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Tesis sobre el tema "Lungs Differentiation"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 29 de enero de 2023

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1

Stogsdill, Jeffrey Alan. "Characterization of Altered Epithelial Cell Turnover and Differentiation in Embryonic Murine Lungs That Over-Express Receptors for Advanced Glycation End-Products (RAGE)." BYU ScholarsArchive, 2012. https://scholarsarchive.byu.edu/etd/3217.

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Receptors for advanced glycation end-products (RAGE) are multi-ligand cell surface receptors highly expressed in the lung that modulate pulmonary inflammation during disease. However, the contributions of RAGE signaling are unknown during pulmonary organogenesis. In order to test the hypothesis that RAGE misexpression adversely affects lung morphogenesis, conditional transgenic mice were generated that over-express RAGE in alveolar type II cells of the lung. When RAGE is over-expressed throughout embryogenesis, severe lung hypoplasia ensues, culminating in perinatal lethality. Flow cytometry a

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2

Lal, Arpita. "Relationship among differentiation of self, relationship satisfaction, partner support, depression, monitoring/blunting style, adherence to treatment and quality of life in patients with chronic lung disease." Columbus, Ohio : Ohio State University, 2006. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1164037503.

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3

Ansari, Naser A. (Naser Awni). "Purification and Characterization of a Differentiation Factor From Rat Lung Conditioned Medium." Thesis, North Texas State University, 1988. https://digital.library.unt.edu/ark:/67531/metadc798062/.

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A Differentiation Factor (DF) was purified from rat lung conditioned medium by a four-steps procedure. The DF has a molecular weight of 27000, and an isoelectric point of 4.70. Although DF is stable up to 60°C, it is sensitive to digestion by trypsin, chymotrypsin and subtilisin. DF forms granulocyte colonies in soft agar. Studies using anti-NRK CSF antibody demonstrated that DF is distinct from GM-CSF.

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4

Ansari, Naser A. (Naser Awni). "Mechanism of myeloid differentiation induced by a differentiation factor isolated from rat lung conditioned medium." Thesis, University of North Texas, 1991. https://digital.library.unt.edu/ark:/67531/metadc798429/.

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5

Berg, Tove. "C/EBP transcription factors in lung cellular differentiation and development /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-586-0/.

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6

Tompkins, David H. "Sox2 is a Master Regulator of Differentiation in Respiratory Epithelium." University of Cincinnati / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1307985600.

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7

Alaqel, Abdullah. "The directed differentiation of human embryonic stem cells to lung cell lineages." Thesis, University of Bath, 2017. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.760955.

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Human embryonic stem cells (hESCs) show significant therapeutic potential in treating degenerative disorders. This is in part because of their ability to produce a limitless supply of starting cells and their potential to differentiate into more than 200 different cell types. The aim of the current research was to generate a robust stage wise protocol for the differentiation of hESCs to respiratory epithelial cells. The epithelial cells could then be used either for transplantation studies or, as an in vitro model for drug toxicity testing. In order to achieve this goal, we must identify the k

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8

METZGER, DAVID EDWARD. "THE ROLE OF THE ETS TRANSCRIPTION FACTOR Elf5 IN LUNG DEVELOPMENT." University of Cincinnati / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1197664589.

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9

Senden, Nicole Hubertina Maria. "NSP-reticulons characterization and use for the detection of neuroendocrine differentiation in lung cancer /." Maastricht : Maastricht : Universitaire Pers Maastricht ; University Library, Maastricht University [Host], 1995. http://arno.unimaas.nl/show.cgi?fid=8353.

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10

Soh, Boon Seng. "Optimization of Human Embryonic Stem Cells Culture and their Differentiation towards the Lung Lineage." Thesis, Imperial College London, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.516176.

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11

Kikuchi, Ryutaro. "Expression of IGF1R is associated with tumor differentiation and survival in patients with lung adenocarcinoma." Kyoto University, 2013. http://hdl.handle.net/2433/174806.

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12

Chen, Gang. "Critical roles of Foxa2 and Spdef in regulating innate immunity and goblet cell differentiation in the lung." University of Cincinnati / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1276537438.

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13

Cassel, Tobias. "Transcriptional regulation of differentiation markers in the distal lung epithelium : a role for C/EBP factors /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4853-4/.

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14

Van, Vranken Benjamin Eugene. "The influence of embryonic lung mesenchyme on the differentiation of embryonic stem cells in co-culture." Thesis, Imperial College London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.416628.

