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Journal articles on the topic 'Heart – Differentiation'

Author: Grafiati

Published: 4 June 2021

Last updated: 13 February 2022

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1

Hildick-Smith, D. J. R. "Echocardiographic differentiation of pathological and physiological left ventricular hypertrophy." Heart 85, no. 6 (2001): 615–19. http://dx.doi.org/10.1136/heart.85.6.615.

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2

Kempf, Tibor, and Kai C. Wollert. "Growth-Differentiation Factor-15 in Heart Failure." Heart Failure Clinics 5, no. 4 (2009): 537–47. http://dx.doi.org/10.1016/j.hfc.2009.04.006.

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3

Chen, J. N., and M. C. Fishman. "Zebrafish tinman homolog demarcates the heart field and initiates myocardial differentiation." Development 122, no. 12 (1996): 3809–16. http://dx.doi.org/10.1242/dev.122.12.3809.

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The fashioning of a vertebrate organ requires integration of decisions of cell fate by individual cells with those that regulate organotypic form. Logical candidates for this role, in an organ such as the heart, are genes that initiate the differentiation process leading to heart muscle and those that define the earliest embryonic heart field, but for neither class are genes defined. We cloned zebrafish Nkx2.5, a homolog of the tinman homeodomain gene needed for visceral and cardiac mesoderm formation in Drosophila. In the zebrafish, its expression is associated with cardiac precursor cells th

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Davis, L. A., and L. F. Lemanski. "Induction of myofibrillogenesis in cardiac lethal mutant axolotl hearts rescued by RNA derived from normal endoderm." Development 99, no. 2 (1987): 145–54. http://dx.doi.org/10.1242/dev.99.2.145.

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A strain of axolotl, Ambystoma mexicanum, that carries the cardiac lethal or c gene presents an excellent model system in which to study inductive interactions during heart development. Embryos homozygous for gene c contain hearts that fail to beat and do not form sarcomeric myofibrils even though muscle proteins are present. Although they can survive for approximately three weeks, mutant embryos inevitably die due to lack of circulation. Embryonic axolotl hearts can be maintained easily in organ culture using only Holtfreter's solution as a culture medium. Mutant hearts can be induced to diff

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5

Kazez, A., I. H. Özercan, and P. S. Erol. "Sacrococygeal heart: a very rare differentiation in teratoma." Journal of Pediatric Surgery 38, no. 6 (2003): 990. http://dx.doi.org/10.1016/s0022-3468(03)00142-8.

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6

Kirkpatrick, Michael A., and Andrew S. Groves. "Verbal Feedback Facilitates Heart Rate Discrimination and Differentiation." European Journal of Behavior Analysis 12, no. 2 (2011): 431–39. http://dx.doi.org/10.1080/15021149.2011.11434393.

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7

Kazez, A., İ. H. Özercan, F. S. Erol, M. Faik Özveren, and E. Parmaksız. "Sacrococcygeal Heart: A Very Rare Differentiation in Teratoma." European Journal of Pediatric Surgery 12, no. 4 (2002): 278–80. http://dx.doi.org/10.1055/s-2002-34483.

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8

THOMPSON, R. P., J. R. LINDROTH, A. J. ALLES, and A. R. FAZEL. "Cell Differentiation Birthdates in the Embryonic Rat Heart." Annals of the New York Academy of Sciences 588, no. 1 Embryonic Ori (1990): 446–48. http://dx.doi.org/10.1111/j.1749-6632.1990.tb13259.x.

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9

Chen, Wei-Qian, Chuanfu Li, Hai-Bin Ruan, Xuan Jiang, Xin Qi, and Xiang Gao. "Myeloid Differentiation Protein-88 Signaling Mediates Heart Failure." Journal of Cardiac Failure 13, no. 6 (2007): S79. http://dx.doi.org/10.1016/j.cardfail.2007.06.395.

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10

Drenckhahn, Jörg-Detlef. "Heart Development: Mitochondria in Command of Cardiomyocyte Differentiation." Developmental Cell 21, no. 3 (2011): 392–93. http://dx.doi.org/10.1016/j.devcel.2011.08.021.

