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Artículos de revistas sobre el tema "Coronary heart disease"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 9 de junio de 2025

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1

Nurmamadovna, Ishankulova Nasiba. "Coronary Heart Disease." American Journal of Medical Sciences and Pharmaceutical Research 03, no. 02 (February 28, 2021): 31–36. http://dx.doi.org/10.37547/tajmspr/volume03issue02-04.

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The article covers the etiology, pathogenesis, classification, diagnosis, clinical picture and treatment of coronary heart disease, provides a literature review. Cardiovascular disease (CVD) represents the leading cause of death among women as well as men. The number of deaths due to CVD in women are greater than in men. There are significant gender-related differences concerning CVD.

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2

Shakhnoza, Iskandarova, and Amilova Asalya. "PREVENTION OF CORONARY HEART DISEASE." American Journal of Medical Sciences and Pharmaceutical Research 04, no. 04 (April 1, 2022): 19–21. http://dx.doi.org/10.37547/tajmspr/volume04issue04-05.

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Primary prevention, based on healthy lifestyle habits that prevent the emergence of risk factors, is the preferred method of reducing cardiovascular risk. Reducing the prevalence of obesity is the most urgent task, and it is pleiotropic in that it affects blood pressure, lipid profile, glucose metabolism, inflammation, progression of atherothrombotic disease. Physical activity also improves several risk factors, with the added potential to lower heart rate.

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3

Sudhakar, B. G. K. "Structural heart disease interventions." Clinical Research and Clinical Trials 3, no. 5 (June 25, 2021): 01–05. http://dx.doi.org/10.31579/2693-4779/042.

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Seed for invasive cardiology was sown in early part of nineties. Cardiac catheterization was actually pioneered by Werner Forssmann in 1929. However, credit for therapeutic interventional cardiology should go to US vascular radiologist, Charles Theodore Dotter for performing first peripheral arterial angioplasty [PTA] in 1964. Subsequently, a German cardiologist by name Andreas Gruentzig adapted the technique in 1974 to suit coronary artery disease and performed the first human coronary balloon angioplasty to treat blockage in coronary artery in 1977.

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4

Windecker, S. "CORONARY DISEASE: Intervention in coronary artery disease." Heart 83, no. 4 (April 1, 2000): 481–90. http://dx.doi.org/10.1136/heart.83.4.481.

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5

Gulnara, Asadova, and Djamalov Abdurakhman. "REMEDIES AND RECOMMENDATIONS CORONARY HEART DISEASES." Eurasian Journal of Medical and Natural Sciences 03, no. 02 (February 1, 2023): 224–28. http://dx.doi.org/10.37547/ejmns-v03-i02-p1-37.

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Prevention of coronary heart disease is a series of complex measures aimed at preventing the onset of the disease, the development and occurrence of possible (predictable) complications, which can be even fatal. Prevention of coronary heart disease is indicated for both sick people and healthy people who are at risk of developing the disease. Patients diagnosed with coronary heart disease are shown non-drug treatment, which is a complete or partial elimination of risk factors. Risk factors are any predisposing factors that increase the likelihood of developing or worsening a disease. Risk factors for coronary heart disease are fundamentally divided into removable and non-removable.

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6

Jha, Dr Sudha, Dr Naved Ahmad, Dr Surya Kant Nagtilak, and Dr Maheshwar Chawla. "Coronary Heart Disease, A Gift of Modern Civilization." International Journal of Scientific Research 2, no. 11 (June 1, 2012): 378–80. http://dx.doi.org/10.15373/22778179/nov2013/122.

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7

Gulyabin, Konstantin Robertovich. "Coronary heart disease." Spravočnik vrača obŝej praktiki (Journal of Family Medicine), no. 1 (2022): 63–66. http://dx.doi.org/10.33920/med-10-2201-09.

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Coronary heart disease is of great clinical importance in medical practice. The development of medical science makes it possible to introduce new modern methods of treating this disease. One of these is coronary artery bypass grafting, a modern method of treating coronary heart disease, which allows to reliably increase myocardial perfusion and prolong the life expectancy of patients with coronary heart disease.

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8

Timmis, A. "Coronary disease: Acute coronary syndromes: risk stratification." Heart 83, no. 2 (February 1, 2000): 241–46. http://dx.doi.org/10.1136/heart.83.2.241.

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9

Lavie, Carl J., Gary Legwold, and Michael E. DeBakey. "Coronary heart disease." Postgraduate Medicine 102, no. 2 (August 1997): 210–15. http://dx.doi.org/10.3810/pgm.1997.08.285.

