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Tesis sobre el tema "H. pylori"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 25 de julio de 2025

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1

Warren, John Robin. "The discovery and pathology of H pylori /." Title page and contents only, 1999. http://web4.library.adelaide.edu.au/theses/09MD/09mdw289.pdf.

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2

Сорокман, Т. В. "Кіотський глобальний консенсус щодо H. pylori-асоційованого гастриту". Thesis, БДМУ, 2017. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/17155.

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3

Leslie, Kevin Alexander. "Investigation of Prophage in Clinical Isolates of H pylori." W&M ScholarWorks, 2013. https://scholarworks.wm.edu/etd/1539626941.

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4

Arebi, Naila. "Identification of novel factors regulating mucosal somatostatin in H. pylori gastritis." Thesis, Imperial College London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.289867.

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5

Zhou, Yong. "An antimicrobial agent from celery seed active against H. pylori." Thesis, Sheffield Hallam University, 2008. http://shura.shu.ac.uk/20709/.

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As well as peptic ulcers, Helicobacter pylori is associated with the development of gastritis, gastric adenocarcinoma and lymphoma, and has been classified as a class I carcinogen in humans (International Agency for Research on Cancer Working Group, 1994). Although the bacteria can be eradicated in up to 90% of patients, side effects, poor compliance and the resistance of the bacteria to antibiotics are common causes of frequent treatment failure. Celery seed extracts (CSE) from a unique source in India has been used as herbal medicine since antiquity and found to have anti-inflammatory and ga
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6

Iwano, Masahiro. "Characteristics of gastric MALT lymphoma resistant to eradication of H. pylori." Kyoto University, 2008. http://hdl.handle.net/2433/124330.

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7

Jimenez-Guerra, Francisco. "Prevalence of, and risk factors for, Helicobacter pylori infection and its effect on growth of children in Mexico." Thesis, London School of Hygiene and Tropical Medicine (University of London), 1999. http://researchonline.lshtm.ac.uk/4649359/.

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Helicobacter pylori (H. pylori) infection causes achlorydria, depressed gastric acid barrier, impaired immune response and is suspected in bacterial overgrowth and diarrhoea. These features of the infection are known to cause significant malabsorption of nutrients and impairment of linear growth in children. The prevalence of H. pylori infection in children is known to be much higher in developing countries, especially among the lower socio-economic groups. The true prevalence of infection in urban children in Mexico and its impact on their growth are largely unknown. This study examined the p
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8

Vallström, Anna. "Helicobacter pylori : molecular mechanisms for variable adherence properties." Doctoral thesis, Umeå universitet, Institutionen för medicinsk kemi och biofysik, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-25931.

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More than half of all people worldwide are infected with H. pylori. The infection always cause a gastric inflammation that may develop into peptic ulcer disease or gastric cancer. Attachment proteins, adhesins, mediate specific adherence of H. pylori to receptor structures on the human gastric mucosa. The best-characterized H. pylori adhesin-receptor interactions are the BabA adhesin and the binding to the fucosylated blood group antigens ABO/Lewis b (Leb) and the SabA adhesin and its binding to the inflammation associated sialyl-Lewis x antigen. During H. pylori infection the availability of
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9

de, Albuquerque Mello Luciano. "Análise histomorfométrica de lesões gástricas relacionadas à infecção pelo Hlicobacter pylori." Universidade Federal de Pernambuco, 2007. https://repositorio.ufpe.br/handle/123456789/9072.

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Made available in DSpace on 2014-06-12T23:04:25Z (GMT). No. of bitstreams: 2 arquivo8874_1.pdf: 1552701 bytes, checksum: ca1f64cd2a9ab7f1dd8b3732c2cc688d (MD5) license.txt: 1748 bytes, checksum: 8a4605be74aa9ea9d79846c1fba20a33 (MD5) Previous issue date: 2007<br>O bacilo Helicobacter pylori é atualmente considerado como o agente etiológico mais importante das gastrites em seres humanos e como um fator de risco para o surgimento das doenças ulcerativas do carcinoma gástrico. Estima-se que 50% da população mundial está infectada por esta bactéria. Atualmente, apenas estudos subjetivos e qualit
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10

Benigno, Tiago Gomes da Silva. "Análise da resposta humoral contra H. pylori em pacientes portadores e não portadores de câncer gástrico." reponame:Repositório Institucional da UFC, 2013. http://www.repositorio.ufc.br/handle/riufc/10642.

