Bibliografías temáticas / Entamoeba histolytica

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Literatura académica sobre el tema "Entamoeba histolytica"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 13 de junio de 2025

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Artículos de revistas sobre el tema "Entamoeba histolytica"

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Quispe-Rodríguez, Gabriel H., Alyssa A. Wankewicz, José Luis Málaga Granda, Ben Lewis, Kassi Stockert, and A. Clinton White. "‘Entamoeba histolytica’ identified by stool microscopy from children with acute diarrhoea in Peru is not E. histolytica." Tropical Doctor 50, no. 1 (2019): 19–22. http://dx.doi.org/10.1177/0049475519881037.

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Entamoeba histolytica is a rare but feared pathogen owing to its related morbidity and mortality. Physicians in an ambulatory clinic in Cusco noted frequent reports of E. histolytica diagnosed by microscopy. Other non-pathogenic species of Entamoeba have an identical microscopic appearance. To determine whether the organisms were actually E. histolytica, faecal specimens from children aged six months to three years with diarrhoea were tested by a species-specific ELISA for E. histolytica antigen. Although 19/73 patients (26.0%) were presumptively diagnosed with amoebiasis based on microscopy, none were confirmed by ELISA. Most cases diagnosed as E. histolytic by microscopy in Peru are not infected by the pathogenic species and are probably colonised by non-pathogenic amoeba such as Entamoeba dispar.
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Begum, Sharmin, Hayley Gorman, Attinder Chadha, and Kris Chadee. "Entamoeba histolytica." Trends in Parasitology 37, no. 7 (2021): 676–77. http://dx.doi.org/10.1016/j.pt.2021.01.001.

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Showler, A. J., and A. K. Boggild. "Entamoeba histolytica." Canadian Medical Association Journal 185, no. 12 (2013): 1064. http://dx.doi.org/10.1503/cmaj.121576.

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Pacheco-Yépez, Judith, Rafael Campos-Rodrı́guez, Jesús Serrano-Luna, et al. "Entamoeba histolytica." Archives of Medical Research 31, no. 4 (2000): S242—S244. http://dx.doi.org/10.1016/s0188-4409(00)00182-x.

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Duong, Thanh Hai, and Dominique Richard-Lenoble. "Entamoeba histolytica." EMC - Biologie Médicale 1, no. 1 (2006): 1–4. http://dx.doi.org/10.1016/s2211-9698(06)76273-2.

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TM. "Entamoeba histolytica." CME 11, no. 1 (2014): 32. http://dx.doi.org/10.1007/s11298-014-0043-9.

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Báez-Camargo, M., A. M. Riverón, D. M. Delgadillo, et al. "Entamoeba histolytica." MGG Molecular & General Genetics 253, no. 3 (1996): 289. http://dx.doi.org/10.1007/s004380050324.

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JIRKŮ-POMAJBÍKOVÁ, KATEŘINA, IVAN ČEPIČKA, BARBORA KALOUSOVÁ, et al. "Molecular identification ofEntamoebaspecies in savanna woodland chimpanzees (Pan troglodytes schweinfurthii)." Parasitology 143, no. 6 (2016): 741–48. http://dx.doi.org/10.1017/s0031182016000263.

