Bibliografías temáticas / Cell wall deficiency

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Literatura académica sobre el tema "Cell wall deficiency"

Autor: Grafiati
Publicado: 22 de febrero de 2025
Última modificación: 31 de julio de 2025

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Artículos de revistas sobre el tema "Cell wall deficiency"

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Liao, Ya-Yun, Thomas J. Buckhout, and Wolfgang Schmidt. "Phosphate deficiency-induced cell wall remodeling." Plant Signaling & Behavior 6, no. 5 (2011): 700–702. http://dx.doi.org/10.4161/psb.6.5.15051.

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Ridge, S. C., J. B. Zabriskie, H. Osawa, T. Diamantstein, A. L. Oronsky, and S. S. Kerwar. "Administration of group A streptococcal cell walls to rats induces an interleukin 2 deficiency." Journal of Experimental Medicine 164, no. 1 (1986): 327–32. http://dx.doi.org/10.1084/jem.164.1.327.

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Intraperitoneal administration of group A streptococcal cell walls to Lewis rats induces a chronic arthritis, whereas the Fischer strain is resistant to the development of the lesion. Spleen cells from cell wall-treated rats (Lewis and Fischer) are deficient in the synthesis of IL-2. Using an mAb directed against the rat IL-2-R, the present studies indicate that the expression of IL-2-R on spleens of cell wall-treated rats is normal. However, the addition of exogenous IL-2 to spleen cells cultured with Con A does not stimulate the mitogenic response.
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Liang, Wensheng, Xiang Gao, Yang She, Xin Jing, Xiaolong Yuan, and Derui Zhu. "Adaptation to Long-Term Nitrogen Starvation in a Biocrust-Derived Microalga Vischeria sp. WL1: Insights into Cell Wall Features and Desiccation Resistance." Microorganisms 13, no. 4 (2025): 903. https://doi.org/10.3390/microorganisms13040903.

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In drylands, microalgae dwelling in the biocrust are inevitably confronted with nitrogen deficiency and desiccation stress, despite the protection afforded by the soil biological complex. However, the environmental adaptive features and mechanisms of these microalgae remain largely unknown. In this study, we explored the adaptive changes of a biocrust-derived unicellular microalga, Vischeria sp. WL1 (Eustigmatophyceae), in the face of long-term nitrogen deficiency. Attention was focused on the alterations in cell wall properties and the associated desiccation resistance. After exposure to long
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Wu, Weiwei, Shengnan Zhu, Qianqian Chen, Yan Lin, Jiang Tian, and Cuiyue Liang. "Cell Wall Proteins Play Critical Roles in Plant Adaptation to Phosphorus Deficiency." International Journal of Molecular Sciences 20, no. 21 (2019): 5259. http://dx.doi.org/10.3390/ijms20215259.

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Phosphorus is one of the mineral nutrient elements essential for plant growth and development. Low phosphate (Pi) availability in soils adversely affects crop production. To cope with low P stress, remodeling of root morphology and architecture is generally observed in plants, which must be accompanied by root cell wall modifications. It has been documented that cell wall proteins (CWPs) play critical roles in shaping cell walls, transmitting signals, and protecting cells against environmental stresses. However, understanding of the functions of CWPs involved in plant adaptation to P deficienc
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Vitković, Ljubiša. "Wall turnover deficiency of Bacillus subtilis Nil5 is due to a decrease in teichoic acid." Canadian Journal of Microbiology 33, no. 6 (1987): 566–68. http://dx.doi.org/10.1139/m87-096.

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Bacillus subtilis Ni15 is deficient in cell wall turnover. This deficiency is removed if the medium contains 0.2 M NaCl, which does not affect growth. The levels of amidase and glucosaminidase, the most likely enzymes involved in turnover, were, in stationary phase Nil5 cells, similar to those in late-exponential phase cells of a standard strain. The Nil5 enzymes were not salt sensitive. However, the Nil5 walls contained 4.7-fold less phosphorus than the walls of the standard strain. Since the phosphorus content of B. subtilis walls reflects the level of teichoic acid, it is proposed that the
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Zhang, Cheng, Mingliang He, Zhexuan Jiang, et al. "The Xyloglucan Endotransglucosylase/Hydrolase Gene XTH22/TCH4 Regulates Plant Growth by Disrupting the Cell Wall Homeostasis in Arabidopsis under Boron Deficiency." International Journal of Molecular Sciences 23, no. 3 (2022): 1250. http://dx.doi.org/10.3390/ijms23031250.

