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Artículos de revistas sobre el tema "Aortic blood"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 21 de febrero de 2023

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1

Laurent, Stéphane, Pierre Boutouyrie, and Elie Mousseaux. "Aortic Stiffening, Aortic Blood Flow Reversal, and Renal Blood Flow." Hypertension 66, no. 1 (July 2015): 10–12. http://dx.doi.org/10.1161/hypertensionaha.115.05357.

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2

Muchada, Raoul, Dominique Cathignol, Bernard Lavandier, Jean Lamazou, and Dominique Haro. "Aortic Blood Flow Measurement." American Journal of Noninvasive Cardiology 2, no. 1-2 (1988): 24–31. http://dx.doi.org/10.1159/000470655.

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3

Gjurich, Breanne, Parésa Taghavie-Moghadam, Klaus Ley, and Elena Galkina. "L-selectin deficiency decreases aortic B1a and Breg subsets and promotes atherosclerosis." Thrombosis and Haemostasis 112, no. 10 (2014): 803–11. http://dx.doi.org/10.1160/th13-10-0865.

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SummaryThere is a significant recruitment of leucocytes into aortas during atherogenesis. L-selectin regulates leucocyte migration into secondary lymphoid and peripheral tissues and was proposed to play a role in leucocyte homing into aortas. Here, we determine the role of L-selectin in atherosclerosis. L-selectin-deficient Apoe -/- (Sell -/- Apoe -/-) mice had a 74% increase in plaque burden compared to Apoe -/- mice fed a chow diet for 50 weeks. Elevated atherosclerosis was accompanied by increased aortic leucocyte content, but a 50% reduction in aortic B cells despite elevated B cell counts

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4

R, VINOTH. "TRANSIENT ANALYSIS OF BLOOD FLOW IN FUSIFORM MODELS OF AORTIC ANEURYSMS." International Journal of Psychosocial Rehabilitation 24, no. 04 (February 29, 2020): 1450–62. http://dx.doi.org/10.37200/ijpr/v24i4/pr201114.

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5

Lehmann, E. D., K. D. Hopkins, R. L. Jones, A. G. Rudd, and R. G. Gosling. "Aortic Distensibility in Patients with Cerebrovascular Disease." Clinical Science 89, no. 3 (September 1, 1995): 247–53. http://dx.doi.org/10.1042/cs0890247.

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1. Non-invasive aortic compliance measurements have been used previously to assess the distensibility of the aorta in several pathological conditions associated with increased cardiovascular risk. We set out to establish whether aortic compliance is abnormal in patients with stroke. 2. Pulse wave velocity measurements of thoracoabdominal aortic compliance were made in 20 stroke patients and 25 age- and sex-matched hospitalized, non-stroke control subjects putatively free of cardiovascular disease. Since compliance varies with non-chronic changes in blood pressure, a blood pressure corrected in

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6

JONES, DAVID R., RICHARD W. BRILL, and DENNIS C. MENSE. "The Influence of Blood Gas Properties on Gas Tensions and pH of Ventral and Dorsal Aortic Blood in Free-Swimming Tuna, Euthynnus Affinis." Journal of Experimental Biology 120, no. 1 (January 1, 1986): 201–13. http://dx.doi.org/10.1242/jeb.120.1.201.

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We have developed a technique for capture, anaesthetization, instrumentation and release of tuna and have made the first determinations of blood gas values in dorsal and ventral aortae of free-swimming tuna. Dorsal aortic Po2 varied from 34.5 to 91.7 mmHg, and Pcoco2 ranged from 3.7 to 7mmHg. Dorsal aortic blood [pHa = 7.77 ± 0.04 (8), mean ± one S.E.M. (N)] was more alkaline than ventral aortic blood [pHv = 7.65 ± 0.02 (7)]. Warming dorsal aortic blood from 25 to 35 °C in a closed system caused Po2 and PCOCO2 to rise and pH to fall. Oxygen-combining curves forwhole blood were sigmoid [mean Hi

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7

Azam, M. A., and S. A. A. Salam. "Three Dimensional Analysis of the Blood Flow Regime within Abdominal Aortic Aneurysm." International Journal of Engineering and Technology 3, no. 6 (2011): 621–27. http://dx.doi.org/10.7763/ijet.2011.v3.295.

