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Artículos de revistas sobre el tema "Amino acid; Neurotransmitter"

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Publicado: 4 de junio de 2021
Última modificación: 14 de febrero de 2022

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1

Marshall, Kenneth C., and Huangui Xiong. "Modulation of amino acid neurotransmitter actions by other neurotransmitters: some examples." Canadian Journal of Physiology and Pharmacology 69, no. 7 (1991): 1115–22. http://dx.doi.org/10.1139/y91-163.

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Developments in the field of central neurotransmission indicate that amino acids serve as important and widespread transmitters throughout the central nervous system. There are increasing indications from recent experimental studies that several of the other central neurotransmitters may exert potent effects on central neurons by modulating the actions of amino acids. Noradrenaline and serotonin have received particular attention as potential modulators, and a wide variety of actions has been reported for them. Modulatory actions have been reported at both pre- and post-synaptic levels, includ
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2

SHIGERI, Yasushi, and Keiko SHIMAMOTO. "Function of Neurotransmitter Amino Acid Transporters." Seibutsu Butsuri 46, no. 1 (2006): 10–14. http://dx.doi.org/10.2142/biophys.46.10.

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3

Akagi, Hiroyuki, and Masayoshi Mishina. "Amino acid neurotransmitter receptors: Introductory remarks." Japanese Journal of Pharmacology 64 (1994): 42. http://dx.doi.org/10.1016/s0021-5198(19)49903-4.

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4

Levi, Giulio, and Maurizio Raiteri. "Neurotransmitter Amino Acid Release from Synaptosomes." Journal of Neurochemistry 53, no. 4 (1989): 1324–26. http://dx.doi.org/10.1111/j.1471-4159.1989.tb07432.x.

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5

Erecińska, Maria. "The neurotransmitter amino acid transport systems." Biochemical Pharmacology 36, no. 21 (1987): 3547–55. http://dx.doi.org/10.1016/0006-2952(87)90001-3.

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6

SCHAEFER, FRANZ, MARCEL VOGEL, GUIDO KERKHOFF, JOHANNES WOITZIK, MARKUS DASCHNER, and OTTO MEHLS. "Experimental Uremia Affects Hypothalamic Amino Acid Neurotransmitter Milieu." Journal of the American Society of Nephrology 12, no. 6 (2001): 1218–27. http://dx.doi.org/10.1681/asn.v1261218.

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Abstract. Chronic renal failure is associated with delayed puberty and hypogonadism. To investigate the mechanisms subserving the reported reduced pulsatile release of gonadotropin-releasing hormone (GnRH) in chronic renal failure, this study examined the amino acid neurotransmitter milieu in the medial preoptic area (MPOA), the hypothalamic region where the GnRH-secreting neurons reside, in 5/6-nephrectomized male rats and in ad libitum-fed or pair-fed controls. All rats were castrated and received either a testosterone or a vehicle implant to evaluate additional effects of the prevailing sex
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7

Mishchenko, Oksana, Natalia Palagina, Yuliia Larianovskaya, Tatyana Gorbach, Viktor Khomenko, and Nataliia Yasna. "Influence of a new derivative of 4-aminobutanoic acid on the level of neuromediatory aminoacids, neuromediators and the state of the rats’ hypocamp in conditions of brain ischemia." ScienceRise: Pharmaceutical Science, no. 2 (30) (April 30, 2021): 64–71. http://dx.doi.org/10.15587/2519-4852.2021.230305.

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The aim: to investigate the effect of a new derivative of 4-aminobutanoic acid (compounds KGM-5) on the level of neurotransmitters and neurotransmitter amino acids and the structural-functional state of the hippocampus of rats with acute cerebrovascular accident (ACVA).
 Materials and methods. ACVA was reproduced in rats by occlusion of the left carotid artery under anesthesia (sodium thiopental (35 mg/kg) intraperitoneally (i/p). 5 groups of animals were used: intact control (IC, n=6), untreated animals with ACVA (CP, n=13); animals with ACVA (n=14), which were treated for 5 days with KG
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8

Nelson, N., and H. Lill. "Porters and neurotransmitter transporters." Journal of Experimental Biology 196, no. 1 (1994): 213–28. http://dx.doi.org/10.1242/jeb.196.1.213.