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TCH4 is a xyloglucan endotransglucosylase/hydrolase (XTH) family member. Extensive studies have shown that XTHs are very important in cell wall homeostasis for plant growth and development. Boron (B), as an essential micronutrient for plants, plays an essential role in the cross-linking of cell wall pectin. However, the effect of B on cell wall organization is unclear. This study aimed to explore the mechanism of plant adaption to B stress by investigating the role of TCH4 in cell wall homeostasis. We conducted both plate and hydroponic cultures of wild-type Col-0 and overexpression and gene k
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Ongenae, Véronique, Ariane Briegel, and Dennis Claessen. "Cell wall deficiency as an escape mechanism from phage infection." Open Biology 11, no. 9 (2021): 210199. http://dx.doi.org/10.1098/rsob.210199.

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The cell wall plays a central role in protecting bacteria from some environmental stresses, but not against all. In fact, in some cases, an elaborate cell envelope may even render the cell more vulnerable. For example, it contains molecules or complexes that bacteriophages recognize as the first step of host invasion, such as proteins and sugars, or cell appendages such as pili or flagella. In order to counteract phages, bacteria have evolved multiple escape mechanisms, such as restriction-modification, abortive infection, CRISPR/Cas systems or phage inhibitors. In this perspective review, we
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Yin, Qi, Lu Kang, Yi Liu, et al. "Boron deficiency disorders the cell wall in Neolamarckia cadamba." Industrial Crops and Products 176 (February 2022): 114332. http://dx.doi.org/10.1016/j.indcrop.2021.114332.

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Claessen, Dennis, and Jeff Errington. "Cell Wall Deficiency as a Coping Strategy for Stress." Trends in Microbiology 27, no. 12 (2019): 1025–33. http://dx.doi.org/10.1016/j.tim.2019.07.008.

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Gutierrez-Armijos, L. Roxana, Rodrigo A. C. Sussmann, Ariel M. Silber, Mauro Cortez, and Agustín Hernández. "Abnormal sterol-induced cell wall glucan deficiency in yeast is due to impaired glucan synthase transport to the plasma membrane." Biochemical Journal 477, no. 24 (2020): 4729–44. http://dx.doi.org/10.1042/bcj20200663.

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Abnormal sterols disrupt cellular functions through yet unclear mechanisms. In Saccharomyces cerevisiae, accumulation of Δ8-sterols, the same type of sterols observed in patients of Conradi–Hünermann–Happle syndrome or in fungi after amine fungicide treatment, leads to cell wall weakness. We have studied the influence of Δ8-sterols on the activity of glucan synthase I, the protein synthetizing the main polymer in fungal cell walls, its regulation by the Cell Wall Integrity (CWI) pathway, and its transport from the endoplasmic reticulum to the plasma membrane. We ascertained that the catalytic
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Tesis sobre el tema "Cell wall deficiency"

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Fuller, Elizabeth R. "Cell wall-deficiency in Staphylococcus aureus and its role in antibiotic resistance." Thesis, University of Newcastle Upon Tyne, 2008. http://hdl.handle.net/10443/1671.

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Cell Wall-Deficiency in Staphylococcus aureus and its Role in Antibiotic Resistance. Elizabeth R. Fuller. Cell wall-deficient bacteria (CWDB) induced from Staphylococcus aureus ATCC 9144 (Oxford strain) were generated on medium with elevated osmolality in the presence of sublethal levels of penicillin G. On removal of antibiotic pressure the cell wall-competent (CWC) revertants along with these CWDB exhibited high-level penicillin and methicillin resistance, which was stable in the revertants. The revertants looked visually different, had an altered Gram stain and growth rate. Their matrixassi
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Dell'Era, Simone. "Morphological and molecular characterization of cell wall-deficient L-forms of L. monocytogenes /." Zürich : ETH, 2007. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=17588.

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Palaiodimou, Lydia. "Molecular insights into Listeria monocytogenes persistence via label-free quantitative proteomics." Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPASL115.

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Listeria monocytogenes (Lm) est un pathogène intracellulaire facultatif responsable de la listériose, une infection alimentaire particulièrement dangereuse pour les femmes enceintes et les personnes immunodéprimées. Connue pour son adaptabilité, Lm persiste dans divers environnements, rendant son contrôle difficile. Lors d'infections prolongées de cellules épithéliales, telles que les hépatocytes et les trophoblastes, Lm passe d'un état réplicatif à un état quiescent au sein de vacuoles de type lysosomal, appelées Listeria-containing vacuoles (LisCVs). Cette transition est associée à la perte
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Libros sobre el tema "Cell wall deficiency"

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Mattman, Lida H. Cell Wall Deficient Forms: Stealth Pathogens. Taylor & Francis Group, 2000.

