Bibliografías temáticas / Cellular Proliferation / Tesis
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Tesis sobre el tema "Cellular Proliferation"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 25 de julio de 2025

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1

Gan, Lisha. "Corneal cellular proliferation and wound healing /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4505-5/.

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Kranc, Kamil. "The role of Cited2 in cellular proliferation." Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.398233.

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3

PIUNTI, ANDREA. "POLYCOMB ROLE IN CELLULAR PROLIFERATION AND TRANSFORMATION." Doctoral thesis, Università degli Studi di Milano, 2014. http://hdl.handle.net/2434/696485.

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The Polycomb Group proteins (PcGs) are present in cells nuclei as two main repressive complexes named Polycomb Repressive Complex 1 (PRC1) and 2 (PRC2). Both have been involved in several cellular functions among which the ability to promote cellular proliferation is the main PcG feature that links their activity to cancer development. Both complexes are directly involved in repressing the transcription of the Ink4aArf locus that encodes for the tumor suppressive proteins p16 and p19/Arf (p14/Arf in humans), potent inhibitors of cell growth via the positive regulation of pRb and p53 functions.
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4

Sangfelt, Olle. "Effects of interferon on cellular proliferation and apoptosis /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19981014sang.

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5

Stacy, Andrew Jared. "Regulation of ΔNp63α by TIP60 promotes cellular proliferation". Wright State University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1596151919161674.

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6

Chakravarthy, Usha. "The effect of gamma radiation on intraocular cellular proliferation." Thesis, Queen's University Belfast, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317046.

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7

Maiti, Baidehi. "E2F and survivin - key players in cellular proliferation and transformation." Columbus, Ohio : Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1173801044.

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8

Khav, Eddie. "Visualizing an RB-E2F Cellular Switch that Controls Cell Proliferation." Thesis, The University of Arizona, 2013. http://hdl.handle.net/10150/297627.

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Mammalian cell proliferation is regulated by an Rb-E2F gene network. The input node of this network, Cyclin D, receives graded growth signals; the output node, E2F, generates an all-or-none response. That is, the Rb-E2F gene network functions as a cellular switch, converting analog growth signals into digital E2F activities. The On or Off of this Rb-E2F switch determines the On or Off of cell proliferation. To help better understand the analog/digital conversion mechanism, we constructed a reporter cell line to visualize the dynamic expression of Cyclin D and E2F genes by red and green fluores
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9

Simmons, Ambrosia. "The Role of Polarity Complex Proteins in Neural Progenitor Proliferation." Diss., Temple University Libraries, 2019. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/552083.

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Biomedical Sciences<br>Ph.D.<br>Cortical malformations arise from defects in any stage of brain development and often result in life-long disability ranging from epilepsy to developmental delay and even perinatal lethality. The neuroepithelium of the emergent cortex lays the foundation on which the future cortex will develop, and as such, neuroepithelial tissue and the neural progenitor cells (NPCs) which comprise it are critical to the proper growth and development of the cortex. Here I demonstrate the significance of neuroepithelial cell polarity determinants in cortical development and how
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10

Reed, Jennifer. "Interferon-gamma increases CD4+ T cell survival and proliferation." Click here for download, 2006. http://wwwlib.umi.com/cr/villanova/fullcit?p1432655.

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11

Anderson, Elizabeth. "Co-ordinate regulation of cellular proliferation and apoptosis in rodent liver." Thesis, University of Surrey, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.441719.

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12

Gardner, David Paul. "Epidermal growth factor receptor: Regulation of cellular proliferation and gene expression." Diss., The University of Arizona, 1993. http://hdl.handle.net/10150/186438.