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Uptake of neurotransmitters involves multiple transporters acting in different brain locations under different physiological conditions. The vesicular transporters are driven by a proton-motive force generated by a V-ATPase and their substrates are taken up via proton/substrate exchange. The plasma membrane transporters are driven by an electrochemical gradient of sodium generated by a Na+/K(+)-ATPase. Two distinct families of transporters were identified in this group. One cotransports sodium with glutamate and other amino acids and requires additionally an outwardly directed potassium gradie
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9

Jones, Eugenia M. C. "Na+ - and Cl−-dependent neurotransmitter transporters in bovine retina: Identification and localization by in situ hybridization histochemistry." Visual Neuroscience 12, no. 6 (1995): 1135–42. http://dx.doi.org/10.1017/s0952523800006775.

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AbstractThe physiological actions of biogenic amine and amino-acid neurotransmitters are terminated by their removal from the synaptic cleft by specific high-affinity transport proteins. The members of the Na+- and Cl−-dependent neurotransmitter transporter family expressed in bovine retina and responsible for the uptake of biogenic amine and amino-acid neurotransmitters were identified using a reverse transcriptase-polymerase chain reaction-based approach. cDNA clones encoding bovine homologues of glycine (GLYT-1), γ-aminobutyric acid (GAT-1) creatine (CreaT), and orphan (NTT4) transporters w
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10

Goldsmith, R. F., J. W. Earl, and A. M. Cunningham. "Determination of delta-aminobutyric acid and other amino acids in cerebrospinal fluid of pediatric patients by reversed-phase liquid chromatography." Clinical Chemistry 33, no. 10 (1987): 1736–40. http://dx.doi.org/10.1093/clinchem/33.10.1736.

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Abstract The reversed-phase liquid-chromatographic system described here is capable of resolving the neurotransmitter amino acids aspartic acid, glutamic acid, and gamma-aminobutyric acid (GABA) plus 21 other amino acids in cerebrospinal fluid (CSF) in a single analysis. The amino acids, derivatized with o-phthalaldehyde, are separated in 65 min. Concentrations of glutamine less than or equal to 600 mumol/L can be measured at the same time as GABA greater than or equal to 10 nmol/L. Using this method, we have determined reference intervals for amino acids, including GABA, in CSF in a group of
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11

Ueda, Tetsufumi. "AMINO ACID NEUROTRANSMITTER UPTAKE SYSTEMS IN SYNAPTIC VESICLES." Japanese Journal of Pharmacology 55 (1991): 11. http://dx.doi.org/10.1016/s0021-5198(19)37733-9.

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12

Nilsson, G. E., and P. L. Lutz. "Release of inhibitory neurotransmitters in response to anoxia in turtle brain." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 261, no. 1 (1991): R32—R37. http://dx.doi.org/10.1152/ajpregu.1991.261.1.r32.

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In mammals a massive release of the excitatory neurotransmitter glutamate, occurring within a few minutes of anoxia/ischemia, is thought to be a major mediator of anoxic brain damage. In contrast to the mammalian brain, the turtle brain is exceptionally anoxia tolerant. Using intracerebral microdialysis in turtle brain striatum, we have found a large increase in the extracellular level of the inhibitory neurotransmitter gamma-aminobutyric acid during anoxia, reaching 90 times the normoxic level after 240 min, whereas no substantial release of glutamate occurred. Moreover, the inhibitory neurot
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13

Bröer, Angelika, Nadine Tietze, Sonja Kowalczuk, et al. "The orphan transporter v7-3 (slc6a15) is a Na+-dependent neutral amino acid transporter (B0AT2)." Biochemical Journal 393, no. 1 (2005): 421–30. http://dx.doi.org/10.1042/bj20051273.

