Bibliografías temáticas / Gastric mucosa Effect of drugs on

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Literatura académica sobre el tema "Gastric mucosa Effect of drugs on"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 29 de enero de 2023

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Artículos de revistas sobre el tema "Gastric mucosa Effect of drugs on"

1

Lavy, Alexandra. "Corrosive Effect of Nifedipine in the Upper Gastrointestinal Tract." Diagnostic and Therapeutic Endoscopy 6, no. 1 (January 1, 1999): 39–41. http://dx.doi.org/10.1155/dte.6.39.

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Upper gastrointestinal tract mucosa is prone to injury. Drugs may disturb gastric mucosa protective mechanisms and cause damage. Injury by NSAIDs is a well described complication. Nifedipine, a widely used drug, was not described before as having a potential to damage gastrointestinal mucosa. We describe here, two patients, who developed esophageal and gastric mucosal damage, probably related to Nifedipine ingestion.
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Lu, Sheng-Yu, Song Guo, Shao-Bin Chai, Jia-Qi Yang, Yuan Yue, Hao Li, Pei-Ming Sun, et al. "Autophagy in Gastric Mucosa: The Dual Role and Potential Therapeutic Target." BioMed Research International 2021 (June 11, 2021): 1–8. http://dx.doi.org/10.1155/2021/2648065.

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The incidence of stomach diseases is very high, which has a significant impact on human health. Damaged gastric mucosa is more vulnerable to injury, leading to bleeding and perforation, which eventually aggravates the primary disease. Therefore, the protection of gastric mucosa is crucial. However, existing drugs that protect gastric mucosa can cause nonnegligible side effects, such as hepatic inflammation, nephritis, hypoacidity, impotence, osteoporotic bone fracture, and hypergastrinemia. Autophagy, as a major intracellular lysosome-dependent degradation process, plays a key role in maintain
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3

Andrews, F. J., C. Malcontenti-Wilson, and P. E. O'Brien. "Effect of nonsteroidal anti-inflammatory drugs on LFA-1 and ICAM-1 expression in gastric mucosa." American Journal of Physiology-Gastrointestinal and Liver Physiology 266, no. 4 (April 1, 1994): G657—G664. http://dx.doi.org/10.1152/ajpgi.1994.266.4.g657.

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Leukocyte adhesion to the endothelium appears to play an important role in gastric injury. This study aimed to develop immunohistochemical staining techniques to investigate the distribution and sequence of expression of both leukocyte [lymphocyte function associated antigen 1 (LFA-1)] and endothelial [intracellular adhesion molecule 1 (ICAM-1)] adhesion molecules in the mucosa after treatment with nonsteroidal anti-inflammatory drugs (NSAIDs). In control rats there were 803 +/- 72 LFA-1-stained cells/mm2 in the deep mucosa, 134 +/- 32 cells/mm2 in the superficial mucosa, and 6.4 +/- 1.2 ICAM-
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4

Fang, Yan Fei, Wen Li Xu, Lan Wang, Qing Wu Lian, Li Feng Qiu, Hui Zhou, and Shu Jie Chen. "Effect of Hydrotalcite on Indometacin-Induced Gastric Injury in Rats." BioMed Research International 2019 (April 11, 2019): 1–9. http://dx.doi.org/10.1155/2019/4605748.

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Background and Aims. Hydrotalcite plays an important role in the therapy of gastric ulcer induced by nonsteroidal anti-inflammatory drugs (NSAIDs), but little is known about the mechanism. We designed two experiments to study the preventive and curative effects of hydrotalcite on NSAIDs-related gastric injury in rats and to investigate the relationship between the protective and curative mechanism of hydrotalcite and the secretion of epidermal growth factor (EGF)/prostaglandin E2 (PGE2). Methods. Two experiments were separately designed to evaluate the preventive and curative effects of hydrot
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5

Ren, Shou-zhong, Jian-sheng Guo, Lin Sheng, Chen Jun, Shui-ping Dai, and Zhi-jian Ma. "PROTECTIVE EFFECT OF FENGLIAO-CHANGWEIKANG EXTRACTS, A TRADITIONAL CHINESE HERBAL MEDICINE FORMULA,ON MUCOSA IN RAT WITH CHRONIC GASTRITIS." African Journal of Traditional, Complementary and Alternative Medicines 13, no. 1 (December 3, 2015): 53–61. http://dx.doi.org/10.21010/ajtcam.v13i1.8.

