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Libros sobre el tema "Cartilage cells"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 5 de marzo de 2023

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1

1933-, Malinin Theodore I., ed. Microscopic and histochemical manifestations of hyaline cartilage dynamics. Jena: Urban & Fischer Verlag, 1999.

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2

Malinin, George I. Microscopic and histochemical manifestations of hyaline cartilage dynamics. Jena, Germany: Urban & Fischer, 1999.

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3

F, Stoltz J., and International Symposium on Mechanobiology of Cartilage and Chondrocyte (1st : 1999 : Sainte-Maxime, France), eds. Mechanobiology: Cartilage and chondrocyte. Amsterdam: IOS Press, 2000.

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4

Khan, Wasim S. Stem cells and cartilage tissue engineering approaches to orthopaedic surgery. Hauppauge, N.Y: Nova Science Publishers, 2009.

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5

International, Workshop on Cells and Cytokines in Bone and Cartilage (3rd 1990 Davos Switzerland). Third International Workshop on Cells and Cytokines in Bone and Cartilage: 8-11 April 1990, Davos, Switzerland : abstracts. New York, N.Y: Springer International, 1990.

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6

International Workshop on Cells and Cytokines in Bone and Cartilage (2nd 1988 Davos, Switzerland). Second International Workshop on Cells and Cytokines in Bone and Cartilage: 9-12 April 1988, Davos, Switzerland : abstracts. New York, N.Y: Springer International, 1988.

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7

International Workshop on Cells and Cytokines in Bone and Cartilage (4th 1992 Davos, Switzerland). Fourth Workshop on Cells and Cytokines in Bone and Cartilage: January 11-14, 1992, Davos, Switzerland : abstracts. New York, N.Y: Springer International, 1992.

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8

F, Stoltz J., ed. Mechanobiology: Cartilage and chondrocyte. Amsterdam: IOS Press, 2006.

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9

Mechanobiology: Cartilage and chondrocyte. Amsterdam: IOS Press, 2008.

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10

Sampat, Sonal Ravin. Optimization of Culture Conditions for Cartilage Tissue Engineering Using Synovium-Derived Stem Cells. [New York, N.Y.?]: [publisher not identified], 2014.

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11

Tetlow, Lynne Christine. Histopathology of the rheumatoid lesion: Mast cells, metalloproteinases and cytokinesat sites of cartilage erosion. Manchester: University of Manchester, 1996.

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12

Regulation of Chondrogenesis in Human Mesenchymal Stem Cells by Cartilage Extracellular Matrix and Therapeutic Applications. [New York, N.Y.?]: [publisher not identified], 2018.

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13

Richard, Barrett-Jolley, Beck F. (Friedrich) 1927-, Bondy Carolyn A, Clascá F, Frotscher M. (Michael) 1947-, Haines Duane E, Hirokawa Nobutaka, et al., eds. Facilitative Glucose Transporters in Articular Chondrocytes: Expression, Distribution and Functional Regulation of GLUT Isoforms by Hypoxia, Hypoxia Mimetics, Growth Factors and Pro-Inflammatory Cytokines. Berlin, Heidelberg: Springer-Verlag Berlin Heidelberg, 2008.

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14

M, Shapiro Irving, Boyan Barbara, and Anderson H. Clarke, eds. The growth plate. Amsterdam: IOS Press, 2002.

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15

Monique, Adolphe, ed. Biological regulation of the chondrocytes. Boca Raton, Fla: CRC Press, 1992.

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16

Embryonic stem cell therapy for osteo-degenerative diseases: Methods and protocols. New York, NY: Humana Press, 2011.

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17

Gregory, Bock, and Goode Jamie, eds. Tissue engineering of cartilage and bone. Chichester, West Sussex: J. Wiley, 2003.

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18

Jūryūshi Ikagaku Sentā. Shinpojūmu "Saisei Iryō to Bunshi Imējingu." Dai 3-kai Jūryūshi Ikagaku Sentā Shinpojūmu Saisei Iryō to Bunshi Imējingu. Chiba-ken Chiba-shi: Hōshasen Igaku Sōgō Kenkyūjo, 2004.