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10

Williams, Samantha. "Coronary heart disease." Nursing Standard 19, no. 51 (August 31, 2005): 67–68. http://dx.doi.org/10.7748/ns.19.51.67.s58.

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11

Ahmed, Munir. "CORONARY HEART DISEASE;." Professional Medical Journal 21, no. 06 (December 10, 2014): 1171–73. http://dx.doi.org/10.29309/tpmj/2014.21.06.2250.

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Objective: This study was done to find any correlation among total cholesterol, triglycerides, low density lipoprotein cholesterol and high density lipoprotein cholesterol estimated in serum of offsprings of coronary heart disease patients. Study Design: A cross sectional comparative study. Patients and Methods: Two hundred and fifty (250) subjects having parents with coronary heart disease were selected from Punjab Institute of Cardiology Lahore. The serum total cholesterol, triglycerides, low density lipoprotein cholesterol, and high density lipoprotein cholesterol were estimated. Coefficient of variation was calculated to find whether observations in one series vary correspondingly with observations in another series. Results: Highly significant positive correlation was found between total cholesterol and triglycerides, and, total cholesterol and low density lipoprotein cholesterol. Highly significant negative correlation was found between low density lipoprotein cholesterol and high density lipoprotein cholesterol. Correlation between TG and LDL-c was also significant. Conclusions: Serum total cholesterol, triglycerides, low density lipoprotein cholesterol and high density lipoprotein cholesterol have strong association with one and other. For prediction, prevention and management of coronary heart disease it is important to estimate and observe the correlation among these parameters.

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12

AHMED, MUNIR, and MOHAMMAD TAYYIB. "CORONARY HEART DISEASE." Professional Medical Journal 16, no. 01 (March 10, 2009): 87–93. http://dx.doi.org/10.29309/tpmj/2009.16.01.2988.

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O b j e c t i v e s : This study was conducted to perform serum lipid profile in off springs of premature coronary heart disease(CHD) patients and age and sex matched normal controls and compare the results of two groups. S t u d y d e s i g n : A cross sectional study.Patients a n d m e t h o d s : 250 off springs of diagnosed premature CHD patients were selected from Punjab institute of cardiology, Lahoreand Services hospital, Lahore. 50 age and sex matched normal controls were selected from different areas of Lahore. Serum totalcholesterol (TC), serum triglycerides (TG) serum low density lipoprotein-cholesterol (LDC-c) and serum high density lipoprotein cholesterol(HDL-c) was performed. Results: Serum TC, TG, LDL-c of off springs of premature CHD patients was increased as compared with normalcontrols. Serum HDL-c of all the subjects of off springs of premature CHD patients was decreased as compared with normal controls.C o n c l u s i o n : Off springs of premature CHD patients are more prone to develop lipid abnormalities as compared with normal controls.

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13

GILANi, MUHAMMAD SHAH. "CORONARY HEART DISEASE." Professional Medical Journal 15, no. 02 (March 10, 2008): 255–59. http://dx.doi.org/10.29309/tpmj/2008.15.02.2759.

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Objective: To assess the value of coronary events reflected by changesin R-wave amplitude after exercise.Setting: Nishtar Hospital, Multan. Duration. One year. Study Design: comparativestudy.Material and Methods: Sample size 146 patients.Sampling Technique: Convenient probability sampling done.Results: All patients were followed up for 6 months, during which time myocardial infarction and death of cardiovascularorigin were considered endpoints. The incidence of events in patients in whom R-wave amplitude decreased (normalresponse) and in those in whom R-wave amplitude did not change or increase (abnormal response) was compared.The incidence of coronary events in patients with a normal response was 23% and in those with an abnormal response,45.8% (p<0.01). Correlating the results with several non invasive and angiographic variables, an abnormal R-waveresponse showed a significantly higher rate of events in the subsets of patients with prior myocardial infarction, absenceof cardiomegaly, maximal functional capacity lower than 4 METs, maximal heart rate higher than 140 beats/min andabnormal left ventricular function. Conclusion: The changes in R-wave amplitude after exercise is a variable thatshould be taken into account when assessing the risk of future events in patients with coronary heart disease.

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14

Smith, Timothy W., and Arthur S. Leon. "Coronary Heart Disease." Journal of Cardiopulmonary Rehabilitation 14, no. 3 (May 1994): 201. http://dx.doi.org/10.1097/00008483-199405000-00013.