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Background: Fengliao-Changwei-Kang(FCK), Chinese patent drug, is a famous traditional Chinese herbal medicine formula, which is effectively used to cure gastrointestinal disease including gastritis, enteritis and diarrhea in the clinic for many years. However, little research has been focused on the protective effects of FCK on the gastric mucosa of chronic gastritis (CG) model rat.
 Objective: The present study aimed to explore the effects of FCK extract on mucosa in rats with Chronic Gastritis.
 Materials and Methods: The CG rat model was induced by synthetic methods. The serum lev
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Villa, Afonso Luiz, Ceneviva Reginaldo, Fernanda Viaro, Fernando Ramalho, Antonio Dorival Campos, and Paulo Roberto B. Evora. "The cytoprotective effect of a nitric oxide donor drug on gastric mucous membrane of rats treated with ketoprofen, a non-steroidal anti-inflammatory drug." Arquivos de Gastroenterologia 43, no. 3 (September 2006): 233–37. http://dx.doi.org/10.1590/s0004-28032006000300015.

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BACKGROUND: Non-steroidal anti-inflammatory drugs are considered today a very important group of medication, with a wide variety of therapeutic use, in different areas of modern medicine. Despite their beneficial effects on the patient, these drugs show a high incidence of side effects, mainly in the gastrointestinal tract. The physiopathological mechanisms of non-steroidal anti-inflammatory drugs induced lesions and the gastric mucosa defense mechanism became an important source for medical research, especially those which try to evaluate the role of nitric oxide as a cytoprotective agent. AI
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Bakalarz, Dominik, Edyta Korbut, Zhengnan Yuan, Bingchen Yu, Dagmara Wójcik, Aleksandra Danielak, Katarzyna Magierowska, et al. "Novel Hydrogen Sulfide (H2S)-Releasing BW-HS-101 and Its Non-H2S Releasing Derivative in Modulation of Microscopic and Molecular Parameters of Gastric Mucosal Barrier." International Journal of Molecular Sciences 22, no. 10 (May 14, 2021): 5211. http://dx.doi.org/10.3390/ijms22105211.

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Hydrogen sulfide (H2S) is an endogenously produced molecule with anti-inflammatory and cytoprotective properties. We aimed to investigate for the first time if a novel, esterase-sensitive H2S-prodrug, BW-HS-101 with the ability to release H2S in a controllable manner, prevents gastric mucosa against acetylsalicylic acid-induced gastropathy on microscopic and molecular levels. Wistar rats were pretreated intragastrically with vehicle, BW-HS-101 (0.5–50 μmol/kg) or its analogue without the ability to release H2S, BW-iHS-101 prior to ASA administration (125 mg/kg, intragastrically). BW-HS-101 was
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Lyra, Anna, Markku Saarinen, Heli Putaala, Kaisa Olli, Sampo J. Lahtinen, Arthur C. Ouwehand, Mari Madetoja, and Kirsti Tiihonen. "Bifidobacterium animalisssp.lactis420 Protects against Indomethacin-Induced Gastric Permeability in Rats." Gastroenterology Research and Practice 2012 (2012): 1–9. http://dx.doi.org/10.1155/2012/615051.

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Gastrointestinal (GI) adverse effects such as erosion and increased permeability are common during the use of nonsteroidal anti-inflammatory drugs (NSAIDs). Our objective was to assess whetherBifidobacterium animalisssp.lactis420 protects against NSAID-induced GI side effects in a rat model. A total of 120 male Wistar rats were allocated into groups designated as control, NSAID, and probiotic. The NSAID and probiotic groups were challenged with indomethacin (10 mg/kg−1; single dose). The probiotic group was also supplemented daily with 1010 CFU ofB. lactis420 for seven days prior to the indome
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9

Bastaki, Salim MA, and John L. Wallace. "Pathogenesis of Nonsteroidal Anti-Inflammatory Drug Gastropathy: Clues to Preventative Therapy." Canadian Journal of Gastroenterology 13, no. 2 (1999): 123–27. http://dx.doi.org/10.1155/1999/738968.