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Transporters of the SLC6 (solute carrier 6) family play an important role in the removal of neurotransmitters in brain tissue and in amino acid transport in epithelial cells. In the present study, we demonstrate that mouse v7-3 (slc6a15) encodes a transporter for neutral amino acids. The transporter is functionally and sequence related to B0AT1 (slc6a19) and was hence named B0AT2. Leucine, isoleucine, valine, proline and methionine were recognized by the transporter, with values of K0.5 (half-saturation constant) ranging from 40 to 200 μM. Alanine, glutamine and phenylalanine were low-affinity
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14

Takemoto, Yumi. "Amino Acids That Centrally Influence Blood Pressure and Regional Blood Flow in Conscious Rats." Journal of Amino Acids 2012 (May 29, 2012): 1–14. http://dx.doi.org/10.1155/2012/831759.

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Functional roles of amino acids have increasingly become the focus of research. This paper summarizes amino acids that influence cardiovascular system via the brain of conscious rats. This paper firstly describes why amino acids are selected and outlines how the brain regulates blood pressure and regional blood flow. This section includes a concise history of amino acid neurotransmitters in cardiovascular research and summarizes brain areas where chemical stimulations produce blood pressure changes mainly in anesthetized animals. This is followed by comments about findings regarding several ne
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15

Harper, Andrew, William R. Blythe, Carlton J. Zdanski, Jiri Prazma, and Harold C. Pillsbury. "Student Research Award 1994: Nitric Oxide in the Rat Vestibular System." Otolaryngology–Head and Neck Surgery 111, no. 4 (1994): 430–38. http://dx.doi.org/10.1177/019459989411100407.

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Nitric oxide is known to function as a neurotransmitter in the central nervous system. It is also known to be involved in the control nervous system excitatory amino acid neurotransmission cascade. Activation of excitatory amino acid receptors causes an influx of calcium, which activates nitric oxide synthase. The resulting increase in intracellular nitric oxide activates soluble guanylate cyclase, leading to a rise in cyclic guanosine monophosphate. The excitatory amino acids giutamate and aspartate are found in the vestibular system and have been postulated to function as vestibular system n
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16

Jung-Klawitter, Sabine, and Oya Kuseyri Hübschmann. "Analysis of Catecholamines and Pterins in Inborn Errors of Monoamine Neurotransmitter Metabolism—From Past to Future." Cells 8, no. 8 (2019): 867. http://dx.doi.org/10.3390/cells8080867.

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Inborn errors of monoamine neurotransmitter biosynthesis and degradation belong to the rare inborn errors of metabolism. They are caused by monogenic variants in the genes encoding the proteins involved in (1) neurotransmitter biosynthesis (like tyrosine hydroxylase (TH) and aromatic amino acid decarboxylase (AADC)), (2) in tetrahydrobiopterin (BH4) cofactor biosynthesis (GTP cyclohydrolase 1 (GTPCH), 6-pyruvoyl-tetrahydropterin synthase (PTPS), sepiapterin reductase (SPR)) and recycling (pterin-4a-carbinolamine dehydratase (PCD), dihydropteridine reductase (DHPR)), or (3) in co-chaperones (DN
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17

Kanner, Baruch I. "Amino acid neurotransmitter reuptake: mechanistics, biochemistry and molecular cloning." Biochemical Society Transactions 19, no. 1 (1991): 92–95. http://dx.doi.org/10.1042/bst0190092.

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18

Feleder, Carlos, Damián Refojo, Silvina Nacht, and Jaime A. Moguilevsky. "Interleukin-1 Stimulates Hypothalamic Inhibitory Amino Acid Neurotransmitter Release." Neuroimmunomodulation 5, no. 1-2 (1998): 1–4. http://dx.doi.org/10.1159/000026319.

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19

Clark, Janet A., and Susan G. Amara. "Amino acid neurotransmitter transporters: Structure, function, and molecular diversity." BioEssays 15, no. 5 (1993): 323–32. http://dx.doi.org/10.1002/bies.950150506.

