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Books on the topic 'Disordered Locomotion'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 February 2022

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1

Measurement of human locomotion. Boca Raton, FL: CRC Press, 2001.

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2

J, Levine Linda, ed. Pelvic locomotor dysfunction: A clinical approach. Gaithersburg, Md: Aspen Publishers, 1996.

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3

Fundamentals of anatomy and movement: A workbook and guide. St. Louis: Mosby, 1997.

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4

Clinical gait analysis: Theory and practice. Edinburgh: Elsevier, 2005.

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5

Lewit, Karel. Manipulative therapy in rehabilitation of the locomotor system. 3rd ed. Oxford: Butterworth-Heinemann, 1999.

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6

Lewit, Karel. Manipulative therapy in rehabilitation of the locomotor system. 2nd ed. Oxford: Butterworth Heinemann, 1991.

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7

Manipulative therapy in rehabilitation of the locomotor system. London: Butterworths, 1985.

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8

Movement: Functional movement systems : screening, assessment, and corrective strategies. Aptos, CA: On Target Publications, 2010.

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9

Ward, Thomas E. Kinetic data extraction and analysis system for human gait. Dublin: University College Dublin, 1996.

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10

American Society for Bone and Mineral Research, ed. Primer on the metabolic bone diseases and disorders of mineral metabolism. 4th ed. Philadelphia: Lippincott Williams & Wilkins, 1999.

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11

Latash, Mark L. Bases neurophysiologiques du mouvement. Paris: De Boeck., 2002.

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12

Neurophysiological basis of movement. 2nd ed. Champaign, IL: Human Kinetics, 2008.

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13

Neurophysiological basis of movement. Champaign, IL: Human Kinetics, 1998.

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14

(Editor), Mark L. Latash, and Mindy F. Levin (Editor), eds. Progress in Motor Control: Effects of Age, Disorder, and Rehabilitation. Human Kinetics Publishers, 2003.

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15

Medved, Vladimir. Measurement of Human Locomotion. CRC, 2000.

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16

Abbott, Richard. Gait disorders. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0046.

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Gait problems can arise from a number of different causes ranging from primary neurological to locomotor conditions. Prevalence increases with age, and causation is often multifactorial. Falls are a frequent consequence. This chapter covers the diagnosis of gait disorders and therapies.

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17

Anne, Betts, ed. The patient with a locomotor disorder. London: Harper & Row, 1985.

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18

Evzen, Ph.D. Ruzicka (Editor), Mark Hallett (Editor), and Joseph Jankovic (Editor), eds. Advances in Neurology, Volume 87: Gait Disorders. Lippincott Williams & Wilkins, 2001.

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19

Sandro, Giannini, ed. Gait analysis: Methodologies and clinical applications. Amsterdam: IOS Press for BTS, Bioengineering Technology & Systems, 1994.

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20

Betts, Anne. Patient with a Locomotor Disorder (Test Your Knowledge M.C.Q.). Longman Higher Education, 1985.

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21

Barbieri, Fabio Augusto, and Rodrigo Vitório. Locomotion and Posture in Older Adults: The Role of Aging and Movement Disorders. Springer, 2017.

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22

Barbieri, Fabio Augusto, and Rodrigo Vitório. Locomotion and Posture in Older Adults: The Role of Aging and Movement Disorders. Springer, 2018.

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23

Kirtley, Christopher. Clinical Gait Analysis: Theory and Practice. Churchill Livingstone, 2006.

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24

Observational Gait Analysis. Los Amigos Research &, 2001.

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25

(Editor), Jeffrey M. Hausdorff, and Neil B. Alexander (Editor), eds. Gait Disorders: Evaluation and Management (Neurological Disease and Therapy). Informa Healthcare, 2005.

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26

Human motion analysis: Current applications and future directions. New York: Institute of Electrical and Electronics Engineers, 1996.

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27

Levin, Mindy F. Progress in Motor Control: Skill Learning, Performance, Health, and Injury. Springer, 2014.

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28

Gotman, Kélina. Choreomania. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190840419.001.0001.

