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Literatura académica sobre el tema "Bacterial Vaginosis"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 29 de mayo de 2022

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Artículos de revistas sobre el tema "Bacterial Vaginosis"

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Djukic, Slobodanka, Natasa Opavski, Vera Mijac y Lazar Ranin. "Current knowledge of bacterial vaginosis". Srpski arhiv za celokupno lekarstvo 139, n.º 5-6 (2011): 402–8. http://dx.doi.org/10.2298/sarh1106402d.

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Bacterial vaginosis, earlier termed nonspecific vaginitis (anaerobic vaginosis) because of the absence of recognized pathogens, is most common vaginal syndrome of women of childbearing age affecting 15-30%. This syndrome, whose aetiology and pathogenesis remains unknown, is characterized by significant changes in the vaginal ecosystem. These changes consist of a decrease in the number of lactobacilli and a large increase in the number of anaerobic organisms. The bacteria adhere to desquamated epithelial cells with a distinctive appearance of clue cells The main complaints of women with symptomatic bacterial vaginosis include vaginal discharge and odour. However, a significant number of all women who have bacterial vaginosis deny symptoms. Bacterial vaginosis is associated with a number of gynaecologic and obstetric complications including cervicitis, cervical neoplasia, pelvic inflammatory disease, postoperative infections, and preterm labour. The diagnosis is most frequently made based on vaginal smear stained according to Gram (Nugent scoring method). Metronidazole and clindamycin are the drugs of choice for treatment of women with bacterial vaginosis. Which women should undergo treatment? According to the prevailing attitude, it should include women with symptoms. Symptomatic women with frequent relapses of bacterial vaginosisas, as a rule, have poor response to the applied therapy. To achieve better efficiency in the treatment of such women, it is necessary to have more extensive understanding of all factors in the pathogenesis of the syndrome.

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Wang, Jeff. "Bacterial vaginosis". Primary Care Update for OB/GYNS 7, n.º 5 (septiembre de 2000): 181–85. http://dx.doi.org/10.1016/s1068-607x(00)00043-3.

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Hay, Phillip. "Bacterial vaginosis". Medicine 38, n.º 6 (junio de 2010): 281–85. http://dx.doi.org/10.1016/j.mpmed.2010.03.008.

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Hay, Phillip. "Bacterial vaginosis". Medicine 42, n.º 7 (julio de 2014): 359–63. http://dx.doi.org/10.1016/j.mpmed.2014.04.011.

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OSBORNE, NEWTON G. "Bacterial Vaginosis". Journal of Gynecologic Surgery 16, n.º 2 (enero de 2000): 93–94. http://dx.doi.org/10.1089/gyn.2000.16.93.

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VEJTORP, MOGENS, ANNE CATHRINE BOLLERUP, LlSSlE VEJTORP, ERIK FANOE, EVA NATHAN, ANITA REITER, MARY E. ANDERSEN, BODIL STROMSHOLT y STEEN STEENBEK SCHRODER. "Bacterial Vaginosis". Obstetrical & Gynecological Survey 44, n.º 6 (junio de 1989): 471–72. http://dx.doi.org/10.1097/00006254-198906000-00020.

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Neri, A., D. Rabinerson y B. Kaplan. "Bacterial Vaginosis". Obstetrical & Gynecological Survey 49, n.º 12 (diciembre de 1994): 809–13. http://dx.doi.org/10.1097/00006254-199412000-00003.

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Young, H. "Bacterial vaginosis". Sexually Transmitted Infections 61, n.º 3 (1 de junio de 1985): 213–14. http://dx.doi.org/10.1136/sti.61.3.213.

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Pattman, R. S. "Bacterial vaginosis". Sexually Transmitted Infections 64, n.º 3 (1 de junio de 1988): 208. http://dx.doi.org/10.1136/sti.64.3.208.

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OʼBrien, Rebecca Flynn. "Bacterial Vaginosis". Postgraduate Obstetrics & Gynecology 25, n.º 23 (noviembre de 2005): 1–7. http://dx.doi.org/10.1097/00256406-200511300-00001.

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Tesis sobre el tema "Bacterial Vaginosis"

1

Al-Mushrif, Shawqi A. "Pathogenicity of bacterial vaginosis". Thesis, University of Sheffield, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.310757.

