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Artículos de revistas sobre el tema "Balance inflammatoire"

Autor: Grafiati

Publicado: 21 de diciembre de 2024

Última modificación: 31 de julio de 2025

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1

Buron, F., P. Malvezzi, E. Villar, et al. "Effet du sirolimus sur la balance inflammatoire en transplantation rénale : étude sirilygre." Néphrologie & Thérapeutique 8, no. 5 (2012): 268. http://dx.doi.org/10.1016/j.nephro.2012.07.305.

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2

SAUVET, F., M. CHENNAOUI, S. BANZET, et al. "Coup de chaleur d’exercice, système cardiovasculaire et vulnérabilité systémique." Médecine et Armées Vol. 40 No. 3, Volume 40, Numéro 3 (June 1, 2012): 231–40. http://dx.doi.org/10.17184/eac.6611.

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L’un des problèmes posés par le coup de chaleur d’exercice réside dans la précocité des troubles neurologiques qui sont la conséquence de mécanismes pernicieux généraux et cérébraux aboutissant à un déficit de perfusion tissulaire et à une ischémie cérébrale. Au niveau général, la redistribution vasculaire et les modifications de la vasomotricité d’origine inflammatoire, favorisent l’apparition d’un collapsus vasculaire. L'ischémie cérébrale, liée à l’oedème cérébral et à l’hypoperfusion, favorise un état de déséquilibre énergétique qui s'accompagne d'une cytotoxicité, d’une inflammation local

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3

Horiuchi, I., Y. Kawano, M. Minohara, and J. Kira. "Th1Th2 balance in inflammatory neurologic diseases." Journal of Neuroimmunology 90, no. 1 (1998): 81. http://dx.doi.org/10.1016/s0165-5728(98)91665-4.

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4

Adrie, Ch, and M. R. Pinsky. "The inflammatory balance in human sepsis." Intensivmedizin und Notfallmedizin 36, no. 5 (1999): 419–28. http://dx.doi.org/10.1007/s003900050260.

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5

Adrie, C., and M. R. Pinsky. "The inflammatory balance in human sepsis." Intensive Care Medicine 26, no. 4 (2000): 364–75. http://dx.doi.org/10.1007/s001340051169.

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6

Tedgui, A., and Z. Mallat. "Pro- and anti-inflammatory balance and atherosclerosis." Atherosclerosis 151, no. 1 (2000): 165. http://dx.doi.org/10.1016/s0021-9150(00)80751-0.

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7

Hutcheson, Jack. "Adipokines influence the inflammatory balance in autoimmunity." Cytokine 75, no. 2 (2015): 272–79. http://dx.doi.org/10.1016/j.cyto.2015.04.004.

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8

Ferrarelli, Leslie K. "New connections: Restoring immune balance." Science Signaling 13, no. 661 (2020): eabf9854. http://dx.doi.org/10.1126/scisignal.abf9854.

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9

Wagener, Frank, Carine Carels, and Ditte Lundvig. "Targeting the Redox Balance in Inflammatory Skin Conditions." International Journal of Molecular Sciences 14, no. 5 (2013): 9126–67. http://dx.doi.org/10.3390/ijms14059126.

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10

Esmaelzadeh, Abbas, Hassan Vosooghinia, Mohammad Reza Sheikhian, et al. "Pro-Oxidant Antioxidant Balance in Inflammatory Bowel Disease." International Journal of Clinical Medicine 07, no. 05 (2016): 334–41. http://dx.doi.org/10.4236/ijcm.2016.75035.

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11

Müller, B., U. Gimsa, N. A. Mitchison, and A. Radbruch. "Modulating the Th1/Th2 balance in inflammatory arthritis." Springer Seminars in Immunopathology 20, no. 1-2 (1998): 181–96. http://dx.doi.org/10.1007/s002810050029.

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12

Goetzl, Laura, Susan Raine, Jose Rivers, Mary Ann Mastrangelo, David J. Tweardy, and Maya S. Suresh. "Maternal inflammatory balance and fetal exposure to hyperthermia." American Journal of Obstetrics and Gynecology 191, no. 6 (2004): S167. http://dx.doi.org/10.1016/j.ajog.2004.10.495.