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Binding of epidermal growth factor (EGF) to the EGF receptor stimulates the tyrosine kinase activity of the receptor and initiates a signal transduction cascade culminating in a mitogenic response. In many tumor derived cell lines which overexpress EGF receptor, exposure to EGF results in growth inhibition. The mechanism for this is unclear. This work involves analysis of growth inhibition by EGF, mechanisms of EGF receptor overexpression and regulation of the EGF receptor gene. The first two studies utilize a cell line (PC-10) that overexpresses EGF receptor without gene amplification. PC-10
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13

Mosca, Matthieu. "Etude du mécanisme d'action de l'interféron alpha dans les néoplasmes myéloprolifératifs classiques." Thesis, Université Paris-Saclay (ComUE), 2018. http://www.theses.fr/2018SACLS516/document.

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Les néoplasmes myéloprolifératifs classiques sont des maladies clonales dues à des mutations acquises de JAK2V617F ou de la Calréticuline (CALRm) au niveau des cellules souches hématopoïétiques (CSH) et conduisant à une surproduction de cellules myéloïdes. L’interféron alpha (IFNa) est le seul traitement curatif qui induit une réponse hématologique et moléculaire. Le but de notre projet est de comprendre son mécanisme d’action en utilisant une cohorte de 47 patients, des lignées cellulaires et des modèles de souris JAK2V617F. Ainsi, nous avons constaté que l’IFNa cible plus
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14

Zettervall, Carl-Johan. "Signaling pathways in the activation and proliferation of Drosophila melanogaster blood cells." Doctoral thesis, Umeå : Umeå centrum för molekylär patogenes (UCMP), 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-513.

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15

Conway-Campbell, Becky Lee. "A novel role for the nuclear growth hormone receptor in cellular proliferation /." St. Lucia, Qld, 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17889.pdf.

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16

Harper, Jane Vera. "Investigation into the role of T-type calcium channels in cellular proliferation." Thesis, University of Reading, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.397848.

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17

Gaisford, Esme Ann. "Rab-Vesicle Trafficking is Required for Ras-Mediated Proliferation." Master's thesis, Temple University Libraries, 2015. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/314879.

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Biomedical Sciences<br>M.S.<br>The Ras superfamily consists of over 150 members including the well-known: H-Ras, K-Ras and N-Ras. We propose to target Ras related proliferation in cancer cells by inhibiting Rab vesicle formation. H-Ras, K-Ras and N-Ras are carcinogenic; activating mutations in Ras signaling are generally associated with increased proliferation and survival in cancer cells. Ras is mutated in up to 30% of all human cancers and represents an early survival mutation in cancer cells. Vesicle-bound Ras is trafficked to the plasma membrane, which facilitates interaction between Raf,
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18

Barnett, David. "Activation antigens in the proliferation and differentiation of normal and malignant human leucocytes." Thesis, Open University, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.293382.

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19

Iqbal, Javaid. "Role of intra-cellular glucocorticoid regulation in vascular lesion development." Thesis, University of Edinburgh, 2010. http://hdl.handle.net/1842/4810.

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Atherosclerosis and post-angioplasty neointimal proliferation, which are leading causes of cardiovascular morbidity and mortality, develop as a result of chronic or acute vascular injury producing inflammatory and proliferative responses in the vessel wall. Glucocorticoids, the stress hormones produced by the adrenal cortex, have anti-inflammatory and anti-proliferative characteristics and can also influence systemic cardiovascular risk factors. The systemic levels of these hormones are controlled by the hypothalamic pituitary adrenal axis. However, there is also a tissue-specific pre-receptor
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20

Zapata, Garin Claire-Alix. "Glycogen regulates cellular proliferation in the context of aging, tumorigenesis, and hepatic regeneration." Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/667163.

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Glycogen is a branched polysaccharide that serves as an intracellular store of glucose that can be mobilized to maintain homeostasis or to fuel cellular processes. Glycogen is synthesized by glycogen synthase, which is present in two different isoforms: liver glycogen synthase (LGS) is mainly expressed in the liver, while muscle glycogen synthase (MGS) is expressed everywhere else. Recent studies are starting to uncover new roles for glycogen besides just being a glucose depot. Importantly, glycogen metabolism has been implicated in the normal aging process in species ranging from Saccharomyce
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21

Millen, Jennifer Elena. "Phosphodiesterase 4 expression and proliferation rates in a cellular model of pulmonary hypertension." Thesis, University of Glasgow, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.425277.