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11

Rugendorff, Astrid, Amelia Younossi-Hartenstein, and Volker Hartenstein. "Embryonic origin and differentiation of the Drosophila heart." Roux's Archives of Developmental Biology 203, no. 5 (1994): 266–80. http://dx.doi.org/10.1007/bf00360522.

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12

Patanè, Salvatore. "Growth Differentiation Factor-15 in Chronic Heart Failure." JACC: Heart Failure 6, no. 2 (2018): 177. http://dx.doi.org/10.1016/j.jchf.2017.10.013.

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13

Voronina, L. P., O. S. Polunina, O. A. Bashkina, E. A. Polunina, and T. V. Prokofieva. "Phenotypic differentiation of patients with chronic heart failure." Medical alphabet, no. 36 (January 13, 2021): 28–33. http://dx.doi.org/10.33667/2078-5631-2020-36-28-33.

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Objective. According to the results of a complex analysis of gender-anamnestic, clinical, biochemical and instrumental parameters using the cluster analysis method to identify phenotypes of chronic heart failure (CHF) in the examined patients.Materials and methods. It was examined 345 patients with CHF with different left ventricular ejection fraction and 60 somatically healthy volunteers. For the study, groups of indicators were formed that most widely characterize the pathogenesis of CHF: gender-anamnestic and clinical, instrumental (echocardiographic study, study of the functional state of

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14

Chang, Yuqiao, Kang Guo, Qiong Li, Cixia Li, Zhikun Guo, and He Li. "Multiple Directional Differentiation Difference of Neonatal Rat Fibroblasts from Six Organs." Cellular Physiology and Biochemistry 39, no. 1 (2016): 157–71. http://dx.doi.org/10.1159/000445613.

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Background/Aims: Fibroblasts are abundantly distributed throughout connective tissues in the body and are very important in maintaining the structural and functional integrity. Recent reports have proved that fibroblasts and mesenchymal stem cells share much more in common than previously recognized. The aim of this study was to investigate comparative studies in fibroblasts on the differences in the expression of molecular markers and differentiation capacity from different organs. Methods: Combined trypsin/collagenase enzymes digestion method was used to isolate and culture the fibroblasts d

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15

Canale, E., J. J. Smolich, and G. R. Campbell. "Differentiation and innervation of the atrioventricular bundle and ventricular Purkinje system in sheep heart." Development 100, no. 4 (1987): 641–51. http://dx.doi.org/10.1242/dev.100.4.641.

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The development of the atrioventricular bundle (AVB) and ventricular Purkinje system and their innervation have been studied in fetal sheep from 27 to 140 days gestation (term is 147 days). The AVB initially consisted of a primordium, which lacked innervation and was composed of small, relatively undifferentiated myocytes. Differentiation of Purkinje-like cells within the AVB began near its distal end and extended towards the atrioventricular node (AVN). Differentiation of the ventricular Purkinje system extended distally from the region of bifurcation of the AVB from cells that were indisting

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16

Takeda, Minako, Yasuo Amano, Masaki Tachi, Hitomi Tani, Kyoichi Mizuno, and Shinichiro Kumita. "MRI differentiation of cardiomyopathy showing left ventricular hypertrophy and heart failure: differentiation between cardiac amyloidosis, hypertrophic cardiomyopathy, and hypertensive heart disease." Japanese Journal of Radiology 31, no. 10 (2013): 693–700. http://dx.doi.org/10.1007/s11604-013-0238-0.

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17

Liu, Jiabao, Peng Wu, Hao Wang, et al. "Necroptosis Induced by Ad-HGF Activates Endogenous C-Kit+ Cardiac Stem Cells and Promotes Cardiomyocyte Proliferation and Angiogenesis in the Infarcted Aged Heart." Cellular Physiology and Biochemistry 40, no. 5 (2016): 847–60. http://dx.doi.org/10.1159/000453144.

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Background/Aims: The discovery of c-kit+ cardiac stem cells (CSCs) provided us with new therapeutic targets to repair the damaged heart. However, the precise mechanisms regulating CSC proliferation and differentiation in the aged heart remained elusive. Necroptosis, a type of regulated cell death, has recently been shown to occur following myocardial infarction (MI); however, its effect on c-kit+ CSCs remains unknown. We investigated the effects of hepatocyte growth factor (HGF) and necroptosis on the proliferation and differentiation of endogenous c-kit+ CSCs in aged rat hearts following MI.

