Literatura académica sobre el tema "Neisseri meningitidis serogroup B"

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Artículos de revistas sobre el tema "Neisseri meningitidis serogroup B"

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Kepenekli Kadayifci, Eda, Deniz Güneşer Merdan, Ahmet Soysal, et al. "Prevalence of Neisseria meningitidis carriage: a small-scale survey in Istanbul, Turkey." Journal of Infection in Developing Countries 10, no. 04 (2016): 413–17. http://dx.doi.org/10.3855/jidc.7483.

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Introduction: The human nasopharynx is the main reservoir of Neisseria meningitidis, and asymptomatic carriage is common. N. meningitidis one of the common causes of bacterial meningitis in Turkey, especially after the implementation of the national immunization program that includes conjugated pneumococcal and Haemophilus influenzae type b vaccines. The purpose of this study was to evaluate the prevalence of meningococcal carriage and determine the leading serogroup, which may help authorities to adapt appropriate meningococal vaccine into the national immunization programme. Methodology: The
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HILSE, R., J. STOEVESANDT, D. A. CAUGANT, H. CLAUS, M. FROSCH, and U. VOGEL. "Distribution of the meningococcal insertion sequence IS1301 in clonal lineages of Neisseria meningitidis." Epidemiology and Infection 124, no. 2 (2000): 337–40. http://dx.doi.org/10.1017/s0950268899003647.

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The distribution of the meningococcal insertion sequence IS1301 was analysed in 496 strains of different serogroups and clonal lineages of Neisseria meningitidis, and in 64 neisserial strains other than N. meningitidis. IS1301 was found in meningococci, but not in apathogenic Neisseria sp. and Neisseria gonorrhoeae. The copy numbers of IS1301 varied between 2 and 17 per genome. IS1301 positive strains were mostly found among the serogroups 29E, W135, X, and Y. Clonal lineages of serogroup A, B, and C meningococci associated with epidemic meningococcal disease were rarely positive for IS1301.
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Kourna Hama, Mamadou, Dam Khan, Boubou Laouali, et al. "Pediatric Bacterial Meningitis Surveillance in Niger: Increased Importance of Neisseria meningitidis Serogroup C, and a Decrease in Streptococcus pneumoniae Following 13-Valent Pneumococcal Conjugate Vaccine Introduction." Clinical Infectious Diseases 69, Supplement_2 (2019): S133—S139. http://dx.doi.org/10.1093/cid/ciz598.

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Abstract Background Meningitis is endemic in Niger. Haemophilus influenzae type b (Hib) vaccine and the 13-valent pneumococcal conjugate vaccine (PCV13) were introduced in 2008 and 2014, respectively. Vaccination campaign against Neisseria meningitidis serogroup A was carried out in 2010–2011. We evaluated changes in pathogen distribution using data from hospital-based surveillance in Niger from 2010 through 2016. Methods Cerebrospinal fluid (CSF) specimens from children <5 years old with suspected meningitis were tested to detect vaccine-preventable bacterial pathogens. Confirmatory identi
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Wang, Quan, Zhujun Shao, Xiaoting Wang, et al. "Genetic Study of Capsular Switching between Neisseria meningitidis Sequence Type 7 Serogroup A and C Strains." Infection and Immunity 78, no. 9 (2010): 3883–88. http://dx.doi.org/10.1128/iai.00363-10.

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ABSTRACT Neisseria meningitidis is a leading cause of septicemia and meningitis worldwide. N. meningitidis capsular polysaccharides have been classified into 13 distinct serogroups which are defined by antibody reactivity and structural analysis, and the capsule plays an important role in virulence. Serogroups A, B, C, W135, and Y have been reported to be clinically important. Several newly identified serogroup C isolates belonging to the unique sequence type 7 (ST-7) were identified in China. Since most ST-7 isolates from China belonged to serogroup A, the newly identified ST-7 serogroup C st
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Abady, N. R., C. J. D. Guglielmino, R. M. Graham, et al. "Genetic characterization of a Neisseria meningitidis cluster in Queensland, Australia." Canadian Journal of Microbiology 63, no. 7 (2017): 644–47. http://dx.doi.org/10.1139/cjm-2017-0017.

