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Libros sobre el tema "Intrinsically disordered"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 12 de febrero de 2022

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1

Uversky, Vladimir N. Intrinsically Disordered Proteins. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-08921-8.

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2

Kragelund, Birthe B. y Karen Skriver, eds. Intrinsically Disordered Proteins. New York, NY: Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0524-0.

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3

Uversky, Vladimir N. y A. Keith Dunker, eds. Intrinsically Disordered Protein Analysis. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-927-3.

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4

Uversky, Vladimir N. y A. Keith Dunker, eds. Intrinsically Disordered Protein Analysis. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-3704-8.

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5

Uversky, Vladimir N. y Sonia Longhi, eds. Instrumental Analysis of Intrinsically Disordered Proteins. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2010. http://dx.doi.org/10.1002/9780470602614.

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6

Felli, Isabella C. y Roberta Pierattelli, eds. Intrinsically Disordered Proteins Studied by NMR Spectroscopy. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-20164-1.

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7

Tompa, Peter. Structure and function of intrinsically disordered proteins. Boca Raton, FL: Chapman & Hall/CRC Press, 2009.

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8

Uversky, Vladimir N. Instrumental analysis of intrinsically disordered proteins: Assessing structure and conformation. Hoboken, N.J: Wiley, 2010.

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9

Instrumental analysis of intrinsically disordered proteins: Assessing structure and conformation. Hoboken, N.J: Wiley, 2010.

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10

Intrinsically Disordered Proteins. Chapman & Hall/CRC, 2009.

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11

Uversky, Vladimir N. Intrinsically Disordered Proteins. Springer, 2014.

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12

Intrinsically Disordered Proteins. Elsevier, 2019. http://dx.doi.org/10.1016/c2018-0-00038-9.

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13

Intrinsically Disordered Proteins. Elsevier, 2018. http://dx.doi.org/10.1016/s0076-6879(18)x0012-3.

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14

Intrinsically Disordered Proteins, Volume 611. Academic Press, 2018.

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15

Measles Virus Nucleoprotein (Intrinsically Disordered Proteins). Nova Science Pub Inc, 2008.

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16

Intrinsically Disordered Proteins and Chronic Diseases. MDPI, 2021. http://dx.doi.org/10.3390/books978-3-0365-1263-1.

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17

Felli, Isabella C. y Roberta Pierattelli. Intrinsically Disordered Proteins Studied by NMR Spectroscopy. Springer, 2016.

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18

P, Creamer Trevor, ed. Unfolded proteins: From denatured to intrinsically disordered. New York: Nova Science, 2008.

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19

Tompa, Peter y Alan Fersht. Structure and Function of Intrinsically Disordered Proteins. Chapman and Hall/CRC, 2009. http://dx.doi.org/10.1201/9781420078930.

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20

Felli, Isabella C. y Roberta Pierattelli. Intrinsically Disordered Proteins Studied by NMR Spectroscopy. Springer, 2015.

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21

Longhi, Sonia y Vladimir Uversky. Instrumental Analysis of Intrinsically Disordered Proteins: Assessing Structure and Conformation. Wiley & Sons, Incorporated, John, 2011.

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22

Weber, Violet. Intrinsically Disordered Proteins: Structural Characterization, Therapeutic Applications and Future Directions. Nova Science Publishers, Incorporated, 2016.

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23

Intrinsically Disordered Protein Analysis: Volume 1, Methods and Experimental Tools. Humana, 2012.

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24

Uversky, Vladimir N. Dancing Protein Clouds: Intrinsically Disordered Proteins in Health and Disease, Part B. Elsevier Science & Technology Books, 2020.

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25

Dancing Protein Clouds: Intrinsically Disordered Proteins in Health and Disease, Part B. Elsevier Science & Technology, 2020.

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26

Dancing protein clouds: Intrinsically disordered proteins in health and disease, Part A. Elsevier, 2019. http://dx.doi.org/10.1016/s1877-1173(19)x0004-6.

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27

Dancing Protein Clouds: Intrinsically Disordered Proteins in Health and Disease, Part B. Elsevier, 2020. http://dx.doi.org/10.1016/s1877-1173(20)x0007-x.

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28

Computational Approaches to Protein Dynamics: From Quantum to Coarse-Grained Methods. Taylor & Francis Group, 2018.

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29

Computational Approaches to Protein Dynamics: From Quantum to Coarse-Grained Methods. Taylor & Francis Group, 2014.

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30

Wetzel, Ronald y Rakesh Mishra. Structural Biology. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199929146.003.0012.

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The 3,144–amino acid huntingtin protein (HTT) folds in water into a structure consisting of compact, organized domains interspersed with intrinsically disordered protein (IDP) elements. The IDPs function as sites of post-translational modifications and proteolysis as well as in targeting, binding, and aggregation. Although the dominant structural motif of HTT is the α‎-helix–rich HEAT repeat, the expanded polyglutamine (polyQ) toxicity responsible for Huntington’s disease is most likely played out within intrinsically disordered HTT exon 1–like fragments consisting of the 16– to 17–amino acid N-terminal HTTNT segment, the polyQ segment, and a proline-rich segment. The physical behavior of HTT exon 1 fragments is dominated by interactive, polyQ repeat length–dependent structural transitions responsible for membrane and protein–protein interactions and the formation of tetramers, higher oligomers, amyloid fibrils, and inclusions. Understanding the basis of this solution behavior may be the key to disease mechanisms and molecular therapeutic strategies.
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31

Siever, Larry J. y Joshua E. Kuluva. Aggression, Impulsivity, and Personality Disorders. Editado por Jon E. Grant y Marc N. Potenza. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195389715.013.0030.

