Bibliografías temáticas / Mouse aorta

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Literatura académica sobre el tema "Mouse aorta"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 12 de febrero de 2022

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Artículos de revistas sobre el tema "Mouse aorta"

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Zhu, Guanglang, Huiying Sun, Jiannan Wang, et al. "In Vivo Detection and Measurement of Aortic Aneurysm and Dissection in Mouse Models Using Microcomputed Tomography with Contrast Agent." Contrast Media & Molecular Imaging 2019 (March 6, 2019): 1–9. http://dx.doi.org/10.1155/2019/5940301.

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Objectives. The aim of this study was to evaluate the potential of microcomputed tomography (micro-CT) using the intravascular contrast agent ExiTron nano 12000 for aorta imaging and monitoring the dynamic changing process of the aorta in mouse models with aortic aneurysm and dissection. Materials and Methods. Experiments were performed on healthy mice and mice with aortic dissection. Mice that were developing aortic dissection and healthy mice underwent micro-CT imaging after injection of ExiTron nano 12000. Time-dependent signal enhancement (at 1, 2, 3, 6, and 12 hours after intravenous inje
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Guo, X., Y. Kono, R. Mattrey, and G. S. Kassab. "Morphometry and strain distribution of the C57BL/6 mouse aorta." American Journal of Physiology-Heart and Circulatory Physiology 283, no. 5 (2002): H1829—H1837. http://dx.doi.org/10.1152/ajpheart.00224.2002.

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The goal of the present study was to obtain a systematic set of data along the length of the mouse aorta to study variations of morphometry (diameter, wall thickness, and curvature), strain, and stress of the mouse aorta. Five mice were imaged with a 13-MHz ultrasound probe to determine the in vivo diameter along the aorta. A cast was made of these aortas to validate the ultrasonic diameter measurements. The root mean squared and systematic errors for these measurements were 12.6% and 6.4% of the mean ultrasound diameter, respectively. The longitudinal variations of geometry, stress, and strai
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Silswal, Neerupma, Nikhil K. Parelkar, Michael J. Wacker, Mostafa Badr та Jon Andresen. "PPARα-Independent Arterial Smooth Muscle Relaxant Effects of PPARαAgonists". PPAR Research 2012 (2012): 1–10. http://dx.doi.org/10.1155/2012/302495.

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We sought to determine direct vascular effects of peroxisome proliferator-activated receptor alpha (PPARα) agonists using isolated mouse aortas and middle cerebral arteries (MCAs). The PPARαagonists GW7647, WY14643, and gemfibrozil acutely relaxed aortas held under isometric tension and dilated pressurized MCAs with the following order of potency: GW7647≫WY14643>gemfibrozil. Responses were endothelium-independent, and the use of PPARαdeficient mice demonstrated that responses were also PPARα-independent. Pretreating arteries with high extracellular K+attenuated PPARαagonist-mediated relaxat
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Hemmeryckx, Bianca, Marc F. Hoylaerts, Eveline Deloose, et al. "Age-associated pro-inflammatory adaptations of the mouse thoracic aorta." Thrombosis and Haemostasis 110, no. 10 (2013): 785–94. http://dx.doi.org/10.1160/th13-01-0022.

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SummaryArterial ageing may be associated with a reduction in vasodilation due to increased reactive oxygen species (ROS) production, whereas endothelial cell activation induces procoagulant changes. However, little is known on the effect of ageing on expression of anticoagulant endothelial markers such as endothelial protein C receptor (EPCR). To study age-associated alterations in smooth muscle cell (SMC) and endothelial cell (EC) structure and function, the aorta was isolated from 10-week-and 12– and 24-month-old C57BL/6J mice and analysed for its expression of genes involved in senescence,
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Agianniotis, A., A. Rachev, and N. Stergiopulos. "Active axial stress in mouse aorta." Journal of Biomechanics 45, no. 11 (2012): 1924–27. http://dx.doi.org/10.1016/j.jbiomech.2012.05.025.

