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Artículos de revistas sobre el tema "Inflammation"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 25 de julio de 2025

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1

Nordstrom, Thomas. "What is Inflammation?" International Journal of Science and Research (IJSR) 11, no. 1 (2022): 992–95. http://dx.doi.org/10.21275/sr22119212044.

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2

Vijayendranath, Nayak S., Gunasheela S, Karthik M, and Hegde Aparna. "Healing by Inflammation - Prolotherapy." Case Reports in Dental Science 1, no. 1 (2020): 9–14. http://dx.doi.org/10.46619/crds.2020.1-1003.

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Temporomandibular disorder (TMD) is a collective term used to describe a group of disorders related to temporomandibular region. It’s considered to be the common cause for orofacial pain. With the advancement of research, prolotherapy is considered to be the one of the treatment modalities to treat TMD, when the conservative management fails.

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3

J, Mancini-Filho. "Natural Antioxidants and Tissue Inflammation." Bioequivalence & Bioavailability International Journal 7, no. 2 (2023): 1–3. http://dx.doi.org/10.23880/beba-16000203.

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The phenolic compounds present in food cover a wide range of structures that have different biological properties. Highlighting its antioxidant properties and the presence mainly of spices, herbs and other foods. Some compounds present in spices can be listed for their antioxidant activity, such as: cloves have eugenol, pinene in their composition, cinnamon also has eugenol, limonene, pinene, catechins and other phenolic compounds in their composition, anise has pinene, rutin, apigenin, oregano has apigenin, quercecin, rosmarinic, caffeic, p-coumaric acids, and others. Rosemary presents the ca

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4

Narula, Jagat, and Eloisa Arbustini. "Inflammation, Superadded Inflammation, and Out-of-Proportion Inflammation in Atherosclerosis." JAMA Cardiology 3, no. 10 (2018): 912. http://dx.doi.org/10.1001/jamacardio.2018.2760.

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5

Djaldetti, Meir. "Piperine – An Immunomodulator and Inflammation Mitigator." Journal of Clinical and Laboratory Research 2, no. 5 (2021): 01–04. http://dx.doi.org/10.31579/2768-0487/027.

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Black pepper, one of the most widespread spices, gained the entitlement “King of spices” founded on its peculiar pungent test and therapeutic properties, both owed to its active alkaloid - piperine. Mounting evidence indicates that piperine possesses immunomodulatory and therapeutic activities. The aim of this mini review was to summarize the role of piperine in abolishing inflammation, its part in the immune activity of peripheral blood mononuclear- and a number of other cells, its capacity to elicit production of inflammatory cytokines and its function as a synergist endorsing the beneficial

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6

H, Khazaei. "Overview of Orbital Inflammation/Unmet Needs." Open Access Journal of Ophthalmology 7, no. 2 (2022): 1–6. http://dx.doi.org/10.23880/oajo-16000245.

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Diseases of the orbit and periorbital eye tissues manifest in a wide variety of clinical presentations. Space occupying lesions in the orbit include infections, inflammations, vascular malformations, and malignancies. The significant variation in presentations is due to the complex anatomy of the orbit and the heterogeneous nature of the multiple disease processes that present themselves as orbital inflammatory processes. Additionally, although specific disease entities often show similar patterns of orbital tissue involvement, there is still a spectrum of clinical presentations within disease

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7

Ado, A. A. "Allergic inflammation." Kazan medical journal 29, no. 11-12 (2022): 966–83. http://dx.doi.org/10.17816/kazmj90287.

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Relatively not so long ago, the doctrine of the inflammatory response was enriched by the concept of "allergic inflammation" (von Pirquet 1907-08). However, the subsequent experimental and clinical study of these inflammatory forms has deepened our understanding of the pathogenesis of each inflammatory reaction so much that it can rightfully be called a stage in the new history of inflammation. From time immemorial, the doctor knew the variety of forms of development and course of inflammation. The construction of these numerous modifications of inflammation in most cases remained unclear. In

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8

Alderton, Gemma, and Seth Thomas Scanlon. "Inflammation." Science 374, no. 6571 (2021): 1068–69. http://dx.doi.org/10.1126/science.abn1721.