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This book traces the emergence and spread of the choreomania concept through colonial medical and ethnographic circles, showing how fantasies of instability—and of the Oriental other—haunted scientific modernity. Scenes from the archives of medical history, neurology, psychiatry, sociology, religion, and popular journalism show how the discursive history of the ‘dancing mania’ moved and transformed with its translations throughout the colonial world. From antiquarian references to ancient Greek bacchanals and medieval St. Vitus’s dances, to scientific reperformances of early modern religious ecstasies, and American government anthropology, ‘choreomania’ arose to signal every sort of gestural and choreographic unrest. Village kermesses, revolutionary crowds, and neuromotor disorders—including hysteria, epilepsy, and chorea—were among the many unruly forms of locomotion indiscriminately compared to bacchanalian turmoil. So too, charges of spontaneous political agitation levied against demonstrators from Africa and South America to the South Seas reveal heightened anxieties about the spread of social disorder. Initially employed to describe ‘contagious’ popular dances, jerking movements, and convulsions, with decolonization, the ‘dancing disease’ increasingly described the fitful drama of anti-European revolt. Closely indebted to the work of Michel Foucault, this book opens a new chapter on the way we think epidemic madness and the organization and disorganization of bodies and disciplines in the modern age. Setting ideas about disruptively moving bodies at the heart of the scientific enterprise, this book argues that disciplines themselves were at once more porous and mobile than is commonly allowed, and that ‘dance’ itself has to be radically reimagined across fields.

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29

R, Sanberg Paul, Ossenkopp Klaus-Peter, and Kavaliers Martin, eds. Motor activity and movement disorders: Research issues and applications. Totowa, N.J: Humana Press, 1996.

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30

J, Favus Murray, Christakos Sylvia, and American Society for Bone and Mineral Research., eds. Primer on the metabolic bone diseases and disorders of mineral metabolism. 3rd ed. Philadelphia: Lippincott-Raven, 1996.

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31

Sanberg, Paul, Klaus-Peter Ossenkopp, and Martin Kavaliers. Motor Activity & Movement Disorders: Research Issues and Applications (Contemporary Neuroscience). Humana Press, 1995.

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32

Stoddard, Frederick J., and Robert L. Sheridan. Wound Healing and Depression. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603342.003.0009.

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Depression and wound healing are bidirectional processes for adults and children consistent with the conception of depression as systemic. This systemic interaction is similar to the “bidirectional impact of mood disorder on risk for development, progression, treatment, and outcomes of medical illness” generally. And, evidence is growing that the bidirectional impact of mood disorder may be true for injuries and for trauma surgery. Animal models have provided some support that treatment of depression may improve wound healing. An established biological model for a mechanism delaying wound healing is increased cortisol secretion secondary to depression and/or stress, and impaired immune response, in addition or together with the other factors such as genetic or epigenetic risk for depression. Cellular models relate both to wound healing and to depression include cytokines, the inflammatory response (Miller et al, 2008), and cellular aging (Telgenhoff and Shroot, 2005) reflected in shorter leukocyte telomere length (LTL) (Verhoeven et al, 2016). Another model of stress impacting wound healing investigated genetic correlates—immediate early gene expression or IEG from the medial prefrontal cortex, and locomotion, in isolation-reared juvenile rats. Levine et al (2008) compared isolation reared to group reared samples, and found that, immediate gene expression in the medial prefrontal cortex (mPFC) was reduced, and behavioral hyperactivity increased, in juvenile rats with 20% burn injuries. Wound healing in the isolation reared rats was significantly impaired. They concluded that these results provide candidates for behavioral biomarkers of isolation rearing during physical injury, i.e. reduced immediate mPFC gene expression and hyperactivity. They suggested that a biomarker such as IEGs might aid in demarcating patients with resilient and adaptive responses to physical illness from those with maladaptive responses

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33

Haruki, Murakami. Best︠s︡vetnyĭ T︠S︡kuru Tadzaki i gody ego stranstviĭ. 2015.

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34

Latash, Mark L. Neurophysiological Basis of Movement. 2nd ed. Human Kinetics Publishers, 2007.

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35

S, Kataria Mohan, ed. Fits, faints, and falls in old age. Lancaster [Lancashire]: MTP Press, 1985.

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36

1919-, Struppler A., and Weindl A, eds. Electromyography and evoked potentials: Theories and applications. Berlin: Springer-Verlag, 1985.

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37

(Editor), M. Fetter, T. Haslwanter (Editor), H. Misslich (Editor), and D. Tweed (Editor), eds. Three-dimensional Kinematics of the Eye, Head and Limb Movements. CRC, 1997.

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38

Three-dimensional kinematics of eye, head and limb movements. Amsterdam, The Netherlands: Harwood Academic Publishers, 1997.

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39

Struppler, A. Electromyography and Evoked Potentials: Theories and Applications (Advances in Applied Neurological Sciences 1). Springer-Verlag, 1985.

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40

Struppler, A. Electromyography and Evoked Potentials: Theories and Applications. Springer, 2011.

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41

Colorless Tsukuru Tazaki And His Years Of Pilgrimage A Novel. Knopf, 2014.

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42

Colorless Tsukuru Tazaki and his years of pilgrimage. Vintage, 2015.

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43

(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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44

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Amsterdam: Elsevier, 1996.

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