Relevant bibliographies by topics / Neuroprostanes

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Neuroprostanes'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Neuroprostanes"

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Quan, Long Guo, and Jin Kun Cha. "Preparation of Isoprostanes and Neuroprostanes." Journal of the American Chemical Society 124, no. 42 (2002): 12424–25. http://dx.doi.org/10.1021/ja027451z.

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Belcastro, Livia, Carolina S. Ferreira, Marcelle A. Saraiva, et al. "Decreased Fatty Acid Transporter FABP1 and Increased Isoprostanes and Neuroprostanes in the Human Term Placenta: Implications for Inflammation and Birth Weight in Maternal Pre-Gestational Obesity." Nutrients 13, no. 8 (2021): 2768. http://dx.doi.org/10.3390/nu13082768.

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The rise in prevalence of obesity in women of reproductive age in developed and developing countries might propagate intergenerational cycles of detrimental effects on metabolic health. Placental lipid metabolism is disrupted by maternal obesity, which possibly affects the life-long health of the offspring. Here, we investigated placental lipid metabolism in women with pre-gestational obesity as a sole pregnancy complication and compared it to placental responses of lean women. Open profile and targeted lipidomics were used to assess placental lipids and oxidised products of docosahexaenoic (D
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Signorini, Cinzia, Silvia Leoncini, Thierry Durand, et al. "Circulating 4-F4t-Neuroprostane and 10-F4t-Neuroprostane Are Related to MECP2 Gene Mutation and Natural History in Rett Syndrome." International Journal of Molecular Sciences 22, no. 8 (2021): 4240. http://dx.doi.org/10.3390/ijms22084240.

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Neuroprostanes, a family of non-enzymatic metabolites of the docosahexaenoic acid, have been suggested as potential biomarkers for neurological diseases. Objective biological markers are strongly needed in Rett syndrome (RTT), which is a progressive X-linked neurodevelopmental disorder that is mainly caused by mutations in the methyl-CpG binding protein 2 (MECP2) gene with a predominant multisystemic phenotype. The aim of the study is to assess a possible association between MECP2 mutations or RTT disease progression and plasma levels of 4(RS)-4-F4t-neuroprostane (4-F4t-NeuroP) and 10(RS)-10-F
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Signorini, Cinzia, Elena Moretti, Daria Noto, et al. "F4-Neuroprostanes: A Role in Sperm Capacitation." Life 11, no. 7 (2021): 655. http://dx.doi.org/10.3390/life11070655.

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F4-neuroprostanes (F4-NeuroPs), derived from the oxidative metabolization of docosahexaenoic acid (DHA), are considered biomarkers of oxidative stress in neurodegenerative diseases. Neurons and spermatozoa display a high DHA content. NeuroPs might possess biological activities. The aim of this in vitro study was to investigate the biological effects of chemically synthetized 4-F4t-NeuroP and 10-F4t-NeuroP in human sperm. Total progressive sperm motility (p < 0.05) and linearity (p = 0.016), evaluated by a computer-assisted sperm analyzer, were significantly increased in samples incubated wi
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Signorini, Cinzia, Claudio De Felice, Silvia Leoncini, et al. "F4-neuroprostanes mediate neurological severity in Rett syndrome." Clinica Chimica Acta 412, no. 15-16 (2011): 1399–406. http://dx.doi.org/10.1016/j.cca.2011.04.016.

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Jarocka-Karpowicz, Iwona, Anna Syta-Krzyżanowska, Jan Kochanowicz, and Zenon Dionizy Mariak. "Clinical Prognosis for SAH Consistent with Redox Imbalance and Lipid Peroxidation." Molecules 25, no. 8 (2020): 1921. http://dx.doi.org/10.3390/molecules25081921.

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Subarachnoid hemorrhage (SAH) accounts for 3% of all strokes. As more and more data indicates the role of oxidative stress in acute brain damage caused by SAH, an attempt was made to correlate the clinical status of patients with systemic level of antioxidants and lipid peroxidation products. The hemorrhage was diagnosed with brain computed tomography (CT) and aneurysm with angio-CT and angiography, while the vasospasm was monitored with transcranial Doppler. Plasma glutathione peroxidase activity (GSH-Px) and vitamin A, E, and C levels were determined spectrophotometrically and by HPLC, respe
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Taber, Douglass F., and L. Jackson Roberts. "Nomenclature systems for the neuroprostanes and for the neurofurans." Prostaglandins & Other Lipid Mediators 78, no. 1-4 (2005): 14–18. http://dx.doi.org/10.1016/j.prostaglandins.2005.07.002.

