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Journal articles on the topic 'Endomysial antibody'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Chan, K. N., A. D. Phillips, R. Mirakian, and J. A. Walker‐Smith. "Endomysial Antibody Screening in Children." Journal of Pediatric Gastroenterology and Nutrition 18, no. 3 (1994): 316–20. http://dx.doi.org/10.1002/j.1536-4801.1994.tb11181.x.

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It has been suggested that endomysial antibodies are specific markers for coeliac disease. In a 13‐month study, we examined the usefulness of screening for these antibodies in the diagnosis of coeliac disease in children. Twenty‐one of 223 (9.4%) serum samples [or 17 of 192 (9%) children undergoing investigation for GI disorders] were found to be positive for serum IgA class endomysial antibodies. These included eight strong positives, eight positives, and five weak positives. One‐hundred‐thirty‐four children had small bowel biopsies performed. Endomysial antibodies were found in all children

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Chan, K. N., A. D. Phillips, R. Mirakian, and J. A. Walker-Smith. "Endomysial Antibody Screening in Children." Journal of Pediatric Gastroenterology and Nutrition 18, no. 3 (1994): 316–20. http://dx.doi.org/10.1097/00005176-199404000-00011.

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3

Murray, Joseph A., Judith Herlein, Frank Mitros, and James A. Goeken. "Serologic Testing for Celiac Disease in the United States: Results of a Multilaboratory Comparison Study." Clinical Diagnostic Laboratory Immunology 7, no. 4 (2000): 584–87. http://dx.doi.org/10.1128/cdli.7.4.584-587.2000.

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ABSTRACT The aim of this study was to compare the efficiencies of six reference laboratories for serologic testing for celiac disease. Serum from 20 patients with untreated celiac disease and from 20 controls was thawed, divided, and distributed to each participating laboratory, which performed endomysial antibody tests. Five laboratories also performed antigliadin antibody tests. Sensitivity for endomysial antibody immunoglobulin A (IgA) varied from 57 to 90%. In all laboratories, the specificity for celiac disease was 100%. The sensitivity and specificity for both IgA and IgG antigliadin ant

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Karabey, Mehmet, Havva Kaya, Alperen Ceylan, Kadir Kaba, Mehmet Özdemir, and Bahadır Feyzioğlu. "The effect of the pandemic on autoantibody rates in the general population." Archives of Rheumatology 39, no. 4 (2024): 541–48. https://doi.org/10.46497/archrheumatol.2024.10330.

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Objectives: The study aimed to investigate the possible effects of coronavirus disease 2019 (COVID-19) on autoantibodies. Patients and methods: Samples of 89,108 individuals (29,033 males, 60,075 females; median: 36 years; range, 0 to 96 years) who underwent autoimmune testing between January 2017 and May 2022 were retrospectively analyzed. The prepandemic period was defined as May 1, 2017, to March 20, 2020, while the pandemic period was defined as March 20, 2020, to May 31, 2022. Results: Of the participants, 0.55% were of foreign nationality. The positivity rate was 18.12%. Autoantibody pos

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Picarelli, Antonio, Luigi Sabbatella, and Marco Di Tola. "Endomysial antibody production after in vitro gliadin challenge." European Journal of Gastroenterology & Hepatology 13, no. 2 (2001): 213. http://dx.doi.org/10.1097/00042737-200102000-00022.

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6

Ciclitira, P. J., F. Biagi, H. J. Ellis, and N. D. G. Parnell. "Endomysial antibody production after in vitro gliadin challenge." European Journal of Gastroenterology & Hepatology 13, no. 2 (2001): 214. http://dx.doi.org/10.1097/00042737-200102000-00023.

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7

Peleg, Roni, Z. Itzhak Ben-Zion, Aya Peleg, et al. "“Bread madness” revisited: screening for specific celiac antibodies among schizophrenia patients." European Psychiatry 19, no. 5 (2004): 311–14. http://dx.doi.org/10.1016/j.eurpsy.2004.06.003.