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18

Kaarbø, Mari, Denis I. Crane, and Wayne G. Murrell. "RhoA Regulation of Cardiomyocyte Differentiation." Scientific World Journal 2013 (2013): 1–12. http://dx.doi.org/10.1155/2013/491546.

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Earlier findings from our laboratory implicated RhoA in heart developmental processes. To investigate factors that potentially regulate RhoA expression, RhoA gene organisation and promoter activity were analysed. Comparative analysis indicated strict conservation of both gene organisation and coding sequence of the chick, mouse, and human RhoA genes. Bioinformatics analysis of the derived promoter region of mouse RhoA identified putative consensus sequence binding sites for several transcription factors involved in heart formation and organogenesis generally. Using luciferase reporter assays,

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19

Fang, Yi-Hsien, Saprina P. H. Wang, Zi-Han Gao, et al. "Efficient Cardiac Differentiation of Human Amniotic Fluid-Derived Stem Cells into Induced Pluripotent Stem Cells and Their Potential Immune Privilege." International Journal of Molecular Sciences 21, no. 7 (2020): 2359. http://dx.doi.org/10.3390/ijms21072359.

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Mature mammalian hearts possess very limited regenerative potential. The irreversible cardiomyocyte loss after heart injury can lead to heart failure and death. Pluripotent stem cells (PSCs) can differentiate into cardiomyocytes for cardiac repair, but there are obstacles to their clinical application. Among these obstacles is their potential for post-transplant rejection. Although human amniotic fluid-derived stem cells (hAFSCs) are immune privileged, they cannot induce cardiac differentiation. Thus, we generated hAFSC-derived induced PSCs (hAFSC-iPSCs) and used a Wnt-modulating differentiati

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20

Wei, Y., D. Bader, and J. Litvin. "Identification of a novel cardiac-specific transcript critical for cardiac myocyte differentiation." Development 122, no. 9 (1996): 2779–89. http://dx.doi.org/10.1242/dev.122.9.2779.

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A novel cDNA, pCMF1, which is expressed exclusively and transiently in the myogenic cells of the differentiating chicken heart was isolated and characterized. The full-length cDNA of pCMF1 has one open reading frame encoding 1538 predicted amino acids. While computer analysis predicts the presence of specific structural motifs, the overall sequence of pCMF1 is unique. The pattern of pCMF1 gene expression during heart formation was determined by whole-mount in situ hybridization. pCMF1 is transiently expressed within the myogenic cells of the primitive heart tube from stages 9 to 18 and is not

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21

Carver, W., R. L. Price, D. S. Raso, L. Terracio, and T. K. Borg. "Distribution of beta-1 integrin in the developing rat heart." Journal of Histochemistry & Cytochemistry 42, no. 2 (1994): 167–75. http://dx.doi.org/10.1177/42.2.8288862.

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Cell-cell and cell-matrix interactions play critical roles in various developmental processes including differentiation, proliferation, and migration. Members of the integrin family of cell surface components are important mediators of these cell-extracellular matrix (ECM) contacts or interactions. The ECM provides signals to individual cells essential for development and differentiation and plays essential roles in establishing and maintaining the complex structure of the vertebrate heart. Integrins provide a fundamental link for transduction of developmental signals to cells. Integrin expres

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22

Foster, Paul S., Daniel G. Webster, and Edward W. L. Smith. "The Psychophysiological Differentiation of Emotional Memories." Imagination, Cognition and Personality 17, no. 2 (1997): 111–22. http://dx.doi.org/10.2190/qu7n-hqyw-86xf-wx56.

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Participants' heart rate and skin resistance responses to emotional memories (fear, anger, joy, sadness, and embarrassment) were studied to determine if the recollection of emotion is sufficient to produce psychophysiological changes, to determine if such changes differ for the various emotions, and to determine the relationship between imaginal abilities and psychophysiological responses to emotional memories. The Absorption Scale of the Multidimensional Personality Questionnaire was used as the measure of imaginal ability [1]. A repeated measures analysis of variance indicated significant di

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23

Jonker, Sonnet S., Lubo Zhang, Samantha Louey, George D. Giraud, Kent L. Thornburg, and J. Job Faber. "Myocyte enlargement, differentiation, and proliferation kinetics in the fetal sheep heart." Journal of Applied Physiology 102, no. 3 (2007): 1130–42. http://dx.doi.org/10.1152/japplphysiol.00937.2006.