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SUMMARYTo address the molecular diversity and occurrence of pathogenic species of the genusEntamoebaspp. in wild non-human primates (NHP) we conducted molecular-phylogenetic analyses onEntamoebafrom wild chimpanzees living in the Issa Valley, Tanzania. We compared the sensitivity of molecular [using a genus-specific polymerase chain reaction (PCR)] and coproscopic detection (merthiolate-iodine-formaldehyde concentration) ofEntamoebaspp. We identifiedEntamoebaspp. in 72 chimpanzee fecal samples (79%) subjected to species-specific PCRs for sixEntamoebaspecies/groups (Entamoeba histolytica, Entamoeba nuttalli, Entamoeba dispar, Entamoeba moshkovskii, Entamoeba coliandEntamoeba poleckiST2). We recorded threeEntamoebaspecies:E. coli(47%),E. dispar(16%),Entamoeba hartmanni(51%). Coproscopically, we could only distinguish the cysts of complexE. histolytica/dispar/moshkovskii/nuttalliandE. coli. Molecular prevalence of entamoebas was higher than the prevalence based on the coproscopic examination. Our molecular phylogenies showed that sequences ofE. disparandE. colifrom Issa chimpanzees are closely related to sequences from humans and other NHP from GenBank. The results showed that wild chimpanzees harbourEntamoebaspecies similar to those occurring in humans; however, no pathogenic species were detected. Molecular-phylogenetic methods are critical to improve diagnostics of entamoebas in wild NHP and for determining an accurate prevalence ofEntamoebaspecies.
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Ikbal, Abu Md Ashif, Bikash Debnath, Amlanjyoti Rajkhowa, Kishan Paul, Rima Majumder, and Kuntal Manna. "Amoebiasis: An Infectious Disease Caused by Entamoeba histolytica." Asian Journal of Basic Science & Research 04, no. 02 (2022): 32–40. http://dx.doi.org/10.38177/ajbsr.2022.4202.

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Amoebiasis is an infectious disease caused by Entamoeba histolytica (E. histolytica), the common symptoms are cramping, abdominal pain, watery or bloody diarrhea, and weight loss. Sometimes patients suffer amoebiasis as an asymptomatic behavior. In 1859 scientist W.D. Lambl first described the amoebiasis vector E. histolytica. In developing countries, millions of people die due to amoebiasis. A doctor diagnosed the disease detection of E. histolytic antigen in stool or antibodies against the parasite in serum. Metronidazole, tinidazole, lodoquinol, diloxanide furoate are given for amoebiasis patients. In this review, we have summarized the statistics, pathogenesis, diagnosis, prevention, and treatment strategy of amoebiasis disease. It also gives information about the life cycle of E. histolytica.
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Orozco, E., C. Gómez, D. G. Pérez, et al. "Entamoeba histolytica Hybrids." Archives of Medical Research 31, no. 4 (2000): S273—S274. http://dx.doi.org/10.1016/s0188-4409(00)00189-2.

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Tesis sobre el tema "Entamoeba histolytica"

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Gaucher, Denis. "DNA vaccination against Entamoeba histolytica." Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=82877.

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Invasive amebiasis, caused by the protozoan parasite Entamoeba histolytica, is one of the leading parasitic causes of mortality worldwide, and there are no vaccines available to control the disease. The heavy subunit of the E. histolytica Gal-lectin is regarded as a potential subunit vaccine candidate. A Th1 (cell-mediated) immune response is protective against invasive amebiasis, and DNA vaccination is a strategy to induce such a response against specific antigens. The objective of this study was to construct and test a Gal-lectin-based DNA vaccine against E. histolytica. DNA encoding as 894--1081 of the Gal-lectin heavy subunit was resynthesized using a gerbil codon frequency bias and inserted in a mammalian expression vector to generate the DNA vaccine pCISToGL6. Balb/c mice vaccinated intradermally developed a Gal-lectin-specific cellular immune response, as well as an anti-Gal-lectin humoral immune response. Serum antibodies recognized a recombinant portion of the Gal-lectin heavy subunit by immunoblot and ELISA, and bound to native Gal-lectin on the surface of live trophozoites, inhibiting adherence to target cells. The Gal-lectin-specific serum antibodies were of the IgG2a isotype, indicating that a Th1 response was stimulated by the vaccine. We were also interested in using DNA encoding IL-12, IL-18 or GM-CSF as genetic adjuvants co-injected with pCISToGL6 to potentiate the immune response. Since the DNA vaccine was destined to confer protection in the gerbil model of invasive amebiasis, we cloned gerbil IL-12 (p35 and p40), IL-18 and its convertase caspase-1, and GM-CSF. The proteins were expressed in mammalian cells and showed bioactivity in vitro. Taken together, these results have laid the foundation to optimize and test a working Gal-lectin with co-stimulatory molecules to elicit a Th1 immune response for protective immunity against invasive amebiasis.
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Di, Paolo Tiziano. "Stress response in Entamoeba histolytica." Thesis, McGill University, 1994. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=68169.

