Artículos de revistas:

1

IGWE, ORISA J., SHANE S. QUE HEE y W. D. WAGNER. "Interaction between 1,2-Dichloroethane and Disulfiram". Toxicological Sciences 6, n.º 4 (1986): 733–46. http://dx.doi.org/10.1093/toxsci/6.4.733.

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2

IGWE, O. "Interaction between 1,2-dichloroethane and disulfiram I. Toxicologic effects". Fundamental and Applied Toxicology 6, n.º 4 (mayo de 1986): 733–46. http://dx.doi.org/10.1016/0272-0590(86)90186-7.

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3

Brittebo, E. B., B. Kowalski, H. Ghantous y I. Brandt. "Epithelial binding of 1,2-dichloroethane in mice". Toxicology 56, n.º 1 (mayo de 1989): 35–45. http://dx.doi.org/10.1016/0300-483x(89)90210-2.

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4

Li, Wenxue, Liping Chen, Yiwei Su, Hua Yin, Yaqin Pang y Zhixiong Zhuang. "1,2-Dichloroethane induced nephrotoxicity through ROS mediated apoptosis in vitro and in vivo". Toxicology Research 4, n.º 5 (2015): 1389–99. http://dx.doi.org/10.1039/c5tx00056d.

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5

Yllner, Sven. "Metabolism of 1,2-Dichloroethane-14C in the Mouse". Acta Pharmacologica et Toxicologica 30, n.º 3-4 (13 de marzo de 2009): 257–65. http://dx.doi.org/10.1111/j.1600-0773.1971.tb00657.x.

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6

Igwe, Orisa J., Shane S. Que Hee y William D. Wagner. "Interaction between 1,2-dichloroethane and tetraethylthiuram disulfide (disulfiram)". Toxicology and Applied Pharmacology 86, n.º 2 (noviembre de 1986): 286–97. http://dx.doi.org/10.1016/0041-008x(86)90059-1.

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7

Zeng, Ni, Zhen Zhang, Hongmei Jiang, Ruobi Li, Chong Chang, Fei Wang, Dandan Xu et al. "LncRNA-241 inhibits 1,2-Dichloroethane-induced hepatic apoptosis". Toxicology in Vitro 61 (diciembre de 2019): 104650. http://dx.doi.org/10.1016/j.tiv.2019.104650.

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8

PAYAN, J. P., A. M. SAILLENFAIT, P. BONNET, J. P. FABRY, I. LANGONNE y J. P. SABATE. "Assessment of the Developmental Toxicity and Placental Transfer of 1,2-Dichloroethane in Rats". Toxicological Sciences 28, n.º 2 (1995): 187–98. http://dx.doi.org/10.1093/toxsci/28.2.187.

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9

Payan, J. "Assessment of the Developmental Toxicity and Placental Transfer of 1,2-Dichloroethane in Rats". Fundamental and Applied Toxicology 28, n.º 2 (diciembre de 1995): 187–98. http://dx.doi.org/10.1006/faat.1995.1159.

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10

Payan, J. P., D. Beydon, J. P. Fabry, M. T. Brondeau, M. Ban y J. de Ceaurriz. "Urinary thiodiglycolic acid and thioether excretion in male rats dosed with 1,2-dichloroethane". Journal of Applied Toxicology 13, n.º 6 (noviembre de 1993): 417–22. http://dx.doi.org/10.1002/jat.2550130608.

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11

Lin, Edith L. C., Joan K. Mattox y Michael A. Pereira. "Glutathione plus cytosol- and microsome-mediated binding of 1,2-dichloroethane to polynucleotides". Toxicology and Applied Pharmacology 78, n.º 3 (mayo de 1985): 428–35. http://dx.doi.org/10.1016/0041-008x(85)90249-2.

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12

Wang, S. Y., Y. C. Kuo, Y. Z. Huang, C. W. Huang y C. M. Kao. "Bioremediation of 1,2-dichloroethane contaminated groundwater: Microcosm and microbial diversity studies". Environmental Pollution 203 (agosto de 2015): 97–106. http://dx.doi.org/10.1016/j.envpol.2015.03.042.

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13

Riebeth, D. M., N. O. Belser y C. E. Castro. "Partial rapid metabolism of 1,2-dichloroethane byMethylosinus trichosporiumOB-3b". Environmental Toxicology and Chemistry 11, n.º 4 (abril de 1992): 497–501. http://dx.doi.org/10.1002/etc.5620110407.