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Aggressivity and impulsivity are traits that are core features of the Cluster B personality disorders. Within these disorders, impulsive aggression leads to a significant amount of morbidity and mortality. This type of behavior is intrinsically linked to violence, suicide, and substance abuse. In this chapter, we will discuss the phenomenology of these traits, the neurobiology of impulsive aggression, and some potential treatment options. We will conclude with some thoughts on the future direction of research in this filed.
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32

Vögele, Claus, Annika P. C. Lutz y E. Leigh Gibson. Mood, Emotions, and Eating Disorders. Editado por W. Stewart Agras y Athena Robinson. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190620998.013.8.

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Mood and emotions are intrinsically involved with eating. This chapter discusses basic mechanisms, findings, and models that help our understanding of the interactions between eating and emotions, in both clinical and nonclinical populations. The finding that negative affect predicts EDs transdiagnostically, and that comorbidity with depressive disorders and anxiety disorders is the norm among patients with EDs suggests that EDs may not necessarily be restricted to domains of eating behavior and body image but may also be associated with significant difficulties in affective functioning. This chapter reviews the evidence relating to the notion that EDs are disturbances of mood regulation, in which regulatory strategies specifically related to eating and the body are used to diminish negative affect associated with food, body image, or stress.
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33

Stafström, Sven y Mikael Unge. Disorder-induced electron localization in molecular-based materials. Editado por A. V. Narlikar y Y. Y. Fu. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780199533046.013.25.

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This article examines disorder-induced electron localization in molecular-based materials, using DNA and pentacene molecular crystals as examples. In DNA, the disorder is intrinsic and strong, resulting in very short localization lengths. The pentacene crystal, on the other hand, is intrinsically homogeneous and the disorder is extrinsic and weak, which makes a metal–insulator transition (MIT) possible. After providing an overview of carbon-based materials for electronic applications, the article explains the methodology for calculating the localization properties of a DNA double strand and a pentacene molecular crystal, namely Hamiltonian, transfer matrix, and finite-size scaling. It also discusses the results, which show a substantial increase in the localization length of the electronic state with correlated disorder as compared to the case of uncorrelated disorder.
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34

Herbert, Beate M. y Olga Pollatos. The relevance of interoception for eating behavior and eating disorders. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198811930.003.0009.

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The importance of interoception for adaptive and maladaptive behavior, as well as for psychopathology, has gained growing interest, and dysfunctional interoception has been recognized as representing a core impairment across psychosomatic and psychiatric disorders. Eating is intrinsically guided by interoceptive signals and is directly associated with homeostatic psychophysiological needs, well-being, and survival. This chapter provides conceptually and empirically drawn conclusions focusing on the relevance of distinguishable dimensions of interoception for shaping eating behavior and body weight, and for eating disorders. Going beyond eating behavior per se, anorexia and bulimia nervosa are conceptualized as characterized by profound impairment of the self, with dysfunctional interoception at its core. Predictive coding models are addressed to integrate conclusions and empirical findings tentatively.
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35

Colameco, Stephen. Pain and Addiction in Patients with Co-Occurring Medical Disorders (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190265366.003.0026.

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Distinct from Chapter 24, on co-occurring psychiatric disorders, this chapter addresses common physical comorbidities that give rise to chronic pain and are notorious for associated substance use disorders. The concept of “pseudo-addiction” is explored as one of several contributors to common misperceptions of the analgesic needs of such patients. Examples of entities discussed are chronic low back pain, sleep apnea, chronic pancreatitis, cirrhosis, and HIV infection or AIDS-related pain. While not intrinsically painful, sleep apnea merits inclusion as it arises in conjunction with sedative-hypnotic, opioid, or nicotine use. Cirrhosis likewise creates obstacles to successful pain or addiction management resulting from altered metabolism of medications and enhanced susceptibility to potentially lethal syndromes (hepato-renal syndrome, gastric hemorrhage, etc.). The management of neuropathic pain in HIV infection (Chapter 15) is amplified here.
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36

Pangarkar, Sanjog S. Pain and Addiction in Patients with Traumatic Brain Injury (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190265366.003.0027.

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Distinct from Chapter 24, on co-occurring psychiatric disorders, this chapter addresses common physical comorbidities that give rise to chronic pain and are notorious for associated substance use disorders. The concept of “pseudo-addiction” is explored as one of several contributors to common misperceptions of the analgesic needs of such patients. Examples of entities discussed are chronic low back pain, sleep apnea, chronic pancreatitis, cirrhosis, and HIV infection or AIDS-related pain. While not intrinsically painful, sleep apnea merits inclusion as it arises in conjunction with sedative-hypnotic, opioid, or nicotine use. Cirrhosis likewise creates obstacles to successful pain or addiction management resulting from altered metabolism of medications and enhanced susceptibility to potentially lethal syndromes (hepato-renal syndrome, gastric hemorrhage, etc.). The management of neuropathic pain in HIV infection (Chapter 15) is amplified here.
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