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13

M�ller, B., U. Gimsa, N. A. Mitchison, A. Radbruch, J. Sieper, and Z. Yin. "Modulating the Th1/Th2 balance in inflammatory arthritis." Springer Seminars in Immunopathology 20, no. 1-2 (1998): 181–96. http://dx.doi.org/10.1007/bf00832006.

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14

Guenin, Sophie, Abrahem Kazemi, Abigail Cline, Steven R. Feldman, and Bijan Safai. "Rethinking the Inflammatory Balance in Psoriasis and Atherosclerosis." Journal of Drugs in Dermatology 22, no. 8 (2023): 832–34. http://dx.doi.org/10.36849/jdd.7082.

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15

Guan, Yulong, Caihong Wan, Shigang Wang, Peng Sun, and Cun Long. "Balanced Ultrafiltration: Inflammatory Mediator Removal Capacity." Artificial Organs 36, no. 10 (2012): 894–900. http://dx.doi.org/10.1111/j.1525-1594.2012.01471.x.

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16

Myers, Jay L., and Jorie C. Allen. "Nutrition and Inflammation." American Journal of Lifestyle Medicine 6, no. 1 (2011): 14–17. http://dx.doi.org/10.1177/1559827611424259.

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Evidence indicates that chronic diseases, such as cardiovascular disease, obesity, and asthma are part of a group of conditions linked by inflammatory dysregulation. One explanation for these associations is that the Western diet is obeseogenic and promotes the secretion of inflammatory biochemical signals. Weight gain results in lipid accumulation and adipocyte stress—factors known to disrupt the balance of systemic cell signaling (adipokines and cytokines) that favors inflammation. Markers are used as indicators of the inflammatory milieu and include the measurement of proinflammatory (C-rea

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17

Kunst, Claudia, Stephan Schmid, Marlen Michalski, et al. "The Influence of Gut Microbiota on Oxidative Stress and the Immune System." Biomedicines 11, no. 5 (2023): 1388. http://dx.doi.org/10.3390/biomedicines11051388.

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The human gastrointestinal tract is home to a complex microbial community that plays an important role in the general well-being of the entire organism. The gut microbiota generates a variety of metabolites and thereby regulates many biological processes, such as the regulation of the immune system. In the gut, bacteria are in direct contact with the host. The major challenge here is to prevent unwanted inflammatory reactions on one hand and on the other hand to ensure that the immune system can be activated when pathogens invade. Here the REDOX equilibrium is of utmost importance. This REDOX

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18

Dr.Raminder, Kaur Kathuriya, and Arijit Kumar Das Dr. "EFFECT OF LOWER LIMBS STRENGTHENING TO IMPROVE BALANCE IN COPD PATIENTS, RANDOMIZED CONTROL TRIAL." VIMS Journal of Physical Therapy 1, no. 2 (2019): 99–104. https://doi.org/10.5281/zenodo.3753835.

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Background: COPD is a progressive disease associated with an amplified chronic inflammatory response to nox-ious particles or gases in the airways and lungs.It’s management includes Pulmonary Rehabilitation .But Pulmonary Rehabilitation stays confined to the rehabilitation of lungs , it does not include treatment for secondary impairments like balance problems in patients with COPD. Aim - To improve the balance of patients suffering from COPD by im-proving the peripheral muscle strength with the help of progressive resisted exercises in addition to regular pulmo-nary rehabilitation. Obje

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19

Rodrigues, Denise, Juliana Romanelli Marçal, Renata Samuel, et al. "Th17/Treg immunoregulatory balance in human periapical lesions (47.5)." Journal of Immunology 184, no. 1_Supplement (2010): 47.5. http://dx.doi.org/10.4049/jimmunol.184.supp.47.5.

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Abstract Cysts and granulomas are chronic periapical lesions mediated by a set of inflammatory mediators that develop in order to contain a periapical infection. This study analyzed the inflammatory infiltrate, mast cells and in situ expression of cytokines, chemokines and FoxP3 in human periapical granulomas and cysts compared to controls. Fifty-five lesions (25 cysts, 25 granulomas and 5 controls) were analyzed. The type of inflammatory infiltrate was evaluated by HE staining and the presence of mast cells was analyzed by toluidine blue staining. Indirect immunohistochemistry was used to eva

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20

Proudfoot, Lorna. "Parasitic helminths tip the balance: potential anti-inflammatory therapies." Immunology 113, no. 4 (2004): 438–40. http://dx.doi.org/10.1111/j.1365-2567.2004.01996.x.