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9

Miyamoto, Terumasa. "Inflammation." Ensho 13, no. 2 (1993): 99–100. http://dx.doi.org/10.2492/jsir1981.13.99.

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10

Miyamoto, Terumasa. "Inflammation." Ensho 9, no. 5 (1989): 357. http://dx.doi.org/10.2492/jsir1981.9.357.

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11

Lederer, Katy. "Inflammation." Colorado Review 43, no. 3 (2016): 128–30. http://dx.doi.org/10.1353/col.2016.0094.

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12

Weiss, Ursula. "Inflammation." Nature 454, no. 7203 (2008): 427. http://dx.doi.org/10.1038/454427a.

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13

Weiss, Ursula. "Inflammation." Nature 420, no. 6917 (2002): 845. http://dx.doi.org/10.1038/nature01319x.

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14

Vergnolle, N., and P. Andrade-Gordon. "Inflammation." Journal of Pain 5, no. 3 (2004): S23. http://dx.doi.org/10.1016/j.jpain.2004.02.060.

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15

Parnes, Ohad. "Inflammation." Lancet 372, no. 9639 (2008): 621. http://dx.doi.org/10.1016/s0140-6736(08)61262-3.

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16

Cone, John B. "Inflammation." American Journal of Surgery 182, no. 6 (2001): 558–62. http://dx.doi.org/10.1016/s0002-9610(01)00822-4.

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17

Penn, Audrey S. "Inflammation." Current Opinion in Neurology 9, no. 3 (1996): 219–20. http://dx.doi.org/10.1097/00019052-199606000-00012.

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18

Johnston, Craig A. "Inflammation." American Journal of Lifestyle Medicine 6, no. 1 (2011): 18–20. http://dx.doi.org/10.1177/1559827611425023.

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19

Mantovani, A. "Inflammation." Research in Immunology 143, no. 8 (1992): 850–52. http://dx.doi.org/10.1016/0923-2494(92)80104-s.

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20

Russo-Marie, Françoise. "Inflammation." EMC - Cosmétologie et dermatologie esthétique 1, no. 1 (2000): 1–6. https://doi.org/10.1016/s2211-0380(20)30008-9.

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21

Johnkennedy, Nnodim, and Okafor Chibuzor Mercy. "Perspective of Inflammation and Inflammation Markers." Journal La Medihealtico 3, no. 1 (2022): 16–26. http://dx.doi.org/10.37899/journallamedihealtico.v3i1.620.

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Progression and occurrence of coronary heart disease can be attributed in part to the presence of inflammation. As a result of our understanding of inflammation's molecular underpinnings, we have identified markers that may also serve as new treatment targets for atherosclerosis. Individuals with and without a history of cardiovascular disease can benefit from monitoring their C-reactive protein (CRP) levels (CVD). The anti-inflammatory characteristics of statins have lately been explored, and they have been shown to significantly lower cardiovascular morbidity and death. C-reactive protein, a

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22

Strukova, Svetlana. "Blood coagulation-dependent inflammation. Coagulation-dependent inflammation and inflammation-dependent thrombosis." Frontiers in Bioscience 11, no. 1 (2006): 59. http://dx.doi.org/10.2741/1780.

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23

Aarabi, Ghazal, Renate Schnabel, Guido Heydecke, and Udo Seedorf. "Potential Impact of Oral Inflammations on Cardiac Functions and Atrial Fibrillation." Biomolecules 8, no. 3 (2018): 66. http://dx.doi.org/10.3390/biom8030066.