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Cacicedo, José M., Marie-Soleil Gauthier, Nathan K. Lebrasseur, Ravi Jasuja, Neil B. Ruderman, and Yasuo Ido. "Acute exercise activates AMPK and eNOS in the mouse aorta." American Journal of Physiology-Heart and Circulatory Physiology 301, no. 4 (2011): H1255—H1265. http://dx.doi.org/10.1152/ajpheart.01279.2010.

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Exercise can prevent endothelial cell (EC) dysfunction and atherosclerosis even in the absence of improvements in plasma lipids. However, the mechanisms responsible for these effects are incompletely understood. In this study we examined in mice whether an acute bout of exercise activates enzymes that could prevent EC dysfunction, such as AMP-activated protein kinase (AMPK) and endothelial nitric oxide synthase (eNOS). We also examined whether exercise alters known regulators of these enzymes. C57BL/6 mice underwent a single bout of exhaustive treadmill exercise after which their aortas were a
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Lalli, M. Jane, Shunichi Shimizu, Roy L. Sutliff, Evangelia G. Kranias, and Richard J. Paul. "[Ca2+]ihomeostasis and cyclic nucleotide relaxation in aorta of phospholamban-deficient mice." American Journal of Physiology-Heart and Circulatory Physiology 277, no. 3 (1999): H963—H970. http://dx.doi.org/10.1152/ajpheart.1999.277.3.h963.

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Phospholamban (PLB), a protein localized in the sarcoplasmic reticulum (SR), inhibits the SR Ca2+-ATPase; phosphorylation of PLB relieves this inhibition. We previously reported significant differences in contractility in aorta from mice in which the gene for PLB was ablated (PLB−). In this study, we measured intracellular Ca2+concentration ([Ca2+]i) with fura 2 in the intact mouse aorta to more directly test the hypothesis that these changes are ascribable to altered SR function in vivo. Ten micromoles per liter of the α-agonist phenylephrine (PE) increased [Ca2+]imonotonically to a steady st
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Amirbekian, Smbat, Robert C. Long, Michelle A. Consolini, et al. "In vivo assessment of blood flow patterns in abdominal aorta of mice with MRI: implications for AAA localization." American Journal of Physiology-Heart and Circulatory Physiology 297, no. 4 (2009): H1290—H1295. http://dx.doi.org/10.1152/ajpheart.00889.2008.

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Abdominal aortic aneurysms (AAA) localize in the infrarenal aorta in humans, while they are found in the suprarenal aorta in mouse models. It has been shown previously that humans experience a reversal of flow during early diastole in the infrarenal aorta during each cardiac cycle. This flow reversal causes oscillatory wall shear stress (OWSS) to be present in the infrarenal aorta of humans. OWSS has been linked to a variety of proatherogenic and proinflammatory factors. The presence of reverse flow in the mouse aorta is unknown. In this study we investigated blood flow in mice, using phase-co
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Racho El-Akouri, R., G. Kurlberg, G. Dindelegan, J. Molne, A. Wallin, and M. Brannstrom. "Heterotopic uterine transplantation by vascular anastomosis in the mouse." Journal of Endocrinology 174, no. 2 (2002): 157–66. http://dx.doi.org/10.1677/joe.0.1740157.

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A method of heterotopic uterine transplantation was developed in the mouse as a model system for studies of uterine function and transplant immunology of the uterus. The model involved transplantation of the right uterine horn and the cervix by vascular anastomosis to a donor animal with the intact native uterus remaining in situ. F1-hybrids of inbred C57BL/6 x CBA/ca (B6 CBAF1) mice of 6-8 weeks of age (n=42) were used. The specific pelvic vascular anatomy of these mice was first examined by intra-aortal injection of a two-component silicon-rubber curing agent. The surgery of the donor animal
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Galkina, Elena, Alexandra Kadl, John Sanders, Danielle Varughese, Ian J. Sarembock, and Klaus Ley. "Lymphocyte recruitment into the aortic wall before and during development of atherosclerosis is partially L-selectin dependent." Journal of Experimental Medicine 203, no. 5 (2006): 1273–82. http://dx.doi.org/10.1084/jem.20052205.