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22

Carpenter, Nicholas. "Design and synthesis of inhibitors of critical target proteins implicated in cellular proliferation." Thesis, Cardiff University, 2005. http://orca.cf.ac.uk/55644/.

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Cyclin-Dependent Kinases (CDKs) are a family of protein kinases that control progression through the eukaryotic cell cycle. CDKs become activated when they form a complex with the appropriate cyclin protein, and are fully activated by phosphorylation. CDK inhibitors regulate the activity of CDK/cyclin complexes through competitive inhibition with ATP for the active site. This inhibition prevents the CDK/cyclin complex phosphorylating its substrates and cell cycle progression stops. Alterations in CDK control through cyclin overexpression, CDK inhibitor underexpression or CDK mutation are respo
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23

Berlato, Davide <1973&gt. "Cellular Proliferation in the Prognosis of Intermediate Grade Mast Cell Tumour in Dogs." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2014. http://amsdottorato.unibo.it/6241/1/Davide.Berlato.Dottorato.2014.pdf.

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This was a retrospective study including ninety samples of dogs with a histological diagnosis of intermediate grade cutaneous mast cell tumour (MCT). The objectives of the study were to validate Minichromosome Maintenance Protein 7 (MCM7) as a prognostic marker in MCTs and to compare the ability of mitotic index (MI), Ki67 and MCM7 to predict outcome. The median survival for the entire population was not reached at 2099 days. The mean survival time was 1708 days. Seventy-two cases were censored after a median follow up of 1136 days and eighteen dogs died for causes related to the MCT after a m
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24

Berlato, Davide <1973&gt. "Cellular Proliferation in the Prognosis of Intermediate Grade Mast Cell Tumour in Dogs." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2014. http://amsdottorato.unibo.it/6241/.

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This was a retrospective study including ninety samples of dogs with a histological diagnosis of intermediate grade cutaneous mast cell tumour (MCT). The objectives of the study were to validate Minichromosome Maintenance Protein 7 (MCM7) as a prognostic marker in MCTs and to compare the ability of mitotic index (MI), Ki67 and MCM7 to predict outcome. The median survival for the entire population was not reached at 2099 days. The mean survival time was 1708 days. Seventy-two cases were censored after a median follow up of 1136 days and eighteen dogs died for causes related to the MCT after a m
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25

Fettig, Amy E. "Identification of cellular targets influenced by ectopic expression of TAL1 and LMO1 genes." Virtual Press, 2001. http://liblink.bsu.edu/uhtbin/catkey/1222830.

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Cancer has been a disease, which has generated intense research interest for many years. Misexpression of two oncoproteins, TAL 1 and LMO 1, has been found to help induce a particular type of leukemia, called T-cell acute lymphoblastic leukemia (T-ALL). Presently, it is not completely understood how these proteins induce leukemogenesis or what other cellular proteins they interact with to drive this progression. In this study, a series of experiments were conducted to identify downstream targets of TALI and LMO1. Using retroviral gene transfer, both genes were introduced, either singly or in c
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26

Brennan, Tracy A. "Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation." Diss., Temple University Libraries, 2011. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/107475.

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Cell Biology<br>Ph.D.<br>Cbl is a multivalent protein that interacts with a number of signaling molecules that affect cell proliferation, migration and apoptosis. Although it is a downstream effector of growth factors, cytokines and integrin signaling all of which influence bone mass, very few studies have examined the role of Cbl in osteoblast proliferation and differentiation. To examine the role(s) of Cbl in the skeletal system we have focused specifically on phosphorylation of CblY737 since it is a unique to Cbl (not present on other family members) and upon phosphorylation by Src family k
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27

Sorensin, Troels Seyffart. "Characterisation of DP-1." Thesis, University College London (University of London), 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.243913.

