Relevant bibliographies by topics / Amifostin

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Amifostin'

Author: Grafiati
Published: 4 June 2021
Last updated: 9 February 2022

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Journal articles on the topic "Amifostin"

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Zafirov, Dimce, Jasmina Trojacanec, Maja Slaninka-Miceska, et al. "Effects of amifostin in prevention of cisplatin induced nephrotoxicity in rats." Macedonian Pharmaceutical Bulletin 52 (April 2006): 25–32. http://dx.doi.org/10.33320/maced.pharm.bull.2006.52.004.

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Amifostine is a relatively new organic cytoprotective drug that is actually a prodrug aimed at reduction of cumulative renal toxicity induced by cisplatin repeated administration. The mechanism of amifostin nephroprotective effects has still not been clarified, but it is considered that this medicine has direct cytoprotective effect on kidney tubuli. The main aim of this study was to reveal the effect of amifostin in prevention of nephrotoxicity induced experimentally with long-term administration of cysplatin at dose of 2 mg/kg/b.w./per week during a period of 8 weeks. The results obtained ha
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Kath, R., H. D. Peters, and K. Höffken. "Amifostin." Der Onkologe 3, no. 2 (1997): 191–98. http://dx.doi.org/10.1007/s007610050113.

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Stojanović, Gordana S., Miroslava Stanković, Igor Ž. Stojanović, Ivan Palić, Vesna Milovanović, and Soflja Rančić. "Clastogenic Effect of Atranorin, Evernic acid, and Usnic Acid on Human Lymphocytes." Natural Product Communications 9, no. 4 (2014): 1934578X1400900. http://dx.doi.org/10.1177/1934578x1400900419.

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Three lichen secondary metabolites atranorin (1), evernic acid (2), and usnic acid (3), were evaluated for their in vitro clastogenic and antiproliferative effects on human lymphocytes using the cytochalasin-B blocked micronucleus (CBMN) assay at concentrations of 2 μg/mL, 4 μg/mL and 6 μg/mL of final culture solution. The frequency of micronucleus (MN) was scored in binucleated cells, and cytokinesis-block proliferation index (CBPI) was calculated. Among the tested compounds, 3 exhibited the most prominent effect decreasing the frequency of MN in the range of 42.5% - 48.9%, that is about doub
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Sagowski, C., S. Wenzel, L. Jenicke, et al. "Reduktion der Spättoxizität durch Amifostin bei fraktionierter Bestrahlung der Rattenspeicheldrüsen." HNO 50, no. 9 (2002): 822–28. http://dx.doi.org/10.1007/s00106-001-0595-3.

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Sagowski, C., S. Wenzel, B. Neumann, et al. "Radioprotektive Wirksamkeit von Amifostin auf die Speicheldrüsen der Ratte unter fraktionierter Bestrahlung." HNO 50, no. 2 (2002): 139–45. http://dx.doi.org/10.1007/s001060100536.

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Koukourakis, M. I., C. Frangiadaki, A. Stavroulaki, and G. Ratalis. "Subcutaneous amifostin during fractionated radiotherapy: a randomized phase II study in pelvic tumours." European Journal of Cancer 35 (September 1999): S98. http://dx.doi.org/10.1016/s0959-8049(99)80767-9.

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Czejka, Martin, Johannes Schüller, and Heidemarie Kletzl. "Zytoprotektion mit Amifostin (Ethyol®) in der Chemotherapie: Meta-Analyse zum pharmakokinetischen Interaktionspotential mit Zytostatika." Scientia Pharmaceutica 69, no. 4 (2001): 289–97. http://dx.doi.org/10.3797/scipharm.aut-01-200.

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The cytoprotective agent amifostine (AMI) is capable to protect healthy cells (contrary to tumor cells) due to higher activity of alkaline phosphatase at the membrane site of normal cells. In seven clinical trials the influence of AMI on the pharmacokinetics of different cytostatics was investigated. Preadministration of AMI increased Cmax of doxorubicin (+ 44 %, p < 0.06), epirubicin (+ 31 %, P < 0.08), mitomycin C (+ 41 %, p < 0.01) and docetaxel (+ 31 % and + 17 %, not significant). In contrary, the peak concentration of pirarubicin , the tetrahydropyranyl-prodrug of doxorubicin wa
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Onalan, G., M. Baris, B. Nilufer, and H. B. Zeyneloglu. "Effect of amifostin-cytoprotective agent on the induction of endometriosis in an experimental rat model." Fertility and Sterility 90 (September 2008): S439—S440. http://dx.doi.org/10.1016/j.fertnstert.2008.07.1140.

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Brizel, David M., Todd H. Wasserman, Michael Henke, et al. "Phase III Randomized Trial of Amifostine as a Radioprotector in Head and Neck Cancer." Journal of Clinical Oncology 18, no. 19 (2000): 3339–45. http://dx.doi.org/10.1200/jco.2000.18.19.3339.