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Porta, Alessio, Maurizio Pasi, Enrico Brunoldi, Giuseppe Zanoni, and Giovanni Vidari. "Biology and chemistry of neuroprostanes. First total synthesis of 17-A4-NeuroP: Validation of a convergent strategy to a number of cyclopentenone neuroprostanes." Chemistry and Physics of Lipids 174 (September 2013): 64–74. http://dx.doi.org/10.1016/j.chemphyslip.2013.07.002.

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Huun, Marianne Ullestad, Håvard T. Garberg, Javier Escobar, et al. "DHA reduces oxidative stress following hypoxia-ischemia in newborn piglets: a study of lipid peroxidation products in urine and plasma." Journal of Perinatal Medicine 46, no. 2 (2018): 209–17. http://dx.doi.org/10.1515/jpm-2016-0334.

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AbstractBackground:Lipid peroxidation mediated by reactive oxygen species is a major contributor to oxidative stress. Docosahexaenoic acid (DHA) has anti-oxidant and neuroprotective properties. Our objective was to assess how oxidative stress measured by lipid peroxidation was modified by DHA in a newborn piglet model of hypoxia-ischemia (HI).Methods:Fifty-five piglets were randomized to (i) hypoxia, (ii) DHA, (iii) hypothermia, (iv) hypothermia+DHA or (v) sham. All groups but sham were subjected to hypoxia by breathing 8% O2. DHA was administered 210 min after end of hypoxia and the piglets w
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Roberts, L. Jackson, Thomas J. Montine, William R. Markesbery, et al. "Formation of Isoprostane-like Compounds (Neuroprostanes)in Vivofrom Docosahexaenoic Acid." Journal of Biological Chemistry 273, no. 22 (1998): 13605–12. http://dx.doi.org/10.1074/jbc.273.22.13605.

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Dissertations / Theses on the topic "Neuroprostanes"

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Fournial, Anaïs. "Synthèse totale de 10-F4t-Neuroprostanes et de Phytoprostanes E1 type I." Montpellier 2, 2007. http://www.theses.fr/2007MON20017.

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Oger, Camille. "Synthèses totales de neuroprostanes de type F, dérivées du DHA, de l'EPA et de l'AdA." Thesis, Montpellier 2, 2010. http://www.theses.fr/2010MON20071.

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L'acide docosahexaénoïque (DHA, C22 :6 w3), l'acide icosapentaénoïque (EPA, C20 : 5, w3) et l'acide adrénique (AdA, C22 :4 w6) sont présents en quantités importantes dans les membranes neuronales. Lors d'un stress oxydant, l'oxydation radicalaire de ces acides gras polyinsaturés conduit à la formation de métabolites nommés neuroprostanes (NeuroPs). Souhaitant avoir accès à de nouveaux biomarqueurs du stress oxydant neuronal, nous sommes intéréssés à la synthèse de NeuroPs de type F, issues du DHA, de l'EPA et de l'AdA<br>Docosahexaenoïc acid (DHA, C22 :6 w3), eicosapentaenoïc acid (EPA, C20 :
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Joumard-Cubizolles, Laurie. "Propriétés anti-athérogènes du DHA : effets nutrigénomiques au niveau aortique et rôle potentiel des métabolites issus de la peroxydation." Thesis, Clermont-Ferrand 1, 2013. http://www.theses.fr/2013CLF1MM17.

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Les acides gras polyinsaturés oméga-3 à longue chaîne (AGPIω3-LC), principalement représentés par l'acide eicosapentaénoïque (EPA) et l'acide docosahexaénoïque (DHA), présentent des effets bénéfiques vis-à-vis de l'athérosclérose. Leur action au niveau vasculaire est suggérée. Les mécanismes d'action sont mal compris du fait de la complexité d'action des AGPIω3-LC au niveau cellulaire. Les AGPIω3-LC affectent de nombreuses protéines y compris les facteurs de transcription, ce qui engendre la modulation de l'expression de nombreux gènes. La complexité s'accroît si l'on considère l'ensemble des
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Magee, Kurtis Michael. "Total Synthesis of 20-F4t-Neuroprostane." Thesis, Boston College, 2011. http://hdl.handle.net/2345/2423.