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14

Cottalasso, Damiano, Antonella Bellocchio, Raffaella Norese, Cinzia Domenicotti, Maria Adelaide Pronzato, Luigi Fontana y Giorgio Nanni. "Effects of vitamin E on dolichol content of rats acutely treated with 1,2-dichloroethane". Toxicology 143, n.º 3 (marzo de 2000): 283–92. http://dx.doi.org/10.1016/s0300-483x(99)00181-x.

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15

Story, David L., Earl F. Meierhenry, Charles A. Tyson y Harry A. Milman. "Differences in Rat Liver Enzyme-Altered Foci Produced By Chlorinated Aliphatics and Phenobarbital". Toxicology and Industrial Health 2, n.º 4 (octubre de 1986): 351–62. http://dx.doi.org/10.1177/074823378600200402.

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Nine chlorinated aliphatics (CAs)—1,1-dichloroethane, 1,2-dichloro ethane, 1,1,1-trichloroethane, 1,1,2-trichloroethane, trichloro ethylene, tetrachloroethylene, 1,1,1,2-tetrachloroethane, 1,1,2,2- tetrachloroethane, and hexachloroethane—were examined in a rat liver foci assay for evidence of initiating and promoting potential. Young adult male Osborne-Mendel rats (ten/group) were given partial hepa tectomies, followed 24 hr later by a single i.p. dose of either diethyl nitrosamine (30 mg/kg body weight) or CA, 1 wk later either a diet containing 0.05% (w/w) phenobarbital or daily oral gavage (5 × /wk) of CA in corn oil for 7 weeks, and sacrificed 1 wk later. Putative preneo plastic markers monitored were foci with increased γ-glutamyltrans peptidase activity [GGT( + )]. CAs were without significant effect in the initiation protocol at the maximum tolerated dose. In the promotion protocol, 1,1-dichloroethane, 1,1,2-trichloroethane, tetrachloro ethylene, 1,1,2,2-tetrachloroethane, and hexachloroethane induced significant increases in GGT( + ) foci above control levels. Two variants of GGT( + ) foci were distinguishable, one associated predominantly with phenobarbital promotion, resembling preneoplastic foci in other models, and the other associated with CA promotion, which was less intensely stained and exhibited branching, resemblingfoci undergoing redifferentiation. The marked differences in response may relate to differences in cytotoxic potential or mechanism of action of the two types of agents.

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16

Lone, Mohammad Iqbal, Nazia Nazam, Aashiq Hussain, Shashank K. Singh, Abid Hamid Dar, Rauf Ahmad Najar, Mohammed Hussein Al-Qahtani y Waseem Ahmad. "Genotoxicity and immunotoxic effects of 1,2-dichloroethane in Wistar rats". Journal of Environmental Science and Health, Part C 34, n.º 3 (26 de mayo de 2016): 169–86. http://dx.doi.org/10.1080/10590501.2016.1193924.

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17

Ma, Jie, Haiyan Li, Richard Spiese, John Wilson, Guangxu Yan y Shaohui Guo. "Vapor intrusion risk of lead scavengers 1,2-dibromoethane (EDB) and 1,2-dichloroethane (DCA)". Environmental Pollution 213 (junio de 2016): 825–32. http://dx.doi.org/10.1016/j.envpol.2016.03.032.

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18

Muralidhara, S., R. Ramanathan, S. M. Mehta, L. H. Lash, D. Acosta y J. V. Bruckner. "Acute, Subacute, and Subchronic Oral Toxicity Studies of 1,1-Dichloroethane in Rats: Application to Risk Evaluation". Toxicological Sciences 64, n.º 1 (1 de noviembre de 2001): 135–45. http://dx.doi.org/10.1093/toxsci/64.1.135.

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19

Riebeth, D. M., N. O. Belser y C. E. Castro. "PARTIAL RAPID METABOLISM OF 1,2-DICHLOROETHANE BY METHYLOSINUS TRICHOSPORIUM OB-3b". Environmental Toxicology and Chemistry 11, n.º 4 (1992): 497. http://dx.doi.org/10.1897/1552-8618(1992)11[497:prmodb]2.0.co;2.