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The heat shock response was studied in the intestinal parasitic protozoan Entamoeba histolytica. Temperature shifts from 37$ sp circ$C to 44$ sp circ$C enhanced the synthesis of five major heat shock (or stress) proteins (HSP) of 100, 50, 42, 37, and 28 kDa. Similarly, exposure of amebae to lymphokine activated macrophages and hydrogen peroxide caused HSP expression. Heat shock caused the reversible inhibition of amebic adherence to Chinese hamster ovary cells and human colonic mucin binding to trophozoites by ${>80 %}$. This was due to a decrease in the surface expression of the Gal/GalNAc adherence lectin and a marked reduction in the lectin mRNA expression. However, the presence of target Chinese hamster ovary cells during recovery at 37$ sp circ$C augmented amebic adherence. These results suggest that E. histolytica trophozoites produce a variety of HSP in response to different stimuli and can modulate the expression of the surface adherence lectin which maybe important in pathogenesis.
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Weston, Christopher John. "The unusual transhydrogenase of Entamoeba histolytica." Thesis, University of Birmingham, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246700.

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Preativatanyou, Kanok. "Molecular characterisation of virulence in Entamoeba histolytica." Thesis, University of Liverpool, 2015. http://livrepository.liverpool.ac.uk/2027779/.

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Entamoeba histolytica is a parasitic protozoan that infects the human digestive tract. Infection results from ingestion of cyst-contaminated food or water. To date, E. histolytica infection remains a major worldwide public health problem in worldwide endemic areas. The spectrum of clinical manifestations ranges from asymptomatic carrier to mucous and bloody diarrhea or even extraintestinal amoebiasis, usually amoebic liver abscess. Several molecular studies have been carried out to reveal novel aspects of E. histolytica infection. However, the studies focused on genomic-wide analysis comparing between E. histolytica strains are still limited. Thus, the aims of this project are to comprehensively study the comparative analysis of the whole and small RNA transcriptomes amongst nonvirulent and virulent strains of laboratory cultured E. histolytica trophozoites as well as to integrate such transcriptomic findings with the genomic data for advanced understanding of the molecular pathogenesis and virulence in amoebiasis. In this study, genome-wide transcriptome analysis using Illumina RNA-Seq technology can illustrate significant expression differences between nonvirulent and virulent E. histolytica strains. Differential gene expression analysis between nonvirulent Rahman strain and other three virulent strains (i.e. PVBM08B, HM-1:IMSS and IULA:1092:1) reveals that transcripts involved in host cell killing and mucosal invasion, nucleic acid interaction and response to oxidative stress are notably upregulated in the virulent trophozoites. InterProScan results show the upregulation of genes encoding proteolysis-related domains and the co-upregulation of cytoskeleton and actin-modulating domains in the virulent strains. Also, process ontologies related to protein degradation, cellular biosynthesis, DNA metabolism, repair and recombination, mitotic cell division, actin dynamics and response to stress are highly enriched as a core metabolism in the virulent strains, indicating the rapid growing and active metabolic state are the main drivers of virulence. However, the striking underrepresentation of ontologies involved in signaling and regulatory processes was observed in the virulent parasites. It could be inferred that reduced regulation of sensing and correctly responding to the environmental stimuli potentially enable the parasites to become virulent and subsequently cause the invasive infection. Also, NanoString validation reveals the spectrum of virulence-associated gene expression among these four strains, reflecting their different degrees of virulence. Gene copy number variation (CNV) is widespread among the genomes of the E. histolytica strains, reflecting genomic plasticity and variability in gene family content. Herein, this present data show that patterns of CNV contribute to differential expression profiles, therefore it can be extrapolated that differences in gene copy number between genomes could contribute to the variation in phenotypic attributes, including virulence, among E. histolytica strains. Also, genome plasticity can also be seen in Trypanosomes and Leishmania, suggesting that CNV is a potentially important mechanism in generating genetic diversity and regulating gene expression levels in almost exclusively asexual parasite group. For small RNA transcriptomics, the size-fractionated sRNA sequencing data demonstrate the inverse relationship between antisense sRNA abundance and target gene expression levels, strongly suggesting the sRNA-mediated regulation. Differential sRNA regulation in virulence-associated gene expression was found among strains, indicating that sRNA-mediated post-transcriptional regulation may be important in shaping the parasite virulence. In addition, this study identified the novel putative miRNA from the sRNA sequencing data using the biogenesis-based bioinformatic analysis and qPCR validation, implying that miRNA potentially play a regulatory role in E. histolytica. In summary, it can be inferred that genomic plasticity and sRNA-mediated regulation are important mechanisms of virulence modulation in E. histolytica.
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Pillai, Dylan Ravindran. "Entamoeba histolytica and Entamoeba dispar, mechanisms of adherence and implications for virulence." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0020/NQ53810.pdf.