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Inflammation may be a risk factor for atrial fibrillation (AF). Oral infections frequently lead to chronic inflammation, such as gingivitis, periodontitis, and endodontic lesions. In this narrative review, we consider five basic pathogenic mechanisms that involve oral infections and inflammations in the pathogenesis of AF: (1) low level bacteremia by which oral bacteria enter the blood stream at inflamed sites of the oral cavity and invade the heart; (2) Systemic inflammation induced by inflammatory mediators, which are released from the sites of oral inflammation into the blood stream, affect

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24

Kuzenko, Yevhen, Anatoly Romanyuk, and Antonina Politun. "Macrophage in periodontal inflammation." Journal of Stomatology 69, no. 6 (2016): 674–79. http://dx.doi.org/10.5604/00114553.1230588.

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Aim of the study. To investigate the expression of CD68 and HSP90AA1 in periodontal inflammation. Methods. A total of twenty-seven patients (giant-cell epulis) and thirty patients (acute and chronic inflammations) have been examined for expression of CD68 and HSP90AA1 by hematoxylin – eosin and immunohistochemistry. Results. Strong giant-cell positivity for CD68 was observed in 100% of patients whereas only 85.31% of giant-cell was positive for HSP90AA1 (p lower than 0.05). Strong macrophage immunoreactivity for CD68 was figured in acute 23.2±1.3%, and in chronic 83.1±5.6% (p lower than 0.05)

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25

Naseer, Sikandar, Gulnaz Tasleem, Bareera Zahoor, Bisma Hadi, Amna Amin, and Alishba Hadi. "Exploring the Interplay Between Cancer, Health, and Inflammation." Journal of Clinical and Nursing Research 8, no. 11 (2024): 230–43. http://dx.doi.org/10.26689/jcnr.v8i11.8638.

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Cancer is a complex disease influenced by various factors, including DNA damage, growth signals, and inflammation. Although inflammation has commonly not been considered carcinogenic, increasing evidence indicates its substantial involvement in the onset and progression of cancer, especially in the presence of chronic microbial infections. This review thoroughly analyzes the complex relationship between cancer, health, and inflammation by introducing pathological and physiological features of inflammation. The study explores the various factors that might enhance inflammation, including infect

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26

John Edward, Nagar, and Elmo Ado. "Curcumin in Turmeric as Treatment for Eye Inflammation." Journal of Optometry, Eye and Health Research 4, no. 2 (2023): 7. http://dx.doi.org/10.57002/joehr.v4i2.301.

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AbstractThe research wants to find out if curcumin in turmeric is effective in treating inflammations of the eye. Theresearch also aims to determine if curcumin is an irritant. To answer these questions, curcumin wasextracted and eyedrops were made to be tested on animal models of eye inflammation. Results show thatsigns of inflammation was alleviated with the use of the eyedrops made from curcumin. The study alsofound out that curcumin is practically non-irritative. The study shows the potential use of curcumin as anophthalmic drop, capable of eliminating eye inflammations.

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27

Titus, Anto Sam Crosslee Louis Sam. "Macrophage Polarization in Tissue Inflammation and Fibrosis." Clinical Pathology & Research Journal 7, no. 1 (2023): 1–3. http://dx.doi.org/10.23880/cprj-16000175.

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Macrophages are mononuclear phagocytic cells, which have varied phenotypes and perform diverse intricate functions ranging from inflammation to their role in onset and progression of fibrotic diseases

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28

Klepikov, Igor. "Pseudosepsis in Acute Inflammation of the Lung." Open Access Journal of Pulmonary & Respiratory Sciences 8, no. 1 (2024): 1–5. http://dx.doi.org/10.23880/oajprs-16000162.

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Sepsis has become one of the global problems in the healthcare system in recent decades. According to the World Health Organization (WHO), 49 million people suffer from sepsis every year, treatment of which leads to death in 11 million cases [1].

