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Artículos de revistas sobre el tema "Graves' disease"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 26 de julio de 2025

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1

Nicolle, Michael W. "Pseudo-Myasthenia Gravis and Thymic Hyperplasia in Graves’ Disease." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 26, no. 3 (1999): 201–3. http://dx.doi.org/10.1017/s0317167100000251.

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Background:Diagnostic confusion between thyroid disease and myasthenia gravis (MG) can arise because the two may have similar clinical features, and also because of the more frequent coexistence of these autoimmune disorders in the same individual. In MG, autoantibodies directed against the acetylcholine receptor result in muscle weakness. Thymic pathology is well recognized in MG, with thymic hyperplasia frequent in early onset MG and thymoma more common in later onset MG. In Graves’ disease, autoantibodies against thyroid antigens result in hyperthyroidism. A seldom-recognized feature of Gra

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2

Udorji, Ogochukwu, Subbulaxmi Trikudanathan, and Anthony Desantis. "PSUN39 A rare presentation of autoimmune thyroid diseases and myasthenia gravis: A clinical case report." Journal of the Endocrine Society 6, Supplement_1 (2022): A851. http://dx.doi.org/10.1210/jendso/bvac150.1760.

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Abstract We report a case of a 37-years-old female that presented with a three-week history of diplopia, ptosis, muscle weakness, difficulty chewing, tachycardia, and fatigue; these clinical symptoms were concerning for acute myasthenia gravis crisis. The diagnosis of Myasthenia gravis was further confirmed by a positive immunology workup. Chest CT imaging during her assessment showed anterior mediastinal mass/enlarged thymus indicative of thymic hyperplasia. Medical therapy with intravenous immune globulin, pyridostigmine and further treatment with high-dose steroid. She had a positive respon

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3

H, Joyce Soo Synn, Sau Wei W, and Loo Ling W. "NOT QUITE HICKAM'S DICTUM." Journal of the ASEAN Federation of Endocrine Societies 34 (July 17, 2019): 59. https://doi.org/10.15605/jafes.034.s102.

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INTRODUCTIONMyasthenia gravis is a rare autoimmune neuromuscular disease. Rarer still is the combination of myasthenia gravis and Graves’ disease, especially so in the paediatrics population, bringing to mind Hickam’s Dictum. However, considering that both diseases are antibody-mediated, it should not be surprising to find them occurring simultaneously. Of more concern, though, is the effect these diseases have on each other’s natural progression, and the challenges in managing them. Indeed, it has been observed that concurrent myasthenia gravis worsens hyperthyroidism, and improved thyroid fu

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4

Bleeker, G. M. "Graves' disease." Orbit 6, no. 3 (1987): 151–52. http://dx.doi.org/10.3109/01676838709036094.

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5

Smith, Terry J., and Laszlo Hegedüs. "Graves’ Disease." New England Journal of Medicine 375, no. 16 (2016): 1552–65. http://dx.doi.org/10.1056/nejmra1510030.

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6

McFarland, Kay F., and Gabe Saleeby. "Graves' disease." Postgraduate Medicine 83, no. 4 (1988): 275–82. http://dx.doi.org/10.1080/00325481.1988.11700201.

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7

Weetman, Anthony P. "Graves' Disease." New England Journal of Medicine 343, no. 17 (2000): 1236–48. http://dx.doi.org/10.1056/nejm200010263431707.

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8

Brent, Gregory A. "Graves' Disease." New England Journal of Medicine 358, no. 24 (2008): 2594–605. http://dx.doi.org/10.1056/nejmcp0801880.

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9

Mellow, Prettysun Ang. "GRAVES DISEASE." Jurnal Widya Medika 9, no. 1 (2023): 66–78. http://dx.doi.org/10.33508/jwm.v9i1.4510.

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Graves' disease is one of the most common autoimmune diseases. The disease is named after the scientist Robert James Graves, who first described it in the 19th century as a syndrome with an enlarged and overactive thyroid gland (hyperthyroidism due to circulating autoantibodies), rapid heart rate, and eye abnormalities. graves’ disease is the most common cause of hyperthyroidism and affects more women than men. Graves’ disease can appear at any age, but it most commonly appears for the first time between the ages of 20 and 40. Factor is a predisposition that is more dominant than environmental

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10

VENTO, JOHN A., JAMES D. SLAVIN, GEORGE E. REARDON, and RICHARD P. SPENCER. "Graves?? Disease." Clinical Nuclear Medicine 11, no. 6 (1986): 404–6. http://dx.doi.org/10.1097/00003072-198606000-00008.

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11

McKenna, T. Joseph. "Graves' disease." Lancet 357, no. 9270 (2001): 1793–96. http://dx.doi.org/10.1016/s0140-6736(00)04906-0.

