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Books on the topic 'Neurological Symptom'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

L, Henry Gregory, and Henry Gregory L, eds. Neurologic emergencies: A symptom-oriented approach. 2nd ed. McGraw-Hill, 2003.

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2

Neal, Little, ed. Neurologic emergencies: A symptom oriented approach. McGraw-Hill, 1985.

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3

1931-, Walsh Thomas J., ed. Neuro-ophthalmology: Clinical signs and symptoms. 4th ed. Williams & Wilkins, 1997.

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4

Fenichel, Gerald M. Clinical pediatric neurology: A signs and symptoms approach. 3rd ed. Saunders, 1997.

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5

Roberts, Margaret Ann. The assessment of intercranial neurological symptoms in the elderly using computerised tomography. University of Birmingham, 1987.

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6

Huk, W. J. Magnetic resonance imaging of central nervous system diseases: Functional anatomy, imaging, neurological symptoms, pathology. Springer-Verlag, 1990.

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7

Brust, John C. M. The practice of neural science: From synapses to symptoms. Appleton & Lange, 1999.

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8

Waldmann, Carl, Neil Soni, and Andrew Rhodes. Neurological disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199229581.003.0022.

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Agitation and confusion 360Status epilepticus 362Meningitis 364Intracerebral haemorrhage 366Subarachnoid haemorrhage 368Ischaemic stroke 370Guillain–Barre syndrome 372Myasthenia gravis 374ICU neuromuscular disorders 376Tetanus 378Botulism 380Neurorehabilitation 382Hyperthermias 384Agitation and confusion are common features in critical illness. Agitation is a symptom or sign of numerous acute and chronic disease states that include pain, anxiety and delirium. Agitation is present in around half of ICU patients, with 15% experiencing severe agitation. Confusion may also be chronic or acute and

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9

Wong, Victoria S. S., and Martin Salinsky. Neurological and Medical Factors. Edited by Barbara A. Dworetzky and Gaston C. Baslet. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190265045.003.0004.

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This chapter addresses the neurological and medical factors associated with psychogenic nonepileptic seizures (PNES). PNES can occur concurrently with epilepsy in 5 to 20% of patients. Traumatic brain injury (TBI) is a major cause of epilepsy, but it is also commonly cited by patients with PNES as the primary cause of their seizures. PNES are also overrepresented in patients with intellectual and learning disabilities. Patients with PNES usually have additional subjective neurological and medical complaints. Pain complaints are overrepresented in patients with PNES and are a major contributor

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10

Harding, Kelli Jane K., and Brian A. Fallon. Somatic Symptom and Related Disorders. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199326075.003.0010.

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This chapter discusses the somatic symptom disorders, which are a heterogeneous group unified by physical symptoms or concerns that are associated with prominent distress or impairment. Somatic symptom disorders are estimated to account for 1 in 10 primary care patient visits. The relative prominence of somatic symptoms is essential to the difference between illness anxiety disorder, which is an example of the obsessional/cognitive subtype (not prominent) and somatic symptom disorder,, in which the somatic symptoms are prominent. Patients with body dysmorphic disorder, also an Obsessional/Cogn

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11

Hawkes, Christopher H., Bettina Balint, and José Biller. Instant Neurological Diagnosis. 3rd ed. Oxford University PressNew York, 2025. https://doi.org/10.1093/med/9780197699461.001.0001.

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Abstract When listening to a patient’s history or performing a neurological examination, certain clues pop out such as the Lhermitte sign (or symptom) that would immediately suggest one lead diagnosis—namely multiple sclerosis. We call these Handles. The opposite are Red Flags where the diagnostic process should come to a halt and merit review. The enviable skill to make a rapid diagnosis in neurological disorders comes with experience, which according to many can only be learnt. We disagree. Our book reveals many of the short-cuts used consciously or otherwise by experienced clinicians to get

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12

Voltz, Raymond, Stefan Lorenzl, and Georg Nübling. Neurological disorders other than dementia. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0155.

