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Artículos de revistas sobre el tema "Neovascularisation"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 25 de julio de 2025

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1

Nazm, Nazneen, Nadeem, and Madhura Mukherjee. "To study the extent of regression of retinal neovascularisation in proliferative diabetic retinopathy following laser photocoagulation of the ischaemic retina." Indian Journal of Clinical and Experimental Ophthalmology 11, no. 1 (2025): 24–30. https://doi.org/10.18231/j.ijceo.2025.005.

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Eyes with proliferative diabetic retinopathy have a variable response to treatment with panretinal photocoagulation (PRP) or anti–vascular endothelial growth factor agents. We undertook this study to find out what is the exact extent of regression of neovascularisation with PRP and what are the factors that correlate with this variation in response. Present Prospective Study was conducted in Subjects with Proliferative Diabetic Retinopathy attending the ophthalmology department at ESI-PGIMSR and Model Hospital, Basaidarapur, New Delhi from October 2019-April 2021. During the above-mentioned pe

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2

Creton, D. "Neovascularisation." Phlebologie 37, no. 03 (2008): 134–41. http://dx.doi.org/10.1055/s-0037-1622223.

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SummaryThere are different causes of neovascularisation: vascular endothelial growth factor, inflammatory process, hypertrophy of pre-existant vessels (lymph node veins), haematomas revascularisation, and high difference in pressure. The latter 3 depend directly on the way the operation is performed hence on the surgeon.Concerning the primary varices patch saphenoplasty do not abolish neovascularisation. It is of first importance to perform a flush ligation only in case of incontinent terminal and pre-terminal valves. In that case an incision as small as possible must be carried out with minim

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3

Holmgren, L. "Malignant Neovascularisation." European Journal of Ultrasound 6 (November 1997): S11. http://dx.doi.org/10.1016/s0929-8266(97)87215-1.

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4

Smith, R. "Neovascularisation again." British Journal of Ophthalmology 74, no. 6 (1990): 323. http://dx.doi.org/10.1136/bjo.74.6.323.

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5

Badal, Josep, Luis Amselem, Ricardo Aleman, and Frederic Huste. "Anti–vascular Endothelial Growth Factor Therapy for Myopic Choroidal Neovascularisation." European Ophthalmic Review 07, no. 02 (2013): 84. http://dx.doi.org/10.17925/eor.2013.07.02.84.

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Pathological myopia represents the most common cause of choroidal neovascularisation in young patients. Its natural course has a devastating prognosis. Several treatments have been assessed, but photodynamic therapy is currently the only approved treatment for subfoveal choroidal neovascularisation related to pathological myopia. Anti-vascular endothelial growth factor therapy has demonstrated promising results in any form and localisation of choroidal neovascularisation, although there is an absence of data obtained from randomised clinical trials. The aim of this article is to compare differ

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6

Shehadeh, Mohammad M., Mohammad T. Akkawi, Vasilios F. Diakonis, Ammar A. Aghbar, and Abdelraheem Abu Shanab. "Extensive Corneal Neovascularisation Treatment by Ultraviolet Corneal Collagen Crosslinking." European Ophthalmic Review 11, no. 01 (2017): 62. http://dx.doi.org/10.17925/eor.2017.11.01.62.

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The purpose of this article is to report a case of extensive corneal neovascularisation that was treated by ultraviolet corneal collagen crosslinking (CXL). The case report is about a 24-year-old man who was referred to the cornea clinic with a case of keratitis. He was treated with topical antibiotics. After full resolution of keratitis, his condition was complicated by extensive corneal neovascularisation. A trial of photochemical corneal collagen CXL with riboflavin/ultraviolet A resulted in a dramatic improvement and resolution of the corneal neovascularisation. Thus, we can conclude that

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7

Chan, Elsie, Jennifer Fan Gaskin, and Elsa C. Chan. "Corneal Neovascularisation and Anti-VEGF Therapy." Targets 3, no. 1 (2025): 9. https://doi.org/10.3390/targets3010009.

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Cornea vascularisation is a significant cause of ocular morbidity. Disease or injury often triggers the development of new blood vessels in the cornea, compromising its clarity and impairing vision. Common causes of corneal neovascularisation include infections, chemical burns, and local and systemic inflammatory disorders. Topical corticosteroid eye drops remain the standard therapy; however, extended use of corticosteroids has been known to cause side-effects including cataracts and raised intraocular pressure. As such, an alternative therapy has been actively sought. Vascular endothelial gr

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8

Imre, G. "Lactic acid neovascularisation." British Journal of Ophthalmology 75, no. 4 (1991): 254. http://dx.doi.org/10.1136/bjo.75.4.254.

