Relevant bibliographies by topics / Fish kidney disease study

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Academic literature on the topic 'Fish kidney disease study'

Author: Grafiati
Published: 4 June 2021
Last updated: 5 February 2022

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Journal articles on the topic "Fish kidney disease study"

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Barman, Ditul, and Rajiv V. Gaikwad. "Evaluation of kidney status by ultrasonography in canines: an experimental study." Research in Agriculture Livestock and Fisheries 1, no. 1 (February 23, 2015): 105–8. http://dx.doi.org/10.3329/ralf.v1i1.22373.

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Kidney is a major organ for homeostasis of the body's extracellular fluids by maintaining a constant internal environment. By playing such crucial responsibility kidney remains vulnerable for various diseases and disease condition. Other than laboratory findings, application of ultrasonography can be considered as one of the finest diagnostic tool to determine the architectural detail of kidney. To compare the study, we have taken two groups of canine, namely healthy and clinical. The later grouped was screened based on clinical signs, Blood Urea Nitrogen (BUN) and Serum Creatinine value. Sonography was performed by using Brightness mode instrument for both the groups. The values were analyzed by Prolate Ellipsoid formula to estimate the volume of kidney. There was significant variation in sizes between the kidneys of both the groups. Moreover, the size of the kidneys of few clinical cases, which has been suffering from renal disorder since long, reveals smaller size kidney than the healthy groups. Thus, this noninvasive method is quite practicable to evaluate the size of kidney amongst various renal failure cases. DOI: http://dx.doi.org/10.3329/ralf.v1i1.22373 Res. Agric., Livest. Fish.1(1): 105-108, Dec 2014
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ABD-ELFATTAH, AHMED, INÊS FONTES, GOKHLESH KUMAR, HATEM SOLIMAN, HANNA HARTIKAINEN, BETH OKAMURA, and MANSOUR EL-MATBOULI. "Vertical transmission ofTetracapsuloides bryosalmonae(Myxozoa), the causative agent of salmonid proliferative kidney disease." Parasitology 141, no. 4 (November 7, 2013): 482–90. http://dx.doi.org/10.1017/s0031182013001650.

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SUMMARYThe freshwater bryozoan,Fredericella sultana, is the main primary host of the myxozoan endoparasite,Tetracapsuloides bryosalmonaewhich causes proliferative kidney disease (PKD) of salmonid fish. Because spores that develop in bryozoan colonies are infectious to fish, bryozoans represent the ultimate source of PKD. Bryozoans produce numerous seed-like dormant stages called statoblasts that enable persistence during unfavourable conditions and achieve long-distance dispersal. The possibility thatT. bryosalmonaemay undergo vertical transmission via infection of statoblasts has been the subject of much speculation since this is observed in close relatives. This study provides the first evidence that such vertical transmission ofT. bryosalmonaeis extensive by examining the proportions of infected statoblasts in populations ofF. sultanaon two different rivers systems and confirms its effectiveness by demonstrating transmission from material derived from infected statoblasts to fish hosts. Vertical transmission in statoblasts is likely to play an important role in the infection dynamics of both bryozoan and fish hosts and may substantially contribute to the widespread distribution of PKD.
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Arslan, Gökhan. "Cytokine Gene Expression, Immune Responses and Disease Resistance of Oncorhynchus mykiss after Raphanus sativus By-products Supplementation." Turkish Journal of Fisheries and Aquatic Sciences 21, no. 10 (June 24, 2021): 521–34. http://dx.doi.org/10.4194/1303-2712-v21_10_05.

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In the present study, we examined the effects of aqueous methanolic extract of radish seed (Raphanus sativus) by-products (RS) on innate immune responses and growth performance of rainbow trout (Oncorhynchus mykiss). The fish was fed diets containing 4 different doses of RS (0 % (Control), 0.1% (RS0.1), 0.5% (RS0.5) and 1% (RS1)) for 14 days. The results showed an increased activity of respiratory burst in fish of treatment groups compared to that of control on 14th day (P<0.05). An enhanced bacterial killing activity was observed in R.05 and RS1 treatment groups compared to control (P<0.05) on 7th day of the study. Lysozyme activity was elevated in fish of RS1 group on 7th day, and in all treatment groups on 14th day compared to that of the control. Myeloperoxidase activity increased significantly in RS1 and RS0.1 groups compared to the control on 7th day. IL-1b was up- regulated in head kidney of fish in RS0.1 group on 7th day and in RS0.5 group on 14th day of the study. In intestine of fish of experimental groups, IL-1b expression was significantly elevated on 7th day compared to the control. IL-12 was also up-regulated both in kidney and intestine of treatment group fish on 7th day. Similar results were observed on IL-10 expression. IL8 was up-regulated both in kidney and intestine of treated fish groups. Growth performance was affected positively in the RS1 group compared to the control. However, FCR value did not vary among different groups. Survival also improved against Aeromonas hydrophila infection in RS administered fish. All these results suggest that supplementation of RS through diets for 7 days could improve immune responses and growth in rainbow trout.
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Schmidt-Posthaus, Heike, Ernst Schneider, Nils Schölzel, Regula Hirschi, Moritz Stelzer, and Armin Peter. "The role of migration barriers for dispersion of Proliferative Kidney Disease—Balance between disease emergence and habitat connectivity." PLOS ONE 16, no. 3 (March 17, 2021): e0247482. http://dx.doi.org/10.1371/journal.pone.0247482.

