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Tesis sobre el tema "Inflammatory bowel diseas"

Autor: Grafiati

Publicado: 1 de febrero de 2025

Última modificación: 31 de julio de 2025

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1

Cotterill, Lynn. "Inflammatory bowel disease genetics." Thesis, University of Manchester, 2011. https://www.research.manchester.ac.uk/portal/en/theses/inflammatory-bowel-disease-genetics(daae1a60-2790-4280-b7d5-ac5ec7533c7c).html.

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Inflammatory bowel disease (IBD), which includes the subtypes Crohn's disease (CD) and ulcerative colitis (UC), is a common disease particularly in the Western world. IBD is characterised by inflammation of the small intestine and/or colon. The two subtypes affect different gut locations but both show an increased intestinal permeability or the 'leaky gut syndrome'. This led to the hypothesis that tight junction (TJ) proteins expressed in the epithelium may affect the intestinal permeability as a cause or effect of IBD.Initially, variants in the CARD15, IL23R and ATG16L1 genes, previously asso

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2

Pozuelo, del Río Marta. "Metagenomics in inflammatory bowel disease." Doctoral thesis, Universitat Autònoma de Barcelona, 2019. http://hdl.handle.net/10803/669437.

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La microbiota intestinal desempeña un papel crucial en el manteniendo la homeostasis intesitnal. Alteraciones en la composición microbiana, también conocidas como disbisosis, pueden poner en peligro el estado de salud e incrementar el riesgo a padecer una enfermedad. Aunque muchas enfermedades se han asociado a cambios en la microbiota intestinal, todavía se desconoce si dichas alteraciones son la causa o la consecuencia de las patologías. La enfermedad inflamatoria intestinal (EII) es una enfermedad inflamatoria crónica que se caracteriza por periodos de inflamación y constituye un problema

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3

Geerling, B. J. "Inflammatory bowel disease and nutrition." Maastricht : Maastricht : Universiteit Maastricht ; University Library, Maastricht University [Host], 1999. http://arno.unimaas.nl/show.cgi?fid=7216.

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4

Smith, Graeme Drummond. "Counselling in inflammatory bowel disease." Thesis, University of Edinburgh, 1997. http://hdl.handle.net/1842/12244.

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Introduction; The inflammatory bowel diseases (IBD), Crohn's disease (CD) and ulcerative colitis (UC), affect well over 100,000 people in the United Kingdom Health related quality of life (HRQOL) is influenced by many factors in IBD including; the nature and severity of the disease, socio-economic factors, age, psychological well-being as well as· the efficacy and complications of treatment. Pilot Studies; Quality of life was assessed in 140 IBD patients (70 CD/70 UC). Diarrhoea was, not surprisingly, the most commonly reported physical symptom in both CD and UC and impaired faecal continence

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5

Gustavsson, Anders. "Therapy in inflammatory bowel disease." Doctoral thesis, Örebro universitet, Institutionen för hälsovetenskap och medicin, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-25599.

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The aim of this thesis is to study treatment of inflammatory bowel disease with respect to an acute severe attack of ulcerative colitis and endoscopic balloon dilation in stricturing Crohn’s disease. A retrospective follow-up was made in 158 patients who were given intensive intravenous corticosteroid treatment due a severe, moderate, or mild attack of ulcerative colitis between 1975 and 1982. After 10 years, the colectomy frequency in the severe disease group was 64%, and 49% and28% in the moderate and mild groups, respectively. Severity of the original attack did not influence the subsequent

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6

Matini, Lawrence. "Adaptation to Inflammatory Bowel Disease." Thesis, University of Surrey, 2017. http://epubs.surrey.ac.uk/841456/.

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Inflammatory bowel disease (IBD) is a term used to describe two chronic diseases of the gastrointestinal tract: Ulcerative Colitis (UC) and Crohn's Disease (CD). Although the efficacy of treatment is continuously improving, Quality of Life (QoL) in this illness population remains low with many patients suffering from psychological and psychiatric comorbidities. Psychological interventions aimed at improving outcomes in these patients have largely demonstrated little improvement. This thesis argues that this may be the result of poor understanding of the experience of living with this condition

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7

Gerasimidis, Konstantinos. "Nutritional aspects and gut microbiota in paediatric inflammatory bowel disease." Thesis, Thesis restricted. Connect to e-thesis to view abstract, 2009. http://theses.gla.ac.uk/826/.

