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Journal articles on the topic 'Cell death pathways'

Author: Grafiati

Published: 4 June 2021

Last updated: 9 February 2022

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1

Fernández-Lázaro, Diego, César Ignacio Fernández-Lázaro, and Martínez Alfredo Córdova. "Cell Death: Mechanisms and Pathways in Cancer Cells." Cancer Medicine Journal 1, no. 1 (2018): 12–23. http://dx.doi.org/10.46619/cmj.2018.1-1003.

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Programmed cell death is an essential physiological and biological process for the proper development and functioning of the organism. Apoptosis is the term that describes the most frequent form of programmed cell death and derives from the morphological characteristics of this type of death caused by cellular suicide. Apoptosis is highly regulated to maintain homeostasis in the body, since its imbalances by increasing and decreasing lead to different types of diseases. In this review, we aim to describe the mechanisms of cell death and the pathways through apoptosis is initiated, transmitted,

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2

Stekovic, Slaven, and Frank Madeo. "Cell death pathways." Biochimica et Biophysica Acta (BBA) - Molecular Cell Research 1833, no. 12 (2013): 3447. http://dx.doi.org/10.1016/j.bbamcr.2013.09.016.

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3

Fulda, Simone. "Alternative Cell Death Pathways and Cell Metabolism." International Journal of Cell Biology 2013 (2013): 1–4. http://dx.doi.org/10.1155/2013/463637.

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While necroptosis has for long been viewed as an accidental mode of cell death triggered by physical or chemical damage, it has become clear over the last years that necroptosis can also represent a programmed form of cell death in mammalian cells. Key discoveries in the field of cell death research, including the identification of critical components of the necroptotic machinery, led to a revised concept of cell death signaling programs. Several regulatory check and balances are in place in order to ensure that necroptosis is tightly controlled according to environmental cues and cellular nee

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4

Choi, Soo Youn, Whaseon Lee-Kwon, Hwan Hee Lee, Jun Ho Lee, Satoru Sanada, and Hyug Moo Kwon. "Multiple cell death pathways are independently activated by lethal hypertonicity in renal epithelial cells." American Journal of Physiology-Cell Physiology 305, no. 10 (2013): C1011—C1020. http://dx.doi.org/10.1152/ajpcell.00384.2012.

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When hypertonicity is imposed with sufficient intensity and acuteness, cells die. Here we investigated the cellular pathways involved in death using a cell line derived from renal epithelium. We found that hypertonicity rapidly induced activation of an intrinsic cell death pathway—release of cytochrome c and activation of caspase-3 and caspase-9—and an extrinsic pathway—activation of caspase-8. Likewise, a lysosomal pathway of cell death characterized by partial lysosomal rupture and release of cathepsin B from lysosomes to the cytosol was also activated. Relationships among the pathways were

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5

Shymanskyy, I. O., O. O. Lisakovska, A. O. Mazanova, D. O. Labudzynskyi, A. V. Khomenko, and M. M. Veliky. "Prednisolone and vitamin D(3) modulate oxidative metabolism and cell death pathways in blood and bone marrow mononuclear cells." Ukrainian Biochemical Journal 88, no. 5 (2016): 38–47. http://dx.doi.org/10.15407/ubj88.05.038.

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6

Wong, Brian, and Yongwon Choi. "Pathways leading to cell death in T cells." Current Opinion in Immunology 9, no. 3 (1997): 358–64. http://dx.doi.org/10.1016/s0952-7915(97)80082-9.

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7

Mansilla, Sylvia, Laia Llovera, and Jose Portugal. "Chemotherapeutic Targeting of Cell Death Pathways." Anti-Cancer Agents in Medicinal Chemistry 12, no. 3 (2012): 226–38. http://dx.doi.org/10.2174/187152012800228805.

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8

Jin, Zhaoyu, and Wafik S. El-Deiry. "Overview of cell death signaling pathways." Cancer Biology & Therapy 4, no. 2 (2005): 147–71. http://dx.doi.org/10.4161/cbt.4.2.1508.

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9

Horowitz, Stuart. "Pathways to Cell Death in Hyperoxia." Chest 116 (July 1999): 64S—67S. http://dx.doi.org/10.1378/chest.116.suppl_1.64s.

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10

MacFarlane, M. "Cell death pathways – potential therapeutic targets." Xenobiotica 39, no. 8 (2009): 616–24. http://dx.doi.org/10.1080/00498250903137990.