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Mattman, Lida H. Cell Wall Deficient Forms: Stealth Pathogens. Taylor & Francis Group, 2009.

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Mattman, Lida H. Cell Wall Deficient Forms: Stealth Pathogens. Taylor & Francis Group, 2000.

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Mattman, Lida H. Cell Wall Deficient Forms: Stealth Pathogens, Fourth Edition. CRC, 2009.

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Mattman, Lida H. Cell Wall Deficient Forms: Stealth Pathogens, Third Edition. 3rd ed. CRC, 2000.

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Mattman, Lida H. Cell Wall Deficient FormsStealth Pathogens. Taylor & Francis Group, 2000.

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Cell wall deficient forms: Stealth pathogens. 3rd ed. CRC Press, 2001.

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Mattman, Lida H. Cell Wall Deficient Forms, Third Edition. Taylor & Francis Group, 2000.

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Cell wall deficient forms: Stealth pathogens. 2nd ed. CRC Press, 1993.

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Cell Wall Deficient Forms: Stealth Pathogens, Fourth Edition. CRC Press LLC, 2014.

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Capítulos de libros sobre el tema "Cell wall deficiency"

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Bonilla, I., H. Perez, G. Cassab, M. Lara, and F. Sanchez. "The effect of boron deficiency on development in determinate nodules: changes in cell wall pectin contents and nodule polypeptide expression." In Boron in Soils and Plants. Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5564-9_42.

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Findeklee, Peter, Monika Wimmer, and Heiner E. Goldbach. "Early effects of boron deficiency on physical cell wall parameters, hydraulic conductivity and plasmalemma-bound reductase activities in young C. pepo and V. faba roots." In Boron in Soils and Plants. Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5564-9_43.

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Koch, Arthur L. "Spirochetes and Spiroplasma and the Special Strategies for CWD (Cell Wall Deficient) Cells." In The Bacteria: Their Origin, Structure, Function and Antibiosis. Springer Netherlands, 2007. http://dx.doi.org/10.1007/978-1-4020-3206-6_15.

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Allan, D. L., J. R. Shann, and P. M. Bertsch. "Role of root cell walls in iron deficiency of soybean (Glycine max) and aluminium toxicity of wheat (Triticum aestivum)." In Plant Nutrition — Physiology and Applications. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-0585-6_58.

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"Fungi Recognizing Wall Deficiency of Fungi." In Cell Wall Deficient Forms. CRC Press, 2000. http://dx.doi.org/10.1201/b16928-31.

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Markova, Nadya. "Cell Wall Deficiency in Mycobacteria: Latency and Persistence." In Understanding Tuberculosis - Deciphering the Secret Life of the Bacilli. InTech, 2012. http://dx.doi.org/10.5772/30919.

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Chambers, James. "I-Cell Disease (Mucolipidosis II)." In Clinical Studies in Medical Biochemistry. Oxford University PressNew York, NY, 2006. http://dx.doi.org/10.1093/oso/9780195176872.003.0017.

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Abstract The patient (Fig. 17-1) was a 38-week-gestation product of a 22-year-old G,P1Abo (gravida 1, first pregnancy; paradelivery, first delivery; abortion, 0) woman and her 31-year-old firstcousin partner. Pregnancy was complicated by oligohydramnios (deficiency in the amount of amniotic fluid); an amniocentesis revealed a normal male karyotype. The infant was delivered by cesarean section because of maternal preeclampsia (development of hypertension due to pregnancy). Apgar scores were 8/9, weight was 2200 g, and length was 18.5 inches. On newborn examination, microcephaly (a broad nasal b
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Chambers, James. "I-Cell Disease (Mucolipidosis II)." In Clinical Studies In Medical Biochemistry. Oxford University PressNew York, NY, 2006. http://dx.doi.org/10.1093/oso/9780195147322.003.0017.

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Abstract The patient (Fig. 17-1) was a 38-week-gestation product of a 22-year-old G,P1Abo (gravida 1, first pregnancy; paradelivery, first delivery; abortion, 0) woman and her 31-year-old first-cousin partner. Pregnancy was complicated by oligohydramnios (deficiency in the amount of amniotic fluid); an amniocentesis revealed a normal male karyotype. The infant was delivered by cesarean section because of maternal preeclampsia (development of hypertension due to pregnancy). Apgar scores were 8/9, weight was 2200 g, and length was 18.5 inches. On newborn examination, microcephaly (a broad nasal
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"Disclosures by Electron Microscopy." In Cell Wall Deficient Forms. CRC Press, 2000. http://dx.doi.org/10.1201/b16928-10.