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29

Chukwuedozie Francis Nwachukwu. "Inflammatory reaction - A posit to the double-edged sword." International Journal of Biological and Pharmaceutical Sciences Archive 1, no. 2 (2021): 197–209. http://dx.doi.org/10.30574/ijbpsa.2021.1.2.0036.

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Response by inflammation is triggered by arrays of causes, which include disrupted cells, toxins, germs, and others. It underlies a wide variety of pathophysiological changes. Many aspects of inflammation as it relates to the pathology of various inflammations are very much understood. Yet the healthy roles of inflammation are widely unknown. Inflammation has a controversial role in health and its meanings are, a matter of viewpoint. It has critical roles in protecting organisms from pathogens and injurious substances likewise causing a driving variety of disease progression. On this ground th

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30

Fehér, János, Illés Kovács, and Corrado Balacco Gabrieli. "Role of gastrointestinal inflammations in the development and treatment of depression." Orvosi Hetilap 152, no. 37 (2011): 1477–85. http://dx.doi.org/10.1556/oh.2011.29166.

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Recent studies have revealed that inflammation, among other factors, may be involved in the pathogenesis of depression. One line of studies has shown that depression is frequently associated with manifest gastrointestinal inflammations and autoimmune diseases as well as with cardiovascular diseases, neurodegenerative diseases, type 2-diabetes and also cancer, in which chronic low-grade inflammation is a significant contributing factor. Thus depression may be a neuropsychiatric manifestation of a chronic inflammatory syndrome. Another line of studies has shown that the primary cause of inflamma

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31

Sharemi, Seyedeh Hajar, Faezeh Fashkhami, Tayebeh Omid Khah, et al. "Prevalence and Risk Factors of Cervical Inflammations Among Low-Income Women in Rasht, Iran." Disease and Diagnosis 13, no. 3 (2024): 124–29. http://dx.doi.org/10.34172/ddj.1601.

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Background: Cervical inflammations are among one of the most genital-related complications. This study aimed to investigate the prevalence of cervical inflammations and identify associated risk factors among low-income women in Rasht, Iran. Materials and Methods: This cross-sectional study was conducted on 300 low-income women who referred to Alzahra Hospital, Rasht, Iran. Demographical and clinical data were collected from patients’ archived medical records. Pap smear tests were performed using the liquid-based method to evaluate cervical lesions, and all data were analyzed using SPSS-19 base

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32

Kondapalli, Lavanya, Javid Moslehi, and Marc P. Bonaca. "Inflammation begets inflammation: cancer and acute MI." European Heart Journal 41, no. 23 (2020): 2194–96. http://dx.doi.org/10.1093/eurheartj/ehz951.

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33

Meirovitz, Amichay, Rachel Goldberg, Adi Binder, Ariel M. Rubinstein, Esther Hermano, and Michael Elkin. "Heparanase in inflammation and inflammation-associated cancer." FEBS Journal 280, no. 10 (2013): 2307–19. http://dx.doi.org/10.1111/febs.12184.

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34

White, Raymond P. "Third Molar Oral Inflammation and Systemic Inflammation." Journal of Oral and Maxillofacial Surgery 63, no. 8 (2005): 5–6. http://dx.doi.org/10.1016/j.joms.2005.05.013.

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35

Fleischman, Angela G. "Inflammation as a Driver of Clonal Evolution in Myeloproliferative Neoplasm." Mediators of Inflammation 2015 (2015): 1–6. http://dx.doi.org/10.1155/2015/606819.

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Our understanding of inflammation’s role in the pathogenesis of myeloproliferative neoplasm (MPN) is evolving. The impact of chronic inflammation, a characteristic feature of MPN, likely goes far beyond its role as a driver of constitutional symptoms. An inflammatory response to the neoplastic clone may be responsible for some pathologic aspects of MPN. Moreover,JAK2V617Fmutated hematopoietic stem and progenitor cells are resistant to inflammation, and this gives the neoplastic clone a selective advantage allowing for its clonal expansion. Because inflammation plays a central role in MPN infla

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36

Shi, Xuan-Zheng, John H. Winston, and Sushil K. Sarna. "Differential immune and genetic responses in rat models of Crohn's colitis and ulcerative colitis." American Journal of Physiology-Gastrointestinal and Liver Physiology 300, no. 1 (2011): G41—G51. http://dx.doi.org/10.1152/ajpgi.00358.2010.