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AbstractPurposeA possible association between gluten consumption and schizophrenia has been reported. The objective was to compare patients with chronic schizophrenia and matched controls for sociodemographic variables, prevalence of celiac-specific anti-endomysial antibodies and disease-related variables.Subjects and methodsThe study group was comprised of 50 consecutive patients diagnosed with schizophrenia, 18 years of age and older attending the out-patient clinic of the Mental Health Center in Beer-Sheva, Israel. The control group was comprised of mentally normal volunteers who came to pr

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8

Biagi, F., P. I. Bianchi, J. Campanella, et al. "The prevalence and the causes of minimal intestinal lesions in patients complaining of symptoms suggestive of enteropathy: a follow-up study: Table 1." Journal of Clinical Pathology 61, no. 10 (2008): 1116–18. http://dx.doi.org/10.1136/jcp.2008.060145.

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Aims:Although they are non-specific, minimal intestinal lesions are at the end of the coeliac histological damage spectrum. To investigate whether minimal intestinal lesions in patients without endomysial antibodies are due to coeliac disease, their prevalence, causes and risk of evolving into frank coeliac disease were studied.Methods:From January 2000 to December 2005, 645 duodenal biopsies were performed. In 209 patients, duodenal biopsies were performed independently of endomysial antibody results. Clinical data and HLA-typing of all the patients negative to endomysial antibodies but with

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Anbardar, Mohammad Hossein, Neda Soleimani, Ehsan Torabi Dashtaki, Naser Honar, Mozhgan Zahmatkeshan, and Sahand Mohammadzadeh. "Do Serological Tests Eliminate the Need for Endoscopic Biopsy for the Diagnosis of Symptomatic Patients with Celiac Disease? A Retrospective Study with Review of Literature." Middle East Journal of Digestive Diseases 15, no. 4 (2023): 263–69. http://dx.doi.org/10.34172/mejdd.2023.356.

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Background: Celiac disease is one of the most common genetic allergies worldwide. The prevalence of celiac disease in Iran is similar to or even higher than the global prevalence. Celiac disease is a chronic inflammatory disease that affects the small intestine. Affected patients are allergic to gluten protein that exists in some grains, such as wheat and barley. Methods: Serological endomysial IgA antibody (EMA-AB) and tissue transglutaminase IgA antibody (TTG-IgA) tests were performed on 114 patients aged the ages of 0–18 years with histopathological findings of celiac disease. The results o

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10

Aktay, Atiye N., P. C. Lee, Vijay Kumar, Elaine Parton, David T. Wyatt, and Steven L. Werlin. "The Prevalence and Clinical Characteristics of Celiac Disease in Juvenile Diabetes in Wisconsin." Journal of Pediatric Gastroenterology and Nutrition 33, no. 4 (2001): 462–65. http://dx.doi.org/10.1002/j.1536-4801.2001.tb07502.x.

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ABSTRACTBackgroundThe relationship between celiac disease and juvenile diabetes has long been known. Only a single study in the United States, from Buffalo, New York, has reported the prevalence of celiac disease in a pediatric diabetic population. This study was conducted to determine the prevalence and clinical presentation of celiac disease in children and adolescents with juvenile diabetes in Wisconsin, U.S.A., using serum antiendomysial antibody as a screening test.MethodsTwo hundred eighteen patients with diabetes (113 males; age range, 4–21 years) and 117 age‐and gender‐matched control

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11

Cummins, Adrian, and Fiona Thompson. "Sensitivity of anti-endomysial antibody in detecting celiac disease." Gastroenterology 122, no. 1 (2002): 246–47. http://dx.doi.org/10.1053/gast.2002.30908.

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12

Prasad, Shyam, Peter Thomas, David S. Nicholas, Nicholas M. Sharer, and Jonathon A. Snook. "Adult endomysial antibody-negative coeliac disease and cigarette smoking." European Journal of Gastroenterology & Hepatology 13, no. 6 (2001): 667–71. http://dx.doi.org/10.1097/00042737-200106000-00009.

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13

HUSSAIN, S. "Does a negative anti-endomysial antibody exclude coeliac disease?" American Journal of Gastroenterology 96, no. 9 (2001): S106. http://dx.doi.org/10.1016/s0002-9270(01)03072-6.