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Natural and uninterrupted water courses are important for biodiversity and fish population stability. Nowadays, many streams and rivers are obstructed by artificial migration barriers, often preventing the migration of fish. On the other hand, distribution of pathogens by migrating fishes is still a point of concern. Pathogen transport and transmission is a driving force in the dynamics of many infectious diseases. The aim of the study was to investigate the possible consequences of the removal of an artificial migration barrier for the upstream transport of Tetracapsuloides bryosalmonae, the causative agent of Proliferative Kidney Disease (PKD) in brown trout, by migrating fish. To test this question, a river system was selected with a migration barrier separating a PKD positive river from a PKD negative tributary. After removal of the barrier, PKD prevalence and pathology was examined during five years after elimination of the barrier. In the tributary, no PKD was recorded at any time of the survey. By means of unidirectional PIT (passive integrated transponder)-tagging, we confirmed upstream migration of adult brown trout into the tributary during the cold season, presumably for spawning. By eDNA, we confirmed presence of T. bryoalmonae and Fredericella sp., the definitive host, DNA in water from the PKD positive river stretch, but not in the PKD negative tributary. Our study illustrates the importance of the connectivity of streams for habitat maintenance. Although migration of brown trout from a PKD-positive river into a PKD-negative tributary, mainly for spawning, was confirmed, upstream spreading of PKD was not observed.
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Gopinath, Bamini, David C. Harris, Victoria M. Flood, George Burlutsky, and Paul Mitchell. "Consumption of long-chain n-3 PUFA, α-linolenic acid and fish is associated with the prevalence of chronic kidney disease." British Journal of Nutrition 105, no. 9 (January 24, 2011): 1361–68. http://dx.doi.org/10.1017/s0007114510005040.

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Due to the anti-inflammatory properties of PUFA, it has been suggested that they may protect against kidney damage in adults. However, relatively few epidemiological studies have examined this hypothesis in human subjects. We investigated the association between dietary intakes of PUFA (n-3, n-6 and α-linolenic acid), fish and the prevalence of chronic kidney disease (CKD). A total of 2600 Blue Mountains Eye Study (1997–9) participants aged ≥ 50 years were analysed. Dietary data were collected using a semi-quantitative FFQ, and PUFA and fish intakes were calculated. Baseline biochemistry including serum creatinine was measured. Moderate CKD was defined as an estimated glomerular filtration rate of < 60 ml/min per 1·73 m2. Participants in the highest quartile of long-chain n-3 PUFA intake had a significantly reduced likelihood of having CKD compared with those in the lowest quartile of intake (multivariable-adjusted OR 0·69, 95 % CI 0·49, 0·99). α-Linolenic acid intake was positively associated with CKD (OR, per standard deviation increase in α-linolenic acid, 1·18, 95 % CI 1·05, 1·32). Total n-3 PUFA or total n-6 PUFA were not significantly associated with CKD. The highest compared with the lowest quartile of fish consumption was associated with a reduced likelihood of CKD (OR 0·68, 95 % CI 0·48, 0·97; P for trend = 0·02). The present study shows that an increased dietary intake of long-chain n-3 PUFA and fish reduces the prevalence of CKD. Hence, a diet rich in n-3 PUFA and fish could have a role in maintaining healthy kidney function, in addition to roles of these nutrients in the prevention and modulation of other diseases.
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Saulnier, D., and P. de Kinkelin. "Antigenic and biochemical study of PKX, the myxosporean causative agent of proliferative kidney disease of salmonid fish." Diseases of Aquatic Organisms 27 (1996): 103–14. http://dx.doi.org/10.3354/dao027103.

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Murwantoko, Murwantoko, Eka Diniarti, and Triyanto Triyanto. "Isolation, Characterization and Pathogenicity of Edwardsiella tarda a Causative Disease on Freshwater Fish in Yogyakarta." Jurnal Perikanan Universitas Gadjah Mada 21, no. 1 (August 2, 2019): 41. http://dx.doi.org/10.22146/jfs.39920.

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Edwarsiella tarda is a cosmopolitan bacterium and is a cause of Edwardsiellosis in various fish species. The bacterial infection causes large losses on aquaculture in Asia, especially Japan. This study was conducted to isolate and characterize E. tarda as causative disease in freshwater fishes, and to determine its pathogenicity to catfish (Pangasius sp.). Bacteria were isolated from kidney of diseased fishes on Tryptone Soya Agar medium. Identification was conducted based on morphological colonies, morphological cells and biochemical tests. Fulfillment of Koch Postulates was done by injecting bacteria intraperitoneally on 7-9 cm fishes at dose of 107 cfu/fish. Pathogenicity test was carried out by intraperitoneal injection at 104, 105, 106, and 107 cfu/fish to 7-9 cm-catfish (Pangasius sp.) and followed by observation of disease signs and mortality every six hours for 7 days. Pathogenicity was determined as Lethal Dosage (LD50) using Dragstedt Behrens method. In this research we have isolated three isolates E. tarda causing disease in fishes. The clinical signs of this disease were lose of pigmentation over the lession, swollen of stomach, haemorhage on fins , small cutaneous lesions, and necrotic on fins area. The LD50 of E. tarda isolate L2, L3, and N3 were 4.64 ± 0.35x105, 1.54 ± 0.07x105, and 1.13 ± 0.13x106 cfu/fish, respectively.
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Carraro, Luca, Enrico Bertuzzo, Lorenzo Mari, Inês Fontes, Hanna Hartikainen, Nicole Strepparava, Heike Schmidt-Posthaus, et al. "Integrated field, laboratory, and theoretical study of PKD spread in a Swiss prealpine river." Proceedings of the National Academy of Sciences 114, no. 45 (October 23, 2017): 11992–97. http://dx.doi.org/10.1073/pnas.1713691114.

