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Artículos de revistas sobre el tema "Intracranial blood volume waves"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 1 de febrero de 2022

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1

Hayashi, Minoru, Hidenori Kobayashi, Yuji Handa, Hirokazu Kawano, and Masanori Kabuto. "Brain blood volume and blood flow in patients with plateau waves." Journal of Neurosurgery 63, no. 4 (1985): 556–61. http://dx.doi.org/10.3171/jns.1985.63.4.0556.

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✓ Plateau waves, characterized by acute transient rises of the intracranial pressure (ICP), are accompanied by a marked decrease of the cerebral perfusion pressure. Patients with plateau waves, however, often show no clinical symptoms of ischemia of the brain stem, such as vasopressor response or impairment of consciousness during the waves. The authors studied brain blood volume and blood flow with dynamic computerized tomography using rapid-sequence scanning in patients with plateau waves identified during continuous ICP recording. Following an intravenous bolus injection of contrast medium,

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2

Czosnyka, Marek, Piotr Smielewski, Stefan Piechnik, et al. "Hemodynamic characterization of intracranial pressure plateau waves in head-injured patients." Journal of Neurosurgery 91, no. 1 (1999): 11–19. http://dx.doi.org/10.3171/jns.1999.91.1.0011.

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Object. Plateau waves of intracranial pressure (ICP) are often recorded during intensive care monitoring of severely head injured patients. They are traditionally interpreted as meaningful secondary brain insults because of the dramatic decrease in cerebral perfusion pressure (CPP). The aim of this study was to investigate both the hemodynamic profile and the clinical consequences of plateau waves.Methods. One hundred sixty head-injured patients were studied using continuous monitoring of ICP; almost 20% of these patients exhibited plateau waves. In 96 patients arterial pressure, ICP, and tran

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3

Ursino, Mauro, and Carlo Alberto Lodi. "A simple mathematical model of the interaction between intracranial pressure and cerebral hemodynamics." Journal of Applied Physiology 82, no. 4 (1997): 1256–69. http://dx.doi.org/10.1152/jappl.1997.82.4.1256.

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Ursino, Mauro, and Carlo Alberto Lodi. A simple mathematical model of the interaction between intracranial pressure and cerebral hemodynamics. J. Appl. Physiol. 82(4): 1256–1269, 1997.—A simple mathematical model of intracranial pressure (ICP) dynamics oriented to clinical practice is presented. It includes the hemodynamics of the arterial-arteriolar cerebrovascular bed, cerebrospinal fluid (CSF) production and reabsorption processes, the nonlinear pressure-volume relationship of the craniospinal compartment, and a Starling resistor mechanism for the cerebral veins. Moreover, arterioles are co

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4

Hayashi, Minoru, Hisamasa Ishii, Yuji Handa, Hidenori Kobayashi, Hirokazu Kawano, and Masanori Kabuto. "Role of the medulla oblongata in plateau-wave development in dogs." Journal of Neurosurgery 67, no. 1 (1987): 97–101. http://dx.doi.org/10.3171/jns.1987.67.1.0097.

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✓ Plateau waves reflect both dilatation of the cerebral vessels and an increase in the cerebral blood volume under increased intracranial pressure (ICP). They are often associated with changes in arterial blood pressure (BP) and respiration, suggesting a role of the brain stem in their development. In experiments conducted on dogs in which intracranial hypertension was induced by occluding the neck veins, the authors stimulated the brain-stem reticular formation in the medulla oblongata and caudal pons to identify the brain sites that produce plateau-like responses. A rise in ICP was observed

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5

Beqiri, Erta, Marek Czosnyka, Afroditi D. Lalou, et al. "Influence of mild-moderate hypocapnia on intracranial pressure slow waves activity in TBI." Acta Neurochirurgica 162, no. 2 (2019): 345–56. http://dx.doi.org/10.1007/s00701-019-04118-6.

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Abstract Background In traumatic brain injury (TBI) the patterns of intracranial pressure (ICP) waveforms may reflect pathological processes that ultimately lead to unfavorable outcome. In particular, ICP slow waves (sw) (0.005–0.05 Hz) magnitude and complexity have been shown to have positive association with favorable outcome. Mild-moderate hypocapnia is currently used for short periods to treat critical elevations in ICP. Our goals were to assess changes in the ICP sw activity occurring following sudden onset of mild-moderate hypocapnia and to examine the relationship between changes in ICP

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6

Austin, George M., George M. Austin, Wouter Schievink, and Richard Williams. "Controlled Pressure-Volume Factors in the Enlargement of Intracranial Aneurysms." Neurosurgery 24, no. 5 (1989): 722–30. http://dx.doi.org/10.1227/00006123-198905000-00011.