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21

SOMERVILLE, KEVIN, and GEORGE NOBLE. "Non-steroidal anti-inflammatory drugs: is the balance shifting?" Age and Ageing 26, no. 6 (1997): 417–22. http://dx.doi.org/10.1093/ageing/26.6.417.

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22

Sheikh, Amania A., and Daniel T. Utzschneider. "IRF2 integrates inflammatory signals to balance T cell exhaustion." Immunity 55, no. 12 (2022): 2225–27. http://dx.doi.org/10.1016/j.immuni.2022.11.010.

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23

Zhou, Yang, and Sabrina Bréchard. "Neutrophil Extracellular Vesicles: A Delicate Balance between Pro-Inflammatory Responses and Anti-Inflammatory Therapies." Cells 11, no. 20 (2022): 3318. http://dx.doi.org/10.3390/cells11203318.

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Extracellular vesicles (EVs) are released in the extracellular environment during cell activation or apoptosis. Working as signal transducers, EVs are important mediators of intercellular communication through the convoying of proteins, nucleic acids, lipids, and metabolites. Neutrophil extracellular vesicles (nEVs) contain molecules acting as key modulators of inflammation and immune responses. Due to their potential as therapeutic tools, studies about nEVs have been increasing in recent years. However, our knowledge about nEVs is still in its infancy. In this review, we summarize the current

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24

Rušanac, Anamarija, Zara Škibola, Mario Matijašić, Hana Čipčić Paljetak, and Mihaela Perić. "Microbiome-Based Products: Therapeutic Potential for Inflammatory Skin Diseases." International Journal of Molecular Sciences 26, no. 14 (2025): 6745. https://doi.org/10.3390/ijms26146745.

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Maintaining a balanced skin microbiota is essential for skin health, whereas disruptions in skin microbiota composition, known as dysbiosis, can contribute to the onset and progression of various skin disorders. Microbiota dysbiosis has been associated with several inflammatory skin conditions, including atopic dermatitis, seborrheic dermatitis, acne, psoriasis, and rosacea. Recent advances in high-throughput sequencing and metagenomic analyses have provided a deeper understanding of the skin microbial communities in both health and disease. These discoveries are now being translated into nove

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25

Tsvinger, S. M., A. V. Govorin, and A. M. Karachenova. "Features of cytokine balance in patients with osteoarthrosis and atherosclerosis." Siberian Medical Review, no. 6 (2020): 70–74. http://dx.doi.org/10.20333/2500136-2020-6-70-74.

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The aim of the research is to study the content of some key pro / anti-inflammatory cytokines in patients with osteoarthritis and to evaluate their role in predicting atherogenesis in this category of patients. Material and methods. Patients with primary osteoarthritis of I-III radiological stages were examined. Doppler vessels ultrasound (Doppler ultrasound) of the neck, upper and lower extremities was performed; blood levels of pro / anti-inflammatory cytokines were examined: IL-1β, TNF-α, IL-6, IL- 8, IL-18, MCP-1, IL-17, IL-10, IL-33 by flow cytometry. Results. According to the thickness o

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26

Rupali Kapoor, Rupali, Sakshi Chauhan, and Swapnil Saini. "Comparative study of Vatarakta w.s.r. to Gouty Arthritis." Journal of Ayurveda and Integrated Medical Sciences 8, no. 12 (2024): 204–11. http://dx.doi.org/10.21760/jaims.8.12.30.

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A healthy lifestyle isn’t just diet and exercise. Today we go over the components of leading a healthy lifestyle and how it’s important to lead a balanced life. In general, most would agree that a healthy person doesn’t smoke, is at a healthy weight, eats a balanced healthy diet, thinks positively, feels relaxed, exercises regularly, has good relationships, and benefits from a good life balance. The World Health Organization in 1946 defined health as ‘A complete state of mental, physical and social well-being not merely the absence of disease’ Vatarakta is a metabolic condition where the most

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27

Tamburini, Bartolo, Marco Pio La Manna, Lidia La Barbera, et al. "Immunity and Nutrition: The Right Balance in Inflammatory Bowel Disease." Cells 11, no. 3 (2022): 455. http://dx.doi.org/10.3390/cells11030455.