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11

Golstein, Pierre, and Guido Kroemer. "A multiplicity of cell death pathways." EMBO reports 8, no. 9 (2007): 829–33. http://dx.doi.org/10.1038/sj.embor.7401042.

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12

Piacentini, M., and G. Kroemer. "Cell death pathways in retroviral infection." Cell Death & Differentiation 12, S1 (2005): 835–36. http://dx.doi.org/10.1038/sj.cdd.4401655.

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13

A Ali Azzwali, Abdu-Alhameed, and Azab Elsayed Azab. "Mechanisms of programmed cell death." Journal of Applied Biotechnology & Bioengineering 6, no. 4 (2019): 156–58. http://dx.doi.org/10.15406/jabb.2019.06.00188.

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The present review aims to spotlight on the mechanisms and stages of programmed cell death. Apoptosis, known as programmed cell death, is a homeostatic mechanism that generally occurs during development and aging in order to keep cells in tissue. It can also act as a protective mechanism, for example, in immune response or if cells are damaged by toxin agents or diseases. In cancer treatment, drugs and irradiation used in chemotherapy leads to DNA damage, which results in triggering apoptosis through the p53 dependent pathway in cancer treatment, drugs and irradiation used in chemotherapy lead

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14

Kerr, Shannic-Le, Cynthia Mathew, and Reena Ghildyal. "Rhinovirus and Cell Death." Viruses 13, no. 4 (2021): 629. http://dx.doi.org/10.3390/v13040629.

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Rhinoviruses (RVs) are the etiological agents of upper respiratory tract infections, particularly the common cold. Infections in the lower respiratory tract is shown to cause severe disease and exacerbations in asthma and COPD patients. Viruses being obligate parasites, hijack host cell pathways such as programmed cell death to suppress host antiviral responses and prolong viral replication and propagation. RVs are non-enveloped positive sense RNA viruses with a lifecycle fully contained within the cytoplasm. Despite decades of study, the details of how RVs exit the infected cell are still unc

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15

Bermpohl, Daniela, Annett Halle, Dorette Freyer, et al. "Bacterial programmed cell death of cerebral endothelial cells involves dual death pathways." Journal of Clinical Investigation 115, no. 6 (2005): 1607–15. http://dx.doi.org/10.1172/jci23223.

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16

de Vries, J. F., P. A. von dem Borne, M. H. M. Heemskerk, R. Willemze, J. H. F. Falkenburg, and R. M. Y. Barge. "Retroviral Interference with Execution Pathways in Target Cells To Unravel Mechanisms of Cytotoxic T Cell-Mediated Cell Death." Blood 104, no. 11 (2004): 1267. http://dx.doi.org/10.1182/blood.v104.11.1267.1267.

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Abstract Cytotoxic T lymphocytes (CTLs) mediate target cell death by different effector mechanisms. We investigated whether a correlation exists between the kinetics of CTL-induced killing of the target cell and the different apoptotic pathways executed by the CTL. Different CTL clones were isolated using from a patient with CML after receiving donor lymphocyte infusions from an HLA-identical donor. These CTL clones recognized minor antigens expressed on the EBV-LCL cells from the patient. Since these clones were not all equally effective in killing the same target cells, we hypothesized that

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17

Wahyunitisari, ManikRetno, NiMade Mertaniasih, Muhammad Amin, WayanT Artama, and EkoB Koendhori. "Vitamin D, cell death pathways, and tuberculosis." International Journal of Mycobacteriology 6, no. 4 (2017): 349. http://dx.doi.org/10.4103/ijmy.ijmy_120_17.

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18

Scott, Iain, and David C. Logan. "Mitochondria and cell death pathways in plants." Plant Signaling & Behavior 3, no. 7 (2008): 475–77. http://dx.doi.org/10.4161/psb.3.7.5678.

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19

Philpott, Karen, and Laura Facci. "MAP Kinase Pathways in Neuronal Cell Death." CNS & Neurological Disorders - Drug Targets 7, no. 1 (2008): 83–97. http://dx.doi.org/10.2174/187152708783885129.

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20

Ricci, M. Stacey, and Wei‐Xing Zong. "Chemotherapeutic Approaches for Targeting Cell Death Pathways." Oncologist 11, no. 4 (2006): 342–57. http://dx.doi.org/10.1634/theoncologist.11-4-342.

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21

Duprez, Linde, Ellen Wirawan, Tom Vanden Berghe, and Peter Vandenabeele. "Major cell death pathways at a glance." Microbes and Infection 11, no. 13 (2009): 1050–62. http://dx.doi.org/10.1016/j.micinf.2009.08.013.