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14

Boger, Christopher P. C., Peter W. Thomas, David S. Nicholas, Sue L. Surgenor, and Jonathon A. Snook. "Determinants of endomysial antibody status in untreated coeliac disease." European Journal of Gastroenterology & Hepatology 19, no. 10 (2007): 890–95. http://dx.doi.org/10.1097/meg.0b013e3282eeb472.

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15

McGowan, Kelly E., Martha E. Lyon, Steven D. Loken, and J. Decker Butzner. "Celiac Disease: Are Endomysial Antibody Test Results Being Used Appropriately?" Clinical Chemistry 53, no. 10 (2007): 1775–81. http://dx.doi.org/10.1373/clinchem.2007.090308.

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Abstract Background: The aim of this study was to retrospectively examine how positive IgA-endomysial antibody (EMA) test results for celiac disease were being interpreted and acted on by physicians in the Calgary Health Region. Methods: We reviewed consecutive EMA test results, with or without a serum IgA, obtained during a 17-month period. Seropositive tests were cross-referenced to the surgical database to determine the number of patients who underwent intestinal biopsy and the results of the biopsy. We sent questionnaires to the ordering physicians of positive tests with no record of intes

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16

James, M. W. "Endomysial antibody in the diagnosis and management of coeliac disease." Postgraduate Medical Journal 76, no. 898 (2000): 466–68. http://dx.doi.org/10.1136/pmj.76.898.466.

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17

Korponay-Szabó, Ilma R., Róbert Király, Judit Gyimesi, and Markku Mäki. "Development of Cell-Assembled Human Endomysial-Type Biomatrix Substrate for the Detection of Celiac Disease Autoantibodies." International Journal of Molecular Sciences 26, no. 3 (2025): 1012. https://doi.org/10.3390/ijms26031012.

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The endomysial antibody (EMA) immunofluorescent test is a highly specific method to detect disease-specific autoantibodies in celiac disease (CD) by their binding to natural transglutaminase-2 autoantigen in tissue sections, and it is used as a compulsory confirmatory test in the non-invasive diagnosis of CD. The classical EMA substrates are the monkey esophagus and the human umbilical cord. It is increasingly difficult to use these tissues due to ethical concerns and animal welfare regulations. In this study, we developed, in cell culture, an endomysium-type extracellular biomatrix assembled

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18

Salmi, T. T., P. Collin, I. R. Korponay-Szabo, et al. "Endomysial antibody-negative coeliac disease: clinical characteristics and intestinal autoantibody deposits." Gut 55, no. 12 (2006): 1746–53. http://dx.doi.org/10.1136/gut.2005.071514.

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19

Rittmeyer, Chris, and J. Marc Rhoads. "IgA Deficiency Causes False-Negative Endomysial Antibody Results in Celiac Disease." Journal of Pediatric Gastroenterology &amp Nutrition 23, no. 4 (1996): 504–6. http://dx.doi.org/10.1097/00005176-199611000-00029.

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20

Ferreira, M., S. L. Davies, M. Butler, D. Scott, M. Clark, and P. Kumar. "Endomysial antibody: is it the best screening test for coeliac disease?" Gut 33, no. 12 (1992): 1633–37. http://dx.doi.org/10.1136/gut.33.12.1633.

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21

Rittmeyer, Chris, and J. Marc Rhoads. "IgA Deficiency Causes False‐Negative Endomysial Antibody Results in Celiac Disease." Journal of Pediatric Gastroenterology and Nutrition 23, no. 4 (1996): 504–6. http://dx.doi.org/10.1002/j.1536-4801.1996.tb01712.x.

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22

Korponay–Szabó, Ilma R., Satu Sulkanen, Tuula Halttunen, et al. "Tissue Transglutaminase Is the Target in Both Rodent and Primate Tissues for Celiac Disease–Specific Autoantibodies." Journal of Pediatric Gastroenterology and Nutrition 31, no. 5 (2000): 520–27. http://dx.doi.org/10.1002/j.1536-4801.2000.tb07175.x.