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ABSTRACT Pressure-volume relations were investigated on a model aneurysm wall made of elastic tissue and collagen. The model wall had a Young's Modulus of 2 × 107 dynes/cm2, approximating the elastance of fresh aneurysm walls obtained at autopsy. The model wall was fixed over the top of a glass T-tube, 6 mm in diameter. Pressure pulse waves of water or outdated human blood entered at the bottom of the T-tube and exited by way of a controlled resistance, while pressure was monitored by a strain gauge and recorded on an ink writer from the other arm. Incremental increases in systolic pressure pr

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7

Luciano, Mark G., Stephen M. Dombrowski, Sara Qvarlander, et al. "Novel method for dynamic control of intracranial pressure." Journal of Neurosurgery 126, no. 5 (2017): 1629–40. http://dx.doi.org/10.3171/2016.4.jns152457.

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OBJECTIntracranial pressure (ICP) pulsations are generally considered a passive result of the pulsatility of blood flow. Active experimental modification of ICP pulsations would allow investigation of potential active effects on blood and CSF flow and potentially create a new platform for the treatment of acute and chronic low blood flow states as well as a method of CSF substance clearance and delivery. This study presents a novel method and device for altering the ICP waveform via cardiac-gated volume changes.METHODSThe novel device used in this experiment (named Cadence) consists of a small

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8

Schmidt, Bernhard, Marek Czosnyka, Jens Jürgen Schwarze, et al. "Cerebral Vasodilatation Causing Acute Intracranial Hypertension: A Method for Noninvasive Assessment." Journal of Cerebral Blood Flow & Metabolism 19, no. 9 (1999): 990–96. http://dx.doi.org/10.1097/00004647-199909000-00006.

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Deep spontaneous vasodilatatory events are frequently recorded in various cerebral diseases, causing dramatic increases (A-waves) in intracranial pressure (ICP) and subsequently provoking ischemic brain insults, The relationship between fluctuations in CBF, ICP, and arterial blood pressure (ABP) is influenced by properties of cerebrovascular control mechanisms and the cerebrospinal pressure-volume compensation, The goal of this study was to construct a mathematical model of this relationship and to assess its ability to predict the occurrence and time course of A-waves, A group of 17 severely

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9

Ursino, Mauro, and Patrizia Di Giammarco. "A mathematical model of the relationship between cerebral blood volume and intracranial pressure changes: The generation of plateau waves." Annals of Biomedical Engineering 19, no. 1 (1991): 15–42. http://dx.doi.org/10.1007/bf02368459.

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10

Langvatn, Erlend Aambø, Radek Frič, Bernt J. Due-Tønnessen, and Per Kristian Eide. "Intracranial volume versus static and pulsatile intracranial pressure values in children with craniosynostosis." Journal of Neurosurgery: Pediatrics 24, no. 1 (2019): 66–74. http://dx.doi.org/10.3171/2019.2.peds18767.

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OBJECTIVEReduced intracranial volume (ICV) and raised intracranial pressure (ICP) are assumed to be principal pathophysiological mechanisms in childhood craniosynostosis. This study examined the association between ICV and ICP and whether ICV can be used to estimate the ICP.METHODSThe authors analyzed ICV and ICP measurements from children with craniosynostosis without concurrent hydrocephalus and from age-matched individuals without craniosynostosis who underwent diagnostic ICP measurement.RESULTSThe study included 19 children with craniosynostosis (mean age 2.2 ± 1.9 years) and 12 reference

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11

Strangman, Gary E., Quan Zhang, Karina Marshall-Goebel, et al. "Increased cerebral blood volume pulsatility during head-down tilt with elevated carbon dioxide: the SPACECOT Study." Journal of Applied Physiology 123, no. 1 (2017): 62–70. http://dx.doi.org/10.1152/japplphysiol.00947.2016.

