985.

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23

Hunter, Duncan James Webb. An epidemiological study of the relationship between mortality and hospital morbidity from cirrhosis of the liver, and per capita consumption of absolute alcohol: Ontario 1978 to 1982. 1987.

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