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22

Vandenabeele, P., T. Vanden Berghe, and N. Festjens. "Caspase Inhibitors Promote Alternative Cell Death Pathways." Science's STKE 2006, no. 358 (2006): pe44. http://dx.doi.org/10.1126/stke.3582006pe44.

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23

Ferri, Karine F., and Guido Kroemer. "Organelle-specific initiation of cell death pathways." Nature Cell Biology 3, no. 11 (2001): E255—E263. http://dx.doi.org/10.1038/ncb1101-e255.

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24

Perlson, Eran, Sandra Maday, Meng-meng Fu, Armen J. Moughamian, and Erika L. F. Holzbaur. "Retrograde axonal transport: pathways to cell death?" Trends in Neurosciences 33, no. 7 (2010): 335–44. http://dx.doi.org/10.1016/j.tins.2010.03.006.

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25

Nakayama, Masafumi, Kazumi Ishidoh, Nobuhiko Kayagaki, et al. "Multiple Pathways of TWEAK-Induced Cell Death." Journal of Immunology 168, no. 2 (2002): 734–43. http://dx.doi.org/10.4049/jimmunol.168.2.734.

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26

Salvesen, G. "ID: 152 Proteolytic pathways in cell death." Journal of Thrombosis and Haemostasis 4, s1 (2006): 26. http://dx.doi.org/10.1111/j.1538-7836.2006.00152.x.

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27

Peterson, Jeanne S., B. Paige Bass, Deborah Jue, Antony Rodriguez, John M. Abrams, and Kimberly McCall. "Noncanonical cell death pathways act duringDrosophila oogenesis." genesis 45, no. 6 (2007): 396–404. http://dx.doi.org/10.1002/dvg.20306.

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28

Zhang, Zhengguo, Ming Wang, Florian Eisel, et al. "UropathogenicEscherichia coliEpigenetically Manipulate Host Cell Death Pathways." Journal of Infectious Diseases 213, no. 7 (2015): 1198–207. http://dx.doi.org/10.1093/infdis/jiv569.

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29

Zlotorynski, Eytan. "At the crossroads of cell death pathways." Nature Reviews Molecular Cell Biology 15, no. 4 (2014): 222. http://dx.doi.org/10.1038/nrm3782.

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30

Hardwick, J. Marie, and Wen-Chih Cheng. "Mitochondrial Programmed Cell Death Pathways in Yeast." Developmental Cell 7, no. 5 (2004): 630–32. http://dx.doi.org/10.1016/j.devcel.2004.10.013.

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31

Peixoto, Milena Simões, Marcos Felipe de Oliveira Galvão, and Silvia Regina Batistuzzo de Medeiros. "Cell death pathways of particulate matter toxicity." Chemosphere 188 (December 2017): 32–48. http://dx.doi.org/10.1016/j.chemosphere.2017.08.076.

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32

Persaud-Sawin, D. A., and R.-M. N. Boustany. "Cell death pathways in juvenile Batten disease." Apoptosis 10, no. 5 (2005): 973–85. http://dx.doi.org/10.1007/s10495-005-0733-6.

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33

Depraetere, Valérie, and Pierre Golstein. "Fas and other cell death signaling pathways." Seminars in Immunology 9, no. 2 (1997): 93–107. http://dx.doi.org/10.1006/smim.1997.0062.

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34

Miyata, Tatsunori, and Laura E. Nagy. "Programmed cell death in alcohol-associated liver disease." Clinical and Molecular Hepatology 26, no. 4 (2020): 618–25. http://dx.doi.org/10.3350/cmh.2020.0142.

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Alcohol-associated liver disease (ALD), which ranges from mild disease to alcohol-associated hepatitis and cirrhosis, is the most prevalent type of chronic liver disease and a leading cause of morbidity and mortality worldwide. Accumulating evidence reveals that programmed cell death (PCD) plays a crucial role in progression of ALD involving crosstalk between hepatocytes and immune cells. Multiple pathways of PCD, including apoptosis, necroptosis, autophagy, pyroptosis and ferroptosis, are reported in ALD. Interestingly, PCD pathways are intimately linked and interdependent, making it difficul

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35

Parsons, Melissa J., and Douglas R. Green. "Mitochondria in cell death." Essays in Biochemistry 47 (June 14, 2010): 99–114. http://dx.doi.org/10.1042/bse0470099.

