Relevant bibliographies by topics / Flufenamic acid

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Flufenamic acid'

Author: Grafiati
Published: 4 June 2021
Last updated: 5 February 2022

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Journal articles on the topic "Flufenamic acid"

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&NA;. "Etofenamate/flufenamic acid." Reactions Weekly &NA;, no. 676 (1997): 8. http://dx.doi.org/10.2165/00128415-199706760-00016.

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Fatima, Rabab, Mousmee Sharma, and Parteek Prasher. "Targeted delivery of flufenamic acid by V-amylose." Therapeutic Delivery 12, no. 8 (2021): 575–82. http://dx.doi.org/10.4155/tde-2021-0020.

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Aim: Controlled release of flufenamic acid by helical V-amylose to achieve enzyme-responsive, targeted release of the cargo drug. Materials & methods: Solid-state cross-polarization magic angle spinning carbon-13 nuclear magnetic resonance (CP/MAS NMR), Fourier transform IR and x-ray diffraction (XRD) analysis validated the entrapment of flufenamic acid inside the helical structure of V-amylose. Scanning electron microscopy (SEM) investigations established the morphology of conjugates in simulated gastric environment (pH 1.2) and simulated intestine media (pH 7.2) containing hydrolyzing en
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Farrugia, G., S. Nitecki, G. J. Harty, M. Camilleri, and J. H. Szurszewski. "The effect of flufenamic acid on gastrointestinal myoelectrical activity and transit time in dogs." Gut 42, no. 2 (1998): 258–65. http://dx.doi.org/10.1136/gut.42.2.258.

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Background—Flufenamic acid, a fenamate, has been shown to alter markedly the membrane potential of small intestinal smooth muscle and increase intracellular calcium in single cells.Aims—To determine the effects of flufenamic acid on myoelectrical motor activity and gastrointestinal transit in the intact animal.Methods—Myoelectrical motor activity was recorded via seromuscular platinum electrodes sutured at regular intervals in the stomach and throughout the small intestine. Fasted and fed gastrointestinal transit was assessed using technetium-99m (99mTc) as the radioactive marker linked to 1 m
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Carasso, Ralph L., Odead Peled, and Shlomo Yehuda. "Flufenamic Acid in Prostaglandin Migraine." International Journal of Neuroscience 27, no. 1-2 (1985): 67–71. http://dx.doi.org/10.3109/00207458509149135.

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Salem, H., and A. A. Kheir. "Atomic Absorption Spectrometry of Flufenamic Acid." Analytical Letters 28, no. 10 (1995): 1833–43. http://dx.doi.org/10.1080/00032719508000361.

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Zhu, X. Y., N. Y. Li, and Z. Y. Song. "The effect of flufenamic acid on arachidonic acid metabolism." European Journal of Pharmacology 183, no. 6 (1990): 2250–51. http://dx.doi.org/10.1016/0014-2999(90)93790-w.

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GE, RUILIANG, LEI HU, YILIN TAI, et al. "Flufenamic acid promotes angiogenesis through AMPK activation." International Journal of Oncology 42, no. 6 (2013): 1945–50. http://dx.doi.org/10.3892/ijo.2013.1891.

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Guinamard, Romain, Christophe Simard, and Christopher Del Negro. "Flufenamic acid as an ion channel modulator." Pharmacology & Therapeutics 138, no. 2 (2013): 272–84. http://dx.doi.org/10.1016/j.pharmthera.2013.01.012.

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Salem, Hesham, Magda El-Maamli, and Abdalla Shalaby. "Atomic Absorption Spectrometry of Acidic Pharmaceutical Constituents by Precipitation Using Co (II), Cd (II) and Mn (II)." Scientia Pharmaceutica 68, no. 4 (2000): 343–55. http://dx.doi.org/10.3797/scipharm.aut-00-32.

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Simple and accurate methods are described for the quantitative determination of flufenamic acid, mefenamic acid and tranexamic acid utilizing precipitation reactions with cobalt, cadmium and manganese. The acidic drugs were precipitated from their neutral alcoholic solutions with cobalt sulphate, cadmium nitrate or manganese chloride standard solutions followed by direct determination of the ions in the precipitate or indirect determination of the ions in the filtrate by atomic absorption spectroscopy. The optimum conditions for precipitation were carefully studied. The molar ratio of the reac
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Zhang, Keke, Noalle Fellah, Vilmalí López-Mejías, and Michael D. Ward. "Polymorphic Phase Transformation Pathways under Nanoconfinement: Flufenamic Acid." Crystal Growth & Design 20, no. 11 (2020): 7098–103. http://dx.doi.org/10.1021/acs.cgd.0c01207.

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Dissertations / Theses on the topic "Flufenamic acid"

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Guo, Minshan. "Investigating the dissolution and permeation properties of flufenamic acid cocrystals." Thesis, De Montfort University, 2018. http://hdl.handle.net/2086/17464.