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Thesis (Ph.D.) - University of Glasgow, 2009.<br>Ph.D. thesis submitted to the Division of Developmental Medicine, Faculty of Medicine, University of Glasgow, 2009. Includes bibliographical references. Print version also available.

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8

Campbell, Simon Scott. "Azathioprine use in inflammatory bowel disease." Thesis, University of Edinburgh, 2003. http://hdl.handle.net/1842/24109.

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9

Ljung, Tryggve. "Nitric oxide in inflammatory bowel disease /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-602-2/.

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10

Parkes, Miles. "The genetics of inflammatory bowel disease." Thesis, University of Oxford, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326030.

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11

Dalton, Harry Richard. "Suppressive phenomena in inflammatory bowel disease." Thesis, University of Oxford, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.305970.

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12

Satsangi, Jack. "The genetics of inflammatory bowel disease." Thesis, University of Oxford, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337627.

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13

Eadala, Praveen. "Lactose sensitivity and inflammatory bowel disease." Thesis, Cardiff University, 2013. http://orca.cf.ac.uk/53992/.

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Controversy still exists as to the incidence, role and impact of lactose sensitivity in inflammatory bowel disease. The thesis shows that there is a higher than previously reported incidence of lactose sensitivity determined by a combination of genotype, breath test and symptoms after a lactose challenge. Lactose sensitivity in patients with inflammatory bowel disease who are in remission is 70%. There was no difference compared to healthy volunteers in terms of lactase genotyping; however there was a significantly greater prevalence of positive breath test and symptoms after lactose challenge

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14

Arsenescu, Razvan I. "NOVEL MECHANISMS IN INFLAMMATORY BOWEL DISEASE." UKnowledge, 2011. http://uknowledge.uky.edu/gradschool_diss/211.

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Inflammatory Bowel Diseases, Crohn's Disease and Ulcerative colitis, are idiopathic chronic conditions with multifactorial determinants. In general, terms, intestinal inflammation results from abnormal host-microbe interactions. Alterations in homeostasis involve host genetic factors, environmental cues and unique luminal microbial niches. We have examined the coordinated expressions of several molecular targets relevant to the mucosal immune system and identified signature biomarkers of IBD. Qualitative and quantitative changes in the composition of microbiota can be related to unique immuno-

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15

Rankin, B. J. "Colonic mucus in inflammatory bowel disease." Thesis, University of Newcastle Upon Tyne, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.320821.

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16

Hamlin, Peter John. "Genetic studies in inflammatory bowel disease." Thesis, University of Leeds, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.400166.

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17

Collins, Carole Elizabeth. "Platelet dysfunction in inflammatory bowel disease." Thesis, King's College London (University of London), 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.362539.

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18

Kamperidis, Nikolaos. "Nutrient effects in inflammatory bowel disease." Thesis, Queen Mary, University of London, 2016. http://qmro.qmul.ac.uk/xmlui/handle/123456789/23488.

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Background: Not only does IBD lead to nutritional deficiencies, but also nutrients influence its pathophysiology: exclusive enteral nutrition (EEN) is an effective primary treatment in Crohn's disease; and vitamin D (VitD) is involved in its pathogenesis and course. Aims: We hypothesised that nutrients impact on the course of IBD. We therefore studied the effect of EEN i) on long term clinical course in children; ii) on CD58, a costimulatory molecule at the intestinal epithelial cell (IEC) lines, iii) adults with Crohn's disease. We examined the possible effect of serum vitamin D levels on the

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19

Khodiyar, Varsha Kumari. "Microarray profiling of inflammatory bowel disease." Thesis, University of Leicester, 2002. http://hdl.handle.net/2381/29415.