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Astronauts aboard the International Space Station (ISS) have exhibited hyperopic shifts, posterior eye globe flattening, dilated optic nerve sheaths, and even optic disk swelling from spaceflight. Elevated intracranial pressure (ICP) consequent to cephalad fluid shifts is commonly hypothesized as contributing to these ocular changes. Head-down tilt (HDT) is frequently utilized as an Earth-based analog to study similar fluid shifts. Sealed environments like the ISS also exhibit elevated CO2, a potent arteriolar vasodilator that could further affect cerebral blood volume (CBV) and cerebral blood

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12

Imberti, Roberto, Marinella Fuardo, Guido Bellinzona, Michele Pagani, and Martin Langer. "The use of indomethacin in the treatment of plateau waves: effects on cerebral perfusion and oxygenation." Journal of Neurosurgery 102, no. 3 (2005): 455–59. http://dx.doi.org/10.3171/jns.2005.102.3.0455.

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Object. Plateau waves are sudden and steep increases in intracranial pressure (ICP) that can develop in patients with cerebral injuries, reduced pressure—volume compensatory reserve, and preserved autoregulation. They are caused by cerebral vasodilation in response to a reduction in cerebral perfusion and are associated with increased cerebral blood volume and reduced cerebral blood flow. The authors evaluated the hypothesis that administration of indomethacin, a potent cerebral arteriolar vasoconstrictor, could interrupt the vicious cycle that occurs during plateau waves, extinguishing these

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13

Bajpai, Basant K., Rolandas Zakelis, Mantas Deimantavicius, and Daiva Imbrasiene. "Comparative Study of Novel Noninvasive Cerebral Autoregulation Volumetric Reactivity Indices Reflected by Ultrasonic Speed and Attenuation as Dynamic Measurements in the Human Brain." Brain Sciences 10, no. 4 (2020): 205. http://dx.doi.org/10.3390/brainsci10040205.

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This is a comparative study of two novel noninvasive cerebrovascular autoregulation (CA) monitoring methods based on intracranial blood volume (IBV) changes in the human brain. We investigated the clinical applicability of the new volumetric reactivity index (VRx2), reflected by intracranial ultrasonic attenuation dynamics for noninvasive CA monitoring. The CA was determined noninvasively on 43 healthy participants by calculating the volumetric reactivity index (VRx1 from time-of-flight of ultrasound, VRx2 from attenuation of ultrasound). The VRx was calculated as a moving correlation coeffici

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14

Varsos, Georgios V., Melissa C. Werndle, Zofia H. Czosnyka, et al. "Intraspinal pressure and spinal cord perfusion pressure after spinal cord injury: an observational study." Journal of Neurosurgery: Spine 23, no. 6 (2015): 763–71. http://dx.doi.org/10.3171/2015.3.spine14870.

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OBJECT In contrast to intracranial pressure (ICP) in traumatic brain injury (TBI), intraspinal pressure (ISP) after traumatic spinal cord injury (TSCI) has not received the same attention in terms of waveform analysis. Based on a recently introduced technique for continuous monitoring of ISP, here the morphological characteristics of ISP are observationally described. It was hypothesized that the waveform analysis method used to assess ICP could be similarly applied to ISP. METHODS Data included continuous recordings of ISP and arterial blood pressure (ABP) in 18 patients with severe TSCI. RES

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15

Cheng, Chun-Yu, Hao-Min Cheng, Shih-Pin Chen, et al. "White matter hyperintensities in migraine: Clinical significance and central pulsatile hemodynamic correlates." Cephalalgia 38, no. 7 (2017): 1225–36. http://dx.doi.org/10.1177/0333102417728751.

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Background The role of central pulsatile hemodynamics in the pathogenesis of white matter hyperintensities in migraine patients has not been clarified. Methods Sixty patients with migraine (20–50 years old; women, 68%) without overt vascular risk factors and 30 demographically-matched healthy controls were recruited prospectively. Cerebral white matter hyperintensities volume was determined by T1-weighted magnetic resonance imaging with CUBE-fluid-attenuated-inversion-recovery sequences. Central systolic blood pressure, carotid-femoral pulse wave velocity, and carotid augmentation index were m

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16

Rossiti, Sandro, and Reinhard Volkmann. "Changes of blood flow velocity indicating mechanical compression of the vertebral arteries during rotation of the head in the normal human measured with transcranial Doppler sonography." Arquivos de Neuro-Psiquiatria 53, no. 1 (1995): 26–33. http://dx.doi.org/10.1590/s0004-282x1995000100005.

