Relevant bibliographies by topics / Heart – Differentiation

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Heart – Differentiation'

Author: Grafiati
Published: 4 June 2021
Last updated: 13 February 2022

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Journal articles on the topic "Heart – Differentiation"

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Hildick-Smith, D. J. R. "Echocardiographic differentiation of pathological and physiological left ventricular hypertrophy." Heart 85, no. 6 (2001): 615–19. http://dx.doi.org/10.1136/heart.85.6.615.

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Kempf, Tibor, and Kai C. Wollert. "Growth-Differentiation Factor-15 in Heart Failure." Heart Failure Clinics 5, no. 4 (2009): 537–47. http://dx.doi.org/10.1016/j.hfc.2009.04.006.

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Chen, J. N., and M. C. Fishman. "Zebrafish tinman homolog demarcates the heart field and initiates myocardial differentiation." Development 122, no. 12 (1996): 3809–16. http://dx.doi.org/10.1242/dev.122.12.3809.

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The fashioning of a vertebrate organ requires integration of decisions of cell fate by individual cells with those that regulate organotypic form. Logical candidates for this role, in an organ such as the heart, are genes that initiate the differentiation process leading to heart muscle and those that define the earliest embryonic heart field, but for neither class are genes defined. We cloned zebrafish Nkx2.5, a homolog of the tinman homeodomain gene needed for visceral and cardiac mesoderm formation in Drosophila. In the zebrafish, its expression is associated with cardiac precursor cells th
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Davis, L. A., and L. F. Lemanski. "Induction of myofibrillogenesis in cardiac lethal mutant axolotl hearts rescued by RNA derived from normal endoderm." Development 99, no. 2 (1987): 145–54. http://dx.doi.org/10.1242/dev.99.2.145.

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A strain of axolotl, Ambystoma mexicanum, that carries the cardiac lethal or c gene presents an excellent model system in which to study inductive interactions during heart development. Embryos homozygous for gene c contain hearts that fail to beat and do not form sarcomeric myofibrils even though muscle proteins are present. Although they can survive for approximately three weeks, mutant embryos inevitably die due to lack of circulation. Embryonic axolotl hearts can be maintained easily in organ culture using only Holtfreter's solution as a culture medium. Mutant hearts can be induced to diff
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Kazez, A., I. H. Özercan, and P. S. Erol. "Sacrococygeal heart: a very rare differentiation in teratoma." Journal of Pediatric Surgery 38, no. 6 (2003): 990. http://dx.doi.org/10.1016/s0022-3468(03)00142-8.

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Kirkpatrick, Michael A., and Andrew S. Groves. "Verbal Feedback Facilitates Heart Rate Discrimination and Differentiation." European Journal of Behavior Analysis 12, no. 2 (2011): 431–39. http://dx.doi.org/10.1080/15021149.2011.11434393.

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Kazez, A., İ. H. Özercan, F. S. Erol, M. Faik Özveren, and E. Parmaksız. "Sacrococcygeal Heart: A Very Rare Differentiation in Teratoma." European Journal of Pediatric Surgery 12, no. 4 (2002): 278–80. http://dx.doi.org/10.1055/s-2002-34483.

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THOMPSON, R. P., J. R. LINDROTH, A. J. ALLES, and A. R. FAZEL. "Cell Differentiation Birthdates in the Embryonic Rat Heart." Annals of the New York Academy of Sciences 588, no. 1 Embryonic Ori (1990): 446–48. http://dx.doi.org/10.1111/j.1749-6632.1990.tb13259.x.

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Chen, Wei-Qian, Chuanfu Li, Hai-Bin Ruan, Xuan Jiang, Xin Qi, and Xiang Gao. "Myeloid Differentiation Protein-88 Signaling Mediates Heart Failure." Journal of Cardiac Failure 13, no. 6 (2007): S79. http://dx.doi.org/10.1016/j.cardfail.2007.06.395.