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15

Emery, Charles F. "Coronary Heart Disease." Journal of Cardiopulmonary Rehabilitation 14, no. 3 (May 1994): 201. http://dx.doi.org/10.1097/00008483-199405010-00013.

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16

Smith, T. W., A. S. Leon, and Robert G. McMurray. "Coronary Heart Disease." Medicine & Science in Sports & Exercise 26, no. 1 (January 1994): 118. http://dx.doi.org/10.1249/00005768-199401000-00023.

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17

Lutz, Wolfgang. "CORONARY HEART DISEASE." Nutrition Today 21, no. 2 (March 1986): 40. http://dx.doi.org/10.1097/00017285-198603000-00007.

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18

Ellsworth, DarrellL, Phyliss Sholinsky, Cashell Jaquish, RichardR Fabsitz, and TeriA Manolio. "Coronary heart disease:." American Journal of Preventive Medicine 16, no. 2 (February 1999): 122–33. http://dx.doi.org/10.1016/s0749-3797(98)00138-x.

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19

Stewart, B. Fendley. "Coronary Heart Disease." Physical Medicine and Rehabilitation Clinics of North America 6, no. 1 (February 1995): 37–53. http://dx.doi.org/10.1016/s1047-9651(18)30477-7.

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20

O’Rourke, Robert A., Kanu Chatterjee, and Jeanne Y. Wei. "Coronary heart disease." Journal of the American College of Cardiology 10, no. 2 (August 1987): 52A—56A. http://dx.doi.org/10.1016/s0735-1097(87)80449-7.

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21

Friesinger, Gottlieb C., and Thomas J. Ryan. "CORONARY HEART DISEASE." Cardiology Clinics 17, no. 1 (February 1999): 93–122. http://dx.doi.org/10.1016/s0733-8651(05)70059-x.

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22

Bishop, Tina. "Coronary heart disease." Primary Health Care 26, no. 5 (May 26, 2016): 14. http://dx.doi.org/10.7748/phc.26.5.14.s18.

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23

Cook, Rosemary. "Coronary heart disease." Primary Health Care 6, no. 6 (June 1988): 25–30. http://dx.doi.org/10.7748/phc.6.6.23.s11.

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24

Robinson, Karen. "Coronary heart disease." Emergency Nurse 9, no. 7 (November 2001): 29–34. http://dx.doi.org/10.7748/en2001.11.9.7.29.c1376.

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25

Augustin, Joi, and Johanna Dwyer. "Coronary heart disease." Topics in Clinical Nutrition 10, no. 1 (December 1994): 1–13. http://dx.doi.org/10.1097/00008486-199410010-00003.

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26

&NA;. "CORONARY HEART DISEASE." Nursing 19, no. 1 (January 1989): 97–99. http://dx.doi.org/10.1097/00152193-198901000-00031.

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27

Tunstall-Pedoe, H. "Coronary heart disease." BMJ 303, no. 6804 (September 21, 1991): 701–4. http://dx.doi.org/10.1136/bmj.303.6804.701.

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28

McMurray, J., and H. J. Dargie. "Coronary heart disease." BMJ 303, no. 6816 (December 14, 1991): 1546–47. http://dx.doi.org/10.1136/bmj.303.6816.1546-a.

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29

Poole-Wilson, P. A., and G. Sutton. "Coronary heart disease." BMJ 304, no. 6825 (February 22, 1992): 504–5. http://dx.doi.org/10.1136/bmj.304.6825.504-b.

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30

Davies, M. J. "CORONARY DISEASE: The pathophysiology of acute coronary syndromes." Heart 83, no. 3 (March 1, 2000): 361–66. http://dx.doi.org/10.1136/heart.83.3.361.

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31

Gershlick, A. H. "CORONARY DISEASE: Role of stenting in coronary revascularisation." Heart 86, no. 1 (July 1, 2001): 104–12. http://dx.doi.org/10.1136/heart.86.1.104.

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32

JURGENSEN, J. S. "Severe aneurysmal coronary artery disease." Heart 86, no. 4 (October 1, 2001): 404. http://dx.doi.org/10.1136/heart.86.4.404.

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33

Saesarwati, Desta, and Prijono Satyabakti. "ANALISIS FAKTOR RISIKO YANG DAPAT DIKENDALIKAN PADA KEJADIAN PJK USIA PRODUKTIF." Jurnal PROMKES 4, no. 1 (September 6, 2017): 22. http://dx.doi.org/10.20473/jpk.v4.i1.2016.22-33.