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BENIGNO, Tiago Gomes da Silva. Análise da resposta humoral contra H. pylori em pacientes portadores e não portadores de câncer gástrico. 2013. 71 f. Dissertação (Mestrado em Cirurgia) - Universidade Federal do Ceará. Faculdade de Medicina, Fortaleza, 2013.<br>Submitted by denise santos (denise.santos@ufc.br) on 2015-02-19T11:35:41Z No. of bitstreams: 1 2013_dis_tgsbenigno.pdf: 896697 bytes, checksum: daa07ef38bffc56879c4e5d12ca866fd (MD5)<br>Approved for entry into archive by denise santos(denise.santos@ufc.br) on 2015-02-19T11:36:17Z (GMT) No. of bitstreams: 1 2013_dis_tgsbenigno.pdf: 896697
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11

Qi, Shuang, and 亓爽. "UreE-Hpn/Hpnl interaction in H. pylori, and the role of cysteines in Hpn." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45962753.

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12

Cavalcante, Meyssa Quezado de Figueiredo. "Genótipos cagA, vacA e alelos de H. pylori em pacientes portadores de gastrite, úlcera péptica e câncer gástrico." reponame:Repositório Institucional da UFC, 2010. http://www.repositorio.ufc.br/handle/riufc/10647.

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CAVALCANTE, Meyssa Quezado de Figueiredo. Genótipos cagA, vacA e alelos de H. pylori em pacientes portadores de gastrite, úlcera péptica e câncer gástrico. 2010. 87 f. Dissertação (Mestrado em Cirurgia) - Universidade Federal do Ceará, Faculdade de Medicina, Fortaleza, 2010.<br>Submitted by denise santos (denise.santos@ufc.br) on 2015-02-19T11:57:02Z No. of bitstreams: 1 2010_dis_mqfcavalcante.pdf: 2039790 bytes, checksum: 22b9b8ce8cedcbdd53a754d52a85c7ec (MD5)<br>Approved for entry into archive by denise santos(denise.santos@ufc.br) on 2015-02-19T11:58:29Z (GMT) No. of bitstreams: 1 2010_dis
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Gonçalves, Maria Helane Rocha Batista. "Genótipos das cepas de H. pylori vacA e Alelos cagA e sítios de fosfolização EPIYA em uma comunidade urbana de Fortaleza." reponame:Repositório Institucional da UFC, 2010. http://www.repositorio.ufc.br/handle/riufc/7616.

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GONÇALVES, Maria Helane Rocha Batista. Genótipos das cepas de H. pylori VacA e alelos, cagA e sítios de fosforilação EPIYA em uma comunidade urbana de Fortaleza utilizando método não endoscópico. 2010. 68 f. Dissertação (Mestrado em Cirurgia) - Universidade Federal do Ceará. Faculdade de Medicina, Fortaleza, 2010.<br>Submitted by denise santos (denise.santos@ufc.br) on 2014-03-11T14:14:46Z No. of bitstreams: 1 2010_dis_mhrbgonçalves.pdf: 841970 bytes, checksum: c3ccf6269ed1300097b844a6e63f0539 (MD5)<br>Approved for entry into archive by denise santos(denise.santos@ufc.br) on 2014-03-11T14:15:4
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14

Goggin, Patrick M. "Human gastric mucosal hydrophobicity : role of mucous and phospholipids, and effect of H. pylori and NSAIDs." Thesis, University of Oxford, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.282494.

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15

Braga, Neto Manuel Bonfim. "Estresse oxidativo e genótipo do H. pylori em pacientes portadores de câncer gástrico e seus familiares." reponame:Repositório Institucional da UFC, 2015. http://www.repositorio.ufc.br/handle/riufc/15467.

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BRAGA NETO, Manuel Bonfim. Estresse oxidativo e genótipo do H. pylori em pacientes portadores de câncer gástrico e seus familiares. 2015. 72 f. Tese (Doutorado em Cirurgia) – Faculdade de Medicina, Universidade Federal do Ceará, Fortaleza, 2015.<br>Submitted by denise santos (denise.santos@ufc.br) on 2016-03-15T15:57:06Z No. of bitstreams: 1 2015_tese_mbbraganeto.pdf: 1082677 bytes, checksum: e07dad10a4915c5d65f1929212d0dedb (MD5)<br>Approved for entry into archive by denise santos(denise.santos@ufc.br) on 2016-03-15T15:58:16Z (GMT) No. of bitstreams: 1 2015_tese_mbbraganeto.pdf: 1082677 bytes
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16

Benigno, Tiago Gomes da Silva. "Analysis of humoral response against H. pylori in patients with gastric cancer patients not." Universidade Federal do CearÃ, 2014. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=13447.