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The generation of new myocytes is an essential process of in utero heart growth. Most, or all, cardiac myocytes lose their capacity for proliferation during the perinatal period through the process of terminal differentiation. An increasing number of studies focus on how experimental interventions affect cardiac myocyte growth in the fetal sheep. Nevertheless, fundamental questions about normal growth of the fetal heart remain unanswered. In this study, we determined that during the last third of gestation the hearts of fetal sheep grew primarily by four processes. 1) Myocyte proliferation con

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24

Chen, Li, Filomena Gabriella Fulcoli, Susan Tang, and Antonio Baldini. "Tbx1 Regulates Proliferation and Differentiation of Multipotent Heart Progenitors." Circulation Research 105, no. 9 (2009): 842–51. http://dx.doi.org/10.1161/circresaha.109.200295.

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25

Maisch, Bernhard, and Jörg Lauschke. "Reply to: Pitfalls in the differentiation between athlete’s heart…" Clinical Research in Cardiology 98, no. 7 (2009): 467–68. http://dx.doi.org/10.1007/s00392-009-0036-y.

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26

Wollert, Kai C., and Tibor Kempf. "Growth Differentiation Factor 15 in Heart Failure: An Update." Current Heart Failure Reports 9, no. 4 (2012): 337–45. http://dx.doi.org/10.1007/s11897-012-0113-9.

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27

Chang, Wei-Ting, Jhih-Yuan Shih, Yu-Wen Lin, Zhih-Cherng Chen, Jun-Neng Roan, and Chih-Hsin Hsu. "Growth differentiation factor-15 levels in the blood around the pulmonary artery is associated with hospitalization for heart failure in patients with pulmonary arterial hypertension." Pulmonary Circulation 10, no. 4 (2020): 204589402096294. http://dx.doi.org/10.1177/2045894020962948.

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Despite no significant differences of growth differentiation factor-15 expressions in peripheral, right atrial, and right ventricular blood, in the pulmonary arterial blood, there was a significantly high level of growth differentiation factor-15 in Group I pulmonary arterial hypertension patients subsequently developing heart failure. During right heart catheterization, collecting pulmonary blood samples is suggested to measure growth differentiation factor-15.

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28

Gutkowska, Jolanta, Malgorzata Miszkurka, Bogdan Danalache, Natig Gassanov, Donghao Wang, and Marek Jankowski. "Functional arginine vasopressin system in early heart maturation." American Journal of Physiology-Heart and Circulatory Physiology 293, no. 4 (2007): H2262—H2270. http://dx.doi.org/10.1152/ajpheart.01320.2006.

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Since the neurohypophyseal hormone 8-arginine vasopressin (AVP) is involved in cardiovascular tissue hypertrophy and myocyte differentiation, it is possible that local AVP plays a role in heart maturation. AVP-specific RIA, RT-PCR, and immunoblot measurement of AVP receptors (VR) were used to investigate heart tissues from newborn and adult rats. To test AVP's role in differentiation and specialization into ventricle-like cardiomyocytes, we studied GFP-P19Cl6 stem cells, which express green fluorescence protein (GFP) reporter under transcriptional control of the myosin light chain-2v promoter.

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29

Dick, H., R. G. E. Murray, and S. Walmsley. "Swarmer cell differentiation of Proteus mirabilis in fluid media." Canadian Journal of Microbiology 31, no. 11 (1985): 1041–50. http://dx.doi.org/10.1139/m85-196.

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After 3–4 h in a rich fluid medium such as brain–heart infusion broth, motile nonseptate filaments developed from normal short rods and formed about 80% of the cell mass of Proteus mirabilis PM23. This developmental pattern was not observed in any of the other nine representatives of the species. These filaments were considered to be equivalent to swarmer cells formed on agar media because these cells ceased tumbling (i.e., chemotaxis was repressed), they developed large numbers of flagella (i.e., flagella synthesis and insertion was derepressed), and the distribution of nuclei in the filament

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Kirby, Margaret L., and Karen L. Waldo. "Molecular Embryogenesis of the Heart." Pediatric and Developmental Pathology 5, no. 6 (2002): 516–43. http://dx.doi.org/10.1007/s10024-002-0004-2.