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Eriksson, Katarina. "Bacterial Vaginosis : Diagnosis, Prevalence, and Treatment". Doctoral thesis, Linköpings universitet, Obstetrik och gynekologi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-68812.

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Bacterial Vaginosis (BV) is a disorder of unknown etiology, characterized by a foul smelling vaginal discharge, loss or reduction of the normal vaginal Lactobacilli, and overgrowth of other anaerobic bacteria. Thus, it presents a formidable problem for clinicians as well as microbiologists researching its etiology, clinical course, treatment, and epidemiology. The present work focuses on the unresolved issues of the epidemiology and treatment of BV in order to provide valid methods for treatment studies of this condition and to describe the prevalence of BV in defined populations. The first study validates the use of PAP-stained smears in the diagnosis of BV. The study assesses the methods of Amsel’s clinical criteria and Nugent criteria on Gram-stain smears, against Pap-stained smears and also validates different observers. The result shows that the PAP-staining of vaginal smears is a good method in BV diagnosis; the kappa value is 0.86 (interobserver weighted kappa index) compared to 0.81 for Gram-stained smears, and 0.70 for rehydrated air-dried smears using the mean Nugent score as the criterion standard. This enables population based studies on archived PAP-stained smears from the screening of cervical cancer. In the second study, we use the knowledge gained from study one to investigate the prevalence of BV in a cohort from the population of Åland. The prevalences of BV on the Åland Islands were: 15.6 %, 11.9 %, 8.7 %, and 8.6% in 1993, 1998, 2003, and 2008, respectively. This means that the prevalence of BV decreased between1993-2008 from 15.6% to 8.6%. The confidence intervals are not overlapping, thus indicating a significant decrease in prevalence from 1993 to 2008. The third study is a prospective, double-blind placebo controlled treatment study of BV. After conventional treatment with clindamycin, the patients were treated with adjuvant treatment of Lactobacilli-loaded tampons or placebo. The study showed no differences between the treatment and the placebo group, indicating that the tampon does not work at all. There are a variety of possible explanations for the result, which are analyzed in this thesis. The fourth study aimed to evaluate whether clindamycin is retained for a long time in the vaginal mucosa, thus disturbing the Lactobacilli in an attempt to reimplant Lactobacilli in the probiotic treatment studies. In conventional treatment, it is also useful to know whether clindamycin is retained, especially when considering the pressure from antibiotics on the antimicrobial sensitivity pattern. In the study, we found that the clindamycin disappears rapidly. Conclusion: BV research requires effort from many different scientific disciplines and the riddle of this condition and its treatment can only be resolved by concerted actions in research and treatment. The vision for the future includes, among other factors, better molecular biology based diagnostic tools, and knowledge of population based bacterial floras.

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Bukusi, Elizabeth Anne. "Bacterial vaginosis : a randomized controlled trial to prevent recurrence /". Thesis, Connect to this title online; UW restricted, 2006. http://hdl.handle.net/1773/10880.

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Brumley, Jessica. "Testing a Model of Bacterial Vaginosis among Black Women". Scholar Commons, 2012. http://scholarcommons.usf.edu/etd/3995.

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Bacterial Vaginosis is an inbalance of vaginal flora which has been associated with increased risk of numerous gynecological and obstetric morbidities including increased risk of acquisition of HIV from an infected partner and increased risk of preterm delivery. Black race has been consistently identified as a risk factor for BV. Black women also suffer from significant disparities in most of the morbidities also associated with BV when compared to women of other ethnicities and races. Traditional predictors of BV such as douching practices and sexual behaviors do not fully account for the racial disparities in BV prevalence. Researchers have begun to explore the potential relationship between stress and BV. Also, perceived racism has been identified as a potential stressor contributing to the health outcomes of Black women. The purpose of this study was to test a predictive model of bacterial vaginosis among Black women. The Allostatic Load Model was the theoretical framework. Participants (N=94) completed a self administered questionnaire and interview including measures of perceived stress, preceived racism, behavioral responses to stress and specific behavioral responses to racism along with traditional predictors of BV. Measurement scales included the Cohen Perceived Stress Scale, the John Henryism Scale of Active Coping, the Everyday Perceived Racial Discrimination Index, the Experiences of Discrimination Scale and the Vines Telephone Administered Perceived Racism Scale (TPRS) which included a behavioral responses to racism subscale. Bacterial vaginosis was diagnosed utilizing a self-collected vaginal swab which was analyzed utilizing the BVBlue point of care testing kit. Twenty percent (N=19) of participants screened positive for bacterial vaginosis. Douching and sexual activity in the last three months and education were significantly associated with bacterial vaginosis. Age, income, hormonal contraceptive use and condom use were not associated with BV. Neither perceived stress nor perceived racism were associated with bacterial vaginosis. After logistic regression analysis, only education continued to be a significant predictor of BV. The lack of an association between BV and the main study variables may have been related to young age of the sample or the low rates of high perceived stress and high perceived racism. Perceived stress was positively associated with perceived racism and behavioral responses to stress. This association is likely a reflection of the stressful nature of perceived racism. Further research is needed to better understand how the stressful nature of racism and behavioral responses to stressors may influence health outcomes and if interventions can be utilized to promote adaptive behavioral responses.