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20

Sweeney, Lisa M., Shakil A. Saghir y Michael L. Gargas. "Physiologically based pharmacokinetic model development and simulations for ethylene dichloride (1,2-dichloroethane) in rats". Regulatory Toxicology and Pharmacology 51, n.º 3 (agosto de 2008): 311–23. http://dx.doi.org/10.1016/j.yrtph.2008.05.002.

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21

McDermott, Catherine y James J. A. Heffron. "Toxicity of Industrially Relevant Chlorinated Organic Solvents In Vitro". International Journal of Toxicology 32, n.º 2 (marzo de 2013): 136–45. http://dx.doi.org/10.1177/1091581813482006.

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The cytotoxic effects of 4 industrially important chlorinated organic solvents, dichloromethane (DCM), 1,2-dichloroethane (DCE), trichloroethylene (TCE), and tetrachloroethylene (PERC) in vitro, were investigated. Jurkat T cells were exposed to the solvents individually for 72 hours and changes in reactive oxygen species (ROS) formation, cell proliferation, intracellular free calcium concentration ([Ca2+]), and caspase-3 activity were measured. There was a concentration-dependent increase in the ROS formation and intracellular free [Ca2+] following exposure to each of the solvents. This was accompanied by a decrease in the cell proliferation. Solvent potency decreased in the following order: PERC > TCE > DCM > DCE. Caspase-3 activity was increased in a concentration-dependent manner by TCE and PERC but was not significantly altered by DCM or DCE. n-Acetyl-l-cysteine pretreatment showed that changes in the intracellular free [Ca2+] and caspase-3 activity were independent of ROS formation. However, increased ROS formation did play a causal role in the decreased cell proliferation observed.

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22

Colacci, Annamaria, Mario Mazzullo, Giancarlo Arfellini, Giorgio Prodi y Sandro Grilli. "In vitro microsome- and cytosol-mediated binding of 1,2-dichloroethane and 1,2-dibromoethane with DNA". Cell Biology and Toxicology 1, n.º 2 (enero de 1985): 45–55. http://dx.doi.org/10.1007/bf00717790.

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23

Baertsch, Amadeus, Werner K. Lutz y Christian Schlatter. "Effect of inhalation exposure regimen on DNA binding potency of 1,2-dichloroethane in the rat". Archives of Toxicology 65, n.º 3 (abril de 1991): 169–76. http://dx.doi.org/10.1007/bf02307305.

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24

Hughes, K., M. E. Meek y I. Caldwell. "1,2‐Dichloroethane: Evaluation of risks to health from environmental exposure in Canada". Journal of Environmental Science and Health, Part C 12, n.º 2 (noviembre de 1994): 293–303. http://dx.doi.org/10.1080/10590509409373448.

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25

Huang, Manqi, Yizhou Zhong, Li Lin, Boxuan Liang, Jun Liu, Junying Jiang, Manjiang Hu et al. "1,2-Dichloroethane induces cerebellum granular cell apoptosis via mitochondrial pathway in vitro and in vivo". Toxicology Letters 322 (abril de 2020): 87–97. http://dx.doi.org/10.1016/j.toxlet.2020.01.004.

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26

Laurent, Bodin, Solal Cécilia, Roudot Alain-Claude, Falcy Michel, Mullot Jean-Ulrich, Enriquez Brigitte y Rousselle Christophe. "Proposal of a new carcinogenic toxicological reference value (TRV) for inhalation exposure to 1,2-dichloroethane". Toxicology Letters 189 (septiembre de 2009): S242. http://dx.doi.org/10.1016/j.toxlet.2009.06.460.

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27

Frederick Frasch, H., Ana M. Barbero, Houda Alachkar y James N. McDougal. "Skin Penetration And Lag Times Of Neat And Aqueous Diethyl Phthalate, 1,2-Dichloroethane And Naphthalene". Cutaneous and Ocular Toxicology 26, n.º 2 (enero de 2007): 147–60. http://dx.doi.org/10.1080/15569520701212274.

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28

Storer, Richard D. y Rory B. Conolly. "An investigation of the role of microsomal oxidative metabolism in the in vivo genotoxicity of 1,2-dichloroethane". Toxicology and Applied Pharmacology 77, n.º 1 (enero de 1985): 36–46. http://dx.doi.org/10.1016/0041-008x(85)90265-0.