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Proliferative kidney disease (PKD) is a major threat to wild and farmed salmonid populations because of its lethal effect at high water temperatures. Its causative agent, the myxozoanTetracapsuloides bryosalmonae, has a complex lifecycle exploiting freshwater bryozoans as primary hosts and salmonids as secondary hosts. We carried out an integrated study of PKD in a prealpine Swiss river (the Wigger). During a 3-year period, data on fish abundance, disease prevalence, concentration of primary hosts’ DNA in environmental samples [environmental DNA (eDNA)], hydrological variables, and water temperatures gathered at various locations within the catchment were integrated into a newly developed metacommunity model, which includes ecological and epidemiological dynamics of fish and bryozoans, connectivity effects, and hydrothermal drivers. Infection dynamics were captured well by the epidemiological model, especially with regard to the spatial prevalence patterns. PKD prevalence in the sampled sites for both young-of-the-year (YOY) and adult brown trout attained 100% at the end of summer, while seasonal population decay was higher in YOY than in adults. We introduce a method based on decay distance of eDNA signal predicting local species’ density, accounting for variation in environmental drivers (such as morphology and geology). The model provides a whole-network overview of the disease prevalence. In this study, we show how spatial and environmental characteristics of river networks can be used to study epidemiology and disease dynamics of waterborne diseases.
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Mastuti, Indah, and Ketut Mahardika. "DISTRIBUTION ANALYSIS OF ENLARGED CELLS DERIVED FROM GROUPER SLEEPY DISEASE IRIDOVIRUS (GSDIV) INFECTED HUMPBACK GROUPER Cromileptes altivelis." Indonesian Aquaculture Journal 7, no. 1 (June 30, 2012): 55. http://dx.doi.org/10.15578/iaj.7.1.2012.55-60.

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Characteristic of Megalocytivirus infection has been known to produce formation of inclusion body bearing cells (IBCs) on internals organs of fish predominantly on spleen and kidney. Megalocytivirus that infected grouper is known as Grouper Sleepy Disease Iridovirus (GSDIV). This study was conducted to answer the effect of entry sites of GSDIV on distribution of enlarged cells formed on the internal organs of humpback grouper Cromileptes altivelis. Enlarged cells were observed histologically under the light microscope on spleen, head kidney, trunk kidney, liver, gill, heart, stomach, intestine, muscle and brain. Entry sites were designated to intramuscularly injection, intraperitoneally injection, dipped gill and inoculum added feed. Enlarged cells were formed on spleen, head kidney, trunk kidney, liver, gill, heart, stomach, muscle, except on intestine and brain. All the entry sites resulted in formation of enlarged cells on spleen, head kidney, trunk kidney, liver, heart. Spleen and head kidney were the most frequent observed organ. These results suggested that distribution of enlarged cells were not affected by the entry site of GSDIV.
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Chang, Chia-Hsuan, Sayuj Poudyal, Theeraporn Pulpipat, Pei-Chi Wang, and Shih-Chu Chen. "Pathological Manifestations of Francisella orientalis in the Green Texas Cichlid (Herichthys cyanoguttatus)." Animals 11, no. 8 (August 3, 2021): 2284. http://dx.doi.org/10.3390/ani11082284.

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Francisella orientalis (Fo) is considered to be one of the major pathogens of tilapia because of the high mortalities observed during outbreaks. Other cichlids belonging to the same family (Cichlidae) as tilapia are also quite susceptible to this pathogen. On various occasions, Fo has also been isolated from other warm water fish, including three-line grunt, hybrid striped bass, French grunt, Caesar grunt, and Indo-Pacific reef fish. However, only a few studies have reported the pathogenicity of Francisella orientalis in ornamental cichlid fish. This study fulfills Koch’s postulates by showing that a strain of Fo obtained from green Texas cichlid (Herichthys cyanoguttatus) was able to produce the same pathogenicity in healthy fish. A mortality of 100% was observed after healthy green Texas cichlid were experimentally injected with Fo at a dose of 8.95 × 105 CFU/fish. DNA extracted from the organs of predilection (spleen, head kidney) gave positive results by PCR for all fish that died during the experimental period. Spleen and head kidney presented with multifocal white nodules in the affected fish, corresponding to typical vacuolated granulomas on histopathological examination of the tissues. Based on the results of this study, it is evident that Fo can indeed infect green Texas cichlid and produce a disease typical of francisellosis.
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Dissertations / Theses on the topic "Fish kidney disease study"

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Connor, T. M. F. "A study of inherited kidney disease." Thesis, University College London (University of London), 2013. http://discovery.ucl.ac.uk/1398923/.