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Coronary heart disease is one of cardiovascular disease dan being number one of mortalitycause in world. Coronay heart disease is begun with atherosclerosis. Atherosclerosis is process ofaccumulation of cholesterol in wall of heart’s blodd vessels that causes stenosis (obstruction). Thesedays coronary heart disease attacked productive people. Some risk factors of coronary heart diseaseare smoking habit, physical inactivity, hypertension, intake of unhealthy food, and stress. The aims ofthis research was to analyse modifi able risk factors of coronary heart disease in productive age thatconsists of smoking habit, physical inactivity, and stress. This study was an analytical study that usedcross sectional design conducted at cardiology clinic RSU Haji Surabaya at March-May 2016. Thepopulation in this study were all patients of cardiology clinic in RSU Haji Surabaya with 91 patientsas samples. The data described that there were no signifi cant correlation between smoking habit (p =0,22), physical inactivity (p = 0,79), dan stress (p = 0,06) with the incident of coronary heart diseasein productive age, and there was signifi cant correlation between passive smoker status with correlationof coronary heart disease in productive age (p = 0,01). The conclusion are there were no signifi cantcorrelation between smoking habit, physical inactivity, and stress with incident of coronary heart diseasein productive age, and there was signifi cant correlation between passive smoker status with incident ofcoronary heart disease in productive age.Keywords: risk factors, coronary heart disease, productive age

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34

Rahman, M., A. Sumin та A. Ankudinov. "СОСТОЯНИЕ КОРОНАРНОГО РУСЛА ПРИ ИШЕМИЧЕСКОЙ БОЛЕЗНИ СЕРДЦА У ПАЦИЕНТОВ С ГИПОТИРЕОЗОМ". Baikal Medical Journal 2, № 4 (25 грудня 2023): 64–71. http://dx.doi.org/10.57256/2949-0715-2023-4-64-71.

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Hypothyroidism is a significant aggravating factor for coronary heart disease (CHD). However, there are no practical recommendations for the management of this group to date. The main method of assessing the severity of coronary artery disease is a morphological assessment of the condition of the coronary arteries. The aim. To carry out comparative assessment of clinical parameters, including the state of the coronary bed in patients with coronary artery disease with newly diagnosed (without hormone replacement therapy) and primary manifest hypothyroidism compared with patients with coronary artery disease without hypothyroidism. Materials and methods. Two hundred and forty patients with CHD were examined. The study group included 90 patients with coronary heart disease and hypothyroidism, the comparison group consisted of 150 patients with coronary artery disease without thyroid pathology. A comprehensive examination of patients, including coronary angiography, was performed. The frequency of cases and characteristics of lesions of the coronary bed, the volume of interventions were assessed. The analysis of the obtained data was carried out in Statistica 10.0 (StatSoft Inc., USA). Results. In the study group (CHD and hypothyroidism), a statistically significantly increased level of myoglobin, a change in lipid profile parameters in relation to patients with CHD without hypothyroidism was found. In the group of patients with coronary artery disease and hypothyroidism, a predominance of the frequency of multivessel lesions of the coronary bed was revealed. The volume of surgical interventions between the examined patients did not reveal significant differences. Conclusion. Patients with coronary heart disease and hypothyroidism should be considered as a group of additional cardiovascular risk and require a more careful approach. It is possible to prescribe higher dosages of statins.

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35

Fox, K. A. A. "CORONARY DISEASE: Acute coronary syndromes: presentation---clinical spectrum and management." Heart 84, no. 1 (July 1, 2000): 93. http://dx.doi.org/10.1136/heart.84.1.93.

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36

León Alvarez, Jorge Luis, Michel Curbelo López, Tania Hidalgo Costa, Miguel Ángel Yanes Quesada, Raúl Orlando Calderín Bouza, and Andrew Sefenu Yao Dzebu. "Circadian Blood Pressure Variability in Hypertensive Patients with Coronary Heart Disease." Clinical Cardiology and Cardiovascular Interventions 3, no. 2 (February 4, 2020): 01–10. http://dx.doi.org/10.31579/2641-0419/039.