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29

Mohsenzadeh, Mahdi, Seyed Ahmad Mirbagheri y Samad Sabbaghi. "Degradation of 1,2-dichloroethane by photocatalysis using immobilized PAni-TiO2 nano-photocatalyst". Environmental Science and Pollution Research 26, n.º 30 (30 de agosto de 2019): 31328–43. http://dx.doi.org/10.1007/s11356-019-06240-5.

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30

Giri, Ashok Kumar y Shane S. Quehee. "In vivo sister chromatid exchange induced by 1,2-dichloroethane on bone marrow cells of mice". Environmental Mutagenesis 12, n.º 3 (1988): 331–34. http://dx.doi.org/10.1002/em.2860120307.

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31

Daniel, F. Bernard, Merrel Robinson, Greg R. Olson, Raymond G. York y L. W. Condie. "Ten and Ninety-Day Toxicity Studies of 1,2-Dichloroethane in Sprague-Dawley Rats". Drug and Chemical Toxicology 17, n.º 4 (enero de 1994): 463–77. http://dx.doi.org/10.3109/01480549409014312.

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32

Siddiqui, M. KJ, U. Nigam, S. Srivastava, D. S. Tejeshwar y Chandrawati. "Association of maternal blood pressure and hemoglobin level with organochlorines in human milk". Human & Experimental Toxicology 21, n.º 1 (enero de 2002): 1–6. http://dx.doi.org/10.1191/0960327102ht198oa.

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Milk samples of women from the general population in Lucknow were analysed by gas–liquid chromatography to determine the concentration of organochlorine pesticides and their possible association with maternal characteristics. p,p0-DDT (dichloro diphenyltrichloroethane),p,p-DDE (dichloro diphenyl trichloroethylene), p,p0-DDD (dichloro diphenyl dichloroethane), ¬, 00ad, fi and flisomers of hexachlorocyclohexane (HCH) and aldrin were detected. The calculated daily intake of DDT and HCH by the neonates exceeded their acceptable daily intake (ADI) set by the WHO. A statistically significant association was noted between ¬-HCH residues in milk and hemoglobin (Hb) level in maternal blood (P<0.001). Also, there was a significant association between fi-HCH and p,p0-DDT levels in milk and diastolic blood pressure of mother (P<0.05). An apparent negative relationship between p,p0-DDE level in mother's milk and birthweight of the baby was also noticed (P<0.05). These results highlight the need for periodic monitoring to determine organochlorine contamination in human milk and assess their impact on mother and on neonatal health.

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33

Thomas, L., B. DeFeo, M. F. Mariani y G. D. V. van Rossum. "Comparison of metabolic effects of carbon tetrachloride and 1,2-dichloroethane added in vitro to slices of rat liver". Toxicology in Vitro 3, n.º 1 (enero de 1989): 59–68. http://dx.doi.org/10.1016/0887-2333(89)90025-8.

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34

Ansari, G. A. S., Jose C. Gan y Brian K. Barton. "Synergistic inactivation of plasma ?1-proteinase inhibitor by aldehydes of cigarette smoke with styrene oxide and 1,2-dichloroethane". Archives of Environmental Contamination and Toxicology 17, n.º 4 (julio de 1988): 533–36. http://dx.doi.org/10.1007/bf01055520.

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35

Eldakroory, SA, DA El Morsi, RH Abdel-Rahman, S. Roshdy, MS Gouida y EO Khashaba. "Correlation between toxic organochlorine pesticides and breast cancer". Human & Experimental Toxicology 36, n.º 12 (8 de enero de 2017): 1326–34. http://dx.doi.org/10.1177/0960327116685887.

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Organochlorines (OCs) are common environmental pollutants that have been linked to cancer. This work aims to assess the role of OCs as a risk factor for breast cancer and to evaluate the cellular changes induced by exposure to such environmental contaminants. The study included 70 cancer patients subjected to thorough history taking and routine investigations. Samples from tumor and normal adjacent tissue were taken to measure OCs’ levels and to perform molecular analysis (some oncogenic and apoptotic markers) by flow cytometry. There were significantly higher concentrations of methoxychlor, dichloro-diphenyl-trichloroethane (DDT), hexa-chlorobenzene (HCB), and chlordane in tumor tissue samples compared to the surrounding normal tissue. There was a positive statistically significant correlation between G2m and dichloro-diphenyl-dichloroethane, DDT, and methoxychlor. There was also a negative correlation between propidium iodide (PI) and heptachlor as well as between PI, B-cell lymphoma 2, and methoxychlor. Annexin showed a negative correlation with HCB and methoxychlor. In conclusion, the higher level of organochlorine pesticides in the tissue specimens of breast cancer and the resultant molecular dysfunction highlight a possible association. Further research is warranted to elucidate the other possible mechanisms involved in the process of carcinogenesis.