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9

Cheng, Jacob YC, Doric WK Wong, and Chong Lye Ang. "Intraocular Avastin (Bevacizumab) for Neovascularisation of the Iris and Neovascular Glaucoma." Annals of the Academy of Medicine, Singapore 37, no. 1 (2008): 72–74. http://dx.doi.org/10.47102/annals-acadmedsg.v37n1p72.

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Introduction: The aim of this study was to determine the effectiveness of intraocular injections of bevacizumab for neovascularisation of the iris and neovascular glaucoma. Clinical Picture: Three patients with neovascularisation of the iris due to various causes were recruited. Treatment: Patients were treated with intraocular bevacizumab. Outcome: Neovascularisation of the iris was noted to have completely regressed as early as 3 days after the injection and in all the patients (100%) within 8 days after injection. They were followed up for at least 1 month with no clinical evidence of recur

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10

Buragohain, Suklengmung, Harsha Bhattacharjee, and Henal Javeri. "Capsular neovascularisation in uveitis." Indian Journal of Ophthalmology - Case Reports 1, no. 3 (2021): 458. http://dx.doi.org/10.4103/ijo.ijo_3625_20.

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11

Faraj, L. A., D. G. Said, and H. S. Dua. "Evaluation of corneal neovascularisation." British Journal of Ophthalmology 95, no. 10 (2011): 1343–44. http://dx.doi.org/10.1136/bjophthalmol-2011-300856.

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12

Stavrou, D. "Neovascularisation in wound healing." Journal of Wound Care 17, no. 7 (2008): 298–302. http://dx.doi.org/10.12968/jowc.2008.17.7.30521.

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13

Sunness, J. S. "Choroidal neovascularisation and atrophy." British Journal of Ophthalmology 90, no. 4 (2006): 398–99. http://dx.doi.org/10.1136/bjo.2005.084830.

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14

Sivaprasad, S., and A. T. Moore. "Choroidal neovascularisation in children." British Journal of Ophthalmology 92, no. 4 (2008): 451–54. http://dx.doi.org/10.1136/bjo.2007.124586.

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15

Binns, Malcolm, and Robert W. H. Pho. "Neovascularisation of the epiphysis." Microsurgery 12, no. 1 (1991): 9–13. http://dx.doi.org/10.1002/micr.1920120103.

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16

Carrozzo, Alessandro, Jobe Shatrov, Abdo El Helou, et al. "Ultrasound-guided electrocoagulation of neovascularisation for persistent patellar tendinopathy in athletes: a cohort study of 25 patients with a mean follow-up of 5 years from the SANTI Study Group." BMJ Open Sport & Exercise Medicine 10, no. 1 (2024): e001900. http://dx.doi.org/10.1136/bmjsem-2024-001900.

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BackgroundPatellar tendinopathy (PT) is a common condition characterised by persistent patellar tendon pain and dysfunction, particularly in athletes. Neovascularisation is frequently observed in the PT and is associated with increased pain. Ultrasound-guided electrocoagulation of neovascularisation has emerged as a minimally invasive alternative treatment for recalcitrant PT.Hypothesis/purposeThe purpose of this study was to evaluate the clinical outcomes of ultrasound-guided electrocoagulation of neovascularisation in athletes with persistent PT.Study designCase series; level of evidence, IV

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17

Wali, Keerti, Shilpa Maled, Ronak Solanki, Apoorva G. Ayachit, and Aniket N. Shastri. "Safety and efficacy of frequency-doubled Nd-YAG laser photocoagulation of different types of corneal neovascularisation (NLPC): a prospective study." BMJ Open Ophthalmology 10, no. 1 (2025): e001734. https://doi.org/10.1136/bmjophth-2024-001734.

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PurposeFrequency doubling of neodymium-doped yttrium aluminum garnet (Nd-YAG) laser results in emission of photocoagulating 532 nm light compared with photolytic 1064 nm emission. The ergonomic benefits of solid-state lasers led to replacement of older coagulating lasers in ophthalmic centres by frequency-doubled Nd-YAG laser. Our study aims to evaluate safety and efficacy of frequency-doubled Nd-YAG laser for photocoagulation of corneal neovascularisation (NLPC).Methods30 quiet eyes of 28 patients with superficial, mid-stromal and deep stromal inactive corneal neovascularisation were subjecte

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18

Bobryshev, Y. V., A. E. Farnsworth, and R. S. A. Lord. "Expression of Vascular Endothelial Growth Factor in Aortocoronary Saphenous Vein Bypass Grafts." Cardiovascular Surgery 9, no. 5 (2001): 492–98. http://dx.doi.org/10.1177/096721090100900515.