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Kidney disease is a common, expensive, and growing worldwide health problem. Genetic factors play an important role in the aetiology of kidney disease. Current research suggests that these genetic factors are predominantly rare variants with large phenotypic effects. In this thesis I have used a range of genetic techniques to identify rare variants in different families with kidney disease, and to study how they might cause disease. The Turkish-Cypriot population of Northern Cyprus has a high incidence of renal disease, much of which is undiagnosed and may be inherited. I collected DNA from the entire population on renal replacement therapy and identified three individuals with the G871C mutation in COL4A3. I used conflicting homozygosity analysis to demonstrate a minimal shared haplotype, thus dating this mutation to 17 generations ago. I used linkage analysis and whole genome sequencing in a large Greek-Cypriot kindred to identify 3 novel non-synonymous variants associated with kidney disease. Expression of these variants was examined in cultured primary urothelial cells from this family. I have studied another large pedigree with maternal transmission of renal disease. Sequencing of the mitochondrial genome demonstrated the presence of a novel polymorphism in the heavy strand promoter region at homoplastic levels. Mitochondrial function in primary dermal fibroblasts demonstrated a significant reduction in baseline oxidative respiration with a compensatory increase in glycolysis. Lastly, I have studied a novel compound heterozygous mutation in VHL. This variant showed abnormal degradation of HIF without activation of HIF target genes in patient-derived B-cells. It is possible that these cells are able to employ some kind of VHL-independent HIF regulatory mechanism. These studies demonstrate, in differing ways, the challenges of linking phenotype to genotype. Understanding the pathological and therapeutic importance of genetic information will become increasingly important to our management of kidney disease in this post-genomic era.
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Manfredi, Eugene Trent. "Immunodiagnostic methods for the detection of bacterial kidney disease in salmonid fishes /." Thesis, Connect to this title online; UW restricted, 1986. http://hdl.handle.net/1773/5282.

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Hamel, Owen Sprague. "The dynamics and effects of bacterial kidney disease in Snake River spring Chinook salmon (Oncorhynchus tshawytscha) /." Thesis, Connect to this title online; UW restricted, 2001. http://hdl.handle.net/1773/6364.

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Bettge, Kathrin. "The proliferative kidney disease of salmonids : dynamics of the parasite in the fish host /." [S.l.] : [s.n.], 2008. http://opac.nebis.ch/cgi-bin/showAbstract.pl?sys=000286556.

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Campos-Perez, Juan Jose. "The role of reactive oxygen and nitrogen species in the immune response of rainbow trout to Renibacterium salmoninarum." Thesis, University of Aberdeen, 1998. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU112258.

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The role of reactive oxygen and nitrogen intermediates in the immune response of rainbow trout to R.s. was investigated. The early events occurring when the pathogen interacted with trout macrophages were assessed in terms of the respiratory burst elicited. Live R.s. elicited a respiratory burst, which was enhanced by heat-killed microorganism. This phenomenon, though, was not observed using UV-killed bacteria. Both responses were enhanced when a combination of LPS and TNF was used to activate the macrophages prior to contact R.s. Further studies demonstrated that both compounds synergised to enhance superoxide (O2) production, and that this was correlated with the ability to kill the pathogen. Opsonisation of R.s. with serum factors also increased the respiratory burst, but no difference was found between normal serum and heat-inactivated serum. The role of NO in the immune response of rainbow trout is also studied. Though no evidence of NO production was found in vitro, i.p. injection of live R.s. produced higher NO levels in serum as compared to controls. Fish injected with a virulent strain showed higher levels of NO than controls and than fish injected with an avirulent strain and other strains of unknown virulence. Fish vaccinated with killed R.s. and FIA also showed a significant increase in NO levels, but only four days after vaccination, decreasing thereafter, at both doses of vaccine tested. Injected of Brivax II, an attenuated strain of Aeromonas salmonicida, did not produce a significant increase of NO. RT-PCR was used to detect the expression of the iNOS in different tissues of rainbow trout. iNOS expression was seen only in gill and kidney after i.p. injection. iNOS was detected in the gills 6 h after injecting live R.s. and the expression was still present at day 5. iNOS was detected in the kidney 24 h after injection but was switched off at day 3. After bath challenge with the bacterium, iNOS was expressed in gill, gut and kidney, but the expression varied in each fish. No iNOS expression was found in macrophages isolated from challenged fish.
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Jansson, Eva. "Bacterial kidney disease in salmonid fish : development of methods to assess immune functions in salmonid fish during infection by Renibacterium salmoninarum /." Uppsala : Swedish Univ. of Agricultural Sciences (Sveriges lantbruksuniv.), 2002. http://epsilon.slu.se/avh/2002/91-576-6352-1.pdf.

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Mazur, Carl François. "Growth, incidence of bacterial kidney disease and immunological function of salmonids reared in captivity." Thesis, University of British Columbia, 1991. http://hdl.handle.net/2429/30127.