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Apoptosis can be thought of as a signalling cascade that results in the death of the cell. Properly executed apoptosis is critically important for both development and homoeostasis of most animals. Accordingly, defects in apoptosis can contribute to the development of autoimmune disorders, neurological diseases and cancer. Broadly speaking, there are two main pathways by which a cell can engage apoptosis: the extrinsic apoptotic pathway and the intrinsic apoptotic pathway. At the centre of the intrinsic apoptotic signalling pathway lies the mitochondrion, which, in addition to its role as the

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36

Wang, Man, Shuai Jiang, Yinfeng Zhang, Peifeng Li, and Kun Wang. "The Multifaceted Roles of Pyroptotic Cell Death Pathways in Cancer." Cancers 11, no. 9 (2019): 1313. http://dx.doi.org/10.3390/cancers11091313.

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Cancer is a category of diseases involving abnormal cell growth with the potential to invade other parts of the body. Chemotherapy is the most widely used first-line treatment for multiple forms of cancer. Chemotherapeutic agents act via targeting the cellular apoptotic pathway. However, cancer cells usually acquire chemoresistance, leading to poor outcomes in cancer patients. For that reason, it is imperative to discover other cell death pathways for improved cancer intervention. Pyroptosis is a new form of programmed cell death that commonly occurs upon pathogen invasion. Pyroptosis is marke

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37

Henshall, D. C. "Apoptosis signalling pathways in seizure-induced neuronal death and epilepsy." Biochemical Society Transactions 35, no. 2 (2007): 421–23. http://dx.doi.org/10.1042/bst0350421.

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Delineating the molecular pathways underlying seizure-induced neuronal death may yield novel strategies for brain protection against prolonged or repetitive seizures. Glutamate-mediated excitotoxicity and necrosis is a primary contributing mechanism but seizures also activate programmed (apoptotic) cell death pathways. Apoptosis signalling pathways are typically initiated following perturbation of intracellular organelle function (intrinsic pathway) or by activated cell-surface-expressed death receptors (extrinsic pathway), with signalling cascades orchestrated in part by the Bcl-2 and caspase

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38

Chowdhury, Dipanjan, and Judy Lieberman. "Death by a Thousand Cuts: Granzyme Pathways of Programmed Cell Death." Annual Review of Immunology 26, no. 1 (2008): 389–420. http://dx.doi.org/10.1146/annurev.immunol.26.021607.090404.

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39

LEE, E. C. Y., and M. TENNISWOOD. "PROGRAMMED CELL DEATH AND SURVIVAL PATHWAYS IN PROSTATE CANCER CELLS." Archives of Andrology 50, no. 1 (2004): 27–32. http://dx.doi.org/10.1080/01485010490250498.

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40

Hay, Stewart, and George Kannourakis. "A time to kill: viral manipulation of the cell death program." Journal of General Virology 83, no. 7 (2002): 1547–64. http://dx.doi.org/10.1099/0022-1317-83-7-1547.

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Many viruses have as part of their arsenal the ability to modulate the apoptotic pathways of the host. It is counter-intuitive that such simple organisms would be efficient at regulating this the most crucial pathway within the host, given the relative complexity of the host cells. Yet, viruses have the potential to initiate or stay the onset of programmed cell death through the manipulation of a variety of key apoptotic proteins. It is the intention of this review to provide an overview of viral gene products that are able to promote or inhibit apoptotic death of the host cell and to discuss

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41

Nakajima, H., P. Golstein, and P. A. Henkart. "The target cell nucleus is not required for cell-mediated granzyme- or Fas-based cytotoxicity." Journal of Experimental Medicine 181, no. 5 (1995): 1905–9. http://dx.doi.org/10.1084/jem.181.5.1905.

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The requirement for target cell nuclei in the two apoptotic death pathways used by cytotoxic lymphocytes was tested using model effector systems in which the granzyme and Fas pathways of target damage are isolated. Mast cell tumors expressing granzymes A and B in addition to cytolysin/perforin lysed tumor target cells about 10-fold more efficiently than comparable effector cells without granzymes. Enucleated cytoplast targets derived from these cells were also lysed with a similar 10-fold effect of granzymes. In contrast to cytoplasts, effector granzyme expression did not influence lysis of re

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42

Tang, Peter S., Marco Mura, Rashmi Seth, and Mingyao Liu. "Acute lung injury and cell death: how many ways can cells die?" American Journal of Physiology-Lung Cellular and Molecular Physiology 294, no. 4 (2008): L632—L641. http://dx.doi.org/10.1152/ajplung.00262.2007.