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The significance of a palliative care approach for patients with neurological disorders other than dementia is increasingly recognized. Whereas the care for these patients, for example, those with amyotrophic lateral sclerosis (ALS) or motor neuron disease, had been incorporated in the initial concept by Dame Cicely Saunders, recent scientific evidence supports this position. The need for palliative care in these patients is underpinned by their frequent wish for hastening death. This chapter describes palliative care approaches for a number of neurological disorders other than dementia. For A

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13

Henry, Gregory L., Andy Jagoda, Neal Little, and Thomas R. Pellegrino. Neurologic Emergencies: A Symptom-Oriented Approach, 2/e. 2nd ed. McGraw-Hill Professional, 2003.

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14

Moen, Vibeke. Neurological complications of neuraxial blockade. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0028.

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Neuraxial techniques for obstetric analgesia and anaesthesia are widespread, and serious complications are extremely rare. The most common of all complications following neuraxial blockade is postdural puncture headache, but headache may also be present in pathological conditions such as pre-eclampsia and sinus vein thrombosis. Headache may also be a symptom of cranial subdural haematoma, meningitis, and epidural abscess, all rare complications of central blockade, thus introducing a potential confounder in the newly delivered woman complaining of headache. Vertebral spinal haematomas are extr

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15

Bäumer, Dirk. Diagnosis in suspected neurological disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0220.

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It is not always clear that a symptom is due to a problem in the nervous system. Delirium, generalized weakness, and fatigue are frequently due to systemic illness. Transient loss of consciousness may have a neurological or cardiac cause. It is, therefore, important to keep an open mind before focusing on a primary neurological diagnosis in these presentations. The principles of the neurological method are described in this chapter, followed by its application in common clinical presentations.

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16

Watson, Max, Caroline Lucas, Andrew Hoy, and Jo Wells. Palliative care in non-malignant neurological disease. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199234356.003.0029.

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This chapter focuses on the symptom management of multiple sclerosis, Parkinson’s disease, multiple system atrophy (MSA), progressive supranuclear palsy (PSP), motor neuron disease, neurological complications of AIDS, Creutzfeldt-Jakob disease (CJD), and useful contacts.

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17

Chong, Sam, and J. Ganesalingham. Acute pain in the neurological patient. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199234721.003.0016.

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Acute pain is a common presenting symptom in patients with neurological conditions. Acute onset headache may indicate a life-threatening underlying condition. Lumbosacral and cervical spine pain are commonly caused by degenerative disease but there are sometimes clues to indicate alternative pathologies. Acute pain arising from the peripheral nervous system and muscles are usually inflammatory in origin. A careful history and examination is crucial to assess patients with neurological pain. Opioids may be used in combination of an anti-epileptic or antidepressant drug in the treatment of acute

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18

Hain, Richard D. W., and Satbir Singh Jassal. Neurological symptoms. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198745457.003.0012.

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This chapter covers a range of neurological symptoms commonly encountered in paediatric palliative care. This includes epilepsy, seizures, spasticity, myoclonus, chorea, dystonia, and akathisia. For most of these conditions, both definitions and details of management are provided.

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19

Kipps, Christopher, and John Hodges. Clinical cognitive assessment. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0010.

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Cognitive symptoms arise from the location of brain dysfunction and are not linked directly to any particular pathology. In the early stages of disease, symptoms may be non-specific, and while certain symptom clusters are commonly seen in particular disorders, atypical presentations are not infrequent. For example, in Alzheimer’s disease, patients may present with a focal language syndrome instead of the more commonly appreciated autobiographical memory disturbance despite identical pathology. In our approach to the cognitive assessment, we maintain a symptom oriented approach. This facilitate

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20

Crum, Brian A., Eduardo E. Benarroch, and Robert D. Brown. Neurologic Disorders Categorized by Anatomical Involvement. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0523.

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Neurological disorders of the brain, spine, and peripheral nervous system are examined. Symptoms and signs related to disorders of the cerebral cortex may lead to alterations in cognition and consciousness. Unilateral neurologic symptoms involving a single neurologic symptom commonly localize to the cerebral cortex. Abnormalities of speech and language are localized to the dominant cerebral hemisphere, whereas abnormalities of the nondominant hemisphere may lead to visuospatial deficits, confusion, or neglect of the contralateral side of the body. The hypothalamus is important in many function

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21

Carson, Alan, and Jon Stone. Functional neurological symptoms. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199658602.003.0004.

