Bibliografías temáticas / Caffeine metabolism rate

Índice

Artículos de revistas

Literatura académica sobre el tema "Caffeine metabolism rate"

Autor: Grafiati

Publicado: 10 de marzo de 2023

Crea una cita precisa en los estilos APA, MLA, Chicago, Harvard y otros

Elija tipo de fuente:

Consulte las listas temáticas de artículos, libros, tesis, actas de conferencias y otras fuentes académicas sobre el tema "Caffeine metabolism rate".

Junto a cada fuente en la lista de referencias hay un botón "Agregar a la bibliografía". Pulsa este botón, y generaremos automáticamente la referencia bibliográfica para la obra elegida en el estilo de cita que necesites: APA, MLA, Harvard, Vancouver, Chicago, etc.

También puede descargar el texto completo de la publicación académica en formato pdf y leer en línea su resumen siempre que esté disponible en los metadatos.

Artículos de revistas sobre el tema "Caffeine metabolism rate"

Lader, Malcolm, Claire Cardwell, Philip Shine, and Nigel Scott. "Caffeine withdrawal symptoms and rate of metabolism." Journal of Psychopharmacology 10, no. 2 (1996): 110–18. http://dx.doi.org/10.1177/026988119601000205.

Texto completo

Los estilos APA, Harvard, Vancouver, ISO, etc.

Ashihara, Hiroshi, Hisayo Shimizu, Yoshiyuki Takeda, Takeo Suzuki, Fiona M. Gillies, and Alan Crozier. "Caffeine Metabolism in High and Low Caffeine Containing Cultivars of Camellia sinensis." Zeitschrift für Naturforschung C 50, no. 9-10 (1995): 602–7. http://dx.doi.org/10.1515/znc-1995-9-1002.

Texto completo

Resumen

Abstract The metabolism of [8-14C ]adenine and [2-14C]caffeine was examined in leaf segments from flush shoots of tea cultivars with high and low caffeine content. The caffeine biosynthesis pathway from AMP via theobromine was operative in both high and low caffeine containing cultivars. There was a m ore rapid rate of caffeine biosynthesis from [8-14C ]adenine in the high caffeine cultivars while the rate of degradation of both adenine nucleotides and caffeine into CO2 was greatest in cultivars with a low endogenous caffeine content. Cell-free p reparations from tea shoots contained an N-methyltransferase, that is a keyenzyme in the caffeine biosynthesis pathway; more in-vitro activity was detected in preparations from high caffeine containing cultivars. The data obtained suggest that the high caffeine containing cultivars have a more rapid rate of caffeine biosynthesis and a slower rate of caffeine catabolism than cultivars with a low endogenous caffeine content

Los estilos APA, Harvard, Vancouver, ISO, etc.

Vaughan, Roger A., Randi Garcia-Smith, Marco Bisoffi, Kristina A. Trujillo, and Carole A. Conn. "Effects of Caffeine on Metabolism and Mitochondria Biogenesis in Rhabdomyosarcoma Cells Compared with 2,4-Dinitrophenol." Nutrition and Metabolic Insights 5 (January 2012): NMI.S10233. http://dx.doi.org/10.4137/nmi.s10233.

Texto completo

Resumen

Purpose This work investigated if treatment with caffeine or 2,4-dinitrophenol (DNP) induce expression of peroxisome proliferator-activated receptor coactivator 1 alpha (PGC-1α) and increase both mitochondrial biosynthesis and metabolism in skeletal muscle. Methods Human rhabdomyosarcoma cells were treated with either ethanol control (0.1% final concentration) caffeine, or DNP at 250 or 500 μM for 16 or 24 hours. PGC-1α RNA levels were determined using quantitative reverse transcriptase polymerase chain reaction (qRT-PCR). PGC-1α protein and mitochondrial content was determined using flow cytometry and immunohistochemistry. Metabolism was determined by quantification of extracellular acidification rate and oxygen consumption rate. Results Treatment with either caffeine or DNP induced PGC-1α RNA and protein as well as mitochondrial content compared with control. Treatment with caffeine and DNP also significantly increased oxidative metabolism and total metabolic rate compared with control. Caffeine similarly increased metabolism and mitochondrial content compared with DNP. Conclusion This work identified that both caffeine and DNP significantly induce PGC-1α, and increase both metabolism and mitochondrial content in skeletal muscle.