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Neovascularisation is a prominent feature of long-term aortocoronary saphenous vein bypass grafts but mechanisms involved in the formation of neovessels have not been previously studied. Vascular Endothelial Growth Factor (VEGF) is an important angiogenic factor that induces migration and proliferation of endothelial cells, enhances permeability and modulates thrombogenecity. This study investigated the expression of VEGF in aortocoronary saphenous vein bypass grafts. Aortocoronary saphenous vein bypass grafts with angiographic luminal stenosis of >75% were explanted from 14 patients at red

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19

Bainbridge, James W., Vanya Loroch, Eric Viaud, and Claus Cursiefen. "Beyond Anti-VEGFs – Anti-Insulin Receptor Substrate-1 Oligonucleotides as a Novel Approach to Ocular Neovascular Disorders." European Ophthalmic Review 06, no. 03 (2012): 190. http://dx.doi.org/10.17925/eor.2012.06.03.190.

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Angiogenesis is a complex process that is vital to health but is also a driving factor behind a broad range of malignant, ischaemic, inflammatory, infectious and immune disorders. For optimal efficacy and safety, therapies aimed at preventing angiogenic-mediated disorders must differentiate between healthy and pathological angiogenesis or neovascularisation. Aganirsen is an antisense oligonucleotide that inhibits the insulin receptor substrate (IRS)-1 angiogenic pathway by targeting the IRS-1 messenger RNA. To date, studies of aganirsen have focused mainly on ocular disorders because of the ab

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20

Diamond, Michael Adam, Sze Wah Samuel Chan, Xun Zhou, et al. "Lymphatic vessels identified in failed corneal transplants with neovascularisation." British Journal of Ophthalmology 103, no. 3 (2018): 421–27. http://dx.doi.org/10.1136/bjophthalmol-2018-312630.

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BackgroundCorneal transplant failure with neovascularisation is a leading indication for full-thickness grafts in patients. Lymphangiogenesis is implicated in the pathology of graft failure, and here we systematically evaluate failed human corneal transplants with neovascularisation for the presence of lymphatic vessels.MethodsNine failed grafts with neovascularisation, based on H&E staining with subsequent immunoperoxidase staining for CD31, a blood vessel marker, were selected. Lymphatics were investigated by immunohistochemical and immunofluorescence approaches using podoplanin as a lym

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21

Oki, Aqsa Sjuhada, Moch Febi Alviansyah, Christian Khoswanto, Retno Pudji Rahayu, and Muhammad Luthfi. "Acceleration of post-tooth extraction socket healing after continuous aerobic and anaerobic physical exercise in Wistar rats (Rattus norvegicus)." Dental Journal (Majalah Kedokteran Gigi) 53, no. 4 (2020): 196. http://dx.doi.org/10.20473/j.djmkg.v53.i4.p196-200.

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Background: Physical exercise has been proven to accelerate wound healing. Physical training itself consists of aerobic (continuous training) and anaerobic (interval training) exercise. The effectiveness of continuous physical exercise on post-tooth extraction wound healing is the focus of this study. Purpose: This study aims to investigate the differences in post-tooth extraction wound healing in Wistar rats (Rattus norvegicus) after aerobic and anaerobic exercise based on the number of fibroblasts and neovascularisation. Methods: Wistar rats were divided into three groups: the control group

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22

Holló, Gábor. "Diabetic neovascularisation and secondary glaucoma." Orvosi Hetilap 152, no. 29 (2011): 1167–70. http://dx.doi.org/10.1556/oh.2011.29042.

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Neovascular glaucoma develops from intraocular neovascularisation in diabetes mellitus. Neovascularisation is a consequence of hypoxia-induced production of vascular endothelial growth factor-A. Neovascular glaucoma is one of the most serious, sight-threatening late complications of diabetes. Several intraocular pressure lowering drugs, surgical and adjunctive laser treatments have been used to treat this disease, but the efficacy of the interventions is limited. The role of vascular endothelial growth factor-A blocking therapy in the treatment of neovascular glaucoma remains to be determined.

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23

Augustin, Albert J., and Indre Offermann. "Combination Therapy for Choroidal Neovascularisation." Drugs & Aging 24, no. 12 (2007): 979–90. http://dx.doi.org/10.2165/00002512-200724120-00002.

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24

Cheung, C. M. G., O. M. Durrani, and P. Stavrou. "Peripapillary choroidal neovascularisation in sarcoidosis." Ocular Immunology and Inflammation 10, no. 1 (2002): 69–73. http://dx.doi.org/10.1076/ocii.10.1.69.10324.