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Pacific salmon reared commercially off of the Coast of British Columbia suffer great mortality losses to Bacterial Kidney Disease (BKD), caused by the diplobacillus bacterium Renibacterium salmqninarum. This thesis investigates the effects of environmental conditions on the growth performance and disease susceptibility of salmonids reared in captivity. I found that growth rate of chinook salmon was significantly higher in fish fed to 100 compared to 67 % of satiation during the first 175 days of saltwater rearing but not during the first winter. Feed coversion rate was significantly higher for fish fed at 100 % of satiation compared to 67 % of satiation and higher during the winter compared to summer and fall, irrespective of feeding level. Mortality rates were significantly higher during the summer than during the fall or winter, irrespective of experimental treatment. The last BKD sampling period (day 263) revealed that infection rates were directly proportional to stocking densities of 1.5 to 4 kg.m⁻₃. Hatchery-reared chinook salmon held in freshwater aquaria had significantly lower hematocrit and plasma cortisol concentration increases in response to increased stocking density than did their wild counterparts. Crowding of hatchery-reared and wild chinook salmon resulted in equally increased mortality rates for both groups of fish. Day 33 plasma cortisol concentrations in Atlantic salmon held at three stocking densities were directly proportional to stocking densities of 8 to 64 kg.m⁻₃. The ability of anterior kidney lymphocytes from these fish to produce antibody-producing cells was inversely proportional to the density at which the fish were held.
Land and Food Systems, Faculty of
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Hill, C. "Diabetic kidney disease : a study of management and outcomes." Thesis, Queen's University Belfast, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.676518.

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Diabetes mellitus is the most common cause of end-stage renal disease requiring renal replacement therapy in the United Kingdom. However, many aspects of the prevalence, management and outcomes of diabetic patients with chronic kidney disease (CKD) remain unclear. This thesis consists of three studies using local (Northern Irish) data, national data (from the National Diabetes Audit) and international data (combined as part of a multi-centre collaborative meta-analysis). Its aims were to assess the survival of Northern Irish diabetic patients with CKD, examine the prevalence and associations of diabetes-related CKD in the UK National Diabetes Audit and to assess the association between glycosylated haemoglobin (HbA1c) and survival in diabetic haemodialysis patients.
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Gast, Christine. "A study of the genetics of chronic kidney disease." Thesis, University of Southampton, 2015. https://eprints.soton.ac.uk/397107/.

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This study examines the prevalence and distribution of genetic kidney diseases in a cohort of chronic kidney disease (CKD) patients. The literature and national registries hold a paucity of information on genetic kidney disease prevalence in adult patients with CKD. Through a questionnaire study of all patients of the Wessex Renal and Transplant Service in CKD stages 3-5 on family history, a systematic database search and patient interviews, this study established a prevalence of genetic kidney diseases other than polycystic kidney disease(PKD) of 7.6% amongst end-stage renal disease patients and 3.8% amongst CKD patients, which is higher than previously reported. The study reveals uromodulin associated kidney disease (UAKD) to be the most prevalent genetic kidney disease after PKD, which has not been reported previously. The prevalence for UAKD in Wessex was established at 8.5 per million by UMOD gene sequencing. This is much higher than the only published prevalence of 1.7 per million in Austria. Established diagnostic biochemical tests for UAKD were evaluated and found to have relatively poor sensitivity and specificity –70 and 45% respectively in the case of the fractional excretion of urate, and 70 and 63% for urinary uromodulin, measured by enzyme linked immunosorbent assay (ELISA). On review of clinical phenotypes, hyperuricaemia and gout as the typical clinical features were not statistically associated with UAKD, highlighting the need for gene testing to establish the diagnosis. 81 patients with the clinico-pathological diagnosis focal segmental glomerulosclerosis (FSGS) were recruited to examine underlying gene mutations by a custom-designed targeted next generation sequencing (NGS) panel. Underlying gene mutations were established in 13-20% of patients, which is higher than in previous adult series. Of relevance, the most frequent mutations occurred in the collagen 4 gene, which was unexpected and changed the clinical diagnosis of these patients to Alport disease. Half of the collagen 4 mutations occurred in COL4A5, which was a previously unpublished finding. Whole exome sequencing (WES) was employed in the search for a genetic diagnosis in a previously undiagnosed family. A systematic analysis of both renal and pan-genomic variants failed to identify a disease-causing variant and illustrated the challenges of WES, leading to a discussion of future genetic investigations by NGS.
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Goldstein, D. Jordi. "Effects of selective manipulation of fatty acids in experimental chronic renal disease." Thesis, Boston University, 1993. https://hdl.handle.net/2144/31818.

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Thesis (D.Sc.N.S.)--Boston University, Henry M. Goldman School of Graduate Dentistry, 1993 (Nutritional Sciences).
Includes bibliography (leaves 176-187)
This dissertation has been presented in two related studies: A. Fish Oil Reduces Proteinuria and Interstitial Injury but not GIomerulosclerosis in the Milan Nomotensive Rat Rats of the Milan Normotensive strain (MNS) spontaneously develop severe Proteinuria and excessive glomemlar thromboxane (Tx)A2 PrOduction at a young age. These are accompanied by podocyte alterations and progressive focal glomerulosclerosis (FGS) and interstitial fibrosis. Since previous studies showed that pharmacologic... [TRUNCATED]
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Books on the topic "Fish kidney disease study"

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Kaattari, S. L. Development of a vaccine for bacterial kidney disease in salmon: Final report. Portland, Or: U.S. Dept. of Energy, Bonneville Power Administration, Division of Fish & Wildlife, 1991.