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15

LIU, CONG. "REGULATION OF LUNG EPITHELIAL DIFFERENTIATION ALVEOLARIZATION AND GENE EXPRESSION BY GATA-6 IN VITRO AND IN VIVO." University of Cincinnati / OhioLINK, 2002. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1026498687.

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16

Nagai, Shinjiro. "A novel classification of MUC1 expression is correlated with tumor differentiation and postoperative prognosis in non-small cell lung cancer." Kyoto University, 2008. http://hdl.handle.net/2433/124238.

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17

Karvonen, H. (Henna). "Ultrastructural and functional characterization of myofibroblasts in lung diseases." Doctoral thesis, Oulun yliopisto, 2014. http://urn.fi/urn:isbn:9789526203560.

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Abstract Pulmonary fibrosis, lung cancer and chronic obstructive pulmonary disease (COPD) are severe diseases and common death causes worldwide. Due to the lack of an effective therapy, the investigation of cell biological mechanisms behind these diseases is essential. An activation of stromal cells, including myofibroblasts, is a main feature found in the pathogenesis of lung diseases. Myofibroblasts express alpha-smooth muscle actin (α-SMA), have specific ultrastructure, produce extracellular matrix proteins and possess contractile capacity. Detailed structure and function of myofibroblasts

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18

Piskulak, Katarzyna Teresa [Verfasser]. "Regulation and role of Notch signaling in epithelial progenitor cell differentiation and proliferation in the normal and the fibrotic lung / Katarzyna Teresa Piskulak." Gießen : Universitätsbibliothek, 2013. http://d-nb.info/106546276X/34.

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19

Tsuji, Hideaki. "Multicenter Prospective Study of the Efficacy and Safety of Combined Immunosuppressive Therapy With High-Dose Glucocorticoid, Tacrolimus, and Cyclophosphamide in Interstitial Lung Diseases Accompanied by Anti-Melanoma Differentiation-Associated Gene 5-Positive Dermatomyositis." Kyoto University, 2021. http://hdl.handle.net/2433/261610.

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20

Vasconcelos, Michelle. "O papel da sinalização Notch na diferenciação do epitélio pulmonar." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-16052012-100050/.

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O epitélio pulmonar é formado por uma grande diversidade celular, que incluí: células secretoras, ciliadas, basais e neuroendócrinas (NE). A distribuição balanceada destes tipos celulares é crucial para a função pulmonar e pode ser dramaticamente alterada em doenças como a asma. Neste trabalho, estudamos o papel de Notch no pulmão em desenvolvimento ao inativar condicionalmente Rbpjk ou Pofut1, componentes críticos da sinalização Notch. Pulmões mutantes apresentaram-se superpopulados por células ciliadas e NE, além da ausência de células de Clara. Nossos dados sugeriram que Notch suprime os pr

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21

Jaberansari, Ziba [Verfasser], Didier [Akademischer Betreuer] [Gutachter] Stainier, and Anna [Akademischer Betreuer] [Gutachter] Starzinski-Powitz. "Identifying regulators of lung epithelial cell differentiation by using a forward genetic screening approach in mouse / Ziba Jaberansari. Betreuer: Didier Stainier ; Anna Starzinski-Powitz. Gutachter: Didier Stainier ; Anna Starzinski-Powitz." Frankfurt am Main : Universitätsbibliothek Johann Christian Senckenberg, 2016. http://d-nb.info/1112601473/34.

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22

Belgacemi, Randa. "CARACTÉRISATION DE LA VOIE HEDGEHOG DANS LA DIFFÉRENCIATION DE L'ÉPITHÉLIUM DES VOIES AÉRIENNES ET ALTÉRATION DANS LA BPCO Airway epithelial cell differentiation relies on deficient Hedgehog signalling in COPD." Thesis, Reims, 2020. http://www.theses.fr/2020REIMS009.

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La voie Hedgehog (HH) est une voie de signalisation cruciale pour l’organogenèse et l’homéostasie. Etant donnée son implication dans la quiescence et la réparation pulmonaire ainsi que son altération dans des maladies respiratoires chroniques, nous avons exploré la présence et l’impact de la voie HH durant la différenciation de l’épithélium des voies aériennes et son incidence sur la genèse du remodelage épithélial caractéristique de la BPCO.Nous avons mis en évidence une corrélation entre l’expression des acteurs de la voie HH et la différenciation in vitro ainsi qu’une sécrétion du ligand pr

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23

Akoum, Rania El. "La phosphorylation de CARM1 empêche l'interaction entre PRMT1 et CARM1, deux « Protein Arginine MethylTransférases » impliquées dans la prolifération dans le cancer du poumon." Thesis, Université de Lorraine, 2013. http://www.theses.fr/2013LORR0128/document.

