Libros: "Chronic pain Chronic pain Quality of life"

7

Denise, Webster, ed. Recrafting a life: Solutions for chronic pain and illness. Brunner-Routledge, 2002.

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8

Roberto, Karen A. Chronic pain in later life: A selectively annotated bibliography. Greenwood Press, 2001.

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9

Rothbart, Brian A., and Linda F. Penzabene. Forever Free From Chronic Pain: The Pain Sufferers Guide to Getting Your Life Back. 2nd ed. Happy About Publishers, 2009.

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10

Linchitz, Richard M. Life without pain: Free yourself from chronic back pain, headache, arthritis pain, and more, without surgery or narcotic drugs. Addison-Wesley Pub. Co., 1987.

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11

Chronic pain: Finding hope in the midst of suffering. Beacon Hill Press of Kansas City, 2014.

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12

Arat, Arsavir. Chronic emotional stress of daily life physically mimics chronic injury aches and pains: How this relates to work and liability injuries. CESDL Press, 2006.

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13

Chronic pain: Living by faith when your body hurts. New Growth Press, 2010.

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14

Living well with chronic pain: Because life won't wait-- the latest news you can use, because life is for living on your own terms! Thornton Pub., 2004.

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15

William, Proctor, ed. Pain-free for life: The 6-week cure for chronic pain-without surgery or drugs. Center Street, 2006.

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16

Moore, Rhonda J. Handbook of pain and palliative care: Biobehavioral approaches for the life course. Springer, 2012.

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17

Pratt, Maureen. Beyond pain: Job, Jesus, and joy. Twenty-Third Publications, 2010.

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18

Vad, Vijay. Stop pain: Inflammation relief for an active life. Hay House, 2010.

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19

Peter, Occhiogrosso, ed. Stop pain: Inflammation relief for an active life. 2nd ed. Hay House, 2011.

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20

Struck down but not destroyed!: A Christian response to chronic illness and pain. Rainbow's End Co., 1996.

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21

Condon, Loraine. A new way of life: A practical guide to managing arthritis and chronic pain. HarperHealth, 1995.

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22

Diet for a pain-free life: A revolutionary plan to lose weight, end inflammation, stop pain, sleep better, and feel great in 21 days. Marlowe & Co., 2007.

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23

White, Catherine. Chronic pain and quality of life. 2002.

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24

Iqbal, Afzaal, and Joel Kent. Chronic Sternal Pain after Cardiac Surgery. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190271787.003.0017.

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Chronic post-sternotomy pain (PSP) is a well-known complication of cardiac surgery that is associated with increased morbidity. The lack of adequate pain relief can compromise the patients’ rehabilitation trajectory and diminish their quality of life. Various factors have been identified that contribute to the development and maintenance of PSP. Local and systemic pain management modalities have been identified to mitigate this condition. It is important to conduct a careful history and exam in these patients in order to rule out other causes of pain and properly guide the patient’s management. Individualized treatment will ultimately result in the most effective relief.

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25

Claes, Nathalie, and Winifred Gebhardt. Chronic Pain, Goal Conflict and Goal Frustration. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190627898.003.0009.

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This chapter argues for extending models of chronic pain within an explicit goal and self-regulatory perspective. A self-regulatory perspective allows one to conceptualize pain as an experience that occurs within the real-life context comprising multiple goals. The chapter presents two fictitious cases, which will be used throughout the chapter to clarify goal concepts. Next, it outlines the possible interrelations between goals, after which it specifically focuses on goal conflict and its role in pain. The chapter then provides a definition and overview of the literature on goal frustration and offers insights into the link between goal conflict and goal frustration. It also presents an overview of interventions that focus on tackling goal conflict and goal frustration to improve quality of life. The chapter then discusses potential implications of the theoretical stance and the empirical findings for existing theories of chronic pain problems. Finally, it formulates suggestions for future research.

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26

Chasen, Martin, and Gordon Giddings. Management issues in chronic pain following cancer therapy. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0135.

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With improved surveillance, diagnoses, and treatment of patients with cancer, an increased life expectancy, and specifically an increased number of ‘cancer cured’ patients, is noted. However, the long-term effects of the disease and treatment have a bearing on obtaining optimal physical, psychological, and cognitive functioning for cancer survivors. Pain impacts on all dimensions of quality of life and is one of the most distressing symptoms for patients. Patients often under-recognize pain and are unsure if optimum pain control is achievable. In addition, members of the interdisciplinary team often fail to assess the patient’s pain adequately, due to a lack of knowledge of the principles of pain relief and side effect management. Treatment requires an interprofessional approach that details a comprehensive assessment, with ongoing reassessment, utilizing both pharmacological and non-pharmacological measures. Empowerment of the cancer survivor, respect for survivors’ individuality and collaboration among team members are key elements of any successful strategy to optimize a patient’s quality of life.