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Santos, Gustavo Miranda Pires. "Padronização de condições experimentais no cultivo e quantificação de Entamoeba histolytica que otimizem ensaios de compostos potencialmente amebicidas." Centro de Pesquisas Gonçalo Moniz, 2011. https://www.arca.fiocruz.br/handle/icict/7153.

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Submitted by Ana Maria Fiscina Sampaio (fiscina@bahia.fiocruz.br) on 2013-10-15T17:14:57Z No. of bitstreams: 1 Gustavo Santos. Padronização de condições experimentais.2011.pdf: 3612850 bytes, checksum: 6681f5cbe674b715c7de996edc26d29d (MD5)<br>Made available in DSpace on 2013-10-15T17:14:57Z (GMT). No. of bitstreams: 1 Gustavo Santos. Padronização de condições experimentais.2011.pdf: 3612850 bytes, checksum: 6681f5cbe674b715c7de996edc26d29d (MD5) Previous issue date: 2011<br>Fundação Oswaldo Cruz. Centro de Pesquisas Gonçalo Moniz. Salvador, BA, Brasil<br>O protozoário, Entamoeba histolytica, constitui a etiologia de milhares de óbitos anuais e, em muitos casos, a falta de saneamento, o grau de instrução e a falta de higiene da população podem favorecer a transmissão e a manutenção desses patógenos em uma comunidade. Por causar tantas mortes e problemas na saúde pública trabalhos que facilitem o estudo deste parasito fazem-se importantes. Uma vez que a padronização de cultivo de E. histolytica em placas de poços vai Existem indicações que mostram que este parasito pode se tornar resistente ao medicamento utilizado no tratamento desta protozoose, por isso, a busca por novas substâncias que possam atuar como tratamento alternativo é de suma importância. Portanto, o objetivo do presente trabalho foi otimizar e padronizar o cultivo e a contagem deste parasito in vitro, além de identificar substâncias com potencial amebicida, que possam ser utilizadas no futuro como fármacos no tratamento da amebíase, sugerindo também uma via possível de ação das substâncias que apresentaram os melhores efeitos. Para tanto, os trofozoítos foram cultivados em placas de 24 poços sobre diferentes condições, quatro métodos de contagem de células foram comparados e 74 (setenta e quatro) substâncias foram testadas. Destas 13 (treze) apresentaram uma inibição na proliferação axênica dos trofozoítos de cerca de 70%. Destas, três compostos foram estudados em mais detalhes, os mesoiônicos derivados da piperina (as MII, MVI e MIX). Estas substâncias pertencem ao grupo dos compostos mesoiônicos, substâncias formadas por um anel heteroatômico composto por nitrogênio, carbono e enxofre, capazes de atravessar membranas e interagir com biomoléculas. Além disso, alguns mesoiônicos são doadores de radicais NO e tais grupamentos são capazes de induzir uma morte celular semelhante à apoptose em E. histolytica, como sugerido pela expressão de fosfatidil-serina revelada por anexina-V. Confirmando os resultados descritos na literatura, estas substâncias foram capazes de induzir uma morte programada, porém observações da ultra-estrutura, tais como figuras de mielina, das células tratadas apontaram para autofagia que também foi evidenciada por testes com MDC gerando apoptose tipo II, que pode ser iniciada pela presença de ROS, que neste caso foram por DCFDA.<br>The protozoan parasite, Entamoeba histolytica, is the etiology of thousands of deaths annually and in many cases lack of sanitation, education level and poor hygiene of the population may facilitate the transmission and maintenance of these pathogens in a community. There are indications showing that this parasite may become resistant to the drug used in treatment of protozoal disease, so the search for new substances that can act as an alternative treatment is of paramount importance. Therefore, the aim of this study was to optimize and standardize the cultivation and enumeration of this parasite in vitro, and identify substances with potential amebicidal, which can be used in future as drugs for the treatment of amoebiasis, suggesting a possible route of action of the compounds that showed the greatest effects. For this purpose, trophozoites were cultured in 24-well plates under different conditions, four methods of cell count were compared and 74 (seventy four) substances were tested. Of these thirteen (13) showed an inhibition in the proliferation of axenic trophozoites of about 70%. Of these, three compounds were studied in more detail, the mesoionic derivatives of piperine (the MII, MVI and MIX). These substances belong to the group of mesoionic compounds, formed by a heteroatomic ring composed of nitrogen, carbon and sulfur, able to traverse membranes and interact with biomolecules. Moreover, some donors are mesoionic radicals NO and such groups are able to induce a cell death similar to apoptosis in E. histolytica, as suggested by the expression of phosphatidyl-serine revealed by annexin-V. Confirming the results described in the literature, these substances were capable to inducing a programmed death, but observations of the ultra-structure, such as myelin figures, treated cells pointed out that autophagy was also evidenced by tests with MDC generating apoptosis type II can be initiated by the presence of ROS, which in this case were by DCFDA.
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Schindler, Stefan Wolfgang. "Vorkommen von Entamoeba histolytica und Entamoeba dispar in der Südosttürkei eine epidemiologische Studie /." [S.l.] : [s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=967595126.