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The dynamical changes of blood flow velocity of the intracranial vertebral arteries (VA's) and proximal basilar artery (BA) provoked by rotation of the head in normal volunteers were measured using pulsed-wave transcranial Doppler sonography (TCD). In another group both VA's were examined simultaneously with 2-channel TCD. Blood flow velocities diminished compared to the neutral position in all vessels, independently of die side. Total obstruction of the flow was not observed. Our findings reveal a definitive decrease of blood flow velocity at the vertebrobasilar artery system provoked by rota

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17

Meyerson, B. A., L. Gunasekera, B. Linderoth, and B. Gazelius. "Bedside Monitoring of Regionald Cortical Blood Flow in Comatose Patients Using Laser Doppler Flowmetry." Neurosurgery 29, no. 5 (1991): 750–55. http://dx.doi.org/10.1227/00006123-199111000-00018.

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Abstract Continuous bedside monitoring of regional cerebrocortical blood flow was carried out using laser Doppler flowmetry (LDF) in four comatose patients. Newly designed thin and flexible optical probes that were easily implanted into the cortex were employed. When the patients were in a deep comatose state, the recordings revealed a conspicuous relationship to intracranial pressure. As the patients improved, the flux values gradually increased, and the recordings showed characteristic changes in the wave pattern, presumably related to the restoration of an impaired cerebral autoregulation.

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18

Hanigan, William C., and Sarah N. Zallek. "Headaches, Shunts, and Obstructive Sleep Apnea: Report of Two Cases." Neurosurgery 54, no. 3 (2004): 764–69. http://dx.doi.org/10.1227/01.neu.0000109539.32277.17.

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Abstract OBJECTIVE This report describes two shunted patients evaluated with continuous intracranial pressure (ICP) monitors for worsening headaches and subsequently diagnosed with obstructive sleep apnea. CLINICAL PRESENTATION AND INTERVENTION ICPs were monitored with strain-gauge sensors inserted into the frontal cortex. After the initial diagnosis of sleep apnea, 8-hour attended polysomnography was performed in each patient. Both patients showed apnea-hypopnea indices greater than 15. Consequently, a “split-night study” was performed to evaluate treatment with titrated nasal continuous posi

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19

Kirkeby, O. J., J. R. Pettersen, K. Ekseth, and I. R. Rise. "Colloidal Blood Volume Expansion During High Intracranial Pressure." Acta Neurochirurgica 141, no. 1 (1999): 37–44. http://dx.doi.org/10.1007/s007010050264.

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20

Newell, David W., Rune Aaslid, Renate Stooss, and Hans J. Reulen. "The relationship of blood flow velocity fluctuations to intracranial pressure B waves." Journal of Neurosurgery 76, no. 3 (1992): 415–21. http://dx.doi.org/10.3171/jns.1992.76.3.0415.

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✓ Intracranial pressure (ICP) and continuous transcranial Doppler ultrasound signals were monitored in 20 head-injured patients and simultaneous synchronous fluctuations of middle cerebral artery (MCA) velocity and B waves of the ICP were observed. Continuous simultaneous monitoring of MCA velocity, ICP, arterial blood pressure, and expired CO2 revealed that both velocity waves and B waves occurred despite a constant CO2 concentration in ventilated patients and were usually not accompanied by fluctuations in the arterial blood pressure. Additional recordings from the extracranial carotid arter

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21

Bazyar, Soha, Joana Ramalho, Cihat Eldeniz, Hongyu An, and Yueh Z. Lee. "Comparison of Cerebral Blood Volume and Plasma Volume in Untreated Intracranial Tumors." PLOS ONE 11, no. 9 (2016): e0161807. http://dx.doi.org/10.1371/journal.pone.0161807.

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22

Cardoso, Erico R., Kesava Reddy, and Deepak Bose. "Effect of subarachnoid hemorrhage on intracranial pulse waves in cats." Journal of Neurosurgery 69, no. 5 (1988): 712–18. http://dx.doi.org/10.3171/jns.1988.69.5.0712.