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Gastric ulceration and bleeding are major impediments to the chronic use of nonsteroidal anti-inflammatory drugs (NSAIDs). The development of effective therapies for prevention of these adverse effects requires better understanding of their pathogenesis. Several features of NSAIDs contribute to the development of damage in the stomach, including the topical irritant effects of these drugs on the epithelium, impairment of the barrier properties of the mucosa, suppression of gastric prostaglandin synthesis, reduction of gastric mucosal blood flow and interference with the repair of superficial i
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Moghal, Naheed, and N. A. Jafarey. "Effects of Nonsteroidal Antiinflammatory Drugs on Gastric and Duodenal Mucosa." Gastroenterology 96, no. 2 (February 1989): 553–54. http://dx.doi.org/10.1016/0016-5085(89)91605-3.

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Tesis sobre el tema "Gastric mucosa Effect of drugs on"

1

黃尚行 and Sheung-hang Wong. "The gastric effects of ethanol and their modulation by drugs in rats." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1989. http://hub.hku.hk/bib/B31232048.

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Wong, Sheung-hang. "The gastric effects of ethanol and their modulation by drugs in rats /." [Hong Kong] : University of Hong Kong, 1989. http://sunzi.lib.hku.hk/hkuto/record.jsp?B12607757.

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Sennello, Kathleen Ann. "Comparison of the Effects of Deracoxib, Buffered Aspirin, and Placebo on the Gastric Mucosa of Healthy Dogs." Thesis, Virginia Tech, 2005. http://hdl.handle.net/10919/31788.

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This study tested the hypothesis that administration of deracoxib, a cyclooxygenase-2 specific (COX-2) inhibitor, would result in lower gastric lesion scores than administration of buffered aspirin and gastric lesion scores similar to placebo when administered to healthy dogs for 28 days. Twenty-four, healthy, random source dogs were divided into three groups. Group I received buffered aspirin, 23.6 mg/kg PO q 8h, group II received deracoxib, 1.6 mg/kg PO q 24h and placebo twice daily PO q 8h after deracoxib administration, and group III received placebo PO q 8h. Gastroscopy was performed on d
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4

Wong, Wing-hong. "The anti-ulcer mechanisms of Cortex moutan against stress-induced gastric mucosal damage in rats /." Hong Kong : University of Hong Kong, 1998. http://sunzi.lib.hku.hk/hkuto/record.jsp?B20265517.

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黃穎康 and Wing-hong Wong. "The anti-ulcer mechanisms of Cortex moutan against stress-induced gastric mucosal damage in rats." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1998. http://hub.hku.hk/bib/B31221981.

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6

Stapleton, Graham Neil. "A study of the effects of sucralfate in the bile duct litigated pig peptic ulcer model with particular reference to the effects on the physico-chemical properties of gastric mucus and including comparisons with famotidine and misoprostol." Master's thesis, University of Cape Town, 1992. http://hdl.handle.net/11427/25727.

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Sucralfate is a drug that effectively heals duodenal, gastric and oesophageal ulcers. It is not absorbed systemically and it has been shown to act locally by coating the ulcer base. However when it was also shown to prevent stress ulcers and ethanolinduced gastric mucosa! lesions, it seemed likely that it acted in some way to improve the effectiveness of the gastric mucosa! barrier. Some investigators suggested that it did so by stimulating local prostaglandin release. The Slomiany group, on the basis of in vitro work on the effects of Sucralfate on pig gastric mucus, claimed that Sucralfate a
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7

Jeffrey, Stuart C. "Epidermal growth factor receptor in equine gastric stratified squamous mucosa : effect of progressive ulceration on receptor density /." Thesis, This resource online, 1996. http://scholar.lib.vt.edu/theses/available/etd-09182008-063547/.

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Lee, Adam Michael. "Impact of genetic and epigenetic variability in response to two test drugs 5-Flurouracil and Lansoprazole." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2009. https://www.mhsl.uab.edu/dt/2009p/lee.pdf.

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Dodridge, M. E. (Miles Edward). "The effects of variable dose methotrexate infusion in the laboratory rat." 1987. http://web4.library.adelaide.edu.au/theses/09DM/09dmd641.pdf.