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Wang, Wei. "The effect of Entamoeba histolytica on macrophage functions." Thesis, McGill University, 1993. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=41170.

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Infections with Entamoeba histolytica are associated with impaired cell mediated immunity by an unknown mechanism. Macrophages are the most important cells in host defense against invasive amebiasis. The present study investigated the effect of E. histolytica on macrophage functions. Macrophages isolated from gerbils with amebic liver abscess and naive macrophages exposed to soluble amebic proteins induced profound alteration of eicosanoid formation both in cyclooxygenase and lipoxygenase pathways by enhanced PGE$ sb2$ and LTC$ sb4$ production. TNF-$ alpha$ production by macrophages was altered locally in amebic granulomas and at systemic sites during the infection and in response to amebic proteins stimulation in vitro. PGE$ sb2$ produced by macrophages in response to amebic proteins was involved in the down-regulation of TNF-$ alpha$ production. E. histolytica-induced dysfunction of macrophage cytotoxicity against amebae and tumor cells occurred by suppressing NO and by enhancing PGE$ sb2$ production. Amebic proteins suppressed the induction. of IFN-$ gamma$-induced bone marrow macrophage class II MHC Ia molecule synthesis and I-A$ beta$ mRNA expression by stimulating PGE$ sb2$ production. Taken together, these results demonstrate that E. histolytica induced PGE$ sb2$ production plays a central role in the suppression of macrophage effector and accessory cell functions.
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Belley, Adam. "Molecular interactions between Entamoeba histolytica and colonic mucins." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=36752.

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The enteric protozoan parasite Entamoeba histolytica is the etiologic agent of the disease amebiasis which is characterized by colitis or hepatic lesions. Amebae colonize the colon by binding to mucous glycoproteins (mucins). Secretory mucins provide the gel nature to mucus and are a vital component of epithelial barrier function. Mucins prevent contact-dependent cytolysis of colonic cells by E. histolytica. To possibly circumvent this barrier, the parasite secretes a potent yet unidentified mucin secretagogue, which could deplete the stored mucin pool and render the mucous layer less protective. The objective of this study was to investigate the molecular mechanisms by which E. histolytica modulates colonic mucin exocytosis. We showed that E. histolytica converts exogenous arachidonic acid to prostaglandin E2 (PGE2), a known mucin secretagogue and potential mechanism by which the parasite evokes mucin secretion. Conversion was via a novel cyclooxygenase-like activity and was inhibitable with the known cyclooxygenase inhibitor aspirin. To study E. histolytica-mucin interactions, we developed an in vitro model of LS174T human colonic epithelial cells that secrete mucin constitutively and in response to mucin agonists. Highly purified mucins isolated from LS174T cells markedly inhibited amebic adherence to target cells and the mucous barrier protected the LS174T monolayers from amebic cytolysis. We have identified that Gal and GalNAc residues (O-linked sugars) of mucins are the protective moiety as O- but not N-linked glycosylation inhibitors decreased their protective effect. To understand how mucins are regulated during intestinal amebiasis and in the inflamed gut, we determined that PGE2 binds the EP4 receptor on LS174T cells and in rat colon to stimulate cyclic adenosine monophosphate-dependent mucin exocytosis. Taken together, these studies delineate how E. histolytica modulates host responses during infection to allow the parasite to survive and persist in th
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Kammanadiminti, Srinivas Jagannadha. "Early interactions between Entamoeba histolytica and mucosal cells." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=102989.