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✓ The influence of vasoconstrictors of intracranial arteries on the amplitude and configuration of the intracranial pulse wave (ICPW) was investigated. Continuous pressure recordings from the descending aorta (systemic arterial pressure) and the third cerebral ventricle (intracranial pressure) were obtained from anesthetized cats. Computerized analysis of the configuration, amplitude, and frequency spectrum of ventricular wave (ICPW) and aortic pulse wave (SAPW) was performed. Artificial cerebrospinal fluid (CSF), blood, or 5-hydroxytryptamine (5-HT) was injected intracisternally. In 24 contro

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23

Cammermeyer, Margarethe. "Brain Blood Volume and Blood Flow in Patients with Plateau Waves." Journal of Neuroscience Nursing 18, no. 2 (1986): 100. http://dx.doi.org/10.1097/01376517-198604000-00017.

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24

Barie, Philip S., Jamshid B. G. Ghajar, Andrew D. Firlik, Victor A. Chang, and Robert J. Hariri. "CONTRIBUTION OF INCREASED CEREBRAL BLOOD VOLUME TO POSTTRAUMATIC INTRACRANIAL HYPERTENSION." Journal of Trauma: Injury, Infection, and Critical Care 35, no. 1 (1993): 88–96. http://dx.doi.org/10.1097/00005373-199307000-00015.

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25

van Westen, Danielle, Esben T. Petersen, Ronnie Wirestam, et al. "Correlation between arterial blood volume obtained by arterial spin labelling and cerebral blood volume in intracranial tumours." Magnetic Resonance Materials in Physics, Biology and Medicine 24, no. 4 (2011): 211–23. http://dx.doi.org/10.1007/s10334-011-0255-x.

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26

Bodo, Michael. "Studies in Rheoencephalography (REG)." Journal of Electrical Bioimpedance 1, no. 1 (2019): 18–40. http://dx.doi.org/10.5617/jeb.109.

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Abstract This article presents an overview of rheoencephalography (REG) – electrical impedance measurements of the brain – and summarizes past and ongoing research to develop medical applications of REG for neuro-critical care and for primary prevention of stroke and cardiovascular disease. The availability of advanced electronics and computation has opened up the potential for use of REG technology as a noninvasive, continuous and inexpensive brain monitor for military and civilian applications. The clinical background information presented here introduces physiological and clinical environme

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27

Nowaczewska, Magdalena, and Henryk Kaźmierczak. "Cerebral Blood Flow in Low Intracranial Pressure Headaches—What Is Known?" Brain Sciences 10, no. 1 (2019): 2. http://dx.doi.org/10.3390/brainsci10010002.

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Headaches attributed to low cerebrospinal fluid (CSF) pressure are described as orthostatic headaches caused by spontaneous or secondary low CSF pressure or CSF leakages. Regardless of the cause, CFS leaks may lead to intracranial hypotension (IH) and influence cerebral blood flow (CBF). When CSF volume decreases, a compensative increase in intracranial blood volume and cerebral vasodilatation occurs. Sinking of the brain and traction on pain-sensitive structures are thought to be the causes of orthostatic headaches. Although there are many studies concerning CBF during intracranial hypertensi

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28

Sawicki, Karol, Michał M. Placek, Tomasz Łysoń, Zenon Mariak, Robert Chrzanowski, and Marek Czosnyka. "Change in Blood Flow Velocity Pulse Waveform during Plateau Waves of Intracranial Pressure." Brain Sciences 11, no. 8 (2021): 1000. http://dx.doi.org/10.3390/brainsci11081000.

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A reliable method for non-invasive detection of dangerous intracranial pressure (ICP) elevations is still unavailable. In this preliminary study, we investigate quantitatively our observation that superimposing waveforms of transcranial Doppler blood flow velocity (FV) and arterial blood pressure (ABP) may help in non-invasive identification of ICP plateau waves. Recordings of FV, ABP and ICP in 160 patients with severe head injury (treated in the Neurocritical Care Unit at Addenbrookes Hospital, Cambridge, UK) were reviewed retrospectively. From that cohort, we identified 18 plateau waves reg

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29

Takagi, Toshinori, Takahiko Imai, Keisuke Mishiro, et al. "Cilostazol ameliorates collagenase-induced cerebral hemorrhage by protecting the blood–brain barrier." Journal of Cerebral Blood Flow & Metabolism 37, no. 1 (2016): 123–39. http://dx.doi.org/10.1177/0271678x15621499.