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10

Hsin-min and 周欣敏. "The preventive effect of Mulberry anthocyanin extracts in diclofenac-induced rat gastric mucosa damage." Thesis, 2009. http://ndltd.ncl.edu.tw/handle/76290790734319523055.

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碩士<br>中山醫學大學<br>生化暨生物科技研究所<br>97<br>Diclofenac is a NSAID drug which causes gastric mucosal lesion complication. Food bioactive compounds were reported could exert beneficial effects in the gastrointestinal tract. In this study, we evaluated whether Mulberry anthocyanin extracts (MAE) reduced Diclofenac-induced injury in the rat gastric mucosa. Rats were induced for gastric mucosal lesion with Diclofenac (DIC, 100 mg/kg, orally) after pretreated with MAE in various dosages (100mg/kg, 200mg/kg, 400mg/kg). A proton pump inhibitor, Esomeprazole, was used as a protection control. The gastroprotect
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Libros sobre el tema "Gastric mucosa Effect of drugs on"

1

Rodolfo, Cheli, ed. Gastric protection. New York: Raven Press, 1988.

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Potent acid suppression-when is it appropriate? Royal Society of Medicine, 1990.

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3

Mozsik, Gyula, Andras Debreceni, and Kazuichi Okazaki. The Capsaicin- And Helicobacter Strains-Induced Cellular Mechanisms of the Gastric Mucosa in Animals and Humans. Akademiai Kiado, 2001.

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4

Daniel, Hollander, and Tytgat G. N. J, eds. Sucralfate: From basic science to the bedside. New York: Plenum Medical Book Co., 1995.

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(Editor), Daniel Hollander, and G. N. Tytgat (Editor), eds. Sucralfate: From Basic Science to Bedside. Springer, 1995.

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6

(Editor), C. H. Cho, and J. Y. Wang (Editor), eds. Gastrointestinal Mucosal Repair and Experimental Therapeutics (Frontiers of Gastrointestinal Research). S. Karger Publishers (USA), 2002.

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7

R, Cheli, ed. Gastric secretion: A physiological and pharmacological approach. Verona: Cortina International, 1986.

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8

Wójcik-Gładysz, Anna. Ghrelin – hormone with many faces. Central regulation and therapy. The Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, 2020. http://dx.doi.org/10.22358/mono_awg_2020.

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Discovered in 1999, ghrelin, is one of the peptides co-creating the hypothalamicgastrointestinal axis, otherwise known as the brain-gut axis. Ghrelin participates in many physiological processes and spectrum of its activity is still being discovered. This 28 amino acid peptide ‒ a product of the ghrl gene, was found in all vertebrates and is synthesized and secreted mainly from enteroendocrine X/A cells located in the gastric mucosa of the stomach. Expression of the ghrelin receptor has been found in many nuclei of the hypothalamus involved in appetite regulation. Therefore it’s presumed that
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Capítulos de libros sobre el tema "Gastric mucosa Effect of drugs on"

1

Collins, P. O., I. A. Tavares, and A. Bennett. "Inhibition of prostanoid synthesis by anti-inflammatory drugs in human gastric mucosa." In Side-Effects of Anti-Inflammatory Drugs, 97–100. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-010-9775-8_8.

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Halter, F., A. Baumgartner, L. Varga, and H. R. Koelz. "Both E Prostaglandins and prolonged indomethacin treatment exert trophic effects on the gastric mucosa." In Side-Effects of Anti-Inflammatory Drugs, 133–42. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-010-9775-8_12.

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Rowe, P. H. "Prostaglandins prevent red streaks in rat gastric mucosa caused by intravenous aspirin but not by salicylate." In Side-Effects of Anti-Inflammatory Drugs, 165–66. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-010-9775-8_18.

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Leyck, S., A. M. Huther, and M. J. Parnham. "Polyene phosphatidylcholine: an inhibitor of NSAID gastric toxicity which increases impaired mucosal PGE2 synthesis." In Side-Effects of Anti-Inflammatory Drugs, 163–64. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-010-9775-8_17.