Los estilos APA, Harvard, Vancouver, ISO, etc.

Bracco, D., J. M. Ferrarra, M. J. Arnaud, E. Jequier, and Y. Schutz. "Effects of caffeine on energy metabolism, heart rate, and methylxanthine metabolism in lean and obese women." American Journal of Physiology-Endocrinology and Metabolism 269, no. 4 (1995): E671—E678. http://dx.doi.org/10.1152/ajpendo.1995.269.4.e671.

Texto completo

Resumen

The magnitude of coffee-induced thermogenesis and the influence of coffee ingestion on substrate oxidation were investigated in 10 lean and 10 obese women, over two 24-h periods in a respiratory chamber. On one occasion the subjects consumed caffeinated coffee and on the other occasion, decaffeinated coffee. The magnitude of thermogenesis was smaller in obese (4.9 +/- 2.0%) than in lean subjects (7.6 +/- 1.3%). The thermogeneic response to caffeine was prolonged during the night in lean women only. The coffee-induced stimulation of energy expenditure was mediated by a concomitant increase in lipid and carbohydrate oxidation. During the next day, in postabsorptive basal conditions, the thermogenic effect of coffee had vanished, but a significant increase in lipid oxidation was observed in both groups. The magnitude of this effect was, however, blunted in obese women (lipid oxidation increased by 29 and 10% in lean and obese women, respectively). Caffeine increased urinary epinephrine excretion. Whereas urinary caffeine excretion was similar in both groups, obese women excreted more theobromine, theophylline, and paraxanthine than lean women. Despite the high levels of urinary methylxanthine excretion, thermogenesis and lipid oxidation were less stimulated in obese than in lean subjects.

Los estilos APA, Harvard, Vancouver, ISO, etc.

Fenster, L., C. Quale, R. A. Hiatt, M. Wilson, G. C. Windham, and N. L. Benowitz. "Rate of Caffeine Metabolism and Risk of Spontaneous Abortion." American Journal of Epidemiology 147, no. 5 (1998): 503–10. http://dx.doi.org/10.1093/oxfordjournals.aje.a009477.

Texto completo

Los estilos APA, Harvard, Vancouver, ISO, etc.

Masi, Camilla, Caterina Dinnella, Nicola Pirastu, John Prescott, and Erminio Monteleone. "Caffeine metabolism rate influences coffee perception, preferences and intake." Food Quality and Preference 53 (October 2016): 97–104. http://dx.doi.org/10.1016/j.foodqual.2016.06.002.

Texto completo

Los estilos APA, Harvard, Vancouver, ISO, etc.

Wu, Shou En, and Wei-Liang Chen. "Exploring the Association between Urine Caffeine Metabolites and Urine Flow Rate: A Cross-Sectional Study." Nutrients 12, no. 9 (2020): 2803. http://dx.doi.org/10.3390/nu12092803.

Texto completo

Resumen

Examination of urine excretion of caffeine metabolites has been a simple but common way to determine the metabolism and effect of caffeine, but the relationship between urinary metabolites and urine flow rate is less discussed. To explore the association between urinary caffeine metabolite levels and urine flow rate, 1571 participants from the National Health and Nutrition Examination Survey (NHANES) 2011–2012 were enrolled in this study. We examined the association between urinary caffeine metabolites and urine flow rate with linear regression models. Separate models were constructed for males and females and for participants aged <60 and ≥60 years old. A positive association was found between concentrations of several urinary caffeine metabolites and urine flow rate. Three main metabolites, namely, paraxanthine, theobromine, and caffeine, showed significance across all subgroups. The number of caffeine metabolites that revealed flow-dependency was greater in males than in females and was also greater in the young than in the elderly. Nevertheless, the general weakness of NHANES data, a cross-sectional study, is that the collection is made at one single time point rather than a long-term study. In summary, urinary concentrations of several caffeine metabolites showed a positive relationship with the urine flow rate. The trend is more noticeable in males and in young subgroups.

Los estilos APA, Harvard, Vancouver, ISO, etc.