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CoordenaÃÃo de AperfeÃoamento de Pessoal de NÃvel Superior<br>Helicobacter pylori have accompanied humanity in all its migration routes across the planet. Being the cosmopolitan H.pylori bacteria that evolved and adapted the most adverse conditions through mutation and genetic exchange that resulted in favorable changes morph physiological. Such changes contributed to the development of highly virulent strains of bacteria which cause different pathophysiological conditions in the host. The high genetic variability of H. pylori hinders research to uncover associations with certain gastrointesti
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17

Neto, Manuel Bonfim Braga. "Oxidative stress and H. pylori genotype in patients with gastric cancer and their families." Universidade Federal do CearÃ, 2015. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=16379.

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CoordenaÃÃo de AperfeiÃoamento de NÃvel Superior<br>Gastric cancer is associated with H. pylori infection, environmental factors and genetic predisposition. Relatives of gastric cancer patients are at increased risk to develop cancer, possibly, due to infection by more virulent H. pylori strains and genetic predisposition associated with pro-inflammatory cytokines, which may, in turn, result in an exaggerated inflammatory response and oxidative stress. The aim of this study was to: 1) evaluate the oxidative stress in dispeptic patients without family history of gastric cancer (NRC), patients w
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18

Berthenet, Elvire. "Genome evolution and virulence in H. pylori : identifying the genes/alleles underlying phenotype variation." Thesis, Swansea University, 2018. https://cronfa.swan.ac.uk/Record/cronfa43683.

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An estimated 50% of all people carry the stomach bacterium Helicobacter pylori (H. pylori). This organism is responsible for gastric problems like gastritis and gastric ulcers, and is one of the major causes of gastric cancer worldwide. Large numbers of people carry this organism asymptomatically and many questions remain about why serious symptoms develop in a subset of infected humans. These extremely recombinant bacteria may take different evolutionary trajectories in different people, and some genomic changes may be associated with gastric cancer. To test this, and learn more about the gen
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19

Gasiorowski, Elise. "Élucidation du rôle des phosphatases membranaires dépendantes de l'undécaprényl pyrophosphate dans la pathogénèse d'Helicobacter pylori HP0851, the main undecaprenyl pyrophosphate phosphatases in H. pylori is essential for colonization of the stomach." Thesis, Sorbonne Paris Cité, 2018. https://wo.app.u-paris.fr/cgi-bin/WebObjects/TheseWeb.woa/wa/show?t=2168&f=13552.

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La biosynthèse des polysaccahrides de l'enveloppe bactérienne nécessite la translocation, au travers de la membrane cytoplasmique, de sous unités de sucre qui sont produits dans le cytoplasme. A la fin de ces étapes, les sucres doivent se lier à un transporteur lipidique (l'undécaprényl phosphate, C55-P) fixant ces intermédiaires à la membrane, qui sont ensuite transloqué à la face externe de la membrane interne. Finalement, les fragments de glycane sont transférés à un polymère accepteur naissant. Cette étape libère le transporteur dans une forme inactive, l'undécaprényl-pyrophosphate (C55-PP
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20

Oliveira, Maria Aparecida Alves de. "Sítios de fosforilação de tirosina da proteína caga e genótipos cage do H. pylori em pacientes com gastrite e úlcera péptica." reponame:Repositório Institucional da UFC, 2014. http://www.repositorio.ufc.br/handle/riufc/8328.

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OLIVEIRA, Maria Aparecida Alves de. Sítios de fosforilação de tirosina da proteína caga e genótipos cage do H. pylori em pacientes com gastrite e úlcera péptica. 2014. 77 f. Tese (Doutorado em Cirurgia) - Universidade Federal do Ceará. Faculdade de Medicina, Fortaleza, 2014.<br>Submitted by denise santos (denise.santos@ufc.br) on 2014-06-24T12:08:16Z No. of bitstreams: 1 2014_tese_maaoliveira.pdf: 1958951 bytes, checksum: d8dc97da5e093a4373277c54ae890f93 (MD5)<br>Approved for entry into archive by denise santos(denise.santos@ufc.br) on 2014-06-24T12:08:58Z (GMT) No. of bitstreams: 1 2014_tese_
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21

Wilson, John Edward, University of Western Sydney, Faculty of Environmental Management and Agriculture, and School of Agriculture and Rural Development. "A comparison of alternate mucosal routes of prophylactic immunisation using a mouse model of Helicobacter infection." THESIS_FEMA_ARD_Wilson_J.xml, 2001. http://handle.uws.edu.au:8081/1959.7/511.