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Development of the heart is a complex process involving primary and secondary heart fields that are set aside to generate myocardial and endocardial cell lineages. The molecular inductions that occur in the primary heart field appear to be recapitulated in induction and myocardial differentiation of the secondary heart field, which adds the conotruncal segments to the primary heart tube. While much is now known about the initial steps and factors involved in induction of myocardial differentiation, little is known about induction of endocardial development. Many of the genes expressed by nasce

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Yong, Hue Mun Au, Erica Minato, Eldho Paul, and Udaya Seneviratne. "006 Can seizure-related heart rate differentiate epileptic seizures from psychogenic non-epileptic seizures?" Journal of Neurology, Neurosurgery & Psychiatry 90, e7 (2019): A2.3—A3. http://dx.doi.org/10.1136/jnnp-2019-anzan.6.

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IntroductionThis study aims to (i)evaluate the diagnostic sensitivity, specificity and predictive values of seizure-related heart rate (HR) in differentiating epileptic seizures(ES) from psychogenic non-epileptic seizures(PNES), (ii)define the most useful point of HR measurement: pre-ictal, ictal-onset, maximal-ictal or post-ictal, and (iii)define the HR cut-off points to differentiate ES from PNES.MethodsAll video EEG(VEEG) at Monash Health from May 2009 to November 2015 were retrospectively reviewed. Baseline(during wakefulness), one-minute pre-ictal, ictal-onset, maximal-ictal and one-minut

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Bondue, Antoine, Simon Tännler, Giuseppe Chiapparo, et al. "Defining the earliest step of cardiovascular progenitor specification during embryonic stem cell differentiation." Journal of Cell Biology 192, no. 5 (2011): 751–65. http://dx.doi.org/10.1083/jcb.201007063.

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During embryonic development and embryonic stem cell (ESC) differentiation, the different cell lineages of the mature heart arise from two types of multipotent cardiovascular progenitors (MCPs), the first and second heart fields. A key question is whether these two MCP populations arise from differentiation of a common progenitor. In this paper, we engineered Mesp1–green fluorescent protein (GFP) ESCs to isolate early MCPs during ESC differentiation. Mesp1-GFP cells are strongly enriched for MCPs, presenting the ability to differentiate into multiple cardiovascular lineages from both heart fie

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Behfar, Atta, Carmen Perez-Terzic, Randolph S. Faustino, et al. "Cardiopoietic programming of embryonic stem cells for tumor-free heart repair." Journal of Experimental Medicine 204, no. 2 (2007): 405–20. http://dx.doi.org/10.1084/jem.20061916.

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Embryonic stem cells have the distinct potential for tissue regeneration, including cardiac repair. Their propensity for multilineage differentiation carries, however, the liability of neoplastic growth, impeding therapeutic application. Here, the tumorigenic threat associated with embryonic stem cell transplantation was suppressed by cardiac-restricted transgenic expression of the reprogramming cytokine TNF-α, enhancing the cardiogenic competence of recipient heart. The in vivo aptitude of TNF-α to promote cardiac differentiation was recapitulated in embryoid bodies in vitro. The procardiogen

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34

Jackson, T., M. F. Allard, C. M. Sreenan, L. K. Doss, S. P. Bishop, and J. L. Swain. "The c-myc proto-oncogene regulates cardiac development in transgenic mice." Molecular and Cellular Biology 10, no. 7 (1990): 3709–16. http://dx.doi.org/10.1128/mcb.10.7.3709.

