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Artículos de revistas sobre el tema "Memory Methylene blue"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 13 de febrero de 2022

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Gonzalez-Lima, F. "Extinction Memory Improvement by the Metabolic Enhancer Methylene Blue". Learning & Memory 11, n.º 5 (1 de septiembre de 2004): 633–40. http://dx.doi.org/10.1101/lm.82404.

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Rojas, Julio C., Aleksandra K. Bruchey y F. Gonzalez-Lima. "Neurometabolic mechanisms for memory enhancement and neuroprotection of methylene blue". Progress in Neurobiology 96, n.º 1 (enero de 2012): 32–45. http://dx.doi.org/10.1016/j.pneurobio.2011.10.007.

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Callaway, Narriman Lee, Penny D. Riha, Aleksandra K. Bruchey, Zeenat Munshi y F. Gonzalez-Lima. "Methylene blue improves brain oxidative metabolism and memory retention in rats". Pharmacology Biochemistry and Behavior 77, n.º 1 (enero de 2004): 175–81. http://dx.doi.org/10.1016/j.pbb.2003.10.007.

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Echevarria, David J., Erika M. Caramillo y Francisco Gonzalez-Lima. "Methylene Blue Facilitates Memory Retention in Zebrafish in a Dose-Dependent Manner". Zebrafish 13, n.º 6 (diciembre de 2016): 489–94. http://dx.doi.org/10.1089/zeb.2016.1282.

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Katzoff, Ayelet, Tziona Ben-Gedalya, Itay Hurwitz, Nimrod Miller, Yehoshua Z. Susswein y Abraham J. Susswein. "Nitric Oxide Signals That Aplysia Have Attempted to Eat, a Necessary Component of Memory Formation After Learning That Food Is Inedible". Journal of Neurophysiology 96, n.º 3 (septiembre de 2006): 1247–57. http://dx.doi.org/10.1152/jn.00056.2006.

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Inhibiting nitric oxide (NO) synthesis during learning that food is inedible in Aplysia blocks subsequent memory formation. To gain insight into the function of NO transmission during learning we tested whether blocking NO synthesis affects aspects of feeding that are expressed both in a nonlearning context and during learning. Inhibiting NO synthesis with L-NAME and blocking guanylyl cyclase with methylene blue decreased the efficacy of ad libitum feeding. D-NAME had no effect. L-NAME also decreased rejection responses frequency, but did not affect rejection amplitude. The effect of L-NAME was explained by a decreased signaling that efforts to swallow are not successful, leading to a decreased rejection rate, and a decreased ability to reposition and subsequently consume food in ad libitum feeding. Signaling that animals have made an effort to swallow is a critical component of learning that food is inedible. Stimulation of the lips with food alone did not produce memory, but stimulation combined with the NO donor SNAP did produce memory. Exogenous NO at a concentration causing memory also excited a key neuron responding to NO, the MCC. Block of the cGMP second-messenger cascade during training by methylene blue also blocked memory formation after learning. Our data indicate that memory arises from the contingency of three events during learning that food is inedible. One of the events is efforts to swallow, which are signaled by NO by cGMP.

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Li, Lei, Li Qin, Hai-long Lu, Ping-Jing Li, Yuan-Jian Song y Rong-Li Yang. "Methylene blue improves streptozotocin-induced memory deficit by restoring mitochondrial function in rats". Brain Research 1657 (febrero de 2017): 208–14. http://dx.doi.org/10.1016/j.brainres.2016.12.024.

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Riha, Penny D., Aleksandra K. Bruchey, David J. Echevarria y F. Gonzalez-Lima. "Memory facilitation by methylene blue: Dose-dependent effect on behavior and brain oxygen consumption". European Journal of Pharmacology 511, n.º 2-3 (marzo de 2005): 151–58. http://dx.doi.org/10.1016/j.ejphar.2005.02.001.

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Al-Tahan, Farid J. "The effect of L-arginine and its antagonist L-NAME and Methylene blue on sensory and cognitive function in mice". Iraqi Journal of Veterinary Medicine 36, n.º 1 (30 de junio de 2012): 99–106. http://dx.doi.org/10.30539/iraqijvm.v36i1.553.

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The present study was done to focus light on possible enhancement of the functional performance of male mice and female in neuronal behaviors by using L-arginine as a precursor of nitric oxide (NO). The results showed increase of latency period to reach the novel object in L-arginine treated groups and decrease in both L-NAME and methylene blue treated groups in both periods of treatment (15 and 30) days and were more prominent in male than in female mice as compared with control groups. Similar results were observed in passive avoidance latency period to enter the dark compartment. There was a reduction in latency period to reach the alternative arm of T-maze test in L-arginine treated groups and increase in both L-NAME and methylene blue treated groups in both periods of treatment (15 and 30) days in both genders. It could be concluded that L-arginine-NO pathway plays an important role in improving memory in male more than female mice.