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Ezer, E. "Evidence for sulphydryl-sensitive process in the mechanisms of acute gastric mucosal injury and defence." In Side-Effects of Anti-Inflammatory Drugs, 113–21. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-010-9775-8_10.

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Konturek, S. J., J. Oleksy, and E. Zielonka. "Comparison of gastric mucosal damage and prostaglandin formation in arthritis patients treated with carprofen and ibuprofen." In Side-Effects of Anti-Inflammatory Drugs, 89–96. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-010-9775-8_7.

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Warrington, S., N. Debbas, M. Farthing, M. Horton, A. Thillainayagam, and A. Umile. "Comparison of effects on gastrointestinal blood loss and gastric mucosal appearance of piroxicam-β-cyclodextrin, piroxicam and placebo." In Side-Effects of Anti-Inflammatory Drugs 3, 89–96. Dordrecht: Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-011-2982-4_13.

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Gyires, K., C. Blandizzi, and M. Del Tacca. "Analysis of the Inhibiting Activity of Presynaptic α2-Adrenoceptors against NSAID-induced Gastric Mucosal Lesions in the Rat." In Side Effects of Anti-Inflammatory Drugs IV, 287–94. Dordrecht: Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5394-2_30.

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Szabo, S., E. Gyomber, R. E. Morales, L. Nagy, and P. Vattay. "Novel strategies of gastric and duodenal mucosal protection against NSAID injury: role of protease inhibitors, muscle relaxants and growth factors." In Side-Effects of Anti-Inflammatory Drugs 3, 115–25. Dordrecht: Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-011-2982-4_15.

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Kromin, A. A. "Lesions of Gastric Mucosa under the Effect of Acute Emotional Stress." In Perspectives on Research in Emotional Stress, 191–204. London: Routledge, 2022. http://dx.doi.org/10.4324/9781315075488-15.

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Actas de conferencias sobre el tema "Gastric mucosa Effect of drugs on"

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M.Hussein, Ali, Nadir Nanakali, and Mohammed M.Hussein. "EFFECT OF HYPERICUM PERFORATUM ON GASTRIC ULCER IN RAT." In 4th International Conference on Biological & Health Sciences (CIC-BIOHS’2022). Cihan University, 2022. http://dx.doi.org/10.24086/biohs2022/paper.742.

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Gastric ulcer is a chronic condition that occurs when the mucosa of the stomach is broken. There is a physiological equilibrium between aggressive factors and mucosal defense. This study aimed to determine the prevention level and efficiency of herbal medicinal plants (Hypericum perforatum) and compared with the omeprazole drug.Many groups were prepared from Albino male rats, first control group (inoculate with H. pylori and fed with standard pellet), Second group, rats inoculated by H. pylori and prevented with aqueous extract H. perforatum in two dosages (250mg/kg, 500mg/kg), Third group ino
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Zhang, Lian, Sungkyoung Kim, Wenping Ding, Leizhen Zheng, Li Zhang, Yingying Tong, and Siyu Chen. "Abstract 5525: Antitumor effect of Realgar in combination with chemotherapy drugs on human gastric cancer cells." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-5525.

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Muguruma, Kazuya, Masakazu Yashiro, Hiroaki Tanaka, Kenjirou Kimura, Hisashi Nagahara, Ryousuke Amano, Eiji Noda, et al. "Abstract 100: The effect of synergic anti-proliferation of DNA methyltransferase inhibitor binding to anti-cancer drugs in gastric cancer." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-100.

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Informes sobre el tema "Gastric mucosa Effect of drugs on"

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Xie, Yunhui, and Peng Pang. A Systematic Review and Network Meta-Analysis: Effect of of GLP-1 drugs on weight loss in obese people. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2022. http://dx.doi.org/10.37766/inplasy2022.6.0074.

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Review question / Objective: 1、Whether GLP-1 drugs have weight loss effect on obese people ? 2、Which GLP-1 drugs are most effective in weight loss among obese people ? Condition being studied: Obesity is an important public health issue that has been on the rise over the last decades. It calls for effective prevention and treatment. Bariatric surgery is the most effective medical therapy for weight loss in morbid obesity, but we are in need for less aggressive treatments. Glucagon-like-peptide-1 receptor agonists are a group of incretin-based drugs that have proven to be productive for obesity
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