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In this study of inflammatory bowel diseases (IBD, i.e. Crohn's disease and ulcerative colitis), the gene transcription profile of colonic IBD resection specimens were analysed by oligonucleotide microarray analysis. A total of 33,625 genes were profiled across 23 colonic mucosa samples; 5 involved Crohn's disease, 4 uninvolved Crohn's disease, 5 involved ulcerative colitis, 3 uninvolved ulcerative colitis and 6 samples from macroscopically normal areas of colorectal cancer resections (controls). A number of data-mining tools, encompassing clustering (e.g. hierarchical & K-means) and matrix-ba

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20

Johnston, Colette. "Metabolomic profiling in inflammatory bowel disease." Thesis, University of Manchester, 2014. https://www.research.manchester.ac.uk/portal/en/theses/metabolomic-profiling-in-inflammatory-bowel-disease(1eb7a48f-af12-4bd2-8497-ebad4eae0e4e).html.

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Introduction: Inflammatory bowel disease is a common, complex relapsing disorder characterised by immune dysregulation, altered intestinal permeability and microbial insult. Limited knowledge is available regarding the metabolic changes observed during progression of the disease, and limited biomarkers of disease available that have been validated and shown to be of sound clinical value. Aim of Study: A two stage metabolomics approach was adopted to determine if metabolic signature profiles, could distinguish inflammatory bowel disease Crohn’s disease (CD) patients from ulcerative colitis (UC)

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21

Hildebrand, Diane Rosemary. "Metabolomic profiling in inflammatory bowel disease." Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/28850.

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Introduction Inflammatory bowel disease (IBD) is a chronic gastrointestinal disorder that encompasses two major subtypes; Crohn’s Disease (CD) and Ulcerative Colitis (UC). Our knowledge regarding disease pathogesis is rapidly increasing. However, these disease entities provide challenges in diagnosis, monitoring of disease activity and assessing individual response to treatment, because there is a lack of validated clinical biomarkers. Metabolomics involves the study of numerous analytes that have very diverse physical and chemical properties and occur in a wide concentration range. Early evid

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22

Tsaprouni, Loukia G. "Histone acetylation and inflammatory mediators in inflammatory bowel disease." Thesis, University of Bedfordshire, 2003. http://hdl.handle.net/10547/620761.

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During cell activation the tightly compacted DNA is made available to DNA-binding proteins allowing the induction of gene transcription. In the resting cell, DNA is packaged into chromatin whose fundamental subunit is the nucleosome, composed of an octamer of four core histones (H) 3, 4, 2A and 2B. During the induction of gene transcription, modification of histones, by acetylation, methylation etc., results in unwinding of the DNA, permitting access of large DNAbinding proteins, such as RNA polymerase II, and subsequent induction of gene transcription. This investigation initially examined th

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23

Bresso, Francesca. "Genetic and molecular determinants in inflammatory bowel disease /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-982-3/.

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24

Bhatti, Mansoor Ahmad. "A study of pro inflammatory mechanisms in inflammatory bowel disease." Thesis, Imperial College London, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.249258.

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25

Salim, Sa'ad Yislam. "Mucosal dendritic cells in inflammatory bowel disease." Doctoral thesis, Linköpings universitet, Kirurgi, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-52234.

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Crohn's disease, a chronic inflammation of the bowel, is a multi-factorial condition where uncontrolled immune responses to luminal bacteria occur in genetically predisposed individuals. The first observable clinical signs are small ulcers that form at a specialised form of epithelium, follicle-associated epithelium (FAB). The FAB covers immune inductive sites, Peyer's patches, which function primarily as sensory areas that sample the externaI gut environment. Dendritic cells are one of the key cells that are involved in sensing luminal contents and orchestrating the gut immune system. The mai

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26

Ingram, John Robert. "Topical nicotine therapy for inflammatory bowel disease." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.491900.

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This thesis covers several areas which are related to the potential therapeutic effect of topical nicotine in ulcerative colitis (UC) and Crohn's colitis. It also examines a possible mechanism which may be pertinent to its mode of action. Transdermal nicotine is of benefit for active UC but adverse events (AEs) are frequent and limit its use. This prompted development of topical delivery systems which made direct application of nicotine to the colonic mucosa possible. Nicotine liquid enemas and delayed-release oral preparations produce lower systemic blood levels of nicotine and initial work w

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27

Irving, Peter Miles. "Platelet-leucocyte aggregates in inflammatory bowel disease." Thesis, Queen Mary, University of London, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.441946.

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28

Rahman, Arman. "Defensins and cytokines in inflammatory bowel disease." Doctoral thesis, Umeå : Univ, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1377.