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Ferreira, Carolina Sanitá Tafner [UNESP]. "Avaliação proteômica do conteúdo vaginal em resposta ao tratamento da vaginose bacteriana". Universidade Estadual Paulista (UNESP), 2015. http://hdl.handle.net/11449/131919.

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Made available in DSpace on 2015-12-10T14:22:34Z (GMT). No. of bitstreams: 0 Previous issue date: 2015-02-12. Added 1 bitstream(s) on 2015-12-10T14:28:40Z : No. of bitstreams: 1 000841294_20160212.pdf: 455091 bytes, checksum: cf5f5b68871cf0cd54c8c50a907eee6f (MD5) Bitstreams deleted on 2016-02-12T10:30:03Z: 000841294_20160212.pdf,. Added 1 bitstream(s) on 2016-02-12T10:30:40Z : No. of bitstreams: 1 000841294.pdf: 1238916 bytes, checksum: 3073f4f03baf2b589d8ee875c0fca572 (MD5)
Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)
Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)
Pró-Reitoria de Pós-Gradução da UNESP (ProPG)
A vaginose bacteriana é o tipo mais comum de flora vaginal anormal e pode ser definida pela diminuição, ou mesmo depleção, dos lactobacilos vaginais. Tal condição está associada ao aumento do risco de parto prematuro e aquisição de diversas infecções sexualmente transmissíveis. A eficácia a curto prazo do tratamento da vaginose bacteriana com metronidazol é baixa. Portanto, o objetivo desse trabalho foi caracterizar o proteoma do fluido cérvico-vaginal de mulheres com vaginose bacteriana e comparar o perfil proteômico entre as mulheres que foram tratadas com sucesso em relação àquelas que falharam em restabelecer a microbiota lactobacilar após 7 dias de metronidazol. A presença da vaginose bacteriana foi definida de acordo com os critérios de Nugent após coloração de Gram dos esfregaços vaginais. Os perfis proteômicos do fluido cérvico-vaginal foram determinados utilizando a metodologia de shotgun LC MS/MS. As análises comparativas do proteoma das 38 mulheres com vaginose bacteriana e 39 com microbiota vaginal normal identificaram e determinaram a abundância relativa de 116 proteínas. Entre elas, catepsina G e a região BRO da cadeia pesada V-III de imunoglobulina foram exclusivas de vaginose bacteriana e o inibidor de elastase de leucócitos, involucrina e a proteína associada a diferenciação de neuroblastos AHNAK exclusivas de microbiota vaginal normal. Além disso, 20 (17.2%) proteínas foram diferencialmente expressas na vaginose bacteriana, das quais 9 são envolvidas na resposta imune. Entretanto, a comparação do proteoma do fluido cérvico-vaginal das 24 mulheres com vaginose bacteriana que foram tratadas com sucesso e 11 que persistiram com esta condição após o tratamento com metronidazol não apresentou diferença. Portanto, pudemos demonstrar que a vaginose bacteriana altera significantemente o proteoma local, mas o perfil proteômico cérvico-vaginal do hospedeiro não influencia a resposta ao...
Bacterial vaginosis is the most common type of abnormal vaginal flora and defined by the depletion of vaginal lactobacilli. This condition increases the risk of premature labor and acquisition of several sexually transmitted infections. Short-term efficacy of bacterial vaginosis metronidazole treatment is low. Thus, we aimed to characterize the cervicovaginal fluid proteome of women with bacterial vaginosis and to compare the proteomic profile between women who were successfully treated with those who failed to reestablish lactobacillar flora after the 7-days course of metronidazole. Presence of bacterial vaginosis was defined according to Nugent criteria on Gram-stained vaginal smears. Proteomic profile of cervicovaginal fluids were determined using shotgun LC MS/MS. Comparative analysis of the proteome of 38 women with bacterial vaginosis and 39 with normal vaginal flora identified and determined the relative abundance of 116 proteins. Among them, cathepsin G and Ig heavy chain V-III region BRO were exclusive of bacterial vaginosis while leukocyte elastase inhibitor, involucrin and neuroblast differentiation-associated protein AHNAK of normal flora. Moreover, 20 (17.2%) proteins were differentially expressed in bacterial vaginosis, of which 9 are involved in immune response. However, the cervicovaginal proteome of 24 women with bacterial vaginosis who were successfully treated and 11 who persisted with this condition did not differ between each other. Therefore, we showed that bacterial vaginosis significantly changes the local proteome, but cervicovaginal host's proteins do not influence the response to metronidazole treatment
FAPESP: 2012/16800-3