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20

Chapman, A. G., and G. P. Hart. "Anticonvulsant drug action and regional neurotransmitter amino acid changes." Journal of Neural Transmission 72, no. 3 (1988): 201–12. http://dx.doi.org/10.1007/bf01243420.

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21

Raymond, Lynn A., Craig D. Blackstone, and Richard L. Huganir. "Phosphorylation of amino acid neurotransmitter receptors in synaptic plasticity." Trends in Neurosciences 16, no. 4 (1993): 147–53. http://dx.doi.org/10.1016/0166-2236(93)90123-4.

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22

Reynolds, J. N., and W. J. Racz. "Effects of methylmercury on the spontaneous and potassium-evoked release of endogenous amino acids from mouse cerebellar slices." Canadian Journal of Physiology and Pharmacology 65, no. 5 (1987): 791–98. http://dx.doi.org/10.1139/y87-127.

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The effects of methylmercury on the spontaneous and potassium-evoked release of endogenous amino acids from mouse cerebellar slices have been examined. Methylmercury induced a concentration-dependent increase in the spontaneous release of glutamate, aspartate, γ-aminobutyric acid, and taurine from mouse cerebellar slices. Glycine release was slightly increased, but not in a concentration-dependent manner. The spontaneous release of glutamine from mouse cerebellar slices was not altered by any concentration of methylmercury examined (10, 20, and 50 μM). The tissue content of glutamate, γ-aminob
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23

Trudeau, L. E., and V. F. Castellucci. "Excitatory amino acid neurotransmission at sensory-motor and interneuronal synapses of Aplysia californica." Journal of Neurophysiology 70, no. 3 (1993): 1221–30. http://dx.doi.org/10.1152/jn.1993.70.3.1221.

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1. Although the gill and siphon withdrawal reflex of Aplysia has been used as a model system to study learning-associated changes in synaptic transmission, the identity of the neurotransmitter released by the sensory neurons and excitatory interneurons of the network mediating this behavior is still unknown. The identification of the putative neurotransmitter of these neurons should facilitate further studies of synaptic plasticity in Aplysia. 2. We report that sensory-motor transmission within this circuit is mediated through the activation of an excitatory amino acid receptor that is blocked
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24

Cordeiro, Dawn, Garrett Bullivant, Ronald D. Cohn, Julian Raiman, and Saadet Mercimek-Andrews. "Outcome of Patients With Inherited Neurotransmitter Disorders." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 45, no. 5 (2018): 571–76. http://dx.doi.org/10.1017/cjn.2018.266.

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AbstractWe report the outcome of 12 patients with inherited neurotransmitter disorders of monoamine, tetrahydrobiopterin and γ amino butyric acid metabolisms from a single Inherited Neurotransmitter Disorder Clinic including tyrosine hydroxylase (n=2), aromatic l-amino acid decarboxylase (n=1), 6-pyruvoyltetrahydropterin synthase, dihydropteridine reductase and succinic semialdehyde dehydrogenase deficiencies. Six patients (with 6-pyruvoyltetrahydropterin synthase, dihydropteridine reductase and tyrosine hydroxylase deficiencies) had normal neurodevelopmental outcome on treatment. Tetrahydrobi
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25

Kanner, B. I. "Sodium-coupled neurotransmitter transport: structure, function and regulation." Journal of Experimental Biology 196, no. 1 (1994): 237–49. http://dx.doi.org/10.1242/jeb.196.1.237.

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The removal of neurotransmitters by their transporters--located in the plasma membranes of nerve terminals and glial cells--plays an important role in the termination of synaptic transmission. In the last 3 years, many neurotransmitter transporters have been cloned. Structurally and functionally they can be divided into two groups: glutamate transporters, of which to date three have been cloned, couple the flow of glutamate to that of sodium and potassium. The second group of transporters includes those for GABA, glycine, taurine, norepinephrine, dopamine and serotonin. They are sodium- and ch
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26

Liu, Lin. "Toxicity of Pesticide on Neurotransmitter." Advanced Materials Research 518-523 (May 2012): 2045–48. http://dx.doi.org/10.4028/www.scientific.net/amr.518-523.2045.

