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Books on the topic 'Sire genetics'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Frederick, Carson Alistair, Dawson Lynne Evelyn Rosemary, Gordon Frederick James, Northern Ireland Agricultural Research and Development Council., and Northern Ireland. Department of Agriculture and Rural Development., eds. The Influence of suckler cow genetics and terminal sire on performance of the suckler herd. AgriSearch, 2004.

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2

Afrooz, Afghani, ed. Family history of osteoporosis. Nova Science Publishers, 2009.

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3

Jipa, Dan. Analiza granulometrică a sedimentelor: Semnificații genetice. Editura Academiei Republicii Socialiste România, 1987.

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4

International Congress on Pharmacogenetics and Anaesthetics (1986 San Remo, Italy). Pharmacogenetics and anaesthetics: Proceedings of the International Congress held in Sanremo, Italy, November 7-8th, 1986. Edited by Cominetti Marco, De Bellis Pasquale, and Rasore Quartino Alberto. Silver Press, 1988.

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5

Beaton, Alan. Left side, right side: A review of laterality research. Yale University Press, 1986.

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6

Beaton, Alan. Left side, right side: A review of laterality research. Batsford Academic and Educational, 1985.

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7

Smoller, Jordan W. The other side of normal: How biology is providing the clues to unlock the secrets of normal and abnormal behavior. HarperLuxe, 2012.

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8

International Symposium on Site-Directed Mutagenesis and Protein Engineering (1990 Tromsø, Norway). Site-directed mutagenesis and protein engineering: Proceedings of the International Symposium on Site-Directed Mutagensis and Protein Engineering, Tromsø, 27-30 August 1990. Edited by el-Gewely M. Rafaat. Elsevier Science Publishers., 1991.

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9

International Symposium on Site-Directed Mutagenesis and Protein Engineering (1990 Tromsø, Norway). Site-directed mutagenesis and protein engineering: Proceedings of the International Symposium on Site-Directed Mutagenesis and Protein Engineering, Tromsø, 27-30 August 1990. Edited by El-Gewely M. Rafaat. Elsevier Science Publishers, 1991.

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10

Maria, Pacifici Gian, and Pelkonen R. O, eds. Interindividual varaiblity in human drug metabolism. Taylor & Francis, 2001.

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11

Chŏng, I.-suk. Simjang pujagyong yujŏnja kŏmsaek kisul hwangnip ŭl tʻonghan anjŏnsŏng yangni yŏnʼgu: Simjang pujagyong yujŏnja kŏmsaekpŏp hwangnip mit simjang pujagyong deitʻŏbeisŭ hwakpo = Safety pharmacology study through the development of genetic marker for the evaluation of the cardiac side effects : Development of genetic marker for the cardiac side effect and evaluation of the cardiac side effects. Sikpʻum Ŭiyakpʻum Anjŏnchʻŏng, 2007.

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12

Zenker, Frank. Bayesian Argumentation: The practical side of probability. Springer Netherlands, 2013.

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13

F, Amábile-Cuevas Carlos, ed. Antibiotic resistance: From molecular basics to therapeutic options. Chapman & Hall, 1996.

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14

Gillham, Nicholas W. A life of Sir Francis Galton: From African exploration to the birth of Eugenics. Oxford University Press, 2001.

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15

Moya, Joaquim. Sire of fetus effects and environmental correlations for dairy cattle production and reproduction. 1985.

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16

Walsh, Bruce, and Michael Lynch. The Genetic Effective Size of a Population. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198830870.003.0003.

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The effects of genetic drift usually assume an idealized population of constant size. This chapter shows how the population size for such an idealized population can be replaced with an effective population size for populations with age structure, unequal sex ratios, a history of expansion or contraction, inbreeding, and population subdivision. These demographic features impact the entire genome more or less equally. A relatively recent understanding is that selection at a site can dramatically reduce the local effective population size experienced by nearby linked sites (the Hill-Robertson ef

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17

Kohn, Alfie. The Brighter Side of Human Nature: Altruism Empathy in Everyday Life. Basic Books, 1992.

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18

Frankham, Richard, Jonathan D. Ballou, Katherine Ralls, et al. Evolutionary genetics of small populations. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198783398.003.0002.

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Genetic management of fragmented populations involves the application of evolutionary genetic theory and knowledge to alleviate problems due to inbreeding and loss of genetic diversity in small population fragments. Populations evolve through the effects of mutation, natural selection, chance (genetic drift) and gene flow (migration). Large outbreeding, sexually reproducing populations typically contain substantial genetic diversity, while small populations typically contain reduced levels. Genetic impacts of small population size on inbreeding, loss of genetic diversity and population differe

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19

Renton, Alan E., and Alison M. Goate. Genetics of Dementia. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0051.