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8

Bulpitt, Christopher J., C. Rajkumar, and James D. Cameron. "Central aortic blood pressure measurements." Journal of Human Hypertension 14, no. 8 (August 2000): 531. http://dx.doi.org/10.1038/sj.jhh.1001066.

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9

Bukoski, R. D., and D. A. McCarron. "Altered aortic reactivity and lowered blood pressure associated with high calcium intake." American Journal of Physiology-Heart and Circulatory Physiology 251, no. 5 (November 1, 1986): H976—H983. http://dx.doi.org/10.1152/ajpheart.1986.251.5.h976.

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The hypothesis that dietary calcium (dCa) alters functional properties of aortic smooth muscle in the spontaneously hypertensive rat (SHR) was tested. At 6 wk of age, Wistar Kyoto (WKY) and (SHR) rats were placed on a control diet containing 1% Ca. The experimental SHR group received a 2%-calcium diet. After an average of either 8 or 15 wk on the diets (WOD), aortic rings were prepared for measurement of passive elastic properties and isometric force development. Differences in blood pressure (BP) were not apparent until after 8 WOD when the BP of SHRs on 2% dCa were 10-15 mmHg lower than SHRs

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10

Zindovic, Igor, Gustaf Edgren, Shahab Nozohoor, and Ammar Majeed. "ABO blood group and the risk of aortic disease: a nationwide cohort study." BMJ Open 10, no. 10 (October 2020): e036040. http://dx.doi.org/10.1136/bmjopen-2019-036040.

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ObjectivesTo analyse the association between ABO blood group and aortic disease using data on blood donors and transfused patients from Sweden.DesignThis was a retrospective study using data from the Swedish portion of the Scandinavian Donations and Transfusions Database. The association between ABO blood group and aortic disease was analysed using log-linear Poisson regression models and presented as incidence rate ratios (IRRs).SettingSwedish population-based study.ParticipantsThe study cohort consisted of 1 164 561 Swedish blood donors and 961 637 transfused patients with a combined follow-

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11

Dziuba, O. S. "Blood coagulation and aortic wall integrity in rats with obesity-induced insulin resistance." Ukrainian Biochemical Journal 90, no. 2 (April 10, 2018): 14–23. http://dx.doi.org/10.15407/ubj90.02.014.

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12

Bogren, Hugo, Michael Buonocore, and David Follette. "Four-Dimensional Aortic Blood Flow Patterns in Thoracic Aortic Grafts." Journal of Cardiovascular Magnetic Resonance 2, no. 3 (2000): 201–8. http://dx.doi.org/10.3109/10976640009146568.

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13

Ye, Dien, Congqing Wu, Hui Chen, Ching-Ling Liang, Deborah A. Howatt, Michael K. Franklin, Jessica J. Moorleghen, et al. "Fludrocortisone Induces Aortic Pathologies in Mice." Biomolecules 12, no. 6 (June 13, 2022): 825. http://dx.doi.org/10.3390/biom12060825.

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Background and Objective: In an experiment designed to explore the mechanisms of fludrocortisone-induced high blood pressure, we serendipitously observed aortic aneurysms in mice infused with fludrocortisone. The purpose of this study was to investigate whether fludrocortisone induces aortic pathologies in both normocholesterolemic and hypercholesterolemic mice. Methods and Results: Male adult C57BL/6J mice were infused with either vehicle (85% polyethylene glycol 400 (PEG-400) and 15% dimethyl sulfoxide (DMSO); n = 5) or fludrocortisone (12 mg/kg/day dissolved in 85% PEG-400 and 15% DMSO; n =

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14

Palmier, Mickael, Quentin Cohen, Benjamin Bottet, and Didier Plissonnier. "Competition between Conflicting Aortic Blood Flows." Journal of Vascular and Interventional Radiology 32, no. 8 (August 2021): 1239. http://dx.doi.org/10.1016/j.jvir.2021.03.547.