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Neisseria meningitidis serogroups B and C have been responsible for the majority of invasive meningococcal disease in Australia, with serogroup B strains causing an increasing proportion of cases in recent years. Serogroup Y has typically caused sporadic disease in Australia. In 2002, a cluster of 4 cases was reported from a rural region in Queensland. Three of these cases were serogroup C, with 1 case diagnosed by molecular detection only, and the fourth case was identified as a serogroup Y infection. Genomic analysis, including antigen finetyping, multilocus sequence typing (MLST), and core
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Li, Yanwen, Yao-hui Sun, Cathy Ison, Myron M. Levine, and Christoph M. Tang. "Vaccination with Attenuated Neisseria meningitidis Strains Protects against Challenge with Live Meningococci." Infection and Immunity 72, no. 1 (2004): 345–51. http://dx.doi.org/10.1128/iai.72.1.345-351.2004.

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ABSTRACT Meningococcal disease is a life-threatening infection caused by Neisseria meningitidis. Currently, there are no vaccines to prevent infection with serogroup B N. meningitidis strains, the leading cause of meningococcal meningitis in Europe and North America. Here we describe the construction and characterization of two attenuated serogroup B N. meningitidis strains, YH102 (MC58Δsia ΔrfaF) and YH103 (MC58Δsia ΔmetH). Both strains are markedly attenuated in their capacity to cause bacteremia in rodent models and have a reduced ability to survive in a human whole-blood assay. Immunizatio
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7

Stabler, Richard A., Gemma L. Marsden, Adam A. Witney, et al. "Identification of pathogen-specific genes through microarray analysis of pathogenic and commensal Neisseria species." Microbiology 151, no. 9 (2005): 2907–22. http://dx.doi.org/10.1099/mic.0.28099-0.

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The release of the complete genome sequences of Neisseria meningitidis MC58 and Z2491 along with access to the sequences of N. meningitidis FAM18 and Neisseria gonorrhoeae FA1090 allowed the construction of a pan-Neisseria microarray, with every gene in all four genomes represented. The microarray was used to analyse a selection of strains including all N. meningitidis serogroups and commensal Neisseria species. For each strain, genes were defined as present, divergent or absent using gack analysis software. Comparison of the strains identified genes that were conserved within N. meningitidis
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Portilho, Amanda Izeli, Gabriela Trzewikoswki de Lima, and Elizabeth De Gaspari. "Neisseria meningitidis: analysis of pili and LPS in emerging Brazilian strains." Therapeutic Advances in Vaccines and Immunotherapy 8 (January 2020): 251513552091919. http://dx.doi.org/10.1177/2515135520919195.

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Background: Neisseria meningitidis is the main cause of bacterial meningitis in Brazil, where the main serogroups isolated are B and C; however, the serogroup W has recently emerged. LPS and type IV pili are important virulence factors that increase meningococci pathogenicity. Methods: The characterization of Lipopolysaccharide (LPS) and type IV pili in 19 meningococci strains of serogroup B, 21 of serogroup C, 45 of serogroup W and 28 of serogroup Y, isolated in Brazil between 2011 and 2017, was conducted using the Enzyme-linked Immunosorbent Assay (Dot- ELISA) technique and monoclonal antibo
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Ceyhan, Mehmet, Yasemin Ozsurekci, Cihangül Bayhan, et al. "682. The Changing Epidemiology of Bacterial Meningitis During 2015–2017 in Turkey: A Hospital-Based Prospective Surveillance Study." Open Forum Infectious Diseases 5, suppl_1 (2018): S246. http://dx.doi.org/10.1093/ofid/ofy210.689.

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Abstract Background The etiology of bacterial meningitis in Turkey has been changed after the implementation of conjugated vaccines against Streptococcus pneumonia and Haemophilus influenzae type b (Hib) in Turkish national immunization schedule. Methods. This prospective study was conducted in 25 hospitals located seven regions of Turkey (representing 30% of Turkey population) and children aged between 1 month and 18 years with suspected meningitis and hospitalized were included. Cerebrospinal fluid samples were collected and bacterial identification was made according to the multiplex PCR as
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Caesar, Nicole M., Kenneth A. Myers, and Xin Fan. "Neisseria meningitidis serogroup B vaccine development." Microbial Pathogenesis 57 (April 2013): 33–40. http://dx.doi.org/10.1016/j.micpath.2013.02.003.

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