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CARM1 et PRMT1 sont 2 Protein Arginine MethylTransferases (PRMTs) impliquées dans la prolifération et dérégulées dans le cancer. La dimérisation est une caractéristique commune aux PRMTs. PRMT1 et CARM1 coopèrent dans la régulation des gènes mais il n'existe pas de données concernant un hétérodimère CARM1/PRMT1. Nous avons trouvé que PRMT1 et CARM1 sont surexprimées dans le cancer du poumon non à petites cellules et dans 2 lignées d'adénocarcinomes pulmonaires, A549 et H1299. Les siPRMT1 réduisent la prolifération cellulaire et facilitent la différentiation. Les siCARM1 produisent un effet sim

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24

Pellet, Mathieu. "Caractérisation non entière de systèmes biologiques : application au muscle squelettique et au poumon." Phd thesis, Université Sciences et Technologies - Bordeaux I, 2013. http://tel.archives-ouvertes.fr/tel-00944614.

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Le thème des travaux qui fait l'objet de ce mémoire de thèse s'inscrit dans le cadre de la caractérisation de systèmes biologiques par modèles non entiers. Cette thèse comporte deux parties qui reposent sur deux collaborations distinctes. La première s'appuie sur une collaboration avec le laboratoire Mouvement Adaptation Cognition de l'Université Bordeaux 2 et l'institut Magendie de l'Inserm. L'objectif de ce travail consiste à étudier l'influence la longueur du muscle sur sa dynamique dans les cas de variations statiques et dynamiques de cette grandeur. La deuxième collaboration est un projet

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25

Stupnikov, Maria Rose. "Genetic regulation of pulmonary progenitor cell differentiation." Thesis, 2019. https://doi.org/10.7916/d8-kgxh-ga80.

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The respiratory system represents a major interface between the body and the external environment. Its design includes a tree-like network of conducting tubules (airways) that carries air to millions of alveoli, where gas exchange occurs. The conducting airways are characterized by their great diversity in epithelial cell types with multiple populations of secretory, multiciliated, and neuroendocrine cells. How these different cell types arise and how these populations are balanced are questions still not well understood. Aberrant patterns of airway epithelial differentiation have been describ

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26

Johnston, Sonya D. (Sonya Denise). "Development of the pulmonary surfactant system in non-mammalian amniotes." 2001. http://web4.library.adelaide.edu.au/theses/09PH/09phj737.pdf.

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"March 2001". Bibliography: leaves 193-238. Relates changes in the development of the pulmonary surfactant system in response to birth strategy, lung morphology and phylogeny in order to determine the extent of conservation in this process, by quantifying the total of phsospholipid, disaturated phospholipid and cholesterol in the lung washings of embryonic and hatchling chickens, oviparous bearded dragons and viviparous sleepy lizards, snapping turtles and green sea turtles throughout the final stages of incubation and gestation. Finds that the pattern of development of pulmonary surfactant l

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27

Johnston, Sonya D. (Sonya Denise). "Development of the pulmonary surfactant system in non-mammalian amniotes / Sonya D. Johnston." Thesis, 2001. http://hdl.handle.net/2440/19859.

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"March 2001".<br>Bibliography: leaves 193-238.<br>vii, 238 leaves : ill. (some col.) ; 30 cm.<br>Relates changes in the development of the pulmonary surfactant system in response to birth strategy, lung morphology and phylogeny in order to determine the extent of conservation in this process, by quantifying the total of phsospholipid, disaturated phospholipid and cholesterol in the lung washings of embryonic and hatchling chickens, oviparous bearded dragons and viviparous sleepy lizards, snapping turtles and green sea turtles throughout the final stages of incubation and gestation. Finds that

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28

YANG, BAN-CHI, and 楊泮池. "GROWTH AND DIFFERENTIATION OF HUMAN AIRWAY EPITHELIUM AND LUNG CARCINOMA." Thesis, 1990. http://ndltd.ncl.edu.tw/handle/21703885933216396770.

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博士<br>國立臺灣大學<br>醫學研究所<br>79<br>Mucociliary clearance plays an important role in prlmonary defense mechanism. Abnormalities in mucociliary clearance are directly or indirectly related to various pulmonary desorders such se chromic bromchitis, bronchiectasis, cystic fibrosis, boinchial asthma, pneumonia and even lung cancer. There are two major functions of human respiratory epithelium: sceretion of viscoelastic mucus which can trap the inhaled potentially harmful particles and ciliary escalation which re

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29

Su, Hsiang-Han, and 蘇湘涵. "The Role of Aryl Hydrocarbon Receptorin Regulation of Lung Fibroblast Differentiation." Thesis, 2011. http://ndltd.ncl.edu.tw/handle/09150113142949332984.