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28

Miettinen, Teemu P. "On connections between Metazoan cellular metabolism and cell size." Thesis, University of Dundee, 2015. https://discovery.dundee.ac.uk/en/studentTheses/3cdc8663-1167-4a8b-ad5c-698c20695664.

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All animal cells maintain cell size homeostasis, where cell growth (increase in size) is balanced with proliferation (reduction in size via cell division). Yet, different cell types have different sizes and there are physiologically relevant situations where animal cells undergo major cell size changes. So how is cell size regulated? And why is cell size regulated? Are there specific cellular processes that have different functionality in different sized cells? This thesis investigates these questions from the perspective of cellular metabolism. Using a Cyclin dependent kinase 1 knockout mouse
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29

Kwon, Jungeun Sarah, and Jungeun Sarah Kwon. "Controlling Depth of Cellular Quiescence by an Rb-E2f Network Switch." Diss., The University of Arizona, 2017. http://hdl.handle.net/10150/625623.

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Development, tissue renewal and longevity of multi-cellular organisms require the ability to switch between a proliferative state and quiescence, a reversible arrest from the cell cycle. The balance of quiescence and proliferation underlies the fundamental feature of generating and maintaining the appropriate number of cells, which is essential for tissue architecture, regeneration, and function. Disruption of quiescence and proliferation balance leads to hypo- or hyper-proliferative diseases. To date, the regulatory mechanism of proliferation has been well established, while cellular quiescen
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30

Haubst, Nicole. "Cellular and Molecular Mechanisms regulating Cell Proliferation during the Forebrain Development of the Mouse." Diss., lmu, 2005. http://nbn-resolving.de/urn:nbn:de:bvb:19-44694.

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31

Cao, Tingting, and 曹婷婷. "Oncogene EIF5A2 promotes cell growth and proliferation by reprograming cellular metabolism in hepatocellular carcinoma." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/208001.

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32

Kumar, Neil. "A computational and experimental study of HER2-signaling effects on cellular migration and proliferation." Thesis, Massachusetts Institute of Technology, 2006. http://hdl.handle.net/1721.1/39263.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Dept. of Chemical Engineering, February 2007.<br>Includes bibliographical references.<br>The fundamental question posed in this thesis is: how does a cell 'decide' to behave in a particular way? The human body is comprised of [approx.] 1014 cells that interpret extracellular information and respond with such behavior as migration, proliferation, apoptosis, or differentiation. Thirty years of research in the related fields of biochemistry, molecular biology, and genetics have demonstrated that, in most cases, the cellular decision-making p
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33

Chen, Jingbo, and 陳靜波. "Calcium signaling pathways and cell proliferation in human cardiac fibroblast." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B41290434.

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34

Chen, Jingbo. "Calcium signaling pathways and cell proliferation in human cardiac fibroblast." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B41290434.

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35

Buschmann, Mary McVey. "Laminin-332-Mediated Proliferation Control: Mechanisms Regulating Formation of the Epithelium." University of Cincinnati / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1275661166.

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36

Tea, Jonathan. "Social Stress Reduces Cellular Proliferation and Neurogenesis in the Forebrain of Male Zebrafish (Danio Rerio)." Thesis, Université d'Ottawa / University of Ottawa, 2017. http://hdl.handle.net/10393/36903.

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Many animals, including zebrafish (Danio rerio), form social hierarchies as a result of competition for limited resources. Socially subordinate fish experience chronic activation of the hypothalamic-pituitary-interrenal (HPI) axis, leading to prolonged elevation of plasma cortisol, the glucocorticoid end-product of HPI axis activation. Elevated cortisol levels can reduce cellular proliferation and neurogenesis in the brain. Thus, the present study tested the hypothesis that social stress suppresses cellular proliferation in the brain of subordinate zebrafish via a cortisol-mediated mechanism.
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37

Raibon, Audrey. "Le facteur d'initiation de la traduction eIF3f dans le muscle squelettique : étude in vitro et obtention de modèles animaux." Thesis, Montpellier 1, 2013. http://www.theses.fr/2013MON1T023/document.