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ABSTRACTBackgroundEndomysial antibodies have recently been shown to react with tissue transglutaminase. This study was undertaken to investigate whether the tissue distribution of transglutaminase is also compatible with reticulin, jejunal, and fibroblast autoantibody binding patterns.MethodsSera from patients with and without celiac disease, monoclonal tissue transglutaminase antibodies, and sera from mice parenterally immunized against commercially available tissue transglutaminase, transglutaminase complexed with gliadin, or gliadin were used in indirect immunofluorescence and double‐staini

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23

Prince, Harry E. "Evaluation of the INOVA Diagnostics Enzyme-Linked Immunosorbent Assay Kits for Measuring Serum Immunoglobulin G (IgG) and IgA to Deamidated Gliadin Peptides." Clinical and Vaccine Immunology 13, no. 1 (2006): 150–51. http://dx.doi.org/10.1128/cvi.13.1.150-151.2006.

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ABSTRACT New assays for antibodies to deamidated gliadin peptides (DGP) expressing celiac disease-specific epitopes were evaluated using 154 sera previously tested for endomysial immunoglobulin A (IgA) (EMA), transglutaminase IgA (TGA), and conventional gliadin antibodies. DGP antibody results showed 97% concordance with EMA and TGA results. Of 56 sera negative for EMA and TGA but positive for conventional gliadin antibodies, 54 (96%) were negative for DGP antibodies.

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24

Bharo, Mumtaz Ali, Kamran Ali Shahani Baloach, Bakhtiar Ahmed Bhanbhro, Hamza Ishfaq, and Kiran Abbas. "Common Presentation and Clinical Correlates of Celiac Disease in Pediatric Population of Pakistan." Pakistan Armed Forces Medical Journal 74, SUPPL-2 (2024): S101–104. http://dx.doi.org/10.51253/pafmj.v74isuppl-2.5082.

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Objective: To explore the various clinical presentations in childhood celiac disease patients of Sindh, Pakistan. Study Design: Cross-sectional study Setting and Duration of Study: Pediatric Department, Pir Abdul Qadir Shah Jeelani Institute of Medical Sciences, Gambat, Khaipur Mirs Pakistan, from Sep 2018 to Mar 2020. Methodology: A total of 140 children aged between 1-15 years, with diagnosed cases of celiac disease were enrolled in the study. Patients with inconclusive diagnosis, with multiple comorbidities, age >15 years, or children whose parents did not give consent to take part in th

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25

Ransford, Rupert A., Michael J. Hall, Martin Palmer, Valerie Bailey, Sara Price, and Mark Hayes. "Controlled prospective endomysial antibody screening for celiac disease in iron deficiency anemia." Gastroenterology 118, no. 4 (2000): A367. http://dx.doi.org/10.1016/s0016-5085(00)83575-1.

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26

Rossi, Thomas M., Vijay Kumar, Aaron Lerner, Leo A. Heitlinger, Neil Tucker, and John Fisher. "Relationship of Endomysial Antibodies to Jejunal Mucosal Pathology." Journal of Pediatric Gastroenterology and Nutrition 7, no. 6 (1988): 858–63. http://dx.doi.org/10.1002/j.1536-4801.1988.tb09655.x.

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SummarySerum immunoglobulin A class antibodies reactive to the endomysial lining of the smooth muscle bundles of the gastrointestinal tract have recently been reported to be specific and sensitive indicators of celiac disease (CD). A total of 203 subjects were examined for serum endomysial antibodies (EmA) and in 103 small bowel biopsies were obtained. EmA were detected in 43 cases including 26 CD patients evaluated during the gluten challenge phase of diagnosis by European Society for Pediatric Gastroenterology and Nutrition criteria, 11 of 53 symptomatic patients, and 6 asymptomatic family m

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27

Carroccio, A., N. Custro, G. Montalto, L. Giannitrapani, M. Soresi, and A. Notarbartolo. "Evidence of Transient IgA Anti-Endomysial Antibody Positivity in a Patient with Graves’ Disease." Digestion 60, no. 1 (1999): 86–88. http://dx.doi.org/10.1159/000007595.

