Bibliografías temáticas / Cervix uteri Gene expression

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Literatura académica sobre el tema "Cervix uteri Gene expression"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 7 de febrero de 2022

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Artículos de revistas sobre el tema "Cervix uteri Gene expression"

1

Kwon, Kyung Ik, Tae Sung Lee, Jiung Ho Rhee, Soon Do Cha, Sang Sook Lee y Young Wook Suh. "Expression of the mutant p53 gene in the carcinoma of the cervix uteri". Korean Journal of Gynecologic Oncology and Colposcopy 5, n.º 4 (1994): 23. http://dx.doi.org/10.3802/kjgoc.1994.5.4.23.

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Miller, C. y D. A. Sassoon. "Wnt-7a maintains appropriate uterine patterning during the development of the mouse female reproductive tract". Development 125, n.º 16 (15 de agosto de 1998): 3201–11. http://dx.doi.org/10.1242/dev.125.16.3201.

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The murine female reproductive tract differentiates along the anteroposterior axis during postnatal development. This process is marked by the emergence of distinct cell types in the oviduct, uterus, cervix and vagina and is dependent upon specific mesenchymal-epithelial interactions as demonstrated by earlier heterografting experiments. Members of the Wnt family of signaling molecules have been recently identified in this system and an early functional role in reproductive tract development has been demonstrated. Mice were generated using ES-mediated homologous recombination for the Wnt-7a gene (Parr, B. A. and McMahon, A. P. (1995) Nature 374, 350–353). Since Wnt-7a is expressed in the female reproductive tract, we examined the developmental consequences of lack of Wnt-7a in the female reproductive tract. We observe that the oviduct lacks a clear demarcation from the anterior uterus, and acquires several cellular and molecular characteristics of the uterine horn. The uterus acquires cellular and molecular characteristics that represent an intermediate state between normal uterus and vagina. Normal vaginas have stratified epithelium and normal uteri have simple columnar epithelium, however, mutant uteri have stratified epithelium. Additionally, Wnt-7a mutant uteri do not form glands. The changes observed in the oviduct and uterus are accompanied by a postnatal loss of hoxa-10 and hoxa-11 expression, revealing that Wnt-7a is not required for early hoxa gene expression, but is required for maintenance of expression. These clustered hox genes have been shown to play a role in anteroposterior patterning in the female reproductive tract. In addition to this global posterior shift in the female reproductive tract, we note that the uterine smooth muscle is disorganized, indicating development along the radial axis is affected. Changes in the boundaries and levels of other Wnt genes are detectable at birth, prior to changes in morphologies. These results suggest that a mechanism whereby Wnt-7a signaling from the epithelium maintains the molecular and morphological boundaries of distinct cellular populations along the anteroposterior and radial axes of the female reproductive tract.
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Riethdorf, Sabine, Lutz Riethdorf, Nicole Richter y Thomas Löning. "Expression of the MCP-1 Gene and the HPV 16 E6/E7 Oncogenes in Squamous Cell Carcinomas of the Cervix uteri and Metastases". Pathobiology 66, n.º 6 (1998): 260–67. http://dx.doi.org/10.1159/000028032.

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Alvarez-Rodriguez, Manuel, Mohammad Atikuzzaman, Heli Venhoranta, Dominic Wright y Heriberto Rodriguez-Martinez. "Expression of Immune Regulatory Genes in the Porcine Internal Genital Tract Is Differentially Triggered by Spermatozoa and Seminal Plasma". International Journal of Molecular Sciences 20, n.º 3 (25 de enero de 2019): 513. http://dx.doi.org/10.3390/ijms20030513.

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Mating or cervical deposition of spermatozoa or seminal plasma (SP) modifies the expression of genes affecting local immune defense processes at the oviductal sperm reservoir in animals with internal fertilization, frequently by down-regulation. Such responses may occur alongside sperm transport to or even beyond the reservoir. Here, immune-related gene expression was explored with cDNA microarrays on porcine cervix-to-infundibulum tissues, pre-/peri-ovulation. Samples were collected 24 h post-mating or cervical deposition of sperm-peak spermatozoa or SP (from the sperm-peak fraction or the whole ejaculate). All treatments of this interventional study affected gene expression. The concerted action of spermatozoa and SP down-regulated chemokine and cytokine (P00031), interferon-gamma signaling (P00035), and JAK/STAT (P00038) pathways in segments up to the sperm reservoir (utero-tubal junction (UTJ)/isthmus). Spermatozoa in the vanguard sperm-peak fraction (P1-AI), uniquely displayed an up-regulatory effect on these pathways in the ampulla and infundibulum. Sperm-free SP, on the other hand, did not lead to major effects on gene expression, despite the clinical notion that SP mitigates reactivity by the female immune system after mating or artificial insemination.
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Goldberg, D. M. y E. P. Diamandis. "Models of neoplasia and their diagnostic implications: a historical perspective". Clinical Chemistry 39, n.º 11 (1 de noviembre de 1993): 2360–74. http://dx.doi.org/10.1093/clinchem/39.11.2360.

