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Artículos de revistas sobre el tema "Autonomic nervous system. Bone Diabetes"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 3 de febrero de 2022

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1

Miyasaka, Naoyuki, Mihoko Akiyoshi y Toshiro Kubota. "Relationship between autonomic nervous system activity and bone mineral density in non-medicated perimenopausal women". Journal of Bone and Mineral Metabolism 32, n.º 5 (8 de noviembre de 2013): 588–92. http://dx.doi.org/10.1007/s00774-013-0534-x.

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2

Ion, Daniela–Elena. "Treading on tricky ground: reconstructive approaches to Charcot neuropathic arthropathy of the foot". Romanian Journal of Orthopaedic Surgery and Traumatology 1, n.º 2 (1 de diciembre de 2018): 112–20. http://dx.doi.org/10.2478/rojost-2018-0089.

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Abstract Introduction and purpose:Charcot neuroarthropathy defines a cluster of progressive lesions affecting the joints and bones, as well as the soft tissues of the foot in the context of diabetes, a pivotal role being attributed to peripheral neuropathy. Loss of sensation and proprioception, subsequent repeated trauma, muscle and autonomic nervous system impairment contribute to the alteration of the foot’s architecture and distribution of pressure, ultimately triggering ulceration and gangrene. The urge to avoid amputation has fueled the development of conservative and reconstructive techniques capable of delaying, if not preventing such negative outcomes. The purpose of this review was to present the most frequently used reconstruction procedures and the challenges arising in adapting them to particular foot morphologies and lesion stages. Methods:Literature search was conducted using PubMed, resulting in around 90 articles, multicenter studies and reviews, 26 of which were considered most relevant in providing the guidelines for orthopedic reconstruction and postoperative care in Charcot foot patients with diabetic neuropathy prevailing over arteriopathy. Results:The tarsometatarsal and metatarsophalangeal joints are most frequently affected. Closed reduction, arthrodesis, and tendon lengthening are key features of an efficient correction, alternatively accompanied by resections and tenotomies. Ulceration and callus debridement may also be necessary, while prolonged casting and immobilization remain obligatory. Conclusions:Most authors agree that stabilizing the deformities, optimizing the pressure on the soft tissues, and promoting the healing of potential lesions are the main purposes of the interventions. Prompt recognition and correction of Charcot foot deformities improve life quality and minimize the prospects of amputation.
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Rossi, Marco, Luigi Ricordi, Emilio Mevio, Gabriele Fornasari, Cesare Orlandi, Pietro Fratino, Giorgio Finardi y Luciano Bernardi. "Autonomic nervous system and microcirculation in diabetes". Journal of the Autonomic Nervous System 30 (julio de 1990): S133—S135. http://dx.doi.org/10.1016/0165-1838(90)90117-2.

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Elefteriou, Florent. "Impact of the Autonomic Nervous System on the Skeleton". Physiological Reviews 98, n.º 3 (1 de julio de 2018): 1083–112. http://dx.doi.org/10.1152/physrev.00014.2017.

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It is from the discovery of leptin and the central nervous system as a regulator of bone remodeling that the presence of autonomic nerves within the skeleton transitioned from a mere histological observation to the mechanism whereby neurons of the central nervous system communicate with cells of the bone microenvironment and regulate bone homeostasis. This shift in paradigm sparked new preclinical and clinical investigations aimed at defining the contribution of sympathetic, parasympathetic, and sensory nerves to the process of bone development, bone mass accrual, bone remodeling, and cancer metastasis. The aim of this article is to review the data that led to the current understanding of the interactions between the autonomic and skeletal systems and to present a critical appraisal of the literature, bringing forth a schema that can put into physiological and clinical context the main genetic and pharmacological observations pointing to the existence of an autonomic control of skeletal homeostasis. The different types of nerves found in the skeleton, their functional interactions with bone cells, their impact on bone development, bone mass accrual and remodeling, and the possible clinical or pathophysiological relevance of these findings are discussed.
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5

Horn, Charles C., Jeffrey L. Ardell y Lee E. Fisher. "Electroceutical Targeting of the Autonomic Nervous System". Physiology 34, n.º 2 (1 de marzo de 2019): 150–62. http://dx.doi.org/10.1152/physiol.00030.2018.

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Autonomic nerves are attractive targets for medical therapies using electroceutical devices because of the potential for selective control and few side effects. These devices use novel materials, electrode configurations, stimulation patterns, and closed-loop control to treat heart failure, hypertension, gastrointestinal and bladder diseases, obesity/diabetes, and inflammatory disorders. Critical to progress is a mechanistic understanding of multi-level controls of target organs, disease adaptation, and impact of neuromodulation to restore organ function.
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6

Millar, Philip J. y John S. Floras. "Statins and the autonomic nervous system". Clinical Science 126, n.º 6 (15 de noviembre de 2013): 401–15. http://dx.doi.org/10.1042/cs20130332.