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36

Boverhof, D. R., J. A. Hotchkiss, M. J. LeBaron, L. M. Segal y C. Howick. "Investigation of the potential non-genotoxic mode of action for 1,2-Dichloroethane (DCE)-induced mammary tumors in female rats". Toxicology Letters 295 (octubre de 2018): S150—S151. http://dx.doi.org/10.1016/j.toxlet.2018.06.750.

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37

Kramers, P. G. N., H. C. A. Mout, B. Bissumbhar y C. R. Mulder. "Inhalation exposure in Drosophila mutagenesis assays: experiments with aliphatic halogenated hydrocarbons, with emphasis on the genetic activity profile of 1,2-dichloroethane". Mutation Research/Environmental Mutagenesis and Related Subjects 252, n.º 1 (febrero de 1991): 17–33. http://dx.doi.org/10.1016/0165-1161(91)90248-7.

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38

SCHULZ, GÜNTER y GISELA BIRKHAHN. "Kinetic Analogies Between Decomposition of 1,2-Dichloroethane and Chloroform Under Non-Thermal Reducing RF Plasma Conditions". Hazardous Waste and Hazardous Materials 13, n.º 4 (enero de 1996): 465–72. http://dx.doi.org/10.1089/hwm.1996.13.465.

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39

Zhang, Lin y Ya‐ping Jin. "Toxic effects of combined treatment of 1,2‐dichloroethane and ethanol on mouse brain and the related mechanisms". Journal of Biochemical and Molecular Toxicology 33, n.º 5 (21 de enero de 2019): e22294. http://dx.doi.org/10.1002/jbt.22294.

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40

Zhong, Yizhou, Boxuan Liang, Manjiang Hu, Jun Liu, Li Lin, Junying Jiang, Xi Lin et al. "MicroRNA-29b-3p aggravates 1,2-dichloroethane-induced brain edema by targeting aquaporin 4 in Sprague-Dawley rats and CD-1 mice". Toxicology Letters 319 (febrero de 2020): 160–67. http://dx.doi.org/10.1016/j.toxlet.2019.11.011.

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41

Liang, Boxuan, Yizhou Zhong, Bo Wang, Li Lin, Jun Liu, Xi Lin, Yuji Huang et al. "1,2-Dichloroethane induces apoptosis in the cerebral cortexes of NIH Swiss mice through microRNA-182-5p targeting phospholipase D1 via a mitochondria-dependent pathway". Toxicology and Applied Pharmacology 430 (noviembre de 2021): 115728. http://dx.doi.org/10.1016/j.taap.2021.115728.

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42

Lund, B. O., Å. Bergman y I. Brandt. "In vitro macromolecular binding of 2-(2-chlorophenyl)-2-(4-chlorophenyl)-1,1-dichloroethane (o,p′-DDD) in the mouse lung and liver". Chemico-Biological Interactions 70, n.º 1-2 (1989): 63–72. http://dx.doi.org/10.1016/0009-2797(89)90063-x.

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43

Suguro, Mayuko, Takamasa Numano, Mayumi Kawabe, Yuko Doi, Norio Imai, Yukinori Mera y Seiko Tamano. "Lung Tumor Induction by 26-week Dermal Application of 1,2-Dichloroethane in CB6F1-Tg rasH2 Mice". Toxicologic Pathology 45, n.º 3 (abril de 2017): 427–34. http://dx.doi.org/10.1177/0192623317701003.

