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Tesis sobre el tema "Lungs Pathophysiology"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 28 de enero de 2023

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1

McLennan, Geoffrey. "Oxygen toxicity and radiation injury to the pulmonary system." Title page, index and forward only, 1997. http://web4.library.adelaide.edu.au/theses/09PH/09phm164.pdf.

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Bibliography: leaves 168-184. The work in this study encompasses oxygen free radical related inflammation in the peripheral lung and in lung cells. Animal and human studies have been used. Methods include cell culture with function studies, protein chemistry, animal and human physiology, and cell and lung structure through histopathology, and various forms of electron microscopy. The work resulting from this thesis has formed an important basis for understanding acute and chronic lung injury.
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2

McNamara, Tracy Renee. "Chlamydia pneumoniae and airways inflammation : an investigation of the host cell-pathogen relationship /." Title page, table of contents and abstract only, 2004. http://web4.library.adelaide.edu.au/theses/09PH/09phm4791.pdf.

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3

Chan, Ching Eunice, and 陳清. "Pathogenetic role of aberrant promoter methylation in lung cancer." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39557819.

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4

Smedley, Jeremy Vance. "A Combined In Vivo and In Vitro Approach to the Study of Endotoxemia in Swine." Thesis, Virginia Tech, 2000. http://hdl.handle.net/10919/33947.

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The cardiopulmonary effects of endotoxin administration (1 microgram/kg) were evaluated in 8-10 week old SPF-derived Yorkshire pigs, both because endotoxemia is a common and important swine problem, and because the pig is a good model for human adult respiratory distress syndrome. Physiological changes included sustained increases in mean pulmonary artery pressure, pulmonary vascular resistance, pulmonary arterial wedge pressure, heart rate, hematocrit, and the arterial partial pressure of carbon dioxide. Transient increases were also observed in central venous pressure and airway pressure.
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5

Kerckx, Yannick. "Modeling nitric oxide production and transport in the human lung." Doctoral thesis, Universite Libre de Bruxelles, 2009. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210324.

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Le travail présenté ici porte sur l’étude de la production et du transport du monoxyde d’azote (NO) dans le poumon humain. Le NO est une molécule dont l’implication dans des processus physiologiques n’a été mis en évidence qu’en 1987. Depuis, il a été démontré que le NO joue de nombreux rôles dans le corps humain. Le NO est un gaz labile (instable) dans les conditions physiologiques, il diffuse très facilement au travers des parois et il a une grande affinité pour l’hémoglobine. La production du NO est liée à 3 isoformes différentes de la protéine appelées synthases du NO ou NO synthases.<p>\<
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6

Bondue, Benjamin. "Role of chemerin and its receptor ChemR23 in the physiopathology of inflammatory lung diseases." Doctoral thesis, Universite Libre de Bruxelles, 2010. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209992.

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Chemoattractant agents play a crucial role in the initiation of immune responses, by regulating the traffic and function of leucocyte populations. Their receptors are therefore considered as potential targets for the development of new therapies in the fields of cancer and inflammatory diseases. ChemR23, a previously orphan receptor discovered in the laboratory, is structurally related to receptors for chemoattractant agents. It is expressed on immature myeloid and plasmacytoid dendritic cells (mDCs and pDCs respectively), as well as on adipocytes, macrophages, NK and endothelial cells. Chemer
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7

Vanderstocken, Gilles. "Caractérisation du rôle des nucléotides extracellulaires et du récepteur purinergique P2Y2 dans la physiopathologie des maladies pulmonaires inflammatoires." Doctoral thesis, Universite Libre de Bruxelles, 2012. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209591.

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Amongst respiratory diseases, inflammatory lung diseases constitute a major part of public <p>health problem. As a consequence, investigating the immune mechanisms that contribute to <p>the pathogenesis of these diseases is essential to identify candidate targets for the <p>development of new therapeutic drugs. Furthermore, over the past 20 years, the growing awareness <p>that purinergic signalling events shape the immune and inflammatory responses to infection and <p>allergic reactions warranted the development of animal models to assess their importance in vivo in <p>acute lung injury and ch
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8

Wang, Jianpu. "Pathophysiology and treatment of chlorine gas-induced lung injury : an experimental study in pigs /." Linköping : Univ, 2004. http://www.bibl.liu.se/liupubl/disp/disp2004/med877s.pdf.

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9

Tiev, Kiet Phong. "Rôle du monoxyde d'azote dans la physiopathologie des atteintes pulmonaires de la sclérodermie systémique." Thesis, Paris Est, 2008. http://www.theses.fr/2008PEST0081.

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La pneumopathie interstitielle (PI) est devenue la principale cause de décès de la sclérodermie systémique (ScS). Au cours de PI, l’activation immunitaire déclenche une forte production du monoxyde d’azote (NO) et l’augmentation de la concentration de NO dans l’air expiré des patients atteints de ScS avec PI suggère que cette méthode pourrait détecter précocement l’alvéolite, afin de traiter à temps la PI pour éviter son évolution vers la fibrose pulmonaire. En utilisant le modèle à deux compartiments séparant le NO alvéolaire (CANO) du NO bronchique, nous avons montré que la CANO est : (1) au
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10

Ridings, Philip Charles. "An investigation into the role of selectins in the pathophysiology of sepsis and sepsis-induced acute lung injury." Thesis, University of Southampton, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266390.