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Bunge, Frank, Sander van den Driesche y Michael J. Vellekoop. "Gas Supply through Agarose Walls in Cell Culturing Microchips". Advances in Science and Technology 100 (octubre de 2016): 115–19. http://dx.doi.org/10.4028/www.scientific.net/ast.100.115.

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We present a novel structure to supply gases to microchambers in microfluidic chips. An exemplary application is the continuous feeding of oxygen and CO2 for on-chip cell cultivation of mammalian cells. In our device, the surrounding air diffuses into the culture medium inside the chip through a porous wall of agarose hydrogel resulting in an easy and robust design. One common method is the usage of gas permeable PDMS chips. However, liquid medium in which the cells grow is absorbed by PDMS causing unknown concentrations and memory effects. Another possibility is a complex setup where medium with already dissolved gas is pumped constantly through the chip. We designed and realized a silicon and borosilicate glass chip containing a gas permeable wall of agarose preventing leakage of medium. In order to precisely position the walls in the chip, we made use of surficial phaseguides (50nm high). The blue-bottle-experiment makes the effective dissipation of oxygen visible when the colorless leucomethylen-blue reacts to methylene-blue. Successful results were achieved when applying 0.5 g/l methylene blue, 10 g/l glucose and a pH of 12.6 set by a buffer solution. As a result a continuous color gradient through the chip was obtained, which reflects the oxygen gradient and confirms the oxygen diffusion.

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Callaway, Narriman Lee, Penny D. Riha, Kathryn M. Wrubel, David McCollum y F. Gonzalez-Lima. "Methylene blue restores spatial memory retention impaired by an inhibitor of cytochrome oxidase in rats". Neuroscience Letters 332, n.º 2 (octubre de 2002): 83–86. http://dx.doi.org/10.1016/s0304-3940(02)00827-3.

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Khan, Muhammad Umair, Gul Hassan y Jinho Bae. "Flexible Resistive Switching Memory with a Schottky Diode Function Based on a Zinc Oxide/Methylene Blue Heterojunction". Journal of Electronic Materials 49, n.º 8 (16 de mayo de 2020): 4764–72. http://dx.doi.org/10.1007/s11664-020-08200-z.

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Mori, Takashi, Naoki Koyama, Tatsuya Segawa, Masahiro Maeda, Nobuhiro Maruyama, Noriaki Kinoshita, Huayan Hou, Jun Tan y Terrence Town. "Methylene Blue Modulates β-Secretase, Reverses Cerebral Amyloidosis, and Improves Cognition in Transgenic Mice". Journal of Biological Chemistry 289, n.º 44 (25 de agosto de 2014): 30303–17. http://dx.doi.org/10.1074/jbc.m114.568212.

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Amyloid precursor protein (APP) proteolysis is required for production of amyloid-β (Aβ) peptides that comprise β-amyloid plaques in the brains of patients with Alzheimer disease (AD). Here, we tested whether the experimental agent methylene blue (MB), used for treatment of methemoglobinemia, might improve AD-like pathology and behavioral deficits. We orally administered MB to the aged transgenic PSAPP mouse model of cerebral amyloidosis and evaluated cognitive function and cerebral amyloid pathology. Beginning at 15 months of age, animals were gavaged with MB (3 mg/kg) or vehicle once daily for 3 months. MB treatment significantly prevented transgene-associated behavioral impairment, including hyperactivity, decreased object recognition, and defective spatial working and reference memory, but it did not alter nontransgenic mouse behavior. Moreover, brain parenchymal and cerebral vascular β-amyloid deposits as well as levels of various Aβ species, including oligomers, were mitigated in MB-treated PSAPP mice. These effects occurred with inhibition of amyloidogenic APP proteolysis. Specifically, β-carboxyl-terminal APP fragment and β-site APP cleaving enzyme 1 protein expression and activity were attenuated. Additionally, treatment of Chinese hamster ovary cells overexpressing human wild-type APP with MB significantly decreased Aβ production and amyloidogenic APP proteolysis. These results underscore the potential for oral MB treatment against AD-related cerebral amyloidosis by modulating the amyloidogenic pathway.

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Telch, Michael J., Aleksandra K. Bruchey, David Rosenfield, Adam R. Cobb, Jasper Smits, Sandra Pahl y F. Gonzalez-Lima. "Effects of Post-Session Administration of Methylene Blue on Fear Extinction and Contextual Memory in Adults With Claustrophobia". American Journal of Psychiatry 171, n.º 10 (octubre de 2014): 1091–98. http://dx.doi.org/10.1176/appi.ajp.2014.13101407.

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Sadovnikova, Irina S., Artem P. Gureev, Daria A. Ignatyeva, Maria V. Gryaznova, Ekaterina V. Chernyshova, Ekaterina P. Krutskikh, Anastasia G. Novikova y Vasily N. Popov. "Nrf2/ARE Activators Improve Memory in Aged Mice via Maintaining of Mitochondrial Quality Control of Brain and the Modulation of Gut Microbiome". Pharmaceuticals 14, n.º 7 (23 de junio de 2021): 607. http://dx.doi.org/10.3390/ph14070607.

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Aging is one of the most serious factors for central nervous dysfunctions, which lead to cognitive impairment. New highly effective drugs are required to slow the development of cognitive dysfunction. This research studied the effect of dimethyl fumarate (DMF), methylene blue (MB), and resveratrol (RSV) on the cognitive functions of 15-month-old mice and their relationship to the maintenance of mitochondrial quality control in the brain and the bacterial composition of the gut microbiome. We have shown that studied compounds enhance mitochondrial biogenesis, mitophagy, and antioxidant defense in the hippocampus of 15-month-old mice via Nrf2/ARE pathway activation, which reduces the degree of oxidative damage to mtDNA. It is manifested in the improvement of short-term and long-term memory. We have also shown that memory improvement correlates with levels of Roseburia, Oscillibacter, ChristensenellaceaeR-7, Negativibacillus, and Faecalibaculum genera of bacteria. At the same time, long-term treatment by MB induced a decrease in gut microbiome diversity, but the other markers of dysbiosis were not observed. Thus, Nrf2/ARE activators have an impact on mitochondrial quality control and are associated with a positive change in the composition of the gut microbiome, which together lead to an improvement in memory in aged mice.

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Jia, Hong Jing, Fang Duan y Cong Bin Fan. "Synthesis and Properties of a New Unsymmetry Diarylethene 1-{2-methyl-[5-(4-methylene-bromine)phenyl]-3-thienyl}-2-[2-methyl-5-(10-phenanthrenyl)-3-thienyl]perfluorocyclopentene". Advanced Materials Research 952 (mayo de 2014): 117–20. http://dx.doi.org/10.4028/www.scientific.net/amr.952.117.

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A new photochromic diarylethene based on a phenanthrene, which is named 1-{2-methyl-[5-(4-methylene-bromine) phenyl]-3-thienyl}-2-[2-methyl-5-(10-phenanthrenyl)-3-thienyl] perfluorocyclopentene 1o, was designed and constructed successfully. We used it to accomplished recording by optical storage technology as memory medium. And its properties have been discussed systematically, including photochromic both in dichloromethane solution and in PMMA film and kinetics experiments. The result indicated that its photochromic behaviors could be modulated by UV/Vis light, changing between colorless and blue in dichloromethane solution and in PMMA film, respectively. What is more, the kinetic experiments illustrated that the cyclization/cycloreversion process of this compound was determined to be the zeroth/first reaction. In addition, the results demonstrated that the unsymmetrical diarylethene compound 1o, which we had synthesized, had attractive properties for potential application in optical storage.

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Orzelska-Górka, Jolanta, Piotr Bernat, Piotr Tutka, Joanna Listos, Ewa Kędzierska, Sylwia Fidecka y Sylwia Talarek. "Modification of NO-cGMP Pathway Differentially Affects Diazepam- and Flunitrazepam-Induced Spatial and Recognition Memory Impairments in Rodents". Neurotoxicity Research 37, n.º 4 (2 de diciembre de 2019): 1036–46. http://dx.doi.org/10.1007/s12640-019-00110-1.

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AbstractThis study investigated the influence of sildenafil and methylene blue (MB), two modulators of the nitric oxide (NO)-cyclic guanosine-3′,5′-monophosphate (cGMP) pathway on amnesic effects of two benzodiazepines (BZs) (diazepam (DZ) and flunitrazepam (FNZ)), in rodents—mice and rats. In the modified elevated plus maze (mEPM) and novel object recognition (NOR) tests, MB given ip at a dose of 5 mg/kg 5 min prior to DZ administration (0.25 or 1 mg/kg, sc) enhanced/induced memory impairment caused by DZ. When MB (2.5, 5, and 10 mg/kg) was applied 5 min prior to FNZ administration (0.05 and 0.1 mg/kg), an effect was opposite and memory impairment induced by FNZ was reduced. When sildenafil (2.5 and 5 mg/kg, ip) was applied 5 min prior to DZ, we observed a reduction of DZ-induced memory deficiency in the mEPM test. A similar effect of sildenafil was shown in the NOR test when the drug was applied at doses of 1.25, 2.5, and 5 mg/kg prior to DZ. In the mEPM test, sildenafil at abovementioned doses had no effects on FNZ-induced memory impairment. In turns, sildenafil administered at doses of 2.5 and 5 mg/kg increased the effect of FNZ on memory impairment in the NOR test. In conclusion, the NO-cGMP pathway is involved differentially into BZs-induced spatial and recognition memory impairments assessed using the NOR and mEPM tests. Modulators of the NO-cGMP pathway affect animal behavior in these tests in a different way depending on what benzodiazepine is applied.

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Zhou, Libin, Joseph Flores, Anastasia Noël, Olivier Beauchet, P. Jesper Sjöström y Andrea C. LeBlanc. "Methylene blue inhibits Caspase-6 activity, and reverses Caspase-6-induced cognitive impairment and neuroinflammation in aged mice". Acta Neuropathologica Communications 7, n.º 1 (diciembre de 2019). http://dx.doi.org/10.1186/s40478-019-0856-6.

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AbstractActivated Caspase-6 (Casp6) is associated with age-dependent cognitive impairment and Alzheimer disease (AD). Mice expressing human Caspase-6 in hippocampal CA1 neurons develop age-dependent cognitive deficits, neurodegeneration and neuroinflammation. This study assessed if methylene blue (MB), a phenothiazine that inhibits caspases, alters Caspase-6-induced neurodegeneration and cognitive impairment in mice. Aged cognitively impaired Casp6-overexpressing mice were treated with methylene blue in drinking water for 1 month. Methylene blue treatment did not alter Caspase-6 levels, assessed by RT-PCR, western blot and immunohistochemistry, but inhibited fluorescently-labelled Caspase-6 activity in acute brain slice intact neurons. Methylene blue treatment rescued Caspase-6-induced episodic and spatial memory deficits measured by novel object recognition and Barnes maze, respectively. Methylene blue improved synaptic function of hippocampal CA1 neurons since theta-burst long-term potentiation (LTP), measured by field excitatory postsynaptic potentials (fEPSPs) in acute brain slices, was successfully induced in the Schaffer collateral-CA1 pathway in methylene blue-treated, but not in vehicle-treated, Caspase-6 mice. Increased neuroinflammation, measured by ionized calcium binding adaptor molecule 1 (Iba1)-positive microglia numbers and subtypes, and glial fibrillary acidic protein (GFAP)-positive astrocytes, were decreased by methylene blue treatment. Therefore, methylene blue reverses Caspase-6-induced cognitive deficits by inhibiting Caspase-6, and Caspase-6-mediated neurodegeneration and neuroinflammation. Our results indicate that Caspase-6-mediated damage is reversible months after the onset of cognitive deficits and suggest that methylene blue could benefit Alzheimer disease patients by reversing Caspase-6-mediated cognitive decline.

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Auchter, Allison M., Douglas W. Barrett, Marie H. Monfils y F. Gonzalez-Lima. "Methylene Blue Preserves Cytochrome Oxidase Activity and Prevents Neurodegeneration and Memory Impairment in Rats With Chronic Cerebral Hypoperfusion". Frontiers in Cellular Neuroscience 14 (20 de mayo de 2020). http://dx.doi.org/10.3389/fncel.2020.00130.

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Handral, Mukund y Manjunath P. M. "Photooxidized Vespa orientalis venom Improves Memory and Learning Activities in Rats". International Journal of Pharmaceutical Sciences and Drug Research 10, n.º 01 (15 de enero de 2018). http://dx.doi.org/10.25004/ijpsdr.2018.100104.

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The work was aimed to study the effect of photooxidized Vespa orientalis venom on memory and learning activity on rats in presence of scopolamine and ondansetron using T maze, Elevated plus maze and passive avoidance. UV radiation exposure of Vespa orientalis venom (VOV) for 15, 30, 45 and 60 min intervals in presence to methylene blue to detoxify venom and studied by change in UV spectrum. Antigenicity study and in in-vivo and in-vitro neutralization study of Photooxidized Vespa orientalis venom (PVOV) against immunoglobulin from hyperimmunized rabbit was performed. The memory and learning activity of PVOV in presence and absence of scopolamine and ondansetron was studied. Forty five minute UV Radiation exposed VOV showed shift in λmax and increase absorbance indicated alternations in venom protein concentration, this in supported when PVOV showed loss of toxicity and decrease in mortality time in neutralization study in mice. Administration of PVOV for 28 days produced a notable improvement in spatial and long memory in rats when subjected in several tasks. When PVOV administered with scopolamine and ondansetron, all the parameters of spatial and long term memory tasks were significantly reduced inferred that PVOV acted by modulating either muscarinic or serotonergic receptors. However, other possibility of low-molecular weight protein and peptides or enzymes, which might also act by serotonergic / cholinergic system that affect CNS action. We concluded that although there is a possibility of employing PVOV in the treatment of depressive and chronic degenerative illnesses as a nonherbal and nonsynthetic alternative for patients not responding to the available therapy.

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