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Statins (3-hydroxy-3-methylglutaryl-CoA reductase inhibitors) reduce plasma cholesterol and improve endothelium-dependent vasodilation, inflammation and oxidative stress. A ‘pleiotropic’ property of statins receiving less attention is their effect on the autonomic nervous system. Increased central sympathetic outflow and diminished cardiac vagal tone are disturbances characteristic of a range of cardiovascular conditions for which statins are now prescribed routinely to reduce cardiovascular events: following myocardial infarction, and in hypertension, chronic kidney disease, heart failure and diabetes. The purpose of the present review is to synthesize contemporary evidence that statins can improve autonomic circulatory regulation. In experimental preparations, high-dose lipophilic statins have been shown to reduce adrenergic outflow by attenuating oxidative stress in central brain regions involved in sympathetic and parasympathetic discharge induction and modulation. In patients with hypertension, chronic kidney disease and heart failure, lipophilic statins, such as simvastatin or atorvastatin, have been shown to reduce MNSA (muscle sympathetic nerve activity) by 12–30%. Reports concerning the effect of statin therapy on HRV (heart rate variability) are less consistent. Because of their implications for BP (blood pressure) control, insulin sensitivity, arrhythmogenesis and sudden cardiac death, these autonomic nervous system actions should be considered additional mechanisms by which statins lower cardiovascular risk.
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Hreidarsson, A. B. y H. J. G. Gundersen. "Reduced Pupillary Unrest: Autonomic Nervous System Abnormality in Diabetes Mellitus". Diabetes 37, n.º 4 (1 de abril de 1988): 446–51. http://dx.doi.org/10.2337/diab.37.4.446.

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Hreidarsson, A. B. y H. J. Gundersen. "Reduced pupillary unrest. Autonomic nervous system abnormality in diabetes mellitus". Diabetes 37, n.º 4 (1 de abril de 1988): 446–51. http://dx.doi.org/10.2337/diabetes.37.4.446.

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9

Irigoyen, M. C., Dulce Elena Casarini, Mariana Morris y Nicola Montano. "Autonomic Nervous System, Inflammation, and Diabetes: Mechanisms and Possible Interventions". Experimental Diabetes Research 2012 (2012): 1–2. http://dx.doi.org/10.1155/2012/894157.

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10

Meretskyy, V. M. y M. M. Korda. "The features of neuroendocrine changes in cranial injury associated with diabetes mellitus". Kazan medical journal 94, n.º 4 (15 de diciembre de 2013): 560–65. http://dx.doi.org/10.17816/kmj1971.

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Aim. To study the features of neuroendocrine regulation in cranial injury associated with diabetes mellitus. Methods. Experiments were carried out on 100 male white rats that were divided into the following groups: the first group (n=10) - control group consisted of 10 intact animals, the second group (n=40) - rats with simulated traumatic cranial injury, the third group (n=10) - rats with experimental diabetes mellitus, the fourth group (n=40) - rats with simulated traumatic cranial injury and experimental diabetes mellitus. Experimental diabetes mellitus was induced by a single streptozotocin solution intraperitoneal injection. Animals were withdrawn from the experiment at 3, 24 hours, 5 and 14 days after the traumatic cranial injury. The mathematical analysis of the cardiac rhythm variability was performed by ECG intervals data analysis. Corticosterone serum levels were measured using ELISA. Results. Based on a comprehensive ECG intervals data analysis, it was found that after experimental traumatic cranial injury the imbalance of autonomic nervous system with increased sympathetic autonomic nervous system function occurs, manifesting as a deregulation of cholinergic and adrenergic effects on sinoatrial node. In diabetes, an increase of the role of sympathetic effects on the sinus node functioning also occurred. Revealed autonomic nervous system deregulation after experimental traumatic cranial injury associated with experimental diabetes mellitus were associated with sympathetic autonomic nervous system over-function, parasympathetic autonomic nervous system exhausting and decreased recovery speed. There was a relevant increase in corticosterone serum concentration reaching its maximum 24 hours after traumatic cranial injury. Conclusion. A significant autonomic nervous system sympathetic shift and autonomic regulation intensifying characterizes the clinical course in traumatic cranial injury associated with diabetes mellitus, a positive correlation of corticosterone serum concentration and sympathetic autonomic nervous system over-function was revealed in animals with experimental traumatic cranial injury, particularly when associated with experimental diabetes mellitus.
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11

Surwit, R. S. y M. N. Feinglos. "Stress and Autonomic Nervous System in Type II Diabetes: A Hypothesis". Diabetes Care 11, n.º 1 (1 de enero de 1988): 83–85. http://dx.doi.org/10.2337/diacare.11.1.83.

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12

Bruinstroop, Eveline, Susanne E. la Fleur, Mariette T. Ackermans, Ewout Foppen, Joke Wortel, Sander Kooijman, Jimmy F. P. Berbée, Patrick C. N. Rensen, Eric Fliers y Andries Kalsbeek. "The autonomic nervous system regulates postprandial hepatic lipid metabolism". American Journal of Physiology-Endocrinology and Metabolism 304, n.º 10 (15 de mayo de 2013): E1089—E1096. http://dx.doi.org/10.1152/ajpendo.00614.2012.