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The genetic architecture of dementia is polygenic and complex, with risk alleles spanning frequency–effect size space. Despite significant progress, most genes influencing these disorders await discovery. Known risk loci implicate perturbed pathways that coalesce around recurring mechanistic themes, notably the autophagosome-lysosome system, the cytoskeleton, endocytosis, innate immunity, lipid metabolism, mitochondria, and the ubiquitin-proteasome system. Phenotypic and pathophysiological pleiotropy suggests some conditions form continuous clinicopathogenetic disease spectra blurring classica

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20

The other side of normal: How biology is providing the clues to unlock the secrets of normal and abnormal behavior. William Morrow, 2012.

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21

Láruson, Áki Jarl, and Floyd Allan Reed. Population Genetics with R. Oxford University Press, 2021. http://dx.doi.org/10.1093/oso/9780198829539.001.0001.

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Population genetics is an inherently quantitative discipline. Because the focus of population genetics studies is usually on abstract concepts like the frequencies of genetic variants over time, it can at first glance be difficult to conceptualize and appropriately visualize. As more and more quantitative models and methods have become established in the discipline, it has become necessary for people just entering the field to quickly develop a good understanding of the many layers of complex approaches, so as to correctly interpret even basic results. An unfortunate side effect of the widespr

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22

Eyre, Steve, and Jane Worthington. Genetics of rheumatoid arthritis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0040.

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A range of epidemiological studies have clearly established that susceptibility to rheumatoid arthritis (RA) is determined by both genetic and environmental factors. Studies over the last five decades have used a variety of approaches to identify the genetic variants associated with disease. HLA DRB1 was the first RA susceptibility locus to be discovered and has the largest effect size. We describe current understanding of the complexities of HLA association for RA. Linkage and small-scale association studies prior to 2007 provided convincing evidence for only one more RA susceptibility locus,

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23

Eyre, Steve, Jane Worthington, and Sebastien Viatte. Genetics of rheumatoid arthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0040_update_003.

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A range of epidemiological studies have clearly established that susceptibility to rheumatoid arthritis (RA) is determined by both genetic and environmental factors. Studies over the last five decades have used a variety of approaches to identify the genetic variants associated with disease. HLA DRB1 was the first RA susceptibility locus to be discovered and has the largest effect size. We describe current understanding of the complexities of HLA association for RA. Linkage and small-scale association studies prior to 2007 provided convincing evidence for only one more RA susceptibility locus,

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24

Turkheimer, Eric. The nature of nature. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198725978.003.0026.

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Chapter 26 covers how Sir Francis Galton was the first to apply the Shakespearian phrase “nature and nurture” to human individual differences. The so-called nature–nurture debate began as a question of whether differences among people could be attributed to in-born characteristics or environmental characteristics. Galton predated both scientific understanding of genetics and contemporary conceptualizations of behavior, but in the century that followed it was learned that: (1) the most general answer is, “both”, (2) genetic differences were more pervasive and powerful than had been anticipated,

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25

U.S. Genetic Testing Market. https://www.fortunebusinessinsights.com/u-s-genetic-testing-market-105034, 2020.

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26

Schutte, Nienke M., Meike Bartels, and Eco JC de Geus. Genetics of physical activity and physical fitness. Edited by Neil Armstrong and Willem van Mechelen. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198757672.003.0020.

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Regular physical activity and fitness are key contributors to children’s health. It is important to understand sources of variation in phenotypes seen among children and adolescents. It is important to calculate the relative importance of genetic and environmental factors to observed individual differences. Heritability estimates of physical activity vary, depending on sample size and measurement instrument, but the overall importance of environmental factors seems to decrease in adolescence, whereas genetic effects become more prominent. Twin and family studies show that individual difference

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27

Haun, Marianna. The X Factor: What It Is & How to Find It: The Relationship Between Inherited Heart Size and Racing Performance. Russell Meerdink Company, 1997.

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28

Haun, Marianna. Understanding the Power of the X Factor: Patterns of Heart Score and Performance. Russell Meerdink Company, 2001.

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29

Mills, M. G. L., and M. E. J. Mills. Morphometrics, demographics, and genetic viability. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198712145.003.0002.

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Morphometric data showed that southern Kalahari male cheetahs are larger than females and coalition males are larger than single males. Both tend to be smaller than cheetahs from other regions. The estimated density was 0.7 adult cheetahs/100 km2. Adult males were either single (43.2%) or in two-male (35.8%) or three-male (20.9%) coalitions. Only two out of seven two-male coalitions were full siblings. Litter sizes at birth and emergence were similar to those in the Serengeti, but age at independence and at first litter were older, and litter size at independence larger. Cub sex ratio was equa

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30

PLANT GENETIC RESOURCES FOR GENETIC TECHNOLOGIES: TO THE 100TH ANNIVERSARY OF PUSHKIN LABORATORIES OF VIR. N.I. Vavilov All-Russian Institute of Plant Genetic Resources, 2022. http://dx.doi.org/10.30901/978-5-907145-84-9.