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Abstract In comparison with normal cells, cancer cells have an enhanced ability to trap both nitrogen and energy; an enhanced operation of the glycolytic and direct oxidative pathways, leading to accumulation of lactate and increased production of NADPH; and a greater content of lysosomal hydrolases. These changes represent a reprogramming of gene expression, which, at its most specific, is accompanied by the reappearance in the cell and ultimately in the body fluids of oncodevelopmental proteins not normally found in mature adult tissues. The most florid stage of this reprogramming leads to the metastatic phenotype, which confers upon the cancer cell the ability to stimulate angiogenesis, invade the bloodstream and lymphatic vessel, and arrest and proliferate in distant tissues. The diagnostic implications of these phenotypic changes are illustrated for cancer of the cervix uteri and cancer of the colon. We also review the classical theories of neoplasia, including the cellular anoxia concept of Warburg, the deletion hypothesis of Potter, and various other mechanisms emphasizing genomic derepression and impaired immunity. The critical steps in chemical carcinogenesis are described, and the Vogelstein-Lane model is presented, emphasizing the stepwise and cumulative genomic changes affecting chromosomes 5q, 17p, 18q, and gene amplification of chromosome 12 as well as genomic instability resulting from reduced DNA methylation. The main consequences of these genomic alterations include overexpression or activation of oncogenes such as c-myc and k-ras, together with mutation or functional inactivation of suppressor genes such as p53. Finally, the implications of these findings for diagnosis and management are illustrated by reference to recent investigations in cancers of the breast, colon, and bladder, in which these genomic alterations can be detected by examination of appropriate cellular material and by detection in serum of antibodies to the p53 gene product.
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Alvarez-Rodriguez, Manuel, Cristina A. Martinez, Dominic Wright y Heriberto Rodriguez-Martinez. "Does the Act of Copulation per se, without Considering Seminal Deposition, Change the Expression of Genes in the Porcine Female Genital Tract?" International Journal of Molecular Sciences 21, n.º 15 (31 de julio de 2020): 5477. http://dx.doi.org/10.3390/ijms21155477.

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Semen—through its specific sperm and seminal plasma (SP) constituents—induces changes of gene expression in the internal genital tract of pigs, particularly in the functional sperm reservoir at the utero-tubal junction (UTJ). Although seminal effects are similarly elicited by artificial insemination (AI), major changes in gene expression are registered after natural mating, a fact suggesting the act of copulation induces per se changes in genes that AI does not affect. The present study explored which pathways were solely influenced by copulation, affecting the differential expression of genes (DEGs) of the pre/peri-ovulatory genital tract (cervix, distal uterus, proximal uterus and UTJ) of estrus sows, 24 h after various procedures were performed to compare natural mating with AI of semen (control 1), sperm-free SP harvested from the sperm-peak fraction (control 2), sperm-free SP harvested from the whole ejaculate (control 3) or saline-extender BTS (control 4), using a microarray chip (GeneChip® porcine gene 1.0 st array). Genes related to neuroendocrine responses (ADRA1, ADRA2, GABRB2, CACNB2), smooth muscle contractility (WNT7A), angiogenesis and vascular remodeling (poFUT1, NTN4) were, among others, overrepresented with distal and proximal uterine segments exhibiting the highest number of DEGs. The findings provide novel evidence that relevant transcriptomic changes in the porcine female reproductive tract occur in direct response to the specific act of copulation, being semen-independent.
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7

Yi, B. R., S. U. Kim y K. C. Choi. "126 POTENTIAL EFFECTS OF ENGINEERED STEM CELLS TRANSDUCED WITH THERAPEUTIC GENES AGAINST HeLa HUMAN CERVICAL CANCER CELLS VIA A SELECTIVE TUMOR TROPISM CAUSED BY VASCULAR ENDOTHELIAL GROWTH FACTOR". Reproduction, Fertility and Development 26, n.º 1 (2014): 177. http://dx.doi.org/10.1071/rdv26n1ab126.