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Intracranial hemorrhage remains a devastating disease. Among antiplatelet drugs, cilostazol, a phosphodiesterase 3 inhibitor, was recently reported to prevent secondary hemorrhagic stroke in patients in a clinical trial. The aim of this study was to evaluate whether pre-treatment with cilostazol could decrease the intracranial hemorrhage volume and examine the protective mechanisms of cilostazol. We evaluated the pre-treatment effects of the antiplatelet drug cilostazol on the collagenase-induced intracranial hemorrhage volume and neurological outcomes in mice. To estimate the mechanism of col

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Artru, Alan A. "Reduction of Cerebrospinal Fluid Pressure by Hypocapnia: Changes in Cerebral Blood Volume, Cerebrospinal Fluid Volume, and Brain Tissue Water and Electrolytes." Journal of Cerebral Blood Flow & Metabolism 7, no. 4 (1987): 471–79. http://dx.doi.org/10.1038/jcbfm.1987.90.

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The study examined the role of cerebral blood volume (CBV), cerebrospinal fluid (CSF) volume, and brain tissue water and electrolytes on CSF pressure during 4 h of hypocapnia in dogs, Group I (n = 6) was examined during hypocapnia (PaCO2 20 mm Hg), with no intracranial mass being present Group II (n = 6) was examined with an intracranial mass present (epidural balloon, CSF pressure 35 cm H2O), but no hypocapnia. In group III (n = 6), an intracranial mass was present, and hypocapnia was used to lower CSF pressure. In group I, hypocapnia initially reduced CBV from 3.4 to 2.4 ml. With continued h

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31

Barie, Philip S., Jamshid B. G. Ghajar, and Robert J. Hariri. "CONTRIBUTION OF INCREASED CEREBRAL BLOOD VOLUME TO POST-TRAUMATIC INTRACRANIAL HYPERTENSION." Journal of Trauma: Injury, Infection, and Critical Care 33, no. 1 (1992): 148. http://dx.doi.org/10.1097/00005373-199207000-00029.

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32

Kirkeby, Ole J., Ingunn R. Rise, Lars Nordsletten, Sigmund Skjeldal, and Cecilie Risøe. "Cardiovascular response to blood loss during high intracranial pressure." Journal of Neurosurgery 83, no. 6 (1995): 1067–71. http://dx.doi.org/10.3171/jns.1995.83.6.1067.

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✓ The authors hypothesized that the combination of hemorrhage and increased intracranial pressure (ICP) has deleterious effects on cardiovascular function. The effect of blood loss during normal and increased ICP was studied in eight pigs. The mean arterial pressure (MAP), pulmonary arterial pressure, pulmonary capillary wedge pressure, cardiac output, and cerebrospinal fluid (CSF) pressure were measured. The regional tissue blood flow was determined with radioactive microspheres labeled with four different nuclides. High ICP (80% of MAP) was induced by infusion of artificial CSF into the cist

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Rosengarten, Bernhard, Damian Rüskes, Irene Mendes, and Erwin Stolz. "A sudden arterial blood pressure decrease is compensated by an increase in intracranial blood volume." Journal of Neurology 249, no. 5 (2002): 538–41. http://dx.doi.org/10.1007/s004150200061.

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Svedung Wettervik, Teodor, Timothy Howells, Per Enblad, and Anders Lewén. "Intracranial pressure variability: relation to clinical outcome, intracranial pressure–volume index, cerebrovascular reactivity and blood pressure variability." Journal of Clinical Monitoring and Computing 34, no. 4 (2019): 733–41. http://dx.doi.org/10.1007/s10877-019-00387-9.

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OHNO, Y. "Increased intracranial blood flow volume in a preeclamptic woman with postpartum photophobia." Obstetrics & Gynecology 101, no. 5 (2003): 1082–84. http://dx.doi.org/10.1016/s0029-7844(02)02252-4.

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Ohno, Yasumasa, Kumi Iwanaga, Atsuo Itakura, and Shigehiko Mizutani. "Increased Intracranial Blood Flow Volume in a Preeclamptic Woman With Postpartum Photophobia." Obstetrics & Gynecology 101, Supplement (2003): 1082–84. http://dx.doi.org/10.1097/00006250-200305001-00013.

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37

Chambers, I. R., G. Daubaris, E. Jarzemskas, et al. "The clinical application of non-invasive intracranial blood volume pulse wave monitoring." Physiological Measurement 26, no. 6 (2005): 1019–32. http://dx.doi.org/10.1088/0967-3334/26/6/011.