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Functional neurological symptoms are a common cause of disability and distress in any neurological service. Varying terminology, including hysteria, conversion disorder, dissociative, non-organic, and psychogenic, reflects the shifting nature of medical thinking, over time, regarding these presentations. They also emphasize the importance of a historical understanding when grappling with this area. This chapter traces this history, from Briquet’s monumental treatise on hysteria in 1859 to a randomized controlled trial of non-epileptic seizures in 2010. In the process, familiar figures such as

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22

Donaghy, Michael. The clinical approach. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0030.

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This chapter describes the appropriate clinical approach to take when presented with a patient reporting a neurological symptom. Just under 10 per cent of the population consult their general practitioner about a neurological symptom each year in the United Kingdom. About 10 per cent of these are referred for a specialist opinion, usually to a neurologist. Nine conditions account for roughly 75 per cent of general neurological referrals and are diagnosed initially on purely clinical grounds, with the other 25 per cent representing the full range of other, potentially very rare, neurological di

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23

Weeber, Edwin J. Angelman Syndrome. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199744312.003.0013.

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Angelman syndrome (AS) is a devastating neurological disorder with a symptom complex that includes but is not limited to severe developmental delay, profound cognitive disruption, motor coordination defects, increased propensity for seizure with a characteristic abnormal electroencephalogram, sleep disruption, behavioral difficulties, a lack of speech, and an overall happy demeanor. Although the disorder was first described in 1965 by British pediatrician Dr. Harry Angelman, because AS is clinically characterized by a wide constellation of symptoms with varying degrees of severity, it is not r

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24

Sedel, Frédéric, and Yann Nadjar. Neurological and Psychiatric Symptoms. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0067.

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Late-onset forms of inborn errors of metabolism (IEM) first presenting in adulthood often display psychiatric or neurological manifestations, including atypical psychosis or depression, unexplained coma, peripheral neuropathy, cerebellar ataxia, spastic paraparesis, dementia, movement disorders, or epilepsy. With the exception of several review articles, most if not all existing books and diagnostic algorithms refer to pediatric forms of these diseases. Late-onset forms of IEM always display attenuated phenotypes, which in some instances are associated with one or more clinical manifestations

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25

Drake, Marcus. Assessment of urinary incontinence. Edited by Christopher R. Chapple. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0037.

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Involuntary loss of urine is subdivided primarily into stress, urgency, or mixed urinary incontinence. The history and examination aim to identify underlying mechanisms, and indicators of more complex causes, or serious medical conditions. Associated lower urinary tract symptoms (LUTS) should be catalogued in detail. History should also cover symptom bother, as this is the prime driver of therapy. Validated questionnaires are the most effective way to capture aspects of incontinence and associated LUTS. Wider pelvic symptoms, such as pelvic organ prolapse, sexual function, and anal symptoms sh

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26

Donaghy, Michael, and Martin Rossor. Psychologically determined disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0153.

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Neurologists frequently see patients with symptoms or inconsistent signs that are not explicable in terms of any recognized neurological disease process. Often it is clear that such symptoms and signs are being manufactured psychologically, either consciously or, more often, by an unconscious process. Such patients are frequently polysymptomatic, and may have a long history of consulting other specialists, particularly abdominal, dental, gynaecological, and otorhinological surgeons. They run the risk of developing secondary abnormalities induced by surgical and other invasive procedures. Psych

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27

Jordan, Nerissa. Neurocutaneous syndromes. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0224.

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The neurocutaneous syndromes comprise a diverse group of rare genetic disorders with both neurological and cutaneous manifestations. Each syndrome has a distinct phenotype. Symptoms are variable and depend on the syndrome. Neurocutaneous syndromes often present in childhood or adolescence; for example, tuberous sclerosis typically presents in early childhood. The age range of presentation is broad, depending on the specific condition and severity of expression. The majority are autosomally inherited conditions. De novo mutations can occur. Most neurocutaneous syndromes do not have a specific t

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28

Keshav, Satish, and Alexandra Kent. Difficulty swallowing. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0021.