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O'Connor, Glenda. Use of ELISA for monitoring bacterial kidney disease in naturally spawning chinook salmon. Salem, Or: Oregon Dept. of Fish and Wildlife, 2006.

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Moffitt, Christine A. FDA approved registration of erythromycin for treatment of bacterial kidney disease (BKD) in juvenile and adult chinook salmon: Annual report, reporting period: year 1, 10 March 1989 - 9 March 1990. Portland, OR: U.S. Dept. of Energy, Bonneville Power Administration, Division of Fish and Wildlife, 1991.

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Abernethy, C. Scott. Feasibility study for evaluating cummulative exposure of downstream migrant juvenile salmonids to total dissolved gas: Final report. Portland, OR: Bonneville Power Administration, 1998.

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Stephen, Kaattari, United States. Bonneville Power Administration. Division of Fish and Wildlife., and Oregon State University. Dept. of Microbiology., eds. Development of a vaccine for bacterial kidney disease in salmon: Annual report FY 1984. Portland, Or: U.S. Dept. of Energy, Bonneville Power Administration, Division of Fish & Wildlife, 1985.

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Hutchison, Alastair J., and Michael L. Picton. Fractures in patients with chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0121.

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Patients with any degree of chronic kidney disease (CKD) have a much higher risk of fractures than the general population, and the risk of death at 1 year post hip fracture in a dialysis patient is over 60%, compared to less than 20% for a non-CKD patient. The assessment of fracture risk and diagnosis of the underlying skeletal pathology in CKD patients is a significant clinical challenge. Non-invasive imaging techniques are not totally reliable in the general population, and the presence of advanced CKD (stages 4, 5, and 5D) renders them largely useless. Bone strength is not determined only by quantity of bone, and renal osteodystrophy can significantly affect bone quality, rendering it liable to fracture even in the presence of a normal bone density measurement. Currently, the only reliable method of assessing both quantity and quality of bone is the examination of trans-iliac bone biopsy, which is generally, but probably incorrectly, perceived to be overly invasive. However, identifying the cause of reduced bone strength and fractures may influence the choice of therapy. For example, in the presence of low-turnover states such as adynamic bone, antiresorptive agents may be ineffective. Pharmaceuticals licensed for the treatment of osteoporosis in the general population can be used similarly in patients with CKD 1–3 without dosage alteration. In CKD 4, post-hoc analyses suggest denosumab is effective and safe, based on a 3-year study that included 73 such patients. In CKD 5 and 5D no dependable data exists to guide therapy, and it should probably be reserved for patients who have already suffered and survived a fracture, and are therefore at high risk of death from a second event.
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Jardine, Alan G., and Rajan K. Patel. Lipid disorders of patients with chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0102.

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The risk of developing cardiovascular (CV) disease is increased in patients with chronic kidney disease (CKD) and although dyslipidaemia is a major contributory factor to the development of premature CV disease, the relationship is complex. Changes in lipid fractions are related to glomerular filtration rate and the presence and severity of proteinuria, diabetes, and other confounding factors. The spectrum of CV disease changes from lipid-dependent, atheromatous coronary disease in early CKD to lipid-independent, non-coronary disease, manifesting as heart failure, and sudden cardiac death in advanced and end-stage renal disease. Statin-based lipid-lowering therapy is proven to reduce coronary events across the spectrum of CKD. The relative reduction in overall CV events, however, diminishes as CKD progresses and the proportion of lipid-dependent coronary events declines. There is nevertheless a strong argument for the use of statin-based therapy across the spectrum of CKD. The argument is particularly strong for those patients with progressive renal disease who will eventually require transplantation, in whom preventive therapy should start as early as possible. The SHARP study established the benefits and endorses the use of lipid-lowering therapy in CKD 3-4 but uncertainty about the value of initiation of statin therapy in CKD 5 remains. There is, however, no rationale for stopping agents started earlier in the course of the illness for compelling indications, particularly in those who will ultimately be transplanted. The place of high-density lipoprotein-cholesterol raising and triglyceride lowering therapy needs to be assessed in trials. Modifying dyslipidaemia in CKD has demonstrated that lipid-dependent atheromatous cardiovascular disease is only one component of the burden of CV disease in CKD patients, that this is proportionately less in advanced CKD, and that modification of lipid profiles is only one part of CV risk management.
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Biggar, Patrick, Hansjörg Rothe, and Markus Ketteler. Epidemiology of calcium, phosphate, and parathyroid hormone disturbances in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0109_update_001.

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Chronic kidney disease-mineral and bone disorders (CKD-MBD), calcium, phosphate, and parathyroid hormone are biomarkers of mortality and cardiovascular risk. Hyperphosphataemia is a prominent and pathophysiologically most plausible risk indicator. Calcium balance and load appear to be more important than serum concentrations. Parathyroid hormone is a less reliable marker with a relatively wide range extending above that applicable for a normal population especially when used as a singular laboratory parameter without additional assessment of bone metabolism, for example, bone-specific alkaline phosphatase and bone biopsy. There is not a single prospective controlled hard-outcome study that provides us with unequivocal evidence that such an isolated laboratory parameter-based treatment approach will lead to significant clinical improvements. As CKD-MBD is complex, clinical decisions would be made easier by informative prospective trials.
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Speeckaert, Marijn, and Jopis Delanghe. Assessment of renal function. Edited by Christopher G. Winearls. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0007.