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Drenckhahn, Jörg-Detlef. "Heart Development: Mitochondria in Command of Cardiomyocyte Differentiation." Developmental Cell 21, no. 3 (2011): 392–93. http://dx.doi.org/10.1016/j.devcel.2011.08.021.

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Dissertations / Theses on the topic "Heart – Differentiation"

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Litster, Caroline Elizabeth. "Heart rate, heart rate variability, electrodermal activity and the differentiation-of-deception /." Title page, table of contents and abstract only, 2002. http://web4.library.adelaide.edu.au/theses/09SSPS/09sspsl7769.pdf.

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Hinds, Heather C. "Evaluating terminal differentiation of porcine valvular interstitial cells in vitro." Link to electronic thesis, 2006. http://www.wpi.edu/Pubs/ETD/Available/etd-050506-113014/.

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O'Brien, Meghan M. "A pilot proteomic analysis : the study of P19 cells in cardiac differentiation /." Connect to resource online, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=ysu1229374725.

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Buccini, Stephanie M. "Cardiogenic differentiation of induced pluripotent stem cells for regeneration of the ischemic heart." University of Cincinnati / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1382373160.

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Younce, Craig. "Zinc-Finger Protein MCPIP in Cell Death and Differentiation." Doctoral diss., University of Central Florida, 2009. http://digital.library.ucf.edu/cdm/ref/collection/ETD/id/2279.

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Monocyte chemotactic protein-1 (MCP-1) plays a critical role in the development of cardiovascular diseases. How MCP-1 contributes to the development of heart disease is not understood. We present evidence that MCP-1 causes death in cardiac myoblasts, H9c2 by inducing oxidative stress, ER stress and autophagy via a novel Zn-finger protein, MCP-1 induced protein (MCPIP). MCPIP expression caused cell death and knockdown of MCPIP, attenuated MCP-1 induced cell death. Expression of MCPIP resulted in induction of iNOS and production of reactive oxygen (ROS). It caused induction of NADPH oxidase subu
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Wei, Wenjie, and 魏闻捷. "Calcium signaling in the cardiac differentiation of mouse embryonic stem cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B49617862.

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　　Intracellular Ca2+ mobilization via secondary messengers modulates multiple cell functions. Cyclic Adenosine 5’-Diphosphate-Ribose (cADPR) is one of the most well recognized endogenous Ca2+ mobilizing messengers. In mammalian, cADPR is mainly formed by CD38, a multi-functional enzyme, from nicotinamide adenine dinucleotide (NAD). It has previously been shown that the cADPR/CD38/Ca2+pathway mediates many cardiac functions, such as regulating the excitation-contraction coupling in cardiac myocytes and modulating the Ca2+ homeostasis during the ischemia injury of the heart. Thus it is reasonab
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Chau, Dinh Le Mary. "Role of Notch1 in Cardiac Cell Differentiation and Migration: A Dissertation." eScholarship@UMMS, 2007. https://escholarship.umassmed.edu/gsbs_diss/338.

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The cardiac conduction system is responsible for maintaining and orchestrating the rhythmic contractions of the heart. Results from lineage tracing studies indicate that precursor cells in the ventricles give rise to both cardiac muscle and conduction cells. Using chick embryonic hearts, we have found that Notch signaling plays an important role in the differentiation of cardiac muscle and conduction cell lineages in the ventricles. Notch1 expression coincides with a conduction marker at early stages of conduction system development. Mis-expression of constitutively active Notch1 (NIC) in earl
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Maliken, Bryan D. B. A. "Gata4-Dependent Differentiation of c-Kit+ Derived Endothelial Cells Underlies Artefactual Cardiomyocyte Regeneration in the Heart." University of Cincinnati / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1535375861364685.

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Huang, Tianfang. "Mechanism of Arsenical Toxicity on TGFβ Signaling and Genetic Regulation During Cardiac Progenitor Cell Differentiation". Diss., The University of Arizona, 2015. http://hdl.handle.net/10150/556428.