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Lewis, Philip M., Peter Smielewski, Jeffrey V. Rosenfeld, John D. Pickard, and Marek Czosnyka. "A Continuous Correlation Between Intracranial Pressure and Cerebral Blood Flow Velocity Reflects Cerebral Autoregulation Impairment During Intracranial Pressure Plateau Waves." Neurocritical Care 21, no. 3 (2014): 514–25. http://dx.doi.org/10.1007/s12028-014-9994-7.

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Artru, Alan A. "Reduction of Cerebrospinal Fluid Pressure by Hypocapnia: Changes in Cerebral Blood Volume, Cerebrospinal Fluid Volume and Brain Tissue Water and Electrolytes. II. Effects of Anesthetics." Journal of Cerebral Blood Flow & Metabolism 8, no. 5 (1988): 750–56. http://dx.doi.org/10.1038/jcbfm.1988.123.

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Part I of these studies (Artru, 1987) examined how cerebral blood volume (CBV), CSF volume, and brain tissue water and electrolytes determined CSF pressure during 4 h of hypocapnia in sedated dogs. The three groups reported were: hypocapnia (PaCO2 20 mm Hg) with no intracranial mass (group 1), intracranial mass (epidural balloon, CSF pressure 35 cm H2O) but no hypocapnia (group 2), and intracranial mass with hypocapnia used to lower CSF pressure (group 3). It was found that in dogs with an intracranial mass (group 3) the CSF pressure-lowering effect of hypocapnia was sustained for 4 h due to i

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40

Bouma, Gerrit J., J. Paul Muizelaar, Kuniaki Bandoh, and Anthony Marmarou. "Blood pressure and intracranial pressure-volume dynamics in severe head injury: relationship with cerebral blood flow." Journal of Neurosurgery 77, no. 1 (1992): 15–19. http://dx.doi.org/10.3171/jns.1992.77.1.0015.

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✓ Increased brain tissue stiffness following severe traumatic brain injury is an important factor in the development of raised intracranial pressure (ICP). However, the mechanisms involved in brain tissue stiffness are not well understood, particularly the effect of changes in systemic blood pressure. Thus, controversy exists as to the optimum management of blood pressure in severe head injury, and diverging treatment strategies have been proposed. In the present study, the effect of induced alterations in blood pressure on ICP and brain stiffness as indicated by the pressure-volume index (PVI

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Warner, David S. "Blood pressure and intracranial pressure-volume dynamics in severe head injury: relationship with cerebral blood flow." Journal of Neurosurgical Anesthesiology 5, no. 1 (1993): 56–57. http://dx.doi.org/10.1097/00008506-199301000-00018.

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42

Pagani-Estévez, Gabriel L., Jeremy K. Cutsforth-Gregory, Jonathan M. Morris, et al. "Procedural predictors of epidural blood patch efficacy in spontaneous intracranial hypotension." Regional Anesthesia & Pain Medicine 44, no. 2 (2019): 212–20. http://dx.doi.org/10.1136/rapm-2018-000021.

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Background and objectiveEpidural blood patch (EBP) is a safe and effective treatment for spontaneous intracranial hypotension (SIH), but clinical and procedural variables that predict EBP efficacy remain nebulous.MethodsThis study is an institutional review board-approved retrospective case series with dichotomized EBP efficacy defined at 3 months. The study included 202 patients receiving 604 EBPs; iatrogenic cerebrospinal fluid leaks were excluded.ResultsOf the EBPs, 473 (78%) were single-level, 349 (58%) lumbar, 75 (12%) bilevel, and 56 (9%) multilevel (≥3 levels). Higher volume (OR 1.64; p

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43

Rosner, Michael J., and Irene B. Coley. "Cerebral perfusion pressure, intracranial pressure, and head elevation." Journal of Neurosurgery 65, no. 5 (1986): 636–41. http://dx.doi.org/10.3171/jns.1986.65.5.0636.