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The program and abstracts of the All-Russian Conference Plant Genetic Resources for Genetic Technologies: To the 100th Anniversary of Pushkin Laboratories of VIR are presented. The conference was held at VIR on June 22–23, 2022, under Agreement No 075-15-2021-1050 of Sept. 28, 2021 (hereinafter: Event/Conference). The new technological setup aimed at the development of bioeconomy increases the demand for bioresource collections (BRC), including collections of plant genetic resources. Currently, new approaches to collection management and studying are being actively introduced. The foundations

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31

Yende, Sachin, and Derek C. Angus. Genetic Determinants of Sepsis Outcomes. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0027.

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Bone and joint processes take second stage to life-threatening organ failure in the setting of critical illness. However, bone and joint disorders can cause significant impairment in survivors of critical illness. Return to pre-admission function is often limited by acquired complications such as joint contractures, heterotopic ossification, and altered bone metabolism. Critical care physicians should maintain a high index of suspicion for joint contractures, as they are often asymptomatic but the source of enduring disability once the critical illness had receded. Research is needed to docume

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32

Goldman, David, Zhifeng Zhou, and Colin Hodgkinson. The Genetic Basis of Addictive Disorders. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0042.

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Addictive disorders are moderately to highly heritable, indicating that alleles transmitted from parents are protective, or enhance risk by whatever mechanisms. However, the inheritance of addictive disorders is complex, involving hundreds of genes and variants that are both common and rare, and that vary in effect size and context of action. Genes altering risk for addictions have been identified by pathway and candidate gene studies in humans and model organisms, and genomic approaches including genome-wide association, meiotic linkage, and sequencing. Genes responsible for shared liability

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33

Biotech time-bomb: The side-effects are the main effects. Cresskill, NJ, 2009.

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34

Frankham, Richard, Jonathan D. Ballou, Katherine Ralls, et al. Loss of genetic diversity reduces ability to adapt. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198783398.003.0004.

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Environmental change is a ubiquitous feature of the conditions faced by species, so they must either evolve, move to avoid threats, or perish. Species require genetic diversity to evolve to cope with environmental change through natural selection (adaptive evolution). The ability of populations to undergo adaptive evolution depends upon the strength of selection, genetic diversity, effective population size, mutation rates and number of generations. Loss of genetic diversity in small populations reduces their ability to evolve to cope with environmental change, thus increasing their extinction

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35

Frankham, Richard, Jonathan D. Ballou, Katherine Ralls, et al. Genetic Management of Fragmented Animal and Plant Populations. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198783398.001.0001.

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The biological diversity of the planet is being rapidly depleted due to the direct and indirect consequences of human activity. As the size of animal and plant populations decrease and fragmentation increases, loss of genetic diversity reduces their ability to adapt to changes in the environment, with inbreeding and reduced fitness inevitable consequences for many species. Many small isolated populations are going extinct unnecessarily. In many cases, such populations can be genetically rescued by gene flow into them from another population within the species, but this is very rarely done. Thi

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36

Cassidy, Jim, Donald Bissett, Roy A. J. Spence OBE, Miranda Payne, Gareth Morris-Stiff, and Madhumita Bhattacharyya. Skin cancers. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199689842.003.0023_update_001.

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Tumours of the central nervous system examines the epidemiology, aetiology, genetics and pathology of these heterogeneous tumours. Clinical presentation reflects the site of origin and rate of growth. Investigation usually comprises imaging (MRI superior to CT for most), and biopsy; requirement for additional staging depends on pathology. The treatment of low-grade gliomas may be delayed if small with few symptoms, otherwise surgery and/or radiotherapy. High grade gliomas may be managed with surgery, radiotherapy, and temozolomide chemotherapy in fit patients. Unfit patients should be offered

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37

Beaton, Alan. Left Side/Right Side: A Review of Laterality Research. Yale University Press, 1986.

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38

Left Side, Right Side: A Review of Laterality Research. New Haven 1985., 1985.

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39

Left Side/Right Side: A Review of Laterality Research. Yale University Press, 1986.

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40

Frankham, Richard, Jonathan D. Ballou, Katherine Ralls, et al. Population fragmentation causes inadequate gene flow and increases extinction risk. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198783398.003.0005.

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Most species now have fragmented distributions, often with adverse genetic consequences. The genetic impacts of population fragmentation depend critically upon gene flow among fragments and their effective sizes. Fragmentation with cessation of gene flow is highly harmful in the long term, leading to greater inbreeding, increased loss of genetic diversity, decreased likelihood of evolutionary adaptation and elevated extinction risk, when compared to a single population of the same total size. The consequences of fragmentation with limited gene flow typically lie between those for a large popul

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41

Chi, Ming-Hui. Genetic analysis of trancriptional silencing in yeast: Characterization of sir2 suppressors and Sir-2interacting factors. 1996.