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Throughout history a diversity of animal species have been used and studied extensively in the development of vaccines for the benefit of humans and animals alike. As mice are a relatively easy species to maintain, handle and manipulate, and have the advantage of being cost effective, they are commonly employed as animal models in the investigation of immunisation strategies against mucosal associated pathogens. Vaccine research against the human gastric pathogen Helicobacter pylori is extensively conducted in a mouse model and typically uses intra-gastric administration for the testing of pot
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22

McCracken, Kyle W. "Mechanisms of endoderm patterning and directed differentiation of human stem cells into foregut tissues." University of Cincinnati / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1406819241.

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23

Abdullah, Norina Binti. "Chemokine expression and regulation in human gastric epithelial cells : association with gastritis and H. pylori." Thesis, University of Bath, 2001. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.392015.

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24

GonÃalves, Maria Helane Rocha Batista. "GenÃtipos das cepas de H. pylori vacA e Alelos cagA e sÃtios de fosfolizaÃÃo EPIYA em uma comunidade urbana de Fortaleza." Universidade Federal do CearÃ, 2010. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=6094.

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FundaÃÃo de Amparo à Pesquisa do Estado do CearÃ<br>O H. pylori infecta atualmente metade da populaÃÃo mundial e à relacionado com o desenvolvimento das afecÃÃes gÃstricas. O Enteroteste à um mÃtodo minimamente invasivo que pode ser usado para detecÃÃo do H. pylori por meio nÃo endoscÃpico. O objetivo deste estudo foi avaliar a sensibilidade e especificidade do Enteroteste frente ao Teste RespiratÃrio e estudar o perfil genÃtico das cepas de H. pylori em indivÃduos residentes na comunidade Parque UniversitÃrio. O suco gÃstrico foi colhido a partir do Enteroteste, e realizada a cultura e a ext
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Goto, Yasuyuki, Kazuko Nishio, Yoshiko Ishida, et al. "No Association Between Helicobacter Pylori Seropositivity and Ornithine Decarboxylase (ODC) A317G Polymorphism, and No Modification by NAD(P)H : Qinone Oxidoreductase 1 (NQO1) C609T." Nagoya University School of Medicine, 2007. http://hdl.handle.net/2237/7474.

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26

Quarantini, S. "Structural and functional study of Helicobacter pylori proteins contributing to stomach colonization." Doctoral thesis, Università degli studi di Padova, 2012. http://hdl.handle.net/11577/3422440.

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Three H. pylori proteins contributing to stomach colonization have been chosen to be structurally characterized: Fur, Hp1454, and HypB. The first one, which is a Fe-dependent transcription regulator was expressed and purified. Unfortunately. after the crystallization test started, the crystal structure of Fur was published by another group. Hp1454 is a secreted protein of unknown function. It has been purified, expressed and crystallized. Since it lacks any sequence similarity with other proteins of known structure, Se-Met derivatives were produced and the structure determined. A function f
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27

Braga, Meyssa Quezado Cavalcante. "Genotypes cagA, vacA and alleles of H. pylori in patients gastritis, peptic ulcer and gastric cancer." Universidade Federal do CearÃ, 2010. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=13463.

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CoordenaÃÃo de AperfeÃoamento de Pessoal de NÃvel Superior<br>The bacterial species H. pylori is closely correlated with the development of major diseases and gastric cancer of the stomach. The strains that have more pathogenic genotype are more involved in bacterial virulence. The objective was to genotype strains of H. pylori from patients with gastritis, gastroduodenal ulcer and gastric cancer as the cagA and vacA genes and alleles by PCR technique. The genotyping of strains of H. pylori was performed by PCR technique. We evaluated 134 strains, 76 of patients with gastritis, 28 peptic ulcer
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28

Rossi, Ana Flávia Teixeira [UNESP]. "Efeito do tratamento da H. pylori na expressão de citocinas e microRNAs associados ao processo inflamatório." Universidade Estadual Paulista (UNESP), 2015. http://hdl.handle.net/11449/127802.