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The liver is a key organ in controlling glucose and lipid metabolism during feeding and fasting. In addition to hormones and nutrients, inputs from the autonomic nervous system are also involved in fine-tuning hepatic metabolic regulation. Previously, we have shown in rats that during fasting an intact sympathetic innervation of the liver is essential to maintain the secretion of triglycerides by the liver. In the current study, we hypothesized that in the postprandial condition the parasympathetic input to the liver inhibits hepatic VLDL-TG secretion. To test our hypothesis, we determined the effect of selective surgical hepatic denervations on triglyceride metabolism after a meal in male Wistar rats. We report that postprandial plasma triglyceride concentrations were significantly elevated in parasympathetically denervated rats compared with control rats ( P = 0.008), and VLDL-TG production tended to be increased ( P = 0.066). Sympathetically denervated rats also showed a small rise in postprandial triglyceride concentrations ( P = 0.045). On the other hand, in rats fed on a six-meals-a-day schedule for several weeks, a parasympathetic denervation resulted in >70% higher plasma triglycerides during the day ( P = 0.001), whereas a sympathetic denervation had no effect. Our results show that abolishing the parasympathetic input to the liver results in increased plasma triglyceride levels during postprandial conditions.
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13

Birajdar, Sidheshwar Virbhadraappa, Sheshrao Sakharam Chavan, Sanjay A. Munde y Yuvraj P. Bende. "A Study of autonomic nervous system dysfunction among patient with diabetes mellitus: a cross sectional study". International Journal of Advances in Medicine 4, n.º 2 (23 de marzo de 2017): 406. http://dx.doi.org/10.18203/2349-3933.ijam20170967.

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Background: Neuropathy is a common complication of diabetes mellitus and it may affect both the peripheral nerves and autonomic nervous system. It’s prevalence ranges from 1% to 90%. The present study is therefore designed to investigate autonomic nervous system involvement in diabetes mellitus by using simple bedside tests and to study its association with other diabetic angiopathies.Methods: 100 patients of diabetes mellitus were selected in the study. In Autonomic function tests for evaluating parasympathetic damage E: I ratio, 30:75 ratio and Valsalva ratio test was performed. Sympathetic damage was diagnosed by Blood pressure response to standing test and Blood pressure response to sustained handgrip test.Results: Abnormal E:I ratio was noticed in only 24 patients. The 30:15 ratio was found to be abnormal in 38 patients while the Valsalva Ratio was abnormal in 34 patients. Postural hypotension was observed in only 8% patients. The sustained hand grip test was abnormal in 10 patients. The prevalence of cardiac autonomic neuropathy was 58%. The association between the presence of autonomic neuropathy and peripheral neuropathy was statistically significant. However, the association between autonomic neuropathy and retinopathy and nephropathy was not statistically significant.Conclusions: The overall prevalence of autonomic neuropathy in diabetes mellitus was 58%. There was parasympathetic preponderance over sympathetic nervous system in the involvement of diabetic autonomic neuropathy. There was statistically significant association of diabetic autonomic neuropathy with peripheral neuropathy as compared to retinopathy and nephropathy.
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14

Parish, Roy C. "Diabetic Autonomic Neuropathy". Journal of Pharmacy Practice 12, n.º 2 (abril de 1999): 142–54. http://dx.doi.org/10.1177/089719009901200207.

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Diabetic autonomic neuropathy (DAN) occurs in approximately half of Caucasian patients with diabetes and perhaps three-fourths of black diabetic patients. This may be asymptomatic for several years, but the majority of patients with DAN eventually exhibit symptoms of diarrhea, cardiac arrhythmias, sexual dysfunction, and abnormal sweating. Prolonged hyperglycemia results in damage to the autonomic nervous system (ANS), particularly the vagus nerve and other parts of the parasympathetic division. DAN is associated with increased risk of sudden death, high-risk cardiac arrhythmias, myocardial infarction, and death from other causes. Objective testing of autonomic nervous system function yields specific information that affects treatment decisions. Drug therapy can effect improvements in ANS function and reduce these risks. Complications of diabetes that result from ANS dysfunction can be partly reversed or their progress can be slowed by appropriate drug therapy. Features, implications, and therapy of the most common complications resulting from DAN are reviewed, and suggestions for pharmacist involvement in the care of these difficult patients are offered.
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15

Zhhilova, N. "Investigation of the autonomic nervous system in patients with chronic heart failure and chronic cerebral ischemia". East European Journal of Neurology, n.º 3(15) (20 de diciembre de 2018): 36–39. http://dx.doi.org/10.33444/2411-5797.2017.3(15).36-39.

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The number of scientific studies which proving that the cause of many chronic diseases is the autonomic nervous system dysfunction has increased. The changes in the autonomic nervous system can be detected before the appearance of clinical symptoms. This is the basis of prevention. Reducing the influence of the parasympathetic nervous system and activating the sympathetic nervous system are predictors of arterial hypertension, cardiovascular diseases, diabetes and others. There are no clear mechanisms for the emergence of hyperactivity of the sympathetic nervous system in cardiovascular pathology. That is why it needs to be studied. And that is why the study of the influence of the autonomic nervous system in chronic conditions is relevant.
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16

Karki, DB, S. Acharya, P. Shrestha, S. Pant, A. Pokhrel y P. Bista. "Autonomic nervous system evaluation in Type 2 diabetic patients and effect of its duration". Journal of Kathmandu Medical College 1, n.º 2 (26 de mayo de 2013): 77–83. http://dx.doi.org/10.3126/jkmc.v1i2.8142.