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Ferreira, Carolina Sanitá Tafner. "Avaliação proteômica do conteúdo vaginal em resposta ao tratamento da vaginose bacteriana /". Botucatu, 2015. http://hdl.handle.net/11449/131919.

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Orientador: Camila Marconi
Coorientador: Márcia Guimarães da Silva
Banca: Rodrigo Pauperio Soares de Camargo
Banca: Lucilene Dalazari dos Santos
Resumo: A vaginose bacteriana é o tipo mais comum de flora vaginal anormal e pode ser definida pela diminuição, ou mesmo depleção, dos lactobacilos vaginais. Tal condição está associada ao aumento do risco de parto prematuro e aquisição de diversas infecções sexualmente transmissíveis. A eficácia a curto prazo do tratamento da vaginose bacteriana com metronidazol é baixa. Portanto, o objetivo desse trabalho foi caracterizar o proteoma do fluido cérvico-vaginal de mulheres com vaginose bacteriana e comparar o perfil proteômico entre as mulheres que foram tratadas com sucesso em relação àquelas que falharam em restabelecer a microbiota lactobacilar após 7 dias de metronidazol. A presença da vaginose bacteriana foi definida de acordo com os critérios de Nugent após coloração de Gram dos esfregaços vaginais. Os perfis proteômicos do fluido cérvico-vaginal foram determinados utilizando a metodologia de shotgun LC MS/MS. As análises comparativas do proteoma das 38 mulheres com vaginose bacteriana e 39 com microbiota vaginal normal identificaram e determinaram a abundância relativa de 116 proteínas. Entre elas, catepsina G e a região BRO da cadeia pesada V-III de imunoglobulina foram exclusivas de vaginose bacteriana e o inibidor de elastase de leucócitos, involucrina e a proteína associada a diferenciação de neuroblastos AHNAK exclusivas de microbiota vaginal normal. Além disso, 20 (17.2%) proteínas foram diferencialmente expressas na vaginose bacteriana, das quais 9 são envolvidas na resposta imune. Entretanto, a comparação do proteoma do fluido cérvico-vaginal das 24 mulheres com vaginose bacteriana que foram tratadas com sucesso e 11 que persistiram com esta condição após o tratamento com metronidazol não apresentou diferença. Portanto, pudemos demonstrar que a vaginose bacteriana altera significantemente o proteoma local, mas o perfil proteômico cérvico-vaginal do hospedeiro não influencia a resposta ao...
Abstract: Bacterial vaginosis is the most common type of abnormal vaginal flora and defined by the depletion of vaginal lactobacilli. This condition increases the risk of premature labor and acquisition of several sexually transmitted infections. Short-term efficacy of bacterial vaginosis metronidazole treatment is low. Thus, we aimed to characterize the cervicovaginal fluid proteome of women with bacterial vaginosis and to compare the proteomic profile between women who were successfully treated with those who failed to reestablish lactobacillar flora after the 7-days course of metronidazole. Presence of bacterial vaginosis was defined according to Nugent criteria on Gram-stained vaginal smears. Proteomic profile of cervicovaginal fluids were determined using shotgun LC MS/MS. Comparative analysis of the proteome of 38 women with bacterial vaginosis and 39 with normal vaginal flora identified and determined the relative abundance of 116 proteins. Among them, cathepsin G and Ig heavy chain V-III region BRO were exclusive of bacterial vaginosis while leukocyte elastase inhibitor, involucrin and neuroblast differentiation-associated protein AHNAK of normal flora. Moreover, 20 (17.2%) proteins were differentially expressed in bacterial vaginosis, of which 9 are involved in immune response. However, the cervicovaginal proteome of 24 women with bacterial vaginosis who were successfully treated and 11 who persisted with this condition did not differ between each other. Therefore, we showed that bacterial vaginosis significantly changes the local proteome, but cervicovaginal host's proteins do not influence the response to metronidazole treatment
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McLean, Nigel W. "The role of lactobacilli in the prevention of bacterial vaginosis". Thesis, Glasgow Caledonian University, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308801.