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Apoptosis has been considered as an underlying mechanism in acute lung injury/acute respiratory distress syndrome and multiorgan dysfunction syndrome. Recently, several alternative pathways for cell death (such as caspase-independent cell death, oncosis, and autophagy) have been discovered. Evidence of these pathways in the pathogenesis of acute lung injury has also come into light. In this article, we briefly introduce cell death pathways and then focus on studies related to lung injury. The different types of cell death that occur and the underlying mechanisms utilized depend on both experim

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43

Dragovich, Tomislav, Charles M. Rudin, and Craig B. Thompson. "Signal transduction pathways that regulate cell survival and cell death." Oncogene 17, no. 25 (1998): 3207–13. http://dx.doi.org/10.1038/sj.onc.1202587.

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44

Graham, Steven H., and Jun Chen. "Programmed Cell Death in Cerebral Ischemia." Journal of Cerebral Blood Flow & Metabolism 21, no. 2 (2001): 99–109. http://dx.doi.org/10.1097/00004647-200102000-00001.

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Programmed cell death (PCD) is an ordered and tightly controlled set of changes in gene expression and protein activity that results in neuronal cell death during brain development. This article reviews the molecular pathways by which PCD is executed in mammalian cells and the potential relation of these pathways to pathologic neuronal cell death. Whereas the classical patterns of apoptotic morphologic change often do not appear in the brain after ischemia, there is emerging biochemical and pharmacologic evidence suggesting a role for PCD in ischemic brain injury. The most convincing evidence

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45

Williams, Thomas J., Luis E. Gonzales-Huerta, and Darius Armstrong-James. "Fungal-Induced Programmed Cell Death." Journal of Fungi 7, no. 3 (2021): 231. http://dx.doi.org/10.3390/jof7030231.

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Fungal infections are a cause of morbidity in humans, and despite the availability of a range of antifungal treatments, the mortality rate remains unacceptably high. Although our knowledge of the interactions between pathogenic fungi and the host continues to grow, further research is still required to fully understand the mechanism underpinning fungal pathogenicity, which may provide new insights for the treatment of fungal disease. There is great interest regarding how microbes induce programmed cell death and what this means in terms of the immune response and resolution of infection as wel

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46

Hartman, Mariusz L. "Non-Apoptotic Cell Death Signaling Pathways in Melanoma." International Journal of Molecular Sciences 21, no. 8 (2020): 2980. http://dx.doi.org/10.3390/ijms21082980.

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Resisting cell death is a hallmark of cancer. Disturbances in the execution of cell death programs promote carcinogenesis and survival of cancer cells under unfavorable conditions, including exposition to anti-cancer therapies. Specific modalities of regulated cell death (RCD) have been classified based on different criteria, including morphological features, biochemical alterations and immunological consequences. Although melanoma cells are broadly equipped with the anti-apoptotic machinery and recurrent genetic alterations in the components of the RAS/RAF/MEK/ERK signaling markedly contribut

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47

Basmaciyan, Louise, and Magali Casanova. "Cell death in Leishmania." Parasite 26 (2019): 71. http://dx.doi.org/10.1051/parasite/2019071.

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Leishmaniases still represent a global scourge and new therapeutic tools are necessary to replace the current expensive, difficult to administer treatments that induce numerous adverse effects and for which resistance is increasingly worrying. In this context, the particularly original organization of the Leishmania parasite in comparison to higher eukaryotes is a great advantage. It allows for the development of new, very specific, and thus non-cytotoxic treatments. Among these originalities, Leishmania cell death can be cited. Despite a classic pattern of apoptosis, key mammalian apoptotic p

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48

Michel, Patrick P., Etienne C. Hirsch, and Stéphane Hunot. "Understanding Dopaminergic Cell Death Pathways in Parkinson Disease." Neuron 90, no. 4 (2016): 675–91. http://dx.doi.org/10.1016/j.neuron.2016.03.038.

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49

Mroz, Pawel, Anastasia Yaroslavsky, Gitika B. Kharkwal, and Michael R. Hamblin. "Cell Death Pathways in Photodynamic Therapy of Cancer." Cancers 3, no. 2 (2011): 2516–39. http://dx.doi.org/10.3390/cancers3022516.

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50

Neitemeier, Sandra, Anja Jelinek, Vincenzo Laino, et al. "BID links ferroptosis to mitochondrial cell death pathways." Redox Biology 12 (August 2017): 558–70. http://dx.doi.org/10.1016/j.redox.2017.03.007.

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