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Inflammatory bowel disease (IBD) is an increasingly urgent medical problem that strongly impairs quality of life for patients. A global rise in incidence has been observed over the last few decades, with the highest incidence rates recorded in North America and Europe. Still, an increased incidence has been reported in the last ten years in newly industrialized countries in Asia, including China and India, both with more than one billion inhabitants. These data underline that IBD is an urgent global health problem. In addition, it is estimated that between 20% and 30% of IBD patients will deve

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28

LIAN, ZEQIN, XIAOYUAN PERRARD, CHRISTIE M. BALLANTYNE, and HUAIZHU WU. "1941-P: Aging Disturbs the Inflammatory Balance in Adipose Tissue." Diabetes 69, Supplement 1 (2020): 1941—P. http://dx.doi.org/10.2337/db20-1941-p.

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29

Mohamadzadeh, Mansour, Lieping Chen, Gene G. Olinger, William D. Pratt, and Alan L. Schmaljohn. "Filoviruses and the Balance of Innate, Adaptive, and Inflammatory Responses." Viral Immunology 19, no. 4 (2006): 602–12. http://dx.doi.org/10.1089/vim.2006.19.602.

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30

Lawrence, Catherine B. "Galanin-like peptide modulates energy balance by affecting inflammatory mediators?" Physiology & Behavior 97, no. 5 (2009): 515–19. http://dx.doi.org/10.1016/j.physbeh.2009.02.041.

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31

Fett, James D., and Aftab A. Ansari. "Inflammatory markers and cytokines in peripartum cardiomyopathy: a delicate balance." Expert Opinion on Therapeutic Targets 14, no. 9 (2010): 895–98. http://dx.doi.org/10.1517/14728222.2010.511181.

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32

Bisschops, Laurens L. A., Cornelia W. E. Hoedemaekers, Tom E. Mollnes, and Johannes G. van der Hoeven. "Rewarming after hypothermia after cardiac arrest shifts the inflammatory balance*." Critical Care Medicine 40, no. 4 (2012): 1136–42. http://dx.doi.org/10.1097/ccm.0b013e3182377050.

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33

Tallman, Richard D., Mark Dumond, and David Brown. "Inflammatory mediator removal by zero-balance ultrafiltration during cardiopulmonary bypass." Perfusion 17, no. 2 (2002): 111–15. http://dx.doi.org/10.1191/0267659102pf540oa.

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The abnormal conditions to which blood is subjected during cardiopulmonary bypass (CPB) trigger an activation of the inflammatory response in all patients to varying degrees. Both complement activation and the release of cytokines characterize this response. Most inflammatory mediators have a molecular weight that is below the membrane pore size of commonly used ultrafilters, which should allow them to be freely filtered. However, some mediators have been shown to fail to cross through the membrane even though they are small enough to cross. The purpose of the present study was to determine wh

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34

Moutusy, Salvinaz Islam, and Seiichiroh Ohsako. "Gut Microbiome-Related Anti-Inflammatory Effects of Aryl Hydrocarbon Receptor Activation on Inflammatory Bowel Disease." International Journal of Molecular Sciences 25, no. 6 (2024): 3372. http://dx.doi.org/10.3390/ijms25063372.

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Inflammatory bowel disease (IBD) is one of the most prevalent chronic inflammations of the gastrointestinal tract (GIT). The gut microbial population, the cytokine milieu, the aryl hydrocarbon receptor (AHR) expressed by immune and nonimmune cells and the intrinsic pathway of Th-cell differentiation are implicated in the immunopathology of IBD. AHR activation requires a delicate balance between regulatory and effector T-cells; loss of this balance can cause local gut microbial dysbiosis and intestinal inflammation. Thus, the study of the gut microbiome in association with AHR provides critical

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35

Warner, Fiona J., John S. Lubel, Geoffrey W. McCaughan, and Peter W. Angus. "Liver fibrosis: a balance of ACEs?" Clinical Science 113, no. 3 (2007): 109–18. http://dx.doi.org/10.1042/cs20070026.