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29

Simmonds, Nicola Jane. "Reactive oxygen metabolites and inflammatory bowel disease." Thesis, University of Oxford, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317911.

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30

Radford-Smith, Graham Lindsay. "Cytokine gene expression in inflammatory bowel disease." Thesis, University of Oxford, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.296991.

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31

van, Heel David Alexander. "Identification of inflammatory bowel disease susceptibility genes." Thesis, University of Oxford, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.249199.

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32

Williams, Horace Richard Timothy. "Urinary metabolic profiling in inflammatory bowel disease." Thesis, Imperial College London, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.508479.

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33

Cornish, Julie Ann. "Inflammatory bowel disease & female reproductive health." Thesis, Imperial College London, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.539280.

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34

Andreoletti, Gaia. "Genomic analyses of paediatric inflammatory bowel disease." Thesis, University of Southampton, 2016. https://eprints.soton.ac.uk/403112/.

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35

Elding, H. "Dissecting the genetics of Inflammatory Bowel Disease." Thesis, University College London (University of London), 2015. http://discovery.ucl.ac.uk/1464062/.

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Inflammatory Bowel Disease (IBD) can be classified into two main subtypes: Crohn’s Disease (CD) and Ulcerative Colitis (UC). The aim of this study is to identify the genetic contribution to the susceptibility to IBD. In the first part of the study, I focused on Crohn’s Disease, the subtype that shows the greatest heritability. Using both pooled and sub-phenotype data, followed by replication, the results reveal substantial genetic heterogeneity and the total number of confirmed CD susceptibility loci was increased from 71 published by others to 200. This was achieved by analyzing the data usin

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36

Armentrout, Paige L. "Exploring Dietary Patterns in Inflammatory Bowel Disease." The Ohio State University, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1555379444416158.

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37

Wong, L. H. J. "Optimising thiopurine therapy in inflammatory bowel disease." Thesis, Exeter and Plymouth Peninsula Medical School, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701322.

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38

Lee, Cheng Hiang. "Paediatric Inflammatory Bowel Disease: Epidemiology and Immunopathogenesis." Thesis, University of Sydney, 2020. https://hdl.handle.net/2123/24013.

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IBD is a chronic inflammatory disorder of the gut where the exact pathogenesis is not fully understood. The thesis makes important contributions to knowledge in this area. It presents previously unavailable information on the incidence of PIBD in NSW from 1968 to 2013. It also presents findings from an investigation of the immunopathogenesis of IBD based on 2 studies of monogenic IBD and lymphocyte immunophenotyping. The incidence of PIBD in NSW in 2013 was estimated to be 5.32 per 100,000 children, which represents a nearly 30-fold increase over the past 5 decades. This finding is consisten

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39

MURGIA, ANTONIO. "INFLAMMATORY BOWEL DISEASE STUDY: A METABOLOMICS APPROACH." Doctoral thesis, Università degli Studi di Cagliari, 2018. http://hdl.handle.net/11584/255960.

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Inflammatory bowel disease represents a group of chronic disorders that affect one or more parts of the intestine. Although recently the incidence of inflammatory bowel disease has noticeably increased, its aetiology is still unclear. No specific pathogen has been defined as a causative agent. Serological biomarkers have been recently proposed for diagnosis, but they remain untested in clinical applications. Moreover, current diagnostic and monitoring practices for inflammatory bowel disease are very invasive. Therefore, accurate tools for the early diagnosis, and in particular non-invasive st

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40

Berrill, James. "Symptoms of irritable bowel syndrome in patients with inflammatory bowel disease." Thesis, Cardiff University, 2014. http://orca.cf.ac.uk/61734/.

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Irritable bowel syndrome (IBS) and inflammatory bowel disease (IBD) are both chronic relapsing intestinal disorders. Their symptom profiles overlap in terms of abdominal discomfort and altered bowel habit. Meta-‐analysis of patients with IBD demonstrates that 25-‐46% of those in clinical remission have symptoms compatible with IBS. These patients report lower quality of life scores compared to their asymptomatic counterparts. There is uncertainty as to the cause of these symptoms, and concern for the influence they may exert on clinical management. The work described in this thesis investiga

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41

Weersma, Rinse Karel. "Genetic susceptibility for inflammatory bowel diseases." [S.l. : Groningen : s.n. ; University Library Groningen] [Host], 2007. http://irs.ub.rug.nl/ppn/304870935.