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Low to moderate level of chronic arsenic exposure contributes to cardiovascular ailments including heart disease and aneurysms. Current research on the etiology and progression of cardiovascular disease focuses mainly on adult which fails to capture the developmental origins of cardiovascular disease. Thus, disruption in morphogenetic events during early development may initiate and pattern the molecular programming of cardiovascular ailments in adulthood. A major contributor to ischemic heart pathologies is coronary artery disease, however the influences by environmental arsenic in this disea
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Momtahan, Nima. "Extracellular Matrix from Whole Porcine Heart Decellularization for Cardiac Tissue Engineering." BYU ScholarsArchive, 2016. https://scholarsarchive.byu.edu/etd/6225.

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Heart failure is one of the leading causes of death in the United States. Every year in the United States, more than 800,000 people are diagnosed with heart failure and more than 375,000 people die from heart disease. Current therapies such as heart transplants and bioartificial hearts are helpful, but not optimal. Decellularization of porcine whole hearts followed by recellularization with patient-specific human cells may provide the ultimate solution for patients with heart failure. Great progress has been made in the development of efficient processes for decellularization, and the design o
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Books on the topic "Heart – Differentiation"

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J, Hearse David, ed. The developing myocardium. Futura Pub. Co., 1991.

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Cell cycle regulation and differentiation in cardiovascular and neural systems. Springer, 2010.

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Watson, Andrea. Heat shock proteins in leukaemia cell differentiation and cell death. Aston University. Departmentof Pharmaceutical Sciences, 1990.

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Lesuisse, Christian. Role of the constitutive heat shock portein HSC70 during differentiation of haemopoieticcells. University of Manchester, 1994.

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B, Clark Edward, Markwald Roger R, and Takao Atsuyoshi, eds. Developmental mechanisms of heart disease. Futura Pub., 1995.

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De la Cruz, María Victoria. and Markwald Roger R, eds. Living morphogenesis of the heart. Birkhäuser, 1998.

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B, Clark Edward, and Takao Atsuyoshi, eds. Developmental cardiology: Morphogenesis and function. Futura Pub. Co., 1990.

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Takao, Atsuyoshi, and Edward Clark. Developmental Cardiology: Morphogenesis and Function. Futura Pub Co, 1990.

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Giordano, Antonio, and Umberto Galderisi. Cell Cycle Regulation and Differentiation in Cardiovascular and Neural Systems. Springer, 2014.

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van den Hoff, Maurice J. B., and Antoon F. M. Moorman. From heart-forming region to ballooning chambers. Edited by Miguel Torres. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0006.

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This chapter describes the formation of the adult four-chambered heart from the precardiac mesodermal cells. The precardiac mesoderm develops into a linear heart tube by the process of folding. The subsequent increase in size of the heart by the addition of precursor cells derived from the first and second heart fields is discussed. For the sake of clarity, the chapter describes the addition of precursor cells to the inflow and outflow, separately. Next, the formation of the ventricular chambers with respect to ballooning and differentiation into a compact and trabecular layer is discussed. Fi
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Book chapters on the topic "Heart – Differentiation"

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Makino, Shinji, and Keiichi Fukuda. "Methods for Differentiation of Bone-Marrow-Derived Stem Cells into Myocytes." In Regenerating the Heart. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-021-8_6.

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Lescroart, Fabienne, and Sigolène M. Meilhac. "Cell Lineages, Growth and Repair of the Mouse Heart." In Results and Problems in Cell Differentiation. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-30406-4_15.

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Linask, Kérsti K. "Regulatory Role of Cell Adhesion Molecules in Early Heart Development." In Formation and Differentiation of Early Embryonic Mesoderm. Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3458-7_24.

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Rajamannan, Nalini M., Muzaffer Cicek, John R. Hawse, Thomas C. Spelsberg, and Malayannan Subramaniam. "Experimental Model of Aortic Valve Calcification to Induce Osteoblast Differentiation." In Molecular Biology of Valvular Heart Disease. Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-6350-3_4.