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There is an increasing body of evidence to suggest that the RAS (renin–angiotensin system) contributes to tissue injury and fibrosis in chronic liver disease. A number of studies have shown that components of a local hepatic RAS are up-regulated in fibrotic livers of humans and in experimental animal models. Angiotensin II, the main physiological effector molecule of this system, mediates liver fibrosis by stimulating fibroblast proliferation (myofibroblast and hepatic stellate cells), infiltration of inflammatory cells, and the release of inflammatory cytokines and growth factors such as TGF

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36

Harshada, V. Patil, S. Jain Akash, R. Patil Divakar., Z. Shaikh Azam, R. Shaikh Sameer, and P. Pawar Sunil. "A Review on Usage of Probiotic in Treatment of Different Diseases." Journal of Advances in Drug Discovery and Development 2, no. 2 (2024): 63–76. https://doi.org/10.5281/zenodo.11255959.

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<em>Probiotic bacteria offer many health advantages in addition to basic nutritional value. They collaborate to keep the delicate balance. balance between the immune system and the digestive system. When this equilibrium is upset, illness and inflammation occur. By remaining to the mucosa, healthy beneficial microflora competed with harmful bacteria to limit inflammation and excessive immune system stimulation. The growth of pathogenic bacteria is prevented, disease processes are changed, and broad inflammatory disorders are prevented in a solid gastrointestinal plot with adequate bodily fluid

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37

Chebotareva, Natalya V., Anatoly A. Vinogradov, Alla A. Gindis, Irina N. Bobkova, Wenjing Cao, and Lidia V. Lysenko. "The balance of proinflammatory cytokines and Treg cells in chronic glomerulonephritis." Terapevticheskii arkhiv 92, no. 6 (2020): 46–52. http://dx.doi.org/10.26442/00403660.2020.06.000671.

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Chronic glomerulonephritis (CGN) is a disease with a steadily progressing course, which is based on inflammation with the activation of immune cells. The severity of the inflammatory reaction in the kidney tissue is determined by the balance of locally pro-inflammatory factors and protective mechanisms, which include anti-inflammatory cytokines and T-regulatory lymphocytes (Treg). The study of processes that can modulate the severity of inflammation in the kidney is of particular interest for understanding the basic patterns of CGN progression. Aim. To determine the clinical significance

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38

Nicola Conran and Erich V. De Paula. "Thromboinflammatory mechanisms in sickle cell disease - challenging the hemostatic balance." Haematologica 105, no. 10 (2020): 2380–90. http://dx.doi.org/10.3324/haematol.2019.239343.

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Sickle cell disease (SCD) is an inherited hemoglobinopathy that is caused by the presence of abnormal hemoglobin S (HbS) in red blood cells, leading to alterations in red cell properties and shape, as the result of HbS dexoygenation and subsequent polymerization. SCD pathophysiology is characterized by chronic inflammatory processes, triggered by hemolytic and vaso-occlusive events, which lead to the varied complications, organ damage and elevated mortality seen in individuals with the disease. In association with activation of the endothelium and leukocytes, hemostatic alterations and thrombo

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39

Musolino, Caterina, Alessandro Allegra, Vanessa Innao, Andrea Gaetano Allegra, Giovanni Pioggia, and Sebastiano Gangemi. "Inflammatory and Anti-Inflammatory Equilibrium, Proliferative and Antiproliferative Balance: The Role of Cytokines in Multiple Myeloma." Mediators of Inflammation 2017 (2017): 1–24. http://dx.doi.org/10.1155/2017/1852517.

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Multiple myeloma (MM) is typically exemplified by a desynchronized cytokine system with increased levels of inflammatory cytokines. We focused on the contrast between inflammatory and anti-inflammatory systems by assessing the role of cytokines and their influence on MM. The aim of this review is to summarize the available information to date concerning this equilibrium to provide an overview of the research exploring the roles of serum cytokines in MM. However, the association between MM and inflammatory cytokines appears to be inadequate, and other functions, such as pro-proliferative or ant

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40

Rosa, Luciana, Giselli Scaini, Camila B. Furlanetto, et al. "Administration of branched‐chain amino acids alters the balance between pro‐inflammatory and anti‐inflammatory cytokines." International Journal of Developmental Neuroscience 48, no. 1 (2015): 24–30. http://dx.doi.org/10.1016/j.ijdevneu.2015.11.002.