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Dysphagia is a difficulty in the process of swallowing. Dysphagia is an alarm symptom, and therefore requires referral to secondary care for investigations. There are multiple causes, divided into oesophageal, neurological, surgical, and extrinsic obstruction. This chapter covers the approach to diagnosis, key diagnostic tests, therapies, prognosis, and dealing with uncertainty.

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29

WILLIAM, Emma. Neurological Disorders: Neurological Conditions and Symptoms to Watch For. Independently Published, 2022.

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30

Zahn, Roland, and Alistair Burns. Dementia disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198779803.003.0001.

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This chapter provides a brief overview of the different forms of dementia syndromes and provides a simple algorithm for initial differential diagnosis. Rapidly progressive dementias have to be excluded which require specific investigations to detect Creutzfeldt–Jakob as well as inflammatory and autoimmune diseases. A lead symptom-based approach in patients with slowly progressive cognitive and behavioural impairments without neurological symptoms is applied: progressive and primary impairments in recent memory are characteristic of typical Alzheimer’s dementia, primary behavioural changes poin

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31

Krengel, Maxine, and Roberta F. White. Neurological Disorders and Symptoms Associated with Psychological/Behavioral Problems. Edited by Phillip M. Kleespies. Oxford University Press, 2015. http://dx.doi.org/10.1093/oxfordhb/9780199352722.013.31.

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Several neurological syndromes and neurocognitive disorders can result in behavioral and affective symptoms that may be present in an emergent situation or are present in the medical context. Clinicians who see patients with new or unusual behavioral symptoms are often faced with the challenge of determining when patients are in need of follow-up diagnostic evaluation. The purpose of this chapter is to discuss the expression of behavioral changes in neurological disease that we have encountered in our own extensive clinical experience. We do not provide an exhaustive review of all disorders or

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32

Török, M. Estée, Fiona J. Cooke, and Ed Moran. Neurological infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199671328.003.0019.

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This chapter covers both acute bacterial and viral, and chronic, meningitis, as well as tuberculous, cryptococcal, coccidioidal, and Histoplasma meningitis, describing meningeal symptoms (headache, neck stiffness, vomiting, photophobia) and cerebral dysfunction (confusion, coma). The chapter also covers neurocysticercosis (including parenchymal and extra-parenchymal cysts), encephalitis (an inflammatory process in the brain characterized by cerebral dysfunction), as well as brain abscess, cerebritis, subdural empyema, epidural abscess, and cerebrospinal fluid shunt infections.

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33

Lipman, Jeffrey, and Robert J. Boots. Diagnosis, assessment, and management of tetanus, rabies, and botulism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0245.

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Tetanus, rabies and botulism are all infections characterized by the production of a neurotoxin, and generally do not give rise to a systemic inflammatory response. Typically tetanus result from the infection of wounds by the ubiquitious soil-borne bacteria Clostridium tetanii, botulism is most commonly due to toxin produced in food contaminated with Clostridium botulinum. Rabies usually results from an animal bite infected with the rabies virus of the Lyssavirus group. Neurological involvement by all three infections is characterized by paralysis and autonomic instability with tetanus also be

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34

Williams, Christopher, Alan Carson, Sharon Smith, Michael Sharpe, Jonathan Cavanagh, and Catriona Kent. Overcoming Functional Neurological Symptoms: A Five Areas Approach. CRC Press, 2017. http://dx.doi.org/10.1201/b13567.

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35

Carson, Williams. Overcoming Functional Neurological Symptoms: A Five Areas Approach. Taylor & Francis Group, 2011.

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36

Overcoming Functional Neurological Symptoms: A Five Areas Approach. Taylor & Francis Group, 2017.

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37

Carson, Alan, Jonathan Cavanagh, Sharon Smith, Michael Sharpe, and Christopher Williams. Overcoming Functional Neurological Symptoms: A Five Areas Approach. Taylor & Francis Group, 2017.

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38

Carson, Alan, Jonathan Cavanagh, Sharon Smith, Michael Sharpe, and Christopher Williams. Overcoming Functional Neurological Symptoms: A Five Areas Approach. Taylor & Francis Group, 2017.