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Rajamannan, Nalini M. "Ex Vivo Model for Bioprosthetic Valve Calcification via Stem Cell Differentiation to Bone." In Molecular Biology of Valvular Heart Disease. Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-6350-3_7.

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Müller, Mathias M., Helmut RumpoId, Gerhard Schopf, and Peter Zilla. "Changes of Purine Metabolism During Differentiation of Rat Heart Myoblasts." In Purine and Pyrimidine Metabolism in Man V. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-1248-2_74.

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Osmond, Mark. "The Effects of Retinoic Acid on Early Heart Formation and Segmentation in the Chick Embryo." In Formation and Differentiation of Early Embryonic Mesoderm. Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3458-7_23.

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Hiraumi, Yoshimi, Chengqun Huang, Allen M. Andres, Ying Xiong, Jennifer Ramil, and Roberta A. Gottlieb. "Myogenic Progenitor Cell Differentiation Is Dependent on Modulation of Mitochondrial Biogenesis through Autophagy." In Etiology and Morphogenesis of Congenital Heart Disease. Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-54628-3_15.

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Rajamannan, Nalini M., Muzaffer Cicek, John R. Hawse, Thomas C. Spelsberg, and Malayannan Subramaniam. "In Vitro Cell Culture Model of Calcification: Molecular Regulation of Myofibroblast Differentiation to an Osteoblast Phenotype." In Molecular Biology of Valvular Heart Disease. Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-6350-3_2.

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Sato, Yohei, Takashi Higuchi, Hiroshi Kobayashi, Susumu Minamisawa, Hiroyuki Ida, and Toya Ohashi. "Lentiviral Gene Transfer to iPS Cells: Toward the Cardiomyocyte Differentiation of Pompe Disease-Specific iPS Cells." In Etiology and Morphogenesis of Congenital Heart Disease. Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-54628-3_48.

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Conference papers on the topic "Heart – Differentiation"

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Hacke, M., J. Signoret-Genest, P. Tovote, and M. Romanos. "Definition, detection and differentiation of acute emotional states using heart rate recording." In Abstracts of the 2nd Symposium of the Arbeitsgemeinschaft für Neuropsychopharmakologie und Pharmakopsychiatrie (AGNP) and Deutsche Gesellschaft für Biologische Psychiatrie (DGBP). Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0039-3403003.

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Yi, Wei-guo, Ming-yu Lu, Zhi Liu, and Hao Xu. "Association Rule Discovery with Fuzzy Decreasing Support on Syndrome Differentiation in Coronary Heart Disease." In 2009 2nd International Conference on Biomedical Engineering and Informatics. IEEE, 2009. http://dx.doi.org/10.1109/bmei.2009.5304789.

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Young, Jennifer L., Kyle Kretchmer, and Adam J. Engler. "Temporally-Stiffening Hydrogel Regulates Cardiac Differentiation via Mechanosensitive Signaling." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14674.

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Stiffness of the extracellular matrix (ECM) surrounding cells plays an integral role in affecting how a cell spreads, migrates, and differentiates, in the case of stem cells. For mature cardiomyocytes, stiffness regulates myofibril striation, beating rate, and fiber alignment, but does not induce de-differentiation [1,2]. Despite improved myocyte function on materials which mimic the ∼10 kPa heart stiffness, the heart does not begin as a contractile ∼10 kPa material, but instead undergoes ∼10-fold myocardial stiffening during development [3]. Thiolated hyaluronic acid (HA) hydrogels have been
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Yi, Weiguo, Jing Duan, and Mingyu Lu. "Association rule discovery with fuzzy decreasing support on Syndrome Differentiation and medication in coronary heart disease." In 2010 3rd International Conference on Biomedical Engineering and Informatics (BMEI). IEEE, 2010. http://dx.doi.org/10.1109/bmei.2010.5639351.