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Introduction and objectives: Coronary heart disease is one of the complications of hypertension. Ambulatory blood pressure monitoring allows studying the different circadian patterns of blood pressure, but there is not enough evidence linking coronary heart disease with different circadian patterns. This research was carried out with the purpose of characterizing the circadian patterns of blood pressure in patients with essential hypertension and coronary heart disease. Methods: Descriptive and transversal research in 68 hypertensive patients with coronary heart disease who underwent ambulatory blood pressure monitoring. The different circadian patterns of blood pressure were related to the different forms of coronary heart disease and to the control of blood pressure. Results: In the sample, women (60.3%) predominated over men (39.7%). The ages were between 45 to 59 (63.3%) and those 60 and over (36.7%). 44.1% did not have their blood pressure under control, 45.6% were overweight and 38.2% were obese. The abnormal patterns were dipper 42.6%, non-dipper 41.2%, extreme dipper 7.4% and riser 8.8%. Morning surge was observed in 29 patients (42.6%), statistically significant in relation to the control of blood pressure (p = 0.033), but not with the different types of coronary heart disease studied. Conclusions: A statistically significant relationship was found between morning surge and control of blood pressure.

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37

Yakovleva, L. N. "Stable coronary heart disease." Medicine of Ukraine, no. 4(230) (March 29, 2019): 45–50. http://dx.doi.org/10.37987/1997-9894.2019.4(230).185661.

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38

Lee, I.-Min, Ralph S. Paffenbarger, and Paul D. Thompson. "Preventing Coronary Heart Disease." Physician and Sportsmedicine 29, no. 2 (February 2001): 37–52. http://dx.doi.org/10.3810/psm.2001.02.366.

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39

Omenn, Gilbert S., Shirley A. A. Beresford, and Arno G. Motulsky. "Preventing Coronary Heart Disease." Circulation 97, no. 5 (February 10, 1998): 421–24. http://dx.doi.org/10.1161/01.cir.97.5.421.

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40

Jairath, N., and Anne Woods. "Managing coronary heart disease." Dimensions of Critical Care Nursing 18, no. 5 (September 1999): 54. http://dx.doi.org/10.1097/00003465-199909000-00016.

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41

Jackson, Rod, John Lynch, and Sam Harper. "Preventing coronary heart disease." BMJ 332, no. 7542 (March 16, 2006): 617–18. http://dx.doi.org/10.1136/bmj.332.7542.617.

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42

King, Kathryn M., and Heather M. Arthur. "Coronary Heart Disease Prevention." Journal of Cardiovascular Nursing 18, no. 4 (September 2003): 274–81. http://dx.doi.org/10.1097/00005082-200309000-00006.

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43

Hayman, Laura L., and Patricia R. Reineke. "Preventing Coronary Heart Disease." Journal of Cardiovascular Nursing 18, no. 4 (September 2003): 294–301. http://dx.doi.org/10.1097/00005082-200309000-00008.

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44

Ockene, I. S., and J. K. Ockene. "Preventing coronary heart disease." Tobacco Control 2, no. 2 (June 1, 1993): 165. http://dx.doi.org/10.1136/tc.2.2.165b.

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45

Assaf, Annlouise R., Kate L. Lapane, Joyce L. McKenney, Sonja McKinlay, and Richard A. Carleton. "Coronary heart disease surveillance." Journal of Clinical Epidemiology 53, no. 4 (April 2000): 419–26. http://dx.doi.org/10.1016/s0895-4356(99)00183-3.

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46

Thompson, GilbertR, Rossitza Naoumova, Paul Sidhu, Richard Underwood, PaulD Flynn, CarolA Seymour, DerekG Cook, F. Game, and R. Neary. "Predicting coronary heart disease." Lancet 343, no. 8898 (March 1994): 670–72. http://dx.doi.org/10.1016/s0140-6736(94)92664-6.

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47

Rose, Geoffrey, M. P. H. Doublet Stewart, ChristopherA Birt, PeterR Croft, and PatrickF James. "CORONARY HEART DISEASE PREVENTION." Lancet 332, no. 8619 (November 1988): 1081–82. http://dx.doi.org/10.1016/s0140-6736(88)90102-x.

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48

Mccormick, JamesS, Petr Skrabanek, J. A. Lee, and R. C. Cottrell. "CORONARY HEART DISEASE PREVENTION." Lancet 332, no. 8621 (November 1988): 1189–90. http://dx.doi.org/10.1016/s0140-6736(88)90252-8.

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49

Henderson, Andrew. "Coronary heart disease: Overview." Lancet 348 (November 1996): S1—S4. http://dx.doi.org/10.1016/s0140-6736(96)98001-0.

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50

Messerli, Franz H. "Ephemeral Coronary Heart Disease." European Heart Journal 40, no. 24 (June 21, 2019): 1906–8. http://dx.doi.org/10.1093/eurheartj/ehz400.

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