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During the maturation of the cardiac myocyte, a transition occurs from hyperplastic to hypertrophic growth. The factors that control this transition in the developing heart are unknown. Proto-oncogenes such as c-myc have been implicated in the regulation of cellular proliferation and differentiation, and in the heart the switch from myocyte proliferation to terminal differentiation is synchronous with a decrease in c-myc mRNA abundance. To determine whether c-myc can influence myocyte proliferation or differentiation, we examined the in vivo effect of increasing c-myc expression during embryog

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Jackson, T., M. F. Allard, C. M. Sreenan, L. K. Doss, S. P. Bishop, and J. L. Swain. "The c-myc proto-oncogene regulates cardiac development in transgenic mice." Molecular and Cellular Biology 10, no. 7 (1990): 3709–16. http://dx.doi.org/10.1128/mcb.10.7.3709-3716.1990.

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During the maturation of the cardiac myocyte, a transition occurs from hyperplastic to hypertrophic growth. The factors that control this transition in the developing heart are unknown. Proto-oncogenes such as c-myc have been implicated in the regulation of cellular proliferation and differentiation, and in the heart the switch from myocyte proliferation to terminal differentiation is synchronous with a decrease in c-myc mRNA abundance. To determine whether c-myc can influence myocyte proliferation or differentiation, we examined the in vivo effect of increasing c-myc expression during embryog

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36

Wu, Mingfu, and Jingjing Li. "Numb family proteins: novel players in cardiac morphogenesis and cardiac progenitor cell differentiation." Biomolecular Concepts 6, no. 2 (2015): 137–48. http://dx.doi.org/10.1515/bmc-2015-0003.

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AbstractVertebrate heart formation is a spatiotemporally regulated morphogenic process that initiates with bilaterally symmetric cardiac primordial cells migrating toward the midline to form a linear heart tube. The heart tube then elongates and undergoes a series of looping morphogenesis, followed by expansions of regions that are destined to become primitive heart chambers. During the cardiac morphogenesis, cells derived from the first heart field contribute to the primary heart tube, and cells from the secondary heart field, cardiac neural crest, and pro-epicardial organ are added to the he

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37

Hamid, Tariq, Yuanyuan Xu, Mohamed Ameen Ismahil, et al. "TNF receptor signaling inhibits cardiomyogenic differentiation of cardiac stem cells and promotes a neuroadrenergic-like fate." American Journal of Physiology-Heart and Circulatory Physiology 311, no. 5 (2016): H1189—H1201. http://dx.doi.org/10.1152/ajpheart.00904.2015.

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Despite expansion of resident cardiac stem cells (CSCs; c-kit+Lin−) after myocardial infarction, endogenous repair processes are insufficient to prevent adverse cardiac remodeling and heart failure (HF). This suggests that the microenvironment in post-ischemic and failing hearts compromises CSC regenerative potential. Inflammatory cytokines, such as tumor necrosis factor-α (TNF), are increased after infarction and in HF; whether they modulate CSC function is unknown. As the effects of TNF are specific to its two receptors (TNFRs), we tested the hypothesis that TNF differentially modulates CSC

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38

Bogdanov, L. A., and A. G. Kutikhin. "Optimization of hematoxylin and eosin staining of heart, blood vessels, liver, and spleen." Fundamental and Clinical Medicine 4, no. 4 (2019): 70–77. http://dx.doi.org/10.23946/2500-0764-2019-4-4-70-77.

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Aim. To optimize hematoxylin and eosin staining protocol for heart, blood vessels, liver, and spleen.Methods. Heart (ventricles), abdominal aorta, liver (right lobe), and spleen (left part) of the Wistar rats were excised, fixed in 10% neutral phosphate buffered formalin for 24 h, washed in tap water for 2 h, dehydrated in ascending ethanol series (70%, 80%, and 95%) and isopropanol, embedded into paraffin and then sectioned (5 μm) using rotary microtome. For regressive staining, incubation time in Harris hematoxylin was 5 or 10 minutes, time of exposure to differentiation alcoholicaqueous eos

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Urhausen, Axel, and Wilfried Kindermann. "Sports-Specific Adaptations and Differentiation of the Athlete??s Heart." Sports Medicine 28, no. 4 (1999): 237–44. http://dx.doi.org/10.2165/00007256-199928040-00002.

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40

Lim, Jeong A., Hye Jung Baek, Moon Sun Jang та ін. "Loss of β2-spectrin prevents cardiomyocyte differentiation and heart development". Cardiovascular Research 101, № 1 (2013): 39–47. http://dx.doi.org/10.1093/cvr/cvt222.