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Keane, Frances Emer. "Studies of the aetiologies of non-gonococcal urethritis and bacterial vaginosis". Thesis, Queen's University Belfast, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.287207.

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Ugwumadu, Austin Hayes Nnamdi. "The influence of bacterial vaginosis and intermediate flora on human pregnancy". Thesis, St George's, University of London, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.433661.

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Yasnikovska, S. M. "Prognostication of placental dysfunction development in pregnant women with bacterial vaginosis". Thesis, БДМУ, 2021. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18724.

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Libros sobre el tema "Bacterial Vaginosis"

1

Parker, James N. y Philip M. Parker. The official patient's sourcebook on bacterial vaginosis. Editado por Icon Group International Inc y NetLibrary Inc. San Diego, Calif: Icon Health Publications, 2002.

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2

Thomason, J. L. Bacterial vaginosis (Current concepts). The Upjohn Co, 1990.

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3

Meijden, W. I. Van Der. Bacterial Vaginosis: Clue Cell-Positive Discharge : Diagnostic, Ultra-Structural, and Therapeutic Aspects. Van Gorcum Ltd, 1987.

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Publications, ICON Health. Bacterial Vaginosis - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References. ICON Health Publications, 2003.

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Swidsinski, Alexander, Mario Vaneechoutte y Nuno Cerca, eds. Bacterial Vaginosis, a Model of True Polymicrobial Infections: Genetics, Evolution, Clinical and Socio-Clinical Implications. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88966-222-7.

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Publications, ICON Health. The Official Patient's Sourcebook on Bacterial Vaginosis: A Revised and Updated Directory for the Internet Age. Icon Health Publications, 2002.

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Török, M. Estée, Fiona J. Cooke y Ed Moran. Sexually transmitted infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199671328.003.0018.

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This chapter covers the diagnosis and management of sexually transmitted infections, including bacterial vaginosis, with causes including vaginal discharge, vulvovaginal candidiasis, and trichomoniasis. The chapter also covers vulvovaginal candidiasis, genital warts or anogenital warts caused by human papillomavirus, tropical genital ulceration (which is commoner in patients presenting with sexually transmitted infections in the developing world and is an important factor in the spread of HIV), genital herpes, pelvic inflammatory disease, toxic shock syndrome, gonorrhoea, chlamydia, trichomoniasis, and syphilis.

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F, Easmon C. S., ed. The diagnosis and management of bacterial vaginosis: Proceedings of a Round Table meeting held at Robinson College, Cambridge, on 4 July 1993. London: Royal Society of Medicine Services, 1993.

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Holst, John. Pelvic Inflammatory Disease and Tubo-Ovarian Abscess. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0040.

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Pelvic inflammatory disease (PID) consists of inflammation in various parts of the upper genital tract and includes endometritis, salpingitis, tubo-ovarian abscess (TOA), and/or pelvic peritonitis. Overt acute PID patients typically present as ill-appearing with pain, fever, chills, purulent vaginal discharge, nausea, vomiting, and elevated white blood cells. “Silent” PID presents with dyspareunia, irregular bleeding, and urinary and gastrointestinal complaints. Bacterial vaginosis (BV) and associated microorganisms are present in acute PID patients. PID coverage is focused on a polymicrobial infection. HIV patients typically have more severe symptoms and are more likely to have a TOA than an immunocompetent patient, but HIV alone does not mandate hospital admission nor does parenteral therapy improve outcomes compared to non-HIV patients. Gonorrhea and chlamydia cases must be reported to the local health department; it is not mandatory for PID patients to remove an intrauterine device at the time of diagnosis.