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12

Delgado Hurtado, Juan Jose, and Marcela Pineda. "Graves' Disease." New England Journal of Medicine 364, no. 20 (2011): 1955. http://dx.doi.org/10.1056/nejmicm1012081.

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13

Bahn, Rebecca, Elliott Levy, and Leonard Wartofsky. "Graves’ Disease." Journal of Clinical Endocrinology & Metabolism 92, no. 11 (2007): E1. http://dx.doi.org/10.1210/jcem.92.11.9993.

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14

Bahn, Rebecca, Elliott Levy, and Leonard Wartofsky. "Graves’ Disease." Journal of Clinical Endocrinology & Metabolism 92, no. 11 (2007): 0. http://dx.doi.org/10.1210/jcem.92.11.9994.

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15

McCann, John. "Graves' Disease,." Survey of Ophthalmology 46, no. 3 (2001): 298–99. http://dx.doi.org/10.1016/s0039-6257(01)00277-6.

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16

Cheetham, Tim, and Rachel Boal. "Graves’ disease." Paediatrics and Child Health 29, no. 7 (2019): 316–20. http://dx.doi.org/10.1016/j.paed.2019.04.006.

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17

Weetman, A. P. "Graves' disease." BMJ 296, no. 6637 (1988): 1670. http://dx.doi.org/10.1136/bmj.296.6637.1670-b.

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18

Kennedy, R. L., and T. H. Jones. "Graves' disease." BMJ 296, no. 6637 (1988): 1670–71. http://dx.doi.org/10.1136/bmj.296.6637.1670-c.

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19

Ferrer, E., and M. A. Moral. "Graves' disease." Drugs of the Future 32, no. 6 (2007): 555. http://dx.doi.org/10.1358/dof.2007.032.06.1101018.

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20

Zhamaldinov, K., M. Ismailova, D. Makataeva, et al. "GRAVES DISEASE." BULLETIN OF SURGERY IN KAZAKHSTAN, no. 4 (December 30, 2022): 47–51. http://dx.doi.org/10.35805/bsk2022iv047.

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Hyperthyroidism (Graves’ disease (GD)), is a relatively rare disease in adults and children. Treatment options for adults and children are antithyroid drugs (ATD), radioactive iodine (RAI), or thyroidectomy, but the risks as well as benefits of each are different.

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21

Nordyke, Robert A. "Graves' Disease." Archives of Internal Medicine 148, no. 3 (1988): 626. http://dx.doi.org/10.1001/archinte.1988.00380030132023.

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22

W, Shiau Hooi, and Song Hai L. "GRAVES' DISEASE." Journal of the ASEAN Federation of Endocrine Societies 34 (July 17, 2019): 62. https://doi.org/10.15605/jafes.034.s109.

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INTRODUCTIONGraves’ disease is an uncommon disease in childhood with prevalence of 0.02%. It accounts for most of the thyrotoxicosis in paediatric population. Treatment option is limited and the reported remission rate is low. METHODOLOGYThis is a descriptive study. All patients with Graves’ disease who attended the endocrine clinic in Sabah Women and Children’s Hospital are enrolled. Data was obtained through review of their medical records. Their clinical features and treatment outcome were described. Results are expressed as numerical values (percentages) for categorical variables and media

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23

Taylor, Selwyn. "Graves of Graves' Disease, 1796—1853." Journal of the Royal College of Physicians of London 20, no. 4 (1986): 298–300. https://doi.org/10.1016/s0035-8819(25)02437-7.

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24

Mayilvaganan, Sabaretnam, VNSSVAMSD Mahalakshmi, Sapana Bothra, Aromal Chekavar, and Mallika Dhanda. "Unilateral Graves' disease." Indian Journal of Nuclear Medicine 34, no. 4 (2019): 353. http://dx.doi.org/10.4103/ijnm.ijnm_100_19.

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25

Wang, David H., Scott M. Koehler, and Cary N. Mariash. "Detecting Graves' Disease." Physician and Sportsmedicine 24, no. 12 (1996): 35–40. http://dx.doi.org/10.3810/psm.1996.12.1250.

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26

Ebner, Susana A. "Conjugal Graves Disease." Annals of Internal Medicine 116, no. 6 (1992): 479. http://dx.doi.org/10.7326/0003-4819-116-6-479.

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27

Bacci, Vincenzo. "“Fiancees'” Graves Disease." Annals of Internal Medicine 118, no. 3 (1993): 232. http://dx.doi.org/10.7326/0003-4819-118-3-199302010-00023.

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28

Wang, David H., Scott M. Koehler, and Cary N. Mariash. "Detecting Graves' Disease." Physician and Sportsmedicine 24, no. 12 (1996): 35–40. http://dx.doi.org/10.1080/00913847.1996.11948058.

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29

Castiglia, Patricia T. "Hyperthyroidism (Graves' disease)." Journal of Pediatric Health Care 11, no. 5 (1997): 227–29. http://dx.doi.org/10.1016/s0891-5245(97)90106-1.