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25

Groeneveld, Erwin, and Paul Eliadis. "Interferon for subfoveal choroidal neovascularisation." Australian and New Zealand Journal of Ophthalmology 21, no. 2 (1993): 133–34. http://dx.doi.org/10.1111/j.1442-9071.1993.tb00769.x.

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26

Kaplan, H. "Submacular surgery for choroidal neovascularisation." British Journal of Ophthalmology 80, no. 2 (1996): 101. http://dx.doi.org/10.1136/bjo.80.2.101.

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27

Lee, M. S. "Choroidal neovascularisation associated with meningioma." British Journal of Ophthalmology 89, no. 10 (2005): 1384–85. http://dx.doi.org/10.1136/bjo.2005.071696.

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28

Postelmans, L., P. Coquelet, C. Verougstraete, and F. Willermain. "Treatment of peripapillary choroidal neovascularisation." British Journal of Ophthalmology 92, no. 5 (2008): 721–22. http://dx.doi.org/10.1136/bjo.2007.135095.

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29

Yip, Vivien C., Leonard W. Yip, and Augustinus Laude. "Subconjunctival bevacizumab for iris neovascularisation." Lancet Diabetes & Endocrinology 2, no. 6 (2014): 449–50. http://dx.doi.org/10.1016/s2213-8587(14)70070-1.

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30

Cann, StephenA, JohannesP van Netten, TearaL Ashby, MichaelJ Ashwood-Smith, and NicholasG van der Westhuizen. "Role of lymphagenesis in neovascularisation." Lancet 346, no. 8979 (1995): 903. http://dx.doi.org/10.1016/s0140-6736(95)92744-1.

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31

Rowson, Neil J., John K. G. Dart, and Roger J. Buckley. "Corneal neovascularisation in acute hydrops." Eye 6, no. 4 (1992): 404–6. http://dx.doi.org/10.1038/eye.1992.83.

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32

Graham, E. M., M. R. Stanford, J. S. Shilling, and M. D. Sanders. "Neovascularisation associated with posterior uveitis." British Journal of Ophthalmology 71, no. 11 (1987): 826–33. http://dx.doi.org/10.1136/bjo.71.11.826.

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33

Ohnishi, Yoshitaka, Tatsuro Ishibashi, and Takuji Sagawa. "Fluorescein gonioangiography in diabetic neovascularisation." Graefe's Archive for Clinical and Experimental Ophthalmology 232, no. 4 (1994): 199–204. http://dx.doi.org/10.1007/bf00184005.

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34

Hummel, T., P. Burger, N. Frings, et al. "High ligation of the saphenofemoral junction is necessary!" Phlebologie 38, no. 03 (2009): 99–102. http://dx.doi.org/10.1055/s-0037-1622261.

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SummaryNeovascularisation can compromise the success of high ligation and resection of the greater saphenous vein. Studies using duplexultrasound to classify recurrent groin veins have described rates of neovascularisation as high as 60% and raised the question whether high ligation is actually able to prevent groin recurrences. In the present study, recurrent groin veins were excised and examined histologically in order to prove whether neovascularisation is the main cause for sapheno-femoral recurrences. Patients, methods: 419 patients accounting for 458 legs with clinically symptomatic groi

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35

De Maeseneer, M. G., I. F. Tielliu, P. E. Van Schil, S. G. De Hert, and E. J. Eyskens. "Clinical Relevance of Neovascularisation on Duplex Ultrasound in the Long-Term Follow-up after Varicose Vein Operation." Phlebology: The Journal of Venous Disease 14, no. 3 (1999): 118–22. http://dx.doi.org/10.1177/026835559901400306.

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Objective: To evaluate the clinical relevance of neovascularisation at the saphenous ligation site. Design: Long-term follow-up after previous varicose vein surgery in a single patient group. Setting: Vascular clinic of a university hospital. Patients: Eighty-two patients (106 limbs) with a mean follow-up period of 56 months after correct saphenous ligation were submitted to duplex scanning. Intervention: Clinical assessment and colour duplex scanning of all the operated limbs. Reintervention in 15 limbs with perioperative evaluation of recurrent veins. Main outcome measures: Limbs with and wi

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36

Haj Najeeb, Bilal, Gabor G. Deak, Ursula Margarethe Schmidt-Erfurth, and Bianca S. Gerendas. "RAP study, report 1: novel subtype of macular neovascularisation type III, cilioretinal MNV3." British Journal of Ophthalmology 105, no. 1 (2020): 113–17. http://dx.doi.org/10.1136/bjophthalmol-2019-315311.