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27

Brown, Matthew. The chronic constriction injury model of neuropathic pain. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0067.

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The landmark paper discussed in this chapter is ‘A peripheral mononeuropathy in rat that produces disorders of pain sensation like those seen in man’, published by Bennett and Xie in 1988. This paper, in which the unilateral sciatic nerve chronic constriction injury (CCI) model was first presented, is one of the earliest and most comprehensive descriptions of a specific animal paradigm that was designed to model human neuropathic pain. The authors realized that human neuropathic pain rarely involves nerve transection but instead involves evoked changes in damaged and preserved nerve fibres. Furthermore, they systematically applied a barrage of sensory testing that demonstrated quantifiable hyperalgesia and cold allodynia reflecting some of the clinical observations of human neuropathic pain phenotype. CCI provided a high-quality template for the development of neuropathic pain models that impelled the subsequent development of other animal models striving to replicate the human condition faithfully and accurately.

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28

Cheatle, Martin D., and Lara Dhingra. Biopsychosocial Approach to Improving Treatment Adherence in Chronic Pain. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190600075.003.0006.

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Up to 53% of patients with chronic nonmalignant pain demonstrate medication nonadherence, and many are nonadherent with behavior-change interventions for pain, presenting a significant challenge to providers managing this population and compromising patient-reported outcomes related to treatment efficacy, symptom control, and quality of life. Patients with chronic pain are often highly complex and present with numerous medical and psychological comorbidities. Many of these comorbidities, including mood, sleep, and substance use disorders, in addition to maladaptive coping with pain and varied clinician, health system, and family-related factors, can influence adherence to pain interventions. This chapter applies a biopsychosocial framework to guide the clinical assessment of nonadherence behaviors in chronic pain, including the identification of risk factors, mechanisms, and underlying processes of nonadherence, and presents strategies providers can potentially implement to enhance patient adherence to pharmacologic and behavioral therapies for pain management.

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29

Weil, Arnold J. Assessing Quality of Life: Focus on Sustained-release Opioids (New Directions in Chronic Pain). Royal Society of Medicine Press, 2004.

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30

Mofeez, Ali, and Upal Hossain. Acute pain in haematological disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199234721.003.0014.

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The use of painkillers ranging from simple analgesics to strong opioids is a common feature in the acute pain management of haematological conditions. However, each disease also has its own specific aetiological factors for pain, requiring specific treatment. Haematological patients with chronic pain on long-term opioid therapy may require multidisciplinary pain management to improve quality of life and prevent chronic escalation of opioid doses. Intramuscular injections should be avoided in all patients. The use of pethidine (meperidine) is not recommended.

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31

Ruskin, David N. Metabolic Therapy and Pain. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0022.

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Chronic pain is associated strongly with poor quality of life. Drug treatments for pain can be problematic; with the understanding that chronic pain syndromes often involve derangement of homeostasis, there is an increased interest in applying nonpharmacological metabolic therapies. This chapter surveys clinical and animal research into the effects of fasting, calorie restriction, ketogenic diet, and polyunsaturated fatty acid supplementation on pain. These dietary treatments can significantly ameliorate pain in inflammatory and neuropathic disorders. The choice among these treatments might depend on the specific pain syndrome and the tolerance of the patient for particular dietary modifications. Several possible mechanisms are discussed, some of which might be in common among these treatments, and some treatments might engage multiple mechanisms. Multiple mechanisms acting together could be ideal for restoring the disordered metabolism underlying some pain syndromes.

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32

McCabe, Candy, Richard Haigh, Helen Cohen, and Sarah Hewlett. Pain and fatigue. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0012.

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Pain and fatigue are the prominent problems for those with a rheumatic disease, and are often underestimated by clinicians. Symptoms may fluctuate in quality and intensity over time and commonly will vary over the course of a day. For pain, clinical signs and symptoms will be dependent on the source of the pain and whether causative underlying pathology is identifiable or not. Fatigue may range from mild effects to total exhaustion and may include cognitive and emotional elements, with a complex, probably multicausal, pathway. Theoretical knowledge of potential mechanistic pathways for pain and fatigue should be used to inform assessment and treatment approaches. Best practice recommends a multidisciplinary and holistic treatment approach with the patient an active participant in the planning of their care, and self-management. Many patients with chronic musculoskeletal conditions will not achieve a pain-free or fatigue-free status. Medication use must therefore balance potential benefit against short- and long-term side effects. Rheumatology centres should offer specific fatigue and pain self-management support as part of routine care. Emphasis should be given to facilitating self-management strategies for both pain and fatigue to help the patient optimize their quality of life over years or a lifetime of symptoms. Interventions should include behaviour change and cognitive restructuring of pain/fatigue beliefs, as well as access to relevant self-help groups and charitable organizations. Referral for specialist advice from regional or national clinics on pain relief and management should be considered if pain interferes significantly with function or quality of life despite local interventions.