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Atherosclerosis is an inflammatory disease of large arteries. Flow cytometry of aortic cell suspensions showed that B and T lymphocytes and some macrophages and dendritic cells are already present in the adventitia of normal/noninflamed mouse aortas. Adoptively transferred lymphocytes constitutively homed to the aorta and resided within the adventitia up to 7 d after transfer. Lymphocyte trafficking into normal/noninflamed or atherosclerosis-prone aortas was partially L-selectin dependent. Antigen-activated dendritic cells induced increased T lymphocyte proliferation within the aorta 72 h afte
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Tesis sobre el tema "Mouse aorta"

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Binagui-Casas, Anahi Liliana. "Analysis of the role of Flk-1 during mouse haematopoietic stem cell development." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/31134.

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In the mouse embryo, the first definitive haematopoietic stem cells (HSCs), capable of repopulating adult irradiated mice, emerge at mid-gestation by embryonic day E11. At this stage, the aorta-gonad-mesonephros (AGM) region is able to initiate and expand HSCs. Recently, it has been shown that the development of HSC in the AGM region results from the maturation of haematopoietic precursors called pre-HSCs. Mounting evidence points at an endothelial origin for these cells, the haematogenic endothelium. Analysis of VEGFs mutants, a critical pathway for endothelial developement, suggested that it
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Davel, Ana Paula Couto. "Alterações vasculares induzidas pelo tratamento crônico com Isoproterenol: Investigação dos subtipos de receptores b-adrenérgicos envolvidos e da possível geração de um processo inflamatório local." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-13032009-171655/.

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Neste estudo investigou-se: 1) os subtipos de receptores b-adrenérgicos (b-AR) envolvidos nas alterações de reatividade vascular induzida pela hiperativação dos receptores b-AR com do uso de camundongos nocaute para os receptores b1- ou b2-AR e selvagens tratados por 7 dias com isoproterenol; 2) a expressão gênica e protéica de mediadores pró-inflamatórios na aorta de ratos tratados por 7 dias com isoproterenol. Os dados demonstram que: em aorta de camundongos selvagens o receptor b1-AR é mais importante em mediar vasodilatação, enquanto o b2AR modula negativamente a contração vascular de mane
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Krishnamurthy, Varun K. "Biomechanical and Molecular Approaches to Aortic Valve Disease in a Mouse Model." University of Cincinnati / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1354297165.

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Pfeiffer, Brent J. "Role of Proa(2)I collagen chains and collagen crosslinking in thoracic aortic biochemical integrity during aging using the OIM mouse model." Diss., Columbia, Mo. : University of Missouri-Columbia, 2006. http://hdl.handle.net/10355/4397.

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Thesis (Ph. D.)--University of Missouri-Columbia, 2006.<br>Title from title screen of research.pdf file (viewed on December 22, 2006). The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Vita. "May 2006" Includes bibliographical references.
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Otto, Anne. "The protection of rosuvastatin and ramipril against the development of nitrate tolerance in the rat and mouse aorta." Doctoral thesis, Universite Libre de Bruxelles, 2006. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210861.

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Organic nitrates, such as nitroglycerine (NTG), are widely used for their potent vasodilator capacity in the management of coronary artery disease and heart failure. Unfortunately, their beneficial effect is rapidly lost due to the development of nitrate tolerance, which is translated by an impaired vasorelaxation to NTG and an increased oxidative stress production. Although the mechanisms of the development of nitrate tolerance are still not fully elucidated, much interest has been focused in treating nitrate-receiving patients together with other drugs in order to overcome the development of
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Ohmura, Koichiro. "Emergence of T,B, and Myeloid Lineage-Committed as well as Multipotent Hemopoietic Progenitors in the Aorta-Gonad-Mesonephros Region of Day 10 Fetuses of the Mouse." Kyoto University, 2000. http://hdl.handle.net/2433/181268.

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Bush, David Roy. "The LC/MS/MS Analysis of Pyridinoline and Desmosines in Hypertensive Mouse Aorta, Elucidation of Arginine and Proline Fragmentation for the Analysis of Arginine Metabolism in Mosquitoes by LC/MS/MS, and Mudpit Identification of B. Pseudomallei Proteins in Urine." Diss., The University of Arizona, 2013. http://hdl.handle.net/10150/293615.