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PURPOSE: Radiotherapy for head and neck cancer causes acute and chronic xerostomia and acute mucositis. Amifositine and its active metabolite, WR-1065, accumulate with high concentrations in the salivary glands. This randomized trial evaluated whether amifostine could ameliorate these side effects without compromising the effectiveness of radiotherapy in these patients. PATIENTS AND METHODS: Patients with previously untreated head and neck squamous cell carcinoma were eligible. Primary end points included the incidence of grade ≥ 2 acute xerostomia, grade ≥ 3 acute mucositis, and grade ≥ 2 lat
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Haritwal, Teena, Noopur Gupta, Mrinalini Tiwari, Sarvesh Surve, and Paban Kumar Agrawala. "Radiation Countermeasures: Current Status." Defence Life Science Journal 2, no. 3 (2017): 278. http://dx.doi.org/10.14429/dlsj.2.11675.

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Deleterious effects of ionising radiation leading to significant morbidity and mortality have been studied elaborately. A range of synthetic, semisynthetic and herbal compounds have been screened as radiation countermeasure agents and a number of promising radiation countermeasure agents are under development. Amifostin is the only drug which has been approved by the United State Food and Drug Administration (US-FDA), but that too for use in a defined population under strict medical supervision. Granulocyte Colony Stimulating Factor/filgrastim, γ-tocotrienol, genistein are at an advanced stage
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Dissertations / Theses on the topic "Amifostin"

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Rick, Oliver. "Therapieoptimierungsverfahren bei Patienten mit rezidivierten oder progredienten Keimzelltumoren." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2004. http://dx.doi.org/10.18452/13921.

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Patienten mit metastasierten Keimzelltumoren, die einen Progress oder ein Rezidiv ihrer Erkrankung nach einer cisplatinhaltigen Vortherapie erleiden, haben eine schlechte Prognose. Unter Verwendung einer erneuten konventionellen Chemotherapie können maximal 15-30% dieser Patienten geheilt werden, so dass die Mehrzahl der Patienten an ihrer Erkrankung verstirbt. Aus diesem Grund ist die Optimierung der therapeutischen Möglichkeiten ein wesentliches Ziel. Unsere Daten zeigen, dass die Hochdosischemotherapie (HDCT) eine wesentliche therapeutische Verbesserung darstellt und mittels dieser Therapie
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Lino, Juliana Arcanjo. "Efeito protetor da amifostina na neuropatia sensitiva perifÃrica experimental induzida por oxaliplatina." Universidade Federal do CearÃ, 2011. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=5978.

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Conselho Nacional de Desenvolvimento CientÃfico e TecnolÃgico<br>Oxaliplatina (OXL) Ã um agente platÃnico de 3Â geraÃÃo com potente atividade citotÃxica em diversos cÃnceres. Tem como toxicidade limitante uma neuropatia sensitiva perifÃrica (NSP) de inÃcio agudo, tornando-se crÃnica com doses cumulativas. Amifostina (AMF) Ã um agente antioxidante de largo espectro, que vem sendo atualmente estudado na citoproteÃÃo dos efeitos adversos da radioterapia e quimioterapia do cÃncer. Esta pesquisa objetivou avaliar o efeito da AMF sobre a hiperalgesia mecÃnica plantar e alodinia tÃrmica, assim como s
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Mota, Maria Lurdemiler Sabóia. "Avaliação dos efeitos do agente citoprotetor amifostina na mucosite oral e disfunção da barreira intestinal : modelos experimentais em ratos e em pacientes portadores de câncer submetidos à quimioterapia antineoplásica." reponame:Repositório Institucional da UFC, 2004. http://www.repositorio.ufc.br/handle/riufc/3723.

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MOTA, Maria Lurdemiler Saboia. Avaliação dos efeitos do agente citoprotetor amifostina na mucosite oral e disfunção da barreira intestinal : modelos experimentais em ratos e em pacientes portadores de câncer submetidos à quimioterapia antineoplástica. 2004. 202 f. Tese (Doutorado em Farmacologia) - Universidade Federal do Ceará, Fortaleza, 2004.<br>Submitted by denise santos (denise.santos@ufc.br) on 2012-09-04T15:50:56Z No. of bitstreams: 1 2004_tese_mlsmota.pdf: 2532364 bytes, checksum: 499205137a454ed9e3d572b6a6ed89c0 (MD5)<br>Approved for entry into archive by Eliene Nascimento(elienegvn@
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Sarkar, Anish Ali. "Inflammation Of The Taste Sensory System: Cyclophosphamide And Amifostine." ScholarWorks @ UVM, 2019. https://scholarworks.uvm.edu/graddis/1168.