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Pesticides not only play an important role in development of agricultural in China, but also bring much neurotoxicity on the safety of human by intervention compound, analyze and metabolize of neurotransmitter. Many pesticides have neurotransmitter toxicity, including inhibition of cholinesterase activity, increasing excitatory amino acid, decreasing the level of Dopamine. The paper made a brief overview on the toxicity of pesticides on neurotransmitter.
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27

Kumar, Pratik, Wei Huang, David Shukhman, Frank M. Camarda, and Scott T. Laughlin. "Stable cyclopropene-containing analogs of the amino acid neurotransmitter glutamate." Tetrahedron Letters 60, no. 22 (2019): 1476–80. http://dx.doi.org/10.1016/j.tetlet.2019.04.046.

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28

Sonnewald, U., H. Waagepetersen, H. Qu, and A. Schousboe. "New aspects of amino acid neurotransmitter synthesis in cultured neurons." Journal of Neurochemistry 81 (June 28, 2008): 43–44. http://dx.doi.org/10.1046/j.1471-4159.2002.00045.x.

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29

Schousboe, A., N. Westergaard, and U. Sonnewald. "4 Role of astrocytes in amino acid neurotransmitter precursor homeostasis." International Journal of Developmental Neuroscience 14 (July 1996): 51. http://dx.doi.org/10.1016/0736-5748(96)80199-7.

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30

Hansson, Elisabeth, and Lars Rönnbäck. "Adrenergic receptor regulation of amino acid neurotransmitter uptake in astrocytes." Brain Research Bulletin 29, no. 3-4 (1992): 297–301. http://dx.doi.org/10.1016/0361-9230(92)90060-b.

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31

Reynolds, Gavin P., and Christopher E. J. Warner. "Amino acid neurotransmitter deficits in adult Down's syndrome brain tissue." Neuroscience Letters 94, no. 1-2 (1988): 224–27. http://dx.doi.org/10.1016/0304-3940(88)90299-6.

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32

Yoneda, Yukio, Koichi Kawada, and Nobuyuki Kuramoto. "Selective Upregulation by Theanine of Slc38a1 Expression in Neural Stem Cell for Brain Wellness." Molecules 25, no. 2 (2020): 347. http://dx.doi.org/10.3390/molecules25020347.

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Theanine is an amino acid abundant in green tea with an amide moiety analogous to glutamine (GLN) rather than glutamic acid (Glu) and GABA, which are both well-known as amino acid neurotransmitters in the brain. Theanine has no polyphenol and flavonoid structures required for an anti-oxidative property as seen with catechins and tannins, which are more enriched in green tea. We have shown marked inhibition by this exogenous amino acid theanine of the uptake of [3H]GLN, but not of [3H]Glu, in rat brain synaptosomes. Beside a ubiquitous role as an endogenous amino acid, GLN has been believed to
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33

Huang, Chun-Ping, Yi-Wen Lin, Der-Yen Lee, and Ching-Liang Hsieh. "Electroacupuncture Relieves CCI-Induced Neuropathic Pain Involving Excitatory and Inhibitory Neurotransmitters." Evidence-Based Complementary and Alternative Medicine 2019 (October 20, 2019): 1–9. http://dx.doi.org/10.1155/2019/6784735.