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✓ Previous investigations have suggested that intracranial pressure waves may be induced by reduction of cerebral perfusion pressure (CPP). Since pressure waves were noted to be more common in patients with their head elevated at a standard 20° to 30°, CPP was studied as a function of head position and its effect upon intracranial pressure (ICP). In 18 patients with varying degrees of intracranial hypertension, systemic arterial blood pressure (SABP) was monitored at the level of both the head and the heart. Intracranial pressure and central venous pressure were assessed at every 10° of head e

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44

Czosnyka, Zofia, Marek Czosnyka, and John D. Pickard. "CSF Pulse Pressure and B Waves." Journal of Neurosurgery 103, no. 4 (2005): 767–68. http://dx.doi.org/10.3171/jns.2005.103.4.0767.

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Abstract Object. The appearance of numerous B waves during intracranial pressure (ICP) registration in patients with idiopathic adult hydrocephalus syndrome (IAHS) is considered to predict good outcome after shunt surgery. The aim of this study was to describe which physical parameters of the cerebrospinal fluid (CSF) system B-waves reflect and to find a method that could replace long-term B-wave analysis. Methods. Ten patients with IAHS were subjected to long-term registration of ICP and a lumbar constant-pressure infusion test. The B-wave presence, CSF outflow resistance (Rout), and relative

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45

Krieger, Steffen Norbert, Markus Nikolar Streicher, Robert Trampel, and Robert Turner. "Cerebral Blood Volume Changes during Brain Activation." Journal of Cerebral Blood Flow & Metabolism 32, no. 8 (2012): 1618–31. http://dx.doi.org/10.1038/jcbfm.2012.63.

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Cerebral blood volume (CBV) changes significantly with brain activation, whether measured using positron emission tomography, functional magnetic resonance imaging (fMRI), or optical microscopy. If cerebral vessels are considered to be impermeable, the contents of the skull incompressible, and the skull itself inextensible, task- and hypercapnia-related changes of CBV could produce intolerable changes of intracranial pressure. Because it is becoming clear that CBV may be useful as a well-localized marker of neural activity changes, a resolution of this apparent paradox is needed. We have explo

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46

Hannerz, J., and T. Jogestrand. "Effects of Increasing the Intracranial Blood Volume in Cluster Headache Patients and Controls." Cephalalgia 15, no. 6 (1995): 499–503. http://dx.doi.org/10.1046/j.1468-2982.1995.1506499.x.

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Eleven patients with episodic cluster headache in period, five patients out of period and six controls were studied concerning the effects of an increase of the intracranial blood volume by tilting. Common carotid artery (CCA) blood flow was similar in all three groups at baseline and during tilting. CCA diameters were similar at baseline and increased during tilting in all three groups, indicating that tilting caused an increase in the extra- and intracranial blood volume. Unilateral pain or sympathetic dysfunction did not appear during tilting in the patients out of period or in the controls

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47

Hannerz, J., and T. Jogestrand. "Effects of Increasing the Intracranial Blood Volume in Cluster Headache Patients and Controls." Cephalalgia 15, no. 6 (1995): 499–503. http://dx.doi.org/10.1046/j.1468-29821995.1506499.x.

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48

Mehta, Bobby, and Jordan Tarshis. "Repeated large-volume epidural blood patches for the treatment of spontaneous intracranial hypotension." Canadian Journal of Anesthesia/Journal canadien d'anesthésie 56, no. 8 (2009): 609–13. http://dx.doi.org/10.1007/s12630-009-9121-y.

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49

Czosnyka, Marek, Peter Smielewski, Ivan Timofeev, et al. "Intracranial Pressure: More Than a Number." Neurosurgical Focus 22, no. 5 (2007): 1–7. http://dx.doi.org/10.3171/foc.2007.22.5.11.

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✓Many doctors involved in the critical care of head-injured patients understand intracranial pressure (ICP) as a number, characterizing the state of the brain pressure–volume relationships. However, the dynamics of ICP, its waveform, and secondarily derived indices portray useful information about brain homeostasis. There is circumstantial evidence that this information can be used to modify and optimize patients' treatment. Secondary variables, such as pulse amplitude and the magnitude of slow waves, index of compensatory reserve, and pressure–reactivity index (PRx), look promising in clinica

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50

KABEYA, Ryusuke, Suguru INAO, Masanori TADOKORO, Masanari NISHINO, and Jun YOSHIDA. "Cerebral Blood Flow During Plateau Waves in a Patient With Benign Intracranial Hypertension. Case Report." Neurologia medico-chirurgica 40, no. 5 (2000): 287–92. http://dx.doi.org/10.2176/nmc.40.287.

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