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15

Narayan, Om, Joshua Casan, Martin Szarski, Anthony M. Dart, Ian T. Meredith, and James D. Cameron. "Estimation of central aortic blood pressure." Journal of Hypertension 32, no. 9 (September 2014): 1727–40. http://dx.doi.org/10.1097/hjh.0000000000000249.

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16

Orliaguet, Gilles A., and Pierre Y. Gueugniaud. "Non-invasive aortic blood flow measurement." Current Opinion in Anaesthesiology 13, no. 3 (June 2000): 307–12. http://dx.doi.org/10.1097/00001503-200006000-00013.

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17

Takagi, Hisato. "Blood group and abdominal aortic aneurysm." European Journal of Preventive Cardiology 27, no. 19 (September 12, 2019): 2195–99. http://dx.doi.org/10.1177/2047487319876044.

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18

Mitchell, G. F. "Aortic Stiffness and Cerebral Blood Flow." American Journal of Hypertension 24, no. 10 (October 1, 2011): 1056. http://dx.doi.org/10.1038/ajh.2011.112.

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19

Heffernan, Kevin S., James E. Sharman, Eun Sun Yoon, Eui Jin Kim, Su Jin Jung, and Sae Young Jae. "Effect of increased preload on the synthesized aortic blood pressure waveform." Journal of Applied Physiology 109, no. 2 (August 2010): 484–90. http://dx.doi.org/10.1152/japplphysiol.00196.2010.

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In the present study, we examined the influence of preload augmentation via passive leg elevation (PLE) on synthesized aortic blood pressure, aortic augmentation index (AIx), and aortic capacitance (a reflection of aortic reservoir function). Central and peripheral hemodynamics were measured via tonometry with a generalized transfer function in 14 young, healthy men (age = 24 yr). Aortic blood flow was calculated from the left ventricular outflow tract (LVOT) velocity-time integral (VTI) using standard two-dimensional echocardiographic-Doppler techniques. Measures were made in the supine posit

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20

Lam, Chen-Fuh, Timothy E. Peterson, Darcy M. Richardson, Anthony J. Croatt, Livius V. d'Uscio, Karl A. Nath, and Zvonimir S. Katusic. "Increased blood flow causes coordinated upregulation of arterial eNOS and biosynthesis of tetrahydrobiopterin." American Journal of Physiology-Heart and Circulatory Physiology 290, no. 2 (February 2006): H786—H793. http://dx.doi.org/10.1152/ajpheart.00759.2005.

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Shear stress, imposed on the vascular endothelium by circulating blood, critically sustains vascular synthesis of nitric oxide (NO). Endothelial NO synthase (eNOS) activity is determined by heat shock protein 90 (HSP90), caveolin-1, and the cofactor tetrahydrobiopterin (BH4). To determine whether increased blood flow concomitantly upregulates eNOS and GTP cyclohydrolase I (GTPCH I, the rate-limiting enzyme in BH4 biosynthesis), an aortocaval fistula model in the rat was employed wherein aortic blood flow is enhanced proximal but decreased distal to the fistula. Eight weeks after the creation o

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21

Sherrah, Andrew, Fraser Callaghan, Rajesh Puranik, Richmond Jeremy, Paul Bannon, Michael Vallely, and Stuart Grieve. "Multi-Velocity Encoding Four-Dimensional Flow Magnetic Resonance Imaging in the Assessment of Chronic Aortic Dissection." AORTA 05, no. 03 (June 2017): 80–90. http://dx.doi.org/10.12945/j.aorta.2017.16.046.