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Neuropathic pain caused by peripheral tissue injuries to the higher brain regions still has no satisfactory therapy. Disruption of the balance of excitatory and inhibitory neurotransmitters is one of the underlying mechanisms that results in chronic neuropathic pain. Targeting neurotransmitters and related receptors may constitute a novel approach for treating neuropathic pain. We investigated the effects of electroacupuncture (EA) on chronic constriction injury- (CCI-) induced neuropathic pain. The mechanical allodynia and thermal hyperalgesia pain behaviors were relieved by 15 Hz EA but not
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34

El-Naggar, Tarek, María Pilar Gómez-Serranillos, Olga María Palomino, Carmen Arce, and María Emilia Carretero. "Nigella sativaL. Seed Extract Modulates the Neurotransmitter Amino Acids Release in Cultured NeuronsIn Vitro." Journal of Biomedicine and Biotechnology 2010 (2010): 1–8. http://dx.doi.org/10.1155/2010/398312.

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Nigella sativaL. (NS) has been used for medicinal purposes since ancient times. This study aimed to investigate the cytotoxicity of NS dry methanolic extract on cultured cortical neurons and its influence on neurotransmitter release, as well as the presence of excitatory (glutamate and aspartate) and inhibitory amino acids (gamma-aminobutyric acid—GABA—and glycine) in NS extract. Cultured rat cortical neurons were exposed to different times and concentrations of NS dry methanolic extract and cell viability was then determined by a quantitative colorimetric method. NS did not induce any toxicit
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35

Matsumoto, Keigo, Eng H. Lo, Allen R. Pierce, Elkan F. Halpern, and Robert Newcomb. "Secondary Elevation of Extracellular Neurotransmitter Amino Acids in the Reperfusion Phase following Focal Cerebral Ischemia." Journal of Cerebral Blood Flow & Metabolism 16, no. 1 (1996): 114–24. http://dx.doi.org/10.1097/00004647-199601000-00014.

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The purpose of this study was to evaluate amino acid neurotransmitter dynamics in the reperfusion phase after transient cerebral ischemia. In vivo microdialysis was used to measure extracellular amino acid levels in a rabbit model of focal ischemia. During 30 min of transient ischemia (n = 5), small but significant (p < 0.05) increases in glutamate, aspartate, γ-aminobutyric acid (GABA), and taurine were noted. These elevations rapidly returned to baseline levels upon recirculation and remained constant for up to 5.5 h of reperfusion. In rabbits subjected to 2 h of transient ischemia (n = 5
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36

MUSTAFA, ASIF K., PAUL M. KIM, and SOLOMON H. SNYDER. "D-Serine as a putative glial neurotransmitter." Neuron Glia Biology 1, no. 3 (2004): 275–81. http://dx.doi.org/10.1017/s1740925x05000141.

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Abundant recent evidence favors a neurotransmitter/neuromodulator role for D-serine. D-serine is synthesized from L-serine by serine racemase in astrocytic glia that ensheath synapses, especially in regions of the brain that are enriched in NMDA-glutamate receptors. D-serine is more potent than glycine at activating the ‘glycine’ site of these receptors. Moreover, selective degradation of D-serine but not glycine by D-amino acid oxidase markedly reduces NMDA neurotransmission. D-serine appears to be released physiologically in response to activation by glutamate of AMPA-glutamate receptors on
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37

Vermeiren, Yannick, Nathalie Le Bastard, An Van Hemelrijck, Wilhelmus H. Drinkenburg, Sebastiaan Engelborghs, and Peter P. De Deyn. "Behavioral correlates of cerebrospinal fluid amino acid and biogenic amine neurotransmitter alterations in dementia." Alzheimer's & Dementia 9, no. 5 (2012): 488–98. http://dx.doi.org/10.1016/j.jalz.2012.06.010.

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38

Justo, Rayssa, Marcelo Cesar, Edimilson Migowski, and Rafael Cisne. "Relation between vitamins of the b complex, GABA and glutamate, and their role in neurocognitive disorders -Brief review." International Journal of Basic and Applied Sciences 5, no. 4 (2016): 229. http://dx.doi.org/10.14419/ijbas.v5i4.6707.