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28

W. Dickey, A. McMillan, D. F. Hughe. "Sensitivity of Serum Tissue Transglutaminase Antibodies for Endomysial Antibody Positive and Negative Coeliac Disease." Scandinavian Journal of Gastroenterology 36, no. 5 (2001): 511–14. http://dx.doi.org/10.1080/00365520118975.

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Dickey, W., A. McMillan, and D. F. Hughes. "Sensitivity of Serum Tissue Transglutaminase Antibodies for Endomysial Antibody Positive and Negative Coeliac Disease." Scandinavian Journal of Gastroenterology 36, no. 5 (2001): 511–14. http://dx.doi.org/10.1080/003655201750153359.

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30

Biagi, Federico, Nick D. J. Parnell, Julia H. Ellis, and Paul J. Ciclitira. "Endomysial antibody production is not related to histological damage after in vitro gluten challenge." European Journal of Gastroenterology & Hepatology 12, no. 1 (2000): 57–60. http://dx.doi.org/10.1097/00042737-200012010-00011.

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31

Yiannakou, J. Y., D. Dell’Olio, M. Saaka, et al. "Detection and Characterisation of Anti-Endomysial Antibody in Coeliac Disease Using Human Umbilical Cord." International Archives of Allergy and Immunology 112, no. 2 (1997): 140–44. http://dx.doi.org/10.1159/000237445.

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32

Balasa, Adriana Luminita, Cristina Maria Mihai, Tatiana Chisnoiu, and Corina Elena Frecus. "Atypical presentations of celiac disease." ARS Medica Tomitana 22, no. 3 (2016): 181–85. http://dx.doi.org/10.1515/arsm-2016-0030.

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Abstract In this study we evaluated the association of celiac disease in 81 children with autoimmune disease and genetic syndromes over a two years periods (January 2014 to July 2016) in Pediatric Clinic in Constanta. Because the extraintestinal symptoms are an atypical presentation of celiac disease we determined in these children the presence of celiac disease antibodies: Anti-tissue Transglutaminase Antibody IgA and IgA total serum level as a screening method followeds in selective cases by Anti-tissue Transglutaminase Antibody IgG, anti-endomysial antibodies, deamidated gliadin antibodies

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33

Ozgenc, F., G. Aksu, S. Aydogdu, et al. "Association between anti-endomysial antibody and total intestinal villous atrophy in children with coeliac disease." Journal of Postgraduate Medicine 49, no. 1 (2003): 21. http://dx.doi.org/10.4103/0022-3859.933.

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34

Biagi, F., A. Andrealli, P. I. Bianchi, et al. "OC3.05.7 A GLUTEN-FREE DIET QUESTIONNAIRE COMPARED TO PERSISTENCE OF VILLOUS ATROPHY AND ENDOMYSIAL ANTIBODY." Digestive and Liver Disease 40 (March 2008): S51. http://dx.doi.org/10.1016/s1590-8658(08)60130-9.

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35

Barkin, J. S. "Correlation of Duodenal Histology With Tissue Transglutaminase and Endomysial Antibody Levels in Pediatric Celiac Disease." Yearbook of Medicine 2008 (January 2008): 434–36. http://dx.doi.org/10.1016/s0084-3873(08)79023-0.

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36

Donaldson, Matthew R., Sean D. Firth, Holly Wimpee, et al. "Correlation of Duodenal Histology With Tissue Transglutaminase and Endomysial Antibody Levels in Pediatric Celiac Disease." Clinical Gastroenterology and Hepatology 5, no. 5 (2007): 567–73. http://dx.doi.org/10.1016/j.cgh.2007.01.003.

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37

W. Dickey, D. F. Hughes, S. A. McMi. "Reliance on Serum Endomysial Antibody Testing Underestimates the True Prevalence of Coeliac Disease by One Fifth." Scandinavian Journal of Gastroenterology 35, no. 2 (2000): 181–83. http://dx.doi.org/10.1080/003655200750024362.