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30

Okawa, Erin R., Frederick D. Grant, and Jessica R. Smith. "Pediatric Graves’ disease." Current Opinion in Pediatrics 27, no. 4 (2015): 442–47. http://dx.doi.org/10.1097/mop.0000000000000241.

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31

Tunbridge, W. M. G. "Atypical Graves' disease." Clinical Endocrinology 37, no. 5 (1992): 469. http://dx.doi.org/10.1111/j.1365-2265.1992.tb02361.x.

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32

Shields, C. L., L. B. Nelson, G. C. Carpenter, and J. A. Shields. "Neonatal Graves' disease." British Journal of Ophthalmology 72, no. 6 (1988): 424–27. http://dx.doi.org/10.1136/bjo.72.6.424.

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33

Rayner, P. H. W. "NEONATAL GRAVES‘ DISEASE." Developmental Medicine & Child Neurology 18, no. 5 (2008): 666–68. http://dx.doi.org/10.1111/j.1469-8749.1976.tb04214.x.

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34

Ramachandran, Puvanesvaran, Arini Nuran Idris, Tee Pian Pian, et al. "NEONATAL GRAVES’ DISEASE." Journal of the ASEAN Federation of Endocrine Societies 37 (July 15, 2022): 56–57. https://doi.org/10.15605/jafes.037.s2.82.

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INTRODUCTION Neonatal Graves’ disease is a rare disorder seen in 1 in 25000 births, caused by transplacental passage of TSH receptor antibody. Despite serious potential multisystem complications, it can be transient. METHODOLOGY We describe a newborn born to a mother with undiagnosed Graves' disease. RESULTS A male neonate was born prematurely at 31-weeks gestation via emergency lower segment caesarean for fetal distress with a birth weight of 1.7 kg. He was admitted to the neonatal intensive care unit due to respiratory distress requiring non-invasive ventilation. Newborn examination revealed

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35

Banga, J., S. Moshkelgosha, U. Berchner-Pfannschmidt, and A. Eckstein. "Modeling Graves’ Orbitopathy in Experimental Graves’ Disease." Hormone and Metabolic Research 47, no. 10 (2015): 797–803. http://dx.doi.org/10.1055/s-0035-1555956.

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36

Ellis, Harold. "Robert Graves: Graves' Disease of the Thyroid." Journal of Perioperative Practice 22, no. 5 (2012): 176. http://dx.doi.org/10.1177/175045891202200507.

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37

Banga, J., S. Moshkelgosha, U. Berchner-Pfannschmidt, and A. Eckstein. "Modeling Graves’ Orbitopathy in Experimental Graves’ Disease." Hormone and Metabolic Research 47, no. 10 (2015): e4-e4. http://dx.doi.org/10.1055/s-0035-1564108.

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38

Troshina, E. A., A. A. Larina, M. S. Sheremeta, and N. M. Malisheva. "The prevalence of newly diagnosed autoimmune diseases among patients with Graves’ disease and autoimmune polyglandular syndrome of adults." Terapevticheskii arkhiv 92, no. 10 (2020): 9–14. http://dx.doi.org/10.26442/00403660.2020.10.000737.

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Gravess disease is a common part of Autoimmune polyglandular syndrome (APS) and among thyroid autoimmune disorders is usually preceded the onset of the syndrome. Aim.The aim of this study was to determine the frequency of occurrence of APS type 2, 3 among patients with Graves disease. Materials and methods.Sera of 94 patients with Gravess disease, 116 patients with APS 24 types and 80 healthy subjects were screened for 21-OH Ab, insulin-Ab (IAA), Islet Cell-Ab (ICA), glutamic acid decarboxylase-Ab (GADA), protein tyrosine phosphatase-Ab (IA2), Zinc Transporter 8-Ab (ZnT8), Anti-gli

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39

Wagih, Sherin. "Graves' Disease Affecting one Thyroid lobe (Unilateral Graves' Disease): Case Report." Egyptian Journal Nuclear Medicine 3, no. 3 (2010): 62–65. http://dx.doi.org/10.21608/egyjnm.2010.5501.

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40

Lanzolla, G., C. Marcocci, and M. Marinò. "Graves’ disease and Graves’ orbitopathy following COVID-19." Journal of Endocrinological Investigation 44, no. 9 (2021): 2011–12. http://dx.doi.org/10.1007/s40618-021-01576-7.

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41

Karoutsou, E., and A. Polymeris. "Pathogenesis of Graves’ disease focusing on Graves’ ophthalmopathy." Endocrine Regulations 45, no. 04 (2011): 209–20. http://dx.doi.org/10.4149/endo_2011_04_209.