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33

McCabe, Candy, Richard Haigh, Helen Cohen, and Sarah Hewlett. Pain and fatigue. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199642489.003.0012_update_001.

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Pain and fatigue are the prominent problems for those with a rheumatic disease, and are often underestimated by clinicians. Symptoms may fluctuate in quality and intensity over time and commonly will vary over the course of a day. For pain, clinical signs and symptoms will be dependent on the source of the pain and whether causative underlying pathology is identifiable or not. Fatigue may range from mild effects to total exhaustion and may include cognitive and emotional elements, with a complex, probably multicausal, pathway. Theoretical knowledge of potential mechanistic pathways for pain and fatigue should be used to inform assessment and treatment approaches. Best practice recommends a multidisciplinary and holistic treatment approach with the patient an active participant in the planning of their care, and self-management. Many patients with chronic musculoskeletal conditions will not achieve a pain-free or fatigue-free status. Medication use must therefore balance potential benefit against short- and long-term side effects. Rheumatology centres should offer specific fatigue and pain self-management support as part of routine care. Emphasis should be given to facilitating self-management strategies for both pain and fatigue to help the patient optimize their quality of life over years or a lifetime of symptoms. Interventions should include behaviour change and cognitive restructuring of pain/fatigue beliefs, as well as access to relevant self-help groups and charitable organizations. Referral for specialist advice from regional or national clinics on pain relief and management should be considered if pain interferes significantly with function or quality of life despite local interventions.

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34

Kucyi, Aaron. Pain and Spontaneous Thought. Edited by Kalina Christoff and Kieran C. R. Fox. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780190464745.013.40.

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Pain is among the most salient of experiences, while also, curiously, being among the most malleable. A large body of research has revealed that a multitude of explicit strategies can be used to effectively alter the attention-demanding quality of acute and chronic pains and their associated neural correlates. However, thoughts that are spontaneous, rather than actively generated, are common in daily life, and so attention to pain can often temporally fluctuate because of ongoing self-generated experiences. Classic pain theories have largely neglected to account for unconstrained fluctuations in cognition, but new studies have demonstrated the behavioral relevance, putative neural basis, and individual variability of interactions between pain and spontaneous thoughts. This chapter reviews behavioral studies of ongoing fluctuations in attention to pain, studies of the neural basis of spontaneous mind-wandering away from pain, and the clinical implications of this research.

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35

Finnerup, Nanna Brix, and Troels Staehelin Jensen. Management issues in neuropathic pain. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0133.

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Neuropathic pain is a common complication to cancer, cancer treatment, HIV, and other conditions that may affect the somatosensory nervous system. Neuropathic pain may be present in up to 40% of cancer patients and may persist independently of the cancer and affect the quality of life in disease-free cancer survivors. Particular surgical treatment and chemotherapy may cause chronic persistent neuropathic pain in cancer survivors. The diagnosis of neuropathic pain can be challenging and requires documentation of a nervous system lesion and pain in areas of sensory changes. The pharmacological treatment may include tricyclic antidepressants, selective serotonin noradrenaline reuptake inhibitors (duloxetine or venlafaxine), calcium channel α2↓ agonists (gabapentin or pregabalin), and opioids. Topical lidocaine and capsaicin, NMDA antagonists, carbamazepine, oxcarbazepine, and cannabinoids may be indicated. Due to limited efficacy or intolerable side effects at maximal doses, combination therapy is often required and careful monitoring of effect and adverse reactions is important.

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36

Qureshi, M. A., J. H. Gan, S. Kunnumpurath, et al. Preventive Analgesia for the Management of General Surgical Pain. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190626761.003.0002.

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Pain created by surgery has the ability to produce both structural and functional changes in pain pathways. These changes may be reduced if timely and adequate pain relief is delivered to the patient. Poor control of pain can result in remodeling of the “hardwired” pathways involved in pain transmission, which can result in central sensitization and hyperalgesia. Furthermore, poorly controlled pain and delay in its recognition may lead to a chronic pain state, further complicating the patient’s recovery and quality of life. A multimodal approach taking into account psychosocial aspects of the patient is more likely to mitigate the development of chronic postsurgical pain (CPSP).