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Crohn's disease and ulcerative colitis are clinically, immunologically, and morphologically distinct forms of inflammatory bowel disease (IBD). However, smooth muscle function is impaired similarly in both diseases, resulting in diarrhea. We tested the hypothesis that differential cellular, genetic, and immunological mechanisms mediate smooth muscle dysfunction in two animal models believed to represent the two diseases. We used the rat models of trinitrobenzene sulfonic acid (TNBS)- and dextran sodium sulfate (DSS)-induced colonic inflammations, which closely mimic the clinical and morphologi

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37

Arulselvan, Palanisamy, Masoumeh Tangestani Fard, Woan Sean Tan, et al. "Role of Antioxidants and Natural Products in Inflammation." Oxidative Medicine and Cellular Longevity 2016 (2016): 1–15. http://dx.doi.org/10.1155/2016/5276130.

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Inflammation is a comprehensive array of physiological response to a foreign organism, including human pathogens, dust particles, and viruses. Inflammations are mainly divided into acute and chronic inflammation depending on various inflammatory processes and cellular mechanisms. Recent investigations have clarified that inflammation is a major factor for the progression of various chronic diseases/disorders, including diabetes, cancer, cardiovascular diseases, eye disorders, arthritis, obesity, autoimmune diseases, and inflammatory bowel disease. Free radical productions from different biolog

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38

Nikolaos, Tzenios, E. Tazanios Mary, ObGyn, and Chahine Mohamad. "Chronic Inflammation and Blood Cancer." International Journal of Pharmaceutical and Clinical Research 14, no. 11 (2022): 835–42. https://doi.org/10.5281/zenodo.13879580.

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Chronic inflammation may have a detrimental impact on human health as it tends to result in cancer. In addition, it is often linked to different steps that participate in tumorigenesis, including cellular transformation, survival, promotion, invasion, proliferation, angiogenesis, and metastasis. Hence, inflammation predisposes cancer development and plays a vital role in promoting all tumorigenesis stages. Inflammation is caused by many factors, such as bacterial and viral infections, tobacco smoking, autoimmune diseases, obesity[21], asbestos exposure, and many others, increasing cancer risk.

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39

Yin, Geng, Ying Wang, Xiao-min Cen, Min Yang, Yan Liang та Qi-bing Xie. "Lipid Peroxidation-Mediated Inflammation Promotes Cell Apoptosis through Activation of NF-κB Pathway in Rheumatoid Arthritis Synovial Cells". Mediators of Inflammation 2015 (2015): 1–10. http://dx.doi.org/10.1155/2015/460310.

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Rheumatoid arthritis (RA) is a systemic autoimmune disease characterized by chronic inflammation of multiple joints. The central pathogenesis of RA is the proliferation of synovial fibroblasts in response to inflammatory cytokines. However, some of the targeted therapies for inflammation reactions do not display significant clinical improvement after initiation of therapy. Thus, the relationship between inflammatory responses and RA therapy is still incompletely understood. In the present study, we proposed to determine whether enhanced inflammations may lead to cell apoptosis in rheumatoid ar

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40

Brito, T. de, and M. F. Franco. "Granulomatous inflammation." Revista do Instituto de Medicina Tropical de São Paulo 36, no. 2 (1994): 185–92. http://dx.doi.org/10.1590/s0036-46651994000200016.

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41

Natsume, Midori. "Polyphenols: Inflammation." Current Pharmaceutical Design 24, no. 2 (2018): 191–202. http://dx.doi.org/10.2174/1381612823666171109104141.