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Introduction: Dysfunction of the autonomic nervous system is common in diabetic patients. Presence of autonomic dysfunction should alert the physicians of its serious consequences that require timely preventive measures. Objectives: This study was done to fi nd out the autonomic nervous system involvement in Type 2 diabetic patients and to see its relation with the duration of diabetes. Methods: This was a hospital based cross-sectional study. All consecutive diabetic patients of both genders attending Kathmandu Medical College and a private clinic, Temple of Healing were included. A battery of six well validated and accepted autonomic nervous system evaluation tests were done to evaluate the autonomic function in 245 Type 2 diabetic patients. Results: Prevalence of autonomic dysfunction was found to be 71.02% with 58.78% patients having mild autonomic dysfunction and 12.24% having moderate dysfunction. Severe autonomic dysfunction was detected in none of the patients. Parasympathetic dysfunction was much more common than sympathetic dysfunction (68.16% vs. 17.96%). No association was found between autonomic dysfunction and duration of diabetes. Conclusions: Autonomic dysfunction is very common in diabetics and can be detected by simple tests. Timely detection of autonomic dysfunction in diabetic patients is advised so that its preventive measures can be effective. DOI: http://dx.doi.org/10.3126/jkmc.v1i2.8142 Journal of Kathmandu Medical College, Vol. 1, No. 2, Oct.-Dec., 2012: 77-83
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17

Gardim, Camila Balsamo, Bruno Affonso P. de Oliveira, Aline Fernanda B. Bernardo, Rayana Loch Gomes, Francis Lopes Pacagnelli, Roselene Modolo R. Lorençoni y Luiz Carlos M. Vanderlei. "Heart rate variability in children with type 1 diabetes mellitus". Revista Paulista de Pediatria 32, n.º 2 (junio de 2014): 279–85. http://dx.doi.org/10.1590/0103-0582201432215513.

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OBJECTIVE:To gather current information about the effects of type 1 diabetes mellitus on children's cardiac autonomic behavior.DATA SOURCES: The search of articles was conducted on PubMed, Ibecs, Medline, Cochrane, Lilacs, SciELO and PEDro databases using the MeSH terms: "autonomic nervous system", "diabetes mellitus", "child", "type 1 diabetes mellitus", "sympathetic nervous system" and "parasympathetic nervous system", and their respective versions in Portuguese (DeCS). Articles published from January 2003 to February 2013 that enrolled children with 9-12 years old with type 1 diabetes mellitus were included in the review.DATA SYNTHESIS: The electronic search resulted in four articles that approached the heart rate variability in children with type 1 diabetes mellitus, showing that, in general, these children present decreased global heart rate variability and vagal activity. The practice of physical activity promoted benefits for these individuals.CONCLUSIONS: Children with type 1 diabetes mellitus present changes on autonomic modulation, indicating the need for early attention to avoid future complications in this group.
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Sharath shanmugam, Oshin mantro, Jagadeesan M, Mariraj I, Prasanna Karthik S, Gowrishankar, Magesh Kumar y Vinoth Kanna S. "A Study On Assessment Of Autonomic Nervous System Function In Patients With Type 2 Diabetes Mellitus". International Journal of Research in Pharmaceutical Sciences 12, n.º 1 (13 de enero de 2021): 699–702. http://dx.doi.org/10.26452/ijrps.v12i1.4163.

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The autonomic nervous system (ANS) innervates the entire neuraxis and influences the functions of all organs. This study was undertaken for evaluating the autonomic dysfunction in diabetic patients using clinical autonomic tests and neuro- electrophysiology. A prospective study was carried out in 66 patients with type II diabetes mellitus in a tertiary care hospital for one year. Systemic examination, necessary investigations, nerve conduction study and clinical testing for the autonomic nervous system were done. The results were noted and analyzed. 65.2% were females, whereas 34.8% were males. Mean duration of diabetes was found to be 9.06 years (SD 4.121). 80.3% population was known to have type 2 diabetes for 5-10 years duration, 13.6% had diabetes for 10-15 years, and only 3.5% had diabetes for more than 15 years. Mean FBS was found to be 196.12(mg/dl) ±77.180 SD and mean PPBS was 303.26(mg/dl) ± 115.385 SD. Mean HbA1c levels were 10.95 ± 2.36 SD. 33.3% showed early parasympathetic involvement for cardiac autonomic neuropathy, 9% had definite parasympathetic involvement, and only 6% had both parasympathetic and sympathetic involvement. 62.12% showed abnormal responses in nerve conduction study, of which 48.78% had autonomic dysfunction. The main factor responsible for the development and progression of autonomic dysfunction is poor glycaemic status. If contributing factors can be detected, early identification of cardiac autonomic neuropathy (CAN) and appropriate management would halt its progression. Aggressive glycaemic monitoring and treatment shall bring down the progression and prolong the time interval in showing abnormal responses in autonomic function testing.
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Pénicaud, Luc, Beatrice Cousin, Corinne Leloup, Anne Lorsignol y Louis Casteilla. "The autonomic nervous system, adipose tissue plasticity, and energy balance". Nutrition 16, n.º 10 (octubre de 2000): 903–8. http://dx.doi.org/10.1016/s0899-9007(00)00427-5.

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Yasuda, Koichiro, Tetsuro Matsunaga, Tetsuya Adachi, Norihiko Aoki, Gozoh Tsujimoto y Kinsuke Tsuda. "Adrenergic receptor polymorphisms and autonomic nervous system function in human obesity". Trends in Endocrinology & Metabolism 17, n.º 7 (septiembre de 2006): 269–75. http://dx.doi.org/10.1016/j.tem.2006.07.001.