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42

Frankham, Richard, Jonathan D. Ballou, Katherine Ralls, et al. A Practical Guide for Genetic Management of Fragmented Animal and Plant Populations. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780198783411.001.0001.

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The biological diversity of the planet is being rapidly depleted due to the direct and indirect consequences of human activity. As the size of wild animal and plant populations decreases and fragmentation increases, inbreeding reduces fitness and loss of genetic diversity reduces their ability to adapt to changes in the environment. Many small isolated populations are going extinct unnecessarily. In many cases, such populations can be genetically rescued by gene flow from another population within the species, but this is very rarely done. This book provides a practical guide to the genetic ma

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43

Saleem, Moin A., and Corinne Antignac. Molecular basis of nephrotic syndrome. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0327_update_001.

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Nephrotic syndrome is broadly a disorder of the glomerular filtration barrier, but in practice the site of dysfunction in the great majority of pathologies is in the podocyte. Genetic causes of nephrotic syndrome provide the strongest proof of this. Almost all the genetic associations with nephrotic syndrome are podocyte proteins. Some basement membrane protein mutations associated with nephrotic syndrome may act through signalling to podocytes, or by causing severe disruption to their environment.

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44

(Editor), Supratim Choudhuri, and David B. Carlson (Editor), eds. Genomics: Fundamentals and Applications. Informa Healthcare, 2008.

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45

Hull, Dwight Ellsworth. Some Methods of Evaluating the Genetic Worth for Milk and Butterfat Transmission of Sires Used in Artificial Breeding. Creative Media Partners, LLC, 2021.

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46

Sayer, John A., and Roslyn J. Simms. Nephronophthisis. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0317_update_001.

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Nephronophthisis (NPHP) is a clinically heterogeneous autosomal recessive cystic kidney disease and the leading genetic cause of end-stage renal failure in children and young adults. Whilst enlarged dysplastic cystic kidneys are associated with infantile NPHP, more typically renal ultrasound reveals normal kidney size and corticomedullary cysts in a child with polyuria and secondary enuresis. Extrarenal manifestations occur in 10–15% including retinal degeneration, cerebellar vermis hypoplasia and liver fibrosis, requiring referral to other specialists. Mutations in 18 genes have been identifi

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47

Wiebe, Peter H., Ann Bucklin, and Mark Benfield. Sampling, Preservation and Counting of Samples II: Zooplankton. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780199233267.003.0010.

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This chapter reviews traditional and new zooplankton sampling techniques, sample preservation, and sample analysis, and provides the sources where in-depth discussion of these topics is addressed. The net systems that have been developed over the past 100+ years, many of which are still in use today, can be categorized into eight groups: non-opening/closing nets, simple opening/closing nets, high-speed samplers, neuston samplers, planktobenthos plankton nets, closing cod-end samplers, multiple net systems, and moored plankton collection systems. Methods of sample preservation include preservat

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48

Keiles, Steven, Margaret Lilley, and Heather MacLeod. Types of Laboratories and Business Relationships. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190604929.003.0001.

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Laboratory genetic counselors work in many different laboratory settings that can represent a variety of clinical specialties. This chapter explores the various types of laboratories and their resulting business relationships. Genetic counselors working in a laboratory will build important relationships with colleagues from within their own company as well as competitors, clients, and others they work with externally. The laboratory’s specialty and its setting will determine who their clients are and is also directly related to their business model. CPT codes and billing and reimbursement issu

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49

Denver, Dee. The Dharma in DNA. Oxford University Press, 2022. http://dx.doi.org/10.1093/oso/9780197604588.001.0001.

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The Dharma in DNA has three objectives: (1) to share the rich but underappreciated history of biology–Buddhism intersections and surprising harmonies between the two traditions, (2) to evaluate Buddhist teachings from a scientific perspective using DNA as the focus of study, and (3) to propose a new approach to science, Bodhi science, as an ethical and operational framework for conducting Buddhist wisdom–guided science and preventing pseudoscience. An interwoven side project examines the life journey of the author, a professor of genetics and father in a transracial adoptive family, who questi

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50

Adami, Hans-Olov, David J. Hunter, Pagona Lagiou, and Lorelei Mucci, eds. Textbook of Cancer Epidemiology. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.001.0001.

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This book offers an overview of the epidemiology and primary prevention for most forms of human cancer. It summarizes concepts and methods of epidemiology, the biology of cancer, cancer genetics, and the emerging potential of biomarkers. It also reviews specific cancer sites in a consistent way, providing clinical and pathological outlines, descriptive epidemiology, and a comprehensive account of traditional and molecular risk factors and their etiological importance. To facilitate reading and use of our Textbook as a reference, we have consistently addressed potential risk factors in the same

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