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Background: Chronic descending thoracic aortic dissection (CDTAD) following surgical repair of ascending aortic dissection requires long-term imaging surveillance. We investigated four-dimensional (4D)-flow magnetic resonance imaging (MRI) with a novel multi-velocity encoding (multi-VENC) technique as an emerging clinical method enabling the dynamic quantification of blood volume and velocity throughout the cardiac cycle. Methods: Patients with CDTAD (n = 10; mean age, 55.1 years; standard deviation (SD) 10.8) and healthy volunteers (n = 9; mean age, 37.1 years; SD 11.4; p < 0.01) underwent

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22

Middeke, Martin. "Zentraler aortaler Blutdruck: Bedeutender Parameter für Diagnostik und Therapie." DMW - Deutsche Medizinische Wochenschrift 142, no. 19 (September 2017): 1430–36. http://dx.doi.org/10.1055/s-0043-113212.

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AbstractIn recent years great emphasis has been placed on the role of central aortic blood pressure as measured non invasively using pulse wave analysis in pathophysiology of cardiovascular diseases and clinical aspects of hypertension. The difference of blood pressure between the central aorta and the brachial artery (amplification) is not constant but varies according to physiological, pathological and pharmacological mechanisms. Central aortic blood pressure is more strongly related to cardiovascular organ damages than does brachial pressure. Several antihypertensive drugs have different ef

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23

Yvernogeau, Laurent, Anna Klaus, Joris Maas, Ismaël Morin-Poulard, Bart Weijts, Stefan Schulte-Merker, Eugene Berezikov, Jan Philipp Junker, and Catherine Robin. "Multispecies RNA tomography reveals regulators of hematopoietic stem cell birth in the embryonic aorta." Blood 136, no. 7 (August 13, 2020): 831–44. http://dx.doi.org/10.1182/blood.2019004446.

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Abstract The defined location of a stem cell within a niche regulates its fate, behavior, and molecular identity via a complex extrinsic regulation that is far from being fully elucidated. To explore the molecular characteristics and key components of the aortic microenvironment, where the first hematopoietic stem cells are generated during development, we performed genome-wide RNA tomography sequencing on zebrafish, chicken, mouse, and human embryos. The resulting anterior-posterior and dorsal-ventral transcriptional maps provided a powerful resource for exploring genes and regulatory pathway

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24

Fatahillah, Arif, Azza Liarista Anggraini, and Susi Setiawani. "Numerical analysis of blood flow in abdominal aortic aneurysm using finite volume method." Desimal: Jurnal Matematika 5, no. 2 (August 30, 2022): 131–42. http://dx.doi.org/10.24042/djm.v5i2.9928.

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There is a deadly cardiovascular disease that can cause swelling of the Abdominal Aorta. This disease is known as Abdominal Aortic Aneurysm (AAA). AAA is believed to be a degenerative process caused by genetic factors, gender, body weight, and age. Changes in collagen and elastin in the aortic wall are the cause of the degeneration process. Therefore, it will cause dilatation of the aortic wall. Swelling of the aortic blood vessels will affect the blood flow velocity in the aortic blood vessels. This research aims to analyze the velocity of blood flow in the Abdominal Aortic Aneurysm based on

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25

Castrejón-Téllez, Vicente, María Esther Rubio-Ruiz, Agustina Cano-Martínez, Israel Pérez-Torres, Leonardo Del Valle-Mondragón, Elizabeth Carreón-Torres, and Verónica Guarner-Lans. "High Sucrose Ingestion during a Critical Period of Vessel Development Promotes the Synthetic Phenotype of Vascular Smooth Muscle Cells and Modifies Vascular Contractility Leading to Hypertension in Adult Rats." International Journal of Hypertension 2022 (June 21, 2022): 1–12. http://dx.doi.org/10.1155/2022/2298329.