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This dissertation presents an investigation into the application of mass spectrometry to the detection and quantitation of proteins and amino acids in complex biological samples. This is accomplished by two dimensional chromatography (strong cation exchange / reverse phase) coupled to tandem mass spectrometry followed by peptide spectrum matching for the detection of Burkholderia pseudomallei proteins in infected patient urine samples and reverse phase liquid chromatography coupled to a triple quadrupole and quadrupole time of flight mass spectrometers for the quantitation of cross-linked or
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Haskett, Darren. "Toward a Method for Biomechanical Determination of Aneurysm Progression in Mouse Models." Thesis, The University of Arizona, 2011. http://hdl.handle.net/10150/144598.

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Aortic aneurysm is a complex disease manifesting in a localized dilation of the aorta developing over years and carries with it a significant chance of rupture resulting in death. As only surgical methods are currently available for treatment, there is a need to understand the underlying mechanisms of the disease and how they develop and lead to expansion and rupture. Thus, the study of the formation and progression of aneurysm has also focused on quantifying any changes observed in fiber realignment and altered mechanical properties leading to vascular disease. Animal models of aneurismal dis
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Xing, Yi. "An ERK-dependent signaling pathway regulated by miRs contributes to an aging-related decrease in smooth muscle contractility by inhibiting caldesmon phosphorylation." Thesis, 2019. https://hdl.handle.net/2144/37004.

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This project focused on extracellular signal-regulated kinase (ERK) and focal adhesion proteins related to ERK activity, and found a novel signaling pathway contributing to aging-related defects in smooth muscle contractility. Previous members of our lab have used ERK inhibitors to demonstrate the role of ERK in smooth muscle contraction. Dr. Nicholson used the ERK inhibitor FR 18024 and noted that, in the presence of this inhibitor phenylephrine (PE) induced a higher stress increase in young mouse aortas compared to old aortas. Inhibition of the kinase ERK abolished this difference. He also
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Hsiao, Shih-Hao, and 蕭士豪. "Effects of Acrylamide on Isolated Rat Aorta and Mouse Phrenic-nerve Diaphragm Toxicity." Thesis, 2019. http://ndltd.ncl.edu.tw/cgi-bin/gs32/gsweb.cgi/login?o=dnclcdr&s=id=%22107NCHU5252009%22.&searchmode=basic.

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碩士<br>國立中興大學<br>食品安全研究所<br>107<br>Purpose: Acrylamide (AA) can be formed during high-temperature frying or baking in the presence of both free asparagine and reducing sugars. It is known that AA displays genotoxicity, carcinogenicity, neurotoxicity and reproductive toxicity in laboratory animals. However, the related mechanisms of acrylamide-induced vasotoxicity and neuro-muscular toxicity are still unclear. This aim of this study was to investigate the possible mechanisms of vasotoxicity and neuro-muscular toxicity of acrylamide. Method: Vascular toxicity was studied by using an isolated rat
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Libros sobre el tema "Mouse aorta"

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Handa, Shivalika. Investigating smooth muscle cell marker gene expression in distinct regions of the mouse aorta during early atherosclerosis by microdissection and mRNA analysis. 2006.

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Capítulos de libros sobre el tema "Mouse aorta"

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Mateo, Jesús, Marina Benito, Samuel España, et al. "Magnetic Resonance Imaging of the Atherosclerotic Mouse Aorta." In Methods in Molecular Biology. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2929-0_29.

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Hoit, Brian D. "Echocardiographic Assessment of the Mouse Heart and Aorta." In Developments in Cardiovascular Medicine. Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1653-8_12.

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Beer, Michael, Sandra Doepping, Markus Hildner, et al. "Laser-Capture Microdissection of Hyperlipidemic/ApoE−/− Mouse Aorta Atherosclerosis." In Methods in Molecular Biology. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-163-5_35.