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The pathogenesis of the enteric protozoan parasite Entamoeba histolytica remains poorly understood. Moreover, the host responses during the early periods of interaction in the gut remain to be clarified. In this study I investigated the cell specific responses to the parasite and the importance of cross talk between epithelial-immune cells that could potentially influence the outcome of infection, with a central focus on Nuclear factor (NF)-kappaB. NF-kappaB is a ubiquitous transcription factor that plays a critical role in mucosal inflammation and its regulation by E. histolytica has not been studied so far. Gal-lectin is a well characterized parasite virulence factor and vaccine candidate. I first characterized the interactions between Gal-lectin and macrophages and found that several proinflammatory genes are upregulated as early as 2h. The Gal-lectin activated NF-kappaB and up-regulated Toll like receptor-2 expression in an NF-kappaB- and p38 Mitogen Activated Protein (MAP) kinase-dependent manner. As intestinal epithelial cells (IEC) form the first line of active host defense against mucosal pathogens, I determined the interaction between ameba soluble proteins and naive IEC. I observed that the parasite could elicit a chemokine response via activation of PI3 kinase and phosphorylation of p65 subunit to induce monocyte chemoattractant protein-1. The consequent recruitment of immune cells could be responsible for colonic inflammation. Finally, I made the novel observation that in macrophage-primed IEC, ameba proteins elicited a cytoprotective stress response. Using a combination of siRNA and over expression studies, I demonstrated that amebic proteins increased the expression and phosphorylation of Heat shock protein (Hsp) 27 thereby enhancing its association with and subsequent inhibition of Inhibitory kappaB kinase (IKK). The resulting inhibition of NF-kappaB could be a potential mechanism that explains the absence of inflammation in the majority of infected individuals. Taken together, the findings of this study open up a new facet in the pathogenesis of amebiasis and unravel a novel paradigm to study host-parasite interactions in the gut.
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Libros sobre el tema "Entamoeba histolytica"

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R, Kretschmer Roberto, ed. Amebiasis: Infection and disease by Entamoeba histolytica. CRC Press, 1990.

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Kretschmer, Roberto R. Amebiasis: Infection and Disease by Entamoeba Histolytica. Taylor & Francis Group, 2020.

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Kretschmer, Roberto R. Amebiasis: Infection and Disease by Entamoeba Histolytica. Taylor & Francis Group, 2020.

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Kretschmer, Roberto R. Amebiasis: Infection and Disease by Entamoeba Histolytica. Taylor & Francis Group, 2020.

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Kretschmer, Roberto R. Amebiasis: Infection and Disease by Entamoeba Histolytica. Taylor & Francis Group, 2020.

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Kretschmer, Roberto R. Amebiasis: Infection and Disease by Entamoeba Histolytica. Taylor & Francis Group, 2019.

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I, Ravdin Jonathan, ed. Amebiasis: Human infection by Entamoeba histolytica. Wiley, 1988.

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I, Ravdin Jonathan, ed. Amebiasis. Wiley, 1988.

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Amebiasis: Infection and disease by Entamoeba histolytica. CRC Press, 1990.

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Kretschmer, Roberto R. Amebiasis: Infection and Disease by Entamoeba Histolytica. Crc Pr I Llc, 1990.