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Cardiometabolic diseases, including hypertension, may result from exposure to high sugar diets during critical periods of development. Here, we studied the effect of sucrose ingestion during a critical period (CP) between postnatal days 12 and 28 of the rat on blood pressure, aortic histology, vascular smooth muscle phenotype, expression of metalloproteinases 2 and 9, and vascular contractility in adult rats and compared it with those of adult rats that received sucrose for 6 months and developed metabolic syndrome (MS). Blood pressure increased to a similar level in CP and MS rats. The diamet

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26

FUJI, Motoaki, Haruhisa HURUMOTO, Tsutomu TAJIKAWA, and Kenkichi OHBA. "0225 Influence of blood flow and blood pressure at aortic valve on pathogeny of aortic valve sclerosis." Proceedings of the Bioengineering Conference Annual Meeting of BED/JSME 2009.22 (2010): 209. http://dx.doi.org/10.1299/jsmebio.2009.22.209.

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27

Gonzalez-Urquijo, Mauricio, Raul Garza de Zamacona, Ana Karen Martinez Mendoza, Miranda Zamora Iribarren, Erika Garza Ibarra, Marcos David Moya Bencomo, and Mario Alejandro Fabiani. "3D Modeling of Blood Flow in Simulated Abdominal Aortic Aneurysm." Vascular and Endovascular Surgery 55, no. 7 (April 27, 2021): 677–83. http://dx.doi.org/10.1177/15385744211012926.

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Background: Besides biological factors, abdominal aortic aneurysm rupture is also caused by mechanical parameters, which are constantly affecting the wall’s tissue due to their abnormal values. The ability to evaluate these parameters could vastly improve the clinical treatment of patients with abdominal aortic aneurysms. The objective of this study was to develop and demonstrate a methodology to analyze the fluid dynamics that cause the wall stress distribution in abdominal aortic aneurysms, using accurate 3D geometry and a realistic, nonlinear, elastic biomechanical model using a computer-ai

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Ben Driss, A., J. Benessiano, P. Poitevin, B. I. Levy, and J. B. Michel. "Arterial expansive remodeling induced by high flow rates." American Journal of Physiology-Heart and Circulatory Physiology 272, no. 2 (February 1, 1997): H851—H858. http://dx.doi.org/10.1152/ajpheart.1997.272.2.h851.

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The effects of chronic increase in aortic blood flow on arterial wall remodeling were investigated in vivo with the use of an aortocaval fistula (ACF) model in rats. Phasic hemodynamics and aortic wall structure upstream and downstream in 30 male Wistar rats with ACF and 30 sham-operated rats were compared immediately and 2 mo after the ACF was opened in anesthetized rats. Opening the ACF upstream acutely decreased aortic pressure (-30%, P < 0.001) and increased aortic blood velocity (x12, P < 0.001), blood flow (x9, P < 0.001), wall shear stress (x10, P < 0.001) and guanosine 3',5

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29

Ghorbani, Niky, Vivek Muthurangu, Abbas Khushnood, Leonid Goubergrits, Sarah Nordmeyer, Joao Filipe Fernandes, Chong-Bin Lee, et al. "Impact of valve morphology, hypertension and age on aortic wall properties in patients with coarctation: a two-centre cross-sectional study." BMJ Open 10, no. 3 (March 2020): e034853. http://dx.doi.org/10.1136/bmjopen-2019-034853.

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ObjectiveWe aimed to investigate the combined effects of arterial hypertension, bicuspid aortic valve disease (BAVD) and age on the distensibility of the ascending and descending aortas in patients with aortic coarctation.DesignCross-sectional study.SettingThe study was conducted at two university medical centres, located in Berlin and London.ParticipantsA total of 121 patients with aortic coarctation (ages 1–71 years) underwent cardiac MRI, echocardiography and blood pressure measurements.Outcome measuresCross-sectional diameters of the ascending and descending aortas were assessed to compute

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30

Tian, Wen, Zhijun Mei, Jian Zhou, Qingsheng Lu, and Zaiping Jing. "A neglected event in endovascular repair of aortic dissection: acute blood pressure variability during aortic angiography." International Journal for Innovation Education and Research 6, no. 8 (August 31, 2018): 235–40. http://dx.doi.org/10.31686/ijier.vol6.iss8.1139.