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Wang, Yu, Lisa A. Cassis, and Sean E. Thatcher. "Use of a Fluorescent Substrate to Measure ACE2 Activity in the Mouse Abdominal Aorta." In Methods in Molecular Biology. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7030-8_5.

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Menon, Prashanthi, and Edward A. Fisher. "Immunostaining of Macrophages, Endothelial Cells, and Smooth Muscle Cells in the Atherosclerotic Mouse Aorta." In Methods in Molecular Biology. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2929-0_9.

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Trachet, B., P. Segers, F. Claes, and A. Berges. "Measuring Mouse Abdominal Aorta Dimensions in Vivo: A Comparison between (3D) Ultrasound and Micro-CT." In IFMBE Proceedings. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-14515-5_107.

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Rivera-Torres, José. "Analysis of Gene and Protein Expression in Atherosclerotic Mouse Aorta by Western Blot and Quantitative Real-Time PCR." In Methods in Molecular Biology. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2929-0_21.

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Andrés-Manzano, M. Jesús, Vicente Andrés, and Beatriz Dorado. "Oil Red O and Hematoxylin and Eosin Staining for Quantification of Atherosclerosis Burden in Mouse Aorta and Aortic Root." In Methods in Molecular Biology. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2929-0_5.

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McArdle, Sara, Ekaterina Koltsova, Grzegorz Chodaczek, and Klaus Ley. "Live Cell Multiphoton Microscopy of Atherosclerotic Plaques in Mouse Aortas." In Cardiovascular Imaging. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09268-3_7.

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McLean, Sean E., Brigham H. Mecham, Cassandra M. Kelleher, Thomas J. Mariani, and Robert P. Mecham. "Extracellular matrix gene expression in the developing mouse aorta." In Extracellular Matrix in Development and Disease. Elsevier, 2005. http://dx.doi.org/10.1016/s1574-3349(05)15003-0.

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Actas de conferencias sobre el tema "Mouse aorta"

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Agianniotis, Aristotelis, Alexander Rachev, and Nikos Stergiopulos. "Active Axial Stress in Mouse Aorta." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80102.

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Arteries exhibit a complex mechanical behavior due to their complex structure and composition. When smooth muscle cells (SMCs) are stimulated to contract, an artery constricts and develops the so called active stress in the arterial wall. Assuming that the SMCs are oriented in the circumferential direction most of studies consider solely development of the circumferential active stress [1]. However, histological findings show existence of SMCs oriented in both the circumferential and axial direction; these vessels manifest biaxial contractile response [2, 3]. In this study we also observed bia
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Mateasik, Anton, Frantisek Uherek, Dusan Chorvat, Jr., D. Tazka, and J. Kyselovic. "Imaging of mouse aorta using OCT." In Saratov Fall Meeting 2000, edited by Valery V. Tuchin. SPIE, 2001. http://dx.doi.org/10.1117/12.431515.

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Bols, Joris, Bram Trachet, Joris Degroote, et al. "Structural Simulation of a Mouse-Specific Abdominal Aorta." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53635.

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In the last years there is an increasing interest in patient-specific simulations of the fluid-structure interaction in aortic aneurysms, a.o. to better understand the growth and development of the aneurysm and to support diagnosis through assessment of its rupture potential. In order to verify these simulations, validation is an important and difficult task. Given ethical constraints, the slow time course of the disease in humans and the absence of true baseline data in a healthy aorta, these studies are difficult to perform in humans. This is particularly true for aneurysm rupture research,
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Haskett, Darren, Greg Johnson, Mohamad Azhar, and Jonathan Vande Geest. "Biomechanical and Microstructural Analysis of Wildtype (C57BL6) Mouse Aorta." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53729.

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It is generally accepted that the formation of an aneurysm in the infrarenal aorta is a complex and multi-factorial disease, however little is known about how biomechanical factors may play a role in the progression of aneurysmal disease. Although it is known that human aneurysmal tissue is remodeled in the disease process [1] and that such reorganization leads to altered function [2], the underlying mechanisms by which such changes remains an important unanswered question in the literature. The purpose of this study is to develop a means for determining the biomechanical alterations that occu
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Wijesinghe, Philip, Niloufer J. Johansen, Andrea Curatolo, David D. Sampson, Ruth Ganss, and Brendan F. Kennedy. "Optical coherence elastography for cellular-scale stiffness imaging of mouse aorta." In International Conference on Biophotonics V, edited by David D. Sampson, Dennis L. Matthews, Jürgen Popp, Halina Rubinsztein-Dunlop, and Brian C. Wilson. SPIE, 2017. http://dx.doi.org/10.1117/12.2269903.