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Le facteur d'initiation de la traduction eIF3f est une des sous-unités constituant le facteur d'initiation de la traduction eIF3. Au niveau musculaire la surexpression de eIF3f dans les myotubes induit une hypertrophie associée à une augmentation de la synthèse protéique. A l'inverse, l'inhibition de l'expression de eIF3f entraîne une atrophie associée à une diminution de la synthèse protéique. Ce travail de thèse a permis (i) in vitro de mettre en évidence les fonctions inhibitrices du facteur eIF3f au cours de la prolifération des myoblastes C2C12 et par une étude transcriptomique sur les fr
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38

Matthews, Benjamin Phillip. "The oncogenic protein E2a-Pbx1 alters cellular proliferation or apoptosis in haematopoietic and fibroblast tissues." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0003/MQ45287.pdf.

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39

Chen, Long-Qi. "The effects of antireflux surgery on esophageal function, cellular proliferation and apoptosis for Barrett's esophagus." [Montréal] : Université de Montréal, 2003. http://wwwlib.umi.com/cr/umontreal/fullcit?pNQ82721.

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Thèse (Ph. D.)--Université de Montréal, 2003.<br>"NQ-82721." "Thèse présentée à la faculté des études supérieures en vue de l'obtention du grade de docteur de philosophie (Ph. D.) en sciences biomédicales." Version électronique également disponible sur Internet.
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40

Wazin, Fatima. "Spred: a negative regulator of cellular processes involved in lens and eye development." Thesis, University of Sydney, 2020. https://hdl.handle.net/2123/22989.

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The transparent and refractive properties of the lens are extremely dependent on the its precise cellular structure, with constant regulation of cell behaviour throughout life. Cell signalling pathways play a crucial role in regulating cellular processes in both mammalian growth and development, and are in turn frequently regulated by inhibitory molecules, including the Spred family, to modulate and attenuate the impact of growth factor stimulation. Due to Spred’s strong expression in lens and its ability to negatively regulate growth factor-induced ERK/MAPK pathways that are essential for man
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41

Suryo, Rahmanto Yohan. "THE PHYSIOLOGICAL AND PATHOPHYSIOLOGICAL ROLES OF MELANOTRANSFERRIN." Thesis, The University of Sydney, 2007. http://hdl.handle.net/2123/2439.

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Melanotransferrin or melanoma tumour antigen p97 (MTf) is a transferrin homologue that is found predominantly bound to the cell membrane via a glycosylphosphatidylinositol anchor. The molecule is a member of the transferrin super-family that binds iron through a single high affinity iron(III)-binding site. Melanotransferrin was originally identified at high levels in melanoma cells and other tumours, but at lower levels in normal tissues. Since its discovery, the function of MTf has remained intriguing, particularly regarding its role in cancer cell iron transport. In fact, considering the cru
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42

Suryo, Rahmanto Yohan. "THE PHYSIOLOGICAL AND PATHOPHYSIOLOGICAL ROLES OF MELANOTRANSFERRIN." Faculty Medicine, Department of Pathology, 2007. http://hdl.handle.net/2123/2439.

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Doctor of Philosophy(PhD)<br>Melanotransferrin or melanoma tumour antigen p97 (MTf) is a transferrin homologue that is found predominantly bound to the cell membrane via a glycosylphosphatidylinositol anchor. The molecule is a member of the transferrin super-family that binds iron through a single high affinity iron(III)-binding site. Melanotransferrin was originally identified at high levels in melanoma cells and other tumours, but at lower levels in normal tissues. Since its discovery, the function of MTf has remained intriguing, particularly regarding its role in cancer cell iron transport.
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43

Koh, Kar Mun. "Effects of Alternating Current Electrical Stimulation on the Cellular Chemistry and Proliferation of C2C12 Muscle Cells." Thesis, North Dakota State University, 2016. https://hdl.handle.net/10365/28058.