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Made available in DSpace on 2015-09-17T15:25:42Z (GMT). No. of bitstreams: 0 Previous issue date: 2015-03-15. Added 1 bitstream(s) on 2015-09-17T15:46:38Z : No. of bitstreams: 1 000844048_20160312.pdf: 733130 bytes, checksum: 4c63392d725a9495e9543a2df54d0223 (MD5) Bitstreams deleted on 2016-03-14T11:16:23Z: 000844048_20160312.pdf,. Added 1 bitstream(s) on 2016-03-14T11:17:05Z : No. of bitstreams: 1 000844048.pdf: 7529493 bytes, checksum: 9dd7b812bf35ed642076bb8874768278 (MD5)<br>Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)<br>Conselho Nacional de Desenvolvimento Científico
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Sigulla, Janine. "The multifactorial regulation of the immune checkpoint PD-L1 in the course of H. pylori infection." Doctoral thesis, Humboldt-Universität zu Berlin, 2021. http://dx.doi.org/10.18452/22545.

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Eines der prävalentesten humanen Pathogene ist das Magenbakterium Helicobacter pylori, welches ca. die Hälfte der Weltbevölkerung infiziert. Die Persistenz geht mit einer chronischen Gastritis einher, welche bis zu Magenkrebs fortschreiten kann. H.pylori bedient sich diverser Mechanismen um sich der Erkennung des Immunsystems zu entziehen und somit eine chronische Infektion zu ermöglichen. Erhöhte Expression des Immunzellinhibitors PD-L1 wurde in Magenepithelzellen gefunden, welche mit diesem Gram-negativen Erreger infiziert wurden. In dieser Arbeit wurde die Regulation auf in vitro Ebene unte
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Bennett, Monique R. "Adaptation of H pylori to Changing Environments Based on Allelic Variation of Sensor Histidine Kinase Arss." W&M ScholarWorks, 2014. https://scholarworks.wm.edu/etd/1539626756.

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Rossi, Ana Flávia Teixeira. "Efeito do tratamento da H. pylori na expressão de citocinas e microRNAs associados ao processo inflamatório /." São José do Rio Preto, 2015. http://hdl.handle.net/11449/127802.

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Orientador: Ana Elizabete Silva<br>Banca: Cláudia Aparecida Rainho<br>Banca: Marília de Freitas Calmon Saiki<br>Resumo: Infecção persistente pela Helicobacter pylori (H. pylori) promove inflamação crônica que resulta em danos na mucosa e o consequente desenvolvimento de lesões gástricas, como a gastrite crônica. Esta bactéria e seus fatores de virulência vacA e cagA influenciam na resposta imune do hospedeiro, desregulando a expressão de mediadores inflamatórios e microRNAs (miRNAs), assim ativando vias relacionadas ao processo carcinogênico. Portanto, a erradicação da H. pylori pode ter um pa
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Munari, Fabio. "Macrophages and neutrophils as major source of B-cells activating factors in H. pylori associated diseases." Doctoral thesis, Università degli studi di Padova, 2012. http://hdl.handle.net/11577/3422161.

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The cytokine APRIL (A proliferation inducing ligand) and the cytokine BLyS (B lymphocyte simulator) are two recently discovered factors that belong to the TNF-(tumor necrosis factor) family. Since their discovery, these cytokines have been intensively studied for their role in B cells biology. Indeed, APRIL and BLyS are involved in the maturation of B cells, but, most importantly, they increase the survival and proliferation of these cells. However, the effect of these cytokines is amplified in neoplastic B cells in lymphomas, and self-reactive B cells in autoimmune diseases. In particular, it
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Moreilhon, Chimène. "Analyse transcriptome des réponses de l'hôte à des infections de l'épithélium respiratoire par S. Aureus et de l'estomac par H. Pylori." Nice, 2005. http://www.theses.fr/2005NICE4009.

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L’objectif de ma thèse a été d’étudier les réponses transcriptionnelles suite à l’infection par Statphylococcus aureus (S. Aureus) et Helicobacter pylori (H. Pylori) grâce aux puces à ADN. Pour identifier les mécanismes sous-jacents à une plus forte sensibilité de l’épithélium respiratoire à S. Aureus lors de la mucoviscidose, j’ai étudié les réponses transcriptionnelles des cellules épithéliales respiratoires MM-39 (CFTR non muté) après contact avec S. Aureus ou avec le surnageant bactérien qui contient les produits de sécrétion. Les facteurs de transcription comme HIF ou NF-kB sont surexprim
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Silva, Carina Siqueira Martelli da. "VITAMINA C NO SORO E SUCO GÁSTRICO DE PACIENTES DISPÉPTICOS COM E SEM INFECÇÃO POR HELICOBACTER PYLORI." Universidade Federal de Santa Maria, 2015. http://repositorio.ufsm.br/handle/1/5863.