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Vitamins, especially the water-soluble complex of vitamins B, are highlighted in the daily clinical practice. Numerous studies emphasize the need for supplementation, mainly in groups with deficiency of these vitamins, such as the elderly, pregnant women, children and patients with diseases associates with cognitive disorder. Thiamine (B1), a vitamin of the diet, is an important cofactor for the three key enzymes involved in the citric acid cycle and the pentose phosphate cycle. Pyridoxine (B6) and cobalamin (B12) act in the CNS as a cofactor in the metabolism reactions of homocysteine. Defici
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Meredith, R. M., B. J. McCabe, K. M. Kendrick, and G. Horn. "Amino acid neurotransmitter release and learning: a study of visual imprinting." Neuroscience 126, no. 2 (2004): 249–56. http://dx.doi.org/10.1016/j.neuroscience.2004.03.046.

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40

Kumar, Pratik, David Shukhman, and Scott T. Laughlin. "A photocaged, cyclopropene-containing analog of the amino acid neurotransmitter glutamate." Tetrahedron Letters 57, no. 51 (2016): 5750–52. http://dx.doi.org/10.1016/j.tetlet.2016.10.106.

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41

Fernandes, Maria J. G., M. Sameiro T. Gonçalves, and Susana P. G. Costa. "Neurotransmitter amino acid—oxobenzo[f]benzopyran conjugates: synthesis and photorelease studies." Tetrahedron 64, no. 49 (2008): 11175–79. http://dx.doi.org/10.1016/j.tet.2008.09.050.

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42

Wafford, K. A., and D. B. Sattelle. "Effects of amino acid neurotransmitter candidates on an identified insect motoneurone." Neuroscience Letters 63, no. 2 (1986): 135–40. http://dx.doi.org/10.1016/0304-3940(86)90050-9.

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43

Bianchi, Mauro, Paolo Ferrario, Antonio Clavenna, Gabriele Biella, and Maria Luisa Sotgiu. "Amino Acid Levelsin the Brain of Rats with Peripheral Nerve Constriction or Section." Pain Research and Management 2, no. 1 (1997): 15–18. http://dx.doi.org/10.1155/1997/535639.

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BACKGROUND: Both excitatory and inhibitory amino acids modulate central neuronal activity following peripheral nerve tissue injury.OBJECTIVE: To establish whether mononeuropathy induced by chronic constriction injury or by sectioning of the sciatic nerve affects neurotransmitter amino acid levels in the rat brain areas.METHODS: Glutamine, glutamic acid, gamma-aminobutyric acid (GABA) and aspartic acid in the hindbrain, midbrain and hypothalamus of rats were measured by high performance liquid chromatography 14 days after surgery.RESULTS: After monolateral nerve ligation an increase in glutamin
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Krivopalov, S. A., B. G. Yushkov, M. Yu Bykova, and K. N. Zabegalov. "Gender differences in the pool of free amino acid neurotransmitters in Krushinsky-Molodkina rats." Biomeditsinskaya Khimiya 66, no. 2 (2020): 124–29. http://dx.doi.org/10.18097/pbmc20206602124.

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The study of the role of neurotransmitter systems in the pathogenesis of epilepsy is one of the priorities of epileptology. New data on the functions of free neurotransmitter-like amino acid in the central nervous system are of the greatest importance and determine the prospects for the development of novel effective anticonvulsants. It is widely believed in clinical medicine that epilepsy has distinct gender characteristics. The aim of this study was to investigate the gender peculiarities in the content of neurotransmitter amino acids in the brain of Krushinsky-Molodkina (KM) rats, which wer
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Camargo, José Augusto, and Paulo Henrique Ferreira Bertolucci. "Quantification of Amino Acid Neurotransmitters in Cerebrospinal Fluid of Patients with Neurocysticercosis." Open Neurology Journal 9, no. 1 (2015): 15–20. http://dx.doi.org/10.2174/1874205x01509010015.