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Thesis advisor: Marc L. Snapper<br>Chapter 1: Oxidative Stress. An overview of oxidative stress and its impact on human health. Various reactive oxygen species and cellular antioxidant defenses are also summarized. Chapter 2: Neuroprostanes. After a survey of fatty acids and their nomenclature, this chapter discusses lipid peroxidation as one of the most significant consequences of oxidative stress in vivo. Lipid metabolites that serve as biomarkers for human diseases are also examined. Chapter 3: First Total Synthesis of 20-F4t-Neuroprostane. Beginning with a brief review of previous neuropro
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Bernad, Stéphane. "Synthèse totale de la 4(RS)-F4c-neuroprostane, molécule impliquée dans la maladie d'Alzheimer." Montpellier 2, 2003. http://www.theses.fr/2003MON20104.

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Chen, Ting Wei, and 陳頲瑋. "Levels of F2-isoprostanes and F4-neuroprostanes in Cerebrospinal Fluid of Patients with Traumatic Brain Injury." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/35259349799205049386.

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碩士<br>長庚大學<br>醫學生物技術暨檢驗學系<br>98<br>Traumatic brain injury (TBI) causes high mortality and morbidity rate in humans. TBI may induce oxidative stress by several mechanism, such as inflammation and hemoglobin release. We have previously shown that levels of F2-isoprostanes (F2-IsoPs) and F4-neuroprostanes (F4-NPs), the most reliable markers of lipid peroxidation derived from arachidonic acid and docosahexaenoic acid, respectively, in cerebrospinal fluid (CSF) predicted poor outcome of aneurysmal subarachnoid hemorrhage in patients. In this study, we first used the specific gas chromatography/n
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Hsieh, Yu Ping, and 謝育萍. "Analysis of F4-neuroprostanes, F2-isoprostanes, or Lipid-soluble Antioxidants in Humans and Rats following Non-traumatic Subarachnoid Hemorrhage." Thesis, 2008. http://ndltd.ncl.edu.tw/handle/38297932565583355704.

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碩士<br>長庚大學<br>醫學生物技術研究所<br>96<br>Aneurysmal subarachnoid hemorrhage (aSAH) caused by aneurysmal rupture is a major type of non-traumatic hemorrhagic stroke in humans. Following aSAH, patients may develope delayed ischemic neurological deficit leading to poor outcome. We hypothesized that oxidative stress would be increased following aSAH due to hemoglobin release and ischemic-reperfusion injury. F2-isoprostanes (F2-IsoPs) and F4-neuroprostanes (F4-NPs) are the most specific markers of lipid peroxidation derived from arachidonic acid and docosahexaenoic acid, respectively. Our previously pu
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Book chapters on the topic "Neuroprostanes"

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De Felice, Claudio, Silvia Leoncini, Cinzia Signorini, et al. "Neuroprostanes and Neurological Severity in Rett Syndrome." In Comprehensive Guide to Autism. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-4788-7_198.

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Reich, Erin E., William R. Markesbery, L. Jackson Roberts, Larry L. Swift, Jason D. Morrow, and Thomas J. Montine. "Quantification of F-Ring and D-/E-Ring Isoprostanes and Neuroprostanes in Alzheimer’s Disease." In Advances in Experimental Medicine and Biology. Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-0667-6_39.

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Reich, Erin E., Thomas J. Montine, and Jason D. Morrow. "Formation of Novel D-Ring and E-Ring Isoprostane-Like Compounds (D4/E4-Neuroprostanes)in Vivo From Docosahexaenoic Acidt." In Advances in Experimental Medicine and Biology. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0193-0_79.

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Arneson, Kyle O., and L. Jackson Roberts. "Measurement of Products of Docosahexaenoic Acid Peroxidation, Neuroprostanes, and Neurofurans." In Methods in Enzymology. Elsevier, 2007. http://dx.doi.org/10.1016/s0076-6879(07)33007-3.

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"Chapter Mass Spectrometry Methods for the Analysis of Oxidant Stress in Biological Fluids and Tissues: Quanti cation of F2-Isoprostanes and F4-Neuroprostanes Using Mass Spectrometry." In Lipid-Mediated Signaling. CRC Press, 2010. http://dx.doi.org/10.1201/9780849381423-15.

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