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Literatura académica sobre el tema "Mitoepigenetics"

Autor: Grafiati

Publicado: 14 de marzo de 2023

Última modificación: 26 de julio de 2025

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Artículos de revistas sobre el tema "Mitoepigenetics"

Cavalcante, Giovanna C., Leandro Magalhães, Ândrea Ribeiro-dos-Santos, and Amanda F. Vidal. "Mitochondrial Epigenetics: Non-Coding RNAs as a Novel Layer of Complexity." International Journal of Molecular Sciences 21, no. 5 (2020): 1838. http://dx.doi.org/10.3390/ijms21051838.

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Mitochondria are organelles responsible for several functions involved in cellular balance, including energy generation and apoptosis. For decades now, it has been well-known that mitochondria have their own genetic material (mitochondrial DNA), which is different from nuclear DNA in many ways. More recently, studies indicated that, much like nuclear DNA, mitochondrial DNA is regulated by epigenetic factors, particularly DNA methylation and non-coding RNAs (ncRNAs). This field is now called mitoepigenetics. Additionally, it has also been established that nucleus and mitochondria are constantly

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Lozano-Rosas, María Guadalupe, Enrique Chávez, Alejandro Rusbel Aparicio-Cadena, Gabriela Velasco-Loyden, and Victoria Chagoya de Sánchez. "Mitoepigenetics and hepatocellular carcinoma." Hepatoma Research 4, no. 6 (2018): 19. http://dx.doi.org/10.20517/2394-5079.2018.48.

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Sadakierska-Chudy, Anna, Małgorzata Frankowska, and Małgorzata Filip. "Mitoepigenetics and drug addiction." Pharmacology & Therapeutics 144, no. 2 (2014): 226–33. http://dx.doi.org/10.1016/j.pharmthera.2014.06.002.

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Ghosh, Sourav, Keshav K. Singh, Shantanu Sengupta, and Vinod Scaria. "Mitoepigenetics: The different shades of grey." Mitochondrion 25 (November 2015): 60–66. http://dx.doi.org/10.1016/j.mito.2015.09.003.

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Cao, Ke, Zhihui Feng, Feng Gao, Weijin Zang, and Jiankang Liu. "Mitoepigenetics: An intriguing regulatory layer in aging and metabolic-related diseases." Free Radical Biology and Medicine 177 (December 2021): 337–46. http://dx.doi.org/10.1016/j.freeradbiomed.2021.10.031.

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Lima, Camila Bruna, and Marc‐André Sirard. "Mitoepigenetics: Methylation of mitochondrial DNA is strand‐biased in bovine oocytes and embryos." Reproduction in Domestic Animals 55, no. 10 (2020): 1455–58. http://dx.doi.org/10.1111/rda.13786.

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Manev, Hari, and Svetlana Dzitoyeva. "Progress in mitochondrial epigenetics." BioMolecular Concepts 4, no. 4 (2013): 381–89. http://dx.doi.org/10.1515/bmc-2013-0005.

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AbstractMitochondria, intracellular organelles with their own genome, have been shown capable of interacting with epigenetic mechanisms in at least four different ways. First, epigenetic mechanisms that regulate the expression of nuclear genome influence mitochondria by modulating the expression of nuclear-encoded mitochondrial genes. Second, a cell-specific mitochondrial DNA content (copy number) and mitochondrial activity determine the methylation pattern of nuclear genes. Third, mitochondrial DNA variants influence the nuclear gene expression patterns and the nuclear DNA (ncDNA) methylation

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Nikolac Perkovic, Matea, Alja Videtic Paska, Marcela Konjevod, et al. "Epigenetics of Alzheimer’s Disease." Biomolecules 11, no. 2 (2021): 195. http://dx.doi.org/10.3390/biom11020195.

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There are currently no validated biomarkers which can be used to accurately diagnose Alzheimer’s disease (AD) or to distinguish it from other dementia-causing neuropathologies. Moreover, to date, only symptomatic treatments exist for this progressive neurodegenerative disorder. In the search for new, more reliable biomarkers and potential therapeutic options, epigenetic modifications have emerged as important players in the pathogenesis of AD. The aim of the article was to provide a brief overview of the current knowledge regarding the role of epigenetics (including mitoepigenetics) in AD, and

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Booz, George W., Gaelle P. Massoud, Raffaele Altara, and Fouad A. Zouein. "Unravelling the impact of intrauterine growth restriction on heart development: insights into mitochondria and sexual dimorphism from a non-hominoid primate." Clinical Science 135, no. 14 (2021): 1767–72. http://dx.doi.org/10.1042/cs20210524.

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Abstract Fetal exposure to an unfavorable intrauterine environment programs an individual to have a greater susceptibility later in life to non-communicable diseases, such as coronary heart disease, but the molecular processes are poorly understood. An article in Clinical Science recently reported novel details on the effects of maternal nutrient reduction (MNR) on fetal heart development using a primate model that is about 94% genetically similar to humans and is also mostly monotocous. MNR adversely impacted fetal left ventricular (LV) mitochondria in a sex-dependent fashion with a greater e

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Salunke, Chaitali* Deokar Shivprasad Dr. Kawade Rajendra Pathade Pratiksha. "Epigenetic Modification in Alzheimer Disease." International Journal of Pharmaceutical Sciences 2, no. 11 (2024): 1357–65. https://doi.org/10.5281/zenodo.14222567.

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Alzheimer’s disease (AD) is a devastating neurodegenerative disorder characterized by progressive cognitive decline and memory loss, imposing a significant burden on affected individuals and their families. Despite the recent promising progress in therapeutic approaches, more needs to be done to understand the intricate molecular mechanisms underlying the development and progression of AD. Alzheimer’s disease is characterized by the formation and deposit abnormal peptides such as amyloid plaques and neurofibrillary tangles in the Brain. There are currently no validated biomarkers w

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Tesis sobre el tema "Mitoepigenetics"

STOCCORO, ANDREA. "Mitoepigenetics investigations in neurodegenerative diseases." Doctoral thesis, Università di Siena, 2019. http://hdl.handle.net/11365/1072183.

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Neurodegenerative diseases (NDs) represent a group of disorders characterized by the progressive neuronal loss in specific areas of the central nervous system. NDs are incurable and often fatal shortly after diagnosis. The global prevalence of these disorders is dramatically increasing worldwide as populations age and life expectancies increase. The identification of valuable biomarkers for an early diagnosis is of outmost importance as it would promote early interventions able to prevent or delay as much as possible the onset of the disease. Despite their high heterogeneity and the diff

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Capítulos de libros sobre el tema "Mitoepigenetics"

Manev, Hari. "Mitoepigenetics and Neuropsychiatric Disorders." In Epigenetics in Psychiatry. Elsevier, 2014. http://dx.doi.org/10.1016/b978-0-12-417114-5.00022-x.

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