Brown, Christopher R., Peyton Jacob III, Margaret Wilson, and Neal L. Benowitz. "Changes in rate and pattern of caffeine metabolism after cigarette abstinence." Clinical Pharmacology and Therapeutics 43, no. 5 (1988): 488–91. http://dx.doi.org/10.1038/clpt.1988.63.

Texto completo

Los estilos APA, Harvard, Vancouver, ISO, etc.

Pascua, Stephanie M., Gabrielle E. McGahey, Ning Ma, Justin J. Wang, and Michelle A. Digman. "Caffeine and Cisplatin Effectively Targets the Metabolism of a Triple-Negative Breast Cancer Cell Line Assessed via Phasor-FLIM." International Journal of Molecular Sciences 21, no. 7 (2020): 2443. http://dx.doi.org/10.3390/ijms21072443.

Texto completo

Resumen

Triple-negative tumor cells, a malignant subtype of breast cancer, lack a biologically targeted therapy. Given its DNA repair inhibiting properties, caffeine has been shown to enhance the effectiveness of specific tumor chemotherapies. In this work, we have investigated the effects of caffeine, cisplatin, and a combination of the two as potential treatments in energy metabolism for three cell lines, triple-negative breast cancer (MDA-MB-231), estrogen-receptor lacking breast cancer (MCF7) and breast epithelial cells (MCF10A) using a sensitive label-free approach, phasor-fluorescence lifetime imaging microscopy (phasor-FLIM). We found that solely using caffeine to treat MDA-MB-231 shifts their metabolism towards respiratory-chain phosphorylation with a lower ratio of free to bound NADH, and a similar trend is seen in MCF7. However, MDA-MB-231 cells shifted to a higher ratio of free to bound NADH when cisplatin was added. The combination of cisplatin and caffeine together reduced the survival rate for MDA-MD231 and shifted their energy metabolism to a higher fraction of bound NADH indicative of oxidative phosphorylation. The FLIM and viability results of MCF10A cells demonstrate that the treatments targeted cancer cells over the normal breast tissue. The identification of energy metabolism alteration could open up strategies of improving chemotherapy for malignant breast cancer.

Los estilos APA, Harvard, Vancouver, ISO, etc.

Lopes, Cátia R., Andreia Oliveira, Ingride Gaspar, et al. "Effects of Chronic Caffeine Consumption on Synaptic Function, Metabolism and Adenosine Modulation in Different Brain Areas." Biomolecules 13, no. 1 (2023): 106. http://dx.doi.org/10.3390/biom13010106.

Texto completo

Resumen

Adenosine receptors mainly control synaptic function, and excessive activation of adenosine receptors may worsen the onset of many neurological disorders. Accordingly, the regular intake of moderate doses of caffeine antagonizes adenosine receptors and affords robust neuroprotection. Although caffeine intake alters brain functional connectivity and multi-omics analyses indicate that caffeine intake modifies synaptic and metabolic processes, it is unclear how caffeine intake affects behavior, synaptic plasticity and its modulation by adenosine. We now report that male mice drinking caffeinated water (0.3 g/L) for 2 weeks were behaviorally indistinguishable (locomotion, mood, memory) from control mice (drinking water) and displayed superimposable synaptic plasticity (long-term potentiation) in different brain areas (hippocampus, prefrontal cortex, amygdala). Moreover, there was a general preservation of the efficiency of adenosine A1 and A2A receptors to control synaptic transmission and plasticity, although there was a tendency for lower levels of endogenous adenosine ensuring A1 receptor-mediated inhibition. In spite of similar behavioral and neurophysiological function, caffeine intake increased the energy charge and redox state of cortical synaptosomes. This increased metabolic competence likely involved a putative increase in the glycolytic rate in synapses and a prospective greater astrocyte–synapse lactate shuttling. It was concluded that caffeine intake does not trigger evident alterations of behavior or of synaptic plasticity but increases the metabolic competence of synapses, which might be related with the previously described better ability of animals consuming caffeine to cope with deleterious stimuli triggering brain dysfunction.

Los estilos APA, Harvard, Vancouver, ISO, etc.

Más fuentes

Ofrecemos descuentos en todos los planes premium para autores cuyas obras están incluidas en selecciones literarias temáticas. ¡Contáctenos para obtener un código promocional único!