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碩士<br>高雄醫學大學<br>醫學研究所<br>99<br>Pulmonary fibrosis is a progressive process that leads to cough, shortening of breath, pulmonary hypertension and cardiopulmonary failure. Fibrosis is defined by the overgrowth, scarring of tissues and is attributed to excess deposition of extracellular matrix including collagen and fibronectin, leading to reduced pulmonary function. Environmental endocrine disruptors are compounds that interfere with human endocrine system and have been shown to be assotiated with many diseases. Therefore, the purpose of our study was to investigate the effects of environmental

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30

Carvalho, Ana Luísa Rodrigues Toste de. "Directed differentiation of human pluripotent stem cells into mature lung epithelium in-vitro." Doctoral thesis, 2020. http://hdl.handle.net/1822/76460.

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Tese de doutoramento em Medicina<br>As doenças de foro respiratório são altamente prevalentes a nível mundial, encontrando-se no grupo das doenças mais fatais em países desenvolvidos. Epitélio pulmonar gerado in-vitro a partir de células estaminais pluripotentes humanas tem aplicações em medicina regenerativa, no estabelecimento de novos modelos de doença, ensaios farmacológicos pré-clínicos e estudo do desenvolvimento humano. Neste trabalho descrevemos uma estratégia para gerar células da via aérea e alveolares maturas a partir de células estaminais pluripotentes humanas. Seguimo

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31

Bhaskaran, Manoj. "Alveolar epithelial trans-differentiation, lung development and disease role of TGF ß1 and microRNAs /." 2008. http://digital.library.okstate.edu/etd/umi-okstate-2807.pdf.

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32

Chung, Chih-Hung, and 鍾志宏. "Functional Oct4B induction by hypoxia promotes lung cancer oncogenesis and epithelial-mesenchymal trans-differentiation." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/06132366207404863001.

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碩士<br>國立陽明大學<br>生化暨分子生物研究所<br>98<br>Hypoxia, a reduction of normal oxygen levels in cells or tissues, creates a variety of changes in cell metabolism including increased oxidative stress and DNA damage. Though Oct4, a homeobox transcription factor essential for self-renewal of stem cells, is expressed in various cancers, little is known about the functional role of Oct4 in tumorigenesis. In this study, we discovered that hypoxia induces a short isoform of Oct4, termed as Oct4B, in lung cancer through a HIF-2? dependent pathway. Overexpression of Oct4B induced cell proliferation and anchorage-i

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33

Huang, Tsai-Wang, and 黃才旺. "The role of Thyroid Transcription Factor-1 and Tumor differentiation in Resected Lung Adenocarcinoma." Thesis, 2017. http://ndltd.ncl.edu.tw/handle/ceva9y.

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博士<br>國防醫學院<br>醫學科學研究所<br>106<br>A total of 520 patients with clinical early stage lung adenocarcinoma who underwent surgical resection were reviewed retrospectively. Clinical data and outcomes were evaluated with an average follow-up of 117 months. The results were validated via lung cancer cell line studies. The clinical parameters did not differ between relapse and nonrelapse patients. Exceptions were tumor differentiation, lymphovascular space invasion, F18-fluorodeoxyglucose maximum standard uptake value, tumor size, and pathological stage (p < 0.001). Poor tumor differentiation was the i

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34

Wright, Kristen. "Role of CCAAT Enhancer Binding Protein Alpha in cell differentiation in leukemia and lung cancer cells." Thesis, 2020. https://hdl.handle.net/2144/41778.

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CCAAT/Enhancer Binding Protein Alpha (C/EBPa) is transcription factor protein involved in the differentiation of many cell types, including granulocytes and pulmonary cells. Studies have found that downregulation of C/EBPa leads to tumor formation in the hematopoietic system, bones, lungs, liver, and other organs. Mutations and post translational modifications can also reduce the function of C/EBPa in humans, leading to cancers. Recent studies have made progress in treating acute promyelocytic leukemia (APL), an M3 subtype of acute myeloid leukemia (AML), by incorporating all trans retinoic ac

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35

Teck-OoiWong and 黃德偉. "The role of osteotropic lung cancer cell-secreted factors in regulating osteoblast and osteoclast differentiation." Thesis, 2018. http://ndltd.ncl.edu.tw/handle/yab57y.