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21

BINZONI, TIZIANO y DIMITRI VAN DE VILLE. "NONINVASIVE PROBING OF THE NEUROVASCULAR SYSTEM IN HUMAN BONE/BONE MARROW USING NEAR-INFRARED LIGHT". Journal of Innovative Optical Health Sciences 04, n.º 02 (abril de 2011): 183–89. http://dx.doi.org/10.1142/s1793545811001447.

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Understanding the mechanisms of interaction between bone/bone marrow, circulatory system and nervous system is of great interest due to the potential clinical impact. In humans, the amount of knowledge in this domain remains relatively limited due to the extreme difficulty to monitor these tissues continuously, noninvasively and for long or repeated periods of time. A typical difficult task would be, for example, to continuously monitor bone/bone marrow blood perfusion, hemoglobin oxygen saturation or blood volume and study their dependence on the activity of the autonomic nervous system. In this review article, we want to show that near-infrared light might be utilized to solve these problems in part. We hope that the present analysis will stimulate future studies in this domain, for which near-infrared light appears as the best available technology today.
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22

Hilsted, J., H. Frandsen, J. J. Holst, N. J. Christensen y S. L. Nielsen. "Plasma glucagon and glucose recovery after hypoglycemia: The effect of total autonomic blockade". Acta Endocrinologica 125, n.º 4 (octubre de 1991): 466–69. http://dx.doi.org/10.1530/acta.0.1250466.

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Abstract. The role of the autonomic nervous system in the glucagon response to hypoglycemia has not been fully clarified. We have studied the effect of total pharmacological blockade of the autonomic nervous system (concomitant α- and β- adrenergic blockade with simultaneous atropine injection) and of isolated α-adrenergic blockade on hormonal responses to hypoglycemia and on blood glucose recovery after hypoglycemia in healthy subjetcs. Neither of the pharmacological blockades had any significant effects on plasma glucagon responses to hypoglycemia nor had they any effect on the rate of blood glucose recovery after hypoglycemia. We conclude that the autonomic nervous system has no major influence on the glucagon response to hypoglycemia in healthy man. Changes in autonomic nervous activity are not essential for blood glucose recovery after hypoglycemia in healthy man.
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Hirsch, Jules y Ronald M. Mackintosh. "Measuring Activity of the Autonomic Nervous System in Humans". Obesity Research 11, n.º 1 (enero de 2003): 2–4. http://dx.doi.org/10.1038/oby.2003.2.

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Aytemir, Kudret, Serdar Aksöyek, Necla Özer, Alper Gürlek y Ali Oto. "QT dispersion and autonomic nervous system function in patients with type 1 diabetes". International Journal of Cardiology 65, n.º 1 (junio de 1998): 45–50. http://dx.doi.org/10.1016/s0167-5273(98)00091-6.

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Stroud, C. R., S. R. Heller, J. D. Ward, C. A. Hardisty y A. P. Weetman. "Analysis of antibodies against components of the autonomic nervous system in diabetes mellitus". QJM 90, n.º 9 (1 de septiembre de 1997): 577–85. http://dx.doi.org/10.1093/qjmed/90.9.577.

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Pöyhönen-Alho, M., M. Viitasalo, M. G. Nicholls, B. M. Lindström, H. Väänänen y R. Kaaja. "Imbalance of the autonomic nervous system at night in women with gestational diabetes". Diabetic Medicine 27, n.º 9 (18 de agosto de 2010): 988–94. http://dx.doi.org/10.1111/j.1464-5491.2010.03062.x.

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Cohen, Michal, Catriona Syme, Brian W. McCrindle y Jill Hamilton. "Autonomic nervous system balance in children and adolescents with craniopharyngioma and hypothalamic obesity". European Journal of Endocrinology 168, n.º 6 (junio de 2013): 845–52. http://dx.doi.org/10.1530/eje-12-1082.

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ObjectiveDysregulation of the autonomic nervous system is thought to be involved in craniopharyngioma-related hypothalamic obesity (CRHO). Increased parasympathetic activity and decreased sympathetic activity have been suggested. We aimed to study autonomic activity using heart rate variability (HRV) and biochemical measures in youth with CRHO compared with controls and to explore relationships between obesity and autonomic indices.DesignA cross-sectional study of 16 youth with CRHO and 16 controls matched for sex, age, and BMI.MethodsAnthropometrics, fasting blood-work, resting energy expenditure (REE), 24-h HRV, and 24-h urine catecholamines were assessed. Quality of life, sleepiness, and autonomic symptoms were evaluated. Power spectral analysis of the HRV was performed.ResultsHRV power spectral analysis parameters of both parasympathetic activity (mean high frequency (HF (ms2)) 611±504 vs 459±336,P=0.325) and sympathetic activity (median low frequency/HF 1.62 (1.37, 2.41) vs 1.89 (1.44, 2.99),P=0.650) did not differ between the groups. Parasympathetic activity negatively correlated with central adiposity in both groups (r=−0.53,P=0.034 andr=−0.54,P=0.029) and sympathetic activity positively correlated with central adiposity in CRHO (r=0.51,P=0.043). Youth with CRHO had significantly lower REE; lower health and activity scores in the quality of life questionnaires, and higher sleepiness scores.ConclusionsAutonomic activity was similar in CRHO and control subjects. The degree of central adiposity correlated negatively with parasympathetic activity and positively with sympathetic activity in children with CRHO. These results provide a new perspective regarding autonomic balance in this unique patient population.
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Brown, F. M., M. Watts y S. L. Rabinowe. "Aggregation of subclinical autonomic nervous system dysfunction and autoantibodies in families with type I diabetes". Diabetes 40, n.º 12 (1 de diciembre de 1991): 1611–14. http://dx.doi.org/10.2337/diabetes.40.12.1611.