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FAAP, Mary Anne Jackson MD. Red Book Informe 2015 del Comite sobre Enfermedades Infecciosas, 30.a edicion. Editado por David W. Kimberlin, Sarah S. Long y Michael T. Brady. American Academy of Pediatrics, 2015. http://dx.doi.org/10.1542/9781581109870.

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Guia autorizada del AAP a la manifestaciones, etiologia, epidemiologia, diagnostico y tratamiento de mas de 200 condiciones de la infancia. El Libro Rojo proporciona una guia basada en la evidencia a la practica de los clinicos en infecciones pediatricas y vacunas basadas en las recomendaciones del comite, asi como la experiencia combinada de los CDC, la FDA y cientos de colaboradores medicos. El Libro Rojo es una referencia esencial para pediatricos enfermedades infecciosas especialistas y pediatras generales, y es util para la medicina de familia y medicos de medicina de emergencia tambien. Los proveedores de salud publica y de salud de la escuela, medicos residentes y estudiantes tambien encontraran una fuente de alto rendimiento de la informacion pediatrica la enfermedad y la vacuna infecciosa. El libro esta dividido en secciones que cubren La inmunizacion activa y pasiva Resumenes de Enfermedades El tratamiento antimicrobiano para el tratamiento y profilaxis Cuidado de los niños en situaciones especiales Informacion y recomendaciones actualizadas que no pueden darse el lujo de estar sin ... Enfoque estandarizado para la prevencion de enfermedades a traves de las vacunas, profilaxis antimicrobiana y las practicas de control de infecciones Nuevo capitulo sobre Fiebres hemorragicas Causada por filovirus se ha añadido Nuevo capitulo sobre infecciones Parechovirus humanos se ha añadido Informacion actualizada sobre las reacciones de hipersensibilidad despues de inmunizaciones Las ultimas sobre las infecciones de transmision sexual (ITS) en los adolescentes y los niños Cobertura actualizada de actinomicosis, amebiases, arbovirus, vaginosis bacteriana, Blastocystis, candidiasis, Clostridium difficile, coronavirus, el dengue, enterovirus, Escherichia coli, Giardia intestinalis, las infecciones gonococicas, infecciones por Helicobacter pylori, enfermedad de Lyme, las infecciones meningococicas, pediculosis capitis, la tos ferina, el neumococo infecciones, rotavirus, y mas Manejo del dolor de inyeccion se ha ampliado de manera significativa Informacion actualizada sobre la hepatitis C Informacion actualizada sobre el grupo B infecciones estreptococicas Actualizado seccion sobre medicamentos para las infecciones parasitarias Significativamente capitulo revisado sobre el virus sincitial respiratorio Recomendaciones para el uso de las vacunas MMR o MMRV se han actualizado El capitulo de la Resistencia a los Antimicrobianos Antimicrobianos y Mayordomia ha broaded significativamente y actualizada Informacion actualizada sobre el VIH !Y mucho mas!

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Capítulos de libros sobre el tema "Bacterial Vaginosis"

1

Sonnex, Christopher. "Bacterial Vaginosis". En In Clinical Practice, 17–23. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-21638-6_4.

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Eschenbach, David A. "Bacterial Vaginosis". En Sexually Transmitted Diseases and Adverse Outcomes of Pregnancy, 101–23. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818210.ch7.

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Rubins, A. "Bacterial Vaginosis". En Sexually Transmitted Infections and Sexually Transmitted Diseases, 203–6. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-14663-3_19.

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Rosca, Aliona y Nuno Cerca. "Bacterial Vaginosis". En Diagnostics to Pathogenomics of Sexually Transmitted Infections, 257–75. Chichester, UK: John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119380924.ch13.

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5

Sobel, Jack D. "Bacterial Vaginosis". En Sexually Transmitted Infections in HIV-Infected Adults and Special Populations, 165–74. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56694-8_9.

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6

Piot, Peter. "Bacterial Vaginosis". En Laboratory Diagnosis of Infectious Diseases, 94–99. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3898-0_9.

