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Relevant bibliographies by topics / Corn Wilt diseases. Erwinia stewartii

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Journal articles
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Academic literature on the topic 'Corn Wilt diseases. Erwinia stewartii'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Corn Wilt diseases. Erwinia stewartii"

1

Brown, A. F., J. A. Juvik, and J. K. Pataky. "Quantitative Trait Loci in Sweet Corn Associated with Partial Resistance to Stewart's Wilt, Northern Corn Leaf Blight, and Common Rust." Phytopathology® 91, no. 3 (2001): 293–300. http://dx.doi.org/10.1094/phyto.2001.91.3.293.

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Partial resistance to Stewart's wilt (Erwina stewartii, syn. Pantoea stewartii), northern corn leaf blight (NCLB) (Exserohilum turcicum), and common rust (Puccinia sorghi) was observed in an F2:3 population developed from a cross between the inbred sweet corn lines IL731a and W6786. The objective of this study was to identify quantitative trait loci (QTL) associated with partial resistance using restriction fragment length polymorphic markers. Phenotypic data were collected for 2 years for Stewart's wilt, NCLB, and common rust but, due to significant family-environment interaction, analysis wa

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2

Pataky, J. K., L. J. du Toit, P. Revilla, and W. F. Tracy. "Reactions of Open-Pollinated Sweet Corn Cultivars to Stewart's Wilt, Common Rust, Northern Leaf Blight, and Southern Leaf Blight." Plant Disease 82, no. 8 (1998): 939–44. http://dx.doi.org/10.1094/pdis.1998.82.8.939.

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Over 800 open-pollinated (OP) varieties of sweet corn were grown and named in the century prior to the development of hybrids, but only a few of the historically important OP cultivars exist today. Alleles that could improve disease resistance of modern sweet corn may be present in the OP cultivars still in existence. The objectives of this research were to compare 36 OP sweet corn cultivars to modern commercial hybrids for reactions to Stewart's wilt, common rust, northern leaf blight (NLB), and southern leaf blight (SLB), and to classify the OP cultivars based on phenotypic reactions to thes

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3

Michener, P. M., J. K. Pataky, and D. G. White. "Transmission of Erwinia stewartii from Plants to Kernels and Reactions of Corn Hybrids to Stewart's Wilt." Plant Disease 86, no. 2 (2002): 167–72. http://dx.doi.org/10.1094/pdis.2002.86.2.167.

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Stewart's wilt reactions of 98 food-grade, white corn hybrids, 3 yellow dent corn hybrids, and 23 sweet corn hybrids and infection of kernels by E. stewartii were evaluated in 1998, 1999, and 2000. Stewart's wilt symptoms were rated from 1 (no appreciable spread of symptoms) to 9 (dead plants) following inoculation. The mean Stewart's wilt ratings for the food-grade, white corn and yellow dent corn hybrids were 1.9, 2.4, and 2.9 in 1998, 1999, and 2000, respectively. The mean Stewart's wilt ratings for the sweet corn hybrids were 3.8, 4.2, and 4.6 in 1998, 1999, and 2000, respectively. Hybrids

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Michener, P. M., J. K. Pataky, and D. G. White. "Rates of Transmitting Erwinia stewartii from Seed to Seedlings of a Sweet Corn Hybrid Susceptible to Stewart's Wilt." Plant Disease 86, no. 9 (2002): 1031–35. http://dx.doi.org/10.1094/pdis.2002.86.9.1031.

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Rates of transmitting Erwinia stewartii from seed to seedlings were estimated from field grow-outs of seedlings grown from seed infected with E. stewartii. Infected seed were produced in 1998, 1999, and 2000 on a Stewart's wilt-susceptible sweet corn hybrid, Jubilee. Seedlings were inoculated repeatedly with pinprick inoculators and suspensions of E. stewartii were injected into ear shanks of the primary ears of each adult plant. Seed from inoculated plants were harvested and bulked. Single kernels were assayed for E. stewartii to estimate the proportion of kernels infected with E. stewartii.

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Ahmad, Musharaf, Doris R. Majerczak, Sharon Pike, Mary Elizabeth Hoyos, Anton Novacky, and David L. Coplin. "Biological Activity of Harpin Produced by Pantoea stewartii subsp. stewartii." Molecular Plant-Microbe Interactions® 14, no. 10 (2001): 1223–34. http://dx.doi.org/10.1094/mpmi.2001.14.10.1223.

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Pantoea stewartii subsp. stewartii causes Stewart's wilt of sweet corn. A hypersensitive response and pathogenicity (Hrp) secretion system is needed to produce water-soaking and wilting symptoms in corn and to cause a hypersensitive response (HR) in tobacco. Sequencing of the hrp cluster revealed a putative harpin gene, hrpN. The product of this gene was overexpressed in Escherichia coli and shown to elicit the HR in tobacco and systemic resistance in radishes. The protein was designated HrpNPnss. Like other harpins, it was heat stable and protease sensitive, although it was three- to fourfold

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6

Meyer, A. C., J. K. Pataky, and J. A. Juvik. "SWEET CORN INBREDS WITH PARTIAL RESISTANCE TO NORTHERN LEAF BLIGHT AND STEWART'S WILT." HortScience 25, no. 9 (1990): 1067a—1067. http://dx.doi.org/10.21273/hortsci.25.9.1067a.