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[Purpose]To investigate acute blood pressure change during aortic angiography in aortic dissection endovascular repair, and analyse the potential risk of this incident.[Method]24 patients with aortic dissection underwent endovascular repair in department of vascular surgery of Changhai hospital between May 2016 and July 2016 were enrolled in this research. Patients were divided into two groups: patients underwent general anesthesia and patients underwent lumbar anesthesia. Blood pressure was monitored by intro-artery catheter. Blood pressure readings were recorded every 10 seconds during the p

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31

Skibitskiy, V. V., A. A. Kiselev, and A. V. Fendrikova. "Effectiveness of Chrono-Pharmacotherapy Depending on the Salt Sensitivity of Patients with Arterial Hypertension and Diabetes Mellitus Type 2." Rational Pharmacotherapy in Cardiology 14, no. 6 (January 5, 2019): 846–51. http://dx.doi.org/10.20996/1819-6446-2018-14-6-846-851.

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Aim. To study the effect of two regimens of combined antihypertensive therapy during the day on daily monitoring of arterial pressure, central aortic pressure, and arterial stiffness, depending on the salt sensitivity of hypertensive patients with diabetes mellitus type 2. Material and methods. 130 hypertensive patients with type 2 diabetes mellitus were included into the study. They were divided into 2 subgroups: salt-sensitive (group 1) and salt-resistant (group 2), and then randomized to subgroups A and B of ongoing therapy: in the morning ramipril and indapamide retard, bedtime – amlodipin

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32

Williams, Bryan, Ewan McFarlane, Dawid Jedrzejewski, and Peter S. Lacy. "Identifying and treating high blood pressure in men under 55 years with grade 1 hypertension: the TREAT CASP study and RCT." Efficacy and Mechanism Evaluation 6, no. 13 (December 2019): 1–90. http://dx.doi.org/10.3310/eme06130.

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Background There is uncertainty regarding whether or not younger (i.e. aged < 55 years), low-risk patients with grade 1 hypertension (i.e. a clinic blood pressure of 140–159/90–99 mmHg) should be treated with blood pressure-lowering medication. This is a heterogeneous group of patients because of variation in systolic/pulse pressure amplification from the central aorta to the brachial artery. It is hypothesised that within grade 1 hypertension, patients can be divided into those with high central aortic systolic pressure and those with low central aortic systolic pressure. Objectives The ai

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33

Axelsson, M., C. Franklin, R. Fritsche, G. Grigg, and S. Nilsson. "The sub-pulmonary conus and the arterial anastomosis as important sites of cardiovascular regulation in the crocodile Crocodylus porosus." Journal of Experimental Biology 200, no. 4 (February 1, 1997): 807–14. http://dx.doi.org/10.1242/jeb.200.4.807.

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We present evidence to support the hypothesis that the arterial anastomosis and the cogteeth-like valves located in the sub-pulmonary conus in the right ventricle are important sites of cardiovascular regulation in the crocodile Crocodylus porosus. The influence of the arterial anastomosis on the development of the 'foramen spike' in the left aortic pressure trace, which occurs at the onset of diastole when the pressures in the right and left aortas become equal, and on gastrointestinal blood flow was examined in unanaesthetised C. porosus using blood vessel occluders. Measurements of blood fl

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34

Petuchova, Aleksandra, and Algirdas Maknickas. "Computational analysis of aortic haemodynamics in the presence of ascending aortic aneurysm." Technology and Health Care 30, no. 1 (December 29, 2021): 187–200. http://dx.doi.org/10.3233/thc-219002.