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Introduction: Helicobater pylori interferes with vitamin C homeostasis and low levels of vitamin C may be a risk factor for H. pylori infection. Objective: to investigate the vitamin C serum and gastric juice levels of patients infected and non-infected by H. pylori. Additionally assess the association of vitamin C levels with H.pylori status, gastric pH and histological changes of gastric mucosa of dyspeptic patients with normal upper GI endoscopy. Methods: We studied 42 dyspeptic patients undergoing upper GI endoscopy had biopsies for diagnosis of H. pylori infection and gastric mucosa chang
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Olofsson, Annelie. "Helicobacter pylori outer membrane vesicles and the host-pathogen interaction." Doctoral thesis, Umeå universitet, Institutionen för medicinsk kemi och biofysik, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-68744.

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Pathak, Amita, Victoria L. Blair, Richard L. Ferrero, Michael Mehring, and Philip C. Andrews. "Bismuth(III) benzohydroxamates: powerful anti-bacterial activity against Helicobacter pylori and hydrolysis to a unique Bi34 oxido-cluster [Bi34O22(BHA)22(H-BHA)14(DMSO)6]." Universitätsbibliothek Chemnitz, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:ch1-qucosa-161727.

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Reaction of BiPh3 or Bi(OtBu)3 with benzohydroxamic acid (H2-BHA) results in formation of novel mono- and di-anionic hydroxamato complexes; [Bi2(BHA)3]∞1, [Bi(H-BHA)3] 2, [Bi(BHA)(H-BHA)] 3, all of which display nM activity against Helicobacter pylori. Subsequent dissolution of [Bi2(BHA)3]∞ in DMSO/toluene results in hydrolysis to the first structurally authenticated {Bi34} oxido-cluster [Bi34O22(BHA)22(H-BHA)14(DMSO)6] 4<br>Dieser Beitrag ist aufgrund einer (DFG-geförderten) Allianz- bzw. Nationallizenz frei zugänglich
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37

Aspholm, Marina. "Adaptation of Helicobacter pylori Adherence Properties in Promotion of Host Tropism and Inflammatory Disease." Doctoral thesis, Umeå universitet, Oral mikrobiologi, 2004. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-388.

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Being among the most prevalent of persistent infectious agents in humans worldwide, Helicobacter pylori induces chronic inflammation (gastritis), which may progress to peptic ulceration and stomach cancer. The ability to adhere to the gastric mucosa is considered to be both a colonization and virulence property of H. pylori. For adherence, H. pylori expresses surface-located attachment proteins (adhesins) that bind to specific receptors in the gastric mucosa. The best characterized H. pylori adhesin-receptor interaction is that between the blood group antigen binding adhesin (BabA) and the fuc
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38

Jordaan, Maraliese. "Diagnosis of helicobacter pylori infection with the 13C-urea breath test : analysis by means of gas chromatography with mass selective detection." Diss., University of Pretoria, 2007. http://hdl.handle.net/2263/27035.

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39

Ayna, Adnan. "Glyceraldehyde-3-phosphate dehydrogenase and fructose-1, 6-bisphosphatase of the enteric pathogens C. jejuni and H. pylori." Thesis, University of Leicester, 2016. http://hdl.handle.net/2381/37425.

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Campylobacter jejuni and Helicobacter pylori are pathogens which cause gastrointestinal diseases and are therefore of significant importance. However, their metabolism and physiology is relatively poorly understood. It had been noted that the genome of these pathogens lack open reading frames for some glycolytic enzymes. Notably, both pathogens lack the gene encoding phosphofructokinase (6-PFK) and thus regulation of the complementary gluconeogenic enzyme fructose-1,6-bisphosphatase (FBPase) might be different to that in the majority of organisms which retain 6-PFK. In order to further underst
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40

Sanches, Bruno Squarcio Fernandes. "Comparação de dois regimes terapêuticos no retratamento da infecção por H. pylori: ensaio clínico aberto, prospectivo e aleatorizado." Universidade Federal de Minas Gerais, 2007. http://hdl.handle.net/1843/ECJS-7EZPMD.