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Glomerular filtration rate (GFR) can be measured as the clearance of exogenous or endogenous filtration markers. Practical formulas permit estimation of creatinine clearance or GFR without timed urine collections in many stable patients with CKD. Standardization of serum creatinine is important for all of these estimation methods and implementing traceability of the assays to the new global SRM 967 standard has led to changes in clinical decision-making criteria. Calibration to an IDMS reference produces a lowering of serum creatinine values by 10–30% for most methods. Serum creatinine concentration depends on age, gender and muscle mass. Cystatin C is an alternative marker of GFR, but estimation is more expensive and it is not clear that it has a useful place in routine practice. The MDRD Study equation was validated in the framework of the Modification of Diet in Renal Diseases study. It is superior to the Cockcroft and Gault formula for estimating Creatinine Clearance in most people. In 2009, the CKD Epidemiology Collaboration (CKD-EPI) formula was introduced, which provides a more accurate estimation for patients with GFR values between 60 and 90 mL/min. In children, the Schwartz formula is frequently used. Some urinary markers of kidney disease are also discussed.
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Book chapters on the topic "Fish kidney disease study"

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Elliott, Diane G., Gregory D. Wiens, K. Larry Hammell, and Linda D. Rhodes. "Vaccination against Bacterial Kidney Disease." In Fish Vaccination, 255–72. Chichester, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118806913.ch22.

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Furuichi, Kengo. "Study at AMED Collecting 600 Biopsy-Proven Diabetic Nephropathies." In Diabetic Kidney Disease, 119–31. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-9301-7_9.

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Schulsinger, David A. "Room #2: The Test I Could Not Study For!" In Kidney Stone Disease, 3–9. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-12105-5_1.

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Morris, David J., and Alexandra Adams. "PCR and in Situ Hybridisation of Tetracapsula Bryosalmonae (PKX), the Causative Agent of Proliferative Kidney Disease." In Reviews: Methods and Technologies in Fish Biology and Fisheries, 299–313. Dordrecht: Springer Netherlands, 2002. http://dx.doi.org/10.1007/978-94-017-2315-2_12.

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Tikariha, Pritha, and Prashant Richhariya. "Comparative Study of Chronic Kidney Disease Prediction Using Different Classification Techniques." In Lecture Notes in Networks and Systems, 195–203. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-8198-9_20.

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Nakanuma, Yasuni, Yasunori Sato, and Kenichi Harada. "Tissue Culture Correlational Study of Genetic Cholangiopathy of Autosomal Recessive Polycystic Kidney Disease." In Methods in Molecular Biology, 303–18. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-125-7_18.

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Guidi, G., C. Rossini, C. Cinelli, V. Meucci, and I. Lippi. "Canine Chronic Kidney Disease: Retrospective Study of a 10-Year Period of Clinical Activity." In Veterinary Science, 115–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-23271-8_19.

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Intas, George, Vasiliki Rokana, Pantelis Stergiannis, Eleftheria Chalari, and Fotios Anagnostopoulos. "Burden and Sleeping Disorders of Family Caregivers of Hemodialysis Patients with Chronic Kidney Disease-End Stage: A Cross-Sectional Study." In Advances in Experimental Medicine and Biology, 33–40. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-32637-1_4.

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Rajapakshe, S. M., S. K. Weragoda, T. Kawakami, and W. B. M. L. I. Weerasekara. "Study of Evidences on Chronic Kidney Disease Due to Unknown Etiology Based on Environmental, Social, Economic and Health Patterns of Selected Population." In Lecture Notes in Civil Engineering, 99–106. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-9749-3_9.

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Ku, Min-Chi, Adrian Schreiber, Paula Ramos Delgado, Philipp Boehm-Sturm, Ralph Kettritz, Thoralf Niendorf, Andreas Pohlmann, and Sonia Waiczies. "Fluorine (19F) MRI for Assessing Inflammatory Cells in the Kidney: Experimental Protocol." In Methods in Molecular Biology, 495–507. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-0978-1_30.

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AbstractInflammation is one underlying contributing factor in the pathology of acute and chronic kidney disorders. Phagocytes such as monocytes, neutrophils and dendritic cells are considered to play a deleterious role in the progression of kidney disease but may also contribute to organ homeostasis. The kidney is a target of life-threatening autoimmune disorders such as the antineutrophil cytoplasmic antibody (ANCA)-associated vasculitides (AAV). Neutrophils and monocytes express ANCA antigens and play an important role in the pathogenesis of AAV. Noninvasive in vivo methods that can quantify the distribution of inflammatory cells in the kidney as well as other organs in vivo would be vital to identify the causality and significance of inflammation during disease progression. Here we describe an noninvasive technique to study renal inflammation in rodents in vivo using fluorine (19F) MRI. In this protocol we chose a murine ANCA-AAV model of renal inflammation and made use of nanoparticles prepared from perfluoro-5-crown-15-ether (PFCE) for renal 19F MRI.This chapter is based upon work from the COST Action PARENCHIMA, a community-driven network funded by the European Cooperation in Science and Technology (COST) program of the European Union, which aims to improve the reproducibility and standardization of renal MRI biomarkers. This experimental protocol chapter is complemented by two separate chapters describing the basic concept and data analysis.
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Conference papers on the topic "Fish kidney disease study"

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Rabby, A. K. M. Shahariar Azad, Rezwana Mamata, Monira Akter Laboni, Ohidujjaman, and Sheikh Abujar. "Machine Learning Applied to Kidney Disease Prediction: Comparison Study." In 2019 10th International Conference on Computing, Communication and Networking Technologies (ICCCNT). IEEE, 2019. http://dx.doi.org/10.1109/icccnt45670.2019.8944799.