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BACKGROUND: The usefulness of numerical modelling of a patient’s cardiovascular system is growing in clinical treatment. Understanding blood flow mechanics can be crucial in identifying connections between haemodynamic factors and aortic wall pathologies. OBJECTIVE: This work investigates the haemodynamic parameters of an ascending aorta and ascending aortic aneurysm in humans. METHODS: Two aortic models were constructed from medical images using the SimVascular software. FEM blood flow modelling of cardiac cycle was performed using CFD and CMM-FSI at different vascular wall parameters. RESULT

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35

Song, Chao, Guanyu Yu, Xiang Feng, Rui Feng, Junmin Bao, Zhiqing Zhao, Yifei Pei, Zaiping Jing, and Qingsheng Lu. "Impact of high blood pressure variability on the occurrence of acute type B aortic dissection." Vascular 28, no. 4 (March 26, 2020): 413–20. http://dx.doi.org/10.1177/1708538120902630.

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Background Acute type B aortic dissection is a life-threatening medical emergency, and hypertension is believed to be an important predictor of aortic dissection; the impact of blood pressure variability on the onset and development of aortic dissection has attracted increasing attention. Methods A total of 120 acute type B aortic dissection patients and 57 hypertensive patients without aortic dissection were consecutively enrolled and retrospectively reviewed between January 2013 and November 2015. There were 60 acute type B aortic dissection patients in both high and low blood pressure varia

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Arai, Tatsuya, Kichang Lee, Robert P. Marini, and Richard J. Cohen. "Estimation of changes in instantaneous aortic blood flow by the analysis of arterial blood pressure." Journal of Applied Physiology 112, no. 11 (June 1, 2012): 1832–38. http://dx.doi.org/10.1152/japplphysiol.01565.2011.

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The purpose of this study was to introduce and validate a new algorithm to estimate instantaneous aortic blood flow (ABF) by mathematical analysis of arterial blood pressure (ABP) waveforms. The algorithm is based on an autoregressive with exogenous input (ARX) model. We applied this algorithm to diastolic ABP waveforms to estimate the autoregressive model coefficients by requiring the estimated diastolic flow to be zero. The algorithm incorporating the coefficients was then applied to the entire ABP signal to estimate ABF. The algorithm was applied to six Yorkshire swine data sets over a wide

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37

Ma, Ben, Elaina Melton, Robert Wiener, Ning Zhou, Wenqian Wu, Lo Lai, Charles Wang, Kevin D. Costa, and Hongyu Qiu. "Age and Blood Pressure Contribute to Aortic Cell and Tissue Stiffness Through Distinct Mechanisms." Hypertension 79, no. 8 (August 2022): 1777–88. http://dx.doi.org/10.1161/hypertensionaha.121.18950.

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Background: Aortic stiffening is strongly associated with both aging and hypertension, but the underlying mechanisms remain unclear. We hypothesized that aging-induced aortic stiffness is mediated by a mechanism differing from hypertension. Methods: We conducted comprehensive in vivo and in vitro experiments using multiple rat models to dissect the different mechanisms of aortic stiffening mediated by aging and hypertension. Results: A time-course study in spontaneously hypertensive rats (SHR) and Wistar-Kyoto (WKY) normotensive rats showed more pronounced aging-associated aortic stiffening in

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38

Schnell, Susanne, Danielle A. Smith, Alex J. Barker, Pegah Entezari, Amir R. Honarmand, Maria L. Carr, S. Chris Malaisrie, et al. "Altered aortic shape in bicuspid aortic valve relatives influences blood flow patterns." European Heart Journal – Cardiovascular Imaging 17, no. 11 (July 26, 2016): 1239–47. http://dx.doi.org/10.1093/ehjci/jew149.

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Farag, Emile S., Luca Peroni, Lukas M. Gottwald, Jeroen Vendrik, S. Matthijs Boekholdt, Aart J. Nederveen, Pim van Ooij, Bas AJM de Mol, and Jolanda Kluin. "Bileaflet Mechanical Aortic Valve Prosthesis Orientation Influences Ascending Aortic Blood Flow Patterns." Structural Heart 3, sup1 (April 9, 2019): 12. http://dx.doi.org/10.1080/24748706.2019.1587948.