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36

Chen, Pei-Yu, and 陳貝瑜. "Genetic and Epigenetic Regulation of PD-L1 in Stem Cell Differentiation and Lung Cancer Cell Progression." Thesis, 2018. http://ndltd.ncl.edu.tw/handle/yg4t76.

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37

Lee, Chih-Chan, and 李志展. "Sox2, an EGFR induced transcriptional factor, modulates cell growth and mesenchymal-epithelial trans-differentiation (MET) of lung cancer." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/91356858696672476371.

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碩士<br>國立陽明大學<br>生化暨分子生物研究所<br>98<br>Aberrant expression and function of epidermal growth factor receptor (EGFR) is reported in most lung cancer cases. Sox2, a core transcription factor regulating self-renewal of stem cells, is highly expressed in lung cancer. We discovered that EGFR and its ligands (TGF-??nand EGF) induce Sox2 expression in lung cancer. Ectopic expression of c-Myc, a key effector of EGFR signaling, induced Sox2 expression; knockdown of c-Myc decreased Sox2 level in lung cancer, suggesting that EGFR induces Sox2 via a c-Myc dependent pathway. Ectopic expression of Sox2 promoted

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38

Sawant, Deepali Vijay. "Control of inflammation, helper T cell responses and regulatory T cell function by Bcl6." Thesis, 2014. http://hdl.handle.net/1805/3829.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Regulatory T (Treg) cells represent an important layer of immune-regulation indispensible for curtailing exuberant inflammatory responses and maintaining self-tolerance. Treg cells have translational potential for autoimmunity, inflammation, transplantation and cancer. Therefore, delineating the molecular underpinnings underlying the development, suppressor function and stability of Tregs is particularly warranted. The transcriptional repressor Bcl6 is a critical arbiter of helper T cell fate, promoting the follicular helper (Tfh) li

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39

Tsai, Wen-Ting, and 蔡文婷. "The role of aryl hydrocarbon receptor (AhR) in growth of human lung cancer cells and the change in AhR expression / activity during differentiation of lung epithelial cells." Thesis, 2004. http://ndltd.ncl.edu.tw/handle/71590337250996204383.

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碩士<br>中山醫學大學<br>毒理學研究所<br>92<br>Our previous studies indicated that aryl hydrocarbon receptor (AhR) expression was up-regulated in human lung adenocarcinoma (AD) tissues and cell lines. It suggested that AhR might play an important role in the development of lung adenocarcinoma. On the other hand, AhR expression was associated with differentiation in some normal cell types, such as keratinocytes. Previously we reported that AhR was present in the human bronchiolar epithelium. There are two objectives in this study: 1) to evaluate the function of AhR in lung adenocarcinoma cells with the RNAi t

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40

Chan, Hsiu-hua, and 詹修華. "Identification of biomarkers for differentiating adenocarcinoma and squamous cell carcinoma of lung for pathological and clinical application." Thesis, 2014. http://ndltd.ncl.edu.tw/handle/gw69zw.

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碩士<br>國立中央大學<br>系統生物與生物資訊研究所<br>102<br>Lung cancer is the leading cause of cancer deaths in the worldwide. The main tumor type includes small cell lung cancer (SCLC ~15% of all lung cancers) and non-small cell lung cancer (NSCLC ~85% of all lung cancers), NSCLC can be classified in adenocarcinoma (AD), squamous-cell carcinoma (SCC), large-cell lung cancer (LCC). Among them, the majority of lung cancer is AD in Taiwan. The different chemotherapy therapeutic drugs can cause the different side effect and prognosis in different kind of lung cancer. Well differentiated AD and SCC can be identify ef

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41

Chiu, Chen-Feng, and 邱振峰. "Short-time dual-phase FDG PET/CT in differentiating lung cancer and benign lung lesion based on chest CT patterns and surgical results:Solid or ground-glass nodules." Thesis, 2011. http://ndltd.ncl.edu.tw/handle/26473990948076177485.

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碩士<br>中國醫藥大學<br>臨床醫學研究所碩士班<br>99<br>Rational and Objectives: In this study, we compare the accuracy of shorter-time dual-phase 18F-FDG PET/CT in evaluating different types of lung nodules classified as solid or ground-glass nodules (GGNs). Materials and Methods: A total of 94 patients were enrolled in this retrospective study. The diagnostic chest CT images were classified as solid or GGNs. The early and delayed maximum standardized uptake value (SUVmax) as well as retention index (RI) of each nodule were determined. Results: Of the 75 solid nodules, 53 were malignant and 22 were benign. Of th

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