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"Public Health and Nosocomial Facets Salmonella and Shigella Carriers." In Cell Wall Deficient Forms. CRC Press, 2000. http://dx.doi.org/10.1201/b16928-11.

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Actas de conferencias sobre el tema "Cell wall deficiency"

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Gruber, Matthew J., Varun Krishnamurthy, D. A. Narmoneva, and Robert B. Hinton. "Elastin Haploinsufficiency Is Associated With Altered Interstitial Phenotype and Progressive Aortopathy." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192891.

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Supravalvular aortic stenosis (SVAS) [1] is a disease of the cardiovascular system that leads to narrowing of the large arteries in humans. Studies have shown [2] that SVAS is caused by mutations or deletions in the elastin gene resulting in elastin haploinsufficiency. Elastin haploinsufficiency results in systemic hypertension [3], thinner and more numerous elastic lamellae [4], and altered arterial mechanics [5]. Genetically modified elastin deficient mice (ELN+/-) recapitulates the human phenotype including obstructive arterial disease and decreased arterial compliance [1,3]. Elastin defici
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Bian, Shiyao, Ying Zheng, Shuichi Takayama, and James B. Grotberg. "Micro-PIV Measurements of an Airway Closure Model." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206831.

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A thin liquid layer coating the airway can be unstable and forms a plug. Airway closure usually happens at the small airways near the end of expiration, often accompanied with hypersecretion or/and surfactant deficiency in the airway in a variety of lung diseases, such as chronic obstructive pulmonary disease (COPD) and acute respiratory distress syndrome (ARDS). Modeling work by Halpern and Grotberg [1] has shown that several forces could contribute to airway closure, such as the surface tension instability and the wall compliance. Experiments in a capillary tube were conducted by Cassidy et
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WAJIMA, T. "THROMBOCYTOPENIA IN ACQUIRED IMMUNE DEFICIENCY SYNDRGME(AIDS)-RELATED COMPLEXES:RESOLUTION DURING HERPES VIRAL INFECTION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644145.

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Immune thrombocytopenia has been recognized as a major hematologic manifestation associated with the acquired immune deficiency syndrome(AIDS) and AIDS-related complexes. The mechanism of thrombocytopenia in human immune deficiency virus infection is probably multifactorial.. The role of platelet-associated immuno-globulin(PAIgG)and circulating immune complexes(CIC) in mediating thrombocytopenia is controversial.. We experienced a case in which immune thrombocytopenia in AIDS-related complexes resolved during herpes zoster infection. A 37 y.o. white homosexual male with AIDS-related complexes
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Tengborn, L., and A. Wallmark. "ALTERATIONS IN THE COAGULATION AND FIBRINOLYTIC SYSTEMS AS PREDISPOSING FACTORS IN THE DEVELOPMENT OF DEEP VENOUS THROMBOSIS (DVT)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644203.

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From Jan. 1982 to Jan. 1987, 1213 patients were investigated either because of thrombotic episode(s) or thrombosis heredity. Diagnosis of DVT was confirmed by phlebography in 567 cases. Patients were first examined at least three months after an acute episode.Methods. Anti thrombin (AT) and plasminogen were assayed, using chromogenic substrates S-2238 and S-2251 , respectively, followed if values were low by immunochemical assessment. Furthermore, fibrinogen, thrombin and reptilase times, APTT, P…P were assessed. Only in the latter study period was protein C activity determined (129 cases). Fi
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Espinosa, Gabriela, Lisa Bennett, William Gardner, and Jessica Wagenseil. "The Effects of Extracellular Matrix Protein Insufficiency and Treatment on the Stiffness of Arterial Smooth Muscle Cells." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14131.

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Increased arterial stiffness is directly correlated with hypertension and cardiovascular disease. Stiffness of the conducting arteries is largely determined by the extracellular matrix (ECM) proteins in the wall, such as collagen and elastin, produced by the smooth muscle cells (SMCs) found in the medial layer. Elastin is deposited as soluble tropoelastin and is later crosslinked into elastin fibers. Newborn mice lacking the elastin protein ( Eln−/−) have increased arterial wall stiffness and SMCs with altered proliferation, migration and morphology [1]. Vessel elasticity is also mediated by o
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Takeshige, T., I. Fuse, I. Hattori, T. Momotsu, A. Shibata, and K. Abe. "NORMAL URINARY PROSTAGLANDIN E2 EXCRETION IN THE PATIENT WITH PLATELET CYCLO-OXYGENASE DEFICIENCY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644879.