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38

Wimpee, Holly, Kris Leiferman, John Zone, et al. "SEVERITY OF HISTOLOGICAL CHANGES IN CELIAC DISEASE CORRELATES WITH IGA ENDOMYSIAL AND TISSUE TRANSGLUTAMINASE ANTIBODY LEVELS." Journal of Pediatric Gastroenterology and Nutrition 41, no. 4 (2005): 493. http://dx.doi.org/10.1097/01.mpg.0000181857.91264.f6.

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39

Shah, Vipul H., Winson Lo, Heidrun Rotterdam, Susie K. Lee, Neeraj Katriyar, and Peter Hr Green. "Correlation of biopsy findings and endomysial antibody status with disease severity in patients with celiac disease." Gastroenterology 118, no. 4 (2000): A371. http://dx.doi.org/10.1016/s0016-5085(00)83591-x.

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40

Leonard, J. N., T. P. Chorzelski, E. H. Beutner, et al. "IgA anti-endomysial antibody detection in the serum of patients with dermatitis herpetiformis following gluten challenge." Archives of Dermatological Research 277, no. 5 (1985): 349–51. http://dx.doi.org/10.1007/bf00509231.

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41

Kapuscinska, Alicja, Tadeusz Zalewski, Tadeusz P. Chorzelski, et al. "Disease Specificity and Dynamics of Changes in IgA Class Anti‐endomysial Antibodies in Celiac Disease." Journal of Pediatric Gastroenterology and Nutrition 6, no. 4 (1987): 529–34. http://dx.doi.org/10.1002/j.1536-4801.1987.tb09346.x.

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SummaryWe evaluated the sensitivity and kinetics of serum IgA class anti‐endomysial antibodies in the diagnosis of celiac disease (CD) as established by the criteria of the European Society for Pediatric Gastroenterology and Nutrition (ESPGAN). Eighty‐four cases that satisfied the ESPGAN criteria for CD were evaluated for IgA‐EmA titers during various phases of establishing the diagnosis. Thirty‐three cases were infants and children <5 years of age undergoing intestinal biopsies for symptoms of CD and 51 were previously diagnosed adults. Of the 33 children, 11 were untreated and symptomatic

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42

Rangnekar, A. S., and W. D. Chey. "Review: tissue transglutaminase test is almost as accurate as endomysial antibody test for diagnosis of coeliac disease." Evidence-Based Medicine 12, no. 1 (2007): 24. http://dx.doi.org/10.1136/ebm.12.1.24.

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43

Atkinson, Kenneth, Sonya Tokmakajian, William Watson, and James Gregor. "Evaluation of the Endomysial Antibody for Celiac Disease: Operating Properties and Associated Cost Implications in Clinical Practice." Canadian Journal of Gastroenterology 11, no. 8 (1997): 673–77. http://dx.doi.org/10.1155/1997/813148.

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OBJECTIVE:To evaluate the operating properties of endomysial antibodies (EMAs) in the diagnosis of celiac disease and to examine, using a cost minimization model, different strategies used in the diagnosis of celiac disease.METHODS:A total of 248 EMA results were reviewed and compared with small bowel biopsy results in 66 patients who had undergone both tests. Regression analysis was used to look for predictors of positive EMA results and positive biopsy results. A cost minimization model from a societal perspective was used to evaluate the cost differences among three different strategies.RES

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44

Almarzooqi, Saeeda, Ronald H. Houston, and Vinay Prasad. "Utility of Tissue Transglutaminase Immunohistochemistry in Pediatric Duodenal Biopsies: Patterns of Expression and Role in Celiac Disease—A Clinicopathologic Review." Pathology Research International 2013 (September 8, 2013): 1–5. http://dx.doi.org/10.1155/2013/602985.

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Tissue transglutaminase (tTG) is a ubiquitous multifunctional protein. It has roles in various cellular processes. tTG is a major target of autoantibodies in celiac disease, and its expression by immunohistochemistry in pediatric celiac disease has not been fully examined. We studied tTG expression in 78 pediatric duodenal biopsies by utilizing an antibody to transglutaminase 2. Serum tTG was positive in all celiac cases evaluated. Serum antiserum endomysial antibody (EMA) and tTG were negative in all control subjects and in inflammatory bowel disease and eosinophilic gastroenteritis. There wa

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Shahzad, Amir, Abdul Aziz Sahto, and Miss Samina. "FREQUENCY OF CELIAC DISEASE." Professional Medical Journal 23, no. 07 (2016): 812–16. http://dx.doi.org/10.29309/tpmj/2016.23.07.1644.