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Argento, G., M. Simonet, C. W. J. Oomens, and F. P. T. Baaijens. "Mechanics of Electrospun Scaffolds: An Application to Heart Valve Tissue Engineering." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80724.

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In the last decade electrospinning has shown its potential of being a feasible technique to manufacture scaffolds for tissue engineering [1]. Previous studies observed that, on a micrometer scale, the topology of the scaffold plays a fundamental role in the spreading and the differentiation of the cells [2], and in the growth of neo-extracellular matrix. On a tissue scale (in the order of cm) the stiffness of the construct enables the possibility of applying mechanical cues for the development of a functional engineered tissue [3]. Studies on scaffold mechanics based on volume-averaging theory
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Salinas, M., R. Lange, and S. Ramaswamy. "Specimen Dynamics and Subsequent Implications in Heart Valve Tissue Engineering Studies." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53346.

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In heart valve tissue engineering, appropriate mechanical preconditioning may provide the necessary stimuli to promote proper tissue formation [1–3]. Previous efforts have focused on a mechanistic heart valve (MHV) bioreactor that can mimic the innate mechanical stress states of flexure, flow and stretch in any combination thereof [1]. A fundamental component pertaining to heart valves is its dynamic behavior. Specific fluid-induced shears stress patterns may play a critical role in up-regulating ECM secretion by progenitor cell sources such as bone marrow derived stem cells [2] and increasing
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Varner, Victor D., and Larry A. Taber. "Not Just Inductive: A Critical Mechanical Role for the Endoderm During Heart Tube Assembly." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80621.

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The heart is the first functioning organ to form during development. Similar to other organ primordia, the embryonic heart forms as a simple tube — in this case, a straight muscle-wrapped tube situated on the ventral side of the embryo. During gastrulation, the cardiac progenitors reside in the lateral plate mesoderm but maintain close contact with the underlying endoderm. In amniotes, these bilateral heart fields are initially organized as a pair of flat epithelia that move toward the embryonic midline and fuse above the anterior intestinal portal (AIP) to form the heart tube. This medial mot
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Salinas, M., D. Schmidt, R. Lange, M. Libera, and S. Ramaswamy. "Computational Prediction of Fluid Induced Stress States in Dynamically Conditioned Engineered Heart Valve Tissues." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80787.

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There is extensive documented evidence that mechanical conditioning plays a significant role in the development of tissue grown in-vitro for heart valve scaffolds [1–3]. Modern custom made bioreactors have been used to study the mechanobiology of engineered heart valve tissues [1]. Specifically fluid-induced shears stress patterns may play a critical role in up-regulating extracellular matrix secretion by progenitor cell sources such as bone marrow derived stem cells (BMSCs) [2] and increasing the possibility of cell differentiation towards a heart valve phenotype. We hypothesize that specific
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Wan, Chen-rei, Seok Chung, Ryo Sudo, and Roger D. Kamm. "Induction of Cardiomyocyte Differentiation From Mouse Embryonic Stem Cells in a Confined Microfluidic Environment." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-203995.

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Embryonic stem cell derived cardiomyocytes are deemed an attractive treatment option for myocardial infarction. Their clinical efficacy, however, has not been unequivocally demonstrated. There is a need for better understanding and characterization of the cardiogenesis process. A microfluidic platform in vitro is used to dissect and better understand the differentiation process. Through this study, we find that while embryoid bodies (EBs) flatten out in a well plate system, differentiated EBs self-assemble into complex 3D structures. The beating regions of EBs are also different. Most beating
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Lahm, H., F. Wirth, M. Dreßen, et al. "Functional Analysis of Candidate Genes Associated with Congenital Heart Disease during Differentiation of Induced Pluripotent Stem Cells and in the Human Embryonic and Adult Heart at Single-Cell Resolution." In 50th Annual Meeting of the German Society for Thoracic and Cardiovascular Surgery (DGTHG). Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1725667.

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