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Platelet (P) Cyclo-oxygenase (CO) deficiency is characterized by bleeding tendency due to platelet release mechanism defect. Pareti et al. have reported a patient with PCO deficiency whose CO activity was defective not only in platelets but also in vessel walls. But it has not been demonstrated whether other cells than platelets do have this enzyme in other.patients with PCO deficiency. Recent data suggest taht hemorrhagic or thrombotic diathesis is dependent on the site(s) (platelet, vessel wall or others) or CO deficiency. In order to determine the CO activity in kidney in a patient with PCO
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Keyes, Joseph T., Stacy Borowicz, Urs Utzinger, Mohamad Azhar, and Jonathan P. Vande Geest. "Quantification of the Biomechanical Differences in Wild-Type and Heterozygous TGF Beta2 Knockout Mice." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19482.

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The use of transgenic mice is an incredibly powerful tool in understanding the underlying etiology of disease. To understand the usefulness of specific transgenic mice, the systems of interest should be characterized. We have created TGFβ2-deficient mouse fetuses that develop widespread aortic and coronary artery aneurysms [1]. Several studies have pointed to a strong connection between elevated TGFβ signaling and aortic aneurysm [2]. In situ hybridization has shown that Tgfb2 and Tgfb3 are major ligands expressed in the aortic medial wall. Further reduction of TGFβ signaling by combining TGFβ
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Teitel, J. M., M. B. Garvey, and J. J. Freedman. "THE ENDOTHELIAL CELL AS THE SII$ OF THE FACTOR VIII BYPASSING ACTIVITY OF PROTHROMBIN COMPLEX CONCENTRATE (PCC)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644731.

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PCC is used empirically for its factor VIII (FVIII) bypassing activity in hemophiliacs with inhibitors to FVIII. The effect of of PCC on plasma coagulation tests is variable and is not predictive of clinical response. We are testing the hypothesis that bypass activity is expressed locally at the vessel wall, which functions as a procoagulant surface at times of hemostatic stress. Monolayer cultures of umbilical vein endothelial cells (EC) were incubated for 1 hour with commerical PCC (1 u/ml), and then extensively washed to remove fluid phase PCC components. Ellagic acid-activated PTT determin
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Dooi jewaard, G., D. J. Binnema, and C. Kluft. "CONTACT ACTIVATION AND SINGLE-CHAIN UROKINASE-TYPE PLASMINOGEN ACTIVATOR." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642958.

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For many years it is known that activation of the factor XII (FXII) -prekallikrein (PKK)- kininogen system of coagulation (contact activation) also may be involved in activation of fibrinolysis. Despite the numerous efforts over the past two decades to clarify this process, our current insights in this matter are far from complete. Also the physiological meaning of this possible interlinkage of coagulation and fibrinolysis is still uncertain; clearcut clinical manifestations in patients deficient in FXII or PKK are not found.No doubt, activation of fibrinolysis is a much more complicated proce
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Informes sobre el tema "Cell wall deficiency"

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Harman, Gary E., and Ilan Chet. Discovery and Use of Genes and Gene Combinations Coding for Proteins Useful in Biological Control. United States Department of Agriculture, 1994. http://dx.doi.org/10.32747/1994.7568787.bard.

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The objectives of the research in this proposal were to (A) identify synergy among proteins that provide enhanced activity over single proteins for control of plant pathogenic fungi, (B) clone and characterize genetic sequences coding for proteins with ability to control pathogenic fungi, (C) produce transgenic organisms with enhanced biocontrol ability using genes and gene combinations and determine their efficiency in protecting plants against plant pathogenic fungi. A related objective was to produce disease-resistant plants. Fungal cell wall degrading enzymes from any source are strongly s
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Thomashow, Linda, Leonid Chernin, Ilan Chet, David M. Weller, and Dmitri Mavrodi. Genetically Engineered Microbial Agents for Biocontrol of Plant Fungal Diseases. United States Department of Agriculture, 2005. http://dx.doi.org/10.32747/2005.7696521.bard.

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The objectives of the project were: a) to construct the site-specific integrative expression cassettes carrying: (i) the chiA gene for a 58-kDa endochitinase, (ii) the pyrrolnitrin biosynthesis operon, and (iii) the acdS gene encoding ACC deaminase; b) to employ these constructs to engineer stable recombinant strains with an expanded repertoire of beneficial activities; c) to evaluate the rhizosphere competence and antifungal activity of the WT and modified strains against pathogenic fungi under laboratory and greenhouse conditions; and d) to monitor the persistence and impact of the introduce
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