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According to the World Health Organization, cancer of cervix uteri is the second most common cancer among women worldwide. Recently, cervical cancer still remains a significant public health problem for women despite the development of the human papilloma virus vaccine. The aim of the present study was to investigate the therapeutic efficacy of genetically engineered stem cells (GESTEC) expressing bacterial cytosine deaminase (CD), human interferon-β (IFN-b) gene, or both against HeLa human cervical cancer and the migration factors of the GESTEC toward the cancer cells. A continuously dividing immortalized cell line of neural stem cells (NSC) from a single clone of human fetal brain, HB1.F3, was developed by introducing v-myc. The further introduction of these NSC with bacterial CD and human IFN-b resulted in the generation of HB1.F3.CD and HB1.F3.CD.IFN-b cells. The anticancer effect of these GESTEC was examined in a co-culture with HeLa cells using MTT assay to measure cell viability. A transwell migration assay was performed to assess the migration capability of the stem cells to cervical cancer cells. Next, several chemoattractant ligands and their receptors related to a selective migration of the stem cells towards HeLa cells were determined by real-time PCR. The cell viability of HeLa cells was decreased in response to 5-fluorocytosine (5-FC), a prodrug, indicating that 5-fluorouracil (5-FU), a toxic metabolite, was converted from 5-FC by the CD gene and it caused the cell death in a co-culture system. When IFN-b was additionally expressed with the CD gene by these GESTEC, the anticancer activity was significantly increased. In the migration assay, the GESTEC selectively migrated to HeLa cells. As results of real-time PCR, chemoattractant ligands such as MCP-1, SCF, and VEGF were expressed in HeLa cells, and several receptors such as uPAR, VEGFR2, and c-kit were produced by the GESTEC. These GESTEC transduced with the CD gene and IFN-b may provide the potential of a novel gene therapy for anti-cervical cancer treatments via their selective tumour tropism derived from VEGF and VEGFR2 expressions between HeLa cells and the GESTEC. This work was supported by a grant from the Next-Generation BioGreen 21 Program (No. PJ009599), Rural Development Administration, Republic of Korea.
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IWAHASHI, MASAAKI y YASUTERU MURAGAKI. "Decreased type III collagen expression in human uterine cervix of prolapse uteri". Experimental and Therapeutic Medicine 2, n.º 2 (2011): 271–74. http://dx.doi.org/10.3892/etm.2011.204.

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Negri, Giovanni, Fabio Vittadello, Fabio Romano, Armin Kasal, Francesco Rivasi, Salvatore Girlando, Christine Mian y Eduard Egarter-Vigl. "P16INK4a expression and progression risk of low-grade intraepithelial neoplasia of the cervix uteri". Virchows Archiv 445, n.º 6 (9 de octubre de 2004): 616–20. http://dx.doi.org/10.1007/s00428-004-1127-9.

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Randall, G. W., J. C. Daniel y B. S. Chilton. "Prolactin enhances uteroglobin gene expression by uteri of immature rabbits". Reproduction 91, n.º 1 (1 de enero de 1991): 249–57. http://dx.doi.org/10.1530/jrf.0.0910249.

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Tesis sobre el tema "Cervix uteri Gene expression"

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Chiu, Pui-man. "Molecular genetics of cervical cancer from chromosome number alterations to aberrant gene expressions /". Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B43085544.

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Liu, Si Stephanie. "Cervical cancer and radiotherapy: study on apoptosis and its related genes, with special interest on p73". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B31245766.

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Chiu, Pui-man y 趙佩文. "Molecular genetics of cervical cancer: from chromosome number alterations to aberrant gene expressions". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43085544.

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Lundberg, Mathias. "Cloning and characterization of human glutaredoxins /". Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-261-2.

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Abelin, Törnblom Susanne. "Mediators of cervical ripening in preterm birth : experimental and clinical investigations /". Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-305-1/.

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Lai, Tung-on Anthony y 黎東安. "PRKAA1 gene amplification in cervical cancer and precursors: a study in cytology samples". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45153048.

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Ho, Kam-tai y 何金娣. "To reveal the gene copy status of MUC1 in cervical neoplasia and precursor lesions by real-time PCR". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B44659799.

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Vadlamuri, Satya Vijayanand. "Genetic evaluation of cytochrome-P450 expression in smoking and nonsmoking women". Morgantown, W. Va. : [West Virginia University Libraries], 2001. http://etd.wvu.edu/templates/showETD.cfm?recnum=1806.

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Thesis (Ph. D.)--West Virginia University, 2001.
Title from document title page. Document formatted into pages; contains xiv, 150 p. : ill. (some col.). Vita. Includes abstract. Includes bibliographical references (p. 104-120).
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Schöffel, Marion. "Die Expression von PAX9 im gesunden und dysplastischen Epithel und in invasiven Karzinomen des Ösophagus und der Cervix uteri eine immunhistochemische Studie /". [S.l.] : [s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=973910062.

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Mo, Bilan. "5-fluorouracil-induced apoptosis and altered expression of apoptosis-regulating proteins in a model system for multistage cervical carcinogenesis". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0031/MQ62401.pdf.

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