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H. pylori treatment failure is a growing problem in daily practice. Aim: to evaluate and compare the efficacy of two new regimes as second-line options in a randomized, prospective study. Methods: Patients previously submitted, without success, to clarithromycin-based regimes were included. 13C-urea breath test (UBT) was performed in all patients. The patients were randomized to receive a combination of rabeprazole 20mg, levofloxacin 500mg and furazolidone 200mg (2 tablets) administered in a single dose for 10 days (RLF) or the association of rabeprazole 20mg, bismuth subcitrate 120mg (2 table
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Silva, Cicero Igor Simoes Moura. "Genótipos cagA , vacA e alelos e sítios de fosfolilação de tirosina da proteína caga do h. pylori em pacientes com e sem história familiar de câncer gástrico." reponame:Repositório Institucional da UFC, 2009. http://www.repositorio.ufc.br/handle/riufc/7298.

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SILVA, Cicero Igor Simoes Moura. Genótipos cagA , vacA e alelos e sítios de fosfolilação de tirosina da proteína caga do h. pylori em pacientes com e sem história familiar de câncer gástrico. 2009. 73 f. Dissertação (Mestrado em Cirurgia) - Universidade Federal do Ceará. Faculdade de Medicina, Fortaleza, 2009.<br>Submitted by denise santos (denise.santos@ufc.br) on 2014-02-18T14:08:21Z No. of bitstreams: 1 2009_dis_cismsilva.pdf: 858659 bytes, checksum: d9ead253a62bca88b77a42f2664ed067 (MD5)<br>Approved for entry into archive by denise santos(denise.santos@ufc.br) on 2014-02-18T14:09:43Z (GMT
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42

Mazzoleni, Felipe. "Eosinófilos duodenais : potencial associação com a infecção pelo Helicobacter pylori e com os sintomas da dispepsia funcional." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2017. http://hdl.handle.net/10183/174772.

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Introdução e objetivos: Eosinofilia duodenal está associada com parasitoses intestinais e com alergias alimentares, e tem sido sugerida como possível fator etiológico da dispepsia funcional, pela capacidade de causar alterações na motilidade e na sensibilidade do aparelho digestivo. Sua relação com o Helicobacter pylori é pouco conhecida, tendo sido avaliada apenas como achado secundário em alguns estudos, com resultados controversos. Esse estudo tem como objetivos avaliar o papel da infecção gástrica pelo H. pylori no número de eosinófilos duodenais e avaliar a relação dos eosinófilos duodena
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43

Amorocho, Cruz Claudia Milena. "Caracterización y potencial probiótico de bacterias lácticas aisladas de leche de oveja guirra." Doctoral thesis, Universitat Politècnica de València, 2011. http://hdl.handle.net/10251/13830.

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Diversos estudios señalan que varias especies pertenecientes a las Bacterias Acido Lácticas (BAL) poseen propiedades probióticas y se encuentran comúnmente en los derivados lácteos. Actualmente, en la Comunidad Valenciana se comercializan quesos frescos y madurados elaborados a partir de la leche de oveja guirra, autóctona de esta comunidad e incluida dentro del catálogo de razas protegidas en España. Sin embargo, hasta el momento no se han realizado ensayos para determinar el potencial probiótico de cepas aisladas de esta leche. En esta tesis, se han aislado 131 BAL de leche de oveja Guirra
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PUCCIO, SIMONE. "DEVELOPMENT OF A WEBTOOL FOR INTERACTOME­SEQUENCING DATA ANALYSIS AND IDENTIFICATION OF H. PYLORI EPITOPES RESPONSIBLE FOR HOST IMMUNO­RESPONSE MODULATION." Doctoral thesis, Università degli Studi di Milano, 2017. http://hdl.handle.net/2434/473104.

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To elucidate the molecular mechanisms involved in persistency/latency of the H. pylori infection or in its progression towards serious diseases, it is necessary to analyse the host pathogen interaction in vivo. The circulating antibody repertoire represents an important source of diagnostic information, serving as biomarker to provide a “disease signature”. The aim of this work is the identification of H. pylori epitopes responsible for host immuno­response modulation through: a discovery­driven approach that couples “phage display” and deep sequencing (interactome-sequencing) and the develo
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45

Mustapha, Pascale. "Etude des interactions entre Helicobacter pylori et les cellules épithéliales gastriques." Phd thesis, Université de Poitiers, 2011. http://tel.archives-ouvertes.fr/tel-00742960.