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19

Lau, Wing Fun. Regulation of cartilage differentiation and formation in a chondrogenic cell line by retinoic acid, dexamethasone and 1,25-dihydroxyvitamin Df3d. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1993.

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20

Cardew, Gail. The molecular basis of skeletogenesis. Chichester: Wiley, 2001.

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21

Taehakkyo, Chŏnbuk, ред. Nano haibŭridŭ sojae wa kanyŏp chulgi sepʻo rŭl iyonghan kol mit yŏnʼgol chaesaeng kisul kaebal e kwanhan yŏnʼgu: Chʻoejong yŏnʼgu kaebal kyŏlgwa pogosŏ = A study on the development of cartilage and bone regeneration by nanohybrid biomaterials with mesenchymal stem cell. [Seoul]: Pogŏn Pokchibu, 2004.

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22

Sabatini, Massimo, Philippe Pastoureau, and Frédéric de Ceuninck. Cartilage and Osteoarthritis. Humana Press, 2010.

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23

Sabatini, Massimo, Philippe Pastoureau, and Frédéric de Ceuninck. Cartilage and Osteoarthritis. Humana Press, 2010.

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24

Cartilage and osteoarthritis. Totowa, NJ: Humana Press, 2004.

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25

(Editor), Massimo Sabatini, Philippe Pastoureau (Editor), and FrTdTric De Ceuninck (Editor), eds. Cartilage and Osteoarthritis (Methods in Molecular Medicine). Humana Press, 2004.

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26

(Foreword), L. Peterson, B. J. Cole (Foreword), and Riley J. Williams (Editor), eds. Cartilage Repair Strategies. Humana Press, 2007.

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27

(Editor), Frédéric De Ceuninck, Massimo Sabatini (Editor), and Philippe Pastoureau (Editor), eds. Cartilage and Osteoarthritis (Methods in Molecular Medicine). Humana Press, 2004.

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28

Mechanobiology: Cartilage and Chondrocyte (Biomedical and Health Research). Ios Pr Inc, 2002.

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29

Stoltz, J. F. Mechanobiology: Cartilage And Chondrocyte (Biomedical and Health Research). Ios Pr Inc, 2004.

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30

Muldrew, Kenneth B. The osmotic rupture hypothesis and its application to the cryopreservation of articular cartilage. 1993.

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31

MECHANOBIOLOG, INTERNATIONAL SYMPOSIUM ON, and J. F. Stoltz. Mechanobiology: Cartilage and Chondrocyte, Vol. 4 (Biomedical and Health Research, Vol. 68) (Biomedical and Health Research). IOS Press, 2006.

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32

The Effect of Titanium Surface Roughness on Growth, Differentiation, and Protein Synthesis of Cartilage and Bone Cells. Storming Media, 1996.

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33

(Editor), Irving M. Shapiro, Barbara Boyan (Editor), and H. Clarke Anderson (Editor), eds. The Growth Plate (Biomedical and Health Research, 54). Ios Pr Inc, 2002.

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34

Adolphe, Monique. Biological Regulation of the Chondrocytes. CRC Press LLC, 2022.

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35

Adolphe, Monique. Biological Regulation of the Chondrocytes. CRC Press LLC, 2022.

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36

Adolphe, Monique. Biological Regulation of the Chondrocytes. CRC Press LLC, 2022.

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37

Adolphe, Monique. Biological Regulation of the Chondrocytes. CRC Press LLC, 2022.

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38

Goldring, Steven R. Pathophysiology of periarticular bone changes in osteoarthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0005.

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Under physiological conditions, the subchondral bone of diarthrodial joints such as the hip, knee, and phalanges forms an integrated biocomposite with the overlying calcified and hyaline articular cartilage that is optimally organized to transfer mechanical load. During the evolution of the osteoarthritic process both the periarticular bone and cartilage undergo marked changes in their structural and functional properties in response to adverse biomechanical and biological signals. These changes are mediated by bone cells that modify the architecture and properties of the bone through active c
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39

Pitzalis, Costantino, Frances Humby, and Michael P. Seed. Synovial pathology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0052.