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Capítulos de libros sobre el tema "Entamoeba histolytica"

1

Coia, John, and Heather Cubie. "Entamoeba histolytica." In The Immunoassay Kit Directory. Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-009-0359-3_13.

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Lamps, Laura W. "Entamoeba histolytica." In Surgical Pathology of the Gastrointestinal System: Bacterial, Fungal, Viral, and Parasitic Infections. Springer US, 2009. http://dx.doi.org/10.1007/978-1-4419-0861-2_28.

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Spice, William M., Jorge A. Cruz-Reyes, and John P. Ackers. "Entamoeba histolytica." In Molecular and Cell Biology of Opportunistic Infections in AIDS. Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-011-1530-8_5.

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Ringelmann, R., and Beate Heym. "Entamoeba histolytica." In Parasiten des Menschen. Steinkopff, 1991. http://dx.doi.org/10.1007/978-3-642-85397-5_41.

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Mehlhorn, Heinz. "Entamoeba histolytica." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_1060-2.

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Tannich, Egbert. "Entamoeba histolytica." In Lexikon der Infektionskrankheiten des Menschen. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-39026-8_305.

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Stark, Damien. "Entamoeba histolytica." In PCR for Clinical Microbiology. Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-90-481-9039-3_62.

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Mehlhorn, Heinz. "Entamoeba histolytica." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_1060.

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Shibayama, Mineko, Nidia León-Sicairos, Jesús Serrano-Luna, and Mireya de la Garza. "Entamoeba histolytica." In Laboratory Models for Foodborne Infections. CRC Press, 2017. http://dx.doi.org/10.1201/9781315120089-39.

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Serrano-Luna, Jesús, Moisés Martínez-Castillo, Nidia Leon-Sicairos, Mineko Shibayama, and Mireya de la Garza. "Entamoeba histolytica." In Handbook of Foodborne Diseases. CRC Press, 2018. http://dx.doi.org/10.1201/b22030-56.

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Actas de conferencias sobre el tema "Entamoeba histolytica"

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Bahlavouni, Alina, and Aleena Paul. "Hemolytic Uremic Syndrome Associated with Entamoeba Histolytica." In AAP National Conference & Exhibition Meeting Abstracts. American Academy of Pediatrics, 2021. http://dx.doi.org/10.1542/peds.147.3_meetingabstract.866.

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Bermudez, Frenz Marjond Q., Mar John R. Palacio, Jocelyn Flores Villaverde, and Jessie Jaye R. Balbin. "Detection of Entamoeba Histolytica using Canny Edge Detection and Watershed Transform." In ICBET 2020: 2020 10th International Conference on Biomedical Engineering and Technology. ACM, 2020. http://dx.doi.org/10.1145/3397391.3397402.

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Jamil, Hussein A., and Nebrass F. Chachain. "The effects of earthworm powder on viability of Entamoeba histolytica (trophozoites) in vitro." In 2ND INTERNATIONAL CONFERENCE ON MATERIALS ENGINEERING & SCIENCE (IConMEAS 2019). AIP Publishing, 2020. http://dx.doi.org/10.1063/5.0000349.

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Santos, Maria Isabel Quaranta Lobão dos, Lucas Martins Silva, Maria Fortes Vieria, Valrer Kolhy Perroni de Melo, Mariana Oliveira Cordeiro, and Kleverson Wessel de Oliveira. "Entamoeba histolytica: uma revisão integrativa dos aspectos patogênicos e delimitações clínicas e sociais." In III Congresso Online Brasileiro de Medicina. Congresse.me, 2022. http://dx.doi.org/10.54265/vaql3196.

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Jimenez Hernandez, Dafne Andrea, Maria del Consuelo Gomez Garcia, Maritere Dominguez Rojas, Fabiola Bello Santos, Olivia Medel Flores, and Guillermo Perez Ishiwara. "In silico Analysis of Post Translational Modifications on the EhHSTF7 factor in Entamoeba histolytica." In 2021 Mexican International Conference on Computer Science (ENC). IEEE, 2021. http://dx.doi.org/10.1109/enc53357.2021.9534809.