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Objectives: The aim behind this study was to determine the frequency of Celiacdisease in patients presenting with Iron deficiency anemia at tertiary care hospital. StudyDesign: Descriptive cross sectional hospital based study. Setting: Non-probability consecutivesampling technique in a department of medicine at Medical Unit II, People’s Medical University,Shaheed Benazeerabad. Period: Six months from 26th October 2014 to 25th April 2015. Patientsand Methods: A standard sample bottle was used to collect the blood sample from the peripheralvein followed by testing for Anti-tissue transglutaminas

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46

Vázquez, Horacio, María de la Paz Temprano, Emilia Sugai, et al. "Prevalence of Celiac Disease and Celiac Autoimmunity in the Toba Native Amerindian Community of Argentina." Canadian Journal of Gastroenterology and Hepatology 29, no. 8 (2015): 431–34. http://dx.doi.org/10.1155/2015/927458.

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BACKGROUND: Celiac disease (CD) is mostly recognized among subjects with a Caucasian ethnic ancestry. No studies have explored conditions predisposing Amerindians to CD.OBJECTIVE: To prospectively assess environmental, genetic and serological conditions associated with CD among members of the Toba native population attending a multidisciplinary sanitary mission.METHODS: An expert nutritionist determined daily gluten intake using an established questionnaire. Gene typing for the human leukocyte antigen (HLA) class II alleles was performed on DNA extracted from peripheral blood (HLA DQ2/DQ8 hapl

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47

Picarelli, A. "31-43 Amino Acid Sequence of the a-Gliadin Induces Anti-Endomysial Antibody Production during in Vitro Challenge." Scandinavian Journal of Gastroenterology 34, no. 11 (1999): 1099–102. http://dx.doi.org/10.1080/003655299750024896.

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48

Chakrabarti, Sabyasachi, Koichi S. Kobayashi, Richard A. Flavell, et al. "Impaired membrane resealing and autoimmune myositis in synaptotagmin VII–deficient mice." Journal of Cell Biology 162, no. 4 (2003): 543–49. http://dx.doi.org/10.1083/jcb.200305131.

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Members of the synaptotagmin family have been proposed to function as Ca2+ sensors in membrane fusion. Syt VII is a ubiquitously expressed synaptotagmin previously implicated in plasma membrane repair and Trypanosoma cruzi invasion, events which are mediated by the Ca2+-regulated exocytosis of lysosomes. Here, we show that embryonic fibroblasts from Syt VII–deficient mice are less susceptible to trypanosome invasion, and defective in lysosomal exocytosis and resealing after wounding. Examination of mutant mouse tissues revealed extensive fibrosis in the skin and skeletal muscle. Inflammatory m

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Jatla, Muralidhar, Caroline Kieserman-Shmokler, and Ritu Verma. "Endomysial Antibody Testing Improves Sensitivity in Screening for Celiac Disease in Young Children; a Five Year Single Center Experience." American Journal of Gastroenterology 102 (September 2007): S550—S551. http://dx.doi.org/10.14309/00000434-200709002-01184.

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Singh, Alka, Atreyi Pramanik, Pragyan Acharya, and Govind K. Makharia. "Non-Invasive Biomarkers for Celiac Disease." Journal of Clinical Medicine 8, no. 6 (2019): 885. http://dx.doi.org/10.3390/jcm8060885.

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Once thought to be uncommon, celiac disease has now become a common disease globally. While avoidance of the gluten-containing diet is the only effective treatment so far, many new targets are being explored for the development of new drugs for its treatment. The endpoints of therapy include not only reversal of symptoms, normalization of immunological abnormalities and healing of mucosa, but also maintenance of remission of the disease by strict adherence of the gluten-free diet (GFD). There is no single gold standard test for the diagnosis of celiac disease and the diagnosis is based on the

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