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Chemotherapeutics are used extensively to treat cancer patients and often induce adverse effects, including taste dysfunctions. Disturbances in taste are detrimental to the overall well-being of cancer patients, causing malnutrition and weight loss that aggravate their condition even further. Inflammation due to an infection of the taste sensory system as previously shown, has detrimental effects on the taste sensation. Our study focused on if chemotherapy induced an inflammatory response in the taste buds using cyclophosphamide (CYP), a pro-drug. Once metabolized by the P450 enzyme complex, i
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Barbosa, Sabrina Carneiro Melo. "Efeito protetor da amifostina sobre a mucosite oral, via inibição de tnf-α, il-1β e nosi, e sobre a hipossalivação induzidas por 5-fluorouracil em hamsters". reponame:Repositório Institucional da UFC, 2011. http://www.repositorio.ufc.br/handle/riufc/4707.

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BARBOSA, Sabrina Carneiro Melo. Efeito protetor da amifostina sobre a mucosite oral, via inibição de TNF-a, IL-1b e NOSi, e sobre a hipossalivação induzidas por 5-fluorouracil em hamsters . 2011. 108 f. Dissertação (Mestrado em Farmacologia) - Universidade Federal do Ceará. Faculdade de Medicina, Fortaleza, 2011.<br>Submitted by denise santos (denise.santos@ufc.br) on 2013-03-15T12:09:19Z No. of bitstreams: 1 2011_dis_scmbarbosa.pdf: 3957690 bytes, checksum: 719ede6aebb7ad2507e0bdb6d6a678f3 (MD5)<br>Approved for entry into archive by Erika Fernandes(erikaleitefernandes@gmail.com) on 2013-03-15
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Mourão, Lívia Talita Cajaseiras. "Envolvimento de óxido nítrico e de canais de potássio dependentes de ATP no efeito protetor da amifostina sobre as alterações motoras funcionais e inflamatórias da cistite hemorrágica induzida por Ifosfamida." reponame:Repositório Institucional da UFC, 2012. http://www.repositorio.ufc.br/handle/riufc/8355.

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MOURÃO, Lívia Talita Cajaseiras. Envolvimento de óxido nítrico e de canais de potássio dependentes de ATP no efeito protetor da amifostina sobre as alterações motoras funcionais e inflamatórias da cistite hemorrágica induzida por ifosfamida. 2012. 116 f. Dissertação (Mestrado em Farmacologia) - Universidade Federal do Ceará. Faculdade de Medicina, Fortaleza, 2012.<br>Submitted by denise santos (denise.santos@ufc.br) on 2014-06-30T12:45:47Z No. of bitstreams: 1 2012_dis_ltcmourão.pdf: 3656317 bytes, checksum: 7e5faa538de3100bf46c80d23bd2feb4 (MD5)<br>Approved for entry into archive by denise s
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Stempak, Diana Christine. "The developmental pharmacology of amifostine, pharmacokinetics and measures of oxidative stress." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ58859.pdf.

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JÃnior, JerÃnimo Junqueira. "Efeito gastroprotetor da amifostina (ETHYOLÂ) na lesÃo gÃstrica induzida por etanol em ratos: papel dos grupos sulfidrÃlicos nÃo-protÃicos e neurÃnios sensoriais aferentes." Universidade Federal do CearÃ, 2008. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=1306.

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CoordenaÃÃo de AperfeiÃoamento de Pessoal de NÃvel Superior<br>INTRODUÃÃO: A amifostina (WR-2721) tem sido largamente estudada como agente citoprotetor em diferentes ÃrgÃos e contra os mais diversos agressores do organismo humano. Recentemente, um efeito gastroprotetor deste fÃrmaco foi observado em modelo de lesÃo gÃstrica induzida por indometacina (MOTA et al., 2007). OBJETIVOS: Este trabalho investigou o efeito da amifostina na lesÃo gÃstrica por etanol e o papel dos neurÃnios sensoriais aferentes, grupos sulfidrÃlicos nÃo-protÃicos, Ãxido nÃtrico, canais de potÃssio sensÃveis ao ATP e cicl
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Cakmak, Gulgun. "The Effects Of Radioprotectant Amifostine On Irradiated Rat Brain And Liver Tissues." Phd thesis, METU, 2010. http://etd.lib.metu.edu.tr/upload/12612591/index.pdf.

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Amifostine is the only approved radioprotective agent by the Food and Drug Administration for reducing the damaging effects of radiation on healthy tissues. In this study, the effects of ionizing radiation on rat liver microsomal membrane and brain tissue and the protecting effects of amifostine on these systems were investigated at molecular level. Sprague-Dawley rats, which were administered amifostine or not, were whole-body irradiated and liver microsomal membranes and different regions of the brain of these rats were analyzed using FTIR spectroscopy, FTIR microspectroscopy and synchrotron
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Freire, Rosemayre Souza. "AvaliaÃÃo do feito citoprotetor da amifostina na cardiotoxicidade aguda induzida por doxorubicina." Universidade Federal do CearÃ, 2008. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=9289.