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42

Nagayama, Yuji, Mami Nakahara, and Norio Abiru. "Animal models of Graves’ disease and Graves’ orbitopathy." Current Opinion in Endocrinology & Diabetes and Obesity 22, no. 5 (2015): 381–86. http://dx.doi.org/10.1097/med.0000000000000186.

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43

Lanzolla, Giulia, Claudio Marcocci, and Michele Marinò. "Oxidative Stress in Graves Disease and Graves Orbitopathy." European Thyroid Journal 9, Suppl. 1 (2020): 40–50. http://dx.doi.org/10.1159/000509615.

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Oxidative stress is involved in the pathogenesis of Graves hyperthyroidism (GH) and Graves orbitopathy (GO) and an antioxidant approach has been proposed for both. In GH, a disbalance of the cell redox state is associated with thyroid hyperfunction and antithyroid medications may reduce oxidative stress. Tissue hypoxia participates in the pathogenesis of GO, and oxygen free radicals are involved in the typical changes of orbital tissues as reported by in vitro and clinical studies. Antioxidant agents, especially selenium, have been proposed as a therapeutic option for GH and GO. A clinical stu

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44

Mutiara, Ade, Dewi S. Soemarko, and Indah S. Widyahening. "Occupational Stress and its Relation to Grave’s Disease." Indonesian Journal of Community and Occupational Medicine 1, no. 2 (2021): 72–7. http://dx.doi.org/10.53773/ijcom.v1i2.14.72-7.

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Background: Graves’ disease is an autoimmune thyroid gland disease that causes increased activity of the thyroid hormones. But, the mechanism by which the autoantibodies are generated is still unclear. Some theory suggests that daily life stress may be a risk factor that triggers Graves’ disease. The aim of this report is to enrol a systematically searching in order to get an answer about the risk factor of the Grave’s disease and the prevention measure.Methods: In this case report, a 47-year-old career woman with prominent occupational stress was diagnosed with Graves’ disease. Literature sea

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45

Galofré, Juan C. "Microchimerism in Graves' Disease." Journal of Thyroid Research 2012 (2012): 1–7. http://dx.doi.org/10.1155/2012/724382.

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Microchimerism is the presence of cells from one individual in another genetically distinct individual. Pregnancy is the main cause of natural microchimerism through transplacental bidirectional cell trafficking between mother and fetus. The consequences of pregnancy-related microchimerism are under active investigation. However, many authors have suggested a close relationship linking fetal microchimerism and the development of autoimmune diseases. It has been more than ten years now since the demonstration of the presence of a significant high number of fetal microchimeric cells residing in

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46

Mikhail, NASSER, and Soma Wali. "Graves’ disease Induced by Sars-Cov-2 Vaccination." Journal of Clinical Research and Reports 11, no. 2 (2022): 01–05. http://dx.doi.org/10.31579/2690-1919/247.

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Background: Several cases of Graves’ disease were recently reported in individuals vaccinated against severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). Objective: To determine characteristics and patterns of Graves’ disease occurring following SARS-CoBV-2 vaccination. Methods: PubMed search up to March 31st, 2022. Search terms are Graves’ disease, SARS-Cov-2, vaccine, COVID-19. Case reports, case series, review articles and pertinent in vitro studies are reviewed. Results: Review of literature revealed 28 cases (19 women) of new onset and 5 cases (4 women) of relapses of Graves’ di

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47

Bury, Anna, and Maria Klatka. "Graves` disease and myasthenia gravis in a teenager – diagnostic problems." Pediatric Endocrinology 14, no. 2 (2015): 89–94. http://dx.doi.org/10.18544/ep-01.14.02.1560.

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48

Beliard, Kara A., Srinidhi Shyamkumarb, Mabel Yau, Cassie Mintz, and Robert Rapaport. "Graves Disease in Infancy." Journal of the Endocrine Society 5, Supplement_1 (2021): A929—A930. http://dx.doi.org/10.1210/jendso/bvab048.1899.

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Abstract Background: Graves disease (GD) is the most common cause of hyperthyroidism worldwide. The usual age of presentation is between 20-30 years, and it is more common in females. Transient hyperthyroidism does occur in infants born to mothers with GD, however, the novo GD in infants is extremely rare. We are aware of only four cases of GD in children under the age of 2 years old previously reported in the literature, with the youngest being of 18 months. Although rare, the complications can be devastating, so identifying and treating GD in infants is vital. We describe an infant who prese

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49

Yamada, T. "Aging and Graves' disease." Nippon Ronen Igakkai Zasshi. Japanese Journal of Geriatrics 28, no. 2 (1991): 129–34. http://dx.doi.org/10.3143/geriatrics.28.129.

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50

Felz, Michael W., and Peter P. Stein. "Tremor in Graves' disease." Postgraduate Medicine 107, no. 1 (2000): 184. http://dx.doi.org/10.3810/pgm.2000.01.1878.

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