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Han, Cheng, Matthew W. Rice y Dongsheng Cai. "Neuroinflammatory and autonomic mechanisms in diabetes and hypertension". American Journal of Physiology-Endocrinology and Metabolism 311, n.º 1 (1 de julio de 2016): E32—E41. http://dx.doi.org/10.1152/ajpendo.00012.2016.

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Interdisciplinary studies in the research fields of endocrinology and immunology show that obesity-associated overnutrition leads to neuroinflammatory molecular changes, in particular in the hypothalamus, chronically causing various disorders known as elements of metabolic syndrome. In this process, neural or hypothalamic inflammation impairs the neuroendocrine and autonomic regulation of the brain over blood pressure and glucose homeostasis as well as insulin secretion, and elevated sympathetic activation has been appreciated as a critical mediator. This review describes the involved physiology and mechanisms, with a focus on glucose and blood pressure balance, and suggests that neuroinflammation employs the autonomic nervous system to mediate the development of diabetes and hypertension.
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Jeanrenaud, B. "Insulin, corticosterone and the autonomic nervous system in animal obesities: a viewpoint". Diabetologia 38, n.º 8 (agosto de 1995): 998–1002. http://dx.doi.org/10.1007/bf00400592.

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Jeanrenaud, B. "Insulin, corticosterone and the autonomic nervous system in animal obesities: a viewpoint". Diabetologia 38, n.º 8 (1 de julio de 1995): 998–1002. http://dx.doi.org/10.1007/s001250050384.

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W. Porges, Stephen. "LOVE: AN EMERGENT PROPERTY OF THE MAMMALIAN AUTONOMIC NERVOUS SYSTEM". Psychoneuroendocrinology 23, n.º 8 (noviembre de 1998): 837–61. http://dx.doi.org/10.1016/s0306-4530(98)00057-2.

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33

Alen, Nicholas V., LillyBelle K. Deer y Camelia E. Hostinar. "Autonomic nervous system activity predicts increasing serum cytokines in children". Psychoneuroendocrinology 119 (septiembre de 2020): 104745. http://dx.doi.org/10.1016/j.psyneuen.2020.104745.

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34

Carnethon, Mercedes R., Sherita H. Golden, Aaron R. Folsom, William Haskell y Duanping Liao. "Prospective Investigation of Autonomic Nervous System Function and the Development of Type 2 Diabetes". Circulation 107, n.º 17 (6 de mayo de 2003): 2190–95. http://dx.doi.org/10.1161/01.cir.0000066324.74807.95.

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35

Hägglund, Harriet, Arja Uusitalo, Juha E. Peltonen, Anne S. Koponen, Jyrki Aho, Katri Pullinen, Ari Suojalehto, Marja Päivinen y Heikki O. Tikkanen. "Dissociation of Cardiac Autonomic Nervous System Function and Aerobic Capacity in Type 1 Diabetes". Medicine & Science in Sports & Exercise 42 (mayo de 2010): 539–40. http://dx.doi.org/10.1249/01.mss.0000385327.54538.06.

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36

Eguchi, Kazuo. "Disrupted diurnal BP variation and cardiovascular disease in diabetes: Relation to autonomic nervous system". Autonomic Neuroscience 135, n.º 1-2 (septiembre de 2007): 34–35. http://dx.doi.org/10.1016/j.autneu.2007.06.039.

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37

Carnethon, M. R., R. J. Prineas, M. Temprosa, Z. M. Zhang, G. Uwaifo y M. E. Molitch. "The Association Among Autonomic Nervous System Function, Incident Diabetes, and Intervention Arm in the Diabetes Prevention Program". Diabetes Care 29, n.º 4 (27 de marzo de 2006): 914–19. http://dx.doi.org/10.2337/diacare.29.04.06.dc05-1729.

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38

Tseng, Kuang-Wen, Mei-Lin Peng, Yang-Cheng Wen, Kang-Jen Liu y Chung-Liang Chien. "Neuronal degeneration in autonomic nervous system of Dystonia musculorum mice". Journal of Biomedical Science 18, n.º 1 (2011): 9. http://dx.doi.org/10.1186/1423-0127-18-9.

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39

Skrapari, Ioanna, Nicholas Tentolouris, Despoina Perrea, Christos Bakoyiannis, Athanasia Papazafiropoulou y Nicholas Katsilambros. "Baroreflex Sensitivity in Obesity: Relationship With Cardiac Autonomic Nervous System Activity*". Obesity 15, n.º 7 (julio de 2007): 1685–93. http://dx.doi.org/10.1038/oby.2007.201.