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Synovial pathology is seen in a variety of disease states, including rheumatoid arthritis (RA), osteoarthritis (OA), psoriatic arthritis, and systemic lupus erythmatosus (SLE). This chapter highlights recent advances that characterize the cellular composition of these tissues according to surface markers and chemokine and cytokine expression, and describes synovial functional status and response to therapeutics. In RA, after initiation, pannus migrates over and under cartilage, and into subchondral bone, in a destructive process. Cartilage-pannus junction (CPJ) is characterized as invasive or
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40

Douglas, Kenneth. Bioprinting. Oxford University Press, 2021. http://dx.doi.org/10.1093/oso/9780190943547.001.0001.

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Abstract: This book describes how bioprinting emerged from 3D printing and details the accomplishments and challenges in bioprinting tissues of cartilage, skin, bone, muscle, neuromuscular junctions, liver, heart, lung, and kidney. It explains how scientists are attempting to provide these bioprinted tissues with a blood supply and the ability to carry nerve signals so that the tissues might be used for transplantation into persons with diseased or damaged organs. The book presents all the common terms in the bioprinting field and clarifies their meaning using plain language. Readers will lear
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41

Shakibaei, Mehdi, Constanze Csaki, and Ali Mobasheri. Diverse Roles of Integrin Receptors in Articular Cartilage. Springer, 2008.

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42

Fernandes, Tiago Lazzaretti, Daniela Franco Bueno, Kazunori Shimomura, Zhenxing Shao, and Andreas H. Gomoll, eds. Tissue Engineering and Cell Therapy for Cartilage Restoration. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-83250-145-0.

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43

Narcisi, Roberto, and Elena Jones, eds. Cell-Based Approaches for Modulating Cartilage and Bone Phenotype. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88971-855-9.

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44

Thomas, Ranjeny, and Andrew P. Cope. Pathogenesis of rheumatoid arthritis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0109.

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In depth molecular and cellular analysis of synovial tissue and fluid from patients with rheumatoid arthritis has provided important insights into understanding disease pathogenesis. Advances in the 1980s and 1990s included modern cloning strategies, sensitive and specific assays for inflammatory mediators, production of high-affinity neutralizing monoclonal antibodies, advances in flow cytometry, and gene targeting and transgenic strategies in rodents. In the 21st century, technological platforms offer unparalleled opportunities for systematic and unbiased interrogation of the disease process
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45

Diverse Roles Of Integrin Receptors In Articular Cartilage. Springer, 2008.

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46

Narcisi, Roberto, and Eric Farrell, eds. Understanding and Modulating Bone and Cartilage Cell Fate for Regenerative Medicine. Frontiers Media SA, 2019. http://dx.doi.org/10.3389/978-2-88945-790-8.

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47

Tissue Engineering of Cartilage and Bone - No. 249 (Novartis Foundation Symposia). Wiley, 2003.

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48

Abhishek, Abhishek, and Michael Doherty. Pathophysiology of calcium pyrophosphate deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0049.

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Calcium pyrophosphate (CPP) dihydrate crystals form extracellularly. Their formation requires sufficient extracellular inorganic pyrophosphate (ePPi), calcium, and pro-nucleating factors. As inorganic pyrophosphate (PPi) cannot cross cell membranes passively due to its large size, ePPi results either from hydrolysis of extracellular ATP by the enzyme ectonucleotide pyrophosphatase/phosphodiesterase 1 (also known as plasma cell membrane glycoprotein 1) or from the transcellular transport of PPi by ANKH. ePPi is hydrolyzed to phosphate (Pi) by tissue non-specific alkaline phosphatase. The level
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49

Control of Tissue Damage (Research Monographs in Cell & Tissue Physiology). Elsevier Science Ltd, 2000.

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50

The Molecular Basis of Skeletogenesis No. 232. Wiley, 2001.

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