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37

Davis, Mary C., Chung Jung Mun, Dhwani Kothari, et al. The Nature and Adaptive Implications of Pain-Affect Dynamics. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190627898.003.0013.

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Because pain is in part an affective experience, investigators over the past several decades have sought to elaborate the nature of pain-affect connections. Our evolving understanding of the intersection of pain and affect is especially relevant to intervention efforts designed to enhance the quality of life and functional health of individuals managing chronic pain. This chapter describes how pain influences arousal of the vigilance/defensive and appetitive/approach motivational systems and thus the affective health of chronic pain patients. The focus then moves to the dynamic relations between changes in pain and other stressors and changes in positive and negative affect as observed in daily life and laboratory-based experiments. A consensus emerges that sustaining positive affect during pain and stress flares may limit their detrimental effects and promote better functional health. The authors consider the implications of increased understanding of the dynamic interplay between pain and affective experience for enhancing existing interventions.

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38

Sullivan, Mark D. Health-Related Quality of Life as a Goal for Clinical Care. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780195386585.003.0005.

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The importance of chronic illness has brought a new focus on patient-reported outcomes of clinical care. Health-Related Qualify of Life (HRQL) is a new goal for clinical care that combines a physician’s view of health as an objective biological fact and the patient’s view of health as a subjective experiential state. The diagnosis of an impersonal and objective disease separable from the patient arose after the French Revolution and helped to delimit the new right to health care. But objective mortality and morbidity metrics are not adequate for capturing the burden of chronic illness. HRQL was invented to capture the burden of chronic illness, but has not been successfully incorporated into clinical trials or clinical care. Chronic low back pain is presented as an example where both objective and subjective metrics of treatment success have failed. We need an openly patient-centered definition of health that is not just a supplement to objective disease diagnosis.

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39

Wickersham, Dr Pendleton B. Managing Life With Chronic Pain. CreateSpace Independent Publishing Platform, 2011.

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40

Navigating Life with Chronic Pain. Oxford University Press, Incorporated, 2020.

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41

Headley, Barbara J. Chronic Pain: Life Out of Balance. 2nd ed. Pain Resources, 1988.

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42

Embracing Life: Living With Chronic Pain. iUniverse, Inc., 2005.

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43

Von Korff, Michael. Fear and depression as remediable causes of disability in common medical conditions in primary care. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780198530343.003.0007.

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This chapter argues that psychological states, in particular fear and depression, are potentially remediable causes of social role disability among primary care patients. Using chronic low back pain as an example, it considers how recognising and treating depression can improve disability and quality of life for primary care patients with this and many other chronic conditions.

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44

(Editor), Dana S. Deboskey, Joyce M. Engel (Editor), and Thomas W. Oleson (Editor), eds. Pain: Making Life Liveable. Hdi Pub, 1996.

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45

Headley, B. Chronic Pain: Life Out of Balance (#6900). hazelden, 1987.

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46

Living a Healthy Life with Chronic Pain. Bull Publishing Company, 2015.

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47

Chronic Pain: Finding a Life Worth Living. Vantage Press, 1994.

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48

Swarm, Robert A., Menelaos Karanikolas, Lesley K. Rao, and Michael J. Cousins. Interventional approaches for chronic pain. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0098.

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Severe, uncontrolled pain remains common in populations with serious or life-threatening illness. Despite the availability of oral opioid therapy in most developed countries, an estimated 10-30% of people with advanced cancer have inadequate pain control. Published guidelines endorse the view that these patients should be considered for procedural, or so-called interventional, pain therapies. Generally accepted indications for interventional pain therapies include (a) uncontrolled pain despite systemic analgesics and (b) unacceptable systemic analgesic adverse effects. This chapter describes these therapies and discusses how they are best used within a multimodal strategy for symptom management. Interventional pain therapies are now incorporated into best practices for cancer pain management. These therapies, especially spinal analgesics, neurolytic coeliac plexus block, and vertebroplasty, have become essential components of palliative care, to control pain that cannot be safely and effectively managed with systemic analgesics.

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49

John M., M.D. Stamatos. Live Your Life Pain Free: Medical Discoveries That Stop Chronic Pain. Magni Company, 2005.

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50

Parker, Samantha. Yoga for Chronic Pain ... Wtf?: Take Control, Combat Pain & Rock Your Life. Neoteric Movement Sytems, 2018.

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Libros sobre el tema "Chronic pain Chronic pain Quality of life"

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