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The objective of this study was to investigate the effects of electrical stimulation on the cellular chemistry and proliferation of C2C12 muscle cell line. The cells were cultured under the condition of AC electrical stimulation using interdigitated electrode arrays. This research was conducted by applying electrical signals for up to 24 hours to C2C12 muscle cells. After 24 hours, the electrical impedance, cell morphology, proliferation and viability were recorded. The results demonstrated that electrical stimulation have a positive impact on the cell growth of C2C12. These results obtained w
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44

Epstein, Andrew Michael. "Fragile X Protein Regulates Cellular Proliferation and Oocyte Polarity by Controlling cb1 Levels During Drosophila Oogenesis." Thesis, The University of Arizona, 2008. http://hdl.handle.net/10150/193434.

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Fragile X Protein (FMRP) is an RNA binding protein linked to the most common form of inherited mental retardation, Fragile X syndrome (FraX). Despite its ubiquitous expression and presence of non-neuronal phenotypes, FMRP function remains understudied outside of neural and synaptic development. In addition to severe cognitive deficits, FraX etiology also includes postpubescent macroorchidism, which is thought to occur due to overproliferation of the germline. Using a Drosophila model for FraX, I have shown that FMRP controls germline proliferation as well as dorso-ventral polarity during oogen
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45

Lindsey, Jenifer Ann. "The consequence of prostanoid synthesis and release by human peripheral blood monocytes on immune function and cell proliferation /." The Ohio State University, 1985. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487259580261459.

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46

Walsh, Erin. "Crossreactivity of alpha9beta1 integrin with p75NTR in modulation of proinvasive activities of glioma cells." Diss., Temple University Libraries, 2011. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/143048.

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Biology<br>Ph.D.<br>Gliomas are the most common and difficult to treat tumors of the central nervous system. Current treatments often fail to slow progression of disease due to the high invasive nature of glioma leading to a high percentage of recurrence. Our previous studies have demonstrated that the levels of alpha; 9 beta; 1 integrin found on high grade glioma were significantly increased in comparison to normal brain tissue where the levels were negligible. We also found that interaction between alpha; 9 beta; 1 integrin and nerve growth factor (NGF) plays a major role in progression of e
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47

Takayama, Sachiko. "Integrating nuclear receptor and signaling pathways involved in cell proliferation and differentiation /." view abstract or download file of text, 2006. http://proquest.umi.com/pqdweb?did=1251819331&sid=1&Fmt=2&clientId=11238&RQT=309&VName=PQD.

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Thesis (Ph. D.)--University of Oregon, 2006.<br>Typescript. Includes vita and abstract. Includes bibliographical references (leaves 88-100). Also available for download via the World Wide Web; free to University of Oregon users.
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48

Mohan, Abhinav. "ROLE OF E-CADHERIN FORCE IN THE SPATIAL REGULATION OF CELL PROLIFERATION." VCU Scholars Compass, 2016. http://scholarscompass.vcu.edu/etd/4659.

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Cell proliferation and contact inhibition play a major role in maintaining epithelial cell homeostasis. A hallmark of epithelial cells is strong cell-cell junctions. These junctions include E-Cadherin, a type of adherens junction that is critical for both barrier function and contact inhibition. Prior experiments by other groups have shown that adherens junctions are subject to mechanical tension. Externally applied forces (e.g. stretch) results in changes in E-Cadherin forces that coordinate proliferation. My current work tests the hypothesis that E-Cadherin forces mediate the spatial regulat
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49

Clubbs, Elizabeth Ann. "INFLUENCE OF SOY ISOFLAVONES ON THE PROLIFERATION AND DIFFERENTIATION OF PROSTATE EPITHELIAL CELLS." The Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=osu1208956436.

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Blessing, Sina Carola [Verfasser]. "The role of Not4 in cellular homeostasis during proliferation and differentiation in Saccharomyces cerevisiae / Sina Carola Blessing." Konstanz : Bibliothek der Universität Konstanz, 2018. http://d-nb.info/1174143207/34.

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