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39

Overcoming Functional Neurological Symptoms A Five Areas Approach. Hodder Arnold Publishers, 2011.

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40

Bleecker, Margit L. Neurological Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190662677.003.0026.

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This chapter describes neurologic disorders related primarily to occupational exposures along with presenting signs and symptoms. Acute or subacute occupational exposure to high levels of neurotoxic compounds, which occurred in the past and resulted in unique presentations of neurological disorders, occur infrequently today. Sections include the evaluation of toxic neuropathies and the approach to neurobehavioral impairment along with the cognitive domains commonly affected with exposure to neurointoxicants. A section describes the approach to a patient with exposure to neurointoxicants that i

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41

Menkes. Neurologic Signs and Symptoms in Children. Mosby-Year Book, 1991.

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42

Solomon, Tom, and Benedict Michael. Neurological infection. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0229.

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Neurological infections can be broadly subdivided into chronic/subacute and acute. Chronic/subacute infection usually presents with global cognitive decline, with the prototypical disease being progressive multifocal leucoencephalopathy due to infection with the JC virus in immunocompromised patients. Acute neurological infections can be defined microbiologically, by the nature of the pathogen; clinically, by the presenting signs and symptoms and initial CSF findings; or anatomically. The anatomical definitions are those occurring intracranially (‘meningitis’, where infection involves the meni

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43

Cader, Sarah. Demographics of neurological disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0222.

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This chapter reviews the epidemiology of common neurological disorders. While neurological symptoms can potentially appear confusing, understanding which diseases affect particular groups can provide diagnostic guidance.

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44

Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Drugs causing neurological disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0018.

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This chapter describes concise lists of common drug causes of neurological symptoms (cerebellar syndrome, chorea, dystonia, myoclonus, myopathy, peripheral neuropathy, posterior reversible encephalopathy syndrome (PRES), tremors, tics, and seizures).

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45

A Dictionary of Neurological Signs. Springer, 2005.

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46

Harley, Kim, and Sue Jones. Neurological and spinal surgery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642663.003.0023.

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Neurological assessment is performed on admission for surgery, as a routine part of medical examination. This is done to diagnose whether symptoms of illness in the patient are due to neurological conditions and, if so, where in the nervous system the pathological lesions are located. Hydrocephalus is either an acute or chronic condition whereby the cerebrospinal fluid pressure rises, causing symptoms of raised intracranial pressure. Patients at risk of raised intracranial pressure should be nursed by staff trained and experienced in neurological assessment using the Glasgow coma scale. This c

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47

Filippi, Massimo, and Jack H. Simon. Imaging Acute Neurologic Disease: A Symptom-Based Approach. Cambridge University Press, 2014.

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48

Imaging Acute Neurologic Disease: A Symptom-Based Approach. Cambridge University Press, 2014.

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49

Miller, Aaron E., and Teresa M. DeAngelis. Sjögren’s Syndrome. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199732920.003.0014.

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Primary Sjögren’s syndrome involves the cardinal siccal syndrome of xerostomia and xeroophthalmia. Neurological manifestations generally affect the peripheral nervous system in the form of neuropathy but can also target the central nervous system and overlap with demyelinating diseases such as multiple sclerosis and neuromyelitis optica. In this chapter, we review the systemic and neurologic features of Sjögren’s syndrome, and how to approach a patient with neurological symptoms secondary to Sjögren’s syndrome.

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50

Perez, David L., and Valerie Voon. The Neurobiology of PNES and Other Functional Neurological Symptoms. Edited by Barbara A. Dworetzky and Gaston C. Baslet. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190265045.003.0006.

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Patients with psychogenic nonepileptic seizures (PNES) and related functional neurological symptoms are highly prevalent yet poorly understood on a neurobiological level. Clinical and research efforts in PNES and other functional neurological symptoms have lagged behind clinical neuroscience advancements in other neuropsychiatric conditions, despite the high frequency with which clinicians encounter PNES and other functional neurological symptoms. In this chapter, systems-level neurobiological studies in PNES are reviewed. Specific emphasis is given to structural and functional neuroimaging, e

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