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Lelii, Mara, Laura Senatore, William Morello, Sara Testa, Francesca Taroni, Barbara Madini, Maria Carola Pensabene, Marinella Lavelli, and Maria Francesca Patria. "Sleep-disordered breathing in children with chronic kidney disease: a pilot study." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.1229.

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Al Khalaf, SY, EJ O’Reilly, FP McCarthy, M. Kublickas, K. Kublickiene, and AS Khashan. "OP70 Cohort study of adverse pregnancy outcomes in women with chronic kidney disease." In Society for Social Medicine and Population Health Annual Scientific Meeting 2020, Hosted online by the Society for Social Medicine & Population Health and University of Cambridge Public Health, 9–11 September 2020. BMJ Publishing Group Ltd, 2020. http://dx.doi.org/10.1136/jech-2020-ssmabstracts.69.

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Gierten, J., F. Loosli, J. Gehrig, C. Pylatiuk, T. Fitzgerald, E. Birney, J. Wittbrodt, and M. Gorenflo. "Systems Genetics Approach to Study Congenital Heart Disease in a Fish Model." In 50th Annual Meeting of the German Society for Pediatric Cardiology (DGPK). Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1628326.

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Mageau, Arthur, Jean-Francois Timsit, Anne Perozziello, Benedicte Giroux Leprieur, Stephane Ruckly, Claire Dupuis, Lila Bouadma, Thomas Papo, and Karim Sacre. "THU0270 THE BURDEN OF CHRONIC KIDNEY DISEASE IN SYSTEMIC LUPUS ERYTHEMATOSUS: A NATIONWIDE EPIDEMIOLOGIC STUDY." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.1359.

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Devika, R., Sai Vaishnavi Avilala, and V. Subramaniyaswamy. "Comparative Study of Classifier for Chronic Kidney Disease prediction using Naive Bayes, KNN and Random Forest." In 2019 3rd International Conference on Computing Methodologies and Communication (ICCMC). IEEE, 2019. http://dx.doi.org/10.1109/iccmc.2019.8819654.

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Yang, Yi, Qiong He, Hong Zhang, Lanyan Qiu, Linxue Qian, Fu-Feng Lee, Zhi Liu, and Jianwen Luo. "Assessment of Diabetic Kidney Disease Using Ultrasound Localization Microscopy: An in Vivo Feasibility Study in Rats." In 2018 IEEE International Ultrasonics Symposium (IUS). IEEE, 2018. http://dx.doi.org/10.1109/ultsym.2018.8579963.

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Huang, Zhongping, Jie Ren, and Anilchandra Attaluri. "Experimental Study of a Hybrid Renal Replacement System." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14326.

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Kidney failure is a major issue in the United States. The numbers of kidney failure patients are rapidly increasing with the simultaneous rise in diabetes, obesity and hypertension1. Kidney transplantation has shown excellent results, but insufficiency of donors has been a limiting factor. Most end-stage renal disease (ESRD) patients depend on hemodialysis (HD) for survival which is highly expensive. On an average ESRD patients receive 3 dialysis treatment a week and 4 hours per treatment, i.e., approximately 12 hours a week. Technology has not yet reached to a state where all the kidney functions can be mimicked. The only major kidney function being performed in HD is toxin removal. Even the toxins are not being continuously removed from the patients. To compensate the toxin and fluid removal of a whole week within 12 hours, high volumes of fluid are removed in HD treatments. Patients suffer due to the high fluid removal in a short period of time. Also the patients are restricted from taking fluids between the HD treatments. More frequent HD can improve both survival rate and life quality of patients with chronic kidney disease since normal people has his kidneys functioning continuously. It is a well known fact that daily dialysis offers many benefits over regular intermittent HD1. But providing daily dialysis is not affordable currently. Therefore, new modes of delivering continuous renal support are required.
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Xun, Liu, Lv Linsheng, Ling Li, and Lou Tanqi. "A Markov Model Study on the Hierarchical Prognosis and Risk Factors in Patients with Chronic Kidney Disease." In 2012 International Conference on Computer Science and Electronics Engineering (ICCSEE). IEEE, 2012. http://dx.doi.org/10.1109/iccsee.2012.104.

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Gomes Ramalho de Oliveira, Juliana, José Eurico Vasconcelos Filho, and Geraldo Bezerra da Silva Junior. "Renal Health - A New Tool for Chronic Kidney Disease - Application Development and a Proposal for Interventional Study." In 11th International Conference on Health Informatics. SCITEPRESS - Science and Technology Publications, 2018. http://dx.doi.org/10.5220/0006533302610265.

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