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11

Aeffner, Famke. "Pathophysiologic Effects of Influenza Infection on the Murine Lung and Evaluation of Novel Therapeutic Targets." The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1384440085.

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12

Yoshioka, Eliane Muta. "Alterações pulmonares e sistêmicas em modelo de lesão pulmonar aguda de etiologia pulmonar e extra pulmonar após ventilação mecânica de curto prazo." Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/5/5144/tde-03092010-144329/.

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A inflamação pulmonar pode variar de acordo com o sitio primário da injuria e poder ser afetado pelo estresse mecânico gerado pela ventilação mecânica. (VM) Objetivos: estudar as eventuais diferenças na reposta pulmonar e sistêmica na lesão pulmonar aguda pulmonar (LPA P ) e extra pulmonar (LPA Exp) após ventilação mecânica. Métodos: Camundongos BALB/c foram divididos em doze grupos. Os grupos controle pulmonar (CP) e extra pulmonar (C Exp) receberam solução salina (SAL) ou Lipopolissacarideo (LPS) via intratraqueal (IT) ou intraperitoneal (IP) respectivamente. Os grupos foram submetidos ou nã
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13

McLennan, Geoffrey. "Oxygen toxicity and radiation injury to the pulmonary system / by Geoffrey McLennan." Thesis, 1997. http://hdl.handle.net/2440/19734.

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Bibliography: leaves 168-184.<br>184 leaves : ill. (chiefly col.) ; 30 cm.<br>The work in this study encompasses oxygen free radical related inflammation in the peripheral lung and in lung cells. Animal and human studies have been used. Methods include cell culture with function studies, protein chemistry, animal and human physiology, and cell and lung structure through histopathology, and various forms of electron microscopy. The work resulting from this thesis has formed an important basis for understanding acute and chronic lung injury.<br>Thesis (Ph.D.)--University of Adelaide, Dept. of Pa
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14

"Cigarette smoking enhances the expression of thromboxane synthase and stimulates lung cancer stem cells, leading to the development of lung cancer." 2015. http://repository.lib.cuhk.edu.hk/en/item/cuhk-1291566.

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Liu, Yi.<br>Thesis Ph.D. Chinese University of Hong Kong 2015.<br>Includes bibliographical references (leaves 154-175).<br>Abstracts also in Chinese.<br>Title from PDF title page (viewed on 25, October, 2016).
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15

Pandya, Pankita Hemant. "HIF-1α regulates CD55 expression in airway epithelium". 2015. http://hdl.handle.net/1805/8002.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Rationale: CD55 down-regulation on airway epithelium correlates with local complement activation observed in hypoxia-associated pulmonary diseases. Therefore, we hypothesized that induction of hypoxia inducible factor 1 alpha (HIF-1α) in hypoxic airway epithelium, mediates CD55 down-regulation. Methods: Chetomin and HIF-1α siRNA inhibited HIF-1α in hypoxic SAECs (1% O2), and mice lungs (10% O2). DMOG mediated HIF-1α stabilization in normoxic SAECs and mice lungs (21% O2). Transduction of SAECs with AdCA5 also stabilized H
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16

Chuang, Kun-Han, and 莊昆翰. "THE ROLES OF GLUCOSAMINE IN LUNG PATHOPHYSIOLOGY." Thesis, 2013. http://ndltd.ncl.edu.tw/handle/36926312671995191205.

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博士<br>國立陽明大學<br>生理學研究所<br>101<br>Acute phase of lung inflammatory diseases, such as acute lung injury (ALI) and acute respiratory distress syndrome (ARDS), is usually accompanied with disruption of alveolar-capillary barrier and induction of inflammatory-related cytokines and chemokines. Glucosamine, a natural occurring amino monosaccharide highly concentrated in connective tissues, is a joint supplement widely used in the treatment of arthritis. Recently, glucosamine has begun to be considered the potential anti-inflammatory properties to treat the other inflammation disease and anti-cell pro
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17

Akhand, Saeed Salehin. "Role of a putative bacterial lipoprotein in Pseudomonas aeruginosa-mediated cytotoxicity toward airway cells." Thesis, 2014. http://hdl.handle.net/1805/5629.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>The patients with Cystic fibrosis (CF), an inherent genetic disorder, suffer from chronic bacterial infection in the lung. In CF, modification of epithelial cells leads to alteration of the lung environment, such as inhibition of ciliary bacterial clearance and accumulation of thickened mucus in the airways. Exploiting these conditions, opportunistic pathogens like Pseudomonas aeruginosa cause lifelong persistent infection in the CF lung by forming into antibiotic-resistant aggregated communities called biofilms. Airway infecti
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