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L'infection par Helicobacter pylori provoque une inflammation qui peut persister de façon asymptomatique ou évoluer vers de nombreuses pathologies gastroduodénales sévères telles que les ulcères gastroduodénaux, le lymphome du MALT et le cancer gastrique. L'îlot de pathogénicité cag est l'un des facteurs de virulence majeurs de cette bactérie. Plusieurs cytokines et peptides antimicrobiens sont impliqués dans la modulation de la réponse inflammatoire de la muqueuse épithéliale gastrique lors de l'infection par H. pylori. Ce travail a porté sur l'étude des interactions entre H. pylori et les ce
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46

Anderson, Kathleen. "CD25+ CTLA-4+ T Cell-Dependent Induction of Anergic CD25- T Cells Limits the Immune Response to H. pylori Infection Resulting in Mild Gastritis and Persistent Colonization." Connect to text online, 2006. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=case1144332338.

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47

Chan, Rebeca, and 陳懿雯. "A systematic review on the effectiveness of the first-line treatment of gastroesophageal reflux disease in H. pylori infected patients." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47559895.

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Helicobacter pylori (H.pylori) had been confirmed by the World Health Organization (WHO) as Group 1 carcinogens, in which it has been identified to be related with the development of gastric carcinoma. Gastroesophageal reflux disease (GERD) is less commonly found in Asia, while the number of H.pylori infection is considerably to be higher than that of the Western population. The relationship between H.pylori and GERD still remains ambiguous nowadays. One of the contributing factors affecting the level of gastric secretion might be due to the genetic cause. The aim of this review is to assess
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Oliveira, Maria Aparecida Alves de. "Sites of tyrosine phosphorylation of CagA protein and cagE genotypes of H. pylori in patients with gastritis and peptic ulcer." Universidade Federal do CearÃ, 2014. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=11915.

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The H. Pylori strains demonstrate a high level of phenotypic and genotypic diversity, the expression of various genes, which confer greater pathogenicity to the bacteria. Most studies have found that the cagE gene is strongly associated with peptic ulcer disease and sites of tyrosine phosphorylation of CagA protei - EPIYA, has not been fully elucidated existing geographical differences. This study aimed to characterize the strains of H. pylori for the sites of tyrosine phosphorylation of CagA protein EPIYA and presence of genes cagE in patients with gastritis and peptic ulcer disease, and to
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49

Aguemon, Badirou. "Helicobacter pylori en Belgique et au Bénin: prévalence, facteurs de risque, évaluation de la résistance aux antibiotiques et efficacité thérapeutique dans les pathologies ulcéro-inflammatoires de la sphère digestive haute." Doctoral thesis, Universite Libre de Bruxelles, 2005. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/211032.

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Le rôle majeur de l’Helicobacter pylori dans l’étiopathogénie des maladies gastroduodénales (gastrite, ulcère gastrique et duodénal, lymphome gastrique) est bien établi aujourd’hui. L’OMS l’a reconnue comme jouant un rôle important dans la survenue des lésions cancéreuses gastriques. La prévalence de l’infection à H. pylori varie selon les pays de 20% à 90% avec des taux supérieurs à 60% dans les pays en développement, dont le Bénin. Les méthodes usuelles de diagnostic sont soit invasives nécessitant une endoscopie gastrique avec biopsies (test rapide à l’urée, histologie, culture et PCR), soi
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Melo, Cynthia Farias Vieira de. "Análise do perfil de metilação dos genes THBS1, GPX3 e COX2 e identificação de H. pylori em amostras de câncer gástrico." Universidade Federal da Paraí­ba, 2013. http://tede.biblioteca.ufpb.br:8080/handle/tede/3645.

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Made available in DSpace on 2015-04-01T14:16:01Z (GMT). No. of bitstreams: 1 ArquivoTotal.pdf: 1987821 bytes, checksum: 0557f7c6083cf4200e6fc6542c383ea7 (MD5) Previous issue date: 2013-05-16<br>Coordenação de Aperfeiçoamento de Pessoal de Nível Superior - CAPES<br>Cancer is a disease with a high mortality rate in Brazil, including, stomach cancer is currently the fourth most common type of cancer worldwide, responsible for countless deaths. Its etiology is multifactorial, because studies suggest associations to various factors such as dietary habits, environmental factors, genetic and
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