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nÃo hÃ<br>Conselho Nacional de Desenvolvimento CientÃfico e TecnolÃgico<br>IntroduÃÃo: A Doxorubicina (DOX), antineoplÃsico antracÃclico, à largamente utilizada no tratamento dos mais diversos tipos de tumores sÃlidos e neoplasias hematolÃgicas. A Cardiotoxicidade provocada pelo uso de antibiÃticos antracÃclicos tem sido observada hà algumas dÃcadas como fator agravante e limitante do uso terapÃutico da DOX. Apesar do conhecimento de inÃmeros fatores que podem mediar a induÃÃo da cardiotoxicidade pela DOX, os mecanismos fisiopatolÃgicos continuam nÃo esclarecidos. Objetivo: Avaliar o efeito da
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Books on the topic "Amifostin"

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Grdina, David J. Molecular mechanisms in cytoprotection and chemoprevention with ETHYOL (amifostine). Pro-Ed Communications, 1997.

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Fouladi, Maryam. Phase I trial of a twice-daily-dose regimen of amifostine with ICE chemotherapy in children with refractory cancer. National Library of Canada, 2002.

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The developmental pharmacology of amifostine: Pharmacokinetics and measures of oxidative stress. National Library of Canada, 2001.

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Book chapters on the topic "Amifostin"

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Strnad, V., and R. Sauer. "Radioprotection of Head and Neck Tissue by Amifostine." In Frontiers of Radiation Therapy and Oncology. KARGER, 2001. http://dx.doi.org/10.1159/000061305.

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Murley, Jeffrey S., Yasushi Kataoka, and David J. Grdina. "Amifostine and the Endogenous Cellular Antioxidant Enzyme Manganese Superoxide Dismutase in Radioprotection." In Oxidative Stress in Cancer Biology and Therapy. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-397-4_8.

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Lv, Wenlong, Mei Zhang, Zhenhuan Zhang, et al. "Amifostine Acts Upon Mitochondria to Stimulate Growth of Bone Marrow and Regulate Cytokines." In Oxygen Transport to Tissue XXXV. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7411-1_27.

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"Amifostin (C5H15N2O3P S)." In Encyclopedia of Genetics, Genomics, Proteomics and Informatics. Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6754-9_629.

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Tew, Kenneth D. "Amifostine." In xPharm: The Comprehensive Pharmacology Reference. Elsevier, 2007. http://dx.doi.org/10.1016/b978-008055232-3.61209-7.

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"Amifostine." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_221.

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"AMIFOSTINE." In Litt's Drug Eruption Reference Manual Including Drug Interactions. CRC Press, 2004. http://dx.doi.org/10.3109/9780203492079-11.

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"Amifostine." In ASHP® Injectable Drug Information™. ASHP, 2021. http://dx.doi.org/10.37573/9781585286850.013.

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E., Vassilis, and John R. "The Cytoprotective Effect of Amifostine Against Radiation Induced Toxicity." In Modern Practices in Radiation Therapy. InTech, 2012. http://dx.doi.org/10.5772/35053.

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Oster, W., B. Scheffler, P. Schein, and R. Capizzi. "Chemoprotective Effects of Amifostine on Hematopoietic and Nonhematopoietic Tissue." In Reduction of Anticancer Drug Toxicity. S. Karger AG, 1995. http://dx.doi.org/10.1159/000424040.

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Conference papers on the topic "Amifostin"

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Takahashi, I., H. Niibe, N. Mitsuhashi, and T. Nagai. "Clinical usefulness of amifostine (YM-08310, WR-2721) as a radioprotective agent." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1988. http://dx.doi.org/10.1109/iembs.1988.95369.

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Paunesku, Tatjana, Jelena Popovic, Andrijana Klajn, et al. "Abstract 456: WR1065, the active metabolite of amifostine modulates chemistry and biology of cisplatin." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-456.

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Fu, Panfeng, Anna Birukova, Jeffrey S. Murley, David J. Grdina, and Konstantin G. Birukov. "Involvement Of Delayed Cytoprotection In The Inhibitory Effect Of Amifostine On Mouse Model Of Ventilator-Induced Lung Injury." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2659.

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Farraj, AK, DW Winsett, N. Haykal-Coates, et al. "Amifostine Pre-Treatment Attenuates Lung Injury, but Not Inflammation, in Rats Intracheally Instilled with Particulate Matter Rich in Transition Metals." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3166.

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Reports on the topic "Amifostin"

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Weil, Michael M. Prevention of Radiation-Induced Breast Cancer by Amifostine. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada418558.

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Weil, Michael M. Prevention of Radiation-Induced Breast Cancer by Amifostine. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada479321.

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