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The purpose of this study is to improve the solubility, dissolution rate and permeability of poorly water-soluble drugs by investigating the dissolution and permeation properties of flufenamic acid cocrystals in the presence of polymers. This study was separated into four tasks: (1) Formation of pharmaceutical cocrystals: Two pharmaceutical cocrystals of poorly water soluble active pharmaceutical ingredient (API) flufenamic acid (FFA) were synthesized, including 1:1 flufenamic acid-nicotinamide cocrystal (FFA-NIC CO) and 1:1 flufenamic acid-theophylline cocrystal (FFA-TP CO). The results of in
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Burt, Rees A. "9-Phenanthrol and flufenamic acid inhibit calcium oscillations in HL-1 mouse cardiomyocytes." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/honors/186.

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Electrical potentials exist across the membranes of nearly every cell type in the body. In addition, excitable cells, such as neurons, myocytes and even some endocrine cells elicit electrochemical fluctuations, action potentials (AP), in the cell membrane to initiate cell-to-cell communication or intracellular processes. The basis for the electrical potential is rooted within an array of complex interactions between monovalent ions and their associated membrane channels and transporters that regulate the flux of these charged species across the hydrophobic bilayer. Here, an expansion of our
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Rubio, Toledano Laia. "Sistemes bicel•lars com a nova estratègia d’aplicació tòpica." Doctoral thesis, Universitat de Barcelona, 2012. http://hdl.handle.net/10803/84071.

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Durant les últimes dècades, hi ha hagut gran interès en l'exploració de noves tècniques per modular l'absorció d’un fàrmac a través de la pell. Aquests estudis apunten a la necessitat d'obtenir vehicles de mida adequada, alta estabilitat i biocompatibilitat. Els sistemes bicel•lars són nanoagregats constituïts per fosfolípids de cadena alquílica llarga i fosfolípids de cadena curta. Depenent de la composició i de la temperatura aquests sistemes poden tenir diferents formes sent les més comunes les nanoestructures discoïdals anomenades bicel•les, on els fosfolípids de cadena llarga estan ordena
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Caniceiro, Ana Beatriz Ramos. "O papel dos halogénios no desenho de fármacos. O caso da ligação de pequenos ligantes à Transtirretina." Master's thesis, 2019. http://hdl.handle.net/10316/87875.

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Dissertação de Mestrado em Química Medicinal apresentada à Faculdade de Ciências e Tecnologia<br>Abnormal protein folding/unfolding followed by protein aggregation is often associated to disease development. That is the case of human Transthyretin (TTR), a homotetrameric protein found in plasma, cerebrospinal fluid and the humour of the eye, responsible for thyroxine and retinol transport, in association with the retinol-binding protein. TTR is known to undergo conformational changes and form soluble aggregates and insoluble highly stable amyloid fibrils that accumulate extracellularly in the
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Pippione, A. C., I. M. Carnovale, D. Bonanni, et al. "Potent and selective aldo-keto reductase 1C3 (AKR1C3) inhibitors based on the benzoisoxazole moiety: application of a bioisosteric scaffold hopping approach to flufenamic acid." 2018. http://hdl.handle.net/10454/16082.

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Yes<br>The aldo-keto reductase 1C3 (AKR1C3) isoform plays a vital role in the biosynthesis of androgens and is considered an attractive target in prostate cancer (PCa). No AKR1C3-targeted agent has to date been approved for clinical use. Flufenamic acid and indomethacine are non-steroidal anti-inflammatory drugs known to inhibit AKR1C3 in a non-selective manner as COX off-target effects are also observed. Recently, we employed a scaffold hopping approach to design a new class of potent and selective AKR1C3 inhibitors based on a N-substituted hydroxylated triazole pharmacophore. Following a sim
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Book chapters on the topic "Flufenamic acid"

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Evanson, Nathan K. "Flufenamic Acid." In xPharm: The Comprehensive Pharmacology Reference. Elsevier, 2007. http://dx.doi.org/10.1016/b978-008055232-3.61758-1.

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"Flufenamic acid and meclofenamic acid." In Meyler's Side Effects of Drugs. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-444-53717-1.00755-1.

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"Flufenamic acid and meclofenamic acid." In Meyler's Side Effects of Drugs: The International Encyclopedia of Adverse Drug Reactions and Interactions. Elsevier, 2006. http://dx.doi.org/10.1016/b0-44-451005-2/01258-4.

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Conference papers on the topic "Flufenamic acid"

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MARIN, Maria Minodora, Ştefania MARIN, Elena DĂNILĂ, et al. "Influence of the Formulation and Preparation Technique on the Flufenamic Acid Release from Different Collagenic Supports Designed for Wound Healing." In The 7th International Conference on Advanced Materials and Systems. INCDTP - Leather and Footwear Research Institute (ICPI), Bucharest, Romania, 2018. http://dx.doi.org/10.24264/icams-2018.i.19.

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Journal articles
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