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PurposeTo report on patients with macular neovascularisation type III (MNV3) arising from cilioretinal arteries (CRAs) (cilioretinal macular neovascularisation type III (cMNV3)).MethodsWe reviewed baseline examinations of patients with neovascular age-related macular degeneration using multimodal imaging. We determined the type and distribution of MNV lesions in each cMNV3 case, the range of distances from the fovea, existence of exudative maculopathy, intraretinal haemorrhage and other morphological characteristics. 50 consecutive eyes with usual MNV3 without CRA were included as a control gr

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37

Dervenis, Nikolaos, Panagiotis Dervenis, and Eleftherios Agorogiannis. "Neovascular age-related macular degeneration: disease pathogenesis and current state of molecular biomarkers predicting treatment response—a scoping review." BMJ Open Ophthalmology 9, no. 1 (2024): e001516. http://dx.doi.org/10.1136/bmjophth-2023-001516.

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Age-related macular degeneration is a major cause of blindness, and the development of anti-vascular endothelial growth factor (VEGF) intravitreal treatments has revolutionised the management of the disease. At the same time, new challenges and unmet needs arose due to the limitations of the current therapeutic options. Neovascularisation development during the course of the disease has a complex pathogenetic mechanism, and several biomarkers and their association with treatment outcomes have been investigated. We reviewed the relevant literature about neovascularisation development and biomar

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38

&NA;. "Changing times for intraocular neovascularisation, oedema." Inpharma Weekly &NA;, no. 1654 (2008): 9. http://dx.doi.org/10.2165/00128413-200816540-00015.

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39

Tuncel, Alper, Merdan Serin, and Mehmet Bayramicli. "Venous component in scar penetrating neovascularisation." Journal of Plastic Surgery and Hand Surgery 52, no. 1 (2017): 47–52. http://dx.doi.org/10.1080/2000656x.2017.1319848.

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40

Lee, D. K. "Serpiginous choroidopathy presenting as choroidal neovascularisation." British Journal of Ophthalmology 87, no. 9 (2003): 1184–85. http://dx.doi.org/10.1136/bjo.87.9.1184.

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41

Berger, Lisa, and Bernhard M. Stoffelns. "Photodynamic Therapy in Subfoveal Choroidal Neovascularisation." Medical Laser Application 17, no. 4 (2002): 331–40. http://dx.doi.org/10.1078/1615-1615-00078.

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42

AI-Husainy, S., P. Stavrou, and M. W. Hope-Ross. "Alkali injury complicated by choroidal neovascularisation." Eye 14, no. 6 (2000): 904–5. http://dx.doi.org/10.1038/eye.2000.247.

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43

Mehta, J. S., A. S. Jacks, V. Maurino, and A. M. P. Hamilton. "Neovascularisation in a patent chorioretinal anastomosis." Eye 14, no. 6 (2000): 916–18. http://dx.doi.org/10.1038/eye.2000.256.

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44

Menzel-Severing, J. "Emerging techniques to treat corneal neovascularisation." Eye 26, no. 1 (2011): 2–12. http://dx.doi.org/10.1038/eye.2011.246.

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45

McLEOD, D. "Entry site neovascularisation after diabetic vitrectomy." British Journal of Ophthalmology 84, no. 8 (2000): 810–12. http://dx.doi.org/10.1136/bjo.84.8.810.

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46

Lee, J. "Preretinal neovascularisation associated with choroidal melanoma." British Journal of Ophthalmology 85, no. 11 (2001): 1309–12. http://dx.doi.org/10.1136/bjo.85.11.1309.

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47

Aisenbrey, S., F. Gelisken, P. Szurman, and K. U. Bartz-Schmidt. "Surgical treatment of peripapillary choroidal neovascularisation." British Journal of Ophthalmology 91, no. 8 (2007): 1027–30. http://dx.doi.org/10.1136/bjo.2006.108118.

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48

Binder, S. "Surgical treatment of peripapillary choroidal neovascularisation." British Journal of Ophthalmology 91, no. 8 (2007): 990–91. http://dx.doi.org/10.1136/bjo.2007.114009.

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49

Bradbury, Jane. "Integrin antagonists may prevent retinal neovascularisation." Lancet 347, no. 9010 (1996): 1249. http://dx.doi.org/10.1016/s0140-6736(96)90751-5.

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50

Gupta, B., S. Parvizi, and M. D. Mohamed. "Bietti crystalline dystrophy and choroidal neovascularisation." International Ophthalmology 31, no. 1 (2010): 59–61. http://dx.doi.org/10.1007/s10792-010-9406-8.

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