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Short-term alternatives to traditional 2-year carcinogenic studies in rodents are being actively pursued. Recently, a 26-week short-term carcinogenicity study using CB6F1-Tg rasH2@Jcl (rasH2) mice has become a worldwide standard for the evaluation of chemical carcinogenesis. However, an acceptable short-term carcinogenic study model for dermally applied products is still lacking. To investigate the suitability of using the rasH2 mouse to test carcinogenic potential, 1,2-dichloroethane (1,2-DCE) was dermally applied to rasH2 mice: 1,2-DCE is a known carcinogen that causes lung bronchiolo-alveolar adenomas and adenocarcinomas when administered topically, orally, or by inhalation exposure; 1,2-DCE at a dose level of 126 mg/mouse in 200 μl acetone or acetone alone (vehicle control) was applied to the dorsal skin of 10 mice of each sex 3 times a week for 26 weeks. As a positive control, 10 mice of each sex received a single intraperitoneal injection of 75 mg/kg of N-methyl- N-nitrosourea. Bronchiolo-alveolar adenomas and adenocarcinomas were significantly increased in 1,2-DCE-treated rasH2 mice of both sexes, and bronchiolo-alveolar hyperplasias were significantly increased in female mice. Overall, almost all mice of each sex developed adenomas and/or adenocarcinomas with 100% of female rasH2 mice developing bronchiolo-alveolar adenocarcinomas.

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44

Wang, Gaoyang, Yuan Yuan, Jun Zhang, Lanyue Gao, Xiaoqiong Tan, Guangqian Yang, Xiuqiang Lv y Yaping Jin. "Roles of aquaporins and matrix metalloproteinases in mouse brain edema formation induced by subacute exposure to 1,2-dichloroethane". Neurotoxicology and Teratology 44 (julio de 2014): 105–12. http://dx.doi.org/10.1016/j.ntt.2014.06.005.

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45

Jin, Xiaoxia, Yingjun Liao, Xiaoqiong Tan, Jingjing Guo, Gaoyang Wang, Fenghong Zhao y Yaping Jin. "Involvement of the p38 MAPK signaling pathway in overexpression of matrix metalloproteinase-9 during the course of brain edema in 1,2-dichloroethane-intoxicated mice". NeuroToxicology 69 (diciembre de 2018): 296–306. http://dx.doi.org/10.1016/j.neuro.2018.07.022.

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46

Leblond, Vincent S. y Alice Hontela. "Effects ofin VitroExposures to Cadmium, Mercury, Zinc, and 1-(2-Chlorophenyl)-1-(4-chlorophenyl)-2,2-dichloroethane on Steroidogenesis by Dispersed Interrenal Cells of Rainbow Trout (Oncorhynchus mykiss)". Toxicology and Applied Pharmacology 157, n.º 1 (mayo de 1999): 16–22. http://dx.doi.org/10.1006/taap.1999.8660.

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47

Zhou, Zhengyuan, Jingyao Huang, Muhammad Danish, Guilu Zeng, Rumin Yang, Xiaogang Gu, Meesam Ali y Shuguang Lyu. "Insights into enhanced removal of 1,2-dichloroethane by amorphous boron-enhanced Fenton system: Performances and mechanisms". Journal of Hazardous Materials 420 (octubre de 2021): 126589. http://dx.doi.org/10.1016/j.jhazmat.2021.126589.

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48

JENSEN, B. "Toxicity, ultrastructural effects, and metabolic studies with 1-(o-chlorophenyl)-1-(p-chlorophenyl)-2,2-dichloroethane(o,p?-DDD) and its methyl analog in the guinea pig and rat*1". Toxicology and Applied Pharmacology 87, n.º 1 (enero de 1987): 1–9. http://dx.doi.org/10.1016/0041-008x(87)90078-0.

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49

Wei, Yu-Ting, Shian-chee Wu, Shi-Wei Yang, Choi-Hong Che, Hsing-Lung Lien y De-Huang Huang. "Biodegradable surfactant stabilized nanoscale zero-valent iron for in situ treatment of vinyl chloride and 1,2-dichloroethane". Journal of Hazardous Materials 211-212 (abril de 2012): 373–80. http://dx.doi.org/10.1016/j.jhazmat.2011.11.018.

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50

Al-Baldawi, Israa Abdulwahab. "Removal of 1,2-Dichloroethane from real industrial wastewater using a sub-surface batch system with Typha angustifolia L". Ecotoxicology and Environmental Safety 147 (enero de 2018): 260–65. http://dx.doi.org/10.1016/j.ecoenv.2017.08.022.

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Artículos de revistas sobre el tema "Dichloroethane – Toxicology"

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