Bibliografías temáticas / Fibroblast-pneumocyte factor

Índice

  1. Artículos de revistas
  2. Tesis

Literatura académica sobre el tema "Fibroblast-pneumocyte factor"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 1 de febrero de 2022

Crea una cita precisa en los estilos APA, MLA, Chicago, Harvard y otros

Elija tipo de fuente:

Consulte las listas temáticas de artículos, libros, tesis, actas de conferencias y otras fuentes académicas sobre el tema "Fibroblast-pneumocyte factor".

Junto a cada fuente en la lista de referencias hay un botón "Agregar a la bibliografía". Pulsa este botón, y generaremos automáticamente la referencia bibliográfica para la obra elegida en el estilo de cita que necesites: APA, MLA, Harvard, Vancouver, Chicago, etc.

También puede descargar el texto completo de la publicación académica en formato pdf y leer en línea su resumen siempre que esté disponible en los metadatos.

Artículos de revistas sobre el tema "Fibroblast-pneumocyte factor"

1

Buch, S., R. N. Han, J. Liu, A. Moore, J. D. Edelson, B. A. Freeman, M. Post, and A. K. Tanswell. "Basic fibroblast growth factor and growth factor receptor gene expression in 85% O2-exposed rat lung." American Journal of Physiology-Lung Cellular and Molecular Physiology 268, no. 3 (March 1, 1995): L455—L464. http://dx.doi.org/10.1152/ajplung.1995.268.3.l455.

Texto completo
Resumen
Lungs exposed to elevated O2 concentrations suffer an initial loss of type I pneumocytes, followed by a reparative type II pneumocyte hyperplasia. We hypothesized that type II pneumocyte hyperplasia after exposure of young adult rats to 85% O2 in vivo would be temporally related to 1) an increased concentration of intrapulmonary basic fibroblast growth factor (bFGF), a potent stimulator of type II pneumocyte DNA synthesis in vitro, and 2) an upregulation of pneumocyte receptors for bFGF (FGF-R). Increased rat lung bFGF mRNA, relative to air-exposed control animals, was observed at 4 days of ex
Los estilos APA, Harvard, Vancouver, ISO, etc.
2

King, George, Megan E. Smith, Max H. Cake, and Heber C. Nielsen. "What is the identity of fibroblast-pneumocyte factor?" Pediatric Research 80, no. 6 (August 8, 2016): 768–76. http://dx.doi.org/10.1038/pr.2016.161.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
3

Torday, John Steven. "Commentary on “What is the identity of fibroblast pneumocyte factor (FPF)?”." Pediatric Research 81, no. 2 (October 24, 2016): 391. http://dx.doi.org/10.1038/pr.2016.209.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
4

Ballard, Philip L. "Commentary on the identity of fibroblast pneumocyte factor: rat vs. human." Pediatric Research 82, no. 1 (May 31, 2017): 4–5. http://dx.doi.org/10.1038/pr.2017.85.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
5

Nielsen, Heber C., George King, and Max H. Cake. "Response to “Commentary on identity of fibroblast pneumocyte factor: rat vs. human”." Pediatric Research 82, no. 1 (May 31, 2017): 6–7. http://dx.doi.org/10.1038/pr.2017.86.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
6

Sakurai, Maromi K., Sang Lee, Danielle A. Arsenault, Vania Nose, Jay M. Wilson, John V. Heymach, and Mark Puder. "Vascular endothelial growth factor accelerates compensatory lung growth after unilateral pneumonectomy." American Journal of Physiology-Lung Cellular and Molecular Physiology 292, no. 3 (March 2007): L742—L747. http://dx.doi.org/10.1152/ajplung.00064.2006.

Texto completo
Resumen
We hypothesize that compensatory lung growth after unilateral pneumonectomy in a murine model is, in part, angiogenesis dependent and can be altered using angiogenic agents, possibly through regulation of endothelial cell proliferation and apoptosis. Left pneumonectomy was performed in mice. Mice were then treated with proangiogenic factors [vascular endothelial growth factor (VEGF); basic fibroblast growth factor (bFGF)], VEGF receptor antibodies (MF-1, DC101), and VEGF receptor small molecule chemical inhibitors. Lung volume and mass were measured. The lungs were analyzed using immunohistoch
Los estilos APA, Harvard, Vancouver, ISO, etc.
7

King, George, Jolanta E. Damas, Max H. Cake, David Berryman та Garth L. Maker. "Influence of glucocorticoids, neuregulin-1β, and sex on surfactant phospholipid secretion from type II cells". American Journal of Physiology-Lung Cellular and Molecular Physiology 306, № 3 (1 лютого 2014): L292—L298. http://dx.doi.org/10.1152/ajplung.00297.2013.