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Background : Neurocysticercosis is a parasitic disease that affects the central nervous system. Its main clinical manifestations are epileptic seizures. The objective of this study was to investigate the correlation between neurotransmitter concentrations in cerebrospinal fluid (CSF) and the different evolutive forms of neurocysticercosis with or without seizures. Methods : Neurotransmitter concentrations (Aspartate, Glutamate, GABA, Glutamine, Glycine, Taurine) were determined in CSF samples from 42 patients with neurocysticercosis divided into patients with the active cystic form (n = 24, 12
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46

de Silva, A. Prasanna, H. Q. Nimal Gunaratne, Catherine McVeigh, Glenn E. M. Maguire, Pamela R. S. Maxwell та Emma O'Hanlon. "Fluorescent signalling of the brain neurotransmitter γ-aminobutyric acid and related amino acid zwitterions". Chem. Commun., № 18 (1996): 2191–92. http://dx.doi.org/10.1039/cc9960002191.

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Kim, Yeji, and Jongsung Kim. "Synthesis of Carbon Dots via Hydrothermal Reaction for Selective Detection of Serotonin." Journal of Nanoscience and Nanotechnology 20, no. 9 (2020): 5365–68. http://dx.doi.org/10.1166/jnn.2020.17646.

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Serotonin (5-HT) is a significant neurotransmitter involved in the regulation of mood, social behavior, depression, and other neurological activities. In this work, we synthesized nitrogen-doped carbon dots using naturally occurring amino acids such as L-aspartic acid and L-cysteine by employing a one-step hydrothermal reaction. The amino acid-derived carbon dots (CDs) are called AACDs. The as-synthesized AACDs showed strong blue-color emission under UV light and photoluminescence (PL) emission maximum wavelength at 457 nm with an excitation wavelength of 344 nm. The AACDs had superior aqueous
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48

Hylland, P., G. E. Nilsson, and D. Johansson. "Anoxic brain failure in an ectothermic vertebrate: release of amino acids and K+ in rainbow trout thalamus." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 269, no. 5 (1995): R1077—R1084. http://dx.doi.org/10.1152/ajpregu.1995.269.5.r1077.

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The release of excitatory amino acids such as glutamate contributes greatly to anoxic and/or ischemic brain damage in mammals. However, for anoxia-intolerant ectothermic vertebrates, there has been no information on how anoxia affects extracellular amino acid levels, or how such changes relate temporally to major ion movements. We have investigated the effects of environmental anoxia on extracellular amino acid and K+ concentrations in rainbow trout thalamus in vivo at 15 degrees C, using microdialysis and K(+)-selective microelectrodes. Systemic blood pressure was also monitored. In separate
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Neuray, Caroline, Reza Maroofian, Marcello Scala, et al. "Early-infantile onset epilepsy and developmental delay caused by bi-allelic GAD1 variants." Brain 143, no. 8 (2020): 2388–97. http://dx.doi.org/10.1093/brain/awaa178.

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Abstract Gamma-aminobutyric acid (GABA) and glutamate are the most abundant amino acid neurotransmitters in the brain. GABA, an inhibitory neurotransmitter, is synthesized by glutamic acid decarboxylase (GAD). Its predominant isoform GAD67, contributes up to ∼90% of base-level GABA in the CNS, and is encoded by the GAD1 gene. Disruption of GAD1 results in an imbalance of inhibitory and excitatory neurotransmitters, and as Gad1−/− mice die neonatally of severe cleft palate, it has not been possible to determine any potential neurological dysfunction. Furthermore, little is known about the conse
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Lamers, Karel JB, and Ron A. Wevers. "Abnormalities of biogenic amines affecting the metabolism of serotonin and catecholamines." Multiple Sclerosis Journal 4, no. 1 (1998): 37–38. http://dx.doi.org/10.1177/135245859800400109.

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This paper describes the relevance of measuring biogenic amine metabolites in cerebrospinal fluid in order to detect inborn errors affecting catecholamines and serotonin biosynthesis. Defects in tetrahydrobiopterin and a deficiency of aromatic L-amino acid decarboxylase, tyrosine hydroxylase or dopamine-b-hydroxylase are candidate inborn errors for neurotransmitter matabolites screening. This investigation has to be considered in any child with motor retardation and extrapyramidal signs.
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