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Joshi, Narahari V., Honorio Medina, H. Urdaneta, and J. Barboza. "Nanoimaging and ultra structure of Entamoeba histolytica and its pseudopods by using atomic force microscope." In BiOS 2000 The International Symposium on Biomedical Optics, edited by Shuming Nie, Eiichi Tamiya, and Edward S. Yeung. SPIE, 2000. http://dx.doi.org/10.1117/12.383352.

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Nayyef, Hanan J., Faheema J. Abo-Alhur, Sinai W. Mohammed, Estabraq A. Taqi, and Samer S. Kahdim. "Prevalence of Entamoeba histolytica among enteric infection in Al-Furat general hospital in Baghdad/Iraq." In 3RD INTERNATIONAL SCIENTIFIC CONFERENCE OF ALKAFEEL UNIVERSITY (ISCKU 2021). AIP Publishing, 2022. http://dx.doi.org/10.1063/5.0066967.

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Varela-Rodríguez, Luis, José Antonio Velázquez-Domínguez, Verónica Ivonne Hernández-Ramírez, et al. "Pharmacological Properties of Linearolactone against the Amoebiasis Caused by Entamoeba histolytica: An In Silico Study." In ECMS 2021. MDPI, 2021. http://dx.doi.org/10.3390/ecms2021-10843.

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Pantzaris, Nikolaos, Vivek Tank, Diane O’Meara, et al. "P53 Entamoeba histolytica testing in the management of inflammatory bowel disease patients at university hospitals Leicester." In Abstracts of the BSG Annual Meeting, 20–23 June 2022. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2022. http://dx.doi.org/10.1136/gutjnl-2022-bsg.113.

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Duarte, Gabriela Reis, Erika Maria Silva Gonzaga, Evellyn Monick da Silveira Aguiar, Emily Karine Martins Mota, and Tiago Henrique Rodrigues Siebert. "PRESENÇA DE ENTEROPARASITAS EM HORTALIÇAS COMERCIALIZADAS NAS PRINCIPAIS FEIRAS LIVRES DA CIDADE DE SANTARÉM, PARÁ, BRASIL." In II Congresso Brasileiro de Parasitologia Humana On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/conbrapah/47.

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Resumen
Introdução: As hortaliças estão ocupando cada vez mais espaço nas refeições dos brasileiros. Entretanto, esses alimentos, quando contaminados pelo meio ambiente ou pelo processo de manipulação, favorecem a cadeia de transmissão de parasitoses intestinais. As verduras podem conter cistos de protozoários, ovos ou larvas de helmintos, levando a um grave problema de saúde pública no Brasil. As manifestações clínicas mais comuns dessas parasitoses são: diarreia, náuseas, dificuldade de ganhar peso, anemia, má absorção de nutrientes, entre outras. Na Amazônia, a cidade de Santarém ocupa o 3º pior lugar no ranking de saneamento básico do país. Objetivos: investigar quais os tipos mais comuns de enteroparasitas presentes na alface, couve e cheiro-verde comercializados em três feiras livres de Santarém, Pará. Além disso, identificar quais hortaliças e feiras têm maior incidência de contaminação. Material e métodos: Foram adquiridas 42 amostras de alface, couve e cheiro-verde. As coletas foram realizadas entre os meses de janeiro e fevereiro de 2022. Em cada feira, as bancas foram escolhidas aleatoriamente. As amostras foram analisadas através das técnicas de sedimentação espontânea e flutuação, com adaptações para olerícolas, totalizando 84 lâminas lidas. Resultados: Todas as variedades de hortaliças examinadas apresentaram algum grau de contaminação, sendo a alface (35%) e o cheiro-verde (35%) os mais frequentes. A feira 3, localizada na zona norte da cidade, foi a que teve taxa mais elevada de contaminação (50%). As espécies de enteroparasitas encontradas foram: Entamoeba histolytica, Entamoeba coli, Endolimax nana, Balantidium coli, Ascaris lumbricoides e Enterobius vermiculares. A espécie mais frequente foi E. histolytica, que causa a disenteria amebiana. Conclusão: É fundamental a adoção de medidas que tragam melhorias na qualidade higiênico-sanitária do armazenamento dessas hortaliças nas feiras. E faz-se necessária a conscientização da população a cerca da forma correta de higienização das verduras no momento do consumo.
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