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Background: Polyphenols widely distributed in plants, fruits and vegetables have received considerable attention on account of their physiological functions, including their antioxidant and anti-inflammatory properties. Some antioxidant components of cacao liquor prepared from fermented and roasted cacao beans, which is a major ingredient of cocoa and chocolate products, have been characterized as flavan-3-ols and procyanidin oligomers. Methods: This review focuses on a specific group of (-)-epicatechins and their oligomers, the procyanidins, in cacao products. Dietary polyphenols in cacao pro

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42

Hussain, Mazhar, Muhammad Bilal Ghafoor, Muhammad Shahbaz Hussain, and Lubna Akhtar. "SUBCLINICAL INFLAMMATION." Professional Medical Journal 23, no. 09 (2016): 1132–37. http://dx.doi.org/10.29309/tpmj/2016.23.09.1710.

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Objectives: To evaluate the anti-inflammatory effects of sitagliptin in type 2diabetic hyperlipidemic patients. Period: 25 August 2015 to 25 November 2015 (12 weeks).Study Design: Randomized clinical trials. Setting: Outdoor of diabetic clinic of Sheikh ZayedMedical College/Hospital, Rahim Yar Khan. Materials and Methods: Diabetic patients (n=46)with poor glycemic control(HbA1c > 7.2%) and deranged lipid profile were selected. The patientreceived sitagliptin 50mg twice daily for 12 weeks. Results: A total of 46 patients completed thestudy. After 12 weeks treatment with sitagliptin, there wa

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43

Gilroy, Derek W. "Resolving inflammation." Nature Reviews Immunology 21, no. 10 (2021): 620–21. http://dx.doi.org/10.1038/s41577-021-00597-w.

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44

Aikawa, Masanori, Ichiro Manabe, Adrian Chester, and Elena Aikawa. "Cardiovascular Inflammation." International Journal of Inflammation 2012 (2012): 1–2. http://dx.doi.org/10.1155/2012/904608.

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45

Wang, Miao, Weijun Jin, Austin Meng Guo, and Jane Stubbe. "Cardiovascular Inflammation." Mediators of Inflammation 2013 (2013): 1–2. http://dx.doi.org/10.1155/2013/123513.

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46

David, Rachel. "Manipulating inflammation." Nature Reviews Microbiology 8, no. 2 (2010): 90. http://dx.doi.org/10.1038/nrmicro2303.

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47

Meuwissen, Martijn, Allard C. van der Wal, Robbert J. de Winter, Karel T. Koch, Anton E. Becker, and Jan J. Piek. "Stent Inflammation." Circulation 106, no. 9 (2002): 1176–77. http://dx.doi.org/10.1161/01.cir.0000029209.89156.88.

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48

Maciel, T. T., E. Merle, A. Fricot, et al. "PATHOLOGY INFLAMMATION." Nephrology Dialysis Transplantation 29, suppl 3 (2014): iii25—iii26. http://dx.doi.org/10.1093/ndt/gfu119.

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49

Flower, Roderick J., and Mauro Perretti. "Controlling inflammation." Journal of Experimental Medicine 201, no. 5 (2005): 671–74. http://dx.doi.org/10.1084/jem.20050222.

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The inflammatory response protects the body against infection and injury but can itself become deregulated with deleterious consequences to the host. It is now clear that several endogenous biochemical pathways activated during defense reactions can counterregulate inflammation. New experimental evidence adds resolvin E1 to this group of endogenous inhibitors and provides further rationale for the beneficial effects of dietary supplementation with fish oils. It also highlights an unexpected twist in the pharmacology of aspirin.

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50

Huerva, Valentín, Francisco J. Ascaso, and Andrzej Grzybowski. "Ocular Inflammation." Mediators of Inflammation 2015 (2015): 1–2. http://dx.doi.org/10.1155/2015/398076.

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