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42

Kalla, Rahul. "Biomarker discovery in inflammatory bowel diseases." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/31040.

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There is an unmet need for novel biomarker discovery in Inflammatory Bowel Diseases (IBD) to aid clinical management in several clinical settings including diagnosis and prognosis. With an ever-advancing repertoire of biological therapies on the horizon, it is important to personalise treatments at an early stage. The aim of this thesis is to explore the clinical utility of novel blood-based biomarkers in diagnosis, disease classification and prognosis in 2 cohorts: newly diagnosed IBD and acute severe colitis. Investigating the circulating methylome, 290 probes exhibited Holm significant IBD-

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43

Neilly, Paul John David. "Inflammatory mediators and amino acid therapy in experimental inflammatory bowel disease." Thesis, Queen's University Belfast, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.361291.

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44

Halfvarson, Jonas. "Inflammatory bowel disease in twins : studies of genetics and environmental factors /." Linköping : Univ, 2005. http://www.bibl.liu.se/liupubl/disp/disp2005/med909s.pdf.

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45

Schreiber, Olof. "Microcirculation, Mucus and Microbiota in Inflammatory Bowel Disease." Doctoral thesis, Uppsala universitet, Integrativ Fysiologi, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-112718.

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Inflammatory bowel diseases, (IBD), are a group of chronic disorders of the gastro-intestinal tract, and include Crohn’s disease (CD) and Ulcerative Colitis (UC). The pathogenesis is not known, but involves at least in part a loss of tolerance towards the commensal colonic microbiota. In this thesis, we show in animal models of CD and UC that the colonic mucosal blood flow increased compared to healthy animals. This blood flow increase is due to an up regulation of endothelial nitric oxide synthase (NOS). Further, we show in the UC model that the thickness of the firmly adherent colonic mucus

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46

Heikius, B. (Bengt). "Pancreatic and hepatobiliary disorders in inflammatory bowel disease." Doctoral thesis, University of Oulu, 2000. http://urn.fi/urn:isbn:9514257561.

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Abstract Extraintestinal manifestations in inflammatory bowel disease (IBD) have been described with varying frequencies. The aim of this study was to estimate the prevalence of pancreatic duct abnormalities, exocrine and endocrine dysfunction, elevated pancreatic enzymes, hepato-biliary disease, coexisting cholangiographic and pancreatographic duct changes, and elevated serum levels of fibrosis markers in IBD, and to correlate the findings with clinical, endoscopic and histologic variables. From a local patient register, 237 patients were randomly selected and studied. Of these, 170 had ulce

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47

Hann, Bradley M. "Colonic epithelial cell apoptosis in inflammatory bowel disease." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/MQ37955.pdf.

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48

Bernard, Nicole Kristine. "Glucagon-like peptide 2 and inflammatory bowel disease." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0016/MQ49596.pdf.

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49

Onnie, Clive Morris. "Genetic determinants of susceptibility to inflammatory bowel disease." Thesis, King's College London (University of London), 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.485696.

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The aim of this thesis was to advance the current knowledge of the genetic determinants ofsusceptibility to inflammatory bowel disease (IBD), and examine their effect on disease phenotype. A DNA source from 1071 well clinically characterized patients with IBD disease (644 Crohn's Disease (CD), 427 ulcerative colitis (UCÃ‚Â» was established. This was used both alone and in combination with an existing IBD collection to investigate candidate susceptibility genes as well as the relationship between genotype and phenotype. . The previously reported association ofthe IBD5 locus with CD was investig

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50

Cook, W. B. "Iron absorption in health and inflammatory bowel disease." Thesis, University of Cambridge, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.597923.

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Chapter 1 provides a general introduction while Chapter 2 investigates the relationships between dietary iron intake, disease activity and quality of life (QOL) in patients with IBD. Results indicated that non-haem iron intake was significantly associated with iron requirements for IBD patients but not healthy controls. Interestingly, for iron replete IBD patients, a significant positive correlation between iron intake and disease activity was noted. Correlation between QOL and iron intake was also observed. Chapter 3 investigated the acute effects of a single oral dose of ferrous sulphate on

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