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41

Barry, Rachael, David Ruano-Gallego, Shiva T. Radhakrishnan, et al. "Faecal neutrophil elastase-antiprotease balance reflects colitis severity." Mucosal Immunology 13, no. 2 (2019): 322–33. http://dx.doi.org/10.1038/s41385-019-0235-4.

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AbstractGiven the global burden of diarrheal diseases on healthcare it is surprising how little is known about the drivers of disease severity. Colitis caused by infection and inflammatory bowel disease (IBD) is characterised by neutrophil infiltration into the intestinal mucosa and yet our understanding of neutrophil responses during colitis is incomplete. Using infectious (Citrobacter rodentium) and chemical (dextran sulphate sodium; DSS) murine colitis models, as well as human IBD samples, we find that faecal neutrophil elastase (NE) activity reflects disease severity. During C. rodentium i

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42

Wang, Chun, Jun Yuan, Hua-xun Wu, et al. "Paeoniflorin inhibits inflammatory responses in mice with allergic contact dermatitis by regulating the balance between inflammatory and anti-inflammatory cytokines." Inflammation Research 62, no. 12 (2013): 1035–44. http://dx.doi.org/10.1007/s00011-013-0662-8.

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43

JEFCOAT, A. M., J. G. WAGNER, and N. E. ROBINSON. "The neutrophil: understanding ancient and powerful responses in the inflammatory balance." Equine Veterinary Journal 35, no. 1 (2010): 5–6. http://dx.doi.org/10.2746/042516403775467559.

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44

Roger, Anais, Ana Reynders, Guillaume Hoeffel, and Sophie Ugolini. "Neuroimmune crosstalk in the skin: a delicate balance governing inflammatory processes." Current Opinion in Immunology 77 (August 2022): 102212. http://dx.doi.org/10.1016/j.coi.2022.102212.

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45

Chang, Yaoyao, Lixiang Zhai, Jiao Peng, Haiqiang Wu, Zhaoxiang Bian, and Haitao Xiao. "Phytochemicals as regulators of Th17/Treg balance in inflammatory bowel diseases." Biomedicine & Pharmacotherapy 141 (September 2021): 111931. http://dx.doi.org/10.1016/j.biopha.2021.111931.

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46

McBride, William T., and Samuel J. McBride. "The balance of pro- and anti-inflammatory cytokines in cardiac surgery." Current Opinion in Anaesthesiology 11, no. 1 (1998): 15–22. http://dx.doi.org/10.1097/00001503-199802000-00004.

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47

Honda, T., H. Domon, T. Okui, K. Kajita, R. Amanuma, and K. Yamazaki. "Balance of inflammatory response in stable gingivitis and progressive periodontitis lesions." Clinical and Experimental Immunology 144, no. 1 (2006): 35–40. http://dx.doi.org/10.1111/j.1365-2249.2006.03028.x.

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48

Ng, Tien MH, and Myron L. Toews. "Impaired norepinephrine regulation of monocyte inflammatory cytokine balance in heart failure." World Journal of Cardiology 8, no. 10 (2016): 584. http://dx.doi.org/10.4330/wjc.v8.i10.584.

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49

Konenkov, Konenkov V. I., Shevchenko A. V. Shevchenko, Koroleva E. G. Koroleva, et al. "Balance of serum cytokines regulating inflammatory activity in patients with leiomyoma." Akusherstvo i ginekologiia 6_2024 (June 28, 2024): 84–90. http://dx.doi.org/10.18565/aig.2024.74.

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50

Yan, Xuan, Sizhe Xie, Xiaowei Yan, et al. "Targets for the Immune Balance of Biologics in Inflammatory Bowel Disease." Nature Cell and Science 000, no. 000 (2024): 000. http://dx.doi.org/10.61474/ncs.2023.00025.

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