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7

Fujisaki, Midori. "Bacterial Vaginosis". En Preterm Labor and Delivery, 175–80. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-9875-9_18.

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8

Marrazzo, Jeanne M. "Bacterial Vaginosis". En Sexually Transmitted Diseases, 54–63. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118314937.ch7.

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9

Tao, Lin, Sylvia I. Pavlova y Ali O. Kiliç. "Phages and Bacterial Vaginosis". En Phages, 256–79. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816506.ch13.

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10

De Seta, Francesco, Manola Comar, Secondo Guaschino y Bryan Larsen. "Bacterial Vaginitis and Vaginosis". En Sexually Transmitted Infections, 277–94. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-02200-6_14.

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Actas de conferencias sobre el tema "Bacterial Vaginosis"

1

Lithgow, K., V. Buchholz, S. Konschuh y L. Sycuro. "O02.5 Secreted Proteolytic Activity of Bacterial Vaginosis-Associated Bacteria". En Abstracts for the STI & HIV World Congress, July 14–17 2021. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/sextrans-2021-sti.60.

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2

Baker, Yolanda S., Rajeev Agrawal, James A. Foster, Daniel Beck y Gerry Dozier. "Detecting bacterial vaginosis using machine learning". En the 2014 ACM Southeast Regional Conference. New York, New York, USA: ACM Press, 2014. http://dx.doi.org/10.1145/2638404.2638521.

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3

Lokken, E. "S01.2 Bacterial vaginosis, vaginal microbiota, and fecundability". En Abstracts for the STI & HIV World Congress, July 14–17 2021. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/sextrans-2021-sti.20.

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4

Zhan, Chengsen, Ting Liu y He Ding. "Design of portable bacterial vaginosis detection system". En 13th International Photonics and OptoElectronics Meetings (POEM 2021), editado por Xinliang Zhang, Perry Shum y Jianji Dong. SPIE, 2022. http://dx.doi.org/10.1117/12.2626577.

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5

Baker, Yolanda S., Rajeev Agrawal, James A. Foster, Daniel Beck y Gerry Dozier. "Applying machine learning techniques in detecting Bacterial Vaginosis". En 2014 International Conference on Machine Learning and Cybernetics (ICMLC). IEEE, 2014. http://dx.doi.org/10.1109/icmlc.2014.7009123.

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6

Eleuterio, Renata Mirian Nunes, José Eleutério Junior, Paulo Giraldo, Ana Katherine Gonçalves, Beatriz Gordiano Valente y Fernanda Queiroz. "P5.28 Diagnosis of bacterial vaginosis with affirm vpiii". En STI and HIV World Congress Abstracts, July 9–12 2017, Rio de Janeiro, Brazil. BMJ Publishing Group Ltd, 2017. http://dx.doi.org/10.1136/sextrans-2017-053264.644.

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7

Muzny, Christina, Kristal Aaron, Angela Pontius, Cheri Aycock y Jane Schwebke. "P375 Risk factors for incident bacterial vaginosis among heterosexual women". En Abstracts for the STI & HIV World Congress (Joint Meeting of the 23rd ISSTDR and 20th IUSTI), July 14–17, 2019, Vancouver, Canada. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/sextrans-2019-sti.477.

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8

Mitchell, Caroline. "S17.2 Treatment of bacterial vaginosis: how, when and how much?" En Abstracts for the STI & HIV World Congress (Joint Meeting of the 23rd ISSTDR and 20th IUSTI), July 14–17, 2019, Vancouver, Canada. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/sextrans-2019-sti.78.

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9

Carter, Joi, Daniel Beck, Henry Williams, Gerry Dozier y James A. Foster. "GA-based selection of vaginal microbiome features associated with bacterial vaginosis". En GECCO '14: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2014. http://dx.doi.org/10.1145/2576768.2598378.

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10

Beamer, May, Leslie Meyn, Melinda Petrina, Lisa Cosentino, Hilary Avolia, Michele Austin, Allison Demarco, Victoria Gould y Sharon Hillier. "P365 Microbial risk factors for acquisition of symptomatic Bacterial Vaginosis (BV)". En Abstracts for the STI & HIV World Congress (Joint Meeting of the 23rd ISSTDR and 20th IUSTI), July 14–17, 2019, Vancouver, Canada. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/sextrans-2019-sti.467.

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