Texto completo
Resumen
Glucocorticoids induce lung fibroblasts to produce fibroblast-pneumocyte factor, a peptide that stimulates type II cells to synthesize pulmonary surfactant. This effect is known to be more apparent in cells derived from female fetuses, a characteristic that has been attributed to sex-linked differences in the fibroblasts. In the current study, it has been shown that dexamethasone enhances both β-adrenergic receptor (β-AR) activity (1.3- to 1.6-fold increase) and (−)-isoproterenol-induced secretion of surfactant (1.8- to 1.9-fold increase) in type II cells. However, fibroblast-conditioned media
Los estilos APA, Harvard, Vancouver, ISO, etc.
8

Colvin, Jennifer S., Andrew C. White, Stephen J. Pratt, and David M. Ornitz. "Lung hypoplasia and neonatal death inFgf9-null mice identify this gene as an essential regulator of lung mesenchyme." Development 128, no. 11 (June 1, 2001): 2095–106. http://dx.doi.org/10.1242/dev.128.11.2095.

Texto completo
Resumen
Mammalian lung develops as an evagination of ventral gut endoderm into the underlying mesenchyme. Iterative epithelial branching, regulated by the surrounding mesenchyme, generates an elaborate network of airways from the initial lung bud. Fibroblast growth factors (FGFs) often mediate epithelial-mesenchymal interactions and mesenchymal Fgf10 is essential for epithelial branching in the developing lung. However, no FGF has been shown to regulate lung mesenchyme. In embryonic lung, Fgf9 is detected in airway epithelium and visceral pleura at E10.5, but is restricted to the pleura by E12.5. We r
Los estilos APA, Harvard, Vancouver, ISO, etc.
9

Tanswell, A. K., R. N. N. Han, S. J. Buch, and L. J. Fraher. "Circulating Factors That Modify Lung Cell DNA Synthesis Following Exposure to Inhaled Oxidants. III. Effect of Plasma on Lung Pneumocyte and Fibroblast DNA Synthesis Following Exposure of Adult Rats to 85% Oxygen." Experimental Lung Research 17, no. 5 (January 1991): 869–86. http://dx.doi.org/10.3109/01902149109064323.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
10

Srisuma, Sorachai, Soumyaroop Bhattacharya, Feng Tu, Barry Starcher, and Thomas J. Mariani. "Fibroblast Growth Factor Receptor Deficiency Results in Abnormal Type II Pneumocyte Gene Expression and Dysregulation of Matrix Production." FASEB Journal 21, no. 5 (April 2007). http://dx.doi.org/10.1096/fasebj.21.5.a10.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.

Tesis sobre el tema "Fibroblast-pneumocyte factor"

1

Maker, Garth Lucas. "Regulation of surfactant production by fetal type II pneumocytes and characterization of fibroblast-pneumocyte factor /." Access via Murdoch University Digital Theses Project, 2007. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20080430.141113.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
2

au, G. Maker@murdoch edu, and Garth Lucas Maker. "Regulation of surfactant production by fetal type II pneumocytes and the characterization of fibroblast-pneumocyte factor." Murdoch University, 2008. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20080430.141113.

Texto completo
Resumen
The fetal lung undergoes extensive physiological and biochemical maturation prior to birth in preparation for its postnatal function as an organ for gas exchange. Pulmonary surfactant, a substance that reduces surface tension and prevents alveolar collapse, is produced by type II pneumocytes within the lung. Reduced ability to produce surfactant leads to neonatal respiratory distress syndrome. Synthesis of the phospholipid component of surfactant, phosphatidylcholine (PC), is stimulated by fibroblast-pneumocyte factor (FPF), a protein expressed by fibroblast cells within the fetal lung. Althou
Los estilos APA, Harvard, Vancouver, ISO, etc.
3

Maker, Garth Lucas. "Regulation of surfactant production by fetal type II pneumocytes and the characterization of fibroblast-pneumocyte factor." Maker, Garth Lucas (2008) Regulation of surfactant production by fetal type II pneumocytes and the characterization of fibroblast-pneumocyte factor. PhD thesis, Murdoch University, 2008. http://researchrepository.murdoch.edu.au/486/.

Texto completo
Resumen
The fetal lung undergoes extensive physiological and biochemical maturation prior to birth in preparation for its postnatal function as an organ for gas exchange. Pulmonary surfactant, a substance that reduces surface tension and prevents alveolar collapse, is produced by type II pneumocytes within the lung. Reduced ability to produce surfactant leads to neonatal respiratory distress syndrome. Synthesis of the phospholipid component of surfactant, phosphatidylcholine (PC), is stimulated by fibroblast-pneumocyte factor (FPF), a protein expressed by fibroblast cells within the fetal lung. Althou
Los estilos APA, Harvard, Vancouver, ISO, etc.
Ofrecemos descuentos en todos los planes premium para autores cuyas obras están incluidas en selecciones literarias temáticas. ¡Contáctenos para obtener un código promocional único!

Pasar a la bibliografía