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40

Hartley, C. J., L. H. Michael, and M. L. Entman. "Noninvasive measurement of ascending aortic blood velocity in mice." American Journal of Physiology-Heart and Circulatory Physiology 268, no. 1 (January 1, 1995): H499—H505. http://dx.doi.org/10.1152/ajpheart.1995.268.1.h499.

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Mice are useful models in numerous research protocols, but monitoring cardiovascular parameters in small animals is difficult. Therefore we evaluated the use of 20-MHz pulsed Doppler ultrasound to measure ascending aortic blood velocity in intact anesthetized mice. Using a 0.5-mm-diameter 20-MHz transducer applied to the right sternal border, we recorded audio Doppler signals from the ascending aorta of 31 mice [24.4 +/- 1.5 (SD) g body wt]. The signals were played back at speed into a fast Fourier transform analyzer from which we measured heart rate (453 +/- 96 beats/min), ejection time (38 +

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MINAMOTO, Yukiko, Masaki WAKAMATSU, and Fukuichiro OKUMURA. "Autologous blood transfusion during abdominal aortic reconstruction." JOURNAL OF JAPAN SOCIETY FOR CLINICAL ANESTHESIA 10, no. 2 (1990): 180–85. http://dx.doi.org/10.2199/jjsca.10.180.

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Schoenhagen, Paul. "IMH aortic arch with mediastinal blood products." ASVIDE 5 (March 2018): 251. http://dx.doi.org/10.21037/asvide.2018.251.

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Colson, Pascal, Jacques Séguin, Bernard Roquefeuil, and Vaul-André Chaptal. "Transesophageal Doppler to Evaluate Aortic Blood Flow." Chest 96, no. 4 (October 1989): 962. http://dx.doi.org/10.1378/chest.96.4.962-a.

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Hashimoto, Junichiro, and Sadayoshi Ito. "Aortic Blood Flow Reversal Determines Renal Function." Hypertension 66, no. 1 (July 2015): 61–67. http://dx.doi.org/10.1161/hypertensionaha.115.05236.

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Viklander, Gertrud, Jonas Wallinder, and Anders E. Henriksson. "ABO blood groups and abdominal aortic aneurysm." Transfusion and Apheresis Science 47, no. 3 (December 2012): 351–53. http://dx.doi.org/10.1016/j.transci.2012.07.016.

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Witteman, JacquelineC M., DiederickE Grobbee, and Albert Hofman. "Relation between aortic atherosclerosis and blood pressure." Lancet 343, no. 8913 (June 1994): 1649. http://dx.doi.org/10.1016/s0140-6736(94)93108-9.

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DeGroff, Curt, Michael Silberbach, David J. Sahn, and Paul Droukas. "Giant blood cyst of the aortic valve." Journal of the American Society of Echocardiography 8, no. 4 (July 1995): 543–45. http://dx.doi.org/10.1016/s0894-7317(05)80343-8.

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Swiers, Gemma, Nancy A. Speck, and Marella F. T. R. de Bruijn. "Visualizing Blood Cell Emergence from Aortic Endothelium." Cell Stem Cell 6, no. 4 (April 2010): 289–90. http://dx.doi.org/10.1016/j.stem.2010.03.007.

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Narayan, Om, Kim H. Parker, Justin E. Davies, Alun D. Hughes, Ian T. Meredith, and James D. Cameron. "Reservoir pressure analysis of aortic blood pressure." Journal of Hypertension 35, no. 10 (October 2017): 2025–33. http://dx.doi.org/10.1097/hjh.0000000000001424.

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Lehmann, Ed, K. D. Hopkins, R. G. Gosling, J. M. Cruickshank, Margaret Mac Mahon, Noirin Sheahan, J. F. Malone, and Davis Coakley. "Relation between aortic atherosclerosis and blood pressure." Lancet 343, no. 8905 (April 1994): 1106–7. http://dx.doi.org/10.1016/s0140-6736(94)90222-4.

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