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Approximately 200 sweet corn inbred lines were screened for two years for resistance to northern leaf blight, caused by Exserohilum turcicum, and Stewart's wilt, caused by Erwinia stewartii. Inbreds with the best levels of partial resistance to races 1 and 2 of E. turcicum included IL11d, IL676a, IL677a, IL685d, IL766a, IL767a and IL797a. Inbreds with the best partial resistance to E. stewartii included IL126b, IL676a, IL767a, IL772a, IL774g, IL797a, IL798a and M6011. Several of these resistant and moderately resistant inbreds had common ancestors; however, inspection of pedigrees suggested th

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7

Coplin, David L., Doris R. Majerczak, Yongxiang Zhang, Won-Sik Kim, Susanne Jock, and Klaus Geider. "Identification of Pantoea stewartii subsp. stewartii by PCR and Strain Differentiation by PFGE." Plant Disease 86, no. 3 (2002): 304–11. http://dx.doi.org/10.1094/pdis.2002.86.3.304.

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Stewart's bacterial wilt and leaf blight of sweet corn and maize is caused by Pantoea stewartii subsp. stewartii. This bacterium can be seed transmitted at a low frequency, so it is subject to quarantine restrictions by many countries. To develop a polymerase chain reaction assay for the identification of this pathogen from field samples and for use in seed health tests, four primer pairs were tested. These were selected from the sequences of hrpS, cpsDE, and the 16S rRNA intergenic transcribed spacer (ITS) region. Under optimal reaction conditions, about 20 and 200 cells of P. stewartii could

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8

Pataky, J. K., P. M. Michener, N. D. Freeman, R. A. Weinzierl, and R. H. Teyker. "Control of Stewart's Wilt in Sweet Corn with Seed Treatment Insecticides." Plant Disease 84, no. 10 (2000): 1104–8. http://dx.doi.org/10.1094/pdis.2000.84.10.1104.

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Corn flea beetles, Chaetocnema pulicaria, vector Erwinia stewartii (synamorph Pantoea stewartii), which causes Stewart's bacterial wilt of corn (Zea mays). A seed treatment insecticide, imidacloprid, killed flea beetles and reduced the number of feeding wounds and Stewart's wilt symptoms per leaf in greenhouse studies. The objective of our research was to evaluate the ability of imidacloprid and thiamethoxam seed treatments to control Stewart's wilt on sweet corn hybrids under field conditions with naturally occurring populations of the corn flea beetle. Six field trials were planted at four l

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Pataky, Jerald K., Lindsey J. du Toit, and Noah D. Freeman. "Stewart's Wilt Reactions of an International Collection of Zea mays Germ Plasm Inoculated with Erwinia stewartii." Plant Disease 84, no. 8 (2000): 901–6. http://dx.doi.org/10.1094/pdis.2000.84.8.901.

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Maize accessions were evaluated in 1997, 1998, and 1999 to identify additional sources of Stewart's wilt resistance and to determine if reactions differed among accessions collected from various regions of the United States and throughout the world. The distributions of Stewart's wilt reactions rated from 1 (no appreciable spread of symptoms) to 9 (dead plants) were relatively similar among groups of accessions from all regions of the world except for those from the Mid-Atlantic/Ohio River Valley region of the United States, the southern United States, and the northeastern United States. The m

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10

Michener, P. M., N. D. Freeman, and J. K. Pataky. "Relationships Between Reactions of Sweet Corn Hybrids to Stewart's Wilt and Incidence of Systemic Infection by Erwinia stewartii." Plant Disease 87, no. 3 (2003): 223–28. http://dx.doi.org/10.1094/pdis.2003.87.3.223.

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Relationships between the reactions of sweet corn hybrids to Stewart's wilt and the incidence of natural, systemic infection by Erwinia stewartii differed among trials in which the prevalence of Stewart's wilt differed. Systemic Stewart's wilt infection was assessed for 262, 296, and 245 hybrids planted in seven trials in central Illinois in June and July 1998, 1999, and 2000, respectively. Incidence of systemic infection was calculated in each trial for all hybrids in each of nine categories of Stewart's wilt reactions (i.e., 1 = resistant and 9 = susceptible). When mean incidence was about 5

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Dissertations / Theses on the topic "Corn Wilt diseases. Erwinia stewartii"

1

Merighi, Massimo. "Molecular biology and biochemistry of regulation of Hrp/type III secretion genes in the corn pathogen Pantoea stewartii pv. stewartii." Connect to this title online, 2004. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1069854564.

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Thesis (Ph. D.)--Ohio State University, 2004.<br>Document formatted into pages; contains xxxiii, 421 p. Includes bibliographical references. Abstract available online via OhioLINK's ETD Center; full text release delayed at author's request until 2005 Dec. 2.

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