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41

Calderon, Damelys, Evan Bardot, and Nicole Dubois. "Probing early heart development to instruct stem cell differentiation strategies." Developmental Dynamics 245, no. 12 (2016): 1130–44. http://dx.doi.org/10.1002/dvdy.24441.

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42

Sapoznikov, Dan, Myron H. Luria, and Mervyn S. Gotsman. "Differentiation of Periodic from Nonperiodic Low-Frequency Heart Rate Fluctuations." Computers and Biomedical Research 27, no. 3 (1994): 199–209. http://dx.doi.org/10.1006/cbmr.1994.1017.

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Anand, Inder S., Tibor Kempf, Thomas S. Rector, et al. "Serial Measurement of Growth-Differentiation Factor-15 in Heart Failure." Circulation 122, no. 14 (2010): 1387–95. http://dx.doi.org/10.1161/circulationaha.109.928846.

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44

Risebro, C. A., N. Smart, L. Dupays, R. Breckenridge, T. J. Mohun, and P. R. Riley. "Hand1 regulates cardiomyocyte proliferation versus differentiation in the developing heart." Development 133, no. 22 (2006): 4595–606. http://dx.doi.org/10.1242/dev.02625.

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45

Millar, Lynne Martina, Zephryn Fanton, Gherardo Finocchiaro, et al. "Differentiation between athlete’s heart and dilated cardiomyopathy in athletic individuals." Heart 106, no. 14 (2020): 1059–65. http://dx.doi.org/10.1136/heartjnl-2019-316147.

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ObjectiveDistinguishing early dilated cardiomyopathy (DCM) from physiological left ventricular (LV) dilatation with LV ejection fraction <55% in athletes (grey zone) is challenging. We evaluated the role of a cascade of investigations to differentiate these two entities.MethodsThirty-five asymptomatic active males with DCM, 25 male athletes in the ‘grey zone’ and 24 male athletes with normal LV ejection fraction underwent N-terminal pro-brain natriuretic peptide (NT-proBNP) measurement, ECG and exercise echocardiography. Grey-zone athletes and patients with DCM underwent cardiovascular magn

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Behfar, Atta, Leonid V. Zingman, Denice M. Hodgson, et al. "Stem cell differentiation requires a paracrine pathway in the heart." FASEB Journal 16, no. 12 (2002): 1558–66. http://dx.doi.org/10.1096/fj.02-0072com.

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47

Breckwoldt, Kaja, David Letuffe-Brenière, Ingra Mannhardt, et al. "Differentiation of cardiomyocytes and generation of human engineered heart tissue." Nature Protocols 12, no. 6 (2017): 1177–97. http://dx.doi.org/10.1038/nprot.2017.033.

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Scharhag, Jürgen, and Wilfried Kindermann. "Pitfalls in the differentiation between athlete’s heart and hypertrophic cardiomyopathy." Clinical Research in Cardiology 98, no. 7 (2009): 465–66. http://dx.doi.org/10.1007/s00392-009-0035-z.

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Lourenço, Patrícia, Filipe M. Cunha, João Ferreira‐Coimbra, Isaac Barroso, João‐Tiago Guimarães, and Paulo Bettencourt. "Dynamics of growth differentiation factor 15 in acute heart failure." ESC Heart Failure 8, no. 4 (2021): 2527–34. http://dx.doi.org/10.1002/ehf2.13377.

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Rajala, Kristiina, Mari Pekkanen-Mattila, and Katriina Aalto-Setälä. "Cardiac Differentiation of Pluripotent Stem Cells." Stem Cells International 2011 (2011): 1–12. http://dx.doi.org/10.4061/2011/383709.

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Abstract:

The ability of human pluripotent stem cells to differentiate towards the cardiac lineage has attracted significant interest, initially with a strong focus on regenerative medicine. The ultimate goal to repair the heart by cardiomyocyte replacement has, however, proven challenging. Human cardiac differentiation has been difficult to control, but methods are improving, and the process, to a certain extent, can be manipulated and directed. The stem cell-derived cardiomyocytes described to date exhibit rather immature functional and structural characteristics compared to adult cardiomyocytes. Thus

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