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40

Fudashkin, A. y B. Usupbekova. "The Effect of Osteopathic Correction to the Vegetative Status in Patients with Diabetes Mellitus of 2nd Type". Russian Osteopathic Journal, n.º 3-4 (30 de diciembre de 2015): 21–28. http://dx.doi.org/10.32885/2220-0975-2015-3-4-21-28.

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The results of instrumental and functional efficiency assessment of osteopathic correction for diabetes mellitus of 2nd type autonomic dysfunctions are presented in this article. Osteopathic examination data are provided and common somatic dysfunctions are identified. Osteopathic correction of the autonomic disorders is associated with the balancing process for both sympathetic and parasympathetic divisions of autonomic nervous system.
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41

Trombetti, Andrea, Laura Richert, François R. Herrmann, Thierry Chevalley, Jean-Daniel Graf y René Rizzoli. "Selective Determinants of Low Bone Mineral Mass in Adult Women with Anorexia Nervosa". International Journal of Endocrinology 2013 (2013): 1–9. http://dx.doi.org/10.1155/2013/897193.

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We investigated the relative effect of amenorrhea and insulin-like growth factor-I (sIGF-I) levels on cancellous and cortical bone density and size. We investigated 66 adult women with anorexia nervosa. Lumbar spine and proximal femur bone mineral density was measured by DXA. We calculated bone mineral apparent density. Structural geometry of the spine and the hip was determined from DXA images. Weight and BMI, but not height, as well as bone mineral content and density, but not area and geometry parameters, were lower in patients with anorexia nervosa as compared with the control group. Amenorrhea, disease duration, and sIGF-I were significantly associated with lumbar spine and proximal femur BMD. In a multiple regression model, we found that sIGF-I was the only significant independent predictor of proximal femur BMD, while duration of amenorrhea was the only factor associated with lumbar spine BMD. Finally, femoral neck bone mineral apparent density, but not hip geometry variables, was correlated with sIGF-I. In anorexia nervosa, spine BMD was related to hypogonadism, whereas sIGF-I predicted proximal femur BMD. The site-specific effect of sIGF-I could be related to reduced volumetric BMD rather than to modified hip geometry.
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42

Quilliot, Didier, Faiez Zannad y Olivier Ziegler. "Impaired response of cardiac autonomic nervous system to glucose load in severe obesity". Metabolism 54, n.º 7 (julio de 2005): 966–74. http://dx.doi.org/10.1016/j.metabol.2005.03.002.

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43

Dunlap, Elizabeth D., Ellis Samols, Leonard C. Waite y Michael A. Pfeifer. "Development of a method to determine autonomic nervous system function in the rat". Metabolism 36, n.º 2 (febrero de 1987): 193–97. http://dx.doi.org/10.1016/0026-0495(87)90017-5.

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44

Taranikanti, Madhuri, Akhila Dronamraju, Sudha Bala, Rohith Kumar Guntuka y Aswin Kumar Mudunuru. "Autonomic Nervous System Derangement as a Predictor of Cardiovascular Disease in Obese Postmenopausal Women". Indian Journal of Cardiovascular Disease in Women WINCARS 04, n.º 01 (febrero de 2019): 008–14. http://dx.doi.org/10.1055/s-0039-1692310.

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Abstract Background and Aim Cardiovascular disease is more prevalent in women after menopause and particularly in those aged > 60 years. Obesity during menopause has become a growing cause of concern as the source of estrogen during this period is extragonadal. Autonomic function tests serve as effective markers of autonomic activity. Any derangement of autonomic functions in obese postmenopausal women would serve as an early marker for the detection of cardiovascular disease. The aims and objectives of this study were to measure autonomic functions tests in obese postmenopausal women and to correlate the autonomic activity with anthropometric parameters and serum estrogen levels. Material and Methods A cross-sectional study of 101 postmenopausal women aged > 45 years was undertaken. Women with heart disease, diabetes mellitus, hypertension, and respiratory disorders were excluded from this study. A digital data acquisition system, Lab Chart Pro 8.0 was used to perform the autonomic function tests. Results Parasympathetic activity decreased significantly with higher body mass index (BMI). Sympathetic activity that includes diastolic blood pressure and blood pressure measured after isometric handgrip test significantly increased with BMI. A strong negative correlation existed between BMI and parasympathetic high-frequency (HF) domain index. There was a strong positive correlation between BMI and serum estradiol levels. Conclusion Measuring the autonomic activity in obese postmenopausal women along with anthropometric indices and estrogen levels would provide strong evidence of a potential target for existing as well as future heart disease. Any autonomic dysfunction, if detected early, would enable early interventions to prevent heart disease.
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45

Souza, Naiara M., Thais R. Giacon, Francis L. Pacagnelli, Marianne P. C. R. Barbosa, Vitor E. Valenti y Luiz C. M. Vanderlei. "Dynamics of heart rate variability analysed through nonlinear and linear dynamics is already impaired in young type 1 diabetic subjects". Cardiology in the Young 26, n.º 7 (3 de febrero de 2016): 1383–90. http://dx.doi.org/10.1017/s104795111500270x.

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AbstractBackgroundAutonomic diabetic neuropathy is one of the most common complications of type 1 diabetes mellitus, and studies using heart rate variability to investigate these individuals have shown inconclusive results regarding autonomic nervous system activation.AimsTo investigate the dynamics of heart rate in young subjects with type 1 diabetes mellitus through nonlinear and linear methods of heart rate variability.MethodsWe evaluated 20 subjects with type 1 diabetes mellitus and 23 healthy control subjects. We obtained the following nonlinear indices from the recurrence plot: recurrence rate (REC), determinism (DET), and Shanon entropy (ES), and we analysed indices in the frequency (LF and HF in ms2 and normalised units – nu – and LF/HF ratio) and time domains (SDNN and RMSSD), through analysis of 1000 R–R intervals, captured by a heart rate monitor.ResultsThere were reduced values (p<0.05) for individuals with type 1 diabetes mellitus compared with healthy subjects in the following indices: DET, REC, ES, RMSSD, SDNN, LF (ms2), and HF (ms2). In relation to the recurrence plot, subjects with type 1 diabetes mellitus demonstrated lower recurrence and greater variation in their plot, inter-group and intra-group, respectively.ConclusionYoung subjects with type 1 diabetes mellitus have autonomic nervous system behaviour that tends to randomness compared with healthy young subjects. Moreover, this behaviour is related to reduced sympathetic and parasympathetic activity of the autonomic nervous system.
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46

Taborsky, Gerald J. y Thomas O. Mundinger. "Minireview: The Role of the Autonomic Nervous System in Mediating the Glucagon Response to Hypoglycemia". Endocrinology 153, n.º 3 (1 de marzo de 2012): 1055–62. http://dx.doi.org/10.1210/en.2011-2040.

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In type 1 diabetes, the impairment of the glucagon response to hypoglycemia increases both its severity and duration. In nondiabetic individuals, hypoglycemia activates the autonomic nervous system, which in turn mediates the majority of the glucagon response to moderate and marked hypoglycemia. The first goal of this minireview is therefore to illustrate and document these autonomic mechanisms. Specifically we describe the hypoglycemic thresholds for activating the three autonomic inputs to the islet (parasympathetic nerves, sympathetic nerves, and adrenal medullary epinephrine) and their magnitudes of activation as glucose falls from euglycemia to near fatal levels. The implication is that their relative contributions to this glucagon response depend on the severity of hypoglycemia. The second goal of this minireview is to discuss known and suspected down-regulation or damage to these mechanisms in diabetes. We address defects in the central nervous system, the peripheral nervous system, and in the islet itself. They are categorized as either functional defects caused by glucose dysregulation or structural defects caused by the autoimmune attack of the islet. In the last section of the minireview, we outline approaches for reversing these defects. Such reversal has both scientific and clinical benefit. Scientifically, one could determine the contribution of these defects to the impairment of glucagon response seen early in type 1 diabetes. Clinically, restoring this glucagon response would allow more aggressive treatment of the chronic hyperglycemia that is linked to the debilitating long-term complications of this disease.
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47

Petrofsky, Jerrold, Lee Berk y Hani Al-Nakhli. "The Influence of Autonomic Dysfunction Associated with Aging and Type 2 Diabetes on Daily Life Activities". Experimental Diabetes Research 2012 (2012): 1–12. http://dx.doi.org/10.1155/2012/657103.

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Type 2 diabetes (T2D) and ageing have well documented effects on every organ in the body. In T2D the autonomic nervous system is impaired due to damage to neurons, sensory receptors, synapses and the blood vessels. This paper will concentrate on how autonomic impairment alters normal daily activities. Impairments include the response of the blood vessels to heat, sweating, heat transfer, whole body heating, orthostatic intolerance, balance, and gait. Because diabetes is more prevalent in older individuals, the effects of ageing will be examined. Beginning with endothelial dysfunction, blood vessels have impairment in their ability to vasodilate. With this and synaptic damage, the autonomic nervous system cannot compensate for effectors such as pressure on and heating of the skin. This and reduced ability of the heart to respond to stress, reduces autonomic orthostatic compensation. Diminished sweating causes the skin and core temperature to be high during whole body heating. Impaired orthostatic tolerance, impaired vision and vestibular sensing, causes poor balance and impaired gait. Overall, people with T2D must be made aware and counseled relative to the potential consequence of these impairments.
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48

Brown, F. M., M. Watts y S. L. Rabinowe. "Aggregation of Subclinical Autonomic Nervous System Dysfunction and Autoantibodies in Families With Type I Diabetes". Diabetes 40, n.º 12 (1 de diciembre de 1991): 1611–14. http://dx.doi.org/10.2337/diab.40.12.1611.

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Kalopita, Stavroula, Stavros Liatis, Petros Thomakos, Ioannis Vlahodimitris, Chyssoula Stathi, Nicholas Katsilambros y Konstantinos Makrilakis. "RELATIONSHIP BETWEEN GLUCOSE VARIABILITY AND AUTONOMIC NERVOUS SYSTEM DYSFUNCTION IN PATIENTS WITH TYPE 2 DIABETES". European Journal of Internal Medicine 22 (octubre de 2011): S44—S45. http://dx.doi.org/10.1016/s0953-6205(11)60183-0.

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Selvaraj, V. y O. Mantro. "Assessment of autonomic nervous system function in type 2 diabetes a war of new era". Journal of the Neurological Sciences 405 (octubre de 2019): 336–37. http://dx.doi.org/10.1016/j.jns.2019.10.1462.

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