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Trachet, Bram, Marjolijn Renard, Joris Bols, Steven Staelens, Bart Loeys, and Patrick Segers. "Hemodynamics in Ascending and Abdominal Aorta Aneurysm Formation in the ApoE−/− Angiotensin II Mouse Model." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80243.

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Aortic aneurysm is a pathological dilatation of the aorta that can be life-threatening when it ruptures. Aneurysms occur throughout the entire aorta but there is a predisposition for the ascending and the abdominal aorta, an observation that cannot be fully explained by the current knowledge of the disease pathophysiology. ApoE −/− mice infused with angiotensin II have recently been reported to develop not only abdominal [1], but also ascending aortic aneurysms [2]. These animals thus provide the perfect model to compare aneurysm progression in both aortic locations and to investigate whether
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Van Doormaal, M. A., X. Zhang, Y. Zhou, D. A. Steinman, and R. M. Henkelman. "In Vivo MRI Versus Ex Vivo CT for Image-Based CFD of the Mouse Aorta." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80118.

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A novel mouse model of surgically induced aortic valve regurgitation (AR) has been developed which leads to altered hemodynamics in the descending thoracic and abdominal aorta, in turn leading to extensive atherosclerotic lesions in these otherwise lesion free areas [1]. Previous work has shown that maps of oscillatory shear index (OSI) and relative residence time (RRT) are consistent with the plaque distribution [2] and plaque severity in AR mice.
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Karim Azer, Haiying Tang, Michael Desiderio, and Haiying Liu. "In Vivo MRI quantification of circumferential wall shear stress in atherosclerotic-prone mouse aorta." In 2007 IEEE 33rd Annual Northeast Bioengineering Conference. IEEE, 2007. http://dx.doi.org/10.1109/nebc.2007.4413344.

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Trachet, Bram, Marjolijn Renard, Gianluca De Santis, et al. "A Quantitative Comparison Between Baseline Hemodynamics and End-Stage Aneurysm Formation in ApoE −/− Mice." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53452.

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The pathogenesis of abdominal aortic aneurysm (AAA) formation still remains debated. Hemodynamics have been suggested to play a (modulating) role, but no follow-up studies have been performed due to (a.o.) a lack of human data before disease initiation. We therefore used an established mouse model of AAA [1] to study whether AAA develops at locations experiencing disturbed flow. We set up a framework to obtain mouse-specific Computational Fluid Dynamics (CFD) simulations of the mouse abdominal aorta, combining: (i) an in vivo assessed geometric model [2] and (ii) in vivo measured boundary cond
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Haskett, Darren, Urs Utzinger, Mohamad Azhar, and Jonathan Vande Geest. "Progressive Alterations in Biomechanical Response of a Mouse Model of Aneurysm." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80321.

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Abdominal aortic aneurysm (AAA) is a complex disease that leads to a localized dilation of the infrarenal aorta, the rupture of which is associated with significant morbidity and mortality, however the underlying mechanisms by which such changes remains an important unanswered question in the literature. Animal models of AAA can be used to study how changes in the microstructural and biomechanical behavior of aortic tissues develop as disease progresses in these animals. We chose here to investigate changes in mechanical characteristics with time in the established Apolipoprotein E deficient (
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Informes sobre el tema "Mouse aorta"

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Granata, Joseph, Hugo Sanchez, Phillip Loeschinger, and Jodi Evans. CD105 Deficiency in Mouse Aorta-Derived Progenitor Cells Promotes an Enhanced Inflammatory Response to Lipopolysaccharide. Journal of Young